Gotowe bibliografie tematyczne / Macrophage activation

Spis treści

  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
  6. Raporty organizacyjne

Gotowa bibliografia na temat „Macrophage activation”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 25 lipca 2025

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Artykuły w czasopismach na temat "Macrophage activation"

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Sharma, Preeti, Shailza Shreshtha, Pradeep Kumar, Rachna Sharma, and T. K. Mahapatra. "A Review on Macrophage Activation Syndrome." Journal of Pure and Applied Microbiology 13, no. 1 (2019): 183–91. http://dx.doi.org/10.22207/jpam.13.1.19.

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Gilbreath, M. J., C. A. Nacy, D. L. Hoover, C. R. Alving, G. M. Swartz, and M. S. Meltzer. "Macrophage activation for microbicidal activity against Leishmania major: inhibition of lymphokine activation by phosphatidylcholine-phosphatidylserine liposomes." Journal of Immunology 134, no. 5 (1985): 3420–25. http://dx.doi.org/10.4049/jimmunol.134.5.3420.

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Abstract Resident peritoneal macrophages from untreated mice develop microbicidal activity against amastigotes of the protozoan parasite Leishmania tropica (current nomenclature = Leishmania major) after in vitro exposure to LK from antigen-stimulated leukocyte culture fluids. This LK-induced macrophage microbicidal activity was completely abrogated by addition of 7:3 phosphatidylcholine: phosphatidylserine liposomes. Liposome inhibition was not due to direct toxic effects against the parasite or macrophage effector cell; factors in LK that induce macrophage microbicidal activity were not adso
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Van Epps, Heather L. "Macrophage activation unveiled." Journal of Experimental Medicine 202, no. 7 (2005): 884. http://dx.doi.org/10.1084/jem.2027fta.

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In the early 1960s, George Mackaness showed that macrophages from mice infected with intracellular bacteria could launch an indiscriminate attack against unrelated bacteria. Thus began an explosion of research on the biology of what Mackaness first termed “macrophage activation.”
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Heidenreich, S., M. Weyers, J. H. Gong, H. Sprenger, M. Nain, and D. Gemsa. "Potentiation of lymphokine-induced macrophage activation by tumor necrosis factor-alpha." Journal of Immunology 140, no. 5 (1988): 1511–18. http://dx.doi.org/10.4049/jimmunol.140.5.1511.

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Abstract In this study, we examined the possible role of TNF-alpha and lymphotoxin (TNF-beta) as cofactors of macrophage activation. The results demonstrate that both TNF were capable of enhancing the cytostatic and cytolytic activity of murine peritoneal macrophages against Eb lymphoma cells. The potentiation of tumor cytotoxicity became apparent when macrophages from DBA/2 mice were suboptimally activated by either a T cell clone-derived macrophage-activating factor or by IFN-gamma plus LPS. Neither TNF-alpha nor TNF-beta could induce tumor cytotoxicity in IFN-gamma-primed macrophages, indic
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Zhao, Yu, Yuteng Jiang, Fengmei Wang, et al. "High glucose promotes macrophage switching to the M1 phenotype via the downregulation of STAT-3 mediated autophagy." PLOS ONE 19, no. 12 (2024): e0314974. https://doi.org/10.1371/journal.pone.0314974.

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Aim Imbalanced M1/M2 macrophage phenotype activation is a key point in diabetic kidney disease (DKD). Macrophages mainly exhibit the M1 phenotype, which contributes to inflammation and fibrosis in DKD. Studies have indicated that autophagy plays an important role in M1/M2 activation. However, the mechanism by which autophagy regulates the macrophage M1/M2 phenotype in DKD is unknown. Thus, the aim of the present study was to explore whether high glucose-induced macrophages switch to the M1 phenotype via the downregulation of STAT-3-mediated autophagy. Methods DKD model rats were established in
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McGee, MP, R. Wallin, FB Wheeler, and H. Rothberger. "Initiation of the extrinsic pathway of coagulation by human and rabbit alveolar macrophages: a kinetic study." Blood 74, no. 5 (1989): 1583–90. http://dx.doi.org/10.1182/blood.v74.5.1583.1583.

