Gotowe bibliografie tematyczne / Human brain tumor cells

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  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
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Gotowa bibliografia na temat „Human brain tumor cells”

Autor: Grafiati
Data publikacji: 3 czerwca 2025
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Human brain tumor cells"

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Roosen, Mieke, Chris Meulenbroeks, Phylicia Stathi, et al. "BIOL-11. PRECLINICAL MODELLING OF PEDIATRIC BRAIN TUMORS USING ORGANOID TECHNOLOGY." Neuro-Oncology 25, Supplement_1 (2023): i8. http://dx.doi.org/10.1093/neuonc/noad073.030.

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Abstract Molecular characterization has resulted in improved classification of pediatric brain tumors, leading to many novel (sub)types with distinct oncodriving events. To study tumor biology and to perform translational research on each of these tumors, preclinical models are essential. However, we are currently lacking sufficient models, especially in vitro, to represent each (sub)type and their heterogeneity. To generate large series of preclinical in vitro models for pediatric brain tumors, we are using organoid technology. Cells from patient samples and patient-derived xenograft samples
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Weiner, Howard L., Hongyun Huang, David Zagzag, Hayden Boyce, Roger Lichtenbaum, and Edward B. Ziff. "Consistent and Selective Expression of the Discoidin Domain Receptor-1 Tyrosine Kinase in Human Brain Tumors." Neurosurgery 47, no. 6 (2000): 1400–1409. http://dx.doi.org/10.1097/00006123-200012000-00028.

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ABSTRACT OBJECTIVE Few molecular targets are both consistently and selectively expressed in a majority of central nervous system (CNS) neoplasms. Receptor tyrosine kinases have been implicated in brain tumor oncogenesis. We previously isolated one such receptor, discoidin domain receptor-1 (DDR1), from high-grade pediatric brain tumors. Here, we analyze the cellular origin and distribution of DDR1 expression in human brain tumors and its expression in tumor cells relative to surrounding brain. METHODS By use of a digoxigenin-labeled DDR1 riboprobe, we investigated the expression of DDR1 messen
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Kunishio, Katsuzo, Nobuya Mishima, Takashi Matsuhisa та ін. "Immunohistochemical demonstration of DNA polymerase α in human brain-tumor cells". Journal of Neurosurgery 72, № 2 (1990): 268–72. http://dx.doi.org/10.3171/jns.1990.72.2.0268.

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✓ The proliferative capacity of brain-tumor cells was analyzed in vitro and in situ using monoclonal antibody (MAb) against deoxyribonucleic acid (DNA) polymerase α. For the in vitro studies, two cultured human glioma cell lines were investigated using MAb against DNA polymerase α, the MAb Ki-67, a serum against proliferating cell nuclear antigen (PCNA/cyclin), bromodeoxyuridine (BUdR), and an anti-BUdR MAb. During exponential growth of the cells, the percentage of polymerase α-positive cells (the “polymerase α score”) ranged from 72.0% to 77.1%, the Ki-67-positive cells (the “Ki-67 score”) ra
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Ghezelbash, Mohsen, Nahid Masoudian, and Mehdi Pooladi. "Beta Actin Expression Profile in Malignant Human Glioma Tumors." International Clinical Neuroscience Journal 5, no. 2 (2018): 72–77. http://dx.doi.org/10.15171/icnj.2018.14.

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Background: Proteomics is considered a new era in neurophysiological/ neuropathological research including brain tumors. Gliomas which are derived from glial cells are the most common type of brain tumor in humans. Methods: In the present study the total protein content of healthy cells of the brain and brain tumor cells was extracted, purified and quantified by Bradford assay. Two-dimensional electrophoresis were used for protein separation followed by statistical analysis. Primary protein detection was performed based on the differences in isoelectric pH, molecular weight of proteins and pro
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Recht, Lawrence, Carmen O. Torres, Thomas W. Smith, Vic Raso, and Thomas W. Griffin. "Transferrin receptor in normal and neoplastic brain tissue: implications for brain-tumor immunotherapy." Journal of Neurosurgery 72, no. 6 (1990): 941–45. http://dx.doi.org/10.3171/jns.1990.72.6.0941.

