Gotowe bibliografie tematyczne / Human ovarian carcinoma cells

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Gotowa bibliografia na temat „Human ovarian carcinoma cells”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Human ovarian carcinoma cells"

Wu, Guang-Jer. "METCAM/MUC18 Decreases the Malignant Propensity of Human Ovarian Carcinoma Cells." International Journal of Molecular Sciences 19, no. 10 (2018): 2976. http://dx.doi.org/10.3390/ijms19102976.

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METCAM/MUC18 is an integral membrane cell adhesion molecule (CAM) in the Ig-like gene super-family. It can carry out common functions of CAMs which is to perform intercellular interactions and interaction of cell with extracellular matrix in tumor microenvironment, to interact with various signaling pathways and to regulate general behaviors of cells. We and other two groups previously suggested that METCAM/MUC18 probably be utilized as a biomarker for predicting the malignant tendency of clinical ovarian carcinomas, since METAM/MUC18 expression appears to associate with the carcinoma at advan

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Imai, Atsushi, Tsukasa Ohno, Kyoko Takahashi, Tatsuro Furui, and Teruhiko Tamaya. "Lack of Evidence for Aromatase Expression in Human Ovarian Epithelial Carcinoma." Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 31, no. 1 (1994): 65–71. http://dx.doi.org/10.1177/000456329403100111.

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It is controversial whether ovarian epithelial carcinoma possesses steroidogenic enzymes. We investigated aromatase expression in ovarian epithelial carcinoma, and compared it with the normal ovary and placenta. Samples were obtained from an ovarian carcinoma cell line SK-OV-3, ovarian tumour tissues from four patients with epithelial carcinoma and one patient with dysgerminoma. Aromatase enzymatic activity was measured in microsome fractions by quantitating 3H2O released from [1-3H]androstenedione and [3H]oestrone converted from [1,2,6,7-3H]androstenedione. Aromatase messenger ribonucleic aci

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Sidell, N., and I. R. Horowitz. "Reduced connexin 43 mRNA levels in human ovarian carcinoma." Journal of Clinical Oncology 24, no. 18_suppl (2006): 15071. http://dx.doi.org/10.1200/jco.2006.24.18_suppl.15071.

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15071 Background: Cancer cells have been shown to exhibit many defects in gap junctions, which contribute to the loss of growth control and tissue homeostasis. In ovarian tissues, it is known that gap junction communication is predominantly mediated by connexin 43 (cx43) proteins. Recent studies have demonstrated that while human ovarian surface epithelial cells exhibit extensive expression of cx43, this protein is nearly absent in the vast majority of ovarian cancers. Differences in cx43 expression between normal and malignant ovarian tissue at the mRNA level has heretofore not been reported.

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Zhang, Li, Po Ding, Hongcheng Lv, et al. "Number of Polyploid Giant Cancer Cells and Expression of EZH2 Are Associated with VM Formation and Tumor Grade in Human Ovarian Tumor." BioMed Research International 2014 (2014): 1–9. http://dx.doi.org/10.1155/2014/903542.

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To investigate the associations among the number of polyploid giant cancer cells (PGCCs) and vasculogenic mimicry (VM), EZH2 expression, and serous ovarian tumor grade, a total of 80 paraffin-embedded serous ovarian tumor samples including 21 cases of primary carcinoma and their metastatic tumors, 26 cases of primary carcinoma without metastasis, and 12 cases of serous borderline cystadenoma were analyzed. PGCCs and VM were detected in human serous ovarian tumor. The metastatic foci of ovarian carcinoma had the highest number of PGCCs and VM. The number of PGCCs and VM increased with the grade

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FISHMAN, David A., Alicia KEARNS, Susan LARSH, Jan J. ENGHILD, and M. Sharon STACK. "Autocrine regulation of growth stimulation in human epithelial ovarian carcinoma by serine-proteinase-catalysed release of the urinary-type-plasminogen-activator N-terminal fragment." Biochemical Journal 341, no. 3 (1999): 765–69. http://dx.doi.org/10.1042/bj3410765.

