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Rozprawy doktorskie na temat „Mast cells”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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1

Dacre, Kirstie Jane. "Involvement of mast cells and mast cell serine proteinases in equine heaves." Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/29721.

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Mast cells release potent mediators upon degranulation, including serine proteinases. These proteinases play a pivotal role in the pathogenesis of human asthma. Due to the similarities between human asthma and equine heaves, a similar role for the mast cell in equine heaves is proposed. Clinical heaves horses had significantly increased BALF tryptase concentrations compared to controls or heaves horses in remission, whereas BALF tryptase concentrations of controls and heaves horses in remission did not significantly differ. Horses with other pulmonary diseases also had significantly elevated B

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2

Leskinen, Markus. "Mast cell-mediated apoptosis of smooth muscle cells and endothelial cells." Helsinki : University of Helsinki, 2003. http://ethesis.helsinki.fi/julkaisut/laa/kliin/vk/leskinen/.

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3

Crummy, F. "Adenosine, mast cells and asthma." Thesis, Queen's University Belfast, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.403238.

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4

Huntley, John Frederick. "Mast cells and intestinal nematodiasis." Thesis, University of Edinburgh, 1991. http://hdl.handle.net/1842/15070.

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Specific enzyme-linked immunosorbent assays (ELISA) for rat mast cell proteinase I and II (RMCP I and II), intestinal mast cell proteinase (IMCP) and sheep mast cell proteinase (SMCP) were developed. Sheep serum or lymph contained potent inhibitory factors which interfered with the ELISA for SMCP, whereas little or no effect was demonstrated in the rodent ELISA by homologous serum. Secretion of SMCP into gastric lymph was noted in immune sheep following oral challenge with <i>Ostertagia circumcincta</i>. Development of immunity to <i>Haemonchus contortus</i> in sheep, expressed as the ability

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5

Alswied, Abdullah M. "Calcium signalling in mast cells." Thesis, University of Oxford, 2015. https://ora.ox.ac.uk/objects/uuid:cc8f5f8b-5cab-4391-bce3-9541ab371002.

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Mast cells play a central role in many allergic and in ammatory conditions. These cells are activated following an intracellular rise in calcium, such as that which occurs after the activation of cell-surface receptors. One such important receptor is cysteinyl leukotriene (CysLT) receptor type 1 (CysLT1), which is activated by lipid mediators such as CysLTs LTC4, LTD4, and LTE4. CysLT1 stimulation leads to the hydrolysis of membrane phospholipids such as phosphatidylinositol 4 5-bisphosphate (PIP2) via phospholipase C-β , which results in the generation of diacylglycerol and inositol trisphosp

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6

Roy, Ananya. "Mast Cells as Sentinels : Role of serglycin and mast cell proteases in infection and inflammation." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-173508.

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Mast cells (MCs), normally classified into connective tissue MCs and mucosal MCs, are highly granulated cells found in the interface between the interior and the exterior environment of our body, e.g. skin, airways and gastro-intestinal tract. They react to bacteria, parasites, viruses, and allergens by degranulation and release of premade and newly synthesized inflammatory mediators. The MC-proteases (tryptases, chymases and carboxypeptidase A), histamine and serglycin (SG) proteoglycans are premade mediators. Among these, SG is also expressed in a variety of other immune and non-immune cells

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7

Friend, Reuben. "SNARE proteins in human mast cells." Thesis, University of Sheffield, 2013. http://etheses.whiterose.ac.uk/5177/.

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Mast cells form an integral part of both innate and adaptive immunity; they help to orchestrate the inflammatory immune response through the release of a variety of inflammatory mediators. Adverse reaction to allergens can lead to activation of mast cells, causing degranulation and release of a range of pro-inflammatory mediators contributing to the onset of allergy. The most studied activation pathway in the adaptive immune response of mast cells is through the Immunoglobulin E (IgE) cell surface receptor FceRI. Crosslinking of FceRI leads to degranulation and de novo synthesis of mediators.

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8

Tree-Booker, Claire. "TRPC channels in human mast cells." Thesis, University of Sheffield, 2011. http://etheses.whiterose.ac.uk/1927/.

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Mast cells have an important role in the immune system, but they are centrally involved in the pathophysiology of asthma, along with a number of other allergic diseases including rhinitis, eczema and irritable bowel disease (Metcalfe et al., 1997; Beaven, 2009). In the allergic response they are activated by IgE binding to high affinity receptors and subsequent crosslinking by antigen. A rise in intracellular Ca2+ is required for mast cells to become activated and release mediators into the surrounding areas, which give rise to the symptoms of allergic disease (Gilfillan & Tkaczyk, 2006). Eluc

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9

Lin, Tzu-Yin. "The world according to mast cells – the role of Kit in normal and neoplastic canine mast cells." The Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=osu1189098916.

