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Gotowe bibliografie tematyczne / Metastasis and autophagy

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Artykuły w czasopismach
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Gotowa bibliografia na temat „Metastasis and autophagy”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Metastasis and autophagy"

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Xu, Funeng, Xilin Li, Xuehui Huang, Jingmei Pan, Yi Wang, and Shaobing Zhou. "Development of a pH-responsive polymersome inducing endoplasmic reticulum stress and autophagy blockade." Science Advances 6, no. 31 (2020): eabb8725. http://dx.doi.org/10.1126/sciadv.abb8725.

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Autophagy is involved in the occurrence and development of tumors. Here, a pH-responsive polymersome codelivering hydroxychloroquine (HCQ) and tunicamycin (Tuni) drugs is developed to simultaneously induce endoplasmic reticulum (ER) stress and autophagic flux blockade for achieving an antitumor effect and inhibiting tumor metastasis. The pH response of poly(β-amino ester) and HCQ synergistically deacidifies the lysosomes, thereby blocking the fusion of autophagosomes and lysosomes and lastly blocking autophagic flux. The function mechanism of regulating autophagy was systematically investigate

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Sahebi, Reza, Nazanin Akbari, Zeynab Bayat, Mohammad Rashidmayvan, Amin Mansoori, and Maria Beihaghi. "A Summary of Autophagy Mechanisms in Cancer Cells." Research in Biotechnology and Environmental Science 1, no. 1 (2022): 28–35. http://dx.doi.org/10.58803/rbes.2022.1.1.06.

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Autophagy is a well-known vital process in cells and plays a significant role in biological evolution, the immune system, and cell death. It can be effective in fatal disorders, such as nervous system degeneration, autoimmune diseases, and cancer. Autophagy has a dual role; on the one hand, it increases cell survival, and on the other hand, it causes cell death in advanced stages although no agreement has yet been accomplished on the role of autophagy in cellular processes. There is evidence that autophagic signaling regulation is inversely related to oncogenic signaling. Numerous commonly act

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Li, Zhennan, Cheng Lu, Fengliang Wang та ін. "Heat treatment-induced autophagy promotes breast cancer cell invasion and metastasis via TGF-β2-mediated epithelial-mesenchymal transitions". PeerJ 11 (12 січня 2023): e14640. http://dx.doi.org/10.7717/peerj.14640.

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Background Insufficient thermal ablation can accelerate malignant behaviors and metastases in some solid tumors, and epithelial-mesenchymal transition (EMT) and autophagy are involved in tumor metastasis. It has been found that TGF-β2 which belongs to the family of transforming growth factors often associated with cancer cell invasiveness and EMT. However, whether the interactions between autophagy and TGF-β2 induce EMT in breast cancer (BC) cells following insufficient microwave ablation (MWA) remains unclear. Methods BC cells were treated with sublethal heat treatment to simulate insufficien

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Denisenko, Tatiana, Anastasia Pivnyuk, and Boris Zhivotovsky. "p53-Autophagy-Metastasis Link." Cancers 10, no. 5 (2018): 148. http://dx.doi.org/10.3390/cancers10050148.

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Mowers, E. E., M. N. Sharifi, and K. F. Macleod. "Autophagy in cancer metastasis." Oncogene 36, no. 12 (2016): 1619–30. http://dx.doi.org/10.1038/onc.2016.333.

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Zhang, Huilin, and Bingjian Lu. "The Roles of ceRNAs-Mediated Autophagy in Cancer Chemoresistance and Metastasis." Cancers 12, no. 10 (2020): 2926. http://dx.doi.org/10.3390/cancers12102926.

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Chemoresistance and metastasis are the main causes of treatment failure and unfavorable outcome in cancers. There is a pressing need to reveal their mechanisms and to discover novel therapy targets. Autophagy is composed of a cascade of steps controlled by different autophagy-related genes (ATGs). Accumulating evidence suggests that dysregulated autophagy contributes to chemoresistance and metastasis via competing endogenous RNA (ceRNA) networks including lncRNAs and circRNAs. ceRNAs sequester the targeted miRNA expression to indirectly upregulate ATGs expression, and thereof participate in au

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Bhattacharyya, Shalmoli, Renaissa De, and Radhika Srinivasan. "Abstract A031: Combination of hedgehog targeting and autophagy inhibitor potentiate cytotoxicity in 3D spheroids of cervical cancer: A promising therapeutic strategy in metastatic disease." Cancer Research 83, no. 2_Supplement_2 (2023): A031. http://dx.doi.org/10.1158/1538-7445.metastasis22-a031.