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Ovarian carcinomas secrete single-chain urinary-type plasminogen activator (scuPA) and expression of uPA is up-regulated relative to normal ovarian epithelium, leading to an enhanced proteolytic capacity which may facilitate invasion. Furthermore, the uPA receptor (uPAR) is present on ovarian carcinoma cells and is occupied in tumour tissues. In the present study, incubation of scuPA with serum-free conditioned medium from ovarian carcinoma cells resulted in release of a 14 kDa polypeptide. N-terminal sequence analysis identified this fragment as the uPA N-terminal fragment (NTF), which contai

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Koon, E. C., P. C. Ma, R. Salgia, et al. "Effect of a c-Met-specific, ATP-competitive small-molecule inhibitor SU11274 on human ovarian carcinoma cell growth, motility, and invasion." International Journal of Gynecologic Cancer 18, no. 5 (2008): 976–84. http://dx.doi.org/10.1111/j.1525-1438.2007.01135.x.

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Increased expression of the receptor tyrosine kinase c-Met has been shown to correlate with enhanced cell proliferation, motility, and invasion. The objectives of this study were to characterize total and activated c-Met expression in both normal and malignant human ovarian epithelial cells and to determine the effects of inhibiting the activation of c-Met on ovarian epithelial cell growth, motility, and invasion. Total c-Met was overexpressed in 82 (68%) of 119 ovarian carcinomas, as shown by immunohistochemistry. Quantitative reverse transcription–polymerase chain reaction and Western blot a

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Zhang, W., N. Gan, and J. Zhou. "Immunohistochemical Investigation of the Correlation between LIM Kinase 1 Expression and Development and Progression of Human Ovarian Carcinoma." Journal of International Medical Research 40, no. 3 (2012): 1067–73. http://dx.doi.org/10.1177/147323001204000325.

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OBJECTIVES: LIM kinase 1 (LIMK1) is implicated in cellular mechanisms regulating tumour cell invasion and may be involved in ovarian carcinoma progression. This retrospective study, therefore, investigated expression of the LIMK1 gene in primary ovarian tumour tissue samples and evaluated the correlation between LIMK1 gene expression and progression of ovarian carcinoma. METHODS: LIMK1 protein levels were detected by immunohistochemistry in ovarian tissue samples from 57 patients with primary ovarian epithelial tumours (benign, n = 13; borderline, n = 14; carcinoma, n = 30) and 10 patients wit

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Nahar, Begum Afrin, Rama Saha, Chhanda Das, and Gourishankar Kamilya. "Immunohistochemical expression of human epididymis 4 in ovarian malignancy." International Journal of Research in Medical Sciences 7, no. 12 (2019): 4493. http://dx.doi.org/10.18203/2320-6012.ijrms20195506.

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Background: Ovarian malignancies has the highest mortality rate among all gynaecological malignancies. Surface epithelial tumors form two thirds of all ovarian neoplasm and 90% of all ovarian cancers are surface epithelial carcinomas. Mortality in case of ovarian malignancy is high due to late diagnosis. Early and accurate diagnosis can improve the case specific management. HE4 (Human Epididymis Protein 4) which is proved to be overexpressed in the ovarian cancer cells, is considered a new biomarker for ovarian cancer diagnosis which helps in early diagnosis and patient management. Aims and ob

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Tassi, R. A., E. Bignotti, M. Falchetti, et al. "Claudin-7 expression in human epithelial ovarian cancer." International Journal of Gynecologic Cancer 18, no. 6 (2008): 1262–71. http://dx.doi.org/10.1111/j.1525-1438.2008.01194.x.

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Claudin-7 (CLDN-7) is a tight junction protein recently found highly differentially expressed in ovarian carcinoma. To evaluate its potential as a novel biomarker, in this study, we quantified and compared claudin-7 expression at messenger RNA and protein level in 110 patients harboring various histologic types of epithelial ovarian carcinomas (EOC). CLDN-7 transcript was found significantly overexpressed in both primary and metastatic EOCs compared to normal human ovarian surface epithelium cell lines (fold change = 111.4, P< 0.001) by reverse transcription–polymerase chain reaction. At th

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Imai, Atsushi, Tsukasa Ohno, Kazuhiro Ohsuye, and Teruhiko Tamaya. "Expression of Gonadotropin-Releasing Hormone Receptor in Human Epithelial Ovarian Carcinoma." Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 31, no. 6 (1994): 550–55. http://dx.doi.org/10.1177/000456329403100604.