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Abstract We examined assembly and expression of the factor X activating complex on human and rabbit alveolar macrophages. Kinetic parameters of the factor X activating reaction were determined by functional titrations of factors VII and X with macrophage tissue factor (TF) added. We found rapid activation of factor X to Xa on alveolar macrophage surfaces. Detection of rapid factor Xa formation on macrophages required addition of exogenous factors VII and X. At plasma concentrations of the purified factors, factor Xa was formed on freshly isolated macrophages at approximately 5.4 pmol/min/10(6)
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McGee, MP, R. Wallin, FB Wheeler, and H. Rothberger. "Initiation of the extrinsic pathway of coagulation by human and rabbit alveolar macrophages: a kinetic study." Blood 74, no. 5 (1989): 1583–90. http://dx.doi.org/10.1182/blood.v74.5.1583.bloodjournal7451583.

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We examined assembly and expression of the factor X activating complex on human and rabbit alveolar macrophages. Kinetic parameters of the factor X activating reaction were determined by functional titrations of factors VII and X with macrophage tissue factor (TF) added. We found rapid activation of factor X to Xa on alveolar macrophage surfaces. Detection of rapid factor Xa formation on macrophages required addition of exogenous factors VII and X. At plasma concentrations of the purified factors, factor Xa was formed on freshly isolated macrophages at approximately 5.4 pmol/min/10(6) cells. A
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Lewis, Brandon W., Sonika Patial, and Yogesh Saini. "In Vitro Screening Method for Characterization of Macrophage Activation Responses." Methods and Protocols 5, no. 5 (2022): 68. http://dx.doi.org/10.3390/mps5050068.

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Macrophage activation refers to the enhanced functionality of macrophages in response to endogenous or exogenous stimuli. Due to the existence of limitless stimuli and a multitude of receptors on macrophage surfaces, the nature of activation (or acquired functioning) can be specific to the encountering stimulus. This article describes a macrophage-activation screening platform in a 96-well format. The methodology involves the generation of bone marrow-derived macrophages, their activation into two extreme activation states, and screening of activated macrophages for expression of bonafide prot
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Rios, Francisco J., Marianna M. Koga, Mateus Pecenin, Matheus Ferracini, Magnus Gidlund, and S. Jancar. "Oxidized LDL Induces Alternative Macrophage Phenotype through Activation of CD36 and PAFR." Mediators of Inflammation 2013 (2013): 1–8. http://dx.doi.org/10.1155/2013/198193.

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OxLDL is recognized by macrophage scavenger receptors, including CD36; we have recently found that Platelet-Activating Factor Receptor (PAFR) is also involved. Since PAFR in macrophages is associated with suppressor function, we examined the effect of oxLDL on macrophage phenotype. It was found that the presence of oxLDL during macrophage differentiation induced high mRNA levels to IL-10, mannose receptor, PPARγand arginase-1 and low levels of IL-12 and iNOS. When human THP-1 macrophages were pre-treated with oxLDL then stimulated with LPS, the production of IL-10 and TGF-βsignificantly increa
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Valledor, Annabel F., Luís Arpa, Ester Sánchez-Tilló та ін. "IFN-γ–mediated inhibition of MAPK phosphatase expression results in prolonged MAPK activity in response to M-CSF and inhibition of proliferation". Blood 112, № 8 (2008): 3274–82. http://dx.doi.org/10.1182/blood-2007-11-123604.

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Abstract Macrophages have the capacity to proliferate in response to specific growth factors, such as macrophage-colony stimulating factor (M-CSF). In the presence of several cytokines and activating factors, macrophages undergo growth arrest, become activated, and participate in the development of an immune response. We have previously observed that activation of extracellularly regulated kinase 1/2 (ERK-1/2) is required for macrophage proliferation in response to growth factors. A short and early pattern of ERK activity correlated with the proliferative response. In contrast, slightly prolon
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Rozprawy doktorskie na temat "Macrophage activation"

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Svensson, Ulf. "Macrophage activation by bacteria signalling to prostaglandin and cytokine responses /." Lund : Dept. of Medical & Physiological Chemistry, Lund University, 1994. http://books.google.com/books?id=sAhrAAAAMAAJ.

