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Artykuły w czasopismach na temat "Heart Effect of drugs on"
Farrell, A. P., i M. S. Graham. "Effects of adrenergic drugs on the coronary circulation of Atlantic salmon (Salmo salar)". Canadian Journal of Zoology 64, nr 2 (1.02.1986): 481–84. http://dx.doi.org/10.1139/z86-071.
Pełny tekst źródłaRorabaugh, Boyd R., Sarah L. Seeley, Albert D. Bui, Lisanne Sprague i Manoranjan S. D'Souza. "Prenatal methamphetamine differentially alters myocardial sensitivity to ischemic injury in male and female adult hearts". American Journal of Physiology-Heart and Circulatory Physiology 310, nr 4 (15.02.2016): H516—H523. http://dx.doi.org/10.1152/ajpheart.00642.2015.
Pełny tekst źródłaNordin, Henrik, Anders M. Galløe, Søren D. Ladefoged i Jørn Badskjær. "The effects of propranolol and verapamil on hyperthyroid heart symptoms and function, assessed by systolic time intervals". Acta Endocrinologica 128, nr 4 (kwiecień 1993): 297–300. http://dx.doi.org/10.1530/acta.0.1280297.
Pełny tekst źródłaHaverkamp, W. "Heart rate effects of antimuscarinic drugs". International Urology and Nephrology 51, nr 10 (27.07.2019): 1783–84. http://dx.doi.org/10.1007/s11255-019-02239-6.
Pełny tekst źródłaFrigy, Attila, Márta Germán-Salló, Lehel Máthé i Mónika Szabó. "Vércukorcsökkentő gyógyszerek biztonságossága szívelégtelenségben". Orvosi Hetilap 158, nr 5 (luty 2017): 163–71. http://dx.doi.org/10.1556/650.2017.30652.
Pełny tekst źródłaBakhteev, R. R. "Effect of retabolil on hyperuricemia in chronic heart failure". Kazan medical journal 68, nr 2 (15.04.1987): 102–3. http://dx.doi.org/10.17816/kazmj96023.
Pełny tekst źródłaGirouard, Catherine, Jean-Pierre Grégoire, Paul Poirier i Jocelyne Moisan. "Effect of contraindicated drugs for heart failure on hospitalization among seniors with heart failure". Medicine 96, nr 9 (marzec 2017): e6239. http://dx.doi.org/10.1097/md.0000000000006239.
Pełny tekst źródłaGatzov, Plamen. "New glucose-lowering drugs in patients with heart failure". Bulgarian Cardiology 27, nr 2 (21.07.2021): 60–64. http://dx.doi.org/10.3897/bgcardio.27.e70144.
Pełny tekst źródłaFehér, Gergely, i Gabriella Pusch. "Role of antihypertensive drugs in the treatment of migraine". Orvosi Hetilap 156, nr 5 (luty 2015): 179–85. http://dx.doi.org/10.1556/oh.2015.30056.
Pełny tekst źródłaBenfey, B. G. "Antifibrillatory effects of α1-adrenoceptor blocking drugs in experimental coronary artery occlusion and reperfusion". Canadian Journal of Physiology and Pharmacology 71, nr 2 (1.02.1993): 103–11. http://dx.doi.org/10.1139/y93-015.
Pełny tekst źródłaRozprawy doktorskie na temat "Heart Effect of drugs on"
Zeitz, Christopher John. "Acute drug effects on the heart-haemodynamic, pharmacologic and metabolic correlations". Title page, contents and abstract only, 1999. http://web4.library.adelaide.edu.au/theses/09PH/09phz48.pdf.
Pełny tekst źródłaMaune, Jerene Mary 1953. "THE EFFECT OF CAFFEINE ON HEART RATE, RHYTHM AND BLOOD PRESSURE". Thesis, The University of Arizona, 1986. http://hdl.handle.net/10150/276372.
Pełny tekst źródłaRamadan, Waile Rushing Ann E. Hartberg W. Keith. "The protective effect of [alpha]-lipoic acid in doxorubicin induced cardiotoxicity in rats". Waco, Tex. : Baylor University, 2008. http://hdl.handle.net/2104/5218.
