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Gotowe bibliografie tematyczne / Interleukin-10/biosynthesis

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Artykuły w czasopismach

Gotowa bibliografia na temat „Interleukin-10/biosynthesis”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Interleukin-10/biosynthesis"

1

Stahl, Matthias, Hua Cheng, Susanne Linde, and Jens Høiriis Nielsen. "Cortisol increases the susceptibility of rat islets to interleukin 1." Acta Endocrinologica 125, no. 4 (1991): 441–48. http://dx.doi.org/10.1530/acta.0.1250441.

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Abstract. Interleukin 1β has been proposed to play an essential role in the pathogenesis of IDDM by direct interaction with the pancreatic beta-cell. Glucocorticoids are widely used as immunosuppressive agents and have been suggested to interfere both with the production and action of interleukin 1. The aim of the present study was to evaluate the interaction between cortisol and interleukin-1 on the pancreatic beta-cell function in vitro. Newborn rat islets were precultured for seven days before they were exposed to interleukin 1 with or without addition of cortisol. The release of insulin to

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Huang, Chun-Jen, Bruce R. Stevens, R. Barton Nielsen, et al. "Interleukin-10 Inhibition of Nitric Oxide Biosynthesis Involves Suppression of CAT-2 Transcription." Nitric Oxide 6, no. 1 (2002): 79–84. http://dx.doi.org/10.1006/niox.2001.0402.

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Fauser, Bart C. J. M., A. Brenda Galway та Aaron J. W. Hsueh. "Inhibitory actions of interleukin-1β on steroidogenesis in primary cultures of neonatal rat testicular cells". Acta Endocrinologica 120, № 4 (1989): 401–8. http://dx.doi.org/10.1530/acta.0.1200401.

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Abstract. Interleukin-1 is an important cytokine produced by activated macrophages. Because macrophages have been localized in the testis and interleukin-1 bioactivity has been observed in the testis, the potential effect of interleukin-1 on gonadotropin-stimulated androgen production was investigated using primary cultures of neonatal rat testis cells. Cells were incubated for 3 days before change of medium and treatment with human chorionic gonadotropin and interleukin-1. After 3 additional days medium testosterone and progesterone levels were determined. Human chorionic gonadotropin treatme

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Petsini, Filio, Maria Detopoulou, Maria Choleva, Ioannis K. Kostakis, Elizabeth Fragopoulou, and Smaragdi Antonopoulou. "Exploring the Effect of Resveratrol, Tyrosol, and Their Derivatives on Platelet-Activating Factor Biosynthesis in U937 Cells." Molecules 29, no. 22 (2024): 5419. http://dx.doi.org/10.3390/molecules29225419.

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Platelet-activating factor (PAF) is a potent lipid mediator, involved in thrombosis, inflammation, and atherosclerosis. The protective effect of wine and olive oil against atherosclerotic diseases is largely attributed to their phenolic compounds and mostly to resveratrol and tyrosol. Both compounds have been reported to inhibit PAF biosynthesis in interleukin-1β (IL-1β)-stimulated monocytes and also to attenuate PAF biosynthesis in cell lysates. The aim of this study was to investigate the effects of resveratrol, tyrosol, and their derivatives on unstimulated U937 cells and to explore the int

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5

Deng, Jingti, Christian H. James, Lisa Patel, et al. "Human tribbles homologue 2 is expressed in unstable regions of carotid plaques and regulates macrophage IL-10 in vitro." Clinical Science 116, no. 3 (2009): 241–48. http://dx.doi.org/10.1042/cs20080058.

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Mammalian orthologues of the Drosophila tribbles protein (Trb1, Trb2 and Trb3) are a recently described family of signalling molecules that regulate gene expression by modulation of protein kinase signalling pathways. In the present study, a screen for mRNA species specifically regulated in vulnerable regions of human atherosclerotic plaque demonstrated the up-regulation of both Trb1 and Trb2, the latter by more than 8-fold. In vitro experiments in primary human monocyte-derived macrophages showed that Trb2 expression was up-regulated by treatment with oxidized LDL (low-density lipoprotein), a

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Lesnova, Ekaterina I., Olga V. Masalova, Kristina Yu Permyakova, et al. "Difluoromethylornithine (DFMO), an Inhibitor of Polyamine Biosynthesis, and Antioxidant N-Acetylcysteine Potentiate Immune Response in Mice to the Recombinant Hepatitis C Virus NS5B Protein." International Journal of Molecular Sciences 22, no. 13 (2021): 6892. http://dx.doi.org/10.3390/ijms22136892.