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Tabata, Yasuhiko. "Macrophage phagocytosis of polymer microspheres and antitumor activation of macrophages." Kyoto University, 1987. http://hdl.handle.net/2433/74704.

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Pound, J. D. "Parameters of human macrophage activation." Thesis, University of Nottingham, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.381442.

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Cano, Antonella. "Characterization of Acanthamoeba macrophage activation." Thesis, University of Strathclyde, 2015. http://oleg.lib.strath.ac.uk:80/R/?func=dbin-jump-full&object_id=25992.

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Hunter, Catriona Mhairi. "MicroRNA regulation of macrophage activation." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/31027.

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Macrophages are mononuclear phagocytic cells that have diverse roles within the body. Tissue specific macrophages, e.g. Kupffer cells, microglia and osteoclasts, have roles in tissue homeostasis, while circulating macrophages play an important role in the innate immune system. Macrophages detect the presence of pathogen associated molecular patterns (PAMPs) via a range of receptors known collectively as pathogen recognition receptors (PRRs). Detection of pathogens causes the macrophages to become ‘activated,’ during which the macrophages undergo extreme morphological and translational changes
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Ghanipour, Ali. "IL-10 regulates macrophage activation through activation of SHIP." Thesis, University of British Columbia, 2007. http://hdl.handle.net/2429/30873.

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IL-10 is a potent anti-inflammatory and immunosuppressive cytokine, which regulates macrophages by activation of the STAT3 pathway. However, several lines of evidence suggest that IL-10 can inhibit macrophage activation independent of STAT3 through currently unknown mechanisms and pathways. Here for the first time, we show that in murine macrophages, IL-10 activates Src Homology 2 Domain-containing Inositol 5'-Phosphatase (SHIP), a molecule with reported anti-inflammatory effects. Activation of SHIP by IL-10 is required for inhibition of Tumor necrosis factor alpha (TNFα) in macrophages
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Wu, Wei-Kang. "Modulating angiogenesis via altering macrophage activation." Thesis, University of Bristol, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539772.

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DUARTE, Madalena Bento. "Macrophage direct activation by Leishmania Spp." Master's thesis, Instituto de Higiene e Medicina Tropical, 2019. http://hdl.handle.net/10362/97877.

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Leishmanioses é um grupo de doenças causadas por parasitas do género Leishmania que apresenta elevada morbidade e de mortalidade. As manifestações clínicas dependem de interações complexas que se estabelecem entre Leishmania spp. e a resposta imunitária do hospedeiro. A doença pode manifestrar-se de maneiras diferentes, desde uma simples úlcera cutânea até ao envolvimento dos órgãos profundos que pode originar doença visceral fatal ou kala-azar. A resistência a drogas terapêuticas é um problema muito evidente em países endémicos, existindo uma crescente procura por alternativas eficazes. A pes
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Sobhani, Kimia. "Proteomic analysis of macrophage proinflammatory programmed cell death and macrophage activation /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/8688.

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Graham, Susan. "Studies on the activation of rainbow trout (Salmo gairdneri) macrophages and the characterization of a macrophage activating factor." Thesis, University of Aberdeen, 1989. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU498113.

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Rainbow trout macrophages were stimulated with PMA to produce 02- and H2O2 as detected by the reduction of nitroblue tetrazolium (NBT) and the oxidation of phenol red respectively. Addition of DDC or nitroprusside, inhibitors of superoxide dismutase (SOD) increased O2-levels and decreased H2O2 levels, whereas addition of exogenous SOD had the reverse effect. Such data are indicative of a respiratory burst pathway in teleost macrophages comparable with that of mammals. Respiratory burst activity, acid phosphatase activity and RNA synthesis in rainbow trout macrophages which have been stimulated
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Książki na temat "Macrophage activation"

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Russell, Stephen W., and Siamon Gordon, eds. Macrophage Biology and Activation. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77377-8.

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W, Russell Stephen, and Gordon Siamon, eds. Macrophage biology and activation. Springer-Verlag, 1992.