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We have previously demonstrated the presence of gonadotropin-releasing hormone (Gn-RH) messenger ribonucleic acid (mRNA) in epithelial ovarian carcinoma. In this study, the expression of Gn-RH receptor (Gn-RHR) was investigated in human ovarian carcinoma and human ovarian carcinoma cell line. Gn-RHR was determined by [3H] Gn-RH binding assay. Gn-RHR mRNA was determined by reverse transcription-polymerase chain reaction using oligonucleotide primers synthesized based on published human Gn-RHR sequence. Specific Gn-RH binding sites were shown to be present in plasma membrane isolated from five o

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Rozprawy doktorskie na temat "Human ovarian carcinoma cells"

Chu, Stephanie Wai Ling Clinical School St George Hospital Faculty of Medicine UNSW. "An investigation into the effects of albendazole on human ovarian carcinoma cells." Awarded by:University of New South Wales. Clinical School - St George Hospital, 2007. http://handle.unsw.edu.au/1959.4/40448.

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Paclitaxel (PTX) is an effective anti-mitotic drug. It stops cancer from spreading by interfering with the microtubule dynamics which in turn leads to cell cycle arrest and eventually cell death. Despite the clinical success in treating different types of cancers, resistance to PTX remains a major hurdle for successful treatment in relapse patients. Albendazole (ABZ) is a popular anthelmintic used world-wide for the treatment of various types of helmintic infections. In helminthes, ABZ binds to ??-tubulin and inhibits microtubule polymerisation. It was subsequently found that ABZ has anti-canc

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Vasey, Paul A. "In vitro studies of p53 and cisplatin-resistance in human ovarian carcinoma cells." Thesis, University of Glasgow, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.387507.

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Virtanen, Siru Sirkku Pauliina. "Phenotypic and functional characterisation of cancer stem cells in human high-grade serous ovarian carcinoma." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648879.

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Abbott, Heather Elizabeth. "Comparisons of the factors influencing intrinsic radiosensitivity between two isogenic human ovarian carcinoma cell lines." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0021/MQ57080.pdf.

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Illuzzi, G. "SPHINGOLIPID METABOLISM'S ALTERATIONS CORRELATED WITH TUMOR CELL INVASIVITY AND FENRETINIDE RESISTANCE IN A HUMAN OVARIAN CARCINOMA CELL LINE." Doctoral thesis, Università degli Studi di Milano, 2010. http://hdl.handle.net/2434/160742.

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Glycosphingolipids (GSL) modulate several signal transduction processes controlling cell proliferation, survival, differentiation and transformation. Alterations in the expression of carbohydrate epitopes associated with GSL are frequent in tumors, and it has been hypothesized that GSL could play important roles in modulating some of the properties of tumor cells. The contribution of transformation-associated changes in GSL composition to the tumor phenotype are very complex and not fully elucidated, and likely implies heterogeneous molecular mechanisms. However, at least two well established

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Joun, George Louis. "Dissecting the Cellular and Molecular Regulation of Chemo-Resistance and Epithelial to Mesenchymal Transition in Human Ovarian Carcinoma." Thesis, The University of Sydney, 2021. https://hdl.handle.net/2123/28748.

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Ovarian carcinoma is the malignant transformation of ovarian or fallopian tube epithelial cells. High-grade serous carcinoma (HGSOC) diagnosis carries a 30% 5-year survival. The development of chemotherapy resistance is common leading to relapse of a resistant tumour. Due to inherent genetic/chromosomal instability in HGSOC, these tumours are multi-clonal leading to treatment resistance and metastasis. It was aimed to understand the clonal heterogeneity in HGSOC cell lines through single cell molecular and physiological dissection. Chapter 3 describes the use of single cell derived strains, si

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Whitfield, Fatima. "Gamma-Delta T-Cells in Patients with Ovarian Carcinoma." Diss., Temple University Libraries, 2008. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/3422.

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Microbiology and Immunology<br>Ph.D.<br>Ovarian cancer is the fifth most common cause of death from all cancers among women in the Western world and the most lethal of all gynecological cancers. The epithelial ovarian carcinomas (EOC) represent approximately 90% of all human ovarian malignant neoplasm. The five-year survival rate for patients with EOC is attributed to late diagnosis and poor response to therapy. T-cells play an important role in tumor immunity of EOC, evidence includes infiltrating CD3+ T-cells in EOC lesions and a specific antigen driven immune response. Human gd TCR + T-cell

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Al-Azraqi, Abdulla Ali. "The P53 tumour suppressor pathway in human ovarian carcinoma." Thesis, University of Newcastle Upon Tyne, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.264421.

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Buckle, Clive. "Production of human monoclonal antibodies to ovarian carcinoma using phage display." Thesis, University of Sheffield, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.541444.

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Touboul, Cyril. "Influence du stroma et des cellules souches mésenchymateuses sur la dissémination et la résistance au traitement des carcinomes ovariens épithéliaux." Phd thesis, Université Paris Sud - Paris XI, 2012. http://tel.archives-ouvertes.fr/tel-00907889.