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Abstract Cervical cancer is the second most common malignancy in women and a major cause of morbidity and mortality worldwide. Statistics show that present biologic therapy has had little impact on survival in recurrent and metastatic disease. Consequently, there is an urgent need to explore the best treatment options for advanced cervical cancer. Hedgehog (Hh) signaling pathway has been shown to play an important role in metastasis, recurrence and drug resistance of cervical cancer. Therefore, inhibition of hedgehog signaling pathway is emerging as a new strategy for the treatment of advanced

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Radovanovic, Milan, Sasenka Vidicevic, Jelena Tasic, et al. "Role of AMPK/mTOR-independent autophagy in clear cell renal cell carcinoma." Journal of Investigative Medicine 68, no. 8 (2020): 1386–93. http://dx.doi.org/10.1136/jim-2020-001524.

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We examined the status and role of autophagy, a process of lysosomal recycling of cellular material, in clear cell renal cell carcinoma (ccRCC). Paired samples of tumor and adjacent non-malignant tissue were collected from 20 patients with ccRCC after radical nephrectomy. The mRNA levels of apoptosis (BAD, BAX, BCL2, BCLXL, BIM) and autophagy (ATG4, BECN1, GABARAP, p62, UVRAG) regulators were measured by RT-qPCR. The protein levels of autophagosome-associated LC3-II, autophagy receptor p62, apoptotic marker PARP, as well as phosphorylation of autophagy initiator Unc 51-like kinase 1 (ULK1), it

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Kim, Hye Min, and Ja Seung Koo. "The Role of Autophagy in Breast Cancer Metastasis." Biomedicines 11, no. 2 (2023): 618. http://dx.doi.org/10.3390/biomedicines11020618.

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Patient morbidity and mortality is significantly increased in metastatic breast cancer. The metastasis process of breast cancer is very complicated and is delicately controlled by various factors. Autophagy is one of the important regulatory factors affecting metastasis in breast cancer by engaging in cell mobility, metabolic adaptation, tumor dormancy, and cancer stem cells. Here, we discuss the effects of autophagy on metastasis in breast cancer and assess the potential use of autophagy modulators for metastasis treatment.

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Yao, Jiaqi, Chi Ma, Kaixuan Feng, Guang Tan, and Qingping Wen. "Focusing on the Role of Natural Products in Overcoming Cancer Drug Resistance: An Autophagy-Based Perspective." Biomolecules 12, no. 11 (2022): 1565. http://dx.doi.org/10.3390/biom12111565.

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Autophagy is a critical cellular adaptive response in tumor formation. Nutritional deficiency and hypoxia exacerbate autophagic flux in established malignancies, promoting tumor cell proliferation, migration, metastasis, and resistance to therapeutic interventions. Pro-survival autophagy inhibition may be a promising treatment option for advanced cancer. Furthermore, excessive or persistent autophagy is cytotoxic, resulting in tumor cell death. Targeted autophagy activation has also shown significant promise in the fight against tumor drug resistance. Several research groups have examined the

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Rozprawy doktorskie na temat "Metastasis and autophagy"

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Flynn, Alyssa La Belle. "6 Phosphofructo-2-Kinase/Fructose-2,6-Biphosphatase 3 (pfkfb3): A Critical Mediator of Breast Cancer Metastasis and Escape from Dormancy." Case Western Reserve University School of Graduate Studies / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case157271391725048.

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Görgülü, Kivanc [Verfasser], Hana [Akademischer Betreuer] Algül, Roland M. [Gutachter] Schmid, Julia [Gutachter] Mayerle, and Stephan [Gutachter] Herzig. "Role of Autophagy-Related 5 Gene in Pancreatic Cancer and Metastasis / Kivanc Görgülü ; Gutachter: Roland M. Schmid, Julia Mayerle, Stephan Herzig ; Betreuer: Hana Algül." München : Universitätsbibliothek der TU München, 2019. http://d-nb.info/1181946840/34.