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✓ The distribution of transferrin receptor (TfR) in normal human brain-tissue obtained at autopsy and in brain-tumor biopsy specimens from 27 patients was determined by immunohistochemistry using two specific murine monoclonal antibodies against human TfR. The tumors studied included 10 glioblastomas multiforme (GBM's), nine other glial tumors, and eight meningiomas. In normal brain, TfR was detected primarily in endothelial cells; rare glial cells also contained immunoreactive product. All tumors contained TfR-positive cells, although the intensity (number of cells stained) and pattern (focal
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Antonica, F., L. Santomaso, G. Aiello, D. Pernici, E. Miele, and L. Tiberi. "OS13.3.A Establishment of a novel system to specifically trace and ablate quiescent/slow cycling cells in high-grade glioma." Neuro-Oncology 23, Supplement_2 (2021): ii16. http://dx.doi.org/10.1093/neuonc/noab180.051.

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Abstract BACKGROUND High-grade gliomas are the most common malignant brain tumors, with poor prognosis due to recurrence and tumor infiltration after surgical removal and chemotherapy. Quiescent/slow cycling stem cells have been proposed to be one of the main players of tumor relapse but their involvement in in the infiltration remain unclear. Despite they have been described in mouse models or after transcriptional profiling of human tumor samples, their direct visualization, targeting and ablation remains a challenge. MATERIAL AND METHODS Tumors were induced over-expressing oncogenic forms o
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Stewart, Patricia A., Kay Hayakawa, Catherine L. Farrell, and Rolando F. Del Maestro. "Quantitative study of microvessel ultrastructure in human peritumoral brain tissue." Journal of Neurosurgery 67, no. 5 (1987): 697–705. http://dx.doi.org/10.3171/jns.1987.67.5.0697.

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✓ The form and function of blood vessels are determined by the cells that constitute their microenvironment. Brain tissue around tumors contains varying numbers of tumor cells that could influence local capillaries to lose their blood-brain barrier (BBB), as they do in the tumor itself. Microvascular permeability cannot be measured directly in humans but can be inferred from a knowledge of vessel ultrastructure. The authors have examined the vascular ultrastructure associated with the BBB in human peritumoral brain tissue for evidence of BBB compromise and to correlate BBB features with the ce
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Nabors, Michael W., Constance A. Griffin, Barbara A. Zehnbauer, et al. "Multidrug resistance gene (MDR1) expression in human brain tumors." Journal of Neurosurgery 75, no. 6 (1991): 941–46. http://dx.doi.org/10.3171/jns.1991.75.6.0941.

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✓ Multidrug resistance for many types of cancer outside the central nervous system (CNS) has been found to be due to the overexpression of the multidrug resistance gene MDR1, of which the gene-product P-glycoprotein acts as a membrane-bound efflux pump for many anticancer drugs. To examine whether brain tumors overexpress the MDR1 gene, 25 brain-tumor specimens were subjected to Northern blot analysis: 10 gliomas, eight meningiomas, three schwannomas, one malignant lymphoma, and three tumors metastatic to the brain. Ten fresh-frozen autopsy specimens of various parts of normal brain were also
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Li, Jianbo, Guojian Zhang, Mingming Zhu, Xuemei Wang, and Xiao-Feng Li. "Establishment and Imaging Studies of Human Lung Cancer-Associated Brain Metastasis Animal Models." Journal of Medical Imaging and Health Informatics 9, no. 6 (2019): 1138–41. http://dx.doi.org/10.1166/jmihi.2019.2707.

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Background: Lung cancer is one of the malignant tumors with fast increase in morbidity and mortality, and great threat to human health and life. Methods: Animal models (n = 5) were performed by injecting 1 × 106 A549 cells in 0.1 mL phosphate buffer saline into nude mice through the left ventricle. Body weight of animals was measured every 3 days, and changes in the appearance and behavior were observed. Brain magnetic resonance imaging of 5 animals were performed using T1, T1 enhancement and T2 scan at the 4th, 6th, and 7th weeks after injected with tumor cells. Animals (n = 5) were sacrifice
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Nakagawa, Takao, Toshihiko Kubota, Masanori Kabuto, et al. "Production of matrix metalloproteinases and tissue inhibitor of metalloproteinases-1 by human brain tumors." Journal of Neurosurgery 81, no. 1 (1994): 69–77. http://dx.doi.org/10.3171/jns.1994.81.1.0069.