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Le cancer épithélial de l'ovaire (EOC) a la particularité d'être diagnostiqué à un stade avancé chez 75% des patientes et de récidiver dans un grand nombre de cas malgré une bonne réponse initiale à la chimiothérapie, expliquant ainsi son pronostic sombre. Le rôle du microenvironnement tumoral semble être de premier plan dans le développement et la survie des cellules cancéreuses mais il existe encore peu de données concernant les cellules mésenchymateuses souches (MSC). Dans ce travail nous avons donc cherché à déterminer les mécanismes moléculaires entre les MSC et les cellules tumorales ova

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Książki na temat "Human ovarian carcinoma cells"

Byrne, Karen. Targeting of radiolabelled antibodies to ovarian carcinoma cells and spheroids. University of Manchester, 1994.

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Weinstein, Ellen Michele. Inhibition of CA 125, a novel high molecular weight glycoprotein expressed by an ovarian carcinoma cell line: 0VCA 433, is related to glucocorticoid effects of altered cell growth, morphology, and growth pattern. s.n.], 1988.

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Linda, Giudice, Santa Eileen, Pool Robert, Institute of Medicine (U.S.). Board on Health Sciences Policy, and National Research Council (U.S.). Board on Life Sciences., eds. Assessing the medical risks of human oocyte donation for stem cell research: Workshop report. National Academies Press, 2007.

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K, Woodruff Teresa, and Snyder Karrie Ann, eds. Oncofertility: Fertility preservation for cancer survivors. Springer Verlag, 2007.

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1963-, Woodruff Teresa K., and Snyder Karrie Ann, eds. Oncofertility: Fertility preservation for cancer survivors. Springer, 2007.

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Hayat, M. A. Handbook of Immunohistochemistry and in Situ Hybridization of Human Carcinomas: Molecular Genetics, Gastrointestinal Carcinoma, and Ovarian Carcinoma. Elsevier Science & Technology Books, 2006.

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Sheldon, Katherine Mary. Characterization of immunoconjugates active against human carcinoma cells "in vitro". 1987.

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Josephs, Debra H., Heather J. Bax, Giulia Pellizzari, James F. Spicer, Ana Montes, and Sophia N. Karagiannis. Antibody Therapeutics for Ovarian Carcinoma and Translation to the Clinic. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0001.

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Despite improvements over the past decade in the treatment of ovarian cancer, many patients are at risk of recurrent disease and emerging drug resistance. The increased selectivity and reduced toxicity of molecularly targeted anti-cancer agents renders them attractive for development in ovarian cancer, and monoclonal antibodies targeting ovarian cancer-specific tumor antigens represent the largest such group investigated in this clinical setting. This chapter describes examples of monoclonal antibodies clinically evaluated for efficacy in ovarian cancer. These agents recognize molecular target

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Jakobson, Eva. Studies on positive and negative growth regulation in human bladder carcinoma cells. 1994.

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Hayat, M. A. Handbook of Immunohistochemistry and in situ Hybridization of Human Carcinomas, Volume 4: Molecular Genetics, Gastrointestinal Carcinoma, and Ovarian Carcinoma ... in Situ Hybridization of Human Carcinomas). Academic Press, 2006.

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Części książek na temat "Human ovarian carcinoma cells"

Colnaghi, M. I. "Use of bifunctional monoclonal antibodies for retargeting human lymphocytes against ovarian carcinoma cells." In Ovarian Cancer 3. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4757-0136-4_32.

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Yanaihara, Nozomu, and Aikou Okamoto. "Molecular Landscape in Ovarian Clear Cell Carcinoma." In Current Human Cell Research and Applications. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-6013-6_9.

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Andrews, P. A., S. C. Mann, S. Velury, and S. B. Howell. "Cisplatin Uptake Mediated Cisplatin-Resistance in Human Ovarian Carcinoma Cells." In Platinum and Other Metal Coordination Compounds in Cancer Chemotherapy. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-1717-3_26.

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Bernacki, R. J. "The Mechanism of Lectin-Mediated Adhesion of Human Ovarian Carcinoma Cells." In Cancer Therapy. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-84613-7_22.

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Kyo, Satoru. "Investigating the Molecular Carcinogenesis of Ovarian High-Grade Serous Carcinoma." In Current Human Cell Research and Applications. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-6013-6_4.

