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Gotowe bibliografie tematyczne / Heart Muscles Diseases

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Gotowa bibliografia na temat „Heart Muscles Diseases”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 31 stycznia 2023

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Artykuły w czasopismach na temat "Heart Muscles Diseases"

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Agafonov, F. D. "The state of the peripheral circulatory system in infectious diseases." Kazan medical journal 25, no. 11 (October 29, 2021): 1210–17. http://dx.doi.org/10.17816/kazmj80472.

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The state of blood circulation and the causes of its disorder, both in chronic and, in particular, in acute infectious diseases, have always been of great interest to both clinicians and pathologists. Since the time of Laennec, who first drew attention to the weakness of the heart muscles in those who died from febrile diseases and emphasized, like Louis, weakness and fragility of the heart muscle, they began to look for the causes of these disorders in the state of the heart muscles. A significant success in the study of diseases of the heart muscle in infectious diseases was the teaching of

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Chandragirish S, Harsha B R, and Girish V. Patil. "Morphometric study on papillary muscles of human tricuspid valve-dissection method." Indian Journal of Clinical Anatomy and Physiology 8, no. 3 (October 15, 2021): 190–93. http://dx.doi.org/10.18231/j.ijcap.2021.043.

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Aim of the present study was to observe the measurements of anterior papillary muscles present in tricuspid valve of human heart. Measurements of anterior papillary muscles in tricuspid valve gains utmost importance in cardiac surgeries because they are the causes of myocardial infarction in recent times because of its variations and detection of these causes by advent in modern technologies which will help in treatment of tricuspid valve diseases. This study was carried out on 96 normal formalin fixed human heart specimens. Dissection was performed according to standard techniques. Anterior p

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Gorkova, N. B., L. M. Starykh, and L. E. Karpova. "A case of early pelvic-brachial progressive muscular dystrophy with severe heart involvement." Kazan medical journal 72, no. 2 (February 15, 1991): 142–44. http://dx.doi.org/10.17816/kazmj106605.

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Progressive muscular dystrophies (PMD) are a group of hereditary diseases characterized by a primary dystrophic process in muscle tissue. The peculiarity and characteristics of cardiac disorders depend both on the nosological form with a characteristic muscle pathology for each of them, and on the stage, severity of the course of the disease and the mass of the affected muscles, and circulatory failure is less pronounced than damage to skeletal muscles.

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Jörgen Sandell and Mark Davies. "Benefits of sauna on lung capacity, neurocognitive diseases, and heart health." World Journal of Advanced Research and Reviews 17, no. 1 (January 30, 2023): 057–62. http://dx.doi.org/10.30574/wjarr.2023.17.1.1414.

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Sauna refers to passive heat therapy that involves exposure of the body to a high-temperature environment for an appropriately short period, contingent on the therapy’s purpose. Ideally, the therapy aims to raise the internal body temperature by a few degrees, and its effect happens in two phases. The first phase occurs during the first ten minutes, encouraging the body to perspire while maintaining a temperature of around 98.6 degrees. The extra heat is dispersed by increased blood circulation, blood pushing on the skin's surface, and sweating. The body enters the second phase after 10-30 min

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Nazir, Muhammad Mudasser, Muhammad Mazhar Ayaz, Atif Nisar Ahmed, Azhar Maqbool, Kamran Ashraf, Muhammad Oneeb, Ghulam Yasin, et al. "Prevalence of Toxoplasma gondii, Neospora caninum, and Sarcocystis Species DNA in the Heart and Breast Muscles of Rock Pigeons (Columbia livia)." Journal of Parasitology Research 2018 (2018): 1–4. http://dx.doi.org/10.1155/2018/6264042.

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Little is known about the prevalence of protozoan parasites in the muscles of rock pigeons (Columbia livia). The muscles from 54 (heart from 45 and breast from 54) rock pigeons were examined for DNA of Toxoplasma gondii, Neospora caninum, and Sarcocystis species using PCR. Twenty-four were female and 30 were males. The birds were part of flocks of pigeons housed at the tombs of saints in Lahore, Pakistan. Birds that died or were euthanized due to poor health were submitted for necropsy at the Department of Parasitology, University of Veterinary and Animal Sciences, Lahore, Pakistan, where DNA

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Shaikh, Rumana M. "Cardiovascular Diseases Prediction Using Machine Learning Algorithms." Turkish Journal of Computer and Mathematics Education (TURCOMAT) 12, no. 6 (April 11, 2021): 1083–88. http://dx.doi.org/10.17762/turcomat.v12i6.2426.

