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Książki na temat „HYPERTENSIVE DRUGS”

Autor: Grafiati

Data publikacji: 11 września 2023

Data aktualizacji: 25 lipca 2025

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1

Switzerland) International Symposium Clonidine in Hypertension (1984 Geneva. Low dose oral and transdermal therapy of hypertension: International Symposium Clonidine in Hypertension, Geneva, 14th-16th June, 1984. Steinkopff, 1985.

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M, Drayer Jan I., Lowenthal David T, and Weber Michael A, eds. Drug therapy in hypertension. Dekker, 1987.

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H, Messerli Franz, and Opie Lionel H, eds. Combination drug therapy for hypertension. Author's Pub. House, 1997.

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4

Mann, Samuel J. Hypertension and you: Old drugs, new drugs, and the right drugs for your high blood pressure. Rowman & Littlefield Publishers, 2012.

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5

van, Zwieten P. A., and Greenlee William J, eds. Antihypertensive drugs. Harwood Academic Publishers, 1997.

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6

1947-, Martin John E., and Dubbert Patricia M, eds. Non-drug treatments for essential hypertension. Pergamon Press, 1988.

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7

Bönner, Gerd. Prostacyclin and hypertension. Springer-Verlag, 1990.

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8

Parsont, Wolfson Rita, ed. How to control high blood pressure without drugs. Scribner, 1986.

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9

Walter, Flamenbaum, and Punzi Henry A, eds. Hypertension. Futura Pub. Co., 1989.

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10

Richard, Trubo, ed. The H.A.R.T. program: Lower your blood pressure without drugs. HarperCollins Publishers, 1992.

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11

Burnier, Michel, ed. Drug Adherence in Hypertension and Cardiovascular Protection. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-76593-8.

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12

Toma, Strasser, Ganten D. 1941-, World Health Organization, and International Society of Hypertension, eds. Mild hypertension: From drug trials to practice. Raven Press, 1987.

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13

Masaru, Minami, Parvez H, and Saitō Hideya 1933-, eds. Antihypertensive drugs today. VSP, 1992.

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14

Moore, Thomas J., M.D., ed. The DASH diet for hypertension: Lower your blood pressure in 14 days--without drugs. Pocket Books, 2003.

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15

Safar, Michel. Arteries in clinical hypertension. Lippincott-Raven, 1996.

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16

K, Hollenberg Norman, ed. Hypertension: Mechanisms and therapy. Current Medicine, 1995.

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17

1937-, Epstein Murray, and Loutzenhiser Rodger, eds. Calcium antagonists and the kidney. Hanley & Belfus, 1990.

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18

NHS Centre for Reviews & Dissemination., ed. Drug treatment of essential hypertension in older people. NHS Centre for Reviews and Dissemination, University of York, 1999.

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19

Jonkman, Fokke Anthonius Maria. Subchronic studies on experimental hypertension and antihypertensive drugs in the rat. [s.n.], 1986.

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20

L, Prisant Michael, ed. Hypertension in the elderly. Humana Press, 2005.

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21

Canadian Hypertension Society Consensus Conference (1993), ed. Guidelines for the treatment of uncomplicated hypertension. Queen's Printer for Ontario, 1995.

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22

Simons, David R. Hypertension: A physician's guide to drug selection and use. Keyed Reviews Publications, 1986.

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23

Nordmann, Alain Joel. Cost-effectiveness of routine echocardiography in hypertensive patients starting antihypertensive drug therapy. National Library of Canada, 2001.

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24

Igakubu, Hokkaidō Daigaku, ed. New advances in antihypertensive drug research: Pathophysiology and pharmacology. Hokkaido University School of Medicine, 1993.

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25

1942-, Black Henry R., ed. Clinical trials in hypertension. Marcel Dekker, 2001.

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26

Guthrie, Robert M. Pharmacological management of hypertension and dyslipidemia. Jones and Bartlett Publishers, 2010.

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27

Nederfors, Tommy. Xerostomia: Prevalence and pharmacotherapy : with special reference to -adrenoceptor antagonists. [Göteborg Univ.], 1996.

