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1

Rogers, Maureen. "Montague B Lewis." Australasian Journal of Dermatology 55, no. 3 (August 2014): 229. http://dx.doi.org/10.1111/ajd.12241.

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Tannen, Terrell. "Edward B Lewis." Lancet 364, no. 9435 (August 2004): 658. http://dx.doi.org/10.1016/s0140-6736(04)16878-5.

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Galperin, Charles. "Lewis Wolpert, Denis Duboule et Edwards B. Lewis." Bulletin d’histoire et d’épistémologie des sciences de la vie Volume 23, no. 2 (2016): 199. http://dx.doi.org/10.3917/bhesv.232.0199.

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Gillanders, I. "Lewis Gillanders." BMJ 326, no. 7390 (March 22, 2003): 662b—662. http://dx.doi.org/10.1136/bmj.326.7390.662/b.

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5

Juhl, B. R. "Immunohistochemical demonstration and localization of Lewis a and Lewis b determinants in human urothelium." Journal of Histochemistry & Cytochemistry 33, no. 4 (April 1985): 309–14. http://dx.doi.org/10.1177/33.4.2579997.

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In order to obtain baseline information about Lewis antigen expression in human urothelium in order that changes during malignant transformation can be evaluated, urothelium from eight individuals of known erythrocyte Lewis types were stained by a Tween-modified indirect immunoperoxidase staining technique using goat antibodies directed toward the Lewis a and Lewis b determinants and mouse monoclonal antibodies directed toward the Lewis a determinant in serial dilutions. To evaluate the value of the method for tissue Lewis typing, eleven ureters from individuals of unknown erythrocyte Lewis ty
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6

Mayerson, Melvin. "Arthur B. Lewis (1909–1996)." American Journal of Orthodontics and Dentofacial Orthopedics 110, no. 3 (September 1996): 329. http://dx.doi.org/10.1016/s0889-5406(96)80021-7.

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7

Scott, Matthew P., and Peter A. Lawrence. "Edward B. Lewis (1918–2004)." Nature 431, no. 7005 (September 2004): 143. http://dx.doi.org/10.1038/431143a.

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8

Bender, Welcome. "Edward B. Lewis 1918–2004." Nature Genetics 36, no. 9 (September 2004): 919. http://dx.doi.org/10.1038/ng0904-919.

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9

Shashidhara, L. S. "Edward B Lewis (1918–2004)." Journal of Genetics 83, no. 2 (August 2004): 219. http://dx.doi.org/10.1007/bf02729900.

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10

Winchester, Guil. "Edward B. Lewis 1918–2004." Current Biology 14, no. 18 (September 2004): R740—R742. http://dx.doi.org/10.1016/j.cub.2004.09.007.

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11

Mishra, Rakesh K. "Edward B Lewis (1918–2004)." Journal of Biosciences 29, no. 3 (September 2004): 231–33. http://dx.doi.org/10.1007/bf02702605.

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12

BLASCHKE, T., and K. GIACOMINI. "Lewis B. Sheiner, 1940-2004." Clinical Pharmacology & Therapeutics 76, no. 6 (December 2004): 650–51. http://dx.doi.org/10.1016/j.clpt.2004.08.027.

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13

Crow, James F., and Welcome Bender. "Edward B. Lewis, 1918–2004." Genetics 168, no. 4 (December 1, 2004): 1773–83. http://dx.doi.org/10.1093/genetics/168.4.1773.

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14

Reddy, G. Naaresh, Rakesh Parida, R. Inostroza-Rivera, Arindam Chakraborty, Puru Jena, and Santanab Giri. "Unique reactivity of B in B[Ge9Y3]3 (Y = H, CH3, BO, CN): formation of a Lewis base." Physical Chemistry Chemical Physics 21, no. 42 (2019): 23301–4. http://dx.doi.org/10.1039/c9cp04361f.

