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Artykuły w czasopismach na temat „Methylphenidate interaction”

Autor: Grafiati

Data publikacji: 3 czerwca 2025

Data aktualizacji: 31 lipca 2025

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 746 (1999): 9. http://dx.doi.org/10.2165/00128415-199907460-00031.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 1173 (2007): 18. http://dx.doi.org/10.2165/00128415-200711730-00054.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 1181 (2007): 25. http://dx.doi.org/10.2165/00128415-200711810-00073.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 1136 (2007): 20. http://dx.doi.org/10.2165/00128415-200711360-00062.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 1152 (2007): 17. http://dx.doi.org/10.2165/00128415-200711520-00055.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 655 (1997): 10. http://dx.doi.org/10.2165/00128415-199706550-00032.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 803 (2000): 10. http://dx.doi.org/10.2165/00128415-200008030-00025.

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&NA;. "Methylphenidate/sertraline interaction." Reactions Weekly &NA;, no. 1200 (2008): 24. http://dx.doi.org/10.2165/00128415-200812000-00067.

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&NA;. "Methylphenidate/pseudoephedrine interaction." Reactions Weekly &NA;, no. 1303 (2010): 25. http://dx.doi.org/10.2165/00128415-201013030-00077.

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&NA;. "Disulfiram/methylphenidate interaction." Reactions Weekly &NA;, no. 1424 (2012): 24. http://dx.doi.org/10.2165/00128415-201214240-00079.

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&NA;. "Aripiprazole/methylphenidate/risperidone interaction." Reactions Weekly &NA;, no. 1348-1349 (2011): 8. http://dx.doi.org/10.2165/00128415-201113480-00023.

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Ghofrani, Mohammad. "Possible Phenytoin-methylphenidate Interaction." Developmental Medicine & Child Neurology 30, no. 2 (2008): 267–68. http://dx.doi.org/10.1111/j.1469-8749.1988.tb04763.x.

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Arda, Karagöl. "Panic Attack Following Addition of Nalmefene to Methylphenidate in a Patient with Comorbid Alcohol Use Disorder and Attention Deficit Hyperactivity Disorder: A Case Report." International Journal of Medical and Pharmaceutical Case Reports 9, no. 3 (2017): 1–5. https://doi.org/10.9734/IJMPCR/2017/34856.

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<strong>Aim: </strong>Our aim is to describe a previously unreported potential interaction of nalmefene with methylphenidate and discuss possible mechanisms and precautions. <strong>Presentation of Case:</strong> A 40-year-old man with ADHD taking long-acting methylphenidate developed alcohol use disorder. Nalmefene was prescribed. Following his first bedtime administration, he awoke with panic attack symptoms and concomitant suicidal and homicidal ideation. These symptoms resolved 45 minutes after he received diazepam. <strong>Discussion:</strong> Hypotheses for this reaction include a previo

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Tobajas, Yaiza, Marc Alemany-Fornés, Iris Samarra, et al. "Interaction of Diamino Oxidase with Psychostimulant Drugs for ADHD Management." Journal of Clinical Medicine 12, no. 14 (2023): 4666. http://dx.doi.org/10.3390/jcm12144666.

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Histamine intolerance occurs when there is an imbalance between histamine production and the capacity for histamine degradation. Diamine oxidase (DAO) is the main enzyme for the catabolism of ingested histamine degradation in the gastrointestinal tract and its deficiency has been linked to allergy-like symptoms. Psychostimulant drugs are commonly used to treat Attention Deficit Hyperactivity Disorder (ADHD), but their interaction with DAO is not well characterized. In this work, we evaluated the effects of psychostimulant drugs (methylphenidate and lisdexamfetamine) on in vitro DAO activity an

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Kimmick, Erica, Robin McGovern, and Alan Gittis. "Interaction of Methylphenidate and External Cue in an ADHD Behavioral Training Model." Journal of Student Research 1, no. 2 (2012): 79–86. http://dx.doi.org/10.47611/jsr.v1i2.83.

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Symptoms of Attention Deficit Hyperactive Disorder (ADHD) include hyperactivity, inattentiveness, and impulsivity, which are caused by dopamine dysfunction in the medial prefrontal cortex. A six-hydroxydopamine lesion model of ADHD was used to examine response accuracy on an attentional task under one of three conditions: cue only, methylphenidate only (MPH), and MPH with cue. Results indicated rats injected with methylphenidate and exposed to an external cue showed a significant increase in response accuracy compared to the MPH and cue only groups. Also, the results indicate animals in the cu

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Gomes, Karin M., Renan P. Souza, Cecília G. Inácio, et al. "Evaluation of light/dark cycle in anxiety- and depressive-like behaviors after regular treatment with methylphenidate hydrochloride in rats of different ages." Revista Brasileira de Psiquiatria 33, no. 1 (2010): 55–58. http://dx.doi.org/10.1590/s1516-44462010005000018.