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✓ The role of matrix metalloproteinases (MMP's) and their inhibitor, tissue inhibitor of metalloproteinases-1 (TIMP-1), in human brain tumor invasion was investigated. Gelatinolytic activity was assayed via gelatin zymography, and four MMP's (MMP-1, MMP-2, MMP-3, and MMP-9) and TIMP-1 were immunolocalized in human brain tumors and in normal brain tissues using monoclonal antibodies. The tissue was surgically removed from 44 patients: glioblastoma (five cases), anaplastic astrocytoma (six cases), astrocytoma (four cases), metastatic tumor (six cases), neurinoma (10 cases), meningioma (10 cases)
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Rozprawy doktorskie na temat "Human brain tumor cells"

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Landolina, Nadine Anna Caterina. ""Interactions and invasiveness of melanomatous human tumor cells within the blood brain barrier endothelium"." Doctoral thesis, Università di Catania, 2013. http://hdl.handle.net/10761/1397.

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Brain metastases occur in about 25% of patients who die of cancer.One of the most common sources of brain metastases in adults is cutaneous melanoma.The malignant potential of melanomatous cell and its adhesion to the cerebral endothelium plays a key role in this process.The aim of this study was to better characterize the interactions between melanoma cells and the cerebral endothelium, in an attempt to mimick, in some way, melanomatous cells crossing through the BBB and the subsequent spread throghout the cerebral parenchyma.
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Pistollato, Francesca. "Oxygen Tension Controls the Expansion and Differentiation of Normal and Tumor-derived Human Neural Stem Cells. Role of oxygen in BMP responsiveness." Doctoral thesis, Università degli studi di Padova, 2007. http://hdl.handle.net/11577/3425173.

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During neural development the generation of diverse cell types involves the response of precursor cells to a wide variety of environmental cues, like soluble factors, the extracellular matrix and oxygen tension. Among these, oxygen tension and oxidation state in particular are important biophysical parameters that control neural precursor proliferation, survival and fate determination, so the dynamic control of oxygen availability regulates self renewal and the generation of cell diversity during development and throughout the life of the organism. However, the mechanisms by which oxygen acts
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Lanser, Brittany. "Characterization of checkpoint adaptation in human fibroblastic glioma cells and an analysis of protein phosphatase inhibitors." Thesis, Lethbridge, Alta. : University of Lethbridge, Dept. of Biological Sciences, c2012, 2012. http://hdl.handle.net/10133/3390.

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This thesis reports that checkpoint adaptation occurs in human brain cancer cells. M059K cells, after treatment with camptothecin (CPT), recruited γ-histone H2AX, phosphorylated Chk1 and arrested in the G2 phase. Strikingly, cells escaped the checkpoint, became rounded and entered mitosis as measured by phospho-histone H3 signals. Lamin A/C immunofluorescence microscopy revealed that 48% of the cells that survived checkpoint adaptation contained micronuclei. These data suggest that brain cancer cells undergo checkpoint adaptation and may have an altered genome. This thesis also explored if pho
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Geremia, Ezequiel. "Spatial random forests for brain lesions segmentation in MRIs and model-based tumor cell extrapolation." Phd thesis, Université Nice Sophia Antipolis, 2013. http://tel.archives-ouvertes.fr/tel-00838795.

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The large size of the datasets produced by medical imaging protocols contributes to the success of supervised discriminative methods for semantic labelling of images. Our study makes use of a general and efficient emerging framework, discriminative random forests, for the detection of brain lesions in multi-modal magnetic resonance images (MRIs). The contribution is three-fold. First, we focus on segmentation of brain lesions which is an essential task to diagnosis, prognosis and therapy planning. A context-aware random forest is designed for the automatic multi-class segmentation of MS lesion
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Letzien, Ulrike. "Effects of Carnosine and L-histidine on Viability and Expression of Pyruvate Dehydrogenase Kinase 4 in Human Glioblastoma Cells." Doctoral thesis, Universitätsbibliothek Leipzig, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-197285.

