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Rozprawy doktorskie na temat „Macrophage”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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1

Svensson, Ulf. "Macrophage activation by bacteria signalling to prostaglandin and cytokine responses /." Lund : Dept. of Medical & Physiological Chemistry, Lund University, 1994. http://books.google.com/books?id=sAhrAAAAMAAJ.

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Higuera, González Laura 1993. "Novel transcription regulators of tissue macrophages and alternative macrophage polarization." Doctoral thesis, TDX (Tesis Doctorals en Xarxa), 2021. http://hdl.handle.net/10803/672702.

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Macrophages play crucial roles in the defense of the organism against a wide range of pathogens. Macrophages can rapidly adapt to perturbations in the microenvironment due to the existence of a network of transcription factors that modulates their responses. While transcription factors that regulate macrophage identity have been widely described in the past decades, the role of transcription regulators that fine-tune tissue macrophage responses in homeostasis and infection is starting to be elucidated. Our group has previously identified transcription regulators of pro-inflammatory macrophag

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Tabata, Yasuhiko. "Macrophage phagocytosis of polymer microspheres and antitumor activation of macrophages." Kyoto University, 1987. http://hdl.handle.net/2433/74704.

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Raborn, Erinn Shenee. "Cannabinoid Modulation of Chemotaxis of Macrophages and Macrophage-like Cells." VCU Scholars Compass, 2007. http://hdl.handle.net/10156/1333.

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5

Adler, Heiko. "Fetal bovine bone marrow-derived macrophages : a model for studying basic aspects of macrophage biology and pathogen-macrophage interaction in cattle /." [S.l.] : [s.n.], 1994. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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6

Grand-Perret, Thierry A. R. "Induction d'une activité anti-tumorale chez les macrophages péritonéaux murins." Paris 11, 1986. http://www.theses.fr/1986PA112301.

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7

Di, Maggio Paula. "Dietary lipids and inflammation : chylomicron remnants suppress pro-inflammatory pathways and activate antioxidant defence mechanisms in human macrophages." Thesis, Royal Veterinary College (University of London), 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618287.

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8

Soe-Lin, Shan. "Macrophage iron recycling." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=66717.

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In an absurd twist of nature, the physiological role of iron is paradoxical. Iron is the most abundant element found on Earth and yet is insoluble under physiological conditions. Furthermore, life is not possible without iron; iron is indispensible for life, as it is a vital co-factor for essential enzymes due to its unique redox abilities. And yet, high concentrations of iron lead to the formation of reactive oxygen species and are toxic. Consequently, living creatures have evolved ingenious strategies for acquiring and managing otherwise insoluble iron atoms, and for tightl

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9

Georges, George Tharwat. "Novel Characteristics of Murine Bone Marrow-Derived Macrophages and Human Macrophage-Like Cells." VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/932.

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These studies provide evidence for novel properties of macrophages derived from bone marrow stem cells. In study 1, treatment of activated mouse bone marrow-derived macrophages (BMM) with either catecholamine synthesis inhibitors (α-methyl-para-tyrosine and fusaric acid) or the β2 adrenergic receptor antagonist ICI 118,551 demonstrated that BMM produce catecholamines. The catecholamines modulated macrophage cytokine production through autocrine actions on adrenergic receptors. In study II, undifferentiated human bone marrow cells were incubated in 30% mouse L929 fibroblast conditioned medium

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10

Sobhani, Kimia. "Proteomic analysis of macrophage proinflammatory programmed cell death and macrophage activation /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/8688.

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11

Awomoyi, Agnes Abiola Oluwatoyin. "Genetics of susceptibility to tuberculosis." Thesis, Open University, 2000. http://oro.open.ac.uk/58012/.

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Convincing evidence that activated macrophages play a critical role in control of mycobacterial diseases has been clearly established from animal and in-vitro studies. Macrophages produce a variety of molecules upon appropriate stimulation, which act in concert towards eventual killing of bacteria. People with SUb-optimal macrophage activation are more susceptible to infection with intracellular pathogens. My project aims to answer two questions relating to genetic regulation of macrophage activation in tuberculosis: do macrophage genes regulate microbial-induced responses and do macrophage ge

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12

Davis, John Beresford. "The macrophage in atherogenesis." Thesis, University of Cambridge, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.276341.