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28

J, Horan Michael, Page Lot B, National Institutes of Health (U.S.), and Workshop on Drug Side Effects, Drug-Drug Interactions, Drug Resistance, and Patient Compliance in the Management of Hypertension (1986 : Bethesda, Md.), eds. Drug side effects, drug-drug interactions, drug resistance and patient compliance in the management of hypertension: Bethesda, Maryland, April 28-29 1986. American Heart Association, 1988.

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29

1958-, Böhm M., and Laragh John H. 1924-, eds. From hypertension to heart failure. Springer, 1998.

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30

Zuckerman, Ilene H. Identifying drug therapy inappropriateness: Determining the validity of drug use review screening criteria. Center on Drugs and Public Policy, School of Pharmacy, University of Maryland at Baltimore, 1997.

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31

Richard, Moore. The K factor: Reversing and preventing high blood pressure without drugs. Pocket Books, 1987.

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32

Webb, Robyn. Eat to beat high blood pressure: Simple, delicious recipes for a long, healthy life : lower your blood pressure 10% or more without drugs : featuring the DASH-plus plan. Reader's Digest Association, 2008.

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33

Webb, Robyn. Eat to beat high blood pressure: Simple, delicious recipes for a long, healthy life : lower your blood pressure 10% or more without drugs : featuring the DASH-plus plan. Reader's Digest Association, 2008.

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34

Johnson, Nathaniel. Why African Americans get high blood pressure: New government report confirms that high blood pressure can be prevented and can often be treated without drugs. N. Johnson, 2001.

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35

Gidwani, Hitesh, and Chenell Donadee. Hypertensive Emergencies (DRAFT). Edited by Raghavan Murugan and Joseph M. Darby. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190612474.003.0009.

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Hypertensive emergencies may be encountered by rapid response teams (RRTs). Various forms of acute organ dysfunction separate hypertensive urgency from hypertensive emergency. These include acute heart failure, acute coronary syndrome, acute aortic dissection, ischemic stroke, hemorrhagic stroke, hypertensive encephalopathy, sympathetic crisis, postoperative hypertension, and hypertensive emergencies in pregnancy. RRTs must be able to rapidly assess the patient’s condition, initiate treatment, and triage the patient to the appropriate level of care. This chapter summarizes the initial evaluati

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36

Wijdicks, Eelco F. M., and Sarah L. Clark. Antihypertensives and Antiarrhythmics. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190684747.003.0013.

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Acute brain injury can precipitate a hypertensive response, which for the most part is the result of stress-induced, increased sympathetic activity. Acute stroke with hypertension may not be a response but more often a prior, untreated hypertension or a patient with no access to medication. This hypertensive response may wane quickly, and aggressive treatment of these temporary surges in blood pressure could have unwanted consequences. Important characteristics of most antihypertensive drugs used in the neurosciences intensive care unit are cost, having a rapid onset with a short duration of a

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37

Paneni, Francesco, and Massimo Volpe. Co-morbidity (HFrEF and HFpEF): hypertension. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198784906.003.0415_update_001.

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Hypertensive heart disease is a major cause of heart failure (HF) and mortality. Hypertension precedes HF occurrence in 75% of cases, and carries a sixfold increase in HF risk as compared to non-hypertensive individuals. Most importantly, a minority of patients survive 5 years after the onset of hypertensive HF. In hypertensive patients, the heart may present different patterns of adaptive remodelling: concentric remodelling, concentric hypertrophy, and eccentric hypertrophy. Although most hypertensive patients are at high risk of developing concentric hypertrophy, a growing proportion of subj

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38

Groeneveld, A. B. J., and Alexandre Lima. Vasodilators in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0035.

