Gotowe bibliografie tematyczne / HlyA

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Gotowa bibliografia na temat „HlyA”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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Artykuły w czasopismach na temat "HlyA"

Robertson, Kirstin P., C. Jeffrey Smith, Andrea M. Gough, and Edson R. Rocha. "Characterization of Bacteroides fragilis Hemolysins and Regulation and Synergistic Interactions of HlyA and HlyB." Infection and Immunity 74, no. 4 (2006): 2304–16. http://dx.doi.org/10.1128/iai.74.4.2304-2316.2006.

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ABSTRACT This study describes the presence of 10 hemolysin orthologs in the genome of the opportunistic human anaerobic pathogen Bacteroides fragilis, which is currently classified as a nonhemolytic bacterium. The hemolysins were designated HlyA through HlyI plus HlyIII. All cloned hemolysin genes were able to confer hemolytic activity to a nonhemolytic Escherichia coli strain on blood agar plates. Interestingly, HlyH was found to be present in the genome of the B. fragilis NCTC9343 strain but absent in strains 638R, YCH46, and Bacteroides thetaiotaomicron VPI-5482. The hemolysins HlyA, HlyB,

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Sugamata, Yasuhiro, and Toshikazu Shiba. "Improved Secretory Production of Recombinant Proteins by Random Mutagenesis of hlyB, an Alpha-Hemolysin Transporter from Escherichia coli." Applied and Environmental Microbiology 71, no. 2 (2005): 656–62. http://dx.doi.org/10.1128/aem.71.2.656-662.2005.

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ABSTRACT Fusion proteins with an alpha-hemolysin (HlyA) C-terminal signal sequence are known to be secreted by the HlyB-HlyD-TolC translocator in Escherichia coli. We aimed to establish an efficient Hly secretory expression system by random mutagenesis of hlyB and hlyD. The fusion protein of subtilisin E and the HlyA signal sequence (HlyA218) was used as a marker protein for evaluating secretion efficiency. Through screening of more than 1.5 × 104 E. coli JM109 transformants, whose hlyB and hlyD genes had been mutagenized by error-prone PCR, we succeeded in isolating two mutants that had 27- a

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Pimenta, A. L., K. Racher, L. Jamieson, M. A. Blight, and I. B. Holland. "Mutations in HlyD, Part of the Type 1 Translocator for Hemolysin Secretion, Affect the Folding of the Secreted Toxin." Journal of Bacteriology 187, no. 21 (2005): 7471–80. http://dx.doi.org/10.1128/jb.187.21.7471-7480.2005.

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ABSTRACT HlyD, a member of the membrane fusion protein family, is essential for the secretion of the RTX hemolytic toxin HlyA from Escherichia coli. Random point mutations affecting HlyA secretion were obtained, distributed in most periplasmic regions of the HlyD molecule. Analysis of the secretion phenotypes of different mutants allowed the identification of regions in HlyD involved in different steps of HlyA translocation. Four mutants, V349-I, T85-I, V334-I and L165-Q, were conditionally defective, a phenotype shown to be linked to the presence of inhibitory concentrations of Ca2+ in extrac

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Stanley, Peter, Vassilis Koronakis, and Colin Hughes. "Acylation of Escherichia coli Hemolysin: A Unique Protein Lipidation Mechanism Underlying Toxin Function." Microbiology and Molecular Biology Reviews 62, no. 2 (1998): 309–33. http://dx.doi.org/10.1128/mmbr.62.2.309-333.1998.

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SUMMARY The pore-forming hemolysin (HlyA) of Escherichia coli represents a unique class of bacterial toxins that require a posttranslational modification for activity. The inactive protoxin pro-HlyA is activated intracellularly by amide linkage of fatty acids to two internal lysine residues 126 amino acids apart, directed by the cosynthesized HlyC protein with acyl carrier protein as the fatty acid donor. This action distinguishes HlyC from all bacterial acyltransferases such as the lipid A, lux-specific, and nodulation acyltransferases, and from eukaryotic transferases such as N-myristoyl tra

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Reimann, Sven, Gereon Poschmann, Kerstin Kanonenberg, Kai Stühler, Sander H. J. Smits, and Lutz Schmitt. "Interdomain regulation of the ATPase activity of the ABC transporter haemolysin B from Escherichia coli." Biochemical Journal 473, no. 16 (2016): 2471–83. http://dx.doi.org/10.1042/bcj20160154.