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13

Falck-Hansen, Mika André. "Macrophage regulation in atherosclerosis." Thesis, Imperial College London, 2013. http://hdl.handle.net/10044/1/29863.

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Atherosclerosis is the leading cause of cardiovascular disease, topping the mortality list in the United Kingdom and the rest of the world. Macrophage activation and polarisation are key steps in host defence and chronic inflammatory diseases, including atherosclerosis. The myeloid glycoprotein receptor CD200R1 belongs to a family of four isoforms and signals after binding to its cognate ligand, CD200. The CD200/CD200R1 interaction blocks pro-inflammatory cytokines and has never before been studied in atherosclerosis. My work has demonstrated that CD200R is weakly expressed in atherosclerotic

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14

Dewhurst, Jennifer. "Macrophage subpopulations in COPD." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/macrophage-subpopulations-in-copd(e0e66fa4-ddff-4591-b785-edee74918539).html.

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Lung macrophage numbers are increased in Chronic Obstructive Pulmonary Disease (COPD). Macrophage subpopulations with differences in cell surface marker expression and function have been identified in sputum. The aim of this thesis was to investigate the role of lower airway macrophage subpopulations in COPD, focussing on their phenotype and function. Alveolar and interstitial cells from human lung resection tissue were isolated by perfusing the airways with saline. Interstitial cells were isolated by mechanically and enzymatically digesting perfused lung tissue. Alveolar and interstitial macr

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15

Davies, Luke C. "Control of macrophage homeostasis." Thesis, Cardiff University, 2014. http://orca.cf.ac.uk/112185/.

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Tissue resident macrophages are extremely heterogeneous, which reflects their unique microenvironments and tissue specific functions. They are a constituent of all tissues, and are involved in homeostatic processes and inflammatory disease. Recent studies have shown that select tissue resident macrophage populations, such as Langerhans cells of the skin and microglia of the brain, are able to self-renew independently from the bone marrow. This is contrary to the prevailing model macrophage origins, the ‘mononuclear phagocyte system’, which dictates that all macrophages are derived from bone ma

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16

Bouchareychas, Laura. "Implication des phagocytes mononuclées dans l'évolution de la plaque d'athérosclérose et relation avec l'homéostasie du cholestérol et des lipoprotéines." Thesis, Paris 6, 2014. http://www.theses.fr/2014PA066282/document.

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L'athérosclérose est un processus physiopathologique chronique impliqué dans la majorité des maladies cardio-vasculaires. Le développement des lésions d'athérosclérose est caractérisé par l'accumulation de lipides extra et intracellulaires dans la paroi artérielle à l'origine d'une forte réponse inflammatoire impliquant notamment les macrophages. Les macrophages sont considérés comme des acteurs clés dans le développement des plaques d'athérosclérose. En effet, de par leur capacité à métaboliser le cholestérol (captation, stockage, efflux), à réguler l'inflammation et à phagocyter les cellules

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17

Lisowski, Zofia Maria. "Targeting the macrophage in equine post-operative ileus." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/33191.

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Post-operative ileus (POI) is the functional inhibition of propulsive intestinal motility which is a frequent occurrence following abdominal surgery in the horse and in humans. Rodent and human-derived data have shown that manipulation-induced activation of the resident muscularis externa (ME) macrophages in the intestine contributes to the pathophysiology of the disease. Most studies of the disease, specifically in the horse, have focussed on identification of risk factors, descriptive studies of the disease or the assessment of the efficacy of various therapeutic and prophylactic interventio

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18

Alhanghari, Mofeda Abdussalam. "The Anti-Apoptotic Effect of HSV-1 ON Murine Macrophages: RAW 246.7Murine Macrophage Cell Line." Wright State University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=wright1472747895.

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19

Lévêque, Manuella. "Résolution de l'inflammation - infection dans les macrophages de patients atteints de mucoviscidose : impact de la membrane." Thesis, Rennes 1, 2016. http://www.theses.fr/2016REN1B041/document.

