Zaloguj się

Gotowe bibliografie tematyczne / Lungs Inflammation / Książki

Książki na temat „Lungs Inflammation”

Kliknij ten link, aby zobaczyć inne rodzaje publikacji na ten temat: Lungs Inflammation.

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 27 stycznia 2023

Utwórz poprawne odniesienie w stylach APA, MLA, Chicago, Harvard i wielu innych

Wybierz rodzaj źródła:

Sprawdź 50 najlepszych książek naukowych na temat „Lungs Inflammation”.

Przycisk „Dodaj do bibliografii” jest dostępny obok każdej pracy w bibliografii. Użyj go – a my automatycznie utworzymy odniesienie bibliograficzne do wybranej pracy w stylu cytowania, którego potrzebujesz: APA, MLA, Harvard, Chicago, Vancouver itp.

Możesz również pobrać pełny tekst publikacji naukowej w formacie „.pdf” i przeczytać adnotację do pracy online, jeśli odpowiednie parametry są dostępne w metadanych.

Przeglądaj książki z różnych dziedzin i twórz odpowiednie bibliografie.

1

Symposium, on Airway Obstruction and Inflammation (1988 Florence Italy). Airway obstruction and inflammation: Present status and perspectives. Basel: Karger, 1990.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

2

Criner, Gerard J., William D. Cornwell i Thomas J. Rogers. Smoking and lung inflammation: Basic, pre-clinical, and clinical research advances. New York: Springer, 2013.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

3

L, Kradin Richard, i Robinson Bruce W. S, red. Immunopathology of lung disease. Boston: Butterworth-Heinemann, 1996.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

4

Bertolini, Renzo. Animal and vegetable dusts as a cause of deep lung inflammation. Hamilton: CCOHS, 1988.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

5

Michel, Chignard, i Conference on Cytokines and Adhesion Molecules in Lung Inflammation (1995 : Paris, France), red. Cytokines and adhesion molecules in lung inflammation. New York: New York Academy of Sciences, 1996.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

6

F, Donner C., red. COPD is/is not a systemic disease? Hauppauge, NY: Nova Science, 2009.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

7

1952-, Strieter Robert M., Kunkel S. L i Standiford Theodore J, red. Chemokines in the lung. New York: Marcel Dekker, Inc., 2003.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

8

Rogers, Thomas J., Gerard J. Criner i William D. Cornwell, red. Smoking and Lung Inflammation. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7351-0.

Pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

9

Dubinett, Steven M., red. Inflammation and Lung Cancer. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2724-1.

Pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

10

Alper, Scott, i William J. Janssen, red. Lung Innate Immunity and Inflammation. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8570-8.

Pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

11

Wang, Yong-Xiao, red. Lung Inflammation in Health and Disease, Volume I. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-63046-1.

Pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

12

Wang, Yong-Xiao, red. Lung Inflammation in Health and Disease, Volume II. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68748-9.

Pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

13

Pokorski, Mieczyslaw. Pulmonary Infection and Inflammation. Springer London, Limited, 2016.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

14

Pokorski, Mieczyslaw. Pulmonary Infection and Inflammation. Springer, 2018.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

15

Pokorski, Mieczyslaw. Pulmonary Infection and Inflammation. Springer, 2016.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

16

Dubinett, Steven M. Inflammation and Lung Cancer. Springer, 2015.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

17

Dubinett, Steven M. Inflammation and Lung Cancer. Springer, 2016.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

18

Dubinett, Steven M. Inflammation and Lung Cancer. Springer, 2015.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

19

Rogers, Thomas J., Gerard J. Criner i William D. Cornwell. Smoking and Lung Inflammation: Basic, Pre-Clinical and Clinical Research Advances. Springer, 2016.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

20

Martin, Thomas. Cytokines in Pulmonary Disease: Infection and Inflammation (Lung Biology in Health & Disease). Informa Healthcare, 2000.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

21

Strieter, Robert M., Steven L. Kunkel i Theodore J. Standiford. Chemokines in the Lung. Taylor & Francis Group, 2003.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

22

Michel, Chignard, red. Cells and cytokines in lung inflammation. New York, NY: New York Academy of Sciences, 1994.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

23

(Editor), Duncan F. Rogers, i Louise E. Donnelly (Editor), red. Human Airway Inflammation: Sampling Techniques and Analytical Protocols (Methods in Molecular Medicine). Humana Press, 2001.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

24

(Editor), Trevor T. Hansel, i Peter J. Barnes (Editor), red. Recent Advances in the Pathophysiology of COPD (Progress in Inflammation Research). Birkhäuser Basel, 2004.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

25

(Editor), Johan Zaagsma, Herman Meurs (Editor) i Ad F. Roffel (Editor), red. Muscarinic Receptors in Airways Diseases (Progress in Inflammation Research). Birkhauser, 2001.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

26

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

27

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

28

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

29

New perspectives in monitoring lung inflammation: Analysis of exhaled breath condensate. Boca Raton, FL: CRC Press, 2006.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

30

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

31

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

32

Acute Lung Injury: From Inflammation to Repair (Biomedical and Health Research). Ios Pr Inc, 2001.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

33

A, Persson C. G., red. Inflammatory indices in chronic bronchitis. Basel: Birkhäuser, 1990.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

34

1942-, Church Martin, i Robinson Clive 1958-, red. Eicosanoids in inflammatory conditions of the lung, skin, and joints. Lancaster: MTP Press, 1988.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

35

Ray, Abhijit, i Punit Kumar Srivastave. Obstructive Airway Diseases. Taylor & Francis Group, 2011.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

36

Ray, Abhijit, i Punit Kumar Srivastava. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2016.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

37

Ray, Abhijit. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2011.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

38

Ray, Abhijit, i Punit Kumar Srivastava. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2016.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

39

Ray, Abhijit, i Punit Kumar Srivastava. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2018.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

40

Ong, Kian Chung, red. Lung Inflammation. InTech, 2014. http://dx.doi.org/10.5772/57068.

Pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

41

Adlam, David. Pericardial disease. Redaktorzy Patrick Davey i David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0109.

Pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

Streszczenie:

The pericardium forms a continuous sac around the heart, analogous to the pleura surrounding the lungs, and the peritoneum surrounding the abdominal viscera. Between the parietal and visceral layers of the serous pericardium is the pericardial space, which normally contains a small volume of pericardial fluid. The clinical spectrum of pericardial diseases can be divided into: pericarditis, caused by acute inflammation; pericardial effusion, or fluid accumulation in the pericardial space, leading to tamponade; and constrictive pericarditis, caused by chronic infiltration or inflammation leading to pericardial constriction.

42

Wick, Mark R., i Henry D. Tazelaar. Neoplastic Mimics in Thoracic and Cardiovascular Pathology. Springer Publishing Company, Incorporated, 2013.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

43

Wick, Mark R., i Henry D. Tazelaar. Neoplastic Mimics in Thoracic and Cardiovascular Pathology. Springer Publishing Company, Incorporated, 2013.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

44

Wiffen, Philip, Marc Mitchell, Melanie Snelling i Nicola Stoner. Therapy-related issues: musculoskeletal diseases. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199603640.003.0025.

Pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

Streszczenie:

Rheumatoid arthritis 548Gout 556Rheumatoid arthritis (RA) is an autoimmune disease which causes joints lined with synovium to become inflamed, swollen, stiff, and painful, and leads to joint erosion. It is a multisystem disorder which can affect many organs including the eyes, lungs, heart, and blood vessels. The aim of treatment is to decrease pain and inflammation, prevent joint damage, and ultimately induce remission of disease....

45

Blend, Doctor Herry. Ketoconazole: Perfect Guide for Most Effective Antifungal Treatment That Completely Cures Jock Itch, Athlete's Foot, Ringworm, Painful Inflammation, Lungs or Skin Diseases and Other Fungal Diseases. Lulu Press, Inc., 2018.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

46

Klepikov, Igor. Didactics of Acute Lung Inflammation. Cambridge Scholars Publishing, 2022.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

47

Kriemler, Susi, Thomas Radtke i Helge Hebestreit. Exercise, physical activity, and cystic fibrosis. Redaktorzy Neil Armstrong i Willem van Mechelen. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757672.003.0027.

Pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

Streszczenie:

Cystic fibrosis (CF) is a genetic disease resulting in an impaired mucociliary clearance, chronic bacterial airway infection, and inflammation. The progressive destruction of the lungs is the main cause of morbidity and premature death. Diverse other organ systems such as heart, muscles, bones, gastro-intestinal tract, and sweat glands are often also affected and interfere with exercise capacity. Hence, exercise capacity is reduced as the disease progresses mainly due to reduced functioning of the muscles, heart, and/or lungs. Although there is still growing evidence of positive effects of exercise training in CF on exercise capacity, decline of pulmonary function, and health-related quality of life, the observed effects are encouraging and exercise should be implemented in all patient care. More research is needed to understand pathophysiological mechanisms of exercise limitations and to find optimal exercise modalities to slow down disease progression, predict long-term adherence, and improve health-related quality of life.

48

Balhara, Kamna S., Basem F. Khishfe i Jamil D. Bayram. Sepsis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0004.

Pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

Streszczenie:

Sepsis is a clinical syndrome characterized by systemic inflammation in the presence of infection. The source of infection may be occult. One must be aware of the epidemiology, presenting features and complications, diagnostic considerations and tests, and the organisms involved. Bacteria (gram positive and negative) are most commonly associated with sepsis, although fungi, viruses, and parasites can cause sepsis. Infections in the lungs, urinary tract, abdomen, skin, brain, and other areas can cause bacteremia and lead to sepsis. Treatment includes airway, breathing, and circulation (ABCs) management; aggressive fluid resuscitation; early administration of broad-spectrum antibiotics; and early goal-directed therapy and severe sepsis resuscitation bundle. Diagnosis can be challenging in pediatric and geriatric populations.

49

Lynn, William S. Inflammatory Cells & Lung Disease. Taylor & Francis Group, 2020.

Znajdź pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

50

Kriemler, Susi. Exercise, physical activity, and cystic fibrosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199232482.003.0033.

Pełny tekst źródła

Style APA, Harvard, Vancouver, ISO itp.

Streszczenie:

Cystic fibrosis (CF) is the most common genetic autosomal recessive disease of the Caucasian race, generally leading to death in early adulthood.1 The frequency of the gene carrier (heterozygote) is 1:20–25 in Caucasian populations, 1:2000 in African-Americans, and practically non-existent in Asian populations. The disease occurs in about 1 in every 2500 life births of the white population. Mean survival has risen from 8.4 years in 1969 to 32 years in 2000 due to improvements in treatment. The genetic defect causes a pathological electrolyte transport through the cell membranes by a defective chloride channel membrane transport protein [cystic fibrosis transmembrane conductance regulator (CFTR)]. With respect to the function, this affects mainly the exocrine glands of secretory cells, sinuses, lungs, pancreas, liver, and the reproductive tract of the human body leading to a highly viscous, water-depleted secretion. The secretion cannot leave the glands and in consequence causes local inflammation and destruction of various organs. The main symptoms include chronic inflammatory pulmonary disease with a progressive loss of lung function, exocrine and sometimes endocrine pancreas insufficiency, and an excessive salt loss through the sweat glands.1 A summary of the signs and symptoms of CF will be given with a special emphasis on the effect of exercise performance and capacity.