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A broad variety of health conditions are involved in heart disease. Several illnesses and disorders come under the heart disease umbrella. Heart disease forms include: In arrhythmia, abnormality of the heart rhythm. Arteriosclerosis, Hardening of the arteries is atherosclerosis. Via cardiomyopathy, this disorder causes muscles in the heart to harden or grow weak. Defects of the congenital heart, heart abnormalities that are present at birth are congenital heart defects. Disease of the coronary arteries (CAD), the accumulation of plaque in the heart's arteries triggers CAD. It's called ischemic

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Dubuisson, Nicolas, Romain Versele, María A. Davis-López de Carrizosa, Camille M. Selvais, Sonia M. Brichard, and Michel Abou-Samra. "Walking down Skeletal Muscle Lane: From Inflammasome to Disease." Cells 10, no. 11 (November 4, 2021): 3023. http://dx.doi.org/10.3390/cells10113023.

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Over the last decade, innate immune system receptors and sensors called inflammasomes have been identified to play key pathological roles in the development and progression of numerous diseases. Among them, the nucleotide-binding oligomerization domain (NOD-), leucine-rich repeat (LRR-) and pyrin domain-containing protein 3 (NLRP3) inflammasome is probably the best characterized. To date, NLRP3 has been extensively studied in the heart, where its effects and actions have been broadly documented in numerous cardiovascular diseases. However, little is still known about NLRP3 implications in musc

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Caron, Marc-André, Richard Debigaré, P. N. Richard Dekhuijzen, and François Maltais. "Comparative assessment of the quadriceps and the diaphragm in patients with COPD." Journal of Applied Physiology 107, no. 3 (September 2009): 952–61. http://dx.doi.org/10.1152/japplphysiol.00194.2009.

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Chronic obstructive pulmonary disease (COPD) and other chronic diseases such as heart failure are accompanied by skeletal muscle alterations that further enhance morbidity and mortality in affected individuals. Several studies have highlighted important structural and biochemical modifications in limb and respiratory muscles in COPD. Reviewing the similarities and differences between the two most studied muscles in COPD, the quadriceps and the diaphragm, may be helpful in providing important clues about the mechanisms underlying muscle changes associated with this disease. Although oxidative s

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Kovalev, Dmitriy. "Myocarditis complicated by therapy-resistant heart failure." Spravočnik vrača obŝej praktiki (Journal of Family Medicine), no. 2 (February 1, 2020): 55–60. http://dx.doi.org/10.33920/med-10-2002-07.

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Myocarditis is an inflammation of the heart muscles, caused by direct or indirect — through immune mechanisms — exposure to infectious, physical and chemical factors, as well as developing in case of autoimmune diseases and heart transplantation. Inflammatory myocardial diseases are one of the rare nosological forms in cardiology, which, despite its nearly two-century history, still does not have clear guidelines for diagnosis and treatment.

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Moalla, Wassim, Grégory Dupont, Abdou Temfemo, Yves Maingourd, Matthew Weston, and Said Ahmaidi. "Assessment of exercise capacity and respiratory muscle oxygenation in healthy children and children with congenital heart diseases." Applied Physiology, Nutrition, and Metabolism 33, no. 3 (June 2008): 434–40. http://dx.doi.org/10.1139/h07-196.

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Muscular and cardiorespiratory dysfunction contributes to exercise intolerance. Therefore, the aim of the present study was to characterize the cardiopulmonary response andrespiratory muscle oxygenation of children with congenital heart diseases (CHD) when compared with those of healthy children. Twelve children with CHD in New York Heart Association (NYHA) class II or III, and 14 healthy children participated in the study. All subjects performed conventional spirographic measurements and a cardiopulmonary exercise test on a cycle ergometer. Oxygen uptake (VO2), carbon dioxide production (VCO2

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Rozprawy doktorskie na temat "Heart Muscles Diseases"

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Gieseking, Elizabeth Robinson. "Control mechanism for the papillary muscles of the mitral valve : an In Vitro study." Thesis, Georgia Institute of Technology, 1989. http://hdl.handle.net/1853/10912.