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Type 1 secretion systems (T1SS) transport a wide range of substrates across both membranes of Gram-negative bacteria and are composed of an outer membrane protein, a membrane fusion protein and an ABC (ATP-binding cassette) transporter. The ABC transporter HlyB (haemolysin B) is part of a T1SS catalysing the export of the toxin HlyA in E. coli. HlyB consists of the canonical transmembrane and nucleotide-binding domains. Additionally, HlyB contains an N-terminal CLD (C39-peptidase-like domain) that interacts with the transport substrate, but its functional relevance is still not precisely defin

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Zaitseva, J., S. Jenewein, C. Oswald, T. Jumpertz, I. B. Holland, and L. Schmitt. "A molecular understanding of the catalytic cycle of the nucleotide-binding domain of the ABC transporter HlyB." Biochemical Society Transactions 33, no. 5 (2005): 990–95. http://dx.doi.org/10.1042/bst0330990.

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The ABC transporter (ATP-binding-cassette transporter) HlyB (haemolysin B) is the central element of a type I secretion machinery, dedicated to the secretion of the toxin HlyA in Escherichia coli. In addition to the ABC transporter, two other indispensable elements are necessary for the secretion of the toxin across two membranes in a single step: the transenvelope protein HlyD and the outer membrane protein TolC. Despite the fact that the hydrolysis of ATP by HlyB fuels secretion of HlyA, the essential features of the underlying transport mechanism remain an enigma. Similar to all other ABC t

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Lee, Woojung, Soohyun Sung, Jina Ha, et al. "Molecular and Genomic Analysis of the Virulence Factors and Potential Transmission of Hybrid Enteropathogenic and Enterotoxigenic Escherichia coli (EPEC/ETEC) Strains Isolated in South Korea." International Journal of Molecular Sciences 24, no. 16 (2023): 12729. http://dx.doi.org/10.3390/ijms241612729.

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Hybrid strains Escherichia coli acquires genetic characteristics from multiple pathotypes and is speculated to be more virulent; however, understanding their pathogenicity is elusive. Here, we performed genome-based characterization of the hybrid of enteropathogenic (EPEC) and enterotoxigenic E. coli (ETEC), the strains that cause diarrhea and mortality in children. The virulence genes in the strains isolated from different sources in the South Korea were identified, and their phylogenetic positions were analyzed. The EPEC/ETEC hybrid strains harbored eae and est encoding E. coli attaching and

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Masin, Jiri, Adriana Osickova, David Jurnecka, et al. "Retargeting from the CR3 to the LFA-1 receptor uncovers the adenylyl cyclase enzyme–translocating segment of Bordetella adenylate cyclase toxin." Journal of Biological Chemistry 295, no. 28 (2020): 9349–65. http://dx.doi.org/10.1074/jbc.ra120.013630.

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The Bordetella adenylate cyclase toxin-hemolysin (CyaA) and the α-hemolysin (HlyA) of Escherichia coli belong to the family of cytolytic pore-forming Repeats in ToXin (RTX) cytotoxins. HlyA preferentially binds the αLβ2 integrin LFA-1 (CD11a/CD18) of leukocytes and can promiscuously bind and also permeabilize many other cells. CyaA bears an N-terminal adenylyl cyclase (AC) domain linked to a pore-forming RTX cytolysin (Hly) moiety, binds the complement receptor 3 (CR3, αMβ2, CD11b/CD18, or Mac-1) of myeloid phagocytes, penetrates their plasma membrane, and delivers the AC enzyme into the cytos

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Wang, Changying, Qianqian Li, Junqiang Lv, et al. "Alpha-hemolysin of uropathogenic Escherichia coli induces GM-CSF-mediated acute kidney injury." Mucosal Immunology 13, no. 1 (2019): 22–33. http://dx.doi.org/10.1038/s41385-019-0225-6.

