Zaloguj się

Gotowe bibliografie tematyczne / Migration Adhesion

Spis treści

Artykuły w czasopismach

Gotowa bibliografia na temat „Migration Adhesion”

Autor: Grafiati

Data publikacji: 6 września 2023

Utwórz poprawne odniesienie w stylach APA, MLA, Chicago, Harvard i wielu innych

Wybierz rodzaj źródła:

Zobacz listy aktualnych artykułów, książek, rozpraw, streszczeń i innych źródeł naukowych na temat „Migration Adhesion”.

Przycisk „Dodaj do bibliografii” jest dostępny obok każdej pracy w bibliografii. Użyj go – a my automatycznie utworzymy odniesienie bibliograficzne do wybranej pracy w stylu cytowania, którego potrzebujesz: APA, MLA, Harvard, Chicago, Vancouver itp.

Możesz również pobrać pełny tekst publikacji naukowej w formacie „.pdf” i przeczytać adnotację do pracy online, jeśli odpowiednie parametry są dostępne w metadanych.

Artykuły w czasopismach na temat "Migration Adhesion"

1

Aguilar-Cuenca, Rocio, Clara Llorente-Gonzalez, Carlos Vicente, and Miguel Vicente-Manzanares. "Microfilament-coordinated adhesion dynamics drives single cell migration and shapes whole tissues." F1000Research 6 (February 17, 2017): 160. http://dx.doi.org/10.12688/f1000research.10356.1.

Pełny tekst źródła

Streszczenie:

Cell adhesion to the substratum and/or other cells is a crucial step of cell migration. While essential in the case of solitary migrating cells (for example, immune cells), it becomes particularly important in collective cell migration, in which cells maintain contact with their neighbors while moving directionally. Adhesive coordination is paramount in physiological contexts (for example, during organogenesis) but also in pathology (for example, tumor metastasis). In this review, we address the need for a coordinated regulation of cell-cell and cell-matrix adhesions during collective cell mig

Style APA, Harvard, Vancouver, ISO itp.

2

Peacock, Justin G., Ann L. Miller, William D. Bradley, Olga C. Rodriguez, Donna J. Webb, and Anthony J. Koleske. "The Abl-related Gene Tyrosine Kinase Acts through p190RhoGAP to Inhibit Actomyosin Contractility and Regulate Focal Adhesion Dynamics upon Adhesion to Fibronectin." Molecular Biology of the Cell 18, no. 10 (2007): 3860–72. http://dx.doi.org/10.1091/mbc.e07-01-0075.

Pełny tekst źródła

Streszczenie:

In migrating cells, actin polymerization promotes protrusion of the leading edge, whereas actomyosin contractility powers net cell body translocation. Although they promote F-actin–dependent protrusions of the cell periphery upon adhesion to fibronectin (FN), Abl family kinases inhibit cell migration on FN. We provide evidence here that the Abl-related gene (Arg/Abl2) kinase inhibits fibroblast migration by attenuating actomyosin contractility and regulating focal adhesion dynamics. arg−/− fibroblasts migrate at faster average speeds than wild-type (wt) cells, whereas Arg re-expression in thes

Style APA, Harvard, Vancouver, ISO itp.

3

González-Tarragó, Víctor, Alberto Elosegui-Artola, Elsa Bazellières, Roger Oria, Carlos Pérez-González та Pere Roca-Cusachs. "Binding of ZO-1 to α5β1 integrins regulates the mechanical properties of α5β1–fibronectin links". Molecular Biology of the Cell 28, № 14 (2017): 1847–52. http://dx.doi.org/10.1091/mbc.e17-01-0006.

Pełny tekst źródła

Streszczenie:

Fundamental processes in cell adhesion, motility, and rigidity adaptation are regulated by integrin-mediated adhesion to the extracellular matrix (ECM). The link between the ECM component fibronectin (fn) and integrin α5β1 forms a complex with ZO-1 in cells at the edge of migrating monolayers, regulating cell migration. However, how this complex affects the α5β1-fn link is unknown. Here we show that the α5β1/ZO-1 complex decreases the resistance to force of α5β1–fn adhesions located at the edge of migrating cell monolayers while also increasing α5β1 recruitment. Consistently with a molecular c

Style APA, Harvard, Vancouver, ISO itp.

4

Longley, R. L., A. Woods, A. Fleetwood, G. J. Cowling, J. T. Gallagher, and J. R. Couchman. "Control of morphology, cytoskeleton and migration by syndecan-4." Journal of Cell Science 112, no. 20 (1999): 3421–31. http://dx.doi.org/10.1242/jcs.112.20.3421.

Pełny tekst źródła

Streszczenie:

Syndecan-4 is a widely expressed transmembrane heparan sulfate proteoglycan which localizes to focal adhesions. Previous studies showed that the syndecan-4 cytoplasmic domain can associate with and potentiate the activity of protein kinase C, which is required for focal adhesion formation. To examine further the role of syndecan-4 in cell adhesion, we expressed syndecan-4 cDNA constructs in CHO-K1 cells. Syndecan-2 transfection was used to confirm effects seen were specific for syndecan-4. Cells overexpressing full length syndecan-4 core protein exhibited a more flattened, fibroblastic morphol

Style APA, Harvard, Vancouver, ISO itp.

5

Ventre, Maurizio, Carlo Fortunato Natale, Carmela Rianna, and Paolo Antonio Netti. "Topographic cell instructive patterns to control cell adhesion, polarization and migration." Journal of The Royal Society Interface 11, no. 100 (2014): 20140687. http://dx.doi.org/10.1098/rsif.2014.0687.