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Poels, L. G., O. C. Boerman, L. Massuger, et al. "Biodistribution, Binding and Internalisation of Monoclonal Antibodies to Human Ovarian Carcinoma Cells." In From Clone to Clinic. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-011-3780-5_9.

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Scanlon, K. J., Y. Lu, M. Kashani-Sabet, J. x. Ma, and E. Newman. "Mechanisms for Cisplatin-FUra Synergism and Cisplatin Resistance in Human Ovarian Carcinoma Cells both in vitro and in vivo." In Advances in Experimental Medicine and Biology. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5607-3_12.

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Draper, Jonathan S., Harry Moore, and Peter W. Andrews. "Embryonal Carcinoma Cells." In Human Embryonic Stem Cells. Humana Press, 2003. http://dx.doi.org/10.1007/978-1-59259-423-8_4.

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Wunderer, G., and I. Walter. "T-Kinin in Human Ovarian Carcinoma Ascites." In Advances in Experimental Medicine and Biology. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4615-9546-5_18.

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Jäger, W., L. Wildt, and N. Lang. "GnRH Analogues in the Treatment of Ovarian Carcinoma." In GnRH Analogues in Cancer and Human Reproduction. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0723-2_16.

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Streszczenia konferencji na temat "Human ovarian carcinoma cells"

Guimarães, Fernando, and Dominic Ayala Biffi. "CAISMOV24, a new human ovarian carcinoma cell line." In XXIII Congresso de Iniciação Científica da Unicamp. Galoá, 2015. http://dx.doi.org/10.19146/pibic-2015-38140.

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Lim, Chuan‐Bian, Nung Ky, Mohamed Sabry Hamza, and Yan Zhao. "Abstract B67: Thapsigargin sensitizes multidrug‐resistant human ovarian carcinoma cells to doxorubicin." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 15-19, 2009; Boston, MA. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/1535-7163.targ-09-b67.

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Cáceres, Antonella Rosario Ramona, Daniela Alejandra Cardone, Marisa Roig Cerdeño, María de los Ángeles Sanhueza, and Myriam Raquel Laconi. "THE ROLE OF PROGESTERONE METABOLITES ON THE PROLIFERATIVE-APOPTOTIC BALANCE OF HUMAN OVARIAN CARCINOMA-DERIVED CELL LINES." In Second Southern Science Conference - 2024. Araucária - Associação Científica, 2024. https://doi.org/10.48141/sscon_44_2024.pdf.

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Ovarian cancer is the most lethal gynecologic malignancy and the fifth leading cause of cancer death in women. Progesterone can have different effects depending on the hormonal conditions or the physiological environment in which it is administered. Its effect varies depending on the hormonal concentrations present in the body, since these can modify the body's response to the drug , particularly in cancer. P4 is metabolized into two groups: the 5?-pregnans, including allopregnanolone (ALLO), and the 4-pregnans, such as 3?-dihydroprogesterone (3?HP) and 20?-dihydroprogesterone (20?HP). Previou

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Brouwer-Visser, Jurriaan, María J. Cossio, Suzan K. Chao, and Gloria S. Huang. "Abstract 1080: Effect of IGF2 overexpression on the tumorigenicity of human ovarian carcinoma cells." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1080.

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Brouwer-Visser, Jurriaan, Suzan K. Chao, and Gloria S. Huang. "Abstract A48: Effect of constitutive IGF2 overexpression on tumorigenicity of human ovarian carcinoma cells." In Abstracts: Second AACR International Conference on Frontiers in Basic Cancer Research--Sep 14-18, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.fbcr11-a48.

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Potočňák, Ivan, Miroslava Litecká, Jitka Prachařová, Monika Hreusová, and Jana Kašpárková. "Anticancer gallium(III) complexes with halogen- and nitroderivatives of 8- hydroxyquinoline." In 2nd International Conference on Chemo and Bioinformatics. Institute for Information Technologies, University of Kragujevac, 2023. http://dx.doi.org/10.46793/iccbi23.451p.