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Boron compounds usually exhibit Lewis acidity at the boron center due to the presence of vacant p-orbitals. But using Zintl-ion based groups (Ge<sub>9</sub>Y<sub>3</sub>, Y = H, CH<sub>3</sub>, BO, CN), we can alter Lewis acid nature of B to a Lewis base.
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15

King, Jovanka R., Jezabel Varadé, and Lennart Hammarström. "Fucosyltransferase Gene Polymorphisms and Lewisb-Negative Status Are Frequent in Swedish Newborns, With Implications for Infectious Disease Susceptibility and Personalized Medicine." Journal of the Pediatric Infectious Diseases Society 8, no. 6 (December 9, 2018): 507–18. http://dx.doi.org/10.1093/jpids/piy085.

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Abstract Background Single-nucleotide polymorphisms (SNPs) in the fucosyltransferase genes FUT2 and FUT3 have been associated with susceptibility to various infectious and inflammatory disorders. FUT variations influence the expression of human histo-blood group antigens (HBGAs) (H-type 1 and Lewis), which are highly expressed in the gut and play an important role in microbial attachment, metabolism, colonization, and shaping of the microbiome. In particular, FUT polymorphisms confer susceptibility to specific rotavirus and norovirus genotypes, which has important global health implications. M
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16

Orntoft, TF, EH Holmes, P. Johnson, S. Hakomori, and H. Clausen. "Differential tissue expression of the Lewis blood group antigens: enzymatic, immunohistologic, and immunochemical evidence for Lewis a and b antigen expression in Le(a-b-) individuals." Blood 77, no. 6 (March 15, 1991): 1389–96. http://dx.doi.org/10.1182/blood.v77.6.1389.1389.

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Abstract The Lewis blood group system comprises two main carbohydrate antigens, Le(a) and Le(b). Lewis typing has traditionally been based on serologic determinations using erythrocytes and saliva. Several recent studies have demonstrated that erythrocyte Lewis phenotype may change during pregnancy or disease, and inappropriate Lewis antigens have been found in both normal and neoplastic tissue. To evaluate whether these observations are in conflict with the presently proposed genetic and biosynthetic basis of the Lewis blood group system, we performed a combined enzymatic, immunohistologic, a
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17

Orntoft, TF, EH Holmes, P. Johnson, S. Hakomori, and H. Clausen. "Differential tissue expression of the Lewis blood group antigens: enzymatic, immunohistologic, and immunochemical evidence for Lewis a and b antigen expression in Le(a-b-) individuals." Blood 77, no. 6 (March 15, 1991): 1389–96. http://dx.doi.org/10.1182/blood.v77.6.1389.bloodjournal7761389.

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The Lewis blood group system comprises two main carbohydrate antigens, Le(a) and Le(b). Lewis typing has traditionally been based on serologic determinations using erythrocytes and saliva. Several recent studies have demonstrated that erythrocyte Lewis phenotype may change during pregnancy or disease, and inappropriate Lewis antigens have been found in both normal and neoplastic tissue. To evaluate whether these observations are in conflict with the presently proposed genetic and biosynthetic basis of the Lewis blood group system, we performed a combined enzymatic, immunohistologic, and immuno
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18

Hong, Yun Ji, Sang Mee Hwang, Taek Soo Kim, Eun Young Song, Kyoung Un Park, Junghan Song, and Kyou-Sup Han. "Significance of Lewis Phenotyping Using Saliva and Gastric Tissue: Comparison with the Lewis Phenotype Inferred fromLewisandSecretorGenotypes." BioMed Research International 2014 (2014): 1–6. http://dx.doi.org/10.1155/2014/573652.

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Lewis phenotypes using various types of specimen were compared with the Lewis phenotype predicted fromLewisandSecretorgenotypes. This is the first logical step in explaining the association between the Lewis expression andHelicobacter pylori. We performed a study of the followings on 209 patients who underwent routine gastroscopy: erythrocyte and saliva Lewis phenotyping, gastric Lewis phenotyping by the tissue array, and theLewisandSecretorgenes genotyping. The results of phenotyping were as follows [Le(a−b−), Le(a+b−), Le(a−b+), and Le(a+b+), respectively, in order]: erythrocyte (12.4%, 25.8
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19

Dolbeare, Kenneth M., and George Q. Flynn. "Lewis B. Hershey, Mr. Selective Service." American Historical Review 91, no. 1 (February 1986): 208. http://dx.doi.org/10.2307/1867408.