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Warner, Anke Sigrid. "The expression, regulation and effects of inducible nitric oxide synthase in hibernating myocardium." Title page, contents and summary only, 2002. http://web4.library.adelaide.edu.au/theses/09PH/09phw279.pdf.

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Amendments inserted at back. "May 2002" Includes bibliographical references (leaves 237-290) Experiments described in this thesis address the potential role of inducible nitric oxide synthase (iNOS) in hibernating myocardium. Specifically it was sought to establish a cellular model of hibernating myocardium and investigate the expression, regulation and effects of iNOS in this model. Experiments were performed using primary cultures of neonatal rat ventricular myocytes.

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Messaggi-Sartor, Monique 1984. "Respiratory muscle dysfunction in respiratory and non-respiratory diseases : clinical and therapeutic approaches." Doctoral thesis, Universitat Pompeu Fabra, 2017. http://hdl.handle.net/10803/565809.

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Respiratory muscle dysfunction is a clinical condition that may be present in both respiratory and non-respiratory diseases. This impairment of muscle function can have a negative effect on clinical outcomes, contributing to a further worsening of the patient’s clinical condition. This doctoral thesis has been directed by the ‘Rehabilitation Research Group’ (RERG) in collaboration with the Muscle Wasting and Cachexia in Chronic Respiratory Diseases and Lung Cancer Group (Lung Cancer and Muscle Research Group) of the Institut Hospital del Mar d’Investigacions Mèdiques (IMIM) in Barcelona. Muscl

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Spinner, Erin M. "Tricuspid valve mechanics: understanding the effect of annular dilatation and papillary muscle displacement." Diss., Georgia Institute of Technology, 2011. http://hdl.handle.net/1853/45754.

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Tricuspid regurgitation (TR), back flow of blood from the right ventricle to the right atrium, has been reported in approximately 85% of the population, with 16% having mild or severe TR. Patients with untreated moderate to severe TR are likely to experience decreased exercise capacity and have increased morbidity and mortality, thus affecting the patient's quality of life. Current methods of repair offer limited rates of success, and many patients require further operations to correct returning levels of TR. Incomplete repair may be due to incomplete understanding of the functional anatomy a

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Yentrapalli, Venkata Ramesh. "Novel radiation targets in the endothelium and heart muscle." Doctoral thesis, Stockholms universitet, Institutionen för molekylär biovetenskap, Wenner-Grens institut, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-90429.

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Worldwide, people are being exposed to natural and man-made sources of radiation. Epidemiological studies have shown an increased risk of vascular diseases in populations that have been exposed to ionizing radiation. Vascular endothelium is implicated as one of the targets for radiation leading to the development of cardiovascular diseases. However, the molecular mechanisms behind the development of radiation-induced cardiovascular disease in acute or chronic exposed people are not fully elucidated. The hypothesis that chronic low dose rate ionizing radiation accelerates the onset of senescenc

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Pandofino, Alexandra. "A molecular analysis of the basis of cardiovirulence of Coxsackievirus B3." Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.267166.

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Colegrave, Melanie. "Expression of #beta#-cardiac myosin in a myogenic cell line." Thesis, King's College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.342254.

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Vesier, Carol Cockerham. "The role of papillary muscle-mitral valve geometry in systolic anterior motion of the mitral valve." Diss., Georgia Institute of Technology, 1991. http://hdl.handle.net/1853/10279.

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Chung, Jae-Hoon. "Regulation of Human Cardiac Muscle Contraction and Relaxation in Health and Disease." The Ohio State University, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=osu1522851185767187.

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Johnson, Andrew William. "Metabolic control of energetics in human heart and skeletal muscle." Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:82c0dce6-a162-4c08-b061-3ea7f2e35134.

