Gotowe bibliografie tematyczne / Hormones, Sex – Receptors

Spis treści

  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji

Gotowa bibliografia na temat „Hormones, Sex – Receptors”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 9 lutego 2022

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Artykuły w czasopismach na temat "Hormones, Sex – Receptors"

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Honma, Naoko, Yoko Matsuda, and Tetuo Mikami. "Carcinogenesis of Triple-Negative Breast Cancer and Sex Steroid Hormones." Cancers 13, no. 11 (May 25, 2021): 2588. http://dx.doi.org/10.3390/cancers13112588.

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Triple-negative breast cancer (TNBC) lacks an effective treatment target and is usually associated with a poor clinical outcome; however, hormone unresponsiveness, which is the most important biological characteristic of TNBC, only means the lack of nuclear estrogenic signaling through the classical estrogen receptor (ER), ER-α. Several sex steroid receptors other than ER-α: androgen receptor (AR), second ER, ER-β, and non-nuclear receptors represented by G-protein-coupled estrogen receptor (GPER), are frequently expressed in TNBC and their biological and clinical importance has been suggested
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Zarazúa, Abraham, Aliesha González-Arenas, Gabriela Ramírez-Vélez, Blanca Bazán-Perkins, Christian Guerra-Araiza, and María G. Campos-Lara. "Sexual Dimorphism in the Regulation of Estrogen, Progesterone, and Androgen Receptors by Sex Steroids in the Rat Airway Smooth Muscle Cells." International Journal of Endocrinology 2016 (2016): 1–11. http://dx.doi.org/10.1155/2016/8423192.

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The role of sex hormones in lung is known. The three main sex steroid receptors, estrogen, progesterone, and androgen, have not been sufficiently studied in airway smooth muscle cells (ASMC), and the sex hormone regulation on these receptors is unknown. We examined the presence and regulation of sex hormone receptors in female and male rat ASMC by Western blotting and flow cytometry. Gonadectomized rats were treated with 17β-estradiol, progesterone, 17β-estradiol + progesterone, or testosterone. ASMC were enzymatically isolated from tracheas and bronchi. The experiments were performed with dou
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Baber, Rod. "Sex hormones, receptors and modulators." Australian and New Zealand Journal of Obstetrics and Gynaecology 57, no. 4 (August 2017): 391–92. http://dx.doi.org/10.1111/ajo.12666.

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Orshal, Julia M., and Raouf A. Khalil. "Gender, sex hormones, and vascular tone." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 286, no. 2 (February 2004): R233—R249. http://dx.doi.org/10.1152/ajpregu.00338.2003.

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The greater incidence of hypertension and coronary artery disease in men and postmenopausal women compared with premenopausal women has been related, in part, to gender differences in vascular tone and possible vascular protective effects of the female sex hormones estrogen and progesterone. However, vascular effects of the male sex hormone testosterone have also been suggested. Estrogen, progesterone, and testosterone receptors have been identified in blood vessels of human and other mammals and have been localized in the plasmalemma, cytosol, and nuclear compartments of various vascular cell
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Fannon, Stacey A., Regina M. Vidaver, and Sherry A. Marts. "Historical Perspectives: An abridged history of sex steroid hormone receptor action." Journal of Applied Physiology 91, no. 4 (October 1, 2001): 1854–59. http://dx.doi.org/10.1152/jappl.2001.91.4.1854.

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The field of steroid hormone action is well established, although it is barely more than four decades old. Pivotal experiments in the late 1950s and 1960s showed that hormone-binding components exist within nuclei of target tissues and that steroid hormones act by regulating gene expression, rather than directly influencing enzymatic processes. The understanding that steroid hormone receptors interact with the general transcription machinery and alter chromatin structure came in the 1970s and 1980s, and details of this mechanism continue to be elucidated. In addition, the discovery of rapid ce
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Simão, Vinícius Augusto, Luiz Gustavo de Almeida Chuffa, and Isabel Cristina Cherici Camargo. "Ovarian sex steroid receptors and sex hormones in androgenized rats." Reproduction 152, no. 5 (November 2016): 545–59. http://dx.doi.org/10.1530/rep-16-0233.

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This study evaluated for the first time the effects of different doses of the anabolic steroid nandrolone decanoate (ND) on the expression of ovarian steroid receptors (AR, ER-α (ESR1) and ER-β (ESR2)) and related sex hormones after treatment and recovery periods in adult rats. The animals were injected subcutaneously with doses of ND (1.87, 3.75, 7.5 or 15 mg/kg b.w.) or mineral oil (control group) for 15 days, and the experimental groups were divided into three periods of evaluation: (a) ND treatment for 15 days, (b) ND treatment and recovery for a period of 30 days and (c) ND treatment and
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Bhatia, Aruna, Harmandeep Kaur Sekhon, and Gurpreet Kaur. "Sex Hormones and Immune Dimorphism." Scientific World Journal 2014 (2014): 1–8. http://dx.doi.org/10.1155/2014/159150.

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The functioning of the immune system of the body is regulated by many factors. The abnormal regulation of the immune system may result in some pathological conditions. Sex hormones of reproductive system are one of the major factors that regulate immune system due to the presence of hormone receptors on immune cells. The interaction of sex hormones and immune cells through the receptors on these cells effect the release of cytokines which determines the proliferation, differentiation, and maturation of different types of immunocytes and as a result the outcome of inflammatory or autoimmune dis
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Simoncini, T., and AR Genazzani. "Non-genomic actions of sex steroid hormones." European Journal of Endocrinology 148, no. 3 (March 1, 2003): 281–92. http://dx.doi.org/10.1530/eje.0.1480281.

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Steroid hormone receptors have been traditionally considered to act via the regulation of transcriptional processes, involving nuclear translocation and binding to specific response elements, and ultimately leading to regulation of gene expression. However, novel non-transcriptional mechanisms of signal transduction through steroid hormone receptors have been identified. These so-called 'non-genomic' effects do not depend on gene transcription or protein synthesis and involve steroid-induced modulation of cytoplasmic or cell membrane-bound regulatory proteins. Several relevant biological actio
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Higa, Gerald M., and Ryan G. Fell. "Sex Hormone Receptor Repertoire in Breast Cancer." International Journal of Breast Cancer 2013 (2013): 1–14. http://dx.doi.org/10.1155/2013/284036.

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Classification of breast cancer as endocrine sensitive, hormone dependent, or estrogen receptor (ER) positive refers singularly to ERα. One of the oldest recognized tumor targets, disruption of ERα-mediated signaling, is believed to be the mechanistic mode of action for all hormonal interventions used in treating this disease. Whereas ERαis widely accepted as the single most important predictive factor (for response to endocrine therapy), the presence of the receptor in tumor cells is also of prognostic value. Even though the clinical relevance of the two other sex hormone receptors, namely, E
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Villablanca, Amparo C., Muthuvel Jayachandran, and Carole Banka. "Atherosclerosis and sex hormones: current concepts." Clinical Science 119, no. 12 (September 1, 2010): 493–513. http://dx.doi.org/10.1042/cs20100248.

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CVD (cardiovascular disease) is the leading cause of death for women. Considerable progress has been made in both our understanding of the complexities governing menopausal hormone therapy and our understanding of the cellular and molecular mechanisms underlying hormone and hormone receptor function. Understanding the interplay of atherosclerosis and sex steroid hormones and their cognate receptors at the level of the vessel wall has important ramifications for clinical practice. In the present review, we discuss the epidemiology of CVD in men and women, the clinical impact of sex hormones on
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Rozprawy doktorskie na temat "Hormones, Sex – Receptors"

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ElBaradie, Khairat Bahgat. "Membrane effects of sex hormones on growth plate chondrocytes." Diss., Georgia Institute of Technology, 2012. http://hdl.handle.net/1853/45956.

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Understanding and studying the normal bone growth and development is causal. Bone and cartilage tissue provide in addition to their mechanical support, they provide a protection for vital organs such as heart, lung and brain. Longitudinal growth is regulated by the activity of chondrocytes in the epiphyseal growth plates of long bones. Many hormones and growth factors are involved in the regulation of this process. Among these, sex steroids are of crucial importance, especially during puberty. In long bones, endochondral bone formation occurs at the growth plate, a region of developing cartila
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Isaksson, Friman Erika. "Hormonal treatments and the breast : effects on sex steroid receptor expression and proliferation /." Stockholm : [Karolinska institutets bibl.], 2002. http://diss.kib.ki.se/2002/91-7349-182-9/.

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Cluning, Carmel. "Steroid receptor-associated immunophilins : influence of targeted knockdown and altered expression on receptor signalling." University of Western Australia. School of Medicine and Pharmacology, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0215.