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Gugnoni, Mila <1988&gt. "Cadherin 6 regulates Epithelial Mesenchymal Transition by restraining autophagy and controlling metabolism in metastatic thyroid cancer." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2016. http://amsdottorato.unibo.it/8275/1/Tesi%20Mila%20Gugnoni.pdf.

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The transdifferentiation of epithelial cells toward a mesenchymal phenotype(EMT) is a multi-step process fundamental for tumor cells to leave the primary lesion and colonize ectopic sites. Cadherins are structural proteins that also transduce extracellular signals regulating many cellular pathways but the molecular mechanisms guiding this function is poorly understood. Cadherin-6(CDH6) is a type-2 cadherin which drives EMT during development and is re-expressed in some tumors. In thyroid cancer, CDH6 is a target of TGFβ signaling and a marker of EMT, suggesting a role for this protein in the p

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Kapoor, Somya. "The role of cellular redox imbalance, ER-stress, and autophagy in adaptation of metastatic melanoma to MAPK pathway inhibition." Diss., University of Iowa, 2018. https://ir.uiowa.edu/etd/6597.

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Melanoma incidence in the United States has grown continuously at a rate of 1.5% each year for the last decade. Disease detected early can be cured by surgery, but metastatic disease is lethal. Recent discoveries have led to promising-targeted MAPK-pathway inhibitors (MAPKih) and immunotherapies. However, low response rates and acquired drug resistance remain as significant challenges to improved outcomes. The mechanisms that drive resistance to MAPKih are elusive. On the other hand, our data suggests that drug-induced alterations in oxidative metabolism and cellular antioxidant systems (e.g.,

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Verykiou, Stamatina. "Cross talk between CD271 expressing tumour subpopulations and autophagy in the drug resistance, invasion and survival of cutaneous metastatic melanoma." Thesis, University of Newcastle upon Tyne, 2017. http://hdl.handle.net/10443/3932.

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Cutaneous malignant melanoma remains an increasing world health problem. Although targeted therapy to hyper-activated MAPK signalling has significantly increased survival for patients with BRAF/NRAS mutant melanomas, the development of acquired resistance is inevitable and associated with the emergence of chemo-resistant subpopulations expressing the neurotrophin receptor CD271. The relationship between drug-induced CD271 expressing subpopulations and autophagy however, remains undefined. The central aim of the present study was to define the relationship between CD271 (both constitutive and d

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Hsin, Min-Chieh, and 辛旻潔. "The Mechanisms of Hispolon in Autophagy Induction and Metastasis Inhibition in Human Cervical Cancer Cells." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/f6v972.

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博士<br>中山醫學大學<br>醫學研究所<br>106<br>Hispolon, a phenolic compound isolated from Phellinus igniarius, induces apoptosis and anti-tumor effects in cancers. However, the molecular mechanism involved in hispolon-mediated tumor-suppressing activities observed in cervical cancer is poorly characterized. Here, we demonstrated that treatment with hispolon inhibited cell metastasis in two cervical cancer cell lines. In addition, the downregulation of the lysosomal protease Cathepsin S (CTSS) was critical for hispolon-mediated suppression of tumor cell metastasis in both in vitro and in vivo models. Moreove

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Huang, Jyun-Ying, and 黃俊穎. "Isokotomolide A and Secokotomolide A from Cinnamomum kotoense induce autophagy and apoptosis and inhibit melanoma metastasis in vivo and in vitro." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/x2f32v.

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碩士<br>國立中興大學<br>生醫工程研究所<br>106<br>Melanoma is an aggressive cancer with high lethality, to find new anticancer agent, we identified isokotomolide A and secokotomolide A isolated from Cinnamomum kotoense to be potential bioactive agents against human melanoma but none anti-oxidant. Cell proliferation assay displayed isokotomolide A and secokotomolide A treated in the normal human skin cells showed high viabilities. It also verified both two of them possess strong anti-melanoma effect in dose-dependent manners, especially on B16F10, A2058, MeWo and A375 cells. Wound healing assay presented their

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Kumar, Hindupur Sravanth. "Stress Signaling In Development And Carcinogenesis : Role Of AMP-Activated Protein Kinase." Thesis, 2011. https://etd.iisc.ac.in/handle/2005/2189.