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OBJECTIVE: Methylphenidate hydrochloride is the most widely used medication for treatment and management of attention-deficit hyperactivity disorder. However, the chronic effects of methylphenidate hydrochloride on anxiety- and depressive-like rat behaviors remain poorly investigated. In this context, the present study evaluated the effects of treatment with methylphenidate hydrochloride on anxiety- and depressive-like behaviors using young and adult rats during the light and the dark cycle. METHOD: Male Wistar rats (25 or 60 days old) received a once-daily (in either the light or dark cycle)

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Romano, Elisa, Raymond H. Baillargeon, Isabel Fortier, et al. "Individual Change in Methylphenidate Use in a National Sample of Children Aged 2 to 11 Years." Canadian Journal of Psychiatry 50, no. 3 (2005): 144–52. http://dx.doi.org/10.1177/070674370505000303.

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Objectives: To determine methylphenidate use in children aged 2 to 13 years. To provide age- and sex-specific estimates of methylphenidate initiation and cessation during a 2-year period. Method: Data from 2 cycles of a Canadian household survey yielded a sample of over 10 000 children aged 2 to 11 years at Cycle 1 who continued to participate at Cycle 2. We used logit modelling to estimate Cycle 2 methylphenidate use, methylphenidate use over a 2-year period, and methylphenidate initiation and cessation from Cycles 1 to 2. Results: In 1996 and 1997, methylphenidate use ranged from 0.32% to 6.

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Jadapalle, Sree Latha Krishna, Edwin McCray, John Azat Masoud, and Michael W. Kortz. "Methylphenidate-Induced Chorea Due to Possible Cytochrome P450 Metabolism Heterogeneity - A Rare Case." CNS Spectrums 26, no. 2 (2021): 176–77. http://dx.doi.org/10.1017/s1092852920002898.

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AbstractBackgroundChorea is defined as a hyperactive movement disorder associated with involuntary, quick, and unpredictable muscle contractions of the limbs, face, and trunk. The unpredictable nature of these movements includes variation in speed, timing, and direction of movement. A wide variety of medications, medical conditions and illicit drugs have been associated with movement disorders. Examples include a multitude of antipsychotic induced movement disorders and dyskinesia related to dopaminergic agents, like levodopa and metoclopramide. Dyskinesias have been associated with psycho-sti

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Cummins, Elizabeth D., Kristen K. Leedy, John M. Dose, et al. "The effects of adolescent methylphenidate exposure on the behavioral and brain-derived neurotrophic factor response to nicotine." Journal of Psychopharmacology 31, no. 1 (2016): 75–85. http://dx.doi.org/10.1177/0269881116681458.

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This study analyzed the interaction of adolescent methylphenidate on the behavioral response to nicotine and the effects of these drug treatments on brain-derived neurotrophic factor in the nucleus accumbens and hippocampus in male and female Sprague-Dawley rats. Animals were intraperitoneal administered 1 mg/kg methylphenidate or saline using a “school day” regimen (five days on, two days off) beginning on postnatal day (P)28 and throughout behavioral testing. In Experiment 1, animals were intraperitoneal administered 0.5 mg/kg (free base) nicotine or saline every second day for 10 days from

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RIRIE, DOUGLAS, KIRSTEN RIRIE, NAVIL SETHNA, and LIZANNE FOX. "Unexpected interaction of methylphenidate (Ritalin ® ) with anaesthetic agents." Pediatric Anesthesia 7, no. 1 (1997): 69–72. http://dx.doi.org/10.1046/j.1460-9592.1997.d01-34.x.

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CACI, HERVÉ, and FRANCK BAYLÉ. "A Case of Disulfiram-Methylphenidate Interaction: Implications for Treatment." American Journal of Psychiatry 164, no. 11 (2007): 1759. http://dx.doi.org/10.1176/appi.ajp.2007.07060892.

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WINHUSEN, T., E. SOMOZA, B. SINGAL, et al. "Methylphenidate and cocaine: A placebo-controlled drug interaction study." Pharmacology Biochemistry and Behavior 85, no. 1 (2006): 29–38. http://dx.doi.org/10.1016/j.pbb.2006.06.023.