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Die Arbeit behandelt die Ergebnisse von Experimenten über die Wirkung des Dipeptides Carnosin (β Alanyl L Histidin) und der Aminosäuren L Histidin und β-Alanin auf Kulturen der humanen Zellreihen U87, T98G und LN405, welche von Zellen des malignen Hirntumors Glioblastoma multiforme abgeleitet sind. Die Vitalität der Zellen nach Inkubation mit Carnosin oder L Histidin wurde anhand der Adenosintriphosphatproduktion und der Dehydrohenaseaktivität für Inkubationszeiträume von 24, 48 und 72 Stunden bestimmt. Dabei zeigte sich eine signifikant niedrigere Vitalität der mit Carnosin oder L Histidin
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Junnikkala, Sami. "Complement resistance mechanisms of human tumor cells." Helsinki : University of Helsinki, 2002. http://ethesis.helsinki.fi/julkaisut/laa/haart/vk/junnikkala/.

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Kaul, Aparna. "Mechanisms of Non-Conventional Cell Death in Brain Tumor Cells." University of Toledo Health Science Campus / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=mco1243364096.

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Najim, Nigar. "A study of the cytotoxic effects of methionine depletion in human brain tumour cell lines." Thesis, University of Salford, 2007. http://usir.salford.ac.uk/26832/.

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The aim of this project was to investigate the importance of methionine depletion as a cause of cytotoxicity for paediatric CNS tumour cell lines, and also to investigate the in vitro cellular biochemical responses, as measured by changes in cellular levels of L-methionine, glutathione and O6 - alkylguanine DNA - alkyltransferase (MGMT) activity in glioma (D54) and medulloblastoma (Daoy) cell lines. A characteristic feature of many solid tumours is their requirement for both endogenous and exogenous L-methionine in order to support cellular proliferation. Normal cells are generally methionine-
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Curtis, Maurice A. "Neural progenitor cells in the Huntington's Disease human brain." Thesis, University of Auckland, 2004. http://hdl.handle.net/2292/3114.

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The recent demonstration of endogenous progenitor cells in the adult mammalian brain raises the exciting possibility that these undifferentiated cells may be able to generate new neurons for cell replacement in diseases such as Huntington's disease (HD). Previous studies have shown that neural stem cells in the rodent brain subependymal layer (SEL), adjacent to the caudate nucleus, proliferate and differentiate into neurons and glial cells but no previous study has characterised the human SEL or shown neurogenesis in the diseased human brain. In this study, histochemical and immunohistochemica
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Carlsten, Mattias. "Molecular specificities of NK cell-mediated recognition of human tumor cells." Stockholm, 2010. http://diss.kib.ki.se/2010/978-91-7409-686-6/.

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Książki na temat "Human brain tumor cells"

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Singh, Sheila K., and Chitra Venugopal, eds. Brain Tumor Stem Cells. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-8805-1.

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Singh, Sheila K., and Chitra Venugopal, eds. Brain Tumor Stem Cells. Springer US, 2025. https://doi.org/10.1007/978-1-0716-4654-0.

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Pfragner, Roswitha, and R. Ian Freshney, eds. Culture of Human Tumor Cells. John Wiley & Sons, Inc., 2003. http://dx.doi.org/10.1002/0471722782.

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1938-, Hay Robert, Park Jae-Gahb, and Gazdar Adi F, eds. Atlas of human tumor cell lines. Academic Press, 1994.

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Ludlow, John W. Tumor suppressors: Involvement in human diseases, viral protein interactions, and growth regulation. R.G. Landes, 1994.

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Kolenik, Steven Andrew. The effects of interleukin-1, granulocyte macrophage colony-stimulating factor, and tumor necrosis factor-α on cultured human langerhans cells and cortical thymocytes. s.n.], 1990.

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Mukherjee, Tinku S. Regulation of the D1 dopamine receptor in rat brain and SK-N-Mc human neuroblastoma cells. National Library of Canada = Bibliothèque nationale du Canada, 1995.

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J, Staal Gerard E., and Veelen, Cees W. M. van., eds. Markers of human neuroectodermal tumors. CRC Press, 1986.

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Wicki, Roland. Characterization of the S100 gene cluster on human chromosome 1q21 and analysis of transcriptional control elements of the potential tumor suppressor gene S100A2 in breast epithelial cells. [s.n.], 1997.

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Liu, Zhi-Jian. The effects of calcium, short chain fatty acids and mammalian lignans on calcium transport, intracellular Ca2+ and intracellular pH in the human colon tumor cells HCT-15. National Library of Canada, 2000.