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10

Wang, Yiyu [Verfasser]. "Analysis of mast cells and mast cell-mediator-related histological features in cholinergic urticaria / Yiyu Wang." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2019. http://d-nb.info/1189139715/34.

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11

Noma, Naruto. "Inhibition of MMP-2-Mediated Mast Cell Invasion by NF-κB Inhibitor DHMEQ in Mast Cells". 京都大学 (Kyoto University), 2017. http://hdl.handle.net/2433/225446.

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12

Kinoshita, Makoto. "Mast cell tryptase in mast cell granules enhances MCP-1 and interleukin-8 production in human endothelial cells." Kyoto University, 2006. http://hdl.handle.net/2433/144319.

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13

Möller, Christine. "Regulation of mast cell survival /." Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-4703.

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14

Lin, Tzu-yin. "The world according to mast cells the role of Kit in normal and neoplastic canine mast cells /." Columbus, Ohio : Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1189098916.

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15

Offiah, I. "Cross-talk between human T cells, mast cells and conjunctival epithelial cells." Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1348498/.

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The ocular surface is continually exposed to the outside environment and is a common site of inflammation. Conjunctival epithelial cells are thought to play a role in innate responses at the ocular surface. The hypothesis of my study is that conjunctival epithelial cells also contribute to T cell and mast cell effector mechanisms in chronic allergic eye disease via secretion of cytokines. In this study we initially demonstrate that the conjunctiva expresses TLRs, and that the TLR3 ligand (poly I:C) activates conjunctival epithelial cells in vitro to secrete inflammatory mediators as part of th

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16

Shende, Vishvesh H. "Role of mast cells in ischaemic stroke." Thesis, University of Strathclyde, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.502318.

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The purpose of the project was to investigate the role of mast cells and oestrogen in ischaemic stroke. Inflammatory cells and mediators may prove to be novel targets for stroke. In addition studies carried out on animals clearly signify that biological and endogenous sex steroids play a role in experimental stroke outcome. Histological staining (toluidine blue) of coronal sections from SHRSP and WKY rats post-middle cerebral artery occlusion (MCAO, an experimental stroke model) was used to observe cerebral mast cells and their anatomical localisation. Mast cells were found in the brain at 2 h

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17

Elkovich, Andrea J. "Mast Cells In Kainate Receptor Knockout Mice." VCU Scholars Compass, 2015. http://scholarscompass.vcu.edu/etd/3944.

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Kainate receptor knockout mice have unique differences within their immune system. They exhibit an attenuated TH2 branch, while maintaining a robust TH1 response. Specifically, blocking the formation of functional kainate receptors affects mast cells and their related pathologies. While they seem to develop and activate normally in vivo and in vitro, KAR KO mast cells release more inflammatory mediators upon degranulation. These mice experience severe anaphylactic shock due to two compounding abnormalities. First, KAR KO mast cells release significantly more histamine in vivo upon IgE-med

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18

Compton, Steven John. "The proinflammatory actions of mast cell tryptase on human endothelial cells." Thesis, University of Southampton, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.245046.

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19

Sundström, Magnus. "Signal transduction in mast cell migration /." Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5130-6/.

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20

Peng, Qi. "The heterogeneity, mechanism of regulation and function of human mast cell tryptase." Thesis, University of Southampton, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285784.

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21

Takano, Hirotsugi. "Terminal differentiation of connective tissue-type mast cells." 京都大学 (Kyoto University), 2008. http://hdl.handle.net/2433/137162.

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22

Zhu, Fu-Gang. "Mechanisms of inflammatory cytokine secretion by mast cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0029/NQ66688.pdf.

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23

Crisp, A. J. "Mast cells and their products in rheumatoid arthritis." Thesis, University of Cambridge, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598149.

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24

Cross, Laurence Joseph Mark. "Mast cells and their role in disease states." Thesis, Queen's University Belfast, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336723.

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25

Hooi, Peh Kheng. "Actions of balsalazide and sulphasalazine on mast cells." Thesis, University College London (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.294805.

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26

Okayama, Yoshimichi. "Immunopharmacological studies on human mast cells and basophils." Thesis, University of Southampton, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.296403.

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27

Wang, Yenfeng. "The role of mast cells in foam cell formation, growth inhibition, and apoptosis of smooth muscle cells." Helsinki : University of Helsinki, 1999. http://ethesis.helsinki.fi/julkaisut/mat/bioti/vk/wang/.

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28

Collmann, Emilie. "Role of phosphoindositide 3-kinases in mast cell activation /." [S.l.] : [s.n.], 2009. http://edoc.unibas.ch/diss/DissB_8779.