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Myocardial and skeletal muscle high energy phosphate metabolism is abnormal in heart failure, but the pathophysiology is not understood. Plasma non-esterified fatty acids (NEFA) increase in heart failure due to increased sympathetic drive, and regulate the transcription of mitochondrial uncoupling protein-3 (UCP3), through peroxisome proliferator-activated receptor-α. The aim of the work in this thesis was to determine whether cardiac PCr/ATP ratios and skeletal muscle PCr kinetics during exercise were related to cardiac and skeletal muscle UCP3 levels respectively, thus providing a mechanism

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Książki na temat "Heart Muscles Diseases"

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Williams, Andrew D. Skeletal muscle in heart failure and type 2 diabetes. New York: Nova Biomedical Books, 2010.

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Goodwin, J. F., ed. Heart Muscle Disease. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4874-7.

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L, Tavazzi, and Di Prampero P. E, eds. The anaerobic threshold: Physiological and clinical significance. Basel: Karger, 1986.

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1962-, Laguna Pablo, ed. Bioelectrical signal processing in cardiac and neurological applications. Amsterdam: Elsevier Academic Press, 2005.

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Nuno, Azóia, and Dobreiro Pedra, eds. Treadmill exercise and its effects on cardiovascular fitness, depression, and muscle aerobic function. Hauppauge, N.Y: Nova Science Publisher, 2009.

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C, Claycomb William, Di Nardo Paolo, and New York Academy of Sciences., eds. Cardiac growth and regeneration. New York, N.Y: New York Academy of Sciences, 1995.

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S, Abraham, and Amitai G, eds. Calcium channel modulators in heart and smooth muscle: Basic mechanisms and pharmacological aspects : proceedings of the 33rd [i.e. 34th] Oholo Conference, Eilat, Israel, 1989. Rehovot, Israel: Balaban Publishers, 1990.

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L, Archer Stephen, and Rusch Nancy Jean, eds. Potassium channels in cardiovascular biology. New York: Kluwer Academic/Plenum, 2001.

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Wang, Tammy, Jocelyn Wong, and Anita Honkanen. Glycogen Storage Diseases. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0048.

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Glycogen storage diseases result from deficiencies of various enzymes or proteins in the pathways of glycogen metabolism. The reduction in effective glucose storage and/or mobilization results in hypoglycemia and accumulation of glycogen in tissues. Diagnosis can occur at any age, from infancy to adulthood, depending on the pathway affected and the degree of enzyme deficiency. The clinical presentation varies, but the most commonly affected organ systems include the heart, liver, and skeletal muscles. In addition to the morbidity that can occur from dysfunction of these organs, important anest

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Ramrakha, Punit, and Jonathan Hill, eds. Heart muscle diseases. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199643219.003.0008.

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Classification 418Dilated cardiomyopathy 420Dilated cardiomyopathy: treatment 422Hypertrophic cardiomyopathy 424Hypertrophic cardiomyopathy: investigations 428Hypertrophic cardiomyopathy: treatment 430Restrictive cardiomyopathy 432Cardiac amyloidosis 434Cardiac amyloidosis: treatment 436Fabry disease 438Arrhythmogenic right ventricular cardiomyopathy (ARVC) 440ARVC: management 442Left ventricular non-compaction ...

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Części książek na temat "Heart Muscles Diseases"

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Bogaert, J., and A. M. Taylor. "Heart Muscle Diseases." In Clinical Cardiac MRI, 275–353. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/174_2011_358.

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Wenger, N. K. "Specific heart muscle diseases." In Heart Muscle Disease, 95–139. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4874-7_5.

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Oakley, C. M. "Amyloid heart disease." In Heart Muscle Disease, 141–53. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4874-7_6.

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Goodwin, J. F. "Cardiomyopathies and specific heart muscle diseases: definition, terminology and classification." In Heart Muscle Disease, 1–5. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4874-7_1.

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Perloff, J. K., J. F. Goodwin, D. D. Sugrue, and W. J. McKenna. "Hypertrophic Cardiomyopathy." In Heart Muscle Disease, 7–56. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4874-7_2.

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Olsen, E. G. J., and R. O. Brandenburg. "Dilated (Congestive) Cardiomyopathy." In Heart Muscle Disease, 57–86. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4874-7_3.

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Davies, J. "Restrictive cardiomyopathy." In Heart Muscle Disease, 87–94. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4874-7_4.

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Criley, J. M., R. J. Siegel, J. P. Murgo, J. W. Miller, H. Kuhn, E. D. Wigle, M. Henderson, Z. Sasson, C. Pollick, and H. Rakowski. "Hypertrophic Cardiomyopathy—The Obstructive Dilemma." In Heart Muscle Disease, 157–262. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4874-7_7.

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Lowry, P. J., W. A. Littler, and H. D. Bolte. "Dilated (Congestive) Cardiomyopathy—The evidence for and Against a Disorder of Cellular Immunity and Infection." In Heart Muscle Disease, 263–84. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4874-7_8.

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Metze, Dieter, Vanessa F. Cury, Ricardo S. Gomez, Luiz Marco, Dror Robinson, Eitan Melamed, Alexander K. C. Leung, et al. "Heart Muscle Diseases, Toxic." In Encyclopedia of Molecular Mechanisms of Disease, 783–85. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1760.

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Streszczenia konferencji na temat "Heart Muscles Diseases"

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Cassino, Theresa R., Masaho Okada, Lauren Drowley, Johnny Huard, and Philip R. LeDuc. "Mechanical Stimulation Improves Muscle-Derived Stem Cell Transplantation for Cardiac Repair." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192941.

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Muscle-derived stem cells (MDSCs) have been successfully transplanted into both skeletal (1) and cardiac muscle (2) of dystrophin-deficient (mdx) mice, and show potential for improving cardiac and skeletal dysfunction in diseases like Duchenne muscular dystrophy (DMD). Our previous study explored the regeneration of dystrophin-expressing myocytes following MDSC transplantation into environments with distinct blood flow and chemical/mechanical stimulation attributes. After MDSC transplantation within left ventricular myocardium and gastrocnemius (GN) muscles of the same mdx mice, significantly

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Moussa, Heba Adel Mohamed Lotfy, Gawaher Saleh Abbas Mahgoub, Mashael Ali H. I. Al-Badr, and Huseyin Cagatay Yalcin. "Investigating the Cardiac Effects of Sildenafil loaded Nanoparticles on Heart Failure using the Zebrafish Embryo Model." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0217.

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Background: Cardiovascular diseases (CVDs) are the first cause of death worldwide. Vasolidator agents are used to relax cardiac muscle, but their extremely short half-lifes limit their effectiveness. Sildenafil is such an agent used to relax the blood vessels muscles and increase the blood flow. The conventional drug can lead to serious problems in patients duo to the systematic drug delivery. Use of Nanomedicine potentially can enhance delivery of this agent while reducing the systematic effect of the drug. Aim: The purpose of the research is to examine the effectiveness sildenafil loaded nan

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Mesihović-Dinarević, Senka. "WHAT IS NEW IN CARDIOVASCULAR MEDICINE?" In Symposium with International Participation HEART AND … Akademija nauka i umjetnosti Bosne i Hercegovine, 2019. http://dx.doi.org/10.5644/pi2019.181.03.

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The rapid pace of change continues to be a hallmark in cardiovascular medicine and many see that pace accelerating in adult cardiovascular medicine as well as in paediatric cardiology medicine. Cardiovascular medicine is an area of clinical practice with a continually rapid expansion of knowledge, guidelines, best practices and new technology. Cardiovascular diseases are the leading cause of mortality in the world and cause major costs for the health sector and economy. Primary care clinicians are challenged to optimally manage a multitude of diseases including congestive heart failure, corona

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Schroer, Alison K., and W. David Merryman. "Integrin-Focal Adhesion Coupling and Substrate Stiffness Affect Smooth Muscle Alpha Actin Expression in Fibroblasts." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80887.

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Fibroblast cells play a key role in producing and maintaining connective tissue throughout the body. The ability of these cells to differentiate into a more active myofibroblastic phenotype is important during development and wound healing, but prolonged myofibroblast activation can lead to overproduction of extracellular matrix proteins and stiffening of the surrounding tissue. This stiffening can cause heightened differentiation of neighboring fibroblast through force transduction pathways and can lead to detrimental fibrotic pathologies in many organ systems. Atherosclerosis, interstitial l

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van der Horst, Arjen, Frits L. Boogaard, Marcel C. M. Rutten, and Frans N. van de Vosse. "A 1D Wave Propagation Model of Coronary Flow in a Beating Heart." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53367.