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20

O'Sullivan, John, and George Q. Flynn. "Lewis B. Hershey, Mr. Selective Service." Journal of American History 72, no. 3 (December 1985): 741. http://dx.doi.org/10.2307/1904396.

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21

Lobdell, George H., and George Q. Flynn. "Lewis B. Hershey, Mr. Selective Service." Military Affairs 50, no. 4 (October 1986): 214. http://dx.doi.org/10.2307/1988018.

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22

WILLIAMS, CHARLES H. "David Byron Lewis, MD." Radiology 190, no. 3 (March 1994): 909. http://dx.doi.org/10.1148/radiology.190.3.909-b.

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23

GARCES, MIGUEL. "Elbert K. Lewis, M.D." Radiology 156, no. 3 (September 1985): 847. http://dx.doi.org/10.1148/radiology.156.3.847-b.

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24

BAKER, GERALD L. "Garner L. Lewis, MD." Radiology 169, no. 3 (December 1988): 879. http://dx.doi.org/10.1148/radiology.169.3.879-b.

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25

Yu, L. C., Y. H. Yang, R. E. Broadberry, Y. H. Chen, Y. S. Chan та M. Lin. "Correlation of a missense mutation in the human Secretorα1,2-fucosyltransferase gene with the Lewis(a+b+) phenotype: a potential molecular basis for the weak Secretor allele (Sew)". Biochemical Journal 312, № 2 (1 грудня 1995): 329–32. http://dx.doi.org/10.1042/bj3120329.

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A missense mutation (A385 to T), predicting an Ile129 to Phe substitution, in the human Secretor alpha 1,2-fucosyltransferase gene was present in double dose in Lewis(a+b+) individuals, but not in Lewis(a-b+) individuals. Co-segregation of the Lewis(a+b+) phenotype with homozygosity for the mutation was also verified. These results yield a potential molecular basis for the weak Secretor allele (Sew) accounting for the Lewis(a+b+) phenotype.
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26

Möricke, Jennifer, Birgit Wibbeling, Constantin G. Daniliuc, Gerald Kehr, and Gerhard Erker. "Design and reactions of a carbon Lewis base/boron Lewis acid frustrated Lewis pair." Philosophical Transactions of the Royal Society A: Mathematical, Physical and Engineering Sciences 375, no. 2101 (July 24, 2017): 20170015. http://dx.doi.org/10.1098/rsta.2017.0015.

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The conjugated dienamine 4 selectively adds Piers' borane [HB(C 6 F 5 ) 2 ] to give the enamine/borane system 5 , which features a boratirane structure by internal enamine carbon Lewis base to boron Lewis acid interaction. Compound 5 behaves as a C/B frustrated Lewis pair and undergoes typical addition reactions to benzaldehyde, several nitriles and to sulfur dioxide. This article is part of the themed issue ‘Frustrated Lewis pair chemistry’.
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27

Chase, Preston A., Patricio E. Romero, Warren E. Piers, Masood Parvez, and Brian O. Patrick. "Fluorinated 9-borafluorenes vs. conventional perfluoroaryl boranes — Comparative Lewis acidity." Canadian Journal of Chemistry 83, no. 12 (December 1, 2005): 2098–105. http://dx.doi.org/10.1139/v05-240.