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Six gallium(III) complexes with halogen- or nitroderivatives of 8-hydroxyquinoline, [Ga(ClQ)3] (1), [Ga(BrQ)3] (2), [Ga(dIQ)3] (3), [Ga(CQ)3] (4), [Ga(NQ)3] (5) and [Ga(ClNQ)3]·MeOH were prepared as possible anticancer drugs (HClQ = 5-chloro-8-hydroxyquinoline, HBrQ = 7-bromo-8-roxyquinoline, HdIQ = 5,7-diiodo-8-hydroxyquinoline, HCQ = 5-chloro-7-iodo-8-ydroxyquinoline, HNQ = 5-nitro-8-hydroxyquinoline and HClNQ = 5-chloro-7-nitro-8-hydroxyquinoline). X-ray single crystal structure analysis of [Ga(ClQ)3]·MeOH (1 – MeOH) and 5 showed that both complexes are molecular complexes with Ga(III) coor

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Tanaka, Kyoko, Daisuke Aoki, Seiji Isonishi, Mikio Mikami, Kazushige Kiguchi, and Masao Iwamori. "Abstract 2241: Possible involvement of glycolipids in anticancer drug resistance of human ovarian serous carcinoma-derived cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2241.

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Wu, Jeffrey J., Alexander J. Neuwelt, Leslie L. Muldoon, and Edward A. Neuwelt. "Abstract 4904: Acetaminophen enhances the chemotherapeutic effect of cisplatin and paclitaxel on human ovarian carcinoma cells in vitro." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4904.

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Wu, Yingjen Jeffrey, Alexander J. Neuwelt, Leslie L. Muldoon, and Edward A. Neuwelt. "Abstract C47: Acetaminophen enhances the chemotherapeutic effect of cisplatin and paclitaxel on human ovarian carcinoma cells in vitro." In Abstracts: Second AACR International Conference on Frontiers in Basic Cancer Research--Sep 14-18, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.fbcr11-c47.

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Karst, Alison M., Keren Levanon, and Ronny Drapkin. "Abstract 1588: Transformation of primary human fallopian tube secretory epithelial cells: A cell of origin for high-grade serous ovarian carcinoma." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-1588.

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Raporty organizacyjne na temat "Human ovarian carcinoma cells"

Jones, Richard. Ovarian Carcinoma Stem Cells. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada508216.

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Jones, Richard. Targeting Ovarian Carcinoma Stem Cells. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada581693.

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Krager, Kimberly J., and Frederick E. Domann. Genetically Targeted Radiotherapy Utilizing the Human Sodium Iodide Symporter in Human Breast Carcinoma Cells. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada436156.

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Krager, Kimberly, and Frederick E. Domann. Genetically Targeted Radiotherapy Utilizing the Human Sodium Iodide Symporter in Human Breast Carcinoma Cells. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada455269.

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Dent, Paul. Radiosensitization of Human Mammary Carcinoma Cells by Specific Inhibition of Signal Transduction Cascades. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada409418.

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Lazo, John S., and Robert L. Rice. Protein Kinase C Processes and Their Relation to Apoptosis in Human Breast Carcinoma Cells. Defense Technical Information Center, 1995. http://dx.doi.org/10.21236/ada301315.

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Wang, Xinrun, Tianye Li, Xuechai Bai, Yun Zhu, and Meiliang Zhang. Therapeutic prospect on umbilical cord mesenchymal stem cells in animal model with primary ovarian insufficiency: A meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.5.0075.

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Review question / Objective: Participants: experiment POI animal models; Interventions: human umbilical cord mesenchymal stem cells; Comparisons: POI animal models without hUCMSC therapy; Outcomes: estrous cycle situation, serum sex hormone level and ovarian follicle count; Studies: randomized controlled animal study; The aim of the review is to figure out whether hUCMSC can recover ovarian function in POI animal models. Condition being studied: Primary ovarian insufficiency (POI) is a syndrome characterized by reduced or absent ovarian function (hypogonadism) and elevated levels of gonadotrop

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Azorsa, David. A Novel Approach to Identify Genes that Modulate Response of Human Ovarian Cancer Cells to Chemotherapeutic Agents Using High-Throughput RNA Interference. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada510407.

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Chanvorachote, Pithi. Roles of nitric oxide, reactive oxygen species, and their derivatives in regulation of lung cancer cell metastasis. Chulalongkorn University, 2013. https://doi.org/10.58837/chula.res.2013.28.

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The capability of cancer cells to resist to anoikis, migrate and invade surrounding tissues is associated with high metastatic potential and advanced stage of cancers. Recently, caveolin-1 (Cav-1) protein has garnered increased attention in implicating the aggressive behavior of cancer cells. We demonstrate herein that nitric oxide and hydrogen peroxide play a role in inhibiting anoikis process of lung cancer cells via caveolin-1 dependent mechanism. The Cav-1 function in inhibition of anoikis was demonstrated to be cause through Mcl-1 dependent mechanism. The present study demonstrated that C

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