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Rozprawy doktorskie na temat „Metabolic syndrome mechanism”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Morris, Tiffany J. "Investigating the molecular mechanisms of the metabolic syndrome." Thesis, University of Cambridge, 2010. https://www.repository.cam.ac.uk/handle/1810/237703.

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This thesis aims to highlight molecular mechanisms that have been altered by prenatal undernutrition and may be involved in the metabolic syndrome. Two separate studies were conducted both using a rat model developed through manipulation of the maternal diet to provoke the key features of the metabolic syndrome in adult offspring. Microarray technology was used to detect changes in gene expression in target tissues between offspring of control (normally fed, AD) and undernourished (UN) mothers to obtain a broader picture of the cellular functions and genetic pathways that may be implicated in

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Crendal, Edward. "Myocardial mechanics in metabolic syndrome and aging populations." Thesis, Australian Catholic University, 2013. https://acuresearchbank.acu.edu.au/download/bdb5ee628a2c89f8e75680d9240bdfaaaf14763b565cd0e9e523c93185e5364e/3074279/CRENDAL_EDWARD_2013.pdf.

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Cardiovascular disease (CVD) is the number one cause of death worldwide. The clustering of metabolic risk factors, defined as metabolic syndrome (MetS), and aging are two separate factors that significantly increase the risk of CVD. Both aging and MetS influence CVD by impairing heart tissue (myocardial) function. However, the exact understanding of mechanisms and evolution of these impairments are incomplete in MetS and aging populations. One of the most accessible and utilised techniques for assessing myocardial function is conventional transthoracic echocardiography, and more recently speck

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Rajkhowa, Madhurima. "Insulin resistance in polycystic ovary syndrome mechanisms and the metabolic consequences." Thesis, Keele University, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.242286.

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Malan, Leoné. "Cardiovascular dysfunction and specific coping mechanisms in Africans / L. Malan." Thesis, North-West University, 2005. http://hdl.handle.net/10394/767.

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Motivation: Cardiovascular dysfunction and hypertension are some of the leading causes of morbidity and mortality in the African population. According to the World Health Organisation the increases in these diseases are escalating in developing countries. Apart from the contributory role of genetics towards the incidence of hypertension, evidence regarding lifestyle as a determinant or marker of cardiovascular diseases in this group is not well known. The interaction of psychological and physiological mechanisms can contribute towards a broader scope of behavioural physiology in the higher pre

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Palacios, Verdú María Gabriela 1983. "Analysis of the evolving clinical phenotype and molecular mechanisms in Williams-Beuren syndrome." Doctoral thesis, Universitat Pompeu Fabra, 2015. http://hdl.handle.net/10803/324646.

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Williams-Beuren syndrome is a neurocognitive disorder with multisystemic manifestations of variable expressivity caused by a 1.55-1.83 Mb at chromosomal band 7q11.23. In this thesis project we have characterized the 7q11.23 deletion to refine the breakpoint location of the deletion, identify hospots and search for putative cis and trans-acting mechanisms involved. We have also characterized novel and previously reported metabolic alterations in a cohort of patients and explored its association with putative candidate genes using different mouse models and gene association analyses. Finally, we

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6

Iglesias, Miguel Angel University of New South Wales /. Garvan Institute of Medical Research Physiology &amp Pharmacology UNSW. "Mechanisms of amelioration of lipid-induced insulin resistance: role of AMP-activated protein kinase." Awarded by:University of New South Wales / Garvan Institute of Medical Research. Physiology and Pharmacology, 2004. http://handle.unsw.edu.au/1959.4/22391.

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Insulin resistance is an early marker of Type II diabetes. Excessive lipid accumulation in muscle and liver leads to insulin resistance, and lowering tissue lipids causes an enhancement of insulin action. The enzyme AMP-activated protein kinase (AMPK) is activated when cellular energy levels are compromised, such as during exercise; this enhances fuel oxidation and inhibits energy consuming processes. The hypothesis in this thesis was that activating AMPK in a lipid-induced insulin resistant state leads to tissue lipid reduction and improved insulin sensitivity. Insulin resistant high-fat fed

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7

Thomas, Amandine. "Hypoxie intermittente et homéostasie glucidique : étude des mécanismes d'action cellulaire A hybrid model to study pathological mutations of the human ADP/ATP carriers Visceral white fat remodeling contributes to intermittent hypoxia-induced atherogenesis The insulin sensitizing effect of topiramate involves KATP channel activation in the central nervous system The Impact of Sleep Disorders on Glucose Metabolism: Endocrine and Molecular Mechanisms Endoplasmic reticulum stress as a novel inducer of hypoxia inducible factor-1 activity: its role in the susceptibility to myocardial ischemia-reperfusion induced by chronic intermittent hypoxia Chronic intermittent hypoxia improves whole-body glucose tolerance by activating skeletal muscle AMP-activated protein kinase in mice Prolyl-4-hydroxylase 1 (PHD1) deficiency impairs whole-body glucose tolerance and insulin sensitivity in mice but does not worsen high-fat diet-induced metabolic dysfunctions Specific transcriptomic signature in response to intermittent hypoxia exposure in liver and fat tissue." Thesis, Université Grenoble Alpes (ComUE), 2015. http://www.theses.fr/2015GREAV044.