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Pelham, William E., David L. Meichenbaum, Bradley H. Smith, Margaret H. Sibley, Elizabeth M. Gnagy, and Oscar Bukstein. "Acute Effects of MPH on the Parent–Teen Interactions of Adolescents With ADHD." Journal of Attention Disorders 21, no. 2 (2016): 158–67. http://dx.doi.org/10.1177/1087054713480833.

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This study explored the nature of interactions between adolescent males with ADHD and their mothers, and the effects of methylphenidate (MPH) on an analogue parent–teen interaction task. Twenty-five adolescent males with ADHD ( M = 13.6 years) and their mothers and 14 non-ADHD adolescent males ( M = 13.4 years) and their mothers completed ratings of perceived dyadic conflict. Behavioral observations of dyads during 10-min conflict-resolution tasks were also collected. The ADHD dyads completed these tasks twice, with adolescents receiving either 0.3 mg/kg MPH or placebo. Videotaped sessions wer

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Peña Guerrero, Patricia, Carolina Magro Fernández, and Eduardo Durán Ferreras. "Epileptic seizure due to drug interaction between sertraline and methylphenidate." Medicina Clínica (English Edition) 147, no. 12 (2016): 568. http://dx.doi.org/10.1016/j.medcle.2016.12.049.

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Öner, Özgür, Ata Akın, Hasan Herken, et al. "Association Among SNAP-25 Gene DdeI and MnlI Polymorphisms and Hemodynamic Changes During Methylphenidate Use." Journal of Attention Disorders 15, no. 8 (2010): 628–37. http://dx.doi.org/10.1177/1087054710374597.

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Objective: To investigate the interaction of treatment-related hemodynamic changes with genotype status for Synaptosomal associated protein 25 (SNAP-25) gene in participants with attention deficit hyperactivity disorder (ADHD) on and off single dose short-acting methylphenidate treatment with functional near-infrared spectroscopy (fNIRS). Method: A total of 15 right-handed adults and 16 right-handed children with DSM-IV diagnosis of ADHD were evaluated. Ten milligrams of short-acting methylphenidate was administered in a crossover design. Results: Participants with SNAP-25 DdeI T/T genotype ha

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KOTAKI, HAJIME, FUTAMI NAKAZATO, TAKAO AOYAMA, YUKIYA SAITOH, and FUJIO NAKAGAWA. "Interaction in tissue distribution between methylphenidate and pemoline. I. Tissue distribution of methylphenidate and its metabolite in the rat." CHEMICAL & PHARMACEUTICAL BULLETIN 36, no. 8 (1988): 3190–95. http://dx.doi.org/10.1248/cpb.36.3190.

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DuPAUL, GEORGE J., RUSSELL A. BARKLEY, and MARY B. McMURRAY. "Response of Children with ADHD to Methylphenidate: Interaction with Internalizing Symptoms." Journal of the American Academy of Child & Adolescent Psychiatry 33, no. 6 (1994): 894–903. http://dx.doi.org/10.1097/00004583-199407000-00016.

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Briegel, Wolfgang. "Psychiatric Comorbidities in 1p36 Deletion Syndrome and Their Treatment—A Case Report." International Journal of Environmental Research and Public Health 18, no. 22 (2021): 12064. http://dx.doi.org/10.3390/ijerph182212064.

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1p36 deletion syndrome represents the most common terminal deletion observed in humans. Major clinical findings comprise developmental delay/intellectual disability, poor or absent expressive language, congenital central muscular hypotonia, brain anomalies, brachydactyly/camptodactyly, short feet, and characteristic facial features like straight eyebrows, deep-set eyes, and midface hypoplasia. So far, there is very limited knowledge about comorbid psychiatric disorders and their effective treatment in this special population. To fill this gap, this case report presents an initially four-year-o

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Le Nedelec, M. J., and R. J. Rosengren. "Methylphenidate inhibits cytochrome P450 in the Swiss Webster mouse." Human & Experimental Toxicology 21, no. 5 (2002): 273–80. http://dx.doi.org/10.1191/0960327102ht245oa.