Pełny tekst źródłaLi, Y. M. J. "An in-vivo study of the anti-arrhythmic and electrophysiological effects of amiodarone, lignocaine and penticainide (CM7857) in the rat". Thesis, De Montfort University, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.233821.
Pełny tekst źródłaHuang, Yi Fei. "Myocardial pharmacokinetics and pharmacodynamics in the sheep /". Title page, contents and abstract only, 1991. http://web4.library.adelaide.edu.au/theses/09PH/09phh8742.pdf.
Pełny tekst źródłaAccompanying diskette contains data of Chapters 3, 4, 5, 7, 8 and 9 (ASCII). Includes bibliographical references (leaves 177-207).
Ritchie, Rebecca Helen. "Acute haemodynamic effects of three cardioactive agents: metoprolol, sotalol and milrinone : influence of myocardial content and systolic interval /". Title page, table of contents and summary only, 1994. http://web4.library.adelaide.edu.au/theses/09PH/09phr611.pdf.
Pełny tekst źródłaShubat, Pamela Jane. "Monocrotaline toxicity and pulmonary arteries". Diss., The University of Arizona, 1988. http://hdl.handle.net/10150/184533.
Pełny tekst źródłaRossouw, Ellen. "The effect of androgenic anabolic steroids on the susceptibility of the rat heart to ischaemia and reperfusion injury". Thesis, Stellenbosch : Stellenbosch University, 2002. http://hdl.handle.net/10019.1/53105.
Pełny tekst źródłaENGLISH ABSTRACT: Background: Athletes use androgenic anabolic steroids (AAS) to enhance their physical performance. The abuse of AAS is however associated with a host of side effects including sudden death due to cardiac arrest. The use of AAS leads to myocardial hypertrophy, which possibly makes the heart more prone to ischaemia/reperfusion injury, since it often develops in the absence of proper vasculature development. Chronic AAS use also disrupts myocardial p-adrenoreceptor function and possibly cAMP, signalling in the heart. Drugs increasing cAMP and decreasing cGMP levels in the ischaemic myocardium exacerbate myocardial ischaemia/reperfusion injury. We also know that AAS causes coronary artery disease secondary to the deleterious alteration of lipid profiles by increasing the LOL cholesterol and decreasing the HOLcholesterol levels. AAS treatment may increase systemic TNFa levels by stimulating lymphocyte TNFa secretion that has been implicated in the depression of myocardial function, myocardial hypertrophy and the worsening of ischaemia/reperfsuion injury. Aims: To determine whether chronic AAS treatment in trained and untrained rats influences: 1) heart function and susceptibility to ischaemia/reperfusion injury, 2) myocardial cyclic nucleotide levels (cAMP and cGMP) and 3) myocardial TNFa levels. Material and methods: Male Sprague-Dawley rats (n=100) were divided into 4 groups: sedentary vehicle (placebo) treated group, sedentary AAS treated group, exercise vehicle (placebo) treated group, and exercise AAS treated group. Steroid treated animals received an intramuscular injection of nandrolone laureate (0.375 mg/kg) once a week, for six weeks. Training consisted of swim sessions 6 days a week for 6 weeks. Swim time was incrementally increased up to a maximum of 50 minutes a day. For biometric parameters heart weight and body weight were documented. Hearts were mounted on a l.anqendorff perfusion apparatus and left ventricular developed pressure (LVDP), heart rate (HR) and coronary flow (CF) was monitored. The hearts were subjected to a period of 20 minutes of global ischaemia, followed by 30 minutes of reperfusion. Functional parameters was again monitored and documented. For biochemical analysis, blood was collected for the determination of serum lipid levels and myocardial tissue samples were collected before, during and after ischaemia for the determination of myocardial TNFa, cGMP and cAMP levels and p38 activity. Conclusions: Results obtained would suggest that AAS exacerbate exercise induced myocardial hypertrophy. It also prevents the exercise-induced improvement in cardiac function. AAS use reduces reperfusion function in treated hearts, which may suggest that AAS exacerbates ischaemie and reperfusion injury. Furthermore it was seen that AAS elevates basal (preischaemie) cyclic nucleotide levels and basal (pre-ischaemic) as well as reperfusion TNFa levels. This may also contribute to the exacerbation of ischaemic and reperfusion injury.
AFRIKAANSE OPSOMMING: Agtergrond: Androgeniese anaboliese steroïede (AAS) word dikwels deur atlete gebruik om sportprestasie te verbeter. Die misbruik van AAS het egter talle newe effekte, insluitende skielike dood wat gewoonlik toegeskryf word aan hartaanvalle. Die gebruik van AAS lei onder andere tot miokardiale hipertrofie wat opsigself, as gevolg van ontoereikende vaskulêre ontwikkeling tydens die ontwikkeling van hipertrofie, die hart nog meer vatbaar vir isgemie/herperfusie skade maak. Kroniese AAS toediening versteur miokardiale beta-adtenoresepter funksie en moontlik die tweede boodskapper, sAMP, seintransduksie in die hart. Ons weet ook dat AAS koronêre hartvatsiektes veroorsaak. Laasgenoemde is sekondêr tot die nadelige lipiedprofiel verandering, wat 'n verhoging in LDL-C en 'n verlaging in HDL-C insluit. Middels wat miokardiale sAMP vlakke verhoog en sGMP vlakke in die isgemiese miokardium verlaag, vererger miokardiale isgemie/herperfusie skade. AAS behandeling kan moontlik ook sistemiese TNFa vlakke verhoog deur limfosiet TNFa sekresie te stimuleer. Die verhoogde TNFa vlakke word verbind aan die onderdrukking van miokardiale funksie, miokardiale hipertrofie en die verergering van isgemie/herperfusie skade. Doelwitte: Die doelwitte van die studie was om te bepaal of kroniese AAS toediening in geoefende en ongeoefende rotte 1) hartfunksie en die hart se vatbaarheid vir isgemie/herperfusie skade beïnvloed, 2) miokardiale sikliese nukleotiedvlakke (sAMP en sGMP) beïnvloed en 3) miokardiale TNFa-vlakke beïnvloed. Materiale en metodes: Manlike Sprague-Dawley rotte (n=100) is gebruik en in 4 groepe verdeel: 'n ongeoefende placebo groep (kontrole); 'n ongeoefende steroïedbehandelde groep; 'n geoefende placebo groep (kontrole) en 'n geoefende steroïedbehandelde groep. Steroïed behandelde diere het 'n intramuskulêre nandroloon lauraat inspuiting (0.375 mg/kg) een keer per week vir ses weke ontvang. Die oefenprogram het bestaan uit ses swemsessies 'n week vir ses weke. Die swemtyd is geleidelik weekliks verhoog tot by 'n maksimum tyd 50 min. Die waterbadtemperatuur is tussen 30 - 32 oe gehandhaaf. Vir biometriese parameters is hartgewig en liggaamsgewig genoteer. Harte is op 'n Langendorff perfusie apparaat gemonteer en linker ventrikulêre ontwikkelde druk (LVOD), koronêre vloei (KV) en harttempo (HT) is genoteer. Die harte is vervolgens blootgestel aan 20 minute van globale isgemie gevolg deur 'n 30 minute herperfusieperiode. LVOD, KV en HT is weer eens noteer. Vir biochemiese doeleindes is bloed voor perfusie versamelom serum lipied vlakke te bepaal. Miokardiale weefsel is versamel voor, tydens en na isgemie vir die bepaling van TNFa, cGMP en AMP vlakke asook p38 aktiwiteit. Gevolgtrekkings: Na aanleiding van resultate verkry wil dit voorkom asof die gebruik van steroïde oefeningsgeïnduseerde miokardiale hipertrofie vererger. Dit verhoed ook oefeningsgeïnduseerde verbetering in miokardiale funksie. AAS lei tot 'n verlaagde herperfusiefunksie in behandelde harte, wat dalk mag dui op MS verergering van isgemie en herperfusie skade. Verder was daar ook waargeneem dat MS basale (pre-isgemiese) sikliese nukleotiedvlakke en basale TNFa-vlakke sowel as herperfusie TNFa vlakke verhoog. Die verhoging in TNF-a vlakke mag dus moontlik ook bydra tot die verergering van isgemie- en herperfusieskade.