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Les macrophages sont en première ligne de la défense innée et jouent un rôle important dans l’initiation de la réponse immunitaire puisque régulant l’inflammation et permettant la clairance des pathogènes. Dans la mucoviscidose, ces phénomènes sont exacerbés et deviennent chronique sans pouvoir être résolus. Les objectifs de cette thèse ont été de déterminer des cibles potentielles responsables des altérations du macrophage dans la mucoviscidose. Dans le contexte inflammatoire, la forme soluble du CD14 (sCD14), dont la sécrétion est augmentée par les macrophages de patients atteints de la muco

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20

Foo, Suan Sin. "Deciphering the Role of Macrophage Subsets and Macrophage-Derived Factors During Arthrogenic Alphaviral Infection." Thesis, Griffith University, 2015. http://hdl.handle.net/10072/365249.

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The current situation of global warming has a serious impact on the control of arthropod-borne infectious diseases. Climate change has led to an increase in conducive breeding habitats for mosquitoes. This change in climate has contributed to the widespread distribution of mosquito-transmitted arthritogenic alphaviruses such as Ross River virus (RRV) and chikungunya virus (CHIKV). RRV is currently endemic to Australia and Papua New Guinea, while CHIKV causes global outbreaks. The recent CHIKV outbreak in the Americas has taken the world by surprise and has affected more than 1 million individu

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21

Merlin, Johanna. "Étude de l’influence de la glutaminolyse des macrophages dans les maladies cardio-métaboliques." Electronic Thesis or Diss., Université Côte d'Azur, 2020. http://theses.univ-cotedazur.fr/2020COAZ6016.

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Les maladies chroniques inflammatoires telles que l’obésité ou l’athérosclérose constituent un problème majeur de santé publique dans les pays occidentaux. Il est désormais bien établi que les cellules immunitaires, et en particulier les macrophages, jouent un rôle essentiel dans l’initiation et la progression de ces maladies métaboliques. En effet, les macrophages résidents au sein des tissus contrôlent leur homéostasie comme par exemple l’expansion du tissu adipeux viscéral ou la thermogenèse du tissu adipeux brun. Cependant, les mécanismes restent mal compris. L’immuno-métabolisme est un no

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22

Reichard, Adam Craig. "The Effects of HSV-1 Challenge on Polarized Murine Macrophages: an In Vitro Model Using the J774A.1 Murine Macrophage Cell Line." Wright State University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=wright1343411395.

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23

Kluftinger, Janet Louise. "Macrophage interaction with Pseudomonas aeruginosa." Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/29129.

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The interactions of macrophages with Pseudomonas aeruginosa were studied. Five monoclonal antibodies specific for porin protein F were tested for their ability to opsonize P. aeruginosa for complement-independent phagocytosis by unelicited mouse peritoneal macrophages, human peripheral blood monocytes and mouse macrophage cell line P388[sub D1]. All five antibodies significantly increased the level of bacterial uptake over that obtained with the non-opsonic controls. The relative effectiveness of the different antibodies was approximately the same in all cell types indicating that the P388[sub

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24

Truman, Lucy. "Macrophage chemotaxis to apoptotic cells." Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/29404.

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The CD14 knockout mouse (CD14-/-) was observed to have increased numbers of free apoptotic cells in the thymus. It was hypothesised that this was due to a defect in macrophage clearance of dying cells. <i>In vitro </i>assays revealed that macrophages from the CD14-/- mouse had a partial defect in the phagocytosis of apoptotic cells. Clearance also involves macrophage movement towards the apoptotic corpse. Therefore, it was hypothesised that, in addition to a phagocytosis defect, CD14-/- had impaired chemotaxis to apoptotic cells. An <i>in vitro </i>transmigration assay was developed using the

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25

Heasman, Sarah Jane. "Glucocorticoid modulation of macrophage function." Thesis, University of Edinburgh, 2005. http://hdl.handle.net/1842/29146.

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In this thesis, I have examined the effects of glucocorticoid-treatment of peripheral blood monocytes which has previously been demonstrated to markedly augment phagocytic capacity for apoptotic cells, an effect which may contribute to anti-inflammatory actions of glucocorticoids. Within the inflammatory site, the cytokine environment governs the differentiation and function of infiltrating leukocytes. I have investigated the effects of combinatorial treatment of monocytes with the principal Th1 and Th2 cytokines, IFN-γ and IL-4. I have demonstrated that whilst glucocorticoids exert a dominant

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26

McMurray, Heather Forbes. "Macrophage growth factors in atherogenesis." Thesis, University of Cambridge, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.306460.