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Vasodilators are commonly used in the intensive care unit (ICU) to control arterial blood pressure, unload the left or the right heart, control pulmonary artery pressure, and improve microcirculatory blood flow. Vasodilator refers to drugs acting directly on the smooth muscles of peripheral vessel walls and drugs are usually classified based on their mechanism (acting directly or indirectly) or site of action (arterial or venous vasodilator). Drugs that have a predominant effect on resistance vessels are arterial dilators and drugs that primarily affect venous capacitance vessels are venous di

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39

Saito, H. Progress in Hypertension: Antihypertensive Drugs Today (Progress in Hypertension). Brill Academic Publishers, 1992.

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40

Shorter, Edward, and Max Fink. The Neuroleptic Malignant Syndrome. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190881191.003.0009.

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Reports of fatal febrile, hypertensive, tachycardic neurotoxic cases followed quickly on the introduction of potent new neuroleptic drugs in the 1970s. Patients became mute, rigid, posturing, and staring, showing the signs of catatonia. Labeled the neuroleptic malignant syndrome (NMS), attention was first given to neuroleptic blockade of dopamine receptors as the cause, but treatments with dopamine agonists (bromocriptine) and muscle relaxants (dantrolene) offered little benefit. When catatonia was recognized, treatments with benzodiazepines (lorazepam, diazepam) and induced seizures (electros

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41

Heart attacks, hypertension, and heart drugs. Rodale Press, 1987.

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42

Barthel, Andreas, and Michael Bauer. Psychotropic drugs and metabolic risk. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198789284.003.0011.

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Increased appetite and weight gain represent a significant problem related with particular antipsychotic drugs, antidepressants, mood stabilizers, and—to a lesser extent—anxiolytic drugs. Psychotropic drug-induced weight gain may contribute to obesity-related metabolic changes and pathological conditions such as dyslipidaemia, type-2-diabetes and hypertension—summarized as the metabolic syndrome—with an increased risk for cardiovascular morbidity and mortality. Interestingly, psychotropic drugs are also used for the treatment of diabetes-related complications. For example, antidepressants are

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43

Messerli, Franz H., and Opie Lionel H. Combination Drug Therapy for Hypertension. Lippincott Williams & Wilkins, 1997.

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44

Clinical Pharmacology of Antihypertensive Drugs. 2nd ed. Elsevier Science Publishing Company, 1988.

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45

Caldwell, Scott. Antihypertensive Drugs: Pharmacology, Medical Uses and Potential Side Effects. Nova Science Publishers, Incorporated, 2015.

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46

Mann, M. J., and Mann, M.D., Samuel J. Hypertension and You: Old Drugs, New Drugs, and the Right Drugs for Your High Blood Pressure. Rowman & Littlefield Publishers, Incorporated, 2012.

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47

Blanchard-Loeb. Hypertension: Expert Drug Therapy Video Series. Delmar Learning, 2000.

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48

Shen, Howard. MemoCharts Pharmacology: Drug Therapy for Hypertension. Minireview, 2014.

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49

Sutter, Johan De, Miguel Mendes, and Oscar H. Franco. Cardioprotective drugs. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656653.003.0019.

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Cardioprotective drugs are important in the treatment of patients at risk for or with documented cardiovascular disease. Beta-blockers are indicated after acute coronary syndromes, stable coronary artery disease, heart failure, and arrhythmias. Angiotensin-converting enzyme inhibitors (ACEi) are important in congestive heart failure, stable angina, post-acute myocardial infarction, and secondary prevention after any event or revascularization. Angiotensin receptor blockers are mainly alternative drugs for the same indications in case of intolerance to ACEi. Calcium channel blockers are first l

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50

Sutter, Johan De, Miguel Mendes, and Oscar H. Franco. Cardioprotective drugs. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199656653.003.0019_update_001.

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Cardioprotective drugs are important in the treatment of patients at risk for or with documented cardiovascular disease. Beta-blockers are indicated after acute coronary syndromes, stable coronary artery disease, heart failure, and arrhythmias. Angiotensin-converting enzyme inhibitors (ACEi) are important in congestive heart failure, stable angina, post-acute myocardial infarction, and secondary prevention after any event or revascularization. Angiotensin receptor blockers are mainly alternative drugs for the same indications in case of intolerance to ACEi. Calcium channel blockers are first l

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