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L'hypoxie intermittente (HI), induite par les apnées du sommeil, conduit à des altérations de la sensibilité à l'insuline et de l'homéostasie glucidique mais les mécanismes impliqués restent mal connus. L'objectif de ce travail était d'étudier les effets et les mécanismes sous jacents d'une exposition chronique à l'HI sur l'homéostasie glucidique. L'HI induit une résistance à l'insuline à la fois systémique et tissulaire, ainsi qu'une amélioration de la tolérance au glucose associée à une activation de l'AMPK musculaire. L'HI cause également des altérations du foie et du tissu adipeux associée

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Dharmalingam, Backialakshmi [Verfasser]. "Immune mediated disturbances of bone, connective tissue and vascular metabolism in Complex Regional Pain Syndrome (CRPS) : a new pathogenic mechanism of therapeutic relevance / Backialakshmi Dharmalingam." Gießen : Universitätsbibliothek, 2015. http://d-nb.info/1076980287/34.

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Nduhirabandi, Frederic. "The role of melatonin in cardioprotection : an investigation into the mechanisms involved in glucose homeostasis, microvascular endothelial function and mitochondrial function in normal and insulin resistant states." Thesis, Stellenbosch : Stellenbosch University, 2014. http://hdl.handle.net/10019.1/86332.

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Thesis (PhD)-- Stellenbosch University, 2014.<br>ENGLISH ABSTRACT: Introduction: The cardioprotective actions of the hormone melatonin against myocardial ischaemiareperfusion injury (IRI) are well-established. It has recently been shown to prevent the harmful effects of hyperphagia-induced obesity on the susceptibility of the heart to IRI as well as many of the harmful effects of obesity and insulin resistance. However, the exact mechanism whereby it exerts its beneficial action is still unknown. The aims of this study were to determine the effects of relatively short-term melatonin tre

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Mesquita, Maria do Céu Vinhas Sérgio Vilão. "Síndrome metabólica em infetados pelo vírus da imunodeficiência humana." Master's thesis, [s.n.], 2014. http://hdl.handle.net/10284/4841.

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Projeto de Pós-Graduação/Dissertação apresentado à Universidade Fernando Pessoa como parte dos requisitos para obtenção do grau de Mestre em Ciências Farmacêuticas<br>A Síndrome metabólica consiste num conjunto de fatores de risco (obesidade abdominal, hipertriglicemia, diminuição do colesterol de alta densidade, hipertensão e hiperglicemia) que predispõem o indivíduo para doenças do foro cardiovascular. O facto da literatura frequentemente associar os indivíduos infetados com o vírus da imunodeficiência humana a um risco aumentado de hipertensão, hiperlipidemia e diabetes, torna importante i

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11

Green, Thomas James. "Investigation into the mechanism and treatment of atherogenic dyslipidemia in HIV + patients with HIV metabolic syndrome." Thesis, 2004. http://hdl.handle.net/2429/15246.

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The use of highly active combination anti-retroviral therapy is associated with serious metabolic side effects including increased serum triglycerides, decreased HDL cholesterol as well as insulin resistance, peripheral fat loss and gain of abdominal visceral fat. There is concern that anti-retroviral therapy imposes a significant risk of heart disease in the HIV positive population. We hypothesize that development of dyslipidemia in response to anti-retroviral therapy is associated with the presence of specific genetic polymorphisms and may relate to changes in lipoprotein lipase and hepatic

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Chen, Gou-Chun, and 陳姤君. "The inducing effect and action mechanism of a high fat diet and the sucrose-containing drinking water on metabolic syndrome." Thesis, 2008. http://ndltd.ncl.edu.tw/handle/49045533852325269126.

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碩士<br>中國醫藥大學<br>營養學系<br>96<br>Metabolic syndrome is a global healthy problem with a soaring morbidity. In Taiwan, a dietary patterm change with an increased high fat diet or/and sucrose-sweetened beverage consumption, accompanies with an absence of exercise are considered to be the risk factors of metabolic syndrome. This study was designed to compare the effect of a high fat diet or / and sucrose-containing drinking water on inducing metabolic syndrome and the possible molecular mechanisms involved were also studied. Male C57BL6J mice and Wistar rats were each divided into one control (chow d

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Lopes, Ricardo José Araújo Ladeiras. "Cardiovascular Impact of Metabolic Syndrome: From Mechanisms to New Treatment Targets." Doctoral thesis, 2020. https://hdl.handle.net/10216/131024.

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Lopes, Ricardo José Araújo Ladeiras. "Cardiovascular Impact of Metabolic Syndrome: From Mechanisms to New Treatment Targets." Tese, 2020. https://hdl.handle.net/10216/131024.