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Abstract Uropathogenic Escherichia coli (UPEC) is the leading cause of urinary tract infections (UTIs), inducing acute pyelonephritis and may result in permanent renal scarring and failure. Alpha-hemolysin (HlyA), a key UPEC toxin, causes serious tissue damage; however, the mechanism through which HlyA induces kidney injury remains unclear. In the present study, granulocyte-macrophage colony-stimulating factor (GM-CSF) secreted by renal epithelial cells was upregulated by HlyA in vitro and in vivo, which induced M1 macrophage accumulation in kidney, and ADAM10 was found involved in HlyA-induce

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Jumpertz, Thorsten, Christian Chervaux, Kathleen Racher, et al. "Mutations affecting the extreme C terminus of Escherichia coli haemolysin A reduce haemolytic activity by altering the folding of the toxin." Microbiology 156, no. 8 (2010): 2495–505. http://dx.doi.org/10.1099/mic.0.038562-0.

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Escherichia coli haemolysin A (HlyA), an RTX toxin, is secreted probably as an unfolded intermediate, by the type I (ABC transporter-dependent) pathway, utilizing a C-terminal secretion signal. However, the mechanism of translocation and post-translocation folding is not understood. We identified a mutation (hlyA99) at the extreme C terminus, which is dominant in competition experiments, blocking secretion of the wild-type toxin co-expressed in the same cell. This suggests that unlike recessive mutations which affect recognition of the translocation machinery, the hlyA99 mutation interferes wi

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Rozprawy doktorskie na temat "HlyA"

González, Antonio Pablo. "IDENTIFICACIÓN DE LOS GENES: Stx1, Stx2, eaeA Y hlyA, EN CEPAS DE Escherichia coli AISLADAS DE CANALES Y CARNE PROCESADA DE OVINOS." Tesis de Licenciatura, Universidad Autónoma del Estado de México, 2018. http://hdl.handle.net/20.500.11799/94389.

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E. coli productora de toxina shiga (ECST) y E. coli enterohemorrágica (ECEH) pertenecen a los grupos patógenos de Escherichia coli causantes de enfermedades diarreicas. La presencia de ECST y ECEH en estas enfermedades diarreicas se atribuye principalmente al consumo de vegetales y carnes provenientes de bovinos y ovinos contaminados con estos grupos patógenos y que actualmente se encuentran dentro de las etiologías más importantes del grupo de Enfermedades Trasmitidas por Alimentos (ETA). ECST y ECEH presentan los genes Stx1, Stx2, eaeA y hlyA que codifican a proteínas con cualidades toxicas

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Holden, James Anthony, and jamesholden@netspace net au. "Vaccination Strategies for the Prevention of Swine Dysentery." RMIT University. Applied Sciences, 2006. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20070112.122102.

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The SmpA outer membrane lipoprotein of B. hyodysenteriae has several characteristics that indicate the potential to protect against swine dysentery (SD). It localises to the outer membrane and antibodies directed against SmpA can prevent the growth of B. hyodysenteriae in vitro. There is some variation observed in the distribution and expression of the SmpA lipoprotein, suggesting that vaccination with SmpA may not provide protection against challenge with a heterologous B. hyodysenteriae strain. This study has characterised the variation at the smpA locus, and in the process has identified

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Diabaté, Mamady. "Étude des relations fonctionnelles entre les toxines CNF1 et alpha-hémolysine (HlyA) des Escherichia coli uropathogènes." Nice, 2011. http://www.theses.fr/2011NICE4039.

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Le facteur cytotoxique nécrosant-1 (CNF1) et l’hémolysine-alpha (HlyA) sont les deux toxines majeures des Escherichia coli uropathogènes (UPEC), premier agent étiologique des infections du tractus urinaire (ITU) et principale cause de bactériémie à E. Coli. Comme plusieurs autres facteurs de virulence, ces toxines ont été associées à l’ITU en raison de leur grande prévalence dans les UPEC par rapport aux E. Coli fécaux, respectivement 30 % et 0. 9 % pour CNF1. Cependant, leurs rôles dans la physiopathologie de l’infection urinaire reste imprécis. Dans un modèle murin de bactériémie, nous avons

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Ha, Thi Quyen. "Analysis of gene encoding haemolysin A of Vibrio cholerae isolated in Vietnam." Technische Universität Dresden, 2018. https://tud.qucosa.de/id/qucosa%3A33123.

