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Książki na temat „Lymphocyte therapy”

Autor: Grafiati
Data publikacji: 2 czerwca 2025
Data aktualizacji: 31 lipca 2025

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1

Raut, Mohan, and Mugdha Raut. Lymphocyte Immunization Therapy (LIT) in Reproductive Failures. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-2960-1.

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2

Alexander, Michael A. Immune-based cancer treatment: The T lymphocyte response. CRC Press, 2011.

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3

Jean-Marie, Andrieu, Lu Wei, and International Symposium on Cellular Approaches to the Control of HIV Disease (1st : 1994 : Paris, France), eds. Cell activation and apoptosis in HIV infection: Implications for pathogenesis and therapy. Plenum Press, 1995.

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4

Alexander, Michael A. Immune-based cancer treatment: The T lymphocyte response. CRC Press, 2011.

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5

1933-, Koldovksy P., ed. Lymphocytes in immunotherapy of cancer. Springer-Verlag, 1989.

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6

Hancock, Sarah. Fludarabine as first line therapy for chronic lymphocytic leukaemia. University of Birmingham, Department of Public Health and Epidemiology, 2003.

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7

Chadi, Nabhan, ed. Chronic lymphocytic leukemia research focus. Nova Science Publishers, 2006.

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8

1956-, Zhang Jingwu, and Cohen Irun R, eds. T-cell vaccination. Nova Biomedical Books, 2008.

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9

I, Gabrilovich Dmitry, and Hurwitz Arthur A, eds. Tumor-induced immune suppression: Mechanisms and therapeutic reversal. Springer, 2008.

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10

C, Hyde, National Co-ordinating Centre for HTA (Great Britain), and Health Technology Assessment Programme, eds. Fludarabine as second-line therapy for B cell chronic lymphocytic leukaemia: A technology assessment. Core Research on behalf of the NCCHTA, 2002.

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11

Giorgio, Parmiani, and Lotze Michael T, eds. Tumor immunology: Molecularly defined antigens and clinical applications. Taylor & Francis, 2002.

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12

Bahiru, Gametchu, ed. Glucocorticoid receptor structure and leukemic cell responses. Springer-Verlag, 1995.

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13

Burkhard, Ludewig, and Hoffmann Matthias W, eds. Adoptive immunotherapy: Methods and protocols. Humana Press, 2005.

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14

1944-, Finke James H., and Bukowski Ronald M, eds. Cancer immunotherapy at the crossroads: How tumors evade immunity and what can be done. Humana Press, 2004.

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15

1944-, Finke James H., and Bukowski Ronald M, eds. Cancer immunotherapy at the crossroads: How tumors evade immunity and what can be done. Humana Press, 2004.

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16

J, McMichael Andrew, Bodmer W. F. 1936-, and Imperial Cancer Research Fund (Great Britain), eds. A New look at tumour immunology. Cold Spring Harbor Laboratory Press, 1992.

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17

Gupta, Sudhir. Mechanisms of Lymphocyte Activation and Immune Regulation VIII: Autoimmunity 2000 and Beyond. Springer London, Limited, 2012.

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18

Gupta, Sudhir. Mechanisms of Lymphocyte Activation and Immune Regulation VIII: Autoimmunity 2000 and Beyond. Springer, 2012.

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19

Raut, Mohan, and Mugdha Raut. Lymphocyte Immunization Therapy in Reproductive Failures: New Horizons. Springer, 2022.

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20

Lymphocyte Immunization Therapy in Reproductive Failures: New Horizons. Springer, 2023.

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21

Jolles, G., ed. T-lymphocyte And Inflammatory Cell Research In Asthma. Academic Press, 1993.

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22

Taylor, Jack H., G. Jolles, and J. A. Karlsson. T-Lymphocyte and Inflammatory Cell Research in Asthma. Elsevier Science & Technology Books, 2012.

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23

T-Lymphocyte and Inflammatory Cell Research in Asthma. Academic Press, 1993.