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Rapidly growing tumor cells outgrow their blood supply resulting in a microenvironment with reduced oxygen and nutrients. Using an in vitro transformation model we found that cancer cells expressing the SV40 ST antigen (+ST cells) are more resistant to glucose deprivation-induced cell death than cells lacking the SV40 ST antigen (−ST cells). Mechanistically, we found that the ST antigen mediates this effect by activating a nutrient-sensing kinase, AMP-activated protein kinase (AMPK). We further show that AMPK mediates its effects, at least in part, by inhibiting mTOR (mammalian target of rapam

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Kumar, Hindupur Sravanth. "Stress Signaling In Development And Carcinogenesis : Role Of AMP-Activated Protein Kinase." Thesis, 2011. http://etd.iisc.ernet.in/handle/2005/2189.

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Rapidly growing tumor cells outgrow their blood supply resulting in a microenvironment with reduced oxygen and nutrients. Using an in vitro transformation model we found that cancer cells expressing the SV40 ST antigen (+ST cells) are more resistant to glucose deprivation-induced cell death than cells lacking the SV40 ST antigen (−ST cells). Mechanistically, we found that the ST antigen mediates this effect by activating a nutrient-sensing kinase, AMP-activated protein kinase (AMPK). We further show that AMPK mediates its effects, at least in part, by inhibiting mTOR (mammalian target of rapam

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Części książek na temat "Metastasis and autophagy"

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Hou, Jing, Zhipeng Han, Naping Zhao, and Lixin Wei. "Autophagy and Tumour Metastasis." In Autophagy: Biology and Diseases. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-4272-5_22.

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Polara, Ruhi, Daphni van Rinsum, and Nirmal Robinson. "Autophagy in Cancer Metastasis." In Autophagy in Stem Cell Maintenance and Differentiation. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-17362-2_11.

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Das, Joyjyoti, and Tapas Kumar Maiti. "Mechanical Stress-Induced Autophagy: A Key Player in Cancer Metastasis." In Autophagy in tumor and tumor microenvironment. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-6930-2_8.

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Bindal, Priya, Rohit Kumar, Shumaila Khan, et al. "Role of Autophagy and Oxidative Stress in Cancer Metastasis." In Cancer Drug Discovery and Development. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-66421-2_8.

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Arora, Sandeep, and Sukhbir Singh. "Autophagy-Modulating Drugs as Complements to Cancer Chemotherapy." In Metastatic Diseases. Apple Academic Press, 2021. http://dx.doi.org/10.1201/9781003043249-3.

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Sloan, Philip. "The Bi-Directional Communication Between Tumour Cells and Other Components of the Tumour Microenvironment." In Critical Issues in Head and Neck Oncology. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-23175-9_1.

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AbstractCross talk between cancer cells and their microenvironment can lead to molecular changes in adjacent tissues that can be utilised as biomarkers. One such example stems from the discovery that malignant melanomas with metastatic potential can downregulate autophagy in the overlying epidermis. Autophagy is essential for normal keratinocyte maturation. AMBRA1 is a key autophagy regulatory molecule and its expression in the epidermis is reduced in the epidermis overlying some early stage melanomas. Expression of AMBRA1 is maintained in the overlying epidermis in a subset of low risk melano

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Hashemi, Mehrdad, Najma Farahani, Ali Moghadas Jafari, et al. "Autophagy in the Regulation of Cancer Metastasis." In Autophagy, Apoptosis and Ferroptosis in Oncology. WORLD SCIENTIFIC, 2025. https://doi.org/10.1142/9789811299322_0004.

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Anto, Nikhil Ponnoor, Amitha Muraleedharan, and Rashmi Mittal. "Molecular Sub-Typing and Exploration of Key Signalling Pathways Involved in Complicating the Disease." In Therapeutic Drug Targets and Phytomedicine For Triple Negative Breast Cancer. BENTHAM SCIENCE PUBLISHERS, 2023. http://dx.doi.org/10.2174/9789815079784123010006.