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[Truncated abstract] Steroid receptors belong to the superfamily of nuclear receptors, and include the androgen receptor (AR), estrogen receptors (ER[alpha] and ER[beta], glucocorticoid receptor (GR), mineralocorticoid receptor (MR), and the progesterone receptors (PRA and PRB). Before binding ligand, the receptor undergoes biochemical and structural modifications through a series of interactions with molecular chaperones and cochaperones all within a receptor heterocomplex. The mature receptor complexes with the major chaperone Hsp90, the stabilising cochaperone p23, and one member of a group
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Goold, Richard David. "Influence of endogenous female sex-steroids on mutagen metabolism." Thesis, Rhodes University, 1985. http://hdl.handle.net/10962/d1004919.

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Cytochrome P-450, the terminal oxidase of the metabolic mono-oxygenase system, is thought to exist in multiple forms, which have differing substrate specificities, and are variably inducible by different enzyme inducers. Many mutagens, themselves unreactive, require metabolic activation by one or more of these cytochrome P-450-dependent microsomal enzymes for mutagenic activity. Such mutagens may be detected in the Salmonella mutagenicity test only by the incorporation of an hepatic microsomal (59) fraction into the assay (as a first approximation to in vivo metabolism). Induction of the micro
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Trout, Amanda L. "SEX DIFFERENCES IN CELL DEATH AND STEROID HORMONE RECEPTORS IN CORTICAL EXPLANTS." UKnowledge, 2013. http://uknowledge.uky.edu/physiology_etds/6.

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Estrogens, such as the biologically active 17-b estradiol (E2) have many actions in the male and female brain. Not only does E2 regulate reproductive behavior in adults, it organizes and activates the brains of younger animals in a sex-specific manner. In addition, many human studies have shown E2 to provide protection against a variety of neurological disorders, including stoke. These studies have been controversial and depend largely on the type and timing of hormone replacement. Animal studies are much less controversial and clearly demonstrate a neuroprotective role for E2 following ischem
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Williams, Maro R. I. 1974. "Dehydroepiandrosterone action in the cardiovascular system." Monash University, Dept. of Medicine, 2002. http://arrow.monash.edu.au/hdl/1959.1/7927.

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Tasende, Celia. "Pituitary and uterine sex steriod receptors in ewes : seasonal and postpartum anoestrus, oestrous cycle and experimentally induced subnormal luteal phases /." Uppsala : Dept. of Biomedical Sciences and Veterinary Public Health, Swedish University of Agricultural Sciences, 2005. http://epsilon.slu.se/200597.pdf.

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Meikle, Ana. "Reproductive endocrinology of prepubertal and anestrous ewes : regulation of uterine sex steroid receptors by ovarian hormones and effects of estradiol on gonadotropin secretion and follicular growth /." Uppsala : Swedish Univ. of Agricultural Sciences (Sveriges lantbruksuniv.), 2001. http://epsilon.slu.se/avh/2001/91-576-5915-X.pdf.

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Coulombe, Marie-andree. "Implication du sexe, des hormones gonadiques et de leurs métabolites dans la réponse nociceptive et la perception de la douleur." Thesis, Strasbourg, 2013. http://www.theses.fr/2013STRAJ099/document.

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Plusieurs variables biologiques, psychologiques, ainsi que des différences culturelles, ont été mises en cause afin d'expliquer la différence de perception de la douleur existante entre les hommes et les femmes. Il est connu que les hormones gonadiques influencent la réponse nociceptive chez l’animal et chez l’humain. Le cerveau a aussi la capacité de synthétiser ses propres "hormones sexuelles", les neurostéroïdes. L'objectif de cette thèse était: 1) évaluer les facteurs physiologiques et psychologiques influençant de perception de la douleur chez les hommes et les femmes, 2) évaluer l'implic
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Pedroso, Luciana de Britto. "Avaliação da possível diferença na sensibilidade dolorosa de ratos machos e fêmeas e da resposta de cada sexo a crotalfina, um analgésico tipo opióide." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/42/42136/tde-23012012-114018/.

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Evidências clínicas e experimentais têm sugerido a existência de diferenças na sensação de dor e na resposta a fármacos analgésicos entre machos e fêmeas. A crotalfina (CRF), um peptídeo inicialmente isolado e caracterizado no veneno de serpentes Crotalus durissus terrificus, apresenta efeito antinociceptivo, quando avaliado em diferentes modelos experimentais de dor aguda e crônica. O efeito deste peptídeo é de longa duração e mediado pela ativação de receptores opióides periféricos do tipo <font face=\"Symbol\">k e <font face=\"Symbol\">d. Contudo, os estudos com a crotalfina foram sempre re
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Książki na temat "Hormones, Sex – Receptors"

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Gronemeyer, H., U. Fuhrmann, and K. Parczyk, eds. Molecular Basis of Sex Hormone Receptor Function. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-03689-1.