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27

Rossi, B. C. "Macrophage function in African trypanosomiasis." Thesis, Brunel University, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.373784.

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28

McCarthy, Sean Patrick. "Studies on human macrophage heterogeneity." Thesis, University of Oxford, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.238184.

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29

Wright, Adam. "The macrophage in cystic fibrosis." Thesis, University of Leicester, 2007. http://hdl.handle.net/2381/8783.

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Background: Cystic fibrosis (CF) is caused by absent/defective CF transmembrane conductance regulator protein (CFTR). CF is characterized by thick airway mucus, chronic infection and neutrophil inflammation leading to respiratory failure. I analysed airway macrophages (MΦs) and their expression of pattern recognition receptors (PRRs) in CF, since these cells are crucial to airway immune defence and they can orchestrate inflammation. I also performed transcript analysis of CF monocyte-derived MΦs (MDMs). Methods: Sputum was induced in CF paediatric and adult cohorts. Phenotype and function of C

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30

Pound, J. D. "Parameters of human macrophage activation." Thesis, University of Nottingham, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.381442.

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31

Reid, Vanessa Claire. "Macrophage toxicity of lipid oxidation." Thesis, University of Cambridge, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308384.

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32

Savill, John Stewart. "Macrophage recognition of senescent neutrophils." Thesis, Imperial College London, 1989. http://hdl.handle.net/10044/1/47641.

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Voelz, Kerstin. "Macrophage – cryptococcus interactions during cryptococcosis." Thesis, University of Birmingham, 2010. http://etheses.bham.ac.uk//id/eprint/1194/.

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The human fungal pathogens Cryptococcus neoformans and C. gattii cause life-threatening infections of the central nervous system. One of the major characteristics of cryptococcal disease is the ability of the pathogen to parasitise upon phagocytic effector cells. Cryptococcus can survive and proliferate within macrophages, and is also capable of escaping into the intracellular environment via a non-lytic mechanism (‘expulsion’) and can be transferred directly from one cell to another (lateral transfer). In the first part of this thesis, I demonstrate that enhanced Th2, but not Th1, cytokine le

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34

Hegyi, Laszlo. "Macrophage apoptosis and human atherosclerosis." Thesis, University of Cambridge, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627017.

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35

Cano, Antonella. "Characterization of Acanthamoeba macrophage activation." Thesis, University of Strathclyde, 2015. http://oleg.lib.strath.ac.uk:80/R/?func=dbin-jump-full&object_id=25992.

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36

Goding, Linda M. "Macrophage Recognition of Xenogeneic Erythrocytes." University of Toledo Health Science Campus / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=mco1197469419.

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37

Erwig, Lars-Peter. "Macrophage programming in inflammatory disease." Thesis, University of Aberdeen, 2004. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU194083.

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Before embarking on the work presented here I showed that certain activating signals, such as IFN-gamma and TNF-alpha programmed macrophages to develop distinct sets of properties in vitro, which included unresponsiveness to other types of activation. This raised the question whether macrophage programming occurs in passive and active renal inflammation and whether the macrophage programme could be biased by systemic administration of cytokines. The data presented here shows that macrophages infiltrating acutely inflamed glomeruli of rats with nephrotoxic nephritis display programmed behaviour

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Hunter, Catriona Mhairi. "MicroRNA regulation of macrophage activation." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/31027.

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Macrophages are mononuclear phagocytic cells that have diverse roles within the body. Tissue specific macrophages, e.g. Kupffer cells, microglia and osteoclasts, have roles in tissue homeostasis, while circulating macrophages play an important role in the innate immune system. Macrophages detect the presence of pathogen associated molecular patterns (PAMPs) via a range of receptors known collectively as pathogen recognition receptors (PRRs). Detection of pathogens causes the macrophages to become ‘activated,’ during which the macrophages undergo extreme morphological and translational changes

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39

Holmes, Benjamin A. "The Construction of a Plasmid for Detecting the Pathway of Arginine Metabolism in Human Macrophages: a Real-Time Assessment of Macrophage Polarity." Wright State University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=wright1348874681.

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40

Shabo, Ivan. "Macrophage antigen expression in breast and colorectal cancers : A consequence of macrophage - tumour cell fusion?" Doctoral thesis, Linköpings universitet, Kirurgi, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-54820.