Leigh, Felicity Suzanne Marshall. "The effects of diet, anorectic drugs and caffeine on various cardiovascular parameters in the rat". Thesis, University of Bath, 1988. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.382595.
Pełny tekst źródłaKalis, Joni Kathryn. "THE EFFECT OF BETA-ADRENERGIC BLOCKADE ON THE DRIFT IN OXYGEN CONSUMPTION WITH PROLONGED EXERCISE". Thesis, The University of Arizona, 1985. http://hdl.handle.net/10150/292014.
Pełny tekst źródłaKsiążki na temat "Heart Effect of drugs on"
H, Opie Lionel, red. Drugs for the heart. Wyd. 3. Philadelphia: W.B. Saunders Co., 1991.
Znajdź pełny tekst źródłaDenis, Noble, i Earm Yung E, red. Ionic channels and effect of taurine on the heart. Boston: Kluwer Academic Publishers, 1993.
Znajdź pełny tekst źródłaG, Reves J., i Hall Kenneth D, red. Common problems in cardiac anesthesia. Chicago: Year Book Medical Publishers, 1987.
Znajdź pełny tekst źródłaR, Watson Ronald, i Myers Adam K, red. Alcohol and heart disease. London: Taylor & Francis, 2002.
Znajdź pełny tekst źródłaDroogmans, Steven. Drug-induced valvular heart disease. Hauppauge, N.Y: Nova Science Publishers, 2010.
Znajdź pełny tekst źródłaI, Simpson Joseph, red. Anesthesia and the patient with co-existing heart disease. Boston: Little, Brown, 1993.
Znajdź pełny tekst źródłaGavin, Katherine T. Investigations of cardiovascular [alpha]-adrenoceptor function. Dublin: University College Dublin, 1997.
Znajdź pełny tekst źródłaM, Muggia Franco, Green Michael D. 1949- i Speyer James L, red. Cancer treatment and the heart. Baltimore: Johns Hopkins University Press, 1992.
Znajdź pełny tekst źródłaMyers, Adam K., i Ronald R. Watson. Alcohol and heart disease. London: Taylor & Francis, 2003.
Znajdź pełny tekst źródłaP, Hacker Miles, Lazo John S i Tritton Thomas R, red. Organ directed toxicities of anticancer drugs: Proceedings of the First International Symposium on the Organ Directed Toxicities of Anticancer Drugs, Burlington, Vermont, USA, June 4-6, 1987. Boston: Nijhoff, 1988.
Znajdź pełny tekst źródłaCzęści książek na temat "Heart Effect of drugs on"
Schwandt, Peter, Markus G. Donner i Werner O. Richter. "Long-Term Effect of Ldl-Apheresis on Coronary Heart Disease". W Drugs Affecting Lipid Metabolism, 513–19. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-0311-1_60.
Pełny tekst źródłaPugliese, Anna Maria, Elisabetta Coppi, Federica Cherchi i Giancarlo Pepeu. "Cardiovascular Adverse Effects of Psychotropic Drugs". W Brain and Heart Dynamics, 707–20. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-28008-6_45.
Pełny tekst źródłaPugliese, Anna Maria, Elisabetta Coppi, Federica Cherchi i Giancarlo Pepeu. "Cardiovascular Adverse Effects of Psychotropic Drugs". W Brain and Heart Dynamics, 1–15. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-90305-7_45-1.
Pełny tekst źródłaYang, Charles I., Pravin Taneja i Peter J. Davis. "Sedative Hypnotic and Anesthetic Agents: Their Effect on the Heart". W Handbook of Pediatric Cardiovascular Drugs, 280–317. London: Springer London, 2008. http://dx.doi.org/10.1007/978-1-84628-953-8_12.
Pełny tekst źródłaGovoni, Stefano. "Psychiatric and Neurological Effects of Cardiovascular Drugs". W Brain and Heart Dynamics, 731–44. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-28008-6_46.
Pełny tekst źródłaGovoni, S. "Psychiatric and Neurological Effects of Cardiovascular Drugs". W Brain and Heart Dynamics, 1–14. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-90305-7_46-1.
Pełny tekst źródłaErlemeier, H. H., i W. Bleifeld. "Effects of Different Positive Inotropic Drugs in Congestive Heart Failure". W Heart Failure Mechanisms and Management, 205–10. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-58231-8_21.