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Części książek na temat "Human brain tumor cells"

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Yoshida, Seiichi, Ryuichi Tanaka, and Ryuya Yamanaka. "Antisense DNA Approach to the Growth of Human Glioma Cells." In Brain Tumor. Springer Japan, 1996. http://dx.doi.org/10.1007/978-4-431-66887-9_46.

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Darling, John L. "In Vitro Culture of Malignant Brain Tumors." In Culture of Human Tumor Cells. John Wiley & Sons, Inc., 2005. http://dx.doi.org/10.1002/0471722782.ch14.

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Bernstein, Jerald J., William J. Goldberg, and Edward R. Laws. "Migration of fresh human malignant astrocytoma cells into hydrated gel wafers in vitro." In Brain Tumor Invasiveness. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2622-3_7.

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Bidlingmaier, Scott, Xiaodong Zhu, Yue Liu, Yang Su, and Bin Liu. "Novel Internalizing Human Antibodies Targeting Brain Tumor Sphere Cells." In Stem Cells and Cancer Stem Cells, Volume 9. Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-5645-8_18.

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Tada, Mitsuhiro, Annie-Claire Diserens, Marie-France Hamou, Rehana Jaufeerally, Erwin G. van Meir, and Nicolas de Tribolet. "Suppressed Expression of T-Cell Costimulatory Molecules B7 and B70 in Human Glioblastomas In Vivo." In Brain Tumor. Springer Japan, 1996. http://dx.doi.org/10.1007/978-4-431-66887-9_34.

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Ozawa, Tomoko, and C. David James. "Human Brain Tumor Cell and Tumor Tissue Transplantation Models." In CNS Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-553-8_8.

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Rahman, Mohsen, Herman Yeger, and Laurence E. Becker. "In vivo characterization of a human neuroectodermal tumor cell line." In Biology of Brain Tumour. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2297-9_15.

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Takahashi, Hiroshi, and Shozo Nakazawa. "Effects of a Human Monoclonal Antibody and Cytokines on Human Malignant Glioma Cells." In Biological Aspects of Brain Tumors. Springer Japan, 1991. http://dx.doi.org/10.1007/978-4-431-68150-2_66.

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Qazi, Maleeha, Aneet Mann, Randy van Ommeren, et al. "Generation of Murine Xenograft Models of Brain Tumors from Primary Human Tissue for In Vivo Analysis of the Brain Tumor-Initiating Cell." In Stem Cells and Tissue Repair. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1435-7_4.

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Shimizu, Keiji, Masanobu Yamada, Kazuyoshi Tamura, et al. "Murine Models with Leptomeningeal Dissemination of Human Medulloblastoma Cells." In Biological Aspects of Brain Tumors. Springer Japan, 1991. http://dx.doi.org/10.1007/978-4-431-68150-2_64.

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Streszczenia konferencji na temat "Human brain tumor cells"

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Chen, Xinrui. "Enhancing Brain Tumor Diagnosis with MedMamba: A Novel Application in Neural Imaging Analysis." In 2024 5th International Conference on Intelligent Computing and Human-Computer Interaction (ICHCI). IEEE, 2024. https://doi.org/10.1109/ichci63580.2024.10807965.

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Khalid, Hafsa, and Cem Direkoglu. "Brain Tumor Classification using Deep Learning: Robustness Against Adversarial Attacks and Defense Strategies." In 2025 7th International Congress on Human-Computer Interaction, Optimization and Robotic Applications (ICHORA). IEEE, 2025. https://doi.org/10.1109/ichora65333.2025.11017313.

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Chasiotis, I., D. C. Street, H. L. Fillmore, and G. T. Gillies. "AFM Studies of Tumor Cell Invasion." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43293.

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Our recent investigations on human brain tumor (glioma) cell micro and nanodynamics via AFM methodologies have shown that brain tumor invadopodia (malignant cytostructural cell extensions with sensory, motility, and invasive characteristics extended by tumor cells into their environment) can assume specific geometries based on cell plating density and the location/distance of neighboring cells indicating strong cell sensing and signaling mechanisms between malignant cells and their surroundings. In certain occasions, cancer cell processes (extensions) have been found to be highly directional m
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Majib, Mohammad Shahjahan, T. M. Shahriar Sazzad, and Md Mahbubur Rahman. "A Framework To Detect Brain Tumor Cells Using MRI Images." In 2020 International Congress on Human-Computer Interaction, Optimization and Robotic Applications (HORA). IEEE, 2020. http://dx.doi.org/10.1109/hora49412.2020.9152893.