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Perfluorinated 9-phenyl-9-borafluorene, 1, is an antiaromatic analog of the well-known tris(pentafluorophenyl)borane. Spectroscopic, structural, and electrochemical studies have been performed on 1 and its Lewis base adducts with MeCN, THF, and PMe3 with a view to assessing its comparative Lewis acid strength relative to B(C6F5)3. For the sterically undemanding Lewis base MeCN, 1 and B(C6F5)3 have comparable LA strengths, while for more sterically prominent THF, 1 is clearly the stronger Lewis acid (LA) based on competition experiments. We conclude that steric factors, rather than antiaromatic
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28

Zhao, Haiyan, Joseph H. Reibenspies, and François P. Gabbaï. "Lewis acidic behavior of B(C6Cl5)3." Dalton Trans. 42, no. 3 (2013): 608–10. http://dx.doi.org/10.1039/c2dt31482g.

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29

Ishihara, Kazuaki. "ChemInform Abstract: Chiral B(III) Lewis Acids." ChemInform 33, no. 20 (May 21, 2010): no. http://dx.doi.org/10.1002/chin.200220270.

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Ishihara, Kazuaki. "ChemInform Abstract: Achiral B(III) Lewis Acids." ChemInform 33, no. 20 (May 21, 2010): no. http://dx.doi.org/10.1002/chin.200220271.

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31

Dipta, Tashmim Farhana, Hafizur Rahman, Ayesha Khatun, and Md Ashadul Islam. "Prevalence of Lewis Blood Group among Bangladeshi Population." Anwer Khan Modern Medical College Journal 7, no. 2 (February 18, 2017): 12–15. http://dx.doi.org/10.3329/akmmcj.v7i2.31638.

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Background: The Lewis system differs from all other blood group systems. It is primarily a system of soluble antigens originate in the tissue of body secretions, such as saliva (glycoprotein) and in plasma (glycolipid) respectively.The ABH secretor status influences red cells of Lewis phenotype and this phenotype may be modified by the ABO phenotype.Methodology: A cross sectional study was done to determine the presence of Lewis antigens in patients and voluntary donor's blood and observe the agglutination pattern during blood cross matching. In this study 350 blood samples were tested by hema
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32

Körte, Leif A., Robin Warner, Yury V. Vishnevskiy, Beate Neumann, Hans-Georg Stammler, and Norbert W. Mitzel. "Intramolecular pyridine-based frustrated Lewis-pairs." Dalton Transactions 44, no. 21 (2015): 9992–10002. http://dx.doi.org/10.1039/c5dt01068c.

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Besides a series of pyridine-based Lewis pairs with B(C<sub>6</sub>F<sub>5</sub>)<sub>2</sub> groups with varying electronic and steric situations and strong intramolecular B–N bonds, 2-[(CH<sub>2</sub>)<sub>4</sub>B(C<sub>6</sub>F<sub>5</sub>)<sub>2</sub>]-6-tBu-pyridine was found to behave as a frustrated Lewis pair.
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33

Erker, Gerhard. "Frustrated Lewis pairs: Some recent developments." Pure and Applied Chemistry 84, no. 11 (August 31, 2012): 2203–17. http://dx.doi.org/10.1351/pac-con-12-04-07.

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The chemistry of some reactive frustrated Lewis pairs (FLPs) is reported. This includes intramolecular P/B and N/B FLPs, some of which were used as catalysts for the hydrogenation of electron-rich olefin substrates. Some advanced intermolecular FLPs are reported, which includes systems derived from very bulky alkenyl boranes obtained from 1,1-carboboration reactions of 1-alkynes with tris(pentafluorophenyl)borane. Some such systems activate dihydrogen and transfer the resulting proton/hydride pair even to some electron-poor alkynes. Eventually, we report on the reaction of our intramolecular e
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34

Körte, Leif A., Sebastian Blomeyer, Shari Heidemeyer, Andreas Mix, Beate Neumann, and Norbert W. Mitzel. "Intramolecular cooperativity in frustrated Lewis pairs." Chemical Communications 52, no. 64 (2016): 9949–52. http://dx.doi.org/10.1039/c6cc05228b.

