Gotowe bibliografie tematyczne / Microthrombi

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Gotowa bibliografia na temat „Microthrombi”

Autor: Grafiati

Data publikacji: 21 grudnia 2024

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Microthrombi"

Olson, Timothy M., David J. Driscoll, William D. Edwards, Francisco J. Puga, and Gordon K. Danielson. "Pulmonary microthrombi." Journal of Thoracic and Cardiovascular Surgery 106, no. 4 (1993): 739–44. http://dx.doi.org/10.1016/s0022-5223(19)33719-5.

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Fitzpatrick, James E., Carl Johnson, M. C. Major, Paul Simon, James Owenby, and Lieutenant Colonel. "Cutaneous Microthrombi." American Journal of Dermatopathology 9, no. 5 (1987): 419–22. http://dx.doi.org/10.1097/00000372-198710000-00008.

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Mihalko, Emily P., Megan Sandry, Nicholas Mininni, et al. "Fibrin-modulating nanogels for treatment of disseminated intravascular coagulation." Blood Advances 5, no. 3 (2021): 613–27. http://dx.doi.org/10.1182/bloodadvances.2020003046.

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Abstract Disseminated intravascular coagulation (DIC) is a pathological coagulopathy associated with infection that increases mortality. In DIC, excessive thrombin generation causes symptoms from formation of microthrombi to multiorgan failure; bleeding risks can also be a concern because of clotting factor consumption. Different clinical events lead to DIC, including sepsis, trauma, and shock. Treatments for thrombotic episodes or bleeding presentation in DIC oppose each other, thus creating therapeutic dilemmas in management. The objective of this study was to develop fibrin-specific core-sh

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Shao, Bojing, Mark G. Wahrenbrock, Longbiao Yao, et al. "Carcinoma mucins trigger reciprocal activation of platelets and neutrophils in a murine model of Trousseau syndrome." Blood 118, no. 15 (2011): 4015–23. http://dx.doi.org/10.1182/blood-2011-07-368514.

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Abstract Trousseau syndrome is classically defined as migratory, heparin-sensitive but warfarin-resistant microthrombi in patients with occult, mucinous adenocarcinomas. Injecting carcinoma mucins into mice generates platelet-rich microthrombi dependent on P- and L-selectin but not thrombin. Heparin prevents mucin binding to P- and L-selectin and mucin-induced microthrombi. This model of Trousseau syndrome explains resistance to warfarin, which inhibits fluid-phase coagulation but not selectins. Here we found that carcinoma mucins do not generate microthrombi in mice lacking P-selectin glycopr

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Jilientasia, Godrace Lilihataa, Pujo Semedia Bambang, Utariania Arie, et al. "Histopathological Examination with Cd61 Confirms D-Dimer as a Predictor of Diffuse Pulmonary Microthrombi Findings in Covid-19 Patients." International Journal of Innovative Science and Research Technology 7, no. 1 (2022): 349–55. https://doi.org/10.5281/zenodo.5910705.

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COVID-19 is a global concern that emerged and became a pandemic at March 2020. It is related to dysregulation of immune system causing excessive immune response known as cytokine storm. Other than that there is hypercoagulation that could progress to thrombosis and embolism in various vital organs. Pulmonary embolism is one of the dire consequences of hypercoagulation. CTPA and V/Q scan are the gold standards in pulmonary embolism diagnosis. However in developing countries like Indonesia these facilities are often absent. Thus a simple blood test like Ddimer could aid the detection of pulmonar

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Park, Sohyung, and Byung-Ha Choi. "Acute Myocardial Infarction with Microthrombi in Cardiac Small Vessels after COVID-19 Vaccination (ChAdOx1 nCov-19): A Case Report." Korean Journal of Legal Medicine 45, no. 4 (2021): 127–32. http://dx.doi.org/10.7580/kjlm.2021.45.4.127.

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We present the postmortem findings of an unexpected death due to acute myocardial infarction (AMI) with microthrombi and thrombosis in other vessels after the first dose of coronavirus disease 2019 (COVID-19) vaccination (ChAdOx1 nCov-19). The deceased was a 69-year-old woman who complained of nonspecific symptoms shortly after vaccination and was found dead on the sixth day. Postmortem examination revealed AMI and complications (left ventricular rupture, hemopericardium) with microthrombi in small cardiac vessels, which are similar to the characteristic findings of myocardial injury caused by

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Neil, Desley A. H. "CD31 highlights platelet-rich microthrombi." Histopathology 54, no. 3 (2009): 387–88. http://dx.doi.org/10.1111/j.1365-2559.2008.03215.x.

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Dienel, Ari, Remya Ammassam Veettil, Sung-Ha Hong, et al. "Microthrombi Correlates With Infarction and Delayed Neurological Deficits After Subarachnoid Hemorrhage in Mice." Stroke 51, no. 7 (2020): 2249–54. http://dx.doi.org/10.1161/strokeaha.120.029753.

