Gotowe bibliografie tematyczne / Megakaryopoiesis

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Gotowa bibliografia na temat „Megakaryopoiesis”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 21 lutego 2023

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Artykuły w czasopismach na temat "Megakaryopoiesis"

Geddis, Amy E. "Megakaryopoiesis." Seminars in Hematology 47, no. 3 (July 2010): 212–19. http://dx.doi.org/10.1053/j.seminhematol.2010.03.001.

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Tozawa, Keiichi, Yukako Ono-Uruga, and Yumiko Matsubara. "Megakaryopoiesis." Clinical & Experimental Thrombosis and Hemostasis 1, no. 2 (November 10, 2014): 54–58. http://dx.doi.org/10.14345/ceth.14014.

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Jeanpierre, Sandrine, Franck Emmanuel Nicolini, Bastien Kaniewski, Charles Dumontet, Ruth Rimokh, Alain Puisieux, and Véronique Maguer-Satta. "BMP4 regulation of human megakaryocytic differentiation is involved in thrombopoietin signaling." Blood 112, no. 8 (October 15, 2008): 3154–63. http://dx.doi.org/10.1182/blood-2008-03-145326.

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Abstract Activin A, BMP2, and BMP4, 3 members of the transforming growth factor-β family, are involved in the regulation of hematopoiesis. Here, we explored the role of these molecules in human megakaryopoiesis using an in vitro serum-free assay. Our results highlight for the first time that, in the absence of thrombopoietin, BMP4 is able to induce CD34+ progenitor differentiation into megakaryocytes through all stages. Although we have previously shown that activin A and BMP2 are involved in erythropoietic commitment, these molecules have no effect on human megakaryopoietic engagement and dif

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Blobel, Gerd A. "Krüppeling megakaryopoiesis." Blood 110, no. 12 (December 1, 2007): 3823–24. http://dx.doi.org/10.1182/blood-2007-09-110999.

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KOZUMA, Yukinori. "Megakaryopoiesis and apoptosis." Japanese Journal of Thrombosis and Hemostasis 23, no. 6 (2012): 552–58. http://dx.doi.org/10.2491/jjsth.23.552.

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Jubinsky, Paul T. "Megakaryopoiesis and thrombocytosis." Pediatric Blood & Cancer 44, no. 1 (2004): 45–46. http://dx.doi.org/10.1002/pbc.20243.

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Feng, Gege, Wen Cui, Wenyu Cai, Tiejun Qin, Yue Zhang, Zefeng Xu, Liwei Fang, et al. "Impact of Megakaryocyte Morphology on Prognosis of Persons with Myelodysplastic Syndromes." Blood 126, no. 23 (December 3, 2015): 2876. http://dx.doi.org/10.1182/blood.v126.23.2876.2876.

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Abstract Purpose: To describe the morphological evolution of megakaryocytic dysplasia by developing a systematic classification and evaluate the impact of our classification of dys-megakaryopoiesis on prognosis of persons with MDS. Patients and methods: 423 consecutive patients who had received no prior therapy with MDS diagnosed from January 2000 to April 2014 were enrolled. Follow-up data were available for 371 subjects (88%). Date of last follow-up was December 15, 2014 or date of last contact. Median follow-up was 22 months (range, 1¨C180 months). Subjects with lower-risk MDS fall into Rev

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Liu, Zhi-Jian, and Martha Sola-Visner. "Neonatal and adult megakaryopoiesis." Current Opinion in Hematology 18, no. 5 (September 2011): 330–37. http://dx.doi.org/10.1097/moh.0b013e3283497ed5.

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Behrens, Kira, and Warren S. Alexander. "Cytokine control of megakaryopoiesis." Growth Factors 36, no. 3-4 (July 4, 2018): 89–103. http://dx.doi.org/10.1080/08977194.2018.1498487.

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Szalai, G., A. C. LaRue, and D. K. Watson. "Molecular mechanisms of megakaryopoiesis." Cellular and Molecular Life Sciences 63, no. 21 (August 11, 2006): 2460–76. http://dx.doi.org/10.1007/s00018-006-6190-8.

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Rozprawy doktorskie na temat "Megakaryopoiesis"

Pan, Jiajia. "The Role of DIAPH1 in the Megakaryopoiesis." Thesis, Paris 11, 2014. http://www.theses.fr/2014PA11T074/document.

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Les mégacaryocytes sont les précurseurs cellulaires hautement spécialisés qui produisent des plaquettes via des extensions cytoplasmiques appelées proplaquettes. La formation des proplaquettes exige de profonds changements dans l’organisation du cytosquelette: microtubules et actine. Les formines sont une famille de protéines hautement conservées chez les eucaryotes composées de plusieurs domaines qui régulent le remodelage et la dynamique du cytosquelette d'actine et des microtubules. La plupart des formines sont des effecteurs protéiques des Rho-GTPase. DIAPH1, un membre de la famille des fo

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Radhakrishnan, Aparna. "Genetic variation studies of megakaryopoiesis, platelet formation and platelet function." Thesis, University of Cambridge, 2014. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708102.