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29

Chugunova, Elena. "Biological function of mast cell chymase : in vitro and in vivo studies: a thorny pathway /." Uppsala : Dept. of Molecular Biosciences, Swedish Univ. of Agricultural Sciences, 2004. http://epsilon.slu.se/v181.pdf.

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30

Serra, Pagès Mariona. "Selective EP2 agonism attenuates hdm-induced murine airway pathology and mast cell activity, and triggers intracellular inhibitory signaling in mast cells." Doctoral thesis, Universitat Autònoma de Barcelona, 2012. http://hdl.handle.net/10803/84009.

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L'asma al lèrgica és una malaltia respiratòria crònica amb una alta prevalença en els països desenvolupats. Els tractaments actuals no aturen el procés al lèrgic subjacent ni aconsegueixen el control dels símptomes de la malaltia. El tractament més eficaç és l'ús de corticoesteroids, que es basen en modificacions químiques de potents hormones endògenes antiinflamatòries. L'estudi de vies antiinflamatòries endògenes és una estratègia experimental eficaç per descobrir noves dianes terapèutiques potencials contra l'asma. Una d'aquestes vies endògenes és la de la ciclooxigenasa (COX). Prost

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31

Meleski, Melissa. "The role of mast cells in fetal wound healing." Connect to resource, 2010. http://hdl.handle.net/1811/45043.

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32

Ghouse, Shanawaz Mohammed. "Role of Mast cells in HPV-induced skin cancer." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2017. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-229004.

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Mast cells (MCs) are long-lived immune cells, which were reported to play an important role in initiating innate and adaptive immune responses against various infections. MCs accumulate in high numbers in the stroma and at the invasion front of various human cancers, suggesting a possible contribution by MCs to tumour growth. Experimental studies using crosses of MC-deficient Kit-mutant mouse strains with mouse models of epithelial cancers have provided evidence for important MC tumour-promoting functions. However, the complex alterations of the immune system that characterize Kit-mutant mice

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33

Price, Leo Sebastian. "Secretion and the actin cytoskeleton in rat mast cells." Thesis, University College London (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.307776.

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34

Williams, Cara Margaret May. "Cytokine expression in mast cells and rat lung tissue." Thesis, University of Liverpool, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295818.

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35

COSTANZA, MASSIMO. "Mast cells in the pathogenesis of experimental multiple sclerosis." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2012. http://hdl.handle.net/10281/29633.

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Mast cell (MC)-deficient c-Kit mutant KitW/W-v mice are protected against experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis, suggesting a detrimental role for MCs in this disease. However studies performed so far on this topic relied on a single model of MC-deficiency, the KitW/W-v, which bears several c-Kit dependent phenotypic abnormalities other than MC-deficiency, such as neutropenia and anaemia. Also, it is not clear how MCs shape the central nervous system (CNS) autoimmune response occurring in EAE. In the first work, we focused on evaluating the cont

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36

Dahlin, Joakim. "Mast Cell Progenitor Trafficking in Allergic Airway Inflammation." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-206608.

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Mast cell progenitors originate from the bone marrow and migrate to the lungs via the blood. During maturation, these cells acquire granules that contain a potent array of bronchoconstrictive mediators. The number of pulmonary mast cells is augmented in asthmatic patients and in mice with allergic airway inflammation, possibly contributing to airway hyperreactivity. An increase in mast cells is likely due to an increased recruitment of committed mast cell progenitors from the blood. However, until now a committed mast cell progenitor population has not been found in adult peripheral blood. We

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37

Olsson, Niclas. "Mast Cell Migration in Inflammatory Diseases." Doctoral thesis, Uppsala University, Department of Genetics and Pathology, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3615.

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<p>Mast cells (MCs) are forceful multifunctional effector cells of the immune system. MCs are normally distributed throughout connective and mucosal tissues, but in several pathological conditions accumulation of MCs occur. This accumulation is probable due to directed migration of MCs and they are subjects for migration at least two different occations: 1) when they are recruited as progenitor cells from the blood into the tissue; and 2) when they as mature MCs are recruited to sites of inflammation. The aim of this study was to investigate MC migration to chemoattractants released <i>in vivo

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38

Zarnegar, Behdad. "Mechanisms triggering the recruitment of mast cell progenitors to the lung and regulation of mast cell degranulation." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-306115.