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1955-, Suttles Jill, ed. T-cell signaling of macrophage activation: Cell contact-dependent and cytokine signals. R.G. Landes, 1995.

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Pulkki, Kari. Activation of synovial fibroblasts by macrophage-derived factors: Characterization of arthritis-associated and cytokine-induced biochemical and morphological changes in cultured synovial fibroblasts. K. Pulkki, 1988.

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Kucey, Daryl Stanton. Modulation of macrophage procoagulant activity by platelet activating factor. National Library of Canada = Bibliothèque nationale du Canada, 1992.

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Adams, Dolph O. Macrophage Activation. Springer, 2013.

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Adams, Dolph O. Macrophage Activation. Springer, 2013.

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Grom, Alexei A., and Athimalaipet V. Ramanan. Macrophage activation syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0168.

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Macrophage activation syndrome (MAS) is a life-threatening condition caused by excessive activation and proliferation of T lymphocytes and haemophagocytic macrophages. Although MAS has been reported in association with almost any rheumatic disease, it is by far most common in systemic juvenile idiopathic arthritis. Flares of the underlying disease or infection are most common triggers of MAS. The pathognomonic feature of MAS is typically found in bone marrow: numerous, well-differentiated macrophagic histiocytes phagocytosing normal haematopoietic elements. The expansion of these histiocytes l
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The human macrophage system: Activity and functional morphology. Karger, 1988.

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Gordon, Siamon, and Stephen W. Russell. Macrophage Biology and Activation. Springer London, Limited, 2011.

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Części książek na temat "Macrophage activation"

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Lewis, J. G. "Macrophage Activation." In Encyclopedia of Immunotoxicology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-54596-2_936.

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Iweala, Onyinye, and Eveline Y. Wu. "Macrophage Activation Syndrome." In Rare Rheumatic Diseases of Immunologic Dysregulation. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-99139-9_1.

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Davì, Sergio, Francesca Minoia, Randy Q. Cron, and Angelo Ravelli. "Macrophage Activation Syndrome." In Pediatric Rheumatology. Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-1750-6_22.

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Eloseily, Esraa M., and Randy Q. Cron. "Macrophage Activation Syndrome." In The Microbiome in Rheumatic Diseases and Infection. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-79026-8_14.

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Vinh, Donald C., and Steven M. Holland. "Macrophage Classical Activation." In Phagocyte-Pathogen Interactions. ASM Press, 2014. http://dx.doi.org/10.1128/9781555816650.ch19.

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Comalada, Mònica, Andree Yeramian, Manuel Modolell, Jorge Lloberas, and Antonio Celada. "Arginine and Macrophage Activation." In Methods in Molecular Biology. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-527-5_16.

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Mehta, Bella, and Petros Efthimiou. "Macrophage Activation Syndrome (MAS)." In Auto-Inflammatory Syndromes. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-96929-9_14.

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Classen, Andrea, Jorge Lloberas, and Antonio Celada. "Macrophage Activation: Classical Vs. Alternative." In Macrophages and Dendritic Cells. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-396-7_3.

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Coates, Anthony R. M., Ana Cehovin, and Yanmin Hu. "Chaperonin 60 and Macrophage Activation." In Novartis Foundation Symposia. John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/9780470754030.ch12.

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Langermans, J. A. M., P. H. Nibbering, M. E. B. Van Der Hulst, and R. Van Furth. "Macrophage activation by recombinant cytokines." In Mononuclear Phagocytes. Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-015-8070-0_80.

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Streszczenia konferencji na temat "Macrophage activation"

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McGee, Maria, and Henry Rothberger. "MECHANISMS OF PROCOAGULANT GENERATION BY ALVEOLAR MACROPHAGES DURING MATURATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643168.