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Background and Purpose: Delayed neurological deficits are a devastating consequence of subarachnoid hemorrhage (SAH), which affects about 30% of surviving patients. Although a very serious concern, delayed deficits are understudied in experimental SAH models; it is not known whether rodents recapitulate the delayed clinical decline seen in SAH patients. We hypothesized that mice with SAH develop delayed functional deficits and that microthrombi and infarction correlate with delayed decline. Methods: Adult C57BL/6J mice of both sexes were subjected to endovascular perforation to induce SAH. Mic

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Taha, Aladdin, Magdolna Nagy, Hajo M. Hund, et al. "Vascular injury and occurrence of microthrombi after endovascular therapy for acute ischaemic stroke in a thromboembolic model." BMJ Neurology Open 7, no. 1 (2025): e000989. https://doi.org/10.1136/bmjno-2024-000989.

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BackgroundEndovascular catheters and devices used for thrombectomy in patients who had a stroke can damage the vessel lumen leading to microthrombi. During stroke recanalisation, microthrombi could migrate distally and occlude cerebral microvasculature, potentially limiting the benefit of recanalisation therapy.ObjectivesTo describe vascular injury occurring after endovascular therapy (EVT), with stent retrievers (SR) and direct aspiration (DA), to open up avenues for further improvement of EVT technique.MethodsSR and DA were performed according to clinical procedures in extracranial vessels i

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Samal, Bibek, Ari Dienel, Sungha Hong, and Devin Mcbride. "1139 Role of Platelets in the Development of Neurological Deficits Resulting From Subarachnoid Hemorrhage in Mice." Neurosurgery 71, Supplement_1 (2025): 175. https://doi.org/10.1227/neu.0000000000003360_1139.

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INTRODUCTION: Microthrombosis (platelet aggregation) has been suggested as a major factor contributing to delayed neurological deterioration in patients after subarachnoid hemorrhage (SAH). However, experimental studies on the role of microthrombi in delayed deficits after SAH have not been investigated in depth. Similarly, the only clinically existing antiplatelet therapy is nimodipine, with other therapies being invasive. METHODS: SAH was induced in adult male and female C57BL/6 mice via endovascular perforation. Mice were randomly assigned into sham (n=6/sex) or SAH groups (n=22-24/sex). A

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Rozprawy doktorskie na temat "Microthrombi"

Picot, Audrey. "Develοpment οf a theranοstic agent fοr micrοthrοmbοsis during ischemic strοke". Electronic Thesis or Diss., Normandie, 2024. http://www.theses.fr/2024NORMC418.

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De nouvelles études suggèrent que l'accident vasculaire cérébral ischémique est fréquemment associé à la formation de microthrombi dans la microcirculation corticale. Ces microthrombi sont associés à une augmentation de la taille de la lésion, à un déclin cognitif et à la démence. Il est difficile de prendre en charge ces microthrombi car les méthodes d'imagerie actuelles ne permettent pas de les détecter. Pour y remédier, un nouvel agent théranostique a été développé, les IO@PDA@tPA, combinant des microparticules d'oxyde de fer (IO) enrobées de polydopamine (PDA) et conjuguées avec l’activate

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Części książek na temat "Microthrombi"

de Chickera, Sonali N., and Shaifali Sandal. "Kidney Grafts with Evidence of Microthrombi in Glomerular Capillaries." In Complications in Kidney Transplantation. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-13569-9_11.

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Shimamura, Kayako, Kuniyuki Oka, Masaki Nakazawa, and Mizu Kojima. "DISTRIBUTION PATTERNS OF MICROTHROMBI IN DISSEMINATED INTRAVASCULAR COAGULATION (DIC)." In Acute pulmonary insufficiency, edited by Tom Saldeen. De Gruyter, 1985. http://dx.doi.org/10.1515/9783110850123-007.

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Ghugare, Sheetal Anil, Kusum D. Jashnani, Lalita Yoganand Patil, and Vandana Rajesh Saravade. "Disseminated Microthrombi in Pregnancy: Not Always DIC, Think of TTP." In Maternal Mortality - Lessons Learnt from Autopsy. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-3420-9_17.

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Leach, Richard. "The Respiratory System." In Oxford Textbook of Respiratory Critical Care. Oxford University PressOxford, 2023. http://dx.doi.org/10.1093/med/9780198766438.003.0002.

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Abstract Summary This chapter reviews lung anatomy, including alveolar microanatomy, pulmonary mechanics, control of breathing, gas exchange, pulmonary circulation, and, although not directly related to the respiratory system, the transport of oxygen to the tissues. It examines lung ventilation–perfusion and heart–lung interactions, the impact of low atmospheric pressure and respiratory disease on these relationships. Finally, it briefly acknowledges the many other respiratory system functions, including chemical and drug clearance from the systemic circulation, breakdown of microthrombi, horm

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Prasad, Kuruswamy Thurai. "COVID-19 and Pulmonary Diseases." In COVID-19: Effects in Comorbidities and Special Populations. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/9789815036367122010010.