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Said, H. M. "Determination human brain tumor marker gene carbonic anhydrase 9 (CA9) gene expression in different type of brain tumor cells." In 2013 ICME International Conference on Complex Medical Engineering (CME 2013). IEEE, 2013. http://dx.doi.org/10.1109/iccme.2013.6548278.

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Choi, Seung Ah, Kyu-Chang Wang, Ji Hoon Phi, et al. "Abstract 2649: Human adipose tissue-derived mesenchymal stem cells can target brain tumor initiating cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2649.

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Day, Emily S., Linna Zhang, Nastassja A. Lewinski, et al. "Photothermal Therapy of Glioma in a Mouse Model With Near-Infrared Excited Nanoshells." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13179.

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Glioblastoma multiforme is the most common and aggressive primary brain tumor, with median survival of approximately 10 months and only 5% of patients surviving greater than 5 years after treatment (1). Surgery and radiotherapy are the main treatment modalities for primary brain tumors, but the associated risks are high when infiltrative tumors are positioned near sensitive regions in the brain. Nanoshells, nanoparticles characterized by a spherical silica core and a gold shell, may provide the opportunity to treat brain tumors in a minimally invasive manner, reducing the risk associated with
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Beasley, Selina A., Timothy A. Raines, and Patrick M. Martin. "Abstract 2933: Inhibition of MAPK signaling prevents Fra-1-mediated CD44 expression in human brain tumor cells." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2933.

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Stokol, Tracy, Mandy B. Esch, Nozomi Nishimura, et al. "Little Channels, Big Disease: Using Microfluidics to Investigate Cancer Metastasis." In ASME 2011 9th International Conference on Nanochannels, Microchannels, and Minichannels. ASMEDC, 2011. http://dx.doi.org/10.1115/icnmm2011-58298.

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The leading cause of death in human patients with malignant cancer is the dissemination of the primary tumor to secondary sites throughout the body. It is well known that cancers metastasize to certain tissues (e.g. breast cancer typically spreads to the lungs. brain and bone), in a pattern that cannot be explained by blood flow from the primary tumor or simple mechanical arrest. Circulating tumor cells usually arrest in the microvasculature of target tissues. At these sites, they must adhere to the endothelium, survive, proliferate and extravasate in order to form a secondary tumor. In vitro
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Guerrero-Cazares, Hugo, Emily Lavell, Gabrielle Drummond, et al. "Abstract 444: Slit2 stimulation induces a chemorepellent effect on the migration of human GBM brain tumor initiating cells." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-444.

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Raporty organizacyjne na temat "Human brain tumor cells"

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Shay, Jerry W. A Comprehensive Repository of Normal and Tumor Human Breast Tissues and Cells. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada375078.

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Chaudhuri, Gautam. Repression of RTK Recycling Pathway By SLUG in Human Breast Tumor Cells. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada541187.

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Chaudhuri, Gautam. Modulation of the Proliferation and Metastasis of Human Breast Tumor Cells by SLUG (IDEA). Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada489798.

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Gupta, Piyush, and Robert A. Weinberg. Contribution of Bone Marrow-Derived Cells to the Tumor Stroma in Human Breast Cancer. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada428526.

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Gupta, Piyush, and Robert A. Weinberg. Contribution of Bone Marrow-Derived Cells to the Tumor Stroma in Human Breast Cancer. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada417609.

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Liu, Xuedong. Identification of the Downstream Promoter Targets of Smad Tumor Suppressors in Human Breast Cancer Cells. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada433854.

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Moritz, Robert. Development of Advanced Technologies for Complete Genomic and Proteomic Characterization of Quantized Human Tumor Cells. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada614224.

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Moritz, Robert. Development of Advanced Technologies for Complete Genomic and Proteomic Characterization of Quantized Human Tumor Cells. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada573716.

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Foltz, Gregory. Development of Advanced Technologies for Complete Genomic and Proteomic Characterization of Quantized Human Tumor Cells. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada574964.

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Moritz, Robert. Development of Advanced Technologies for Complete Genomic and Proteomic Characterization of Quantized Human Tumor Cells. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada583585.

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