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Johnson, Lacey Nicole St George Clinical School UNSW. "Molecular regulation of Megakaryopoiesis: the role of Fli-1 and IFI16." Awarded by:University of New South Wales. St George Clinical School, 2006. http://handle.unsw.edu.au/1959.4/26819.

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Megakaryocytes (Mks) are unique bone marrow cells, which produce platelets. Dysregulated Mk development can lead to abnormal platelet number and the production of functionally defective platelets, causing bleeding, thrombotic events, and leukaemia. Understanding the molecular mechanisms driving megakaryopoiesis may yield insights into the molecular genetics and cellular pathophysiology of a diversity of disorders. The primary aim of this thesis was to gain insight into the molecular events required for normal Mk development. As transcription factors and cytokines play a central role in driving

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Houwerzijl, Ewout Johan. "Studies on megakaryopoiesis in patients with myelodysplasia and idiopathic thrombocytopenic purpura." [S.l. : Groningen : s.n. ; University Library of Groningen] [Host], 2008. http://irs.ub.rug.nl/ppn/306088665.

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Xiong, Y. "Regulation of T-cell adhesion and megakaryopoiesis by immune adaptor ADAP." Thesis, University of Liverpool, 2017. http://livrepository.liverpool.ac.uk/3011772/.

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The immune adaptor ADAP possesses versatile roles in a variety of immune cells, including T cells, dendritic cells, macrophages, and platelets, etc. The most extensivelystudied role of ADAP is that it couples TCR activation to integrin activation and T-cell adhesion. However, the regulation of this adaptor during integrin activation and T-cell adhesion remains unclear. Meantime, the functions of ADAP linked to other immune cells are largely unknown. Work in this thesis have identified Ubc9, the sole SUMO E2 conjugase, as an essential regulator of ADAP in T-cell adhesion. We show that ADAP inte

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Mazzi, Stefania. "Study of the role of the methyltransferase EZH2 in normal and pathological megakaryopoiesis." Thesis, Sorbonne Paris Cité, 2018. https://theses.md.univ-paris-diderot.fr/MAZZI_Stefania_2_complete_20180926.pdf.

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Le processus qui aboutit à la formation de plaquettes est appelé mégacaryopoïèse. Les mégacaryocytes (MK) sont de grandes cellules de la moelle osseuse qui par fragmentation dans la circulation sanguine produisent des plaquettes. La régulation extrinsèque ou intrinsèque de ce processus a été largement étudiée. Cependant la régulation épigénétique reste mal connue bien que de nombreuses mutations dans des gènes de régulateurs épigénétiques soient retrouvées dans les hémopathies malignes de la lignée MK. En particulier des mutations du gène de la méthyltransférase EZH2, composant catalytique du

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Ye, Jieyu, and 叶洁瑜. "The role of platelet-derived molecules: PDGF and serotonin in the regulation of megakaryopoiesis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47244446.

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Investigations on platelet-derived growth factor (PDGF) and serotonin (5-HT), molecules stored in platelet granules, imply their potential effects in regulating megakaryopoiesis, which also intimates the existence of an autocrine and/or paracrine loop constructed by megakaryocytes/platelets and their granular constituents. In addition, numerous reports indicate that melatonin, a derivative from serotonin effectively enhances platelet counts in patients with thrombocytopenia. However, their exact roles on human megakaryocytes and the underlying mechanisms remain unknown. Present studies sh

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Fock, Ee-Ling Clinical School St George Hospital Faculty of Medicine UNSW. "Molecular regulation and enhancement of megakaryopoiesis and thrombopoiesis by the p45 subunit of NF-E2." Publisher:University of New South Wales. Clinical School - St George Hospital, 2008. http://handle.unsw.edu.au/1959.4/42885.

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Megakaryocytes (MKs) are a rare population of haematopoietic cells, which produce platelets. Platelet production is a complex process that is tightly regulated at the transcriptional level by lineage specific transcription factors such as p45 NF-E2. Understanding how transcriptional regulators operate is imperative to advance our knowledge of disease pathophysiology and to propose novel treatment options. Therefore, the aims of this study were to: i) study the effects of p45 NF-E2 overexpression on various stages of megakaryopoiesis; (ii) elucidate the nuclear transport mechanisms of p45 NF-E2

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Chen, Jianliang, and 陈健良. "The inhibitory effects of human cytomegalovirus on megakaryopoiesis : megekaryocytic cells and bone marrow derived mesenchymal stormal cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/193520.

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Thrombocytopenia is one of the most common hematologic presentations of active human cytomegalovirus (HCMV) infection, especially in recipients of allogeneic hematopoietic stem cell transplantations and newborns of congenital HCMV infection. However, mechanisms of HCMV-induced thrombocytopenia have not been well understood. The precursor of circulating platelets – megakaryocyte, is derived from hematopoietic stem/progenitor cell in bone marrow. We postulate that inhibition to megakaryocytic development is the major pathogenesis of HCMV-induced thrombocytopenia. Megakaryocytic cells as well as

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El, khoury Mira. "Rôle de la calréticuline dans les néoplasmes myéloprolifératifs." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCC227.

