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Gotowe bibliografie tematyczne / Inflammation Mediators / Rozprawy doktorskie

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Rozprawy doktorskie na temat „Inflammation Mediators”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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1

Farrell, Adrian J. "Mediators of synovial inflammation." Thesis, University of Bristol, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.284326.

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Woollard, Kevin J. "Mediators of monocyte activity in inflammation." Thesis, Aston University, 2003. http://publications.aston.ac.uk/11001/.

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The effects of incubation of CRP with human primary and monocytic cell lines were examined using monocytic cytokine expression, adhesion molecule expression and adhesion to endothelial cells and intracellular peroxide formation, as end points. Monocytic intracellular signalling events were investigated after interaction of CRP with specific CRP receptors on monocytes. These initial signalling events were examined for their role in modulating monocyric adhesipn molecule and cytokine expression. Monocyte recruitment and retention in the vasculature is also influenced by oxidative stress. Therefo

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3

Robinson, Emily. "Mediators of inflammation in acute neurotoxicity." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/mediators-of-inflammation-in-acute-neurotoxicity(a657668f-f6de-4fb6-878d-65846fc18e66).html.

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Neuroinflammation is a major feature of most neurodegenerative conditions, and can leadto the exacerbation of neuronal injury. Inflammatory challenges in the central nervoussystem (CNS) have been shown to activate peripheral immune cells, which subsequentlyinfiltrate into the brain. Concurrently, resident inflammatory cells in the CNS, such asmicroglia, become activated and release inflammatory mediators, including cytokines.The pro-inflammatory cytokine interleukin-1 (IL-1) is a key mediator of neuronal injury.Although two IL-1 agonists exist, IL-1α and IL-1β, the majority of research has foc

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4

Wesseldijk, Feikje. ""Inflammatory Soup" mediators of inflammation in CRPS." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2008. http://hdl.handle.net/1765/13553.

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5

Wilson, Susan Jane. "Mucosal inflammation in allergic rhinitis." Thesis, University of Southampton, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295233.

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6

McKay, Anne. "The role of immune mediators in airway inflammation." Thesis, University of Glasgow, 2004. http://theses.gla.ac.uk/4828/.

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Asthma is a chronic inflammatory condition of the airways characterised by reversible airflow obstruction, airway hyper-responsiveness and inflammatory infiltrates in the airway walls containing eosinophils, T lymphocytes and mast cells. T helper (Th) lymphocyte subsets, defined by the cytokines they secrete, are thought to play a key role in the in the initiation and perpetuation of chronic airway inflammation. Th2 cells, producing interleukin (IL)-4, IL-5, IL-9 and IL-13, are thought to be of particular importance. In contrast, Thl cells producing interferon (IFN)-y may counteract the develo

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7

李蕙琛 and Wai-sum Rachel Li. "Control of adenosine in human umbilical vein endothelial cells during inflammation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39557479.

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8

Airila-Månsson, Stella. "Progression of periodontitis and influence of periodontal bacteria on release of inflammatory markers in Swedish adults /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-622-0/.

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9

Khan, Sarah Basir. "Mediators of inflammation and fibrosis in experimental crescentic glomerulonephritis." Thesis, Imperial College London, 2005. http://hdl.handle.net/10044/1/11303.

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10

Porter, John Robert Stephen. "Microvesicles as mediators of inflammation in severe burn injury." Thesis, Imperial College London, 2015. http://hdl.handle.net/10044/1/43963.

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The host response to a severe burn injury is characterised by exaggerated systemic inflammation. Early clinical manifestations include shock, respiratory failure, renal failure and immunosuppression. The signalling pathways that propagate the inflammatory response are unclear but have traditionally been thought to involve overspill of proinflammatory cytokines. The importance of microvesicles, sub-cellular membrane-bound particles, is increasingly being recognised in the context of intercellular communication. Although circulating microvesicles are elevated in proinflammatory states such as se

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11

Andersson, Åsa. "Macrophages as central inflammatory mediators and as targets for therapeutic interventions /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-618-2/.

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12

Djukanovic, Ratko. "Cellular and soluble mediators of mucosal inflammation in bronchial asthma." Thesis, University of Southampton, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295878.

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13

Bhagat, Kiran. "Vascular inflammation and its effects on endothelial and smooth muscle function, a study in healthy volunteers." Thesis, University College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263301.

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14

Baxter, Edith. "Potential mediators of the decrease in CYP1A activity during CNS inflammation." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0018/MQ57182.pdf.

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15

Nicholson, Tara. "Potential mediators of the downregulation of cytochrome P450 during central inflammation." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/NQ66639.pdf.