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Vibrio cholerae is the cholera causing agent, divided into two biotypes, including the classical biotype and ElTor biotype. Both of these biotypes caused cholera epidemics in the world. The classical biotype caused 6th cholera pandemic (from 1921 to 1961), and ElTor biotype caused 7th cholera pandemic (from 1961 to the 70s). Haemolysin A, a hemolytic protein of V. cholerae ElTor biotype, is encoded by the hlyA gene. This gene is often used for analyzing genetic relationship between strains in the same species or between species in the same Vibrio genus. Results of analyzing nucleotide and amin

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Guzmán-Verri, Caterina. "Virulence mechanisms of two Gram negative bacteria : studies on Escherichia coli hemolysin HlyA and on the interaction of Brucella abortus with non-phagocytic cells /." Stockholm : Karolinska Univ. Press, 2002. http://diss.kib.ki.se/2002/91-7349-114-4.

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Franco, Roger Teixeira. "Caracterização de amostras de Escherichia coli eae positivas isoladas de crianças com diarreia aguda e sem diarreia em Belo Horizonte: tipagem de intimina e pesquisa de hlyA, iha e toxB." Universidade Federal de Minas Gerais, 2012. http://hdl.handle.net/1843/ENMS-8RWQMM.

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Infectious diarrhea is still a main cause of morbidity and mortality among children from the less developed areas of the world. Among several etiologic agents, a group of diarrheagenic Escherichia coli (DEC) named attaching and effacing E. coli (AEEC), associated to the genesis of the intimin-mediated A/E lesion in enterocytes, should be mentioned. AEEC includes typical (tEPEC) and atypical (aEPEC) subgroups of enteropathogenic E. coli (EPEC) and the pathotype enterohemorrhagic E. coli (EHEC) that also expresses shiga toxin. All of them, mainly aEPEC, are genetically diverse, especially in reg

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Ping, Ivan Chang Kok. "HLA performance measurement." Thesis, Monterey, Calif. : Springfield, Va. : Naval Postgraduate School ; Available from National Technical Information Service, 2000. http://handle.dtic.mil/100.2/ADA376484.

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Thesis (M.S. in Modeling, Virtual Environments and Simulation (MOVES))--Naval Postgraduate School, March 2000.<br>Thesis advisor(s): Zyda, Michael ; Bachmann, Eric. "March 2000." Includes bibliographical references (p. 77). Also available in print.

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Lachaud, Laurence. "Reconnaissance allogénique HLA." Montpellier 1, 1995. http://www.theses.fr/1995MON11145.

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FODIL, NASSIMA. "Nouvelle diversite hla." Université Louis Pasteur (Strasbourg) (1971-2008), 2001. http://www.theses.fr/2001STR1A001.

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Au cours de ces 10 dernieres annees, de nombreux genes homologues aux molecules de classes i du complexe majeur d'histocompatibilite (cmh) ont ete mis en evidence. Ils exhibent, pour la plupart, une expression tissulaire restreinte et ne semblent pas avoir pour fonction la presentation peptidique aux cellules t. La famille des genes mic (mhc class i chain-related genes) a recemment ete caracterisee au sein meme de la region de classe i du cmh. Elle se compose de 2 genes fonctionnels (mica et micb) ainsi que de 5 pseudogenes (micc-g). Les travaux presentes dans cette these decrivent le polymorp

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Brown, Juliette. "HLA-DR and HLA-DQ polymorphism and associations in different populations." Thesis, Queen Mary, University of London, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.287875.

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Książki na temat "HlyA"

Boegel, Sebastian, ed. HLA Typing. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8546-3.

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Boegel, Sebastian, ed. HLA Typing. Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3874-3.

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Zelenjuk, Marʹjana. Hlyna sliv: Zbirka viršiv. Zerna, 2018.

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Honda, Yutaka, and Takeo Juji, eds. HLA in Narcolepsy. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-83387-8.

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Lee, John, ed. The HLA System. Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3454-8.

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Dupont, Bo, ed. Immunobiology of HLA. Springer New York, 1989. http://dx.doi.org/10.1007/978-1-4612-3552-1.

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Dupont, Bo, ed. Immunobiology of HLA. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-662-39946-0.

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J, Crumpton M., ed. HLA in medicine. Churchill Livingstone for the British Council, 1987.

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Bo, Dupont, Ceppellini Ruggero, and International Histocompatibility Workshop and Conference (10th : 1987 : Princeton, N.J. and New York, N.Y.), eds. Immunobiology of HLA. Springer-Verlag, 1989.

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Ping, Ivan Chang Kok. HLA performance measurement. Naval Postgraduate School, 2000.

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Części książek na temat "HlyA"

Spierings, Eric, Alejandro Madrigal, and Katharina Fleischhauer. "Histocompatibility." In The EBMT Handbook. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-44080-9_9.