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Mast cells stem from the bone marrow and migrate via the blood as mast cell progenitors. Upon arrival in peripheral tissues, they develop into mast cells. These rare immune cells have numerous granules that contain large amounts of pro-inflammatory mediators. Mast cells accumulate at certain sites in the asthmatic lung, and once activated they release mediators that are thought to induce symptoms. In mouse models of allergic airway inflammation, the increase in lung mast cells in asthma can be mimicked and is mainly caused by the recruitment of mast cell progenitors to the lung. However, wheth

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39

GacÌ§a, Marianna Danuta Aleksandria. "The bi-directional relationship between mast cells and hepatic stellate cells in liver fibrosis." Thesis, University of Southampton, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.323958.

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40

Hara, Masatake. "Mast cells cause apoptosis of cardiomyocytes and proliferation of order intramyocardial cells in vitro." Kyoto University, 2000. http://hdl.handle.net/2433/180841.

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41

Stelekati, Erietta [Verfasser]. "The role of mast cells in CD8+ T cell-mediated immune responses / Erietta Stelekati." Kiel : Universitätsbibliothek Kiel, 2010. http://d-nb.info/1019951877/34.

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42

Kranenburg, Tanya Ann School of Medicine UNSW. "Isolation and characterisation of intact RBL-2H3 mast cell granules ~ phosphorylation events during secretion." Awarded by:University of New South Wales. School of Medicine, 2005. http://handle.unsw.edu.au/1959.4/23445.

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Mediators released from the granules of antigen-activated mast cells contribute to allergies, inflammation and diseases such as asthma. One of the major models used to study mucosal mast cells is the RBL-2H3 mast cell line. While there has been considerable research on the initial signalling events following IgE receptor (Fc??RI) cross-linking, the movement of granules to sites of exocytosis is poorly understood. Understanding the mechanisms that control granule movement to and fusion with the plasma membrane could provide novel targets for improved asthma and allergy therapeutics. To this end

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43

Eskandari, Nahid. "Characterisation of phosphodiesterases in human lung mast cells and basophils." Thesis, University of Sheffield, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.434577.

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44

Alfaiz, Faiz Abdulaziz. "Evaluation of the role of mast cells in parasitic infection." Thesis, University of Strathclyde, 2017. http://digitool.lib.strath.ac.uk:80/R/?func=dbin-jump-full&object_id=29560.

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Mast cells (MCs) are important for controlling both bacterial and parasitic infections, including those caused by helminths. However, their role in combatting these infections is unclear. MCs have been shown to be essential for the successful expulsion of Trichinella spiralis worms and play a role in mounting the immune response towards a T helper 2 type. Intestinal infection with parasitic worms is associated with mastocytosis and the activation and release of specific mediators and cytokines. In this case, MCs play an essential role in the successful expulsion of gastrointestinal worms via t

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45

Panop, Wilainam Parnpen Viriyavejakul. "Response of mast cells in skin biopsy of falciparum malaria /." Abstract, 2003. http://mulinet3.li.mahidol.ac.th/thesis/2546/46E-Panop-W.pdf.

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46

Farrington, Jasmine. "Calcium release activated calcium channels in human lung mast cells." Thesis, University of Sheffield, 2014. http://etheses.whiterose.ac.uk/6609/.

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47

Taruselli, Marcela. "Fluvastatin and microRNA-146a alter interleukin-33 mediated mast cell functions." VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/5859.

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Mast cells are tissue-resident immune cells known as effector cells for the innate and adaptive immune systems. Mast cells contribute to host defenses against parasites such as large roundworm parasites, bacterial pathogens, and toxins, and participate in wound healing, but they are mostly known for their role in allergic diseases. It has been well established that during allergic diseases, mast cells are stimulated by IgE cross-linkage to release proinflammatory mediators. However, a newly discovered cytokine, IL-33 has also been implicated in allergic disease. Recently, IL-33 has been implic

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48

Sime, Wondossen. "The diverse role of laminin isoforms in neuronal cells, human mast cells and blood platelets /." Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/978-91-7357-122-7/.

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49

Kitawaki, Toshio. "IgE-activated mast cells in combination with pro-inflammatory factors induce Th2-promoting dendritic cells." Kyoto University, 2007. http://hdl.handle.net/2433/135901.

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50

Sundström, Magnus. "Signal Transduction in Mast Cell Migration." Doctoral thesis, Uppsala University, Department of Genetics and Pathology, 2001. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-1474.

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<p>Mast cells are essential effector cells in the immune system as they release several inflammatory mediators. An accumulation of mast cells has been described in inflammatory conditions such as asthma and allergic rhinitis. Increased mast cell number, in the skin and other organs, is also a characteristic in mastocytosis, a disease without an effective treatment. One explanation for the increase in mast cell number is migration of mast cells in the tissue. In our studies we utilised mast cell lines, including HMC-1; cell lines transfected with the <i>c-kit</i> gene; and <i>in vitro</i> devel

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