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Hepatitis C virus (HCV) is one of the main triggers of chronic liver disease. Despite tremendous progress in the HCV field, there is still no vaccine against this virus. Potential vaccines can be based on its recombinant proteins. To increase the humoral and, especially, cellular immune response to them, more effective adjuvants are needed. Here, we evaluated a panel of compounds as potential adjuvants using the HCV NS5B protein as an immunogen. These compounds included inhibitors of polyamine biosynthesis and urea cycle, the mTOR pathway, antioxidants, and cellular receptors. A pronounced sti

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Fan, ST, K. Hsia, and TS Edgington. "Upregulation of human immunodeficiency virus-1 in chronically infected monocytic cell line by both contact with endothelial cells and cytokines." Blood 84, no. 5 (1994): 1567–72. http://dx.doi.org/10.1182/blood.v84.5.1567.bloodjournal8451567.

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Cells of monocytic lineage (Mo) persistently infected with human immunodeficiency virus (HIV) have been suspected to be a major reservoir for in vivo transmission of virus to susceptible target cells. Cellular events and mechanisms that upregulate viral gene expression in such cells are important issues. Because the traffic of such cells is central to biodistribution of HIV, we have explored the impact of interaction of endothelium with HIV-1-infected U1 promonocytic cells. Coculturing of U1 with human umbilical endothelial cells (HUVEC) for 24 to 72 hours in the absence of stimulation induced

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Cheng, Yi-Lin, Chi-Yun Wang, Wei-Ching Huang та ін. "Staphylococcus aureus Induces Microglial Inflammation via a Glycogen Synthase Kinase 3β-Regulated Pathway". Infection and Immunity 77, № 9 (2009): 4002–8. http://dx.doi.org/10.1128/iai.00176-09.

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ABSTRACT A proinflammatory role for glycogen synthase kinase 3β (GSK-3β) has been demonstrated. Here, we addressed its roles on heat-inactivated Staphylococcus aureus-induced microglial inflammation. Heat-inactivated S. aureus induced tumor necrosis factor alpha (TNF-α) and nitric oxide (NO) production, at least in part, via a Toll-like receptor 2-regulated pathway. Neutralization of TNF-α largely blocked heat-inactivated S. aureus-induced NO. Heat-inactivated S. aureus activated GSK-3β, and inhibiting GSK-3β reduced TNF-α production as well as inducible NO synthase (iNOS)/NO biosynthesis. Whi

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Huang, Wei-Ching, Yee-Shin Lin, Chi-Yun Wang, et al. "Glycogen synthase kinase-3 negatively regulates anti-inflammatory interleukin-10 for lipopolysaccharide-induced iNOS/NO biosynthesis and RANTES production in microglial cells." Immunology 128, no. 1pt2 (2009): e275-e286. http://dx.doi.org/10.1111/j.1365-2567.2008.02959.x.

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10

Jenkins, C., W. Garcia, M. J. Godwin, et al. "Immunomodulatory Properties of a Viral Homolog of Human Interleukin-10 Expressed by Human Cytomegalovirus during the Latent Phase of Infection." Journal of Virology 82, no. 7 (2008): 3736–50. http://dx.doi.org/10.1128/jvi.02173-07.

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ABSTRACT Human cytomegalovirus (HCMV) establishes a latent infection in hematopoietic cells, from which it can reactivate to cause significant disease in immunocompromised individuals. HCMV expresses a functional homolog of the immunosuppressive cytokine interleukin-10 (termed cmvIL-10), and alternate splicing of the cmvIL-10 transcript results in expression of a latency-associated cmvIL-10 transcript (LAcmvIL-10). To determine whether LAcmvIL-10 encodes immunosuppressive functions, recombinant LAcmvIL-10 protein was generated, and its impact on major histocompatibility complex class II (MHC-I

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