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1

Tabery, Helena M. Varicella-Zoster Virus Epithelial Keratitis in Herpes Zoster Ophthalmicus: In Vivo Morphology in the Human Cornea. Springer-Verlag Berlin Heidelberg, 2011.

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Tabery, Helena M. Varicella-Zoster Virus Epithelial Keratitis in Herpes Zoster Ophthalmicus. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-14487-5.

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Centers for Disease Control (U.S.). Dental Disease Prevention Activity. Preventing the transmission of hepatitis B, AIDS, and herpes in dentistry. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control, Center for Preventive Services, Dental Disease Prevention Activity, 1985.

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Gilden, Don, Randall J. Cohrs, Ravi Mahalingam, and Maria A. Nagel. Varicella Zoster Virus Infection of the Nervous System. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0149.

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Varicella zoster virus (VZV) is a human herpesvirus that causes varicella (chickenpox), after which virus becomes latent in ganglionic neurons along the entire neuraxis. Reactivation of VZV due to a decline in the cell-mediated immune response to VZV in elderly or immunocompromised individuals causes zoster (shingles), frequently complicated by chronic pain (postherpetic neuralgia) and serious neurological disease (meningoencephalitis, myelitis and VZV vasculopathy due to retrograde spread of virus after zoster. Here, we describe clinical, laboratory and pathological features of neurological c
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Tabery, Helena M. Varicella-Zoster Virus Epithelial Keratitis in Herpes Zoster Ophthalmicus: In Vivo Morphology in the Human Cornea. Springer, 2016.

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6

Tabery, Helena M. Varicella-Zoster Virus Epithelial Keratitis in Herpes Zoster Ophthalmicus: In Vivo Morphology in the Human Cornea. Springer, 2011.

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7

Bunker, Professor Christopher, and Dr Arani Chandrakumar. Dermatological diseases and emergencies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.00017.

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Chapter 17 covers dermatological diseases and emergencies including a general introduction to the subject, followed by information on erythroderma, drug eruptions, angio-oedema, Kawasaki disease, staphylococcal toxic shock syndrome, Streptococcal toxic shock syndrome (streptococcal TSS), staphylococcal scalded skin syndrome, necrotizing fasciitis, psoriasis, eczema and dermatitis, cutaneous vasculitis, immunobullous disorders, pyoderma gangrenosum, scarring alopecia, herpes simplex viruses 1 and 2, varicella zoster virus infection, bacterial infections affecting the skin, fungal infections aff
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8

Bale, James F. Congenital and Perinatal Viral Infections. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0160.

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Despite remarkable advancements in the treatment and prevention of infectious diseases, congenital (also known as intrauterine) and perinatal (also known as neonatal) infections remain major causes of permanent neurodevelopmental disabilities worldwide. Fortunately, relatively few viral pathogens can infect the developing fetus or the newborn postnatally and induce neurological disease. These pathogens include cytomegalovirus, rubella virus, herpes simplex virus types 1 and 2, varicella zoster virus, lymphocytic choriomeningitis virus, the nonpolio enteroviruses, parechovirus, and human immuno
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9

Picone, Olivier, Christelle Vauloup-Fellous, and Laurent Mandelbrot. Varicella-Zoster Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0014.

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Chickenpox in a pregnant woman is uncommon, but it is a major concern for patients and their families, as well as for clinicians caring for pregnant women. Varicella infection during pregnancy is usually benign, but there can be serious consequences for both mother and child. Notably, fetal varicella syndrome (FVS) can happen when infection occurs before 21 weeks of gestation. It can present with serious neurological anomalies and unusual cicatricial skin lesions. Later in pregnancy, primary neonatal varicella may occur when the mother is infected in the peripartum period, and it can be life-t
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10

(Editor), A. A. Gershon, C. H. Calisher (Editor), and A. M. Arvin (Editor), eds. Immunity to and Prevention of Herpes Zoster (Archives of Virology. Supplementa). Springer, 2001.

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11

(Editor), Gerd Gross, and Hans Wilhelm Doerr (Editor), eds. Herpes Zoster: Recent Aspects of Diagnosis and Control (Monographs in Virology). S. Karger AG (Switzerland), 2006.

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12

Brown, David W. G. Herpes B virus (Cercopithecine Herpes 1). Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0036.