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Drug interactions have previously been reported following the co-administration of methylphenidate (MPH) and drugs metabolized by the cytochrome P450 (CYP450) system such as imipramine. Therefore, this study used the Swiss Webster mouse to determine the effect of MPH on CYP450 isozymes likely to be important in the interaction between MPH and imipramine. Single high doses of MPH (25, 50 and 100 mg/kg, i.p.) were administered to simulate the abuse of MPH. Under these conditions, MPH decreased total hepatic CYP450 to 50% of control. Additionally, MPH inhibited the catalytic activity of CYP1A and

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Hotha, Kishore Kumar, Swapan Roychowdhury, and Veerappan Subramanian. "Drug-Excipient Interaction of Methylphenidate with Glycerin in Methylphenidate Oral Solution and Identification of its Transesterification Products by UPLC-MS/MS." American Journal of Analytical Chemistry 07, no. 02 (2016): 151–64. http://dx.doi.org/10.4236/ajac.2016.72013.

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Kim, Ju Yeon, Eunji Jung, Taeyeop Lee, Kee Jeong Park, Yoo-Sook Joung, and Hyo-Won Kim. "Effects of Methylphenidate and Atomoxetine Treatment on Improvement of Motor Coordination in Children With Attention-Deficit/Hyperactivity Disorder." Psychiatry Investigation 22, no. 1 (2025): 84–92. https://doi.org/10.30773/pi.2024.0198.

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Objective To investigate the effects of methylphenidate and atomoxetine treatment on motor coordination in children with attention-deficit/hyperactivity disorder (ADHD).Methods In this single-site, open-label, naturalistic follow-up study, 157 children (7.6±1.4 years; 139 males) with ADHD were recruited between March 2015 and May 2020 from the Department of Psychiatry, Asan Medical Center, and treated for 12 weeks with methylphenidate (n=48) or atomoxetine (n=109). Children completed the Advanced Test of Attention (ATA), and caregivers completed the ADHD Rating Scale (ARS) questionnaire and De

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Talishinsky, Aleksandr D., Celine Nicolas, and Satoshi Ikemoto. "Interaction of chronic food restriction and methylphenidate in sensation seeking of rats." Psychopharmacology 234, no. 14 (2017): 2197–206. http://dx.doi.org/10.1007/s00213-017-4625-6.

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Grau-López, Lara, Carlos Roncero, Maria C. Navarro, and Miquel Casas. "Psychosis Induced by the Interaction Between Disulfiram and Methylphenidate May Be Dose Dependent." Substance Abuse 33, no. 2 (2012): 186–88. http://dx.doi.org/10.1080/08897077.2011.634968.

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Beery, Susan H., Herbert C. Quay, and William E. Pelham. "Differential Response to Methylphenidate in Inattentive and Combined Subtype ADHD." Journal of Attention Disorders 21, no. 1 (2016): 62–70. http://dx.doi.org/10.1177/1087054712469256.

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Objective: To examine response to methylphenidate (MPH) assessed by direct observation of ecologically valid behaviors in boys with ADHD with high hyperactivity-impulsivity (HI) and those with predominantly inattentive symptoms (ADHD/I). Method: Sixty-three boys ages 7 to 13 participated in an ADHD Summer Treatment Program and received a double-blind placebo-controlled assessment of .3 mg/kg of MPH on problem behaviors and individualized behavior goals. Medication effect sizes were calculated for each child for each behavior. Results: Children with ADHD/HI ( n = 21) displayed larger MPH effect

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Hetherington, L., EJ Dommett, AC Turner, TB Riley, JX Haensel, and PG Overton. "Effect of methylphenidate on visual responses in the superior colliculus in the anaesthetised rat: Role of cortical activation." Journal of Psychopharmacology 31, no. 10 (2017): 1347–61. http://dx.doi.org/10.1177/0269881117730661.

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The mechanism of action of psychostimulant drugs in the treatment of Attention Deficit Hyperactivity Disorder is still largely unknown, although recent evidence suggests one possibility is that the drugs affect the superior colliculus (SC). We have previously demonstrated that systemically administered d-amphetamine attenuates/abolishes visual responses to wholefield light flashes in the superficial layers of the SC in anaesthetised rats, and the present study sought to extend this work to methylphenidate (MPH). Anaesthetised rats were administered MPH at a range of doses (or saline) and subje

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van der Veen-Mulders, Lianne, Barbara J. van den Hoofdakker, Maaike H. Nauta, Paul Emmelkamp, and Pieter J. Hoekstra. "Methylphenidate Has Superior Efficacy Over Parent–Child Interaction Therapy for Preschool Children with Disruptive Behaviors." Journal of Child and Adolescent Psychopharmacology 28, no. 1 (2018): 66–73. http://dx.doi.org/10.1089/cap.2017.0123.