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Lin, Chih-Li. "The study of tea polyphenols preventive mechanisms on Alzheimer's disease and metabolic syndrome." 2007. http://www.cetd.com.tw/ec/thesisdetail.aspx?etdun=U0001-1207200703020700.

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Lin, Chih-Li, and 林志立. "The study of tea polyphenols preventive mechanisms on Alzheimer''s disease and metabolic syndrome." Thesis, 2007. http://ndltd.ncl.edu.tw/handle/72541447662212192514.

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博士<br>國立臺灣大學<br>生物化學暨分子生物學研究所<br>95<br>Tea is the most consumed drink in the world containing many compounds particularly polyphenols shown to reduce the risk of a variety of diseases in epidemiological studies. In the present study, we demonstrated the possible preventive mechanisms of tea polyphenols on Alzheimer''s disease and metabolic syndrome model. In part I, the results deminstrated that treatment with EGCG reduced the Aβ levels by enhancing endogenous APP nonamyloidogenic proteolytic processing in Aβ-induced AD model both in vitro and in vivo. Moreover, our results indicated that the

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Tai, Huai-Ching, and 戴槐青. "Metabolic Syndrome, Bladder Ischemia and Lower Urinary Tract Symptoms: The Epidemiological Evidences and Pathophysiological Mechanisms." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/37z34c.

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博士<br>國立臺灣大學<br>臨床醫學研究所<br>105<br>Background Lower urinary tract symptoms (LUTS) and metabolic syndrome (MS) are both highly prevalent public health problems, especially in the elderly population. Many recent epidemiological evidences have shown that MS is one of the possible factors for the development of LUTS. In the Third National Health and Nutrition Examination Survey conducted on men > 60 years, the risks of having LUTS increased significantly in men with ≥ 3 MS components when compared with their control counterparts (OR 1.80; 95% CI 1.11–2.94). Men with MS also had a higher growth rate

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Silva, Daniela Maria Miranda da. "The Heart under Pressure: Mechanisms underlying HEpEF Secondary to chronic pressure Overload or Metabolic Syndrome." Doctoral thesis, 2019. https://hdl.handle.net/10216/125424.

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Silva, Daniela Maria Miranda da. "The Heart under Pressure: Mechanisms underlying HEpEF Secondary to chronic pressure Overload or Metabolic Syndrome." Tese, 2019. https://hdl.handle.net/10216/125424.

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Burt, Morton Garth St Vincent's Clinical School UNSW. "Mechanisms underlying glucocorticoid-induced protein wasting and potential treatment with anabolic hormoness." 2007. http://handle.unsw.edu.au/1959.4/41293.

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Protein wasting is a complication of glucocorticoid (GC) therapy. It causes substantial morbidity and there is no treatment. This thesis investigates the metabolic mechanisms underlying GC-induced protein wasting and the potential for anabolic hormones to reverse protein loss. The models of GC excess were Cushing's syndrome and GC therapy. Whole body protein metabolism was assessed using the leucine turnover technique and body composition by dual-energy X-ray absorptiometry to estimate lean body mass (LBM) and fat mass (FM). As previous studies demonstrated that LBM and FM influenced rates of

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Silva, Daniela Rosendo da. "The gut-adipose tissue crosstalk: unravelling the mechanisms of ghrelin-mediated regulation of adipose tissue angiogenesis." Master's thesis, 2020. http://hdl.handle.net/10316/93881.

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Dissertação de Mestrado em Biologia Celular e Molecular apresentada à Faculdade de Ciências e Tecnologia<br>Introdução e Objetivos: A disfunção do tecido adiposo e a desregulação da secreção de hormonas intestinais constituem dois dos principais pilares da síndrome metabólica e da diabetes mellitus tipo 2. Terapêuticas que têm como base a remodelação do trato gastrointestinal resultam numa melhoria substancial da função do tecido adiposo, ao mesmo tempo que induzem a normalização do perfil de secreção das hormonas intestinais, nomeadamente da grelina, uma hormona associada à fome e à ativação

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Weiner, Juliane, Konstanze Zieger, Jan Pippel, and John T. Heiker. "Molecular mechanisms of vaspin action: from adipose tissue to skin and bone, from blood vessels to the brain." 2020. https://ul.qucosa.de/id/qucosa%3A38022.

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Visceral adipose tissue derived serine protease inhibitor (vaspin) or SERPINA12 according to the serpin nomenclature was identified together with other genes and gene products that were specifically expressed or overexpressed in the intra abdominal or visceral adipose tissue (AT) of the Otsuka Long-Evans Tokushima fatty rat. These rats spontaneously develop visceral obesity, insulin resistance, hyperinsulinemia and ‐glycemia, as well as hypertension and thus represent a well suited animal model of obesity and related metabolic disorders such as type 2 diabetes. The follow-up study reporti

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