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Herpes B virus or Cercopithecine herpes 1 as it is formally classified causes a persistent infection of monkeys of the Macaca genus. In monkey colonies and social groups, it is transmitted by close contact and sexually. Human infection is rare with less than 50 human cases described it has been seen in monkey handlers exposed to infected monkeys following bites, scratches and abraded skin. Infection has also been recognized in two cases following exposure through laboratory work. Following an incubation period of 9-59 days typically an ascending encephalomyelitis develops which is fatal in 80%
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13

Alyazidi, Raidan, and Soren Gantt. Herpes simplex Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0007.

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Herpes simplex virus (HSV) types 1 and 2 cause several important syndromes, including congenital and perinatal infections that can cause devastating consequences in newborns (i.e., neonatal HSV). Most neonatal HSV infections are acquired intrapartum in the infected maternal birth canal. Since genital HSV infections are common, neonatal HSV is an important complication in infected women, even if maternal symptoms are absent. As a result of the developmental status of the fetal and newborn immune system, neonatal HSV infection is associated with life-threatening disease. This chapter reviews the
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14

Arvin, Ann M., and Anne A. Gershon. Varicella-Zoster Virus: Virology and Clinical Management. Cambridge University Press, 2010.

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15

Oliver, Stefan L., Ann M. Arvin, Allison Abendroth, and Jennifer F. Moffat. Varicella-Zoster Virus: Genetics, Pathogenesis and Immunity. Springer International Publishing AG, 2022.

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16

Arvin, Ann M., and Anne A. Gershon. Varicella-Zoster Virus: Virology and Clinical Management. Cambridge University Press, 2011.

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(Editor), Ann M. Arvin, and Anne A. Gershon (Editor), eds. Varicella-Zoster Virus: Virology and Clinical Management. Cambridge University Press, 2001.

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18

Bryant, Kevin Francis. Regulation of translation during herpes simplex virus-1 infection. 2007.

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19

(), David Hommes. Herpes & Shingles: How to Relieve the Pain of Herpes Zoster, Treat the Virus and Prevent Future Outbreak. Independently Published, 2019.

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Hommes, David. Herpes and Shingles: How to Relief the Pain of Herpes Zoster, Treat the Virus and Prevent Future Outbreak. Independently Published, 2018.

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21

(Editor), Manfred H. Wolff, S. Schunemann (Editor), and A. Schmidt (Editor), eds. Vericella-Zoster Virus: Molecular Biology, Pathogenesis, and Clinical Aspects (Contributions to Microbiology, 3). S. Karger AG (Switzerland), 1999.

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22

BURMER, Karl. Cbd Oil for Shingles: Your Therapeutic Guide to Curing Varicella-Zoster Virus Infection. Independently Published, 2019.

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23

Sayler, Tobi. Shingles Relief : Relieve Pain of Herpes Zoster, Treat the Herpes Virus, and Prevent Future Outbreaks: What Can Be Mistaken for Shingles. Independently Published, 2021.

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24

Bronson, Vincent. Guide to Dr. Sebi Diet for Herpes: Genital Herpes Is a Common Sexually Transmitted Infection Caused by the Herpes Simplex Virus . Independently Published, 2020.

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25

Genital Herpes. Exon Publications, 2024. http://dx.doi.org/10.36255/genital-herpes.

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Genital Herpes is a common sexually transmitted infection caused by the herpes simplex virus, leading to painful sores or blisters in the genital and anal areas. This article serves as a guide for the public to understand the essential aspects of genital herpes. It is organized into key sections, beginning with an introduction to genital herpes and its causes. The article then discusses the strains of the herpes simplex virus and the risk factors that increase susceptibility to the infection. The prevalence of genital herpes is explored, highlighting its widespread nature. Signs and symptoms a
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26

Silva, Lindsey Marie. Role for the nuclear lamina during early stages of herpes simplex virus 1 infection. 2010.

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27

Struyf, Frank. Genetic Analysis Of Herpesvirus Entry Receptors And Host Susceptibility To Herpes Simplex Virus Infection. Leuven Univ Pr, 2004.

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28

Candido, Kenneth D., Teresa M. Kusper, and Nebojsa Nick Knezevic. Chronic Chest Wall Pain in Postherpetic Neuralgia. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190271787.003.0014.

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Postherpetic neuralgia (PHN) is a debilitating condition that frequently arises after herpes zoster (HZ) caused by the varicella-zoster virus. It is characterized by severe neuropathic pain and sensory disturbances persisting after the resolution of characteristic vesicular skin lesions. Most commonly affected are the thoracic dermatomes. Trigeminal (V1), cervical, and lumbar nerves are other frequently affected sites. Early treatment shortens the duration of acute HZ and may prevent the onset of PHN. A variety of modalities are utilized to treat PHN, including chemical compounds, intervention
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Jones, Mark R., Matthew Novitch, Graham R. Hadley, Alan D. Kaye, and Sudhir A. Diwan. Thoracic Spine Pain. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190626761.003.0008.