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Sex steroid receptors: Handbook on receptor research. Rome: Field Educational Italia, Acta Medica, 1985.

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O'Malley, Bert. Receptors for Reproductive Hormones. Springer, 2012.

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Moudgil, Virinder K. Molecular Mechanism of Steroid Hormone Action: Recent Advances. Walter De Gruyter Inc, 1985.

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1945-, Moudgil V. K., ed. Molecular mechanism of steroid hormone action: Recent advances. Berlin: Walter de Gruyter, 1985.

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Moudgil, Virinder K. Molecular Mechanism of Steroid Hormone Action: Recent Advances. Walter de Gruyter, 1985.

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Chen, George G. Female Sex Hormones and Cancers. Nova Science Publishers, Incorporated, 2010.

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C, Spelsberg T., and Kumar Rajiv 1949-, eds. Steroid and sterol hormone action. Boston: Nijhoff, 1987.

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Cao, Zhiping. Chemical signaling in neural circuits that mediate sexual behaviors. 1994.

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H, Gronemeyer, Fuhrmann U. 1959-, and Parczyk K. 1960-, eds. Molecular basis of sex hormone receptor function: New targets for intervention. Berlin: Springer-Verlag, 1998.

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Części książek na temat "Hormones, Sex – Receptors"

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Migliaccio, Antimo, Gabriella Castoria, and Ferdinando Auricchio. "Non-Genomic Action of Sex Steroid Hormones." In Nuclear Receptors, 365–79. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-3303-1_15.

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Callard, Ian P., and Gloria V. Callard. "Sex Steroid Receptors and Non-Receptor Binding Proteins." In Hormones and Reproduction in Fishes, Amphibians, and Reptiles, 355–84. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1869-9_12.

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Kovats, Susan, Esther Carreras, and Hemant Agrawal. "Sex Steroid Receptors in Immune Cells." In Sex Hormones and Immunity to Infection, 53–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-02155-8_3.

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Döhler, K. D. "The special case of hormonal imprinting, the neonatal influence of sex." In Development of Hormone Receptors, 103–23. Basel: Birkhäuser Basel, 1987. http://dx.doi.org/10.1007/978-3-0348-9291-9_7.

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Thomas, Peter, Christopher Tubbs, Hakan Berg, and Gwen Dressing. "Sex steroid hormone receptors in fish ovaries." In The Fish Oocyte, 203–33. Dordrecht: Springer Netherlands, 2007. http://dx.doi.org/10.1007/978-1-4020-6235-3_8.

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Spelsberg, Thomas C., Merry Jo Oursler, James P. Landers, Malayannam Subramaniam, Steven A. Harris, and B. Lawrence Riggs. "Human Bone Cells: Newly Discovered Target Cells for Sex Steroids." In Steroid Hormone Receptors: Basic and Clinical Aspects, 447–72. Boston, MA: Birkhäuser Boston, 1994. http://dx.doi.org/10.1007/978-1-4615-9849-7_18.

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Friend, Keith E., and Ian E. McCutcheon. "Estrogen Receptor Expression in the Pituitary Gland." In Sex-Steroid Interactions with Growth Hormone, 301–7. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4612-1546-2_29.

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Pfaff, Donald, Elena Choleris, and Sonoko Ogawa. "Genes for sex hormone receptors controlling mouse aggression." In Novartis Foundation Symposia, 78–95. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470010703.ch6.

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Heine, M. J. S., and H. Gronemeyer. "Nuclear Receptors as Targets for Drug Design: New Options and Old Challenges." In Molecular Basis of Sex Hormone Receptor Function, 1–41. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-03689-1_1.

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Giangrande, P., G. Pollio, and D. P. McDonnell. "Functional and Pharmacological Analysis of the A and B Forms of the Human Progesterone Receptor." In Molecular Basis of Sex Hormone Receptor Function, 179–201. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-03689-1_10.

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Streszczenia konferencji na temat "Hormones, Sex – Receptors"

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Jönsson, Jenny-Maria, Nicolai Arildsen, Susanne Malander, Anna Måsbäck, Linda Werner Hartman, Mef Nilbert, and Ingrid Hedenfalk. "Abstract 1834: Sex hormone receptor expression affects ovarian cancer survival." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-1834.