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24

Andrieu, Jean-Marie, and Wei Lu. Cell Activation and Apoptosis in HIV Infection: Implications for Pathogenesis and Therapy. Springer London, Limited, 2012.

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25

Immunotherapy of cancer with sensitized T lymphocytes. R.G. Landes, 1994.

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26

Hjalgrim, Henrik, Ellen T. Chang, and Sally L. Glaser. Hodgkin Lymphoma. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0039.

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Hodgkin lymphoma (HL) is a malignant neoplasm of the lymphatic system. The malignant cell clone derives from germinal center B lymphocytes in ~98% of cases, the rest being of T-lymphocyte origin. Each year, HL is diagnosed in roughly 66,000 individuals worldwide. HL is curable with modern therapy in the vast majority of patients, with five-year survival rates exceeding 90% for early-stage disease. However, so far this excellent prognosis has been achieved at the expense of a high incidence of severe long-term treatment complications such as secondary malignancies, and endocrine and cardiovascu
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27

Davidson, Ben, Pinar Firat, and Claire W. Michael. Serous Effusions: Etiology, Diagnosis, Prognosis and Therapy. Springer, 2019.

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28

Davidson, Ben, Claire W. Michael, and Pİnar Fİrat. Serous Effusions: Etiology, Diagnosis, Prognosis and Therapy. Springer, 2016.

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Fİrat, Pİnar, Ben Davidson, and Claire W. Michael. Serous Effusions: Etiology, Diagnosis, Prognosis and Therapy. Springer London, Limited, 2011.

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30

Davidson, Ben, Pinar Firat, and Claire W. Michael. Serous Effusions: Etiology, Diagnosis, Prognosis and Therapy. Springer, 2018.

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31

Hull, Mark, and Steven C. Reynolds. HIV in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0291.

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It has been over 30 years since the recognition of the acquired immune deficiency syndrome (AIDS), linked to infection with human immunodeficiency virus (HIV). Opportunistic infections arise in the setting of decreases in the CD4+ T-lymphocyte count. Advances in the safety, and effectiveness of combination antiretroviral therapy (cART) have led to substantial improvements in life-expectancy for individuals accessing successful therapy. As such individuals are likely to be admitted to the intensive care unit (ICU) for conditions un-related to HIV, although presentations due to opportunistic inf
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32

(Editor), Susan O'Brien, and John G. Gribben (Editor), eds. Chronic Lymphocytic Leukemia. Informa Healthcare, 2008.

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33

Henggeller, Michelle. Infections in the HIV Patient. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0055.

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The hallmark of the human immunodeficiency virus (HIV) patient with a cluster of differentiation 4 (CD4) T lymphocyte count below 200 is the development of opportunistic infections. Although the use of antiretroviral therapy (ART) has decreased the incidence of these infections, they continue to be a major case of morbidity and mortality in the patient with HIV. These infections can be respiratory in nature and present with cough or shortness of breath: Pneumocystis pneumonia (PCP), tuberculosis (TB), aspergillosis, and coccidioidomycosis. Neurological infections, which can present with change
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34

Misbah, Siraj. Immunosuppressive therapy and therapeutic monoclonal antibodies. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0302.

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The term immunosuppressive therapy encompasses all forms of treatment that dampens function of the recipient’s immune system, with a view to controlling severe autoimmune, inflammatory, or allergic disease. The predominant targets of these agents are T-lymphocytes with multiple downstream effects, including containment of T-cell activation, inhibition of cytokine production, restriction of clonal expansion, and varying degrees of suppression of B-cell function. This chapter reviews the clinical use of monoclonal antibodies and other immunosuppressive agents, and their mechanisms of action.
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35

(Editor), Doriano Fabbro, and Frank McCormick (Editor), eds. Protein Tyrosine Kinases: From Inhibitors to Useful Drugs (Cancer Drug Discovery and Development). Humana Press, 2005.

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36

McCormick, Frank. Protein Tyrosine Kinases: From Inhibitors to Useful Drugs. Humana Press, 2005.

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McCormick, Frank, and Doriano Fabbro. Protein Tyrosine Kinases: From Inhibitors to Useful Drugs. Humana Press, 2010.