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16

Pépin, Marion. "Caractérisation de nouveaux médiateurs entre cancer, inflammation et thrombose : ADAMTS13, PSGL-1 et Siglec-5." Thesis, Université Paris-Saclay (ComUE), 2016. http://www.theses.fr/2016SACLS571/document.

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La maladie cancéreuse et la maladie thrombotique sont des problématiques fréquentes et graves rencontrées en pathologie humaine et en santé publique. Elles ont une relation réciproque médiée par des interactions cellulaires et des mécanismes moléculaires complexes faisant intervenir des acteurs de l’hémostase et de l’immunité. Tout ou partie de ces phénomènes rendent en effet compte du processus d’inflammation et de réponse de l’organisme à l’agression. La caractérisation de certains médiateurs moléculaires ayant des rôles connus dans l’hémostase et l’inflammation peut nous permettre d’amélior

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17

Hasan, Hisham Ahmed. "The effects of glucocorticoids and other pharmacological agents on the production of cytokines and prostaglandin Eâ†2 by human cells in vitro." Thesis, University of Sheffield, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265572.

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18

Fehrenbacher, Jill Christine. "The contribution of inflammation and inflammatory mediators to sensitization of sensory neurons." [Bloomington, Ind.] : Indiana University, 2005. http://wwwlib.umi.com/dissertations/fullcit/3183931.

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Thesis (Ph.D.)--Indiana University, 2005.<br>Source: Dissertation Abstracts International, Volume: 66-07, Section: B, page: 3648. Chairperson: Michael R. Vasko. Title from dissertation home page (viewed Oct. 5, 2006).

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19

Li, Shun. "Crosstalk between mediators of inflammation and the IGF axis in liver metastasis." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=104626.

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Liver metastases are a frequent and often fatal occurrence in cancer patients. A better understanding of the biology of liver metastasis is essential to improve survival statistics for these patients. Although the contribution of IGF-IR to malignancy has been recognized for over 2 decades, the role that the IGF axis plays in metastasis is not yet fully understood. Using a murine Lewis lung carcinoma-based model, our laboratory has previously shown that the IGF-I receptor (IGF-IR) plays the key role in liver metastasis by regulating matrix metalloproteinase (MMP) expression and by increasing t

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20

Coras, Roxana Georgiana. "Identification of Metabolites as Biomarkers and Mediators of Inflammation in Inflammatory Arthritis." Doctoral thesis, Universitat Autònoma de Barcelona, 2021. http://hdl.handle.net/10803/673867.

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L’artritis inflamatòria representa una gran càrrega social i econòmica tot i els recents avenços terapèutics. Malgrat un millor coneixement dels mecanismes patogénics, l’elecció de l’tractament encara es realitza a manera de prova, el que condueix a una manca de control de l’activitat de la malaltia en aproximadament el 30% dels pacients i una alta taxa d’efectes secundaris. La identificació de mediadors de malaltia i predictors de resposta a el tractament és necesaria per permetre el tractament adequat i aconseguir la remissió clínica o al menys una baixa activitat de la malaltia. És poc prob

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21

Zulkhernain, Nursafwana Syazwani. "Identifying Perivascular Macrophages as Crucial Mediators of Leukocyte Homing in Sterile Inflammation." Thesis, The University of Sydney, 2021. https://hdl.handle.net/2123/25758.

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Background: Numerous studies have elucidated key molecular mechanisms underlying leukocyte recruitment; however, little is known about the coordination between infiltrating leukocytes and perivascular immune cells during sterile inflammation. As perivascular macrophages (PVM) have been implicated in the pathogenesis of various sterile inflammatory diseases, it is noteworthy to explore their function in this setting. The recruitment of cytotoxic T lymphocytes (CTLs) has been implicated in shaping the immune milieu within various tissues. Although the Arp2/3 complex has been shown to be impor

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22

Fredriksson, Lars. "Local and systemic inflammatory mediators and their relation to pressure-pain threshold and pain of the temporomandibular joint /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-876-2/.

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23

Mullins, Gail E. "Studies of high mobility group box chromosomal protein 1 as a pro-inflammatory cytokine /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-333-7/.

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24

Bao, Lei. "Immunomodulation and immunopathogenesis in the autoimmune disease with emphasis on autoimmune neuritis and arthritis /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-447-X/.

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25

Loo, Tjing Yung, and 魯慶榮. "Profiles of cytokines and inflammatory mediators: implications in periodontal assessment." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B45893305.

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26

Abelin, Törnblom Susanne. "Mediators of cervical ripening in preterm birth : experimental and clinical investigations /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-305-1/.