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The doubly Lewis-acid functionalised aniline PhN[(CH<sub>2</sub>)<sub>3</sub>B(C<sub>6</sub>F<sub>5</sub>)<sub>2</sub>]<sub>2</sub> features two competing boron functions in fast exchange for binding to the central Lewis base. In contrast to the mono acid-functionalised PhMeN[(CH<sub>2</sub>)<sub>3</sub>B(C<sub>6</sub>F<sub>5</sub>)<sub>2</sub>], it is an active frustrated Lewis pair.
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35

Kelleher, C. "Health promotion: shades of Lewis Carroll." Journal of Epidemiology & Community Health 49, no. 1 (February 1, 1995): 1–4. http://dx.doi.org/10.1136/jech.49.1.1-b.

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36

Choi, Hong Dae, Ji Bong Jang, Pil Ja Seo, Byeng Wha Son, and Uk Lee. "2,9-Dimethyl-3,8-bis(methylsulfanyl)naphtho[2,1-b:3,4-b′]difuran." Acta Crystallographica Section E Structure Reports Online 62, no. 5 (April 11, 2006): o1785—o1786. http://dx.doi.org/10.1107/s1600536806012098.

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The title compound, C18H16O2S2, was prepared by the Lewis acid-catalysed reaction of 2,3-dihydroxynaphthalene with α-chloro-α-(methylsulfanyl)acetone. The naphtho[2,1-b:3,4-b′]difuran unit is nearly planar, with a mean deviation from the least-squares plane of 0.028 Å. In the crystal structure, aromatic π–π stacking interactions are observed between adjacent molecules.
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37

Beebee, Helen. "Counterfactual Dependence and Broken Barometers: A Response to Flichman's Argument." Crítica (México D. F. En línea) 29, no. 86 (January 8, 1997): 107–19. http://dx.doi.org/10.22201/iifs.18704905e.1997.1065.

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El artículo consiste en una defensa del análisis contrafáctico de la causación de David Lewis en contra de un argumento presentado por primera vez por Eduardo Flichman. El argumento de Flichman involucra una situación en la cual tienen lugar los tres sucesos siguientes:&#x0D; p&#x0D; &#x0D; : una presión atmosférica de 1000mb&#x0D; &#x0D; b&#x0D; &#x0D; : el funcionamiento correcto del barómetro&#x0D; &#x0D; r&#x0D; &#x0D; : una lectura en el barómetro de 1000mb&#x0D; &#x0D; &#x0D; Si el análisis de Lewis ha de tener éxito, la fórmula contrafáctica&#x0D; (1) ~O(r) □→ ~O(p)&#x0D; debe ser falsa
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Chang, Jui-Cheng, Che-Hsuan Yang, I.-Wen Sun, Wen-Yueh Ho, and Tzi-Yi Wu. "Synthesis and Properties of Magnetic Aryl-Imidazolium Ionic Liquids with Dual Brønsted/Lewis Acidity." Materials 11, no. 12 (December 13, 2018): 2539. http://dx.doi.org/10.3390/ma11122539.

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A series of unique tunable aryl-imidazolium magnetic ionic liquids (MILs) with dual acidity that contain both Brønsted and Lewis acidic sites (abbreviated as B-L MILs) were synthesized and characterized using nuclear magnetic resonance and mass spectrometry. Physical properties, such as thermal properties, magnetic susceptibility, and Brønsted and Lewis acidity, were measured. These properties were found to depend on the cation structure. These B-L MILs had good solubility in many organic solvents, good thermal stability, and low melting points, and exhibited magnet-like behavior. For these B-
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39

Ting, I.-Wen, Tze-Wah Kao, Yen-Ling Chiu, Shyh-Chyi Lo, Fu-Chang Hu, and Kwan-Dun Wu. "The Relationship of P1 and Lewis Antigens with Peritoneal Dialysis–Related Escherichia Coli Peritonitis." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 28, no. 3_suppl (June 2008): 172–78. http://dx.doi.org/10.1177/089686080802803s32.