Pełny tekst źródłaAsteggiano, Riccardo. "Physiopathology and Toxic Heart Effects of Chemotherapy Drugs". W Cardiac Management of Oncology Patients, 23–97. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15808-2_2.
Pełny tekst źródłaGupta, Suresh Kumar, Meenu Sharma, Ipseeta Mohanty i Dharamvir S. Arya. "Molecular Basis for the Cardioprotective Effect of Herbal Drugs in Ischemic Heart Disease: An Experimental Study". W Myocardial Ischemia and Preconditioning, 235–48. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0355-2_17.
Pełny tekst źródłaEriksson, L. O., B. Beermann i M. Kallner. "Aspects of the Effects or NSAID’s on Renal Function in Congestive Heart Failure". W Non-steroidal Anti-Inflammatory Drugs Basis for Variability in Response, 99–103. Basel: Birkhäuser Basel, 1985. http://dx.doi.org/10.1007/978-3-0348-7720-6_12.
Pełny tekst źródłaStreszczenia konferencji na temat "Heart Effect of drugs on"
Serša, Gregor. "CLINICAL APPLICATIONS OF ELECTROCHEMOTHERAPY". W Symposium with International Participation HEART AND … Akademija nauka i umjetnosti Bosne i Hercegovine, 2019. http://dx.doi.org/10.5644/pi2019.181.01.
Pełny tekst źródłaMoussa, Heba Adel Mohamed Lotfy, Gawaher Saleh Abbas Mahgoub, Mashael Ali H. I. Al-Badr i Huseyin Cagatay Yalcin. "Investigating the Cardiac Effects of Sildenafil loaded Nanoparticles on Heart Failure using the Zebrafish Embryo Model". W Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0217.
Pełny tekst źródłaRizqhilmi, Muhammad Faris, Muhammad Ikrar Hermanadi, Muhammad Yusuf, Rani Wardani Hakim, Siti Farida i Erni Hernawati Purwaningsih. "The effect of Centella asiatica ethanolic extract on heart interleukin-10 levels of aged Sprague-Dawley rats". W THE 6TH BIOMEDICAL ENGINEERING’S RECENT PROGRESS IN BIOMATERIALS, DRUGS DEVELOPMENT, AND MEDICAL DEVICES: Proceedings of the 6th International Symposium of Biomedical Engineering (ISBE) 2021. AIP Publishing, 2022. http://dx.doi.org/10.1063/5.0100545.
Pełny tekst źródłaKurnikova, Irina, Shirin Gulova, Tatiana Nikishova, Nikolay Kislyy, Sophia Buturlina i Oksana Demina. "Frequency Indicators of Heart Rate Variability in Assessing the Effectiveness of Antihypertensive Therapy in Patients With Metabolic Disorders". W Human Interaction and Emerging Technologies (IHIET-AI 2022) Artificial Intelligence and Future Applications. AHFE International, 2022. http://dx.doi.org/10.54941/ahfe100875.
Pełny tekst źródłaAlkawari, Fatima, Wigdan Ali, Fatiha Benslimane i Huseyin Yalcin. "Investigating the Cardiac effects of New Generation Anti-Diabetic Drug Empagliflozin using Zebrafish Embryo Model". W Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0211.
Pełny tekst źródłaHaddad, Batoul, Zainab El-hashmi, Huseyin Yalcin, Fatiha Benslimane i Hana A. Hussain. "Investigating the Efficacy of Natural Blood Glucose Lowering Compounds in the Prevention of Congenital Heart Defects during Maternal Diabetes using the Chick Embryo". W Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0195.
Pełny tekst źródłaMcGoron, Anthony J., i Alicia Fernandez-Fernandez. "Development of a Multiple Indicator Dilution Technique Using Fluorescent Dyes to Measure Cardiac Capillary Permeability". W ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192955.
Pełny tekst źródłaAl-Ansari, Dana E., Nura A. Mohamed, Isra Marei, Huseyin Yalcin i Haissam Abou-Saleh. "Assessment of Metal Organic Framework as Potential Drug Carriers in Cardiovascular Diseases". W Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0127.