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27

Lau, Yuen-chi Roy, and 劉源智. "Role of cyclooxygenases in monocrotaline induced pulmonary injury." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B45010146.

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28

Pak, Anne. "The influence of inflammation and inflammatory mediators on p-glycoprotein expression in rats." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0024/MQ40858.pdf.

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29

Ford, Louise. "Wolbachia mediators of inflammation and their role in the immune response to filariasis." Thesis, University of Liverpool, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.273984.

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30

Faull, Randall James. "Studies of vascular endothelial cell surface antigens relevant to the alloimmune response." Title page, table of contents and abstract only, 1991. http://web4.library.adelaide.edu.au/theses/09PH/09phf2634.pdf.

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Bibliography: leaves 234-314. Examines the role of vascular endothelial cells in inflammation with particular reference to their participation in the immune response directed against a vascularised allograft (kidney)

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31

Kim, Sungwon. "Functional Characterization of the Avian Inflammatory Mediators Nod1, MIF and IL-22." Diss., Virginia Tech, 2011. http://hdl.handle.net/10919/77232.

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Inflammation can be initiated by an innate immune sensor, followed by activation of a signal mediator, resulting in control of immune response by a signal regulator. Mammalian nucleotide-binding oligomerization domain protein 1 (Nod1) and Nod2 initiate host innate immune response by recognition of specific bacterial molecules, resulting in the production of pro-inflammatory cytokines, chemokines, and anti-microbial peptides. A candidate sequence of chicken Nod1 (ChNod1) was identified with no current evidence of ChNod2. Stimulation of transiently overexpressed ChNod1 and its mutants with mamma

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32

Tsaprouni, Loukia G. "Histone acetylation and inflammatory mediators in inflammatory bowel disease." Thesis, University of Bedfordshire, 2003. http://hdl.handle.net/10547/620761.

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During cell activation the tightly compacted DNA is made available to DNA-binding proteins allowing the induction of gene transcription. In the resting cell, DNA is packaged into chromatin whose fundamental subunit is the nucleosome, composed of an octamer of four core histones (H) 3, 4, 2A and 2B. During the induction of gene transcription, modification of histones, by acetylation, methylation etc., results in unwinding of the DNA, permitting access of large DNAbinding proteins, such as RNA polymerase II, and subsequent induction of gene transcription. This investigation initially examined th

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33

AL-Hashimi, Najat AL-Sayed. "Experimental studies on effects of orthodontic forces in generation of immune responses : possible roles for immunoregulating molecules in the control of alveolar bone remodeling /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-985-4/.

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34

Yamen, Eric. "Inflammation and atherosclerosis: Serum amyloid A as an inflammatory mediator in coronary artery disease, and the contribution of coronary plaque to inflammation in atherosclerosis." Thesis, The University of Sydney, 2011. https://hdl.handle.net/2123/29219.

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The relationship between inflammation and atherosclerosis is increasingly well recognised. Systemic inflammatory states, either due to auto-immune illness, or subclinical inflammation measured by inflammatory markers, are associated with increased prevalence of coronary artery disease (CAD) and acute coronary syndromes (ACS). Serum amyloid A (SAA) is an inflammatory marker associated with CAD in some studies, and in vitro work has suggested that it may induce a pro-coagulant state in leucocytes. Additionally, pathological specimens of atheroma contain large numbers of leucocytes which ex

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35

Perkins, Douglas Jay. "Soluble mediators of inflammation and their effect on cyclooxygenase-2: Implications in preterm labor /." The Ohio State University, 1997. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487943610782714.

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36

Nopp, Anna. "Characterisation of eosinophil activity markers : relation to allergic inflammation and apoptosis /." Stockholm : Karolinska Univ. Press, 2002. http://diss.kib.ki.se/2002/91-7349-129-2.

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37

Hamawy, Majed Mahmood. "Antigen induced modulation of autonomic nervous system responses in immunoglobulin-E - sensitized rabbit lung." Diss., The University of Arizona, 1988. http://hdl.handle.net/10150/184590.

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The major objective of this project was to examine the potential for mediators of IgE-mediated allergic reactions to alter neural activity. The project was divided into three parts. In Part I, the ability of endogenously released chemical mediators to alter neural activity in vitro was assessed by measuring isometric contractile responses to electrical field stimulation (EFS) (2-128 Hz, 20 V, 0.5 msec. duration) of sensitized rabbit bronchi before and after exposure to the antigen horseradish peroxidase (HRP). Antigen enhanced bronchial responses to EFS with low frequencies: mean log (± S.E.)