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AbstractHuman leukocyte antigen (HLA) molecules are the most important histocompatibility antigens, due to their genetic polymorphism and their key role in peptide antigen presentation and T-cell alloreactivity. While full matching for the most relevant HLA loci had been regarded as a prerequisite for successful transplantation until recently, the introduction of posttransplant cyclophosphamide (PTCy) as immune prophylaxis has also allowed successful transplantation across multiple HLA mismatches, thus also enabling access to transplantation for patients without a fully compatible donor. The r

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Bodmer, Walter F. "HLA 1987." In Immunobiology of HLA. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-662-39946-0_1.

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Tiwari, Jawahar L., and Paul I. Terasaki. "HLA Nomenclature." In HLA and Disease Associations. Springer New York, 1985. http://dx.doi.org/10.1007/978-1-4613-8545-5_1.

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Hoppe, Immo. "HLA-Antigene." In INSTAND-Schriftenreihe. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-69800-2_38.

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Stöcker, W. "HLA-Allele." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_1459-1.

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Kleesiek, K., C. Götting, J. Diekmann, J. Dreier, and M. Schmidt. "HLA-Antikörper." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-49054-9_1460-1.

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Krüger, C., and W. Stöcker. "HLA-B27." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_1462-1.

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Kleesiek, K., C. Götting, J. Diekmann, J. Dreier, and M. Schmidt. "HLA-Crossmatch." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-49054-9_1463-1.

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Renz, H., and B. Gierten. "HLA-DR." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_1464-1.

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Stöcker, W. "HLA-Allele." In Springer Reference Medizin. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_1459.

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Streszczenia konferencji na temat "HlyA"

Wu, Lingwei, Quanjun Liu, Zhongwei Wu, and Zuhong Lu. "Detection of hlyA Gene of Listeria Monocytogenes with Electrochemical DNA Biosensor." In 2008 2nd International Conference on Bioinformatics and Biomedical Engineering. IEEE, 2008. http://dx.doi.org/10.1109/icbbe.2008.95.

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Silva, IMT, LA Caldeira, LO Sant’anna, LF Seabra, LS Santos, and MRB Araújo. "RELEVÂNCIA DAS METODOLOGIAS AUTOMATIZADAS E ESTUDO GÊNICO NO ISOLAMENTO E NA IDENTIFICAÇÃO DE VIBRIO CHOLERAE." In Resumos do 54º Congresso Brasileiro de Patologia Clínica/Medicina Laboratorial. Zeppelini Editorial e Comunicação, 2022. http://dx.doi.org/10.5327/1516-3180.140s1.6152.

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Objetivo: Reportar a identificação de V. cholerae em hemocultura. Método: Relato de caso a partir das análises microbiológicas de amostra de sangue com identificação de V. cholerae confirmada por espectrometria de massa MALDI-TOF. Conclusão: Paciente do sexo masculino, 57 anos, renal crônico, dialítico, apresentando febre alta, mialgia, diarreia aguda e sonolência, evoluindo com tosse seca, dispneia e anemia crônica, foi encaminhado a UTI. A hemocultura positivou pelo sistema BactAlert®, e ao Gram, visualizaram-se bacilos Gram negativos curvos. O isolado bacteriano foi identificado como V. cho

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Slukin, P. V., L. V. Kolupaeva, N. A. Slukina, N. N. Podgornaja, and N. K. Fursova. "VIRULENCE OF UROPATHOGENIC ESCHERICHIA COLI CARRYING HLYA AND CNF1 GENES FOR GALLERIA MELLONELLA LARVAE." In Molecular Diagnostics and Biosafety. Federal Budget Institute of Science 'Central Research Institute for Epidemiology', 2020. http://dx.doi.org/10.36233/978-5-9900432-9-9-241.

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Torres, Margareth Afonso, Gabriella Camerini Maciel, Eliane Aparecida Rosseto Welter, et al. "Avaliação da acurácia da genotipagem HLA-DQ e detecção de autoanticorpos no diagnóstico da doença celíaca." In Resumos do 56º Congresso Brasileiro de Patologia Clínica/Medicina Laboratorial. Zeppelini Editorial e Comunicação, 2024. https://doi.org/10.5327/1516-3180.142s1.12082.