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It has been known that the P1 and Lewis antigens on red blood cells (RBCs) affect the risk of Escherichia coli–related urinary tract infection. In the present study, we investigated the associations between those antigens and peritoneal dialysis (PD)–related peritonitis and E. coli peritonitis. We recruited 155 patients (66 men, 89 women) who were under PD treatment in July 2005, checked the P1 and Lewis antigen status of their RBCs, reviewed their medical charts, and recorded the dates and the causative pathogens of peritonitis episodes. The relationships between peritonitis and the antigens
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Gowda, Ravikumar R., та Eugene Y. X. Chen. "Chemoselective Lewis pair polymerization of renewable multivinyl-functionalized γ-butyrolactones". Philosophical Transactions of the Royal Society A: Mathematical, Physical and Engineering Sciences 375, № 2101 (24 липня 2017): 20170003. http://dx.doi.org/10.1098/rsta.2017.0003.

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Multivinyl-functionalized γ-butyrolactones, γ-vinyl-γ-methyl-α-methylene-γ-butyrolactone ( γ VMMBL) and γ-allyl-γ-methyl-α-methylene-γ-butyrolactone ( γ AMMBL), have been synthesized from biorenewable ethyl levulinate and effectively polymerized by Lewis pairs consisting of an organic N -heterocyclic carbene Lewis base and a strong organo-Lewis acid E(C 6 F 5 ) 3 (E = Al, B). This Lewis pair polymerization is quantitatively chemoselective, proceeds exclusively via polyaddition across the conjugated α-methylene double bond without participation of the γ-vinyl or γ-allyl double bond, and produce
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41

Bharath, R. Raj, and P. Arumugam. "The prevalence of secretor status and co-expression of lewis antigen in voluntary blood donors." Asian Journal of Medical Sciences 7, no. 5 (August 31, 2016): 93–96. http://dx.doi.org/10.3126/ajms.v7i5.14848.

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Background: Blood group substances are present in soluble form in a majority of individuals in secretion such as saliva and body fl uids. Secretor status refers to the presence (SeSe and Sese) or absence (sese) of secretor gene which secrete ABH soluble substances. Secretor status can be used to resolve ABO discrepancies of people whose blood group cannot be identified by routine blood grouping and it can also help in identifying patients who may be a high risk group for getting certain diseases. Aims and Objectives:Our aim and objectives of the study is to fi nd out the Prevalence of Secretor
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42

Rothenbacher, Dietrich, Maria Weyermann, Gunter Bode, Murrat Kulaksiz, Bernd Stahl, and Hermann Brenner. "Role of Lewis A and Lewis B Blood Group Antigens in Helicobacter pylori Infection." Helicobacter 9, no. 4 (August 2004): 324–29. http://dx.doi.org/10.1111/j.1083-4389.2004.00236.x.

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43

Li, Qiuxia, Chao Shi, Manli Huang, Xing Wei, Hong Yan, Chuluo Yang, and Aihua Yuan. "B- and N-embedded color-tunable phosphorescent iridium complexes and B–N Lewis adducts with intriguing structural and optical changes." Chemical Science 10, no. 11 (2019): 3257–63. http://dx.doi.org/10.1039/c8sc04252g.

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44

Hatakeyama, Masanori. "A Sour Relationship between BabA and Lewis b." Cell Host & Microbe 21, no. 3 (March 2017): 318–20. http://dx.doi.org/10.1016/j.chom.2017.02.014.

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45

Brittain, James E. "Electrical Engineering Hall of Fame: Lewis B. Stillwell." Proceedings of the IEEE 96, no. 3 (March 2008): 532–35. http://dx.doi.org/10.1109/jproc.2007.913559.

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46

Wang, Long, Shunxi Dong, Constantin G. Daniliuc, Lei Liu, Stefan Grimme, Robert Knitsch, Hellmut Eckert, Michael Ryan Hansen, Gerald Kehr, and Gerhard Erker. "Formation of macrocyclic ring systems by carbonylation of trifunctional P/B/B frustrated Lewis pairs." Chemical Science 9, no. 6 (2018): 1544–50. http://dx.doi.org/10.1039/c7sc04394e.