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The coronavirus disease 2019 (COVID-19) primarily affects the respiratory system, commonly manifesting as pneumonia. The clinical presentation of COVID-19 is challenging to distinguish from community-acquired pneumonia due to other etiologies and respiratory exacerbations of pre-existing chronic respiratory diseases. Fortunately, the majority of patients have an asymptomatic or mild illness. However, some patients may develop profound hypoxemia secondary to diffuse alveolar damage and occlusion of alveolar capillaries by microthrombi. When patients with compromised lung function due to pre-exi

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Streszczenia konferencji na temat "Microthrombi"

Santos, João Vitor Ribeiro dos, Mariana Spitz, and Ana Carolina Andorinho. "Stroke secondary to thrombotic microangiopathy." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.300.

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Introduction: Thrombotic thrombocytopenic purpura (TTP) is a hematological disease resulting from the ADAMTS 13 plasmatic protein deficit. It can be congenital or sporadic, and is usually autoimmune. Pathological platelet adhesion occurs, leading to microthrombi in capillary and arterial circulation, microangiopathic anemia and ischemia. The clinical picture includes thrombocytopenia, renal dysfunction, fluctuating neurological symptoms, microangiopathic hemolytic anemia, and fever. Methods: Case report of a 51-year-old male hypertensive patient, diagnosed with idiopathic thrombocytopenic purp

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Kanamori, Y., I. Yada, I. Yuasa, M. Kusagawa, and K. Deguchi. "PHYSIOLOGICAL ROLE OF THE ENHANCED FIBRINOLYTIC ACTIVITY DURING CARDIOPULMONARY BYPASS IN OPEN HEART SURGERY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644658.

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Fibrinolytic activity is reportedly increased during cardiopulmonary bypass( CPB ), and this increase has been considered to be related to the bleeding complications in open heart surgery. The purpose of this study was to clarify the nature of the fibrinolytic activity duringCPB. Twenty patients with valve replacement or aortocoronary bypass surgery were examined. The following parameters were determined: fibrinogen, plasminogen, fibrinopeptide A( FPA ), fibrinopeptide B β 15-42 ( FPB β15-42 ), and tissue-type plasminogen activator ( t-PA ). For further characterization of the fibrinolytic act

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Perktold, Karl, Gerhard Karner, and Gerhard Rappitsch. "Flow Effects on the Concentration of Platelet Active Substances in the Vicinity of Mural Platelet Aggregates." In ASME 1996 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1996. http://dx.doi.org/10.1115/imece1996-1228.

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Abstract The influence of convective flow on the transport of prothrombin, thrombin, antithrombin, ADP and thromboxane in the vicinity of a mural microthrombus has been analysed applying computer simulation. The basis of the study is a suitable reaction, emission and transport model. The reaction kinetics and the mass transport are modelled mathematically with a coupled system of convection-diffusion equations. The description of the flow field near the microthrombus uses the three-dimensional Navier-Stokes equations. The numerical approach applies the finite element method.

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Conhaim, RL, KE Watson, WF Dovi, and BA Harms. "Microthrombus Formation Continues in Rat Lungs for up to 24 Hours Following Acute Hemorrhage." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3870.

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Araújo, Victor Oliveira, Isadora Mônica Ponte de Oliveira, Lara Maria de Oliveira Paiva Freitas, and Júlio César Claudino dos Santos. "“Brain fog” in the post-acute phase of Covid-19." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.681.

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Introduction: Covid-19 infection can affect not only the airways but also other organs such as the brain. Individuals that tested positive for SARSCoV-2 may be asymptomatic, but can also have symptoms - such as “brain fog” - during the acute phase and/or the post-acute phase (being the last ones defined as long haulers). “Brain fog” is a set of symptoms characterized by cognitive dysfunction that includes the inability to concentrate, executive function deficits, anterograde and retrograde amnesia. Objective: Review the pathophysiology of individuals with Covid-19 post-acute phase brain fog. D

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Fujimori, T., T. Saeki, K. Harada, M. Sato, and N. Ohshima. "ANTI-THROMBOTIC EFFECTS OF E-5510 IN EXPERIMENTAL THROMBOSIS MODELS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643429.

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A new agent developed in our laboratory, 4-cyano-5,5-bis(4-methoxyphenyl)-4-pentenoic acid (E-5510), suppressed various human platelet functions in vitro. The compound also showed quite potent ex vivo anti-platelet effects in many experimentalanimals. It is well known that anti-platelet effects are not always parallel to anti-thrombotic effects. Thus, in order to predict the efficacy of E-5510 in thrombotic disorders, the anti-thrombotic effects were examined in 3 different animal models of thrombosis.(1) Anti-thrombotic effect in an extracorporeal shunt model Two hrs after oral administration

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