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Objetivo: A doença celíaca é uma condição inflamatória imunomediada desencadeada pela exposição ao glúten. Os heterodímeros HLA-DQ2 (HLA-DQ2.5: DQA1*05:01/DQB1*02:01 e HLA-DQ2.2:DQA1*02:01/DQB1*02:02) e HLA-DQ8 (DQA1*03:01/DQB1*03:02) estão associados ao maior risco do desenvolvimento da doença celíaca. O diagnóstico é realizado com a pesquisa dos autoanticorpos anti-transglutaminase tecidual IgA (anti-tTG), anti-endomísio e biópsia duodenal. O objetivo deste estudo foi avaliar a acurácia da identificação de HLA-DQ2/DQ8, a detecção de anticorpos anti-tTG IgA e anti-endomísio IgA em relação à b

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Allen, Robert. "HLA." In Joint proceedings of the second international software architecture workshop (ISAW-2) and international workshop on multiple perspectives in software development (Viewpoints '96). ACM Press, 1996. http://dx.doi.org/10.1145/243327.243626.

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Kieffer, N., M. Titeux, A. Henri, J. Breton-Gorius, and W. Vainchenker. "MEGAKARYOCYTIC ORIGIN OF PLATELET HLA CLASS I ANTIGEN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643546.

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The existence of HLA class I antigens on human platelets is well established. However, several authors have suggested that platelet HLA antigens are not integral membrane components but are acquired from soluble plasma sources and adsorbed to the platelet surface.In the present study, we used the monoclonal antibody W6/32, directed against a monomorphic epitope of the HLA class I antigen for the immunochemical characterization of platelet HLA. Immunoprecipitation experiments, performed after in vitro metabolic radiolabeling of human platelets revealed a band of molecular weight 44,000 identica

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Galli, Emanuele, Gaetano Cavarretta, and Salvatore Tucci. "HLA-OMNET++: An HLA Compliant Network Simulator." In 2008 12th IEEE International Symposium on Distributed Simulation and Real-Time Applications (DS-RT). IEEE, 2008. http://dx.doi.org/10.1109/ds-rt.2008.44.

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dos Santos, Gabriel Cardoso, and Raul Ceretta Nunes. "An Approach to Build Source Code for HLA-based Distributed Simulations." In Anais Estendidos do Simpósio Brasileiro de Engenharia de Sistemas Computacionais. Sociedade Brasileira de Computação - SBC, 2022. http://dx.doi.org/10.5753/sbesc_estendido.2022.228111.

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IEEE 1516-2010 High Level Architecture (HLA) is a standard used to build simulators that support interoperability. This standard requests a set of rules described in a Federation Object Model (FOM), which is an agreement for the simulation interoperability. In this context, developing a simulation with several simulators is a challenging task for developers due to the complexity of the HLA in handling the data provided in the FOM. Tools and techniques that seek to optimize the development process of simulators based on HLA have been emerging in recent years, bringing different types of approac

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Liu, Pingan, Lei Li, Wei Heng, and Boyuan Wang. "HLDA based text clustering." In 2012 IEEE 2nd International Conference on Cloud Computing and Intelligence Systems (CCIS). IEEE, 2012. http://dx.doi.org/10.1109/ccis.2012.6664628.

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Chandiok, Prateek, Sanjay Poonia, Soumen Kundu, and Anil Kant Bharti. "Hydraulic Lash Adjuster Dynamic Behavior Prediction Using Multibody Dynamic Simulation and Co-Relation with Testing to Improve Valve Train Design Robustness." In 11th SAEINDIA International Mobility Conference (SIIMC 2024). SAE International, 2024. https://doi.org/10.4271/2024-28-0184.

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<div class="section abstract"><div class="htmlview paragraph">In recent years, world-wide automotive manufacturers have been continuously working to improve the fuel efficiency of IC engine and valve train friction contribute up to 30% of overall friction loss. Oil viscosity plays an important role in reducing overall engine friction, but it adversely affects the function of Valve train in terms of wear and reliability.</div><div class="htmlview paragraph">Now a days HLA/RFF type (Type-II) valve train is mostly used in Internal Combustion engine to reduce friction and a

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Raporty organizacyjne na temat "HlyA"

หังสสูต, ปกรัฐ, та ประทานพร แก้วปรีดี. ประสิทธิภาพในฐานะเป็นกลไกหลบหลีกภูมิคุ้มกันผ่านทีเซลล์ต่อเอชไอวี. จุฬาลงกรณ์มหาวิทยาลัย, 2017. https://doi.org/10.58837/chula.res.2017.13.