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Due to recent developments in miniaturized sensors on guide-wires, assessment of coronary artery disease with intracoronary pressure and flow measurements has become available. However, direct quantification is still limited to the large epicardial vessels, which means that microvascular disease can only be determined from upstream measurements using an appropriate model of the vessels and their interaction with the cardiac muscle [1].

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Raskina, Tatiana, Inessa Grigoreva, Olga Barbarash, Alexander Kokov, and Vladislava Masenko. "AB0848 BMD AND MUSCLE MASS IN PATIENTS WITH ISCHEMIC HEART DISEASE." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.3662.

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Etebari, Ali, Olga Pierrakos, and Pavlos P. Vlachos. "Automatic MRI Image Segmentation and Left Ventricle Surface Reconstruction for Characterizing Myocardial Muscle Function." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-32251.

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Normal left ventricular function is characterized by synchronous firing of myocardial muscle fibers. Healthy myocardial function is essential in maintaining cardiac output, and any disturbance to the contracting muscle cells can be detrimental to the health of the heart. A method for non-invasively monitoring left ventricle (LV) operation is necessary for documenting the progression of heart disease and malfunction, such that the point at which disease becomes irreversible can be determined.

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Dolensky, Joseph R., Lauren D. C. Casa, and Ajit P. Yoganathan. "The Effect of Pulmonary Hypertension on Tricuspid Valve Coaptation in Normal and Pathologic Valve Geometries: An In Vitro Study." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80184.

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Pulmonary hypertension (PHTN) is a pathological condition defined as a mean pulmonary artery pressure (mPAP) greater than 25 mmHg. PHTN can result from a number of lung and heart pathologies, including abnormalities of the pulmonary vasculature, left heart disease, chronic lung disease, and chronic thrombotic disease [1]. Regardless of the cause, the increased afterload on the right heart results in right ventricle (RV) hypertrophy and dilatation and tricuspid regurgitation (TR) [2]. RV dilatation is thought to result in the displacement of the tricuspid valve (TV) papillary muscles (PM) and d

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Sewell-Loftin, M. K., and W. David Merryman. "The Role of SRC in Strain- and Ligand- Dependent Phenotypic Modulation of Mouse Embryonic Fibroblasts." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53604.

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Connective tissue fibrosis represents a significant portion of mortality and morbidity in our society. These diseases include many illnesses such as heart valve disease, atherosclerosis, macular degeneration, and cirrhosis, meaning that millions of lives are affected by these conditions each year. Fibrotic tissues form when quiescent fibroblasts activate becoming myofibroblasts, the phenotype of active tissue construction and fibrosis. During this process, the cells produce smooth muscle α-actin (αSMA), a contractile element considered to be the hallmark of cellular activation [1]. Following t

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Ivanov, Zhanna A., Robert C. Scott, Jenna Rosano, Barbara Krynska, and Mohammad F. Kiani. "Engineering Cardiac Tissue Using Stem Cell Therapy to Mend the Broken Heart." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-203624.

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Myocardial infarction (MI) is one of the most severe forms of coronary artery disease and is the leading cause of death in the United States [1]. Current treatments for an MI are either highly invasive, such as coronary artery bypass grafting and stent angioplasty, or might have undesirable long-term effects as is the case with pharmacological interventions. However, newly emerging methodologies, such as a less invasive stem cell therapy, aim to cure the disease rather than just alleviate its symptoms. This new tissue engineering technology has shown promise in restoring the homeostasis of the

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Raporty organizacyjne na temat "Heart Muscles Diseases"

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Kanner, Joseph, Edwin Frankel, Stella Harel, and Bruce German. Grapes, Wines and By-products as Potential Sources of Antioxidants. United States Department of Agriculture, January 1995. http://dx.doi.org/10.32747/1995.7568767.bard.

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Several grape varieties and red wines were found to contain large concentration of phenolic compounds which work as antioxidant in-vitro and in-vivo. Wastes from wine production contain antioxidants in large amounts, between 2-6% on dry material basis. Red wines but also white wines were found to prevent lipid peroxidation of turkey muscle tissues stored at 5oC. The antioxidant reaction of flavonoids found in red wines against lipid peroxidation were found to depend on the structure of the molecule. Red wine flavonoids containing an orthodihydroxy structure around the B ring were found highly

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