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38

Harkin, Damien Gerard. "Morphological responses of neutrophils in suspension to plasma components and chemotactic factors /." Title page, contents and summary only, 1992. http://web4.library.adelaide.edu.au/theses/09PH/09phh282.pdf.

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39

Redington, Anthony Edward. "Fibrogenic mediators in atopic asthma : expression of endothelin, transforming growth factor-beta and basic fibroblast growth factor." Thesis, University of Southampton, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.242665.

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40

Xia, Qiong. "The role of interleukin 22 and non-ELR-CXC chemokines in human carotid plaque." Thesis, The University of Sydney, 2010. https://hdl.handle.net/2123/28894.

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Objective: Inflammation plays an important role in atherogenesis, especially in plaque destabilisation. IL-22 enhances the innate immunity of tissue cells, favours the anti-microbial defence and protects tissue from damage. IL-22 is described to have a protective role in many chronic inflammatory disorders, but its role in atherosclerosis is unclear. Previous work in our laboratory demonstrated that there was high level both anti— and pro- inflammatory cytokines production in the plaques, particularly from symptomatic patients. However the underlying mechanism of the inflammation in the

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41

Moore, Andrea Rossi. "COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis." Diss., Temple University Libraries, 2010. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/81890.

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Microbiology and Immunology<br>Ph.D.<br>Rheumatoid arthritis (RA) is a chronic disease characterized by cycles of inflammation and resolution. Previously, it was believed that the resolution of inflammation is simply dissipation of pro-inflammatory signals, although current research indicates that resolution is an active process. Acute inflammation follows defined phases of induction, inflammation and resolution, and resolution occurs by an active process that requires COX-2 activity. This study aims to address whether this paradigm extends to a recognized model of chronic inflammation. We dem

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42

McDaniel, J., Karen A. Massey, and Anna Nicolaou. "Fish oil supplementation alters levels of lipid mediators of inflammation in microenvironment of acute human wounds." Wiley, 2010. http://hdl.handle.net/10454/4577.

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no<br>Chronic wounds often result from prolonged inflammation involving excessive polymorphonuclear leukocyte activity. Studies show that the omega-3 polyunsaturated fatty acids eicosapentaenoic and docosahexaenoic acids found in fish oils generate bioactive lipid mediators that reduce inflammation and polymorphonuclear leukocyte recruitment in numerous inflammatory disease models. The purpose of this study was to test the hypotheses that boosting plasma levels of eicosapentaenoic and docosahexaenoic acids with oral supplementation would alter lipid mediator levels in acute wound microenviro

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43

Nist, Marliese Dion. "Inflammatory Mediators of Stress Exposure and Neurodevelopment in Very Preterm Infants." The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1565718071063954.

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44

Blaho, Victoria Alison. "Lipid mediators in the development and resolution of experimental lyme arthritis." Diss., Columbia, Mo. : University of Missouri-Columbia, 2007. http://hdl.handle.net/10355/4819.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2007.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. "May 2007" Includes bibliographical references.

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45

Sylvin, Helena. "Allergen-induced airway reactions in the pig in vivo : inflammatory mediators as targets for asthma therapy /." Stockholm : [Karolinska institutets bibl.], 2002. http://diss.kib.ki.se/2002/91-7349-157-8.

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46

Malamis, Dimitrios. "Systemic levels of inflammatory mediators in periodontitis." The Ohio State University, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=osu1436961245.

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47

Ehrs, Per Olof. "Quality of life and markers of inflammation : a study of asthma in primary care /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-539-9/.

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48

Moëll, Annika. "Inflammatory mediators and enterovirus infections in human islets of Langerhans /." Uppsala : Acta Universitatis Upsaliensis : Universitetsbiblioteket [distributör], 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-8501.

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49

Hosseini, Abolfazl. "Nitric oxide : a marker for inflammation in the lower urinary tract /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-920-X/.

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50

Van, de Vyver Mari. "The contribution of inflammatory mediators to delayed secondary muscle damage." Thesis, Stellenbosch : Stellenbosch University, 2013. http://hdl.handle.net/10019.1/79787.

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Thesis (PhD)--Stellenbosch University, 2013.<br>ENGLISH ABSTRACT: Background: Understanding the contribution of divergent individual response patterns remains a key objective in identifying mechanisms of inflammation and potential factors limiting the resolution of inflammation. The purpose of this research project was to investigate downstream effects of inflammation following exercise-induced muscle damage in human subjects. Methods: For three different studies, a total of 53 untrained healthy male participants were recruited and divided into a non-exercising control (n=13) and exercise-ind

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