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During maturation in vivo and in vitro alveolar macrophages generate procoagulant(s) capable of activating the extrinsic pathway. It is generally agreed that at least part of the activity is due to TF (tissue factor). However, whether or not macrophages also generate functional factor VII or X is controversial. To characterize procoagulant activity increases, we measured kinetic parameters defining interactions between components of the TF-VII complex on membranes of alveolar macrophages either freshly isolated or cultured in serum free medium. In incubation mixtures with fixed concentrations
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"02 Macrophage activation syndrome in SLE." In 8th ANNUAL MEETING OF THE LUPUS ACADEMY, Warsaw, Poland, September 6–8, 2019. Lupus Foundation of America, 2019. http://dx.doi.org/10.1136/lupus-2019-la.5.

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Kumar, Ashna. "657 Macrophage activation syndrome and sepsis." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 15 June 2021–17 June 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-rcpch.124.

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Kuis, W., NM Wulffraat, and GT Rijkers. "SP0075 Macrophage activation syndrome and systemic jia." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.26.

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Di Vincenzo, Serena, Maria Ferraro, Valentina Lazzara, et al. "TSLP blocking modifies cross-talk between airway epithelium and macrophages reducing macrophage activation." In ERS Congress 2024 abstracts. European Respiratory Society, 2024. http://dx.doi.org/10.1183/13993003.congress-2024.oa1968.

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Jain, D., Y. Tomer, A. Malur, MJ Thomassen, and MF Beers. "SP-D Modulates Alveolar Macrophage Polarization and Activation." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a6270.

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Song, Sheng, Feifan Zhou, Wei R. Chen, and Da Xing. "Direct imaging of macrophage activation during PDT treatment." In Photonics and Optoelectronics Meetings 2011. SPIE, 2012. http://dx.doi.org/10.1117/12.919005.

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Oliveira, Renato Lucindo Bolelli de, Maria Clara de Castro e. Caetano, Natalia Lamas Rosario, et al. "ADULT-ONSET STILL’S DISEASE WITH MACROPHAGE ACTIVATION SYNDROME." In XXXIX Congresso Brasileiro de Reumatologia. Sociedade Brasileiro de Reumatologia, 2022. http://dx.doi.org/10.47660/cbr.2022.1992.

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Chang, Haocai. "Photobiomodulation enhances macrophage phagocytic capacity via Rac1 activation." In Twelfth International Conference on Information Optics and Photonics, edited by Yue Yang. SPIE, 2021. http://dx.doi.org/10.1117/12.2604421.

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Tenorio, Elizabeth, Del Donehoo, Sanjeevani Sahu, Achu Byju, Shreya A. Raghavan, and Balakrishna Haridas. "Evaluating Macrophage Immune Response to Degradation Byproducts from Magnesium Alloy Wire." In 2025 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2025. https://doi.org/10.1115/dmd2025-1062.

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Abstract Magnesium alloys have recently gained increasing attention for biomedical applications due to their unique biodegradation and biocompatibility profiles. However, investigations of the interactions between magnesium alloy degradation byproducts and host immune cells (particularly macrophages) are still relatively unexplored. Here, we study the interaction between degradation byproducts and macrophage response in vitro. An extract-based in vitro study was developed to examine macrophage response to different degradation byproducts formed at various time points of the corrosion process w
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Raporty organizacyjne na temat "Macrophage activation"

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Schwartz, Bertha, Vaclav Vetvicka, Ofer Danai, and Yitzhak Hadar. Increasing the value of mushrooms as functional foods: induction of alpha and beta glucan content via novel cultivation methods. United States Department of Agriculture, 2015. http://dx.doi.org/10.32747/2015.7600033.bard.

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During the granting period, we performed the following projects: Firstly, we differentially measured glucan content in several pleurotus mushroom strains. Mushroom polysaccharides are edible polymers that have numerous reported biological functions; the most common effects are attributed to β-glucans. In recent years, it became apparent that the less abundant α-glucans also possess potent effects in various health conditions. In our first study, we explored several Pleurotus species for their total, β and α-glucan content. Pleurotuseryngii was found to have the highest total glucan concentrati
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MicroRNA-mediated calcineurin signaling activation induces CCL2, CCL3, CCL5, IL8 and chemotactic activities in 4,4'-methylene diphenyl diisocyanate exposed macrophages (dataset). U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 2022. http://dx.doi.org/10.26616/nioshrd-1027-2021-0.

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