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Artykuły w czasopismach na temat "Muscle rigidity"

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Matsuki, Yuka. "Muscle Rigidity Associated with Pregabalin." Pain Physician 3;15, no. 3;5 (2012): E350. http://dx.doi.org/10.36076/ppj.2012/15/e350.

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Jenkins, J. G. "Masseter muscle rigidity after vecuronium." European Journal of Anaesthesiology 16, no. 2 (1999): 137–39. http://dx.doi.org/10.1097/00003643-199902000-00011.

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Smith, H. L., and G. R. Park. "Muscle rigidity in meningococcal meningitis." Anaesthesia 48, no. 12 (2007): 1103–4. http://dx.doi.org/10.1111/j.1365-2044.1993.tb07544.x.

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Manners, J. M. "Muscle rigidity in miningococcal meningitis." Anaesthesia 49, no. 6 (1994): 544. http://dx.doi.org/10.1111/j.1365-2044.1994.tb03537.x.

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Jenkins, J. G. "Masseter muscle rigidity after vecuronium." European Journal of Anaesthesiology 16, no. 2 (1999): 137–39. http://dx.doi.org/10.1046/j.1365-2346.1999.00448.x.

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Markelov, Grigory I. "To the symptomatology of trembling paralysis." Neurology Bulletin XVI, no. 2 (2022): 237–48. http://dx.doi.org/10.17816/nb101118.

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The main feature in the clinical picture of trembling paralysis must be recognized as increased rigidity of the muscles, muscle stiffness. This latter imposes on the patient that peculiar imprint, which often makes it possible to recognize this disease by appearance alone. Giving in various cases one or another fluctuation in distribution and intensity, this increased rigidity of the muscles is the most characteristic symptom of Parkinson's disease.
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Vankova, Miriana E., Matthew B. Weinger, Dong-Yi Chen, J. Brian Bronson, Valerie Motis, and George F. Koob. "Role Central Mu, Delta-1, and Kappa-1 Opioid Receptors in Opioid-induced Muscle Rigidity in the Rat." Anesthesiology 85, no. 3 (1996): 574–83. http://dx.doi.org/10.1097/00000542-199609000-00017.

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Background Opioids appear to produce their physiologic effects by binding to at least three types of opioid receptors, the mu (mu), delta (delta), and kappa (kappa) receptors. Muscle rigidity occurs after administration of supra-analgesic doses of potent mu-preferring agonists like alfentanil. The role of different supraspinal opioid receptors in this rigidity has been addressed only recently. To elucidate the contribution of central mu, delta, and kappa receptors to muscle rigidity, the effects of intracerebroventricularly administered opioid receptor-selective agonists and antagonists on alf
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Muralinath, E., Sharlet E. Sony, Naidu K. Mohan, et al. "Exploring the Impact of Drugs on Decerebrate Rigidity." Journal of Advances in Drug Discovery and Development 2, no. 2 (2024): 45–47. https://doi.org/10.5281/zenodo.11174365.

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<em>Decerebrate rigidity happens because of the damage to the brain stem, disrupting the normal inhibitory pathways that regulate muscle tone .Decerebrate rigidity is a state of disputed signals between the brain and the Spinal cord particularly involving the reticular formation in the brain stem. A very few drugs can target various transmitter systems participated in motor control such as GABA, glutamate , dopamine and serotonin. Levo dopa or dopamine agonists have been investigating d for their potential to improve motor symptoms associated with decerebrate rigidity. NMDA receptor antagonist
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Khan, Bashir A., Mazhar U. Khan, Md Umar Majid, and Md Mohib Hussain. "Masseter Muscle Rigidity Following Succinylcholine Administration." Journal of Contemporary Medicine and Dentistry 2, no. 2 (2014): 64–68. http://dx.doi.org/10.18049/jcmad/229a14.

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&NA;. "Benzodiazepines attenuate alfentanil-induced muscle rigidity." Inpharma Weekly &NA;, no. 972 (1995): 13. http://dx.doi.org/10.2165/00128413-199509720-00028.

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Rozprawy doktorskie na temat "Muscle rigidity"

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張劍強 and Kim-keung Cheung. "The effect of hamstring stretching technique on hamstring flexibility and isokinetic strength." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B3125715X.

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Cheung, Kim-keung. "The effect of hamstring stretching technique on hamstring flexibility and isokinetic strength /." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk:8888/cgi-bin/hkuto%5Ftoc%5Fpdf?B23425374.

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Saidi, Azadeh. "Robotic Evaluation Of Rigidity In Parkinson's As A Function Of Speed-Comparison To Clinical Scales." VCU Scholars Compass, 2005. http://scholarscompass.vcu.edu/etd_retro/147.

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Rigidity is one of the cardinal symptoms in Parkinson's disease, along with Bradykinesia, tremor and postural instability. Rigidity in PD has been understudied, but its pathophysiological basis remains unclear. Various types of neurophysiological and biomechanical approach have been developed in order to investigate the neural control of muscle tone. A common approach is to observe the sensitivity of muscle resistance in response to stretch velocity or displacement [Kamper, Rea, He]. A recent study on elbow flexors in patients with spasticity and rigidity showed a velocity dependent increase i
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Dowler, Elizabeth Safety Science Faculty of Science UNSW. "Effects of neutral posture on muscle tension, pain and performance for computer users." Awarded by:University of New South Wales, 1998. http://handle.unsw.edu.au/1959.4/37113.

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This study focuses on developing a new approach to seated work positions. It was conducted on 67 office workers who use the Video Display Terminal (VDT) as a major function of their working day. Muscle tension was measured by surface electromyography when subjects were asked to adopt four selected working postures. Pain was measured before and after ergonomic intervention on the Nordic scale, which was modified for this study. Performance was measured on timed typing tests. A psychosocial questionnaire was used to determine influences of job demands, and a diagnostic assessment was performed t
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Graham, Daniel Joseph. "The Long Term Effects of Short-Wave Diathermy and Long-Duration Static Stretch on Hamstring Flexibility." Diss., CLICK HERE for online access, 2004. http://contentdm.lib.byu.edu/ETD/image/etd624.pdf.

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Stecco, Antonio. "Ialuronidasi per la rigidità muscolare nella spasticità Hyaluronidase for muscle stiffness in spasticity." Doctoral thesis, Università degli studi di Padova, 2016. http://hdl.handle.net/11577/3424238.

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Background: Spasticity is a common neurological impairment after injury to the central nervous system, but the neural and biomechanical contributions to it are still poorly understood. Histