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Gotowe bibliografie tematyczne / Insulin-deficient patien

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Gotowa bibliografia na temat „Insulin-deficient patien”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Insulin-deficient patien"

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Arnold, Amy C., Emily M. Garland, Jorge E. Celedonio та ін. "Hyperinsulinemia and Insulin Resistance in Dopamine β-Hydroxylase Deficiency". Journal of Clinical Endocrinology & Metabolism 102, № 1 (2016): 10–14. http://dx.doi.org/10.1210/jc.2016-3274.

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Abstract Context: Dopamine β-hydroxylase (DBH) deficiency is a rare genetic disorder characterized by failure to convert dopamine to norepinephrine. DBH-deficient patients lack sympathetic adrenergic function and are therefore predisposed to orthostatic hypotension. DBH-deficient mice exhibit hyperinsulinemia, lower plasma glucose levels, and insulin resistance due to loss of tonic sympathetic inhibition of insulin secretion. The impact of DBH deficiency on glucose homeostasis in humans is unknown. Case Description: We describe the metabolic profile of an adolescent female DBH-deficient patien

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Gutiérrez-Fernández, Diego, Álvaro Moreno-Ancillo, Antonio Foncubierta-Fernández, Isabel López-Martínez, Alfonso Lechuga-Sancho, and María José Fernández-Anguita. "Desensitization to mecasermin in an insulin-like growth factor 1–deficient patient." Annals of Allergy, Asthma & Immunology 114, no. 6 (2015): 523–25. http://dx.doi.org/10.1016/j.anai.2015.03.004.

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Paz-Filho, Gilberto, Karin Esposito, Barry Hurwitz, et al. "Changes in insulin sensitivity during leptin replacement therapy in leptin-deficient patients." American Journal of Physiology-Endocrinology and Metabolism 295, no. 6 (2008): E1401—E1408. http://dx.doi.org/10.1152/ajpendo.90450.2008.

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Leptin replacement rescues the phenotype of morbid obesity and hypogonadism in leptin-deficient adults. However, leptin's effects on insulin resistance are not well understood. Our objective was to evaluate the effects of leptin on insulin resistance. Three leptin-deficient adults (male, 32 yr old, BMI 23.5 kg/m2; female, 42 yr old, BMI 25.1 kg/m2; female, 46 yr old, BMI 31.7 kg/m2) with a missense mutation of the leptin gene were evaluated during treatment with recombinant methionyl human leptin (r-metHuLeptin). Insulin resistance was determined by euglycemic hyperinsulinemic clamps and by or

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Hawari, Iman, Johan Ericsson, Basirudeen Syed Ahamed Kabeer, et al. "Understanding the Mechanism of Diabetes Mellitus in a LRBA-Deficient Patient." Biology 11, no. 4 (2022): 612. http://dx.doi.org/10.3390/biology11040612.

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The scope of this study is to show that DM in a LRBA-deficient patient with a stop codon mutation (c.3999 G > A) was not mediated through autoimmunity. We have evaluated the ability of the proband’s T cells to be activated by assessing their CTLA-4 expression. A nonsignificant difference was seen in the CTLA-4 expression on CD3+ T cells compared to the healthy control at basal level and after stimulation with PMA/ionomycin. Blood transcriptomic analysis have shown a remarkable increase in abundance of transcripts related to CD71+ erythroid cells. There were no differences in the expression

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Ashraf, Soliman, Alyafei Fawzia, De Sanctis Vincenzo, et al. "The impact of growth hormone (GH) therapy on glucose metabolism: A narrative review mainly focused on GH-deficient (GHD) children and adolescents." World Journal of Advanced Research and Reviews 22, no. 1 (2024): 627–36. https://doi.org/10.5281/zenodo.14209105.

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<strong>Introduction</strong>: Growth hormone (GH) deficiency (GHD) in children and adolescents is traditionally managed with GH therapy, which, while effective for promoting growth, poses potential metabolic repercussions, particularly concerning glucose metabolism. This review delineates the complex interplay between GH therapy, insulin sensitivity, and glucose homeostasis. <strong>Objective</strong>: To synthesize existing literature on the effects of GH therapy on glucose metabolism, insulin secretion, and insulin sensitivity in GH-deficient pediatric populations, aiming to illuminate the

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Radicioni, Antonio F., Matteo Spaziani, Gilda Ruga, et al. "Growth Hormone Deficiency During the Transition Phase." US Endocrinology 08, no. 02 (2012): 118. http://dx.doi.org/10.17925/use.2012.08.02.118.