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38

Campath - 1 H: Emerging Frontline Therapy in Chronic Lymphocytic Leukemia. Informa Healthcare, 2001.

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39

Nabhan, Chadi. Chronic Lymphocytic Leukemia Research Focus. Nova Science Pub Inc, 2007.

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40

Hawkins, Robert E. Cellular Therapy of Cancer: Development of Gene Therapy Based Approaches. World Scientific Publishing Co Pte Ltd, 2014.

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Cellular Therapy Of Cancer. World Scientific Publishing Company, 2010.

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42

OLDSTONE. Cytotoxic T-Lymphocytes in Human Viral & Malaria Infections. Springer, 1994.

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43

Vigil, Karen J. Non-opportunistic Infections. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0036.

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Lymphocytic interstitial pneumonitis is a common respiratory complication of HIV infection in children but a rare complication in HIV-infected adults. The diagnosis requires histologic confirmation by biopsy. Antiretroviral therapy has been used with success for treatment. Nonspecific interstitial pneumonitis (NSIP) presents with dyspnea, nonproductive cough, and fever in patients with CD4+ T cell counts >200 cells/mm3. The diagnosis of NSIP requires histologic confirmation by biopsy. The optimum treatment remains unclear. The prevalence of pulmonary arterial hypertension (PAH) is higher in
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44

Gabrilovich, Dmitry I., and Arthur Andrew Hurwitz. Tumor-Induced Immune Suppression: Mechanisms and Therapeutic Reversal. Springer, 2016.

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45

Gabrilovich, Dmitry I., and Arthur Andrew Hurwitz. Tumor-Induced Immune Suppression: Mechanisms and Therapeutic Reversal. Springer, 2010.

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46

Gabrilovich, Dmitry I., and Arthur Andrew Hurwitz. Tumor-Induced Immune Suppression: Mechanisms and Therapeutic Reversal. Springer London, Limited, 2014.

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47

Gabrilovich, Dmitry I., and Arthur Andrew Hurwitz. Tumor-Induced Immune Suppression: Mechanisms and Therapeutic Reversal. Springer, 2014.

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48

Bauer, Jan, and Christian G. Bien. Rasmussen Encephalitis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0096.

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Rasmussen encephalitis (RE) is a rare epileptic disorder that is characterized by the presence of unihemispheric seizures coinciding with inflammation. The disease mostly presents in children. Clinically, patients often reach a residual stage with drug resistant seizures and severe neurological deficits. Pathologically, at this stage, the brain shows variable neuronal loss. The etiology is unknown but the infiltration of large numbers of CD8 positive T lymphocytes suggests that this is an autoimmune disease. Treatment consists of anti-inflammatory therapy (IVIG or tacrolimus), which, however,
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49

Hasbun, Rodrigo, Richard Dunham, Joseph S. Kass, et al. HIV-Associated Neurocognitive Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0038.

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HIV causes a chronic form of encephalitis (HIVE) that is clinically characterized by either dementia or mild neurocognitive impairment. Since the introduction of antiretroviral therapy in 1996, the incidence of HIV dementia has decreased by 50%, but the prevalence of mild neurocognitive disorder has increased up to 39%. HIVE is the result of direct microglial infection, interruption of trophic factors, or caused by inflammatory cytokines. HIV enters the brain primarily by the “Trojan horse mechanism”; it is carried by monocytes and lymphocytes that cross the blood–brain barrier. HIV has a pred
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50

Farghaly, Samir A. Adoptive Cell Immunotherapy for Epithelial Ovarian Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0005.

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The standard management for epithelial ovarian cancer (EOC) is a combination of aggressive debulking surgery with residual tumor of less than 1 cm and platinum-based chemotherapy. However, a high percentage of patients experience disease recurrence. Extensive efforts to find new therapeutic options have been made, albeit cancer cells develop drug resistance and malignant progression occurs. Novel therapeutic strategies are needed to enhance progression-free survival and overall survival of patients with advanced EOC. Several preclinical and clinical studies investigated feasibility and efficac
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