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Thoracic spinal pain (TSP) tends to receive less attention from clinical, epidemiologic, and genetic research communities owing to a reduced incidence in comparison to pain arising from cervical and lumbar derangement. Nevertheless, TSP can be similarly disabling to other forms of spinal pain, imposing significant burdens on the individual and society. Thoracic pain may arise from a multitude of underlying pathologies, including angina pectoris, herpes zoster infection, thoracic disc herniations, pulmonary or pleural tumors, and aneurysms. This chapter focuses on TSP of musculoskeletal origin;
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30

van Assen, Sander, and Marc Bijl. Vaccination in immunocompromised adults. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0094.

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This chapter addresses all important questions regarding vaccination of patients with autoimmune inflammatory rheumatic diseases (AIIRD). First, the incidence of vaccine-preventable infections in these patients is discussed. Pulmonary infections, including influenza and pneumococcal infection, occur more often in AIIRD patients; herpes zoster and human papillomavirus are also more frequent. The efficacy of vaccination for all European registered vaccines is discussed. Treatment with disease-modifying anti-rheumatic drugs (DMARDs) and biologicals (in particular TNFα‎-blocking agents) do not ham
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31

Shingles. Exon Publications, 2024. http://dx.doi.org/10.36255/shingles.

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Shingles is a comprehensive article that explains the causes, symptoms, diagnosis, and treatment options for this viral infection caused by the reactivation of the varicella-zoster virus. The article begins with an overview of shingles, detailing how the virus that causes chickenpox can later reactivate in the body, leading to painful rashes and complications. It explores the risk factors, such as age and weakened immune systems, that make individuals more susceptible to shingles. The guide thoroughly covers the symptoms, including the typical rash and potential complications like postherpetic
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Keshav, Satish, and Palak Trivedi. Viral hepatitis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0212.

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Hepatitis means ‘inflammation of the liver’ and is manifest with symptoms that include malaise, anorexia, fever, flu-like symptoms, and pain in the right upper quadrant of the abdomen, with the pain being caused by swelling of the liver and its capsule. Elevations in circulating hepatic enzymes, particularly aspartate transaminase and alanine transaminase, are common, with jaundice occurring some time after the onset of other symptoms and signs. There are five viruses that primarily cause viral hepatitis: hepatitis A, B, C, D, and E viruses, abbreviated HAV, HBV, HCV, HDV, and HEV, respectivel
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DeAugustinas, M., and A. Kiely. Infectious Keratitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0016.

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Keratitis is an inflammation of the cornea, which can lead to corneal opacification or ulceration. The most common cause of infectious keratitis is herpes simplex virus type 1 (HSV-1). Noninfectious corneal infiltrates related to trauma, collagen vascular disease, autoimmune inflammation, vasculitis, or atopy (which predisposes to HSV keratitis) must be considered. HSV-associated stromal keratitis is the most common cause of infectious corneal blindness in the United States, yet its presentation can be fairly subtle. For this reason, symptoms out of proportion to exam findings or a history con
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Van Calsteren, Kristel. Chronic maternal infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0050.

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Pregnant women diagnosed with chronic infections are a worldwide problem. In developed countries, the most frequently encountered are hepatitis B and C, toxoplasmosis, syphilis, herpes simplex, and Cytomegalovirus infections. In developing countries, human immunodeficiency virus and malaria are also seen commonly in pregnant women. Maternal infections are associated with various complications in pregnant women, but also with congenital infections with or without structural anomalies and long-term sequelae, fetal growth restriction, preterm delivery, and perinatal mortality. Moreover, increasin
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Sexually transmitted diseases sourcebook: Basic consumer health information about sexual health and the screening, diagnosis, treatment, and prevention of common sexually transmitted diseases (STDs), including chancroid, chlamydia, gonorrhea, herpes, hepatitis, human immunodeficiency virus/acquired immunodeficiency syndrome (HIV/AIDS), human papillomavirus (HPV), syphilis, and trichomoniasis ; along with facts about risk factors and complications, trends and disparities in infection rates, tips for discussing STDs with sexual partners, a glossary of related terms, and resources for additional help and information. 5th ed. Omnigraphics, Inc., 2013.

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