Pełny tekst źródła

Streszczenie:

Topographic patterns are known to affect cellular processes such as adhesion, migration and differentiation. However, the optimal way to deliver topographic signals to provide cells with precise instructions has not been defined yet. In this work, we hypothesize that topographic patterns may be able to control the sensing and adhesion machinery of cells when their interval features are tuned on the characteristic lengths of filopodial probing and focal adhesions (FAs). Features separated by distance beyond the length of filopodia cannot be readily perceived; therefore, the formation of new adh

Style APA, Harvard, Vancouver, ISO itp.

6

Powner, Dale, Petra M. Kopp, Susan J. Monkley, David R. Critchley, and Fedor Berditchevski. "Tetraspanin CD9 in cell migration." Biochemical Society Transactions 39, no. 2 (2011): 563–67. http://dx.doi.org/10.1042/bst0390563.

Pełny tekst źródła

Streszczenie:

Tetraspanin CD9 is associated with integrin adhesion receptors and it was reported that CD9 regulates integrin-dependent cell migration and invasion. Pro- and anti-migratory effects of CD9 have been linked to adhesion-dependent signalling pathways, including phosphorylation of FAK (focal adhesion kinase) and activation of phosphoinositide 3-kinase, p38 MAPK (mitogen-activated protein kinase) and JNK (c-Jun N-terminal kinase). In the present paper, we describe a novel mechanism whereby CD9 specifically controls localization of talin1, one of the critical regulators of integrin activation, to fo

Style APA, Harvard, Vancouver, ISO itp.

7

Banisadr, Afsheen, Mariam Eick, Pranjali Beri, et al. "EGFRvIII uses intrinsic and extrinsic mechanisms to reduce glioma adhesion and increase migration." Journal of Cell Science 133, no. 24 (2020): jcs247189. http://dx.doi.org/10.1242/jcs.247189.

Pełny tekst źródła

Streszczenie:

ABSTRACTA lack of biological markers has limited our ability to identify the invasive cells responsible for glioblastoma multiforme (GBM). To become migratory and invasive, cells must downregulate matrix adhesions, which could be a physical marker of invasive potential. We engineered murine astrocytes with common GBM mutations, e.g. Ink4a (Ink) or PTEN deletion and expressing a constitutively active EGF receptor truncation (EGFRvIII), to elucidate their effect on adhesion. While loss of Ink or PTEN did not affect adhesion, counterparts expressing EGFRvIII were significantly less adhesive. EGFR

Style APA, Harvard, Vancouver, ISO itp.

8

Yamana, Norikazu, Yoshiki Arakawa, Tomohiro Nishino, et al. "The Rho-mDia1 Pathway Regulates Cell Polarity and Focal Adhesion Turnover in Migrating Cells through Mobilizing Apc and c-Src." Molecular and Cellular Biology 26, no. 18 (2006): 6844–58. http://dx.doi.org/10.1128/mcb.00283-06.

Pełny tekst źródła

Streszczenie:

ABSTRACT Directed cell migration requires cell polarization and adhesion turnover, in which the actin cytoskeleton and microtubules work critically. The Rho GTPases induce specific types of actin cytoskeleton and regulate microtubule dynamics. In migrating cells, Cdc42 regulates cell polarity and Rac works in membrane protrusion. However, the role of Rho in migration is little known. Rho acts on two major effectors, ROCK and mDia1, among which mDia1 produces straight actin filaments and aligns microtubules. Here we depleted mDia1 by RNA interference and found that mDia1 depletion impaired dire

Style APA, Harvard, Vancouver, ISO itp.

9

Wang, Shujie, Takashi Watanabe, Kenji Matsuzawa, et al. "Tiam1 interaction with the PAR complex promotes talin-mediated Rac1 activation during polarized cell migration." Journal of Cell Biology 199, no. 2 (2012): 331–45. http://dx.doi.org/10.1083/jcb.201202041.

Pełny tekst źródła

Streszczenie:

Migrating cells acquire front-rear polarity with a leading edge and a trailing tail for directional movement. The Rac exchange factor Tiam1 participates in polarized cell migration with the PAR complex of PAR3, PAR6, and atypical protein kinase C. However, it remains largely unknown how Tiam1 is regulated and contributes to the establishment of polarity in migrating cells. We show here that Tiam1 interacts directly with talin, which binds and activates integrins to mediate their signaling. Tiam1 accumulated at adhesions in a manner dependent on talin and the PAR complex. The interactions of ta

Style APA, Harvard, Vancouver, ISO itp.

10

Kim, Sarah Hyun Ji, and Daniel A. Hammer. "Integrin crosstalk allows CD4+ T lymphocytes to continue migrating in the upstream direction after flow." Integrative Biology 11, no. 10 (2019): 384–93. http://dx.doi.org/10.1093/intbio/zyz034.

Pełny tekst źródła

Streszczenie:

Abstract In order to perform critical immune functions at sites of inflammation, circulatory T lymphocytes must be able to arrest, adhere, migrate and transmigrate on the endothelial surface. This progression of steps is coordinated by cellular adhesion molecules (CAMs), chemokines, and selectins presented on the endothelium. Two important interactions are between Lymphocyte Function-associated Antigen-1 (LFA-1) and Intracellular Adhesion Molecule-1 (ICAM-1) and also between Very Late Antigen-4 (VLA-4) and Vascular Cell Adhesion Molecule-1 (VCAM-1). Recent studies have shown that T lymphocytes

Style APA, Harvard, Vancouver, ISO itp.

Więcej źródeł

Oferujemy zniżki na wszystkie plany premium dla autorów, których prace zostały uwzględnione w tematycznych zestawieniach literatury. Skontaktuj się z nami, aby uzyskać unikalny kod promocyjny!