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The growth hormone (GH)-insulin-like growth factor-1 (IGF-1) axis has several roles. While achievement of a satisfactory height is probably the most important and well-known, it is now clear that it also affects body composition, metabolism, muscle mass, and bone density during the transition period. Recombinant-growth hormone (Rec-GH) therapy is normally administered to GH-deficient children to achieve a reasonable final height. Retesting with a provocative test (insulin tolerance test or growth-hormone-releasing hormone + arginine test) is necessary during the transition period, after measur

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Radicioni, Antonio F., Matteo Spaziani, Gilda Ruga, et al. "Growth Hormone Deficiency During the Transition Phase." European Endocrinology 8, no. 2 (2010): 112. http://dx.doi.org/10.17925/ee.2012.08.02.112.

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The growth hormone (GH)-insulin-like growth factor-1 (IGF-1) axis has several roles. While achievement of a satisfactory height is probably the most important and well-known, it is now clear that it also affects body composition, metabolism, muscle mass and bone density during the transition period. Recombinant-growth hormone (Rec-GH) therapy is normally administered to GH-deficient children to achieve a reasonable final height. Retesting with a provocative test (insulin tolerance test or growth-hormone-releasing hormone + arginine test) is necessary during the transition period, after measuri

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Schleis, Gregory, and Neda Rasouli. "Two Extremes of Dysglycemia- a Rare Case of Diabetes Complicated With a Non-Functional Pancreatic Neuroendocrine Tumor Transforming to an Insulinoma." Journal of the Endocrine Society 5, Supplement_1 (2021): A404—A405. http://dx.doi.org/10.1210/jendso/bvab048.823.

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Abstract Background: Diabetes is characterized by hyperglycemia with heterogeneous pathophysiological features and varied presentation and consequences. Here, we present a rare case of diabetes that was complicated with insulinoma making glycemia management complicated. Clinical Case: A 48-year-old white man was diagnosed with diabetes when he presented with weight loss, polyuria and polydipsia and HbA1c of 11%. He was treated with oral agents for few months without a good response then switched to insulin. He was assumed to have type 1 diabetes and had fairly well controlled glycemia on multi

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Li, Xia, Shuting Yang, Chuqing Cao, et al. "Validation of the Swedish Diabetes Re-Grouping Scheme in Adult-Onset Diabetes in China." Journal of Clinical Endocrinology & Metabolism 105, no. 10 (2020): e3519-e3528. http://dx.doi.org/10.1210/clinem/dgaa524.

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Abstract Context This study applied the Swedish novel data-driven classification in Chinese newly diagnosed diabetic patients and validated its adoptability. Objective This study aimed to validate the practicality of the Swedish diabetes regrouping scheme in Chinese adults with newly diagnosed diabetes. Design Patients were classified into 5 subgroups by K-means and Two-Step methods according to 6 clinical parameters. Setting Ambulatory care. Patients A cross-sectional survey of 15 772 patients with adult-onset newly diagnosed diabetes was conducted in China from April 2015 to October 2017. In

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Kutoh, Eiji, Jyunka Hayashi, and Alexandra N. Kuto. "Efficacy and safety of dulaglutide in patients with absolute insulin deficiency." Endocrine Regulations 53, no. 3 (2019): 187–90. http://dx.doi.org/10.2478/enr-2019-0019.

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AbstractObjective. While dulaglutide has been approved inpatients with type 2 diabetes (T2DM) in combination with insulin, it has not been studied in insulin-deficient patients, not whether they have type 1 diabetes (T1DM) or T2DM. The aim of this study is to assess the efficacy and safety of dulaglutide 0.75 mg/once weekly (QW) in patients with absolute insulin deficiency (n=10).Subjects and Results. Significant reductions of HbA1c (9.30±1.03% to 8.61±1.21%; p<0.02) and body mass index (BMI; 23.61±3.95 to 23.41±4.24; p<0.02) levels were observed at 3 months with the addition of dulaglut

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Streszczenia konferencji na temat "Insulin-deficient patien"

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Freeman, Kenneth A., Qian Wang, Peter Molenaar, Jan Ulbrecht, Carol H. Gold, and Mike Rovine. "Development of an Adaptive Dual Controller for Glucose Management in Patients With Insulin-Deficient Diabetes." In ASME 2012 5th Annual Dynamic Systems and Control Conference joint with the JSME 2012 11th Motion and Vibration Conference. ASME, 2012. http://dx.doi.org/10.1115/dscc2012-movic2012-8803.

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