Pełny tekst źródłaDa Silva, Alberto Vianna Dias, i Vaninha Vieira. "BBAware - A Context-Aware Mobile and Wearable Architecture for Monitoring Beta-Blocked Cardiac Patients". W VIII Simpósio Brasileiro de Computação Ubíqua e Pervasiva. Sociedade Brasileira de Computação - SBC, 2020. http://dx.doi.org/10.5753/sbcup.2016.9444.
Pełny tekst źródłaKumar, A., J. Fareed, W. H. Wehrmacher, D. Hoppensteadt, O. Ulutin i J. M. Walenga. "ENDOTHELIAL FUNCTION MODULATION AND CONTROL OF VASCULAR AND THROMBOTIC DISORDERS: EXPERIMENTAL RESULTS WITH A POLYDEOXY RIBONUCLEOTIDE AGENT DEFIBROTIDE". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643149.
Pełny tekst źródłaRaporty organizacyjne na temat "Heart Effect of drugs on"
Wu, Xiaoqi, Jisen Zhao, Maoxia Fan i Dongo Guo. Quality of Evidence Supporting the Effects of Xinmailong injection in Heart Failure: An Overview of Systematic Reviews and Meta-Analyses. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, październik 2022. http://dx.doi.org/10.37766/inplasy2022.10.0023.
Pełny tekst źródłaBlack, Sandra, Paul Devereux i Kjell Salvanes. Losing Heart? The Effect of Job Displacement on Health. Cambridge, MA: National Bureau of Economic Research, grudzień 2012. http://dx.doi.org/10.3386/w18660.
Pełny tekst źródłaLichtenberg, Frank. The Effect of New Drugs on Mortality from Rare Diseases and HIV. Cambridge, MA: National Bureau of Economic Research, grudzień 2001. http://dx.doi.org/10.3386/w8677.
Pełny tekst źródłaTreadwell, Jonathan R., James T. Reston, Benjamin Rouse, Joann Fontanarosa, Neha Patel i Nikhil K. Mull. Automated-Entry Patient-Generated Health Data for Chronic Conditions: The Evidence on Health Outcomes. Agency for Healthcare Research and Quality (AHRQ), marzec 2021. http://dx.doi.org/10.23970/ahrqepctb38.
Pełny tekst źródłaBrusseau, Valentin, I. Tauveron, R. Bagheri, U. Ugbolue, V. Magnon, J. B. Bouillon-Minois, V. Navel i F. Dutheil. Effect of hyperthyroidism treatments on heart rate variability: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, sierpień 2022. http://dx.doi.org/10.37766/inplasy2022.8.0062.
Pełny tekst źródłaChou, Roger, Jesse Wagner, Azrah Y. Ahmed, Ian Blazina, Erika Brodt, David I. Buckley, Tamara P. Cheney i in. Treatments for Acute Pain: A Systematic Review. Agency for Healthcare Research and Quality (AHRQ), grudzień 2020. http://dx.doi.org/10.23970/ahrqepccer240.
Pełny tekst źródłaCrosland, Richard D. Effect of Drugs on the Lethality in Mice of the Venoms and Neurotoxins from Sundry Snakes. Fort Belvoir, VA: Defense Technical Information Center, lipiec 1990. http://dx.doi.org/10.21236/ada228245.
Pełny tekst źródłaGeorgitsis, Micheal. The Effect of Lymph Sac Pressure on Lymph Heart Pressure Development in the Toad Bufo Marinus. Portland State University Library, styczeń 2000. http://dx.doi.org/10.15760/etd.7232.
Pełny tekst źródłaZhang, Bin, Ming Zhang, Cao-Jie Lu, Li-Zhi Liu, Chen Jiang i Ming-Jin Zhu. Effect of Znic Supplementation on Patients with Chronic Heart Failure: A systematic Review and Meta Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, styczeń 2022. http://dx.doi.org/10.37766/inplasy2022.1.0100.
Pełny tekst źródłaLleras-Muney, Adriana, i Frank Lichtenberg. The Effect of Education on Medical Technology Adoption: Are the More Educated More Likely to Use New Drugs. Cambridge, MA: National Bureau of Economic Research, wrzesień 2002. http://dx.doi.org/10.3386/w9185.
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