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Les néoplasmes myéloprolifératifs (NMPs) classiques BCR-ABL négatifs regroupent la Polyglobulie de Vaquez, la Thrombocytémie Essentielle et la Myélofibrose Primaire. Ce sont des pathologies malignes clonales entraînées par la signalisation constitutive de la voie JAK2/STAT en raison de mutations somatiques acquises qui affectent trois gènes, JAK2, CALR et MPL. Il s’agit des mutations “motrices” de la maladie responsable du syndrome myéloprolifératif et du phénotype. Cependant CALR n’est pas une molécule de signalisation mais une chaperonne du réticulum endoplasmique. En utilisant des lignées d

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Części książek na temat "Megakaryopoiesis"

Kowata, Shugo, and Yoji Ishida. "Megakaryopoiesis and Thrombopoiesis." In Autoimmune Thrombocytopenia, 9–19. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-4142-6_2.

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Italiano, Joseph E. "Megakaryopoiesis and Platelet Biogenesis." In Molecular and Cellular Biology of Platelet Formation, 3–22. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39562-3_1.

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Kaushansky, K. "Thrombopoietin: Biological Effects Beyond Megakaryopoiesis." In Cytokines and Growth Factors in Blood Transfusion, 161–64. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4613-1137-9_16.

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Dame, Christof, Viola Lorenz, and Martha Sola-Visner. "Fetal and Neonatal Megakaryopoiesis and Platelet Biology." In Molecular and Cellular Biology of Platelet Formation, 267–91. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39562-3_12.

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Rey, Lisette Johana Latorre, and Ute Modlich. "Strategies for the Gene Modification of Megakaryopoiesis and Platelets." In Molecular and Cellular Biology of Platelet Formation, 421–60. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39562-3_20.

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Yu, Ming, and Alan B. Cantor. "Megakaryopoiesis and Thrombopoiesis: An Update on Cytokines and Lineage Surface Markers." In Methods in Molecular Biology, 291–303. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-61779-307-3_20.

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Freson, Kathleen. "Insights in Megakaryopoiesis and Platelet Biogenesis from Studies of Inherited Thrombocytopenias." In Molecular and Cellular Biology of Platelet Formation, 307–26. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39562-3_14.

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Varga, Eszter, Marten Hansen, Emile van den Akker, and Marieke von Lindern. "Erythropoiesis and Megakaryopoiesis in a Dish." In Cell Culture. IntechOpen, 2019. http://dx.doi.org/10.5772/intechopen.80638.

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Águila, Sonia, Ernesto Cuenca-Zamora, Constantino Martínez, and Raúl Teruel-Montoya. "MicroRNAs in Platelets: Should I Stay or Should I Go?" In Platelets. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.93181.

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In this chapter, we discuss different topics always using the microRNA as the guiding thread of the review. MicroRNAs, member of small noncoding RNAs family, are an important element involved in gene expression. We cover different issues such as their importance in the differentiation and maturation of megakaryocytes (megakaryopoiesis), as well as the role in platelets formation (thrombopoiesis) focusing on the described relationship between miRNA and critical myeloid lineage transcription factors such as RUNX1, chemokines receptors as CRCX4, or central hormones in platelet homeostasis like TPO, as well as its receptor (MPL) and the TPO signal transduction pathway, that is JAK/STAT. In addition to platelet biogenesis, we review the microRNA participation in platelets physiology and function. This review also introduces the use of miRNAs as biomarkers of platelet function since the detection of pathogenic situations or response to therapy using these noncoding RNAs is getting increasing interest in disease management. Finally, this chapter describes the participation of platelets in cellular interplay, since extracellular vesicles have been demonstrated to have the ability to deliver microRNAs to others cells, modulating their function through intercellular communication, redefining the extracellular vesicles from the so-called “platelet dust” to become mediators of intercellular communication.

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Streszczenia konferencji na temat "Megakaryopoiesis"

Saultier, P., S. Cabantous, M. Puceat, F. Peiretti, N. Saut, JC Bordet, M. Canault, et al. "GATA1 pathogenic variants disrupt MYH10 silencing during megakaryopoiesis." In 65th Annual Meeting of the Society of Thrombosis and Haemostasis Research. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1728206.

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Pindur, G., E. Seifried, and H. Rasche. "FIBRIN DEPOSITS IN BONE MARROW AND CHANGES IN HAEMOPOIESIS AFTER ENDOTOXIN ADMINISTRATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644256.

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Thrombotic occlusion of microcirculation during DIC has already been studied in numerous organs, but little is known about analogous findings in the bone marrow. Therefore in rats under the influence of endotoxin the defibrination was examined for its relationship to changes inthe bone marrow and haemopoiesis. Bone marrow specimens were studied histologically by fibrin staining methods. Blood cells were measured automatically on the Coulter counter, fibrinogen by clotting assay. A fall in the thrombocyte and fibrinogen level was induced through a single injection of endotoxin with a maximum af

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