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Triple-negative breast cancer is characterized by distinct molecular profiles, unique metastatic patterns, aggressive behavior, lacks the targeted therapeutic approach, and caused significant mortality worldwide. The molecular complexity of angiogenesis, autophagy, apoptosis, and metastasis process in TNBC has fostered research efforts to unleash the molecular, pathological, and genetic drivers of their lethal cascade. This complex disease entity involves PI3k/Akt/mTOR, NF-kB, ERRs, and miRNA trafficking which has further worsened the clinical outcome. Due to their heterogeneous nature, none o

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Ahmed, Mahmoud, and Deok Ryong Kim. "Anti-cancer effect of RKIP via modulating autophagy during metastasis." In Prognostic and Therapeutic Applications of RKIP in Cancer. Elsevier, 2020. http://dx.doi.org/10.1016/b978-0-12-819612-0.00015-8.

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I. Rushdi, Mohammed. "Quercetin: A Flavonoid with Diverse Chemo Preventive Properties against Cancer." In Quercetin - Effects on Human Health. IntechOpen, 2024. http://dx.doi.org/10.5772/intechopen.1004133.

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Quercetin, an exceptional and extraordinary flavonoid possessing bioactive properties, presents a plethora of benefits for the promotion of good health. The anti-tumor characteristics of quercetin have been well-documented in various in vitro and in vivo investigations, encompassing a wide range of cell lines and animal models. Quercetin, through the activation of caspase-3 and inhibition of the phosphorylation of Akt, mTOR, and ERK, as well as the reduction of β-catenin and stabilization of HIF-1α, augments apoptosis and autophagy in cancer. Additionally, quercetin curbs cancer cell metastasi

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Streszczenia konferencji na temat "Metastasis and autophagy"

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Lim, Maegan M., Ziqiang Teo, Chee Chong Choo, Pengcheng Zhu, and Nguan Soon Tan. "Abstract 1442: Understanding the role of autophagy during cancer metastasis." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-1442.

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Seok, Jin Kyung, Eun-Hee Hong, and Joo Young Lee. "Abstract PO037: Oxidized phosphatidylcholines induce tumorigenesis and metastasis through autophagy." In Abstracts: AACR Virtual Special Conference: The Evolving Tumor Microenvironment in Cancer Progression: Mechanisms and Emerging Therapeutic Opportunities; in association with the Tumor Microenvironment (TME) Working Group; January 11-12, 2021. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.tme21-po037.

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Maycotte, Paola, Jackie Thorburn, and Andrew Thorburn. "Abstract 4849: Role of autophagy in breast cancer metastasis and treatment." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-4849.

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Shen, Tong, Lingdong Cai, Yuhong Liu, and Jianming Li. "Abstract A48: Ube2v1 promotes EMT and metastasis by suppression of autophagy." In Abstracts: AACR Special Conference: Advances in Modeling Cancer in Mice: Technology, Biology, and Beyond; September 24-27, 2017; Orlando, Florida. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.mousemodels17-a48.

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Ndoye, Abibatou, Anna Budina, Marie Webster, et al. "Abstract 2906: Role of autophagy in Wnt5A-mediated melanoma invasion and metastasis." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-2906.

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Barnard, Rebecca A., Paola Maycotte, Ryan J. Hansen, Daniel L. Gustafson, and Andrew Thorburn. "Abstract C19: The effect of autophagy inhibition on anchorage-independent cell growth." In Abstracts: AACR Special Conference on Tumor Invasion and Metastasis - January 20-23, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tim2013-c19.

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Gomes, Luciana R., Alexandre T. Vessoni, and Carlos FM Menck. "Abstract A34: Microenvironment controls breast cancer cells sensitivity to doxorubicin by autophagy inhibition." In Abstracts: AACR Special Conference on Tumor Invasion and Metastasis - January 20-23, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tim2013-a34.

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Sharifi, Marina N., Christopher Collier, Lauren Drake, Hong Chen, Stephanie Mui, and Kay F. Macleod. "Abstract 321: Autophagy is required for focal adhesion turnover, tumor cell motility and metastasis." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-321.

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Ray, Upasana, Debarshi Roy, Ling Jin, et al. "Abstract 1936: Autophagy-dependent therapeutic targeting of group III phospholipase A2 attenuates ovarian cancer metastasis." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-1936.

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Cho, Tae Jin, Vok Hee Woo, Jeom Il Choi, et al. "Abstract B13:Porphyromonas gingivalis-induced autophagy suppresses cell proliferation through G1 arrest in oral cancer cells." In Abstracts: AACR Special Conference on Tumor Invasion and Metastasis - January 20-23, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tim2013-b13.

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