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Babkoff, Harvey, Tamsin L. Kelly, Larry T. Matteson, et al. "Pemoline and Methylphenidate: Interaction With Mood, Sleepiness, and Cognitive Performance During64 Hours of Sleep Deprivation." Military Psychology 4, no. 4 (1992): 235–65. http://dx.doi.org/10.1207/s15327876mp0404_3.

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Dodds, Chris, Ulrich Müller, and Tom Manly. "Effects of Psychostimulants on Alertness and Spatial Bias in Healthy Participants." Journal of Cognitive Neuroscience 21, no. 3 (2009): 529–37. http://dx.doi.org/10.1162/jocn.2009.21046.

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Converging evidence from neuropsychological and neuroimaging studies suggests that the ability to maintain an alert, ready-to-respond state is mediated by a network of right-hemisphere frontal and parietal cortical areas. This right lateralization may help to explain why visuospatial hemineglect, a cluster of deficits in detecting and responding to contralesional stimuli, is more common and persistent after right-hemisphere lesions. Indeed, it has been hypothesized that this asymmetry reflects a direct, functional link between alertness and spatial attention. In the present study, we investiga

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Schreiber, Shaul, Miaad Bader, Vardit Rubovitch, and Chaim G. Pick. "Interaction between methylphenidate, methadone and different antidepressant drugs on antinociception in mice, and possible clinical implications." World Journal of Biological Psychiatry 18, no. 4 (2015): 300–307. http://dx.doi.org/10.3109/15622975.2015.1086492.

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Markowitz, John S., Hao-Jie Zhu, and Kennerly S. Patrick. "Isopropylphenidate: An Ester Homolog of Methylphenidate with Sustained and Selective Dopaminergic Activity and Reduced Drug Interaction Liability." Journal of Child and Adolescent Psychopharmacology 23, no. 10 (2013): 648–54. http://dx.doi.org/10.1089/cap.2013.0074.

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Dar, Dalit E., Cheryl Mayo, and George R. Uhl. "The interaction of methylphenidate and benztropine with the dopamine transporter is different than other substrates and ligands." Biochemical Pharmacology 70, no. 3 (2005): 461–69. http://dx.doi.org/10.1016/j.bcp.2005.04.032.

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Kang, Jewook, Bum-Sung Choi, and Bongseog Kim. "The Role of Gene–Gene Interaction Between ADRA2A and SLC6A2 Polymorphisms in Attention System and Treatment Outcomes for Children with ADHD." Children 12, no. 6 (2025): 704. https://doi.org/10.3390/children12060704.

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Background and Objectives: Most genetic studies have focused on catecholamine system genes to identify etiology in attention-deficit/hyperactivity disorder (ADHD), and there is growing evidence that the interaction of several genes may synergistically or antagonistically affect disease outcomes. We investigated the interaction between the alpha-2 adrenergic receptor (ADRA2A) and its transporter (SLC6A2) to determine the etiology and treatment outcomes for ADHD. Materials and Methods: Children with ADHD (age 8.3 ± 2.0 y, 72 boys and 11 girls) were assessed using the Kiddie Schedule for Affectiv

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Casiraghi, Andrea, Francesca Longhena, Gaia Faustini, et al. "Methylphenidate Analogues as a New Class of Potential Disease-Modifying Agents for Parkinson’s Disease: Evidence from Cell Models and Alpha-Synuclein Transgenic Mice." Pharmaceutics 14, no. 8 (2022): 1595. http://dx.doi.org/10.3390/pharmaceutics14081595.

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Parkinson’s disease (PD) is characterized by dopaminergic nigrostriatal neurons degeneration and Lewy body pathology, mainly composed of α-synuclein (αSyn) fibrillary aggregates. We recently described that the neuronal phosphoprotein Synapsin III (Syn III) participates in αSyn pathology in PD brains and is a permissive factor for αSyn aggregation. Moreover, we reported that the gene silencing of Syn III in a human αSyn transgenic (tg) mouse model of PD at a pathological stage, manifesting marked insoluble αSyn deposits and dopaminergic striatal synaptic dysfunction, could reduce αSyn aggregate

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Mohamoud, Mohamed, Qi Chen, David Croteau, et al. "Acute Hyperkinetic Movement Disorders as a Multifactorial Pharmacodynamic Drug Interaction Between Methylphenidate and Risperidone in Children and Adolescents." Journal of Clinical Psychopharmacology 42, no. 3 (2022): 238–46. http://dx.doi.org/10.1097/jcp.0000000000001547.