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วัตถุประสงค์: เพื่อเปรียบเทียบการตอบสนองของทีเซลล์และหากลไกที่เชื้อไวรัสเอชไอวีใช้ในการหลบหลีกจากภูมิคุ้มกัน วิธีดำเนินการทดลอง: รวบรวมอาสาสมัครที่ติดเชื้อเอชไอวี ที่มีระดับซีดีสี่มากกว่า 450 cells/µL จากคลินิกนิรนาม สภากาชาดไทย โดยแบ่งอาสาสมัครเป็น 2 กลุ่ม ตามปริมาณไวรัสในกระแสเลือด (VL) เป็น viraemic controllers (VC) (VL<2,000 cp/mL) และ non-controllers (NC) (VL>2,000 cp/mL) จากนั้นเก็บเลือดเพื่อนำมาทดสอบการตอบสนองต่อโปรตีนของเชื้อเอชไอวีส่วน Gag p24 ด้วยวิธี IFNy ELISpot assay และทำการหาลำดับสารพันธุกรรมเพื่อใช้ในการวิเคราะห์กลไกที่ใช้ในการหลบหลีกจากภูมิคุ้มกัน ผลการทดลอง: VC มีจำนวน

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หังสสูต, ปกรัฐ, та นวพล เตชะเกรียงไกร. บทบาทของทีเซลล์ต่อการวิวัฒนาการของเอชไอวี : ข้อมูลสำคัญสำหรับการพัฒนาวัคซีนป้องกันโรคเอดส์ : รายงานการวิจัย. จุฬาลงกรณ์มหาวิทยาลัย, 2011. https://doi.org/10.58837/chula.res.2011.31.

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ในการพัฒนาวัคซีน HIV เราจำเป็นต้องมีความรู้ในกลไกการป้องกันการติดเชื้อไวรัสชนิดนี้ก่อนการศึกษาภูมิคุ้มกันในผู้ติดเชื้อ HIV ที่มีความสามารถในการควบคุมปริมาณ HIV ได้ตามธรรมชาติซึ่งมีปริมาณ HIV-RNA น้อยกว่า 2000 copies/ml สร้างโอกาสในการวิเคราะห์และค้นหาว่ากลไกอะไรที่ทำให้ผู้ติดเชื้อเหล่านี้สามารถควบคุมไวรัสได้ ผู้วิจัยรับสมัครผู้ที่ควบคุมไวรัสได้ดี (viraemic controllers, VC) จำนวน 13 คน และผู้ที่ควบคุมไวรัสได้ตามปกติ (typical progressor, TP) 32 คน อาสาสมัครทุกรายได้รับการวิเคราะห์ complete blood count, CD4 และ CD8 ตลอดจนปริมาณ plasma HIV-RNA ผู้วิจัยวิเคราะห์ลำดับกรดอะมิโนของโปรตีน p24 จากอาสาสม

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O'Day, Stephen C., and John W. McMaster. The ACETEF HLA Interface. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada375781.

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GOTTLIEB, ERIC JOSEPH, MICHAEL J. MCDONALD, and FRED J. OPPEL, III. Umbra's High Level Architecture (HLA) Interface. Office of Scientific and Technical Information (OSTI), 2002. http://dx.doi.org/10.2172/800785.

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Coppo, Patricia A., Judy W. Davis, and Steve M. Spellman. HLA Typing for Bone Marrow Transplantation. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada462775.

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Black, Jerry. Data Collection in an HLA Federation. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada378558.

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Spellman, Stephen. HLA Typing for Bone Marrow Transplantation. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada546709.

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Setterholm, Michelle, Judy W. Davis, and Steve M. Spellman. HLA Typing for Bone Marrow Transplantation. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada473611.

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Paterson, Daniel J., Eric Anschuetz, Mark Biddle, Dave Kotick, and Thai Nguyen. Architecture Issues for DIS-TO-HLA Conversion. Defense Technical Information Center, 1997. http://dx.doi.org/10.21236/ada332944.

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Dingel, Juergen, David Garlan, and Craig A. Damon. A Feasibility Study of the HLA Bridge. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada461048.

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