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The trifunctional P/B/B frustrated Lewis pairs11a–cfeaturing bulky aryl groups at phosphorus [Dmesp (a), Tipp (b), Mes* (c)] were synthesized. Compounds11a,breact with carbon monoxide and form the macrocyclic dimers17a,b, while the carbonylation reaction of the Mes*P/B/B FLP11cgives the macrocyclic trimer18c.
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47

Abdul, Fatima Rammadan. "Biofilm formation by Staphylococcus aureus isolation from atopic dermatitis patients on defined Lewis types saliva." Journal of University of Babylon 26, no. 5 (March 12, 2018): 233–40. http://dx.doi.org/10.29196/jub.v26i5.921.

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Bacteria adherence and biofilm formation vary depending on the strain of microorganism as well as the substrate.Here, we evaluated the ability of different isolates of Staphylococcus aureus (S. aureus), isolated. from atopic dermatitis patients skin and stool to form a biofilm on different Lewis types saliva, including Le (a) , (b) ,(c) and Le a+b+ saliva. S. aureus isolates were cultured on tryptose soy broth.The bacteria were used to form a biofilm on 96- well flat bottom Microtitration plate that was coated with the respective, optimally diluted, processed saliva of Lewis blood groups (a),(
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48

Abdul, Fatima Rammadan. "Biofilm formation by Staphylococcus aureus Isolation from atopic Dermatitis Patients on Defined Lewis Types saliva." JOURNAL OF UNIVERSITY OF BABYLON for Pure and Applied Sciences 26, no. 5 (March 12, 2018): 233–40. http://dx.doi.org/10.29196/jubpas.v26i5.921.

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Bacteria adherence and biofilm formation vary depending on the strain of microorganism as well as the substrate.Here, we evaluated the ability of different isolates of Staphylococcus aureus (S. aureus), isolated. from atopic dermatitis patients skin and stool to form a biofilm on different Lewis types saliva, including Le (a) , (b) ,(c) and Le a+b+ saliva. S. aureus isolates were cultured on tryptose soy broth.The bacteria were used to form a biofilm on 96- well flat bottom Microtitration plate that was coated with the respective, optimally diluted, processed saliva of Lewis blood groups (a),(
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49

Cui, Peng, Cezar C. Comanescu, and Vlad M. Iluc. "Frustrated Lewis pair-like reactions of nucleophilic palladium carbenes with B(C6F5)3." Chemical Communications 51, no. 28 (2015): 6206–9. http://dx.doi.org/10.1039/c5cc00868a.

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Nucleophilic palladium carbenes react with the strong Lewis acid B(C<sub>6</sub>F<sub>5</sub>)<sub>3</sub>via remote nucleophilic attack at the para-carbon of the supporting ligand or C–F activation of B(C<sub>6</sub>F<sub>5</sub>)<sub>3</sub>; these reactions indicate frustrated Lewis pair-like behaviour given the steric inaccessibility of the nucleophilic carbene center.
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Singh, Bishal K., Mila M. Leuthold, and Grant S. Hansman. "Human Noroviruses' Fondness for Histo-Blood Group Antigens." Journal of Virology 89, no. 4 (November 26, 2014): 2024–40. http://dx.doi.org/10.1128/jvi.02968-14.

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ABSTRACTHuman noroviruses are the dominant cause of outbreaks of gastroenteritis around the world. Human noroviruses interact with the polymorphic human histo-blood group antigens (HBGAs), and this interaction is thought to be important for infection. Indeed, synthetic HBGAs or HBGA-expressing enteric bacteria were shown to enhance norovirus infection in B cells. A number of studies have found a possible relationship between HBGA type and norovirus susceptibility. The genogroup II, genotype 4 (GII.4) noroviruses are the dominant cluster, evolve every other year, and are thought to modify their
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