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Lin, H., H.-C. Fu, Y.-C. Ou, C.-H. Wu, and H.-Y. Kang. "P140 The roles of sex hormone receptors and chemosensitivity in ovarian cancer." In ESGO Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-esgo.202.

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Farhat, G., S. Cummings, N. Parimi, A. Huang, J. Cauley, T. Rohan, F. Hubbell, et al. "Sex Hormones and Risk of Estrogen Receptor (ER)-Positive and ER-Negative Breast Cancer." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-907.

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Ferzel, Laura Ramos Silva, Kiara de Medeiros Braga Cruz Pessanha, Renata Magliano Marins, Meire Cardoso da Mota Bastos, and Isabelly dos Santos Belo. "Carcinoma metaplásico com diferenciação mesenquimal: relato de caso." In 44° Congresso da SGORJ - XXIII Trocando Ideias. Zeppelini Editorial e Comunicação, 2020. http://dx.doi.org/10.5327/jbg-0368-1416-2020130241.

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Introdução: Equivalendo a menos de 5% de todos os cânceres de mama, o carcinoma metaplásico representa um grupo morfologicamente heterogêneo das neoplasias malignas mamárias, que acomete, em sua maioria, mulheres na pós-menopausa. A principal manifestação clínica é nódulo palpável de crescimento progressivo e rápido. Histologicamente, é caracterizado por combinar componente epitelial e componente escamoso ou sarcomatoide. Manifesta-se tipicamente negativo para receptores hormonais e Human Epidermal growth factor Receptor-type 2 (HER2), independentemente do subtipo histológico. Objetivo: Relata
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Sakoda, Lori C., Noel S. Weiss, Matt Barnett, Gary E. Goodman, and Chu Chen. "Abstract 2858: Relation of androgen receptor polymorphisms and prostate cancer risk by level of sex steroid hormones." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2858.

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Noriega, Yamilet, Miguel Rivas, and Elizabeth Langley. "Abstract C56: Characterization of PinX1 as a coregulator of steroid hormone receptors." In Abstracts: Second AACR International Conference on Frontiers in Basic Cancer Research--Sep 14-18, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.fbcr11-c56.

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Jain, Raksha, Jennifer Ray, and Steven L. Brody. "Sex Hormone-Dependent Activation Of The Progesterone Receptor In Airway Epithelial Cells Inhibits Cilia Beat Frequency." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a1226.

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Menamin, Una CMc, James Trainor, Helen G. Coleman, Damian T. McManus, Brian T. Johnston, and Richard Turkington. "PTH-117 Sex hormone receptor expression in oesophageal adenocarcinoma and recurrence and survival: a retrospective cohort study." In British Society of Gastroenterology, Annual General Meeting, 4–7 June 2018, Abstracts. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2018. http://dx.doi.org/10.1136/gutjnl-2018-bsgabstracts.291.

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Novello, Silvia, Valentina Monica, Marina Longo, Sara Demichelis, Benedetta Crida, Matteo Giaj Levra, Simonetta G. Rapetti, and Enrica Capelletto. "Sex-Linked Hormone Receptor Expression In Lung Cancer Patients: Pilot Study In A Consecutive Group Of Patients With Advanced Lung Cancer." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5091.

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Spolador, Maria Eduarda Granucci, Maria Teresa Vasconcelos, Pedro Henrique Gunha Basílio, Samya Hamad Mehanna, and Victória Gayoso Neves Pereira. "TRATAMENTO DO CÂNCER DE MAMA TRIPLO NEGATIVO: O QUE DIZ A LITERATURA?" In I Congresso Nacional Multidisciplinar de Oncologia On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1581.

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Introdução: O câncer de mama triplo negativo (TNBC) caracteriza-se pela não expressão de receptores hormonais de estrogênio e progesterona, e negatividade do fator de crescimento epitelial humano 2 (HER2), correspondendo até 15% dos cânceres mamários. Este tipo molecular apresenta elevado índice proliferativo, sendo mais agressivo e capaz de produzir metástases e recidivas, apresentando pior prognóstico. O tratamento cirúrgico está indicado somado, principalmente, à quimioterapia. Objetivos: Identificar tratamentos atuais do TNBC e possíveis quimiorresistências. Material e métodos: Revisão bib
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