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Carcinogenesis is a sophisticated biological process consisting of a series of progressive changes in somatic cells from premalignant to malignant phenotype. Despite the vast information available about cancer cells, the origin of cancer and cause of metastasis still remain enigmatic. The hypothesis of cell fusion is one of several models explaining the evolution of neoplasia into clinically significant cancer. This theory states that cancer cells through heterotypic fusion with host cells generate hybrids expressing traits from both parental cells, and acquire metastatic potentials and growth

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41

Park, Young Mi. "Interaction between CD36 and Oxidized LDL Modulates Macrophage Cytoskeletal Functions: A Mechanism of Macrophage Trapping." Case Western Reserve University School of Graduate Studies / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=case1270624670.

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42

Ellouze, Mehdi. "Identification des mécanismes anti-inflammatoires de GILZ dans les monocytes/macrophages et de son potentiel thérapeutique dans le choc septique." Thesis, Université Paris-Saclay (ComUE), 2015. http://www.theses.fr/2015SACLS239/document.

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Le Sepsis et le choc septique, associés à une inflammation systémique sévère et incontrôlée, sont les principales causes de mortalité dans les unités de soins intensifs. Les macrophages jouent un rôle central dans ces pathologies. Ils participent à l'initiation et à la régulation de l'inflammation. Lors d'une infection bactérienne, ils reconnaissent le LPS de la paroi bactérienne par l’intermédiaire du TLR4 ce qui déclenche l’activation des MAPK, des facteurs de transcription NF-kB et AP1 et, in fine, la production des cytokines pro-inflammatoires dont le TNF et l'IL6. L’expression de la proté

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Delfini, Marcello. "Jun regulates monocyte-derived macrophage accumulation and tumour progression." Thesis, Aix-Marseille, 2019. http://www.theses.fr/2019AIXM0076.

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Les macrophages sont des cellules immunitaires innées présentes dans chaque organe. Ils sont des cibles thérapeutiques dans de nombreuses maladies, dont le cancer. En dépit de travaux récents sur l'origine des macrophages, les mécanismes régulant leur différenciation sont mal définis. L'expression de Jun, membre de la famille AP-1, augmente pendant la différenciation des macrophages, mais son rôle dans ce processus n'est pas connu.Au cours de mon doctorat, nous avons caractérisé le rôle de Jun dans le développement et l'homéostasie des macrophages, dans un modèle de souris avec délétion condit

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44

Chanez, Pascal. "Hétérogéneité morphologique et fonctionnelle des macrophages des voies aériennes de l'asthmatique." Montpellier 1, 1994. http://www.theses.fr/1994MON1T038.

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Rivier, Agnès. "Activation des phagocytes mononucléés chez les sujets normaux et les sujets asthmatiques/ Agnès Rivier." Montpellier 1, 1994. http://www.theses.fr/1994MON1T020.

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46

Haddad, Elias K. "Study of the role of macrophage activation and macrophage derived cytotoxic factors in early embryo loss." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ29928.pdf.

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47

Haddad, Elias K. "Study of the role of macrophage activation and macrophage derived cytoxic factors in early embryo loss." Thesis, McGill University, 1997. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=42023.

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Using murine models of spontaneous and induced embryo resorption, we have investigated the role of macrophages in the mechanism of early embryo loss. The results showed that macrophage derived nitric oxide was associated with embryo resorption, and that decidual macrophages could be triggered by lipopolysaccharide (LPS) to produce nitric oxide, indicating that the decidual mononuclear cells were primed in situ. Using double immunostaining, we have shown that macrophages were the cellular source of the inducible nitric oxide production. We further showed that embryo abortion can be significantl

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48

Warby, Tammra. "Interactions between granulocyte-macrophage colony-stimulating factor and human monocyte-derived macrophages following infection with HIV-1 /." [St. Lucia, Qld.], 2006. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19318.pdf.

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49

Finney-Hayward, Tricia Kate. "Monocyte-derived macrophages as a lung macrophage model in chronic obstructive pulmonary disease : characterisation and functional output." Thesis, Imperial College London, 2005. http://hdl.handle.net/10044/1/8330.

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50

Walter, Michaela Roylene Valerie. "Macrophages in bovine footrot, the effect of Porphyromonas levii on macrophage function and pro-inflammatory cytokine production." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/MQ65143.pdf.

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