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Coelho, Cideli de Paula, Bruna Oliveira, Larissa Cristina Ares Silveira da Motta, Amanda Sousa, and Maria Martha Bernardi. "Evaluation of the ultradiluted medication MPD 30 CH in the offspring of mice mothers treated with methylphenidate during lactation." International Journal of High Dilution Research - ISSN 1982-6206 15, no. 4 (2021): 20–21. http://dx.doi.org/10.51910/ijhdr.v15i4.844.

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Background: Methylphenidate (MPD) is a non-stimulating amphetamine that has being used for some time in the treatment of Attention Deficit Hyperactivity Disorder (ADHD) and, in adequate doses, it promotes the remission of symptoms and the improvement of important aspects, as social interaction and academic performance1, in patients with ADHD. Literature data indicates that MPD attenuates maternal behavior in mices2. According to this line of study, the work â€œRepeated methylphenidate administration during lactation reduces maternal behavior, induces maternal tolerance, and increases anxiety-l

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Angenoorth, Thomas J. F., Stevan Stankovic, Marco Niello, et al. "Interaction Profiles of Central Nervous System Active Drugs at Human Organic Cation Transporters 1–3 and Human Plasma Membrane Monoamine Transporter." International Journal of Molecular Sciences 22, no. 23 (2021): 12995. http://dx.doi.org/10.3390/ijms222312995.

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Many psychoactive compounds have been shown to primarily interact with high-affinity and low-capacity solute carrier 6 (SLC6) monoamine transporters for norepinephrine (NET; norepinephrine transporter), dopamine (DAT; dopamine transporter) and serotonin (SERT; serotonin transporter). Previous studies indicate an overlap between the inhibitory capacities of substances at SLC6 and SLC22 human organic cation transporters (SLC22A1–3; hOCT1–3) and the human plasma membrane monoamine transporter (SLC29A4; hPMAT), which can be classified as high-capacity, low-affinity monoamine transporters. However,

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Karim, Tahseen J., Cruz Reyes-Vazquez, and Nachum Dafny. "Comparison of the VTA and LC response to methylphenidate: a concomitant behavioral and neuronal study of adolescent male rats." Journal of Neurophysiology 118, no. 3 (2017): 1501–14. http://dx.doi.org/10.1152/jn.00145.2017.

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Methylphenidate (MPD), also known as Ritalin, is a psychostimulant used to treat attention deficit hyperactivity disorder. However, it is increasingly being misused by normal adolescents for recreation and academic advantage. Therefore, it is important to elucidate the behavioral and neurophysiological effects of MPD in normal subjects. MPD inhibits the reuptake of catecholamines, mainly found in the ventral tegmental area (VTA) and locus coeruleus (LC). The VTA and LC normally mediate attention, motivation, and drug reward behaviors. Selective neuronal connections between the VTA and LC have

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Li, Conghui, Xiaolu Han, Xiaoxuan Hong, et al. "Study on the Complexation and Release Mechanism of Methylphenidate Hydrochloride Ion Exchange Resin Complex." Polymers 13, no. 24 (2021): 4394. http://dx.doi.org/10.3390/polym13244394.

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Since the advent of ion exchange resin, it has been widely used in many fields, including drug delivery systems. The drug binds to the resin through an exchange reaction to form a drug–resin complex, which can gradually release drugs through the exchange of physiological ions in the gastrointestinal tract, to realize functions such as taste masking and regulating release. In this study, the complexes of methylphenidate hydrochloride and Amberlite IRP69 were prepared and evaluated to explore the mechanism of complexation, influencing factors and release mechanism at a molecular level. Firstly,

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Robinson, Andrea M., and David J. Bucci. "Individual and combined effects of physical exercise and methylphenidate on orienting behavior and social interaction in spontaneously hypertensive rats." Behavioral Neuroscience 128, no. 6 (2014): 703–12. http://dx.doi.org/10.1037/bne0000015.

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Herrera-Isaza, Laura, Karen Corredor, Fernando Cardenas, Santiago Zarate, and Angela Gomez. "Role of environmental enrichment in locomotion, anxiety, memory and social interaction of Wistar rats under chronic treatment of methylphenidate." IBRO Reports 6 (September 2019): S329. http://dx.doi.org/10.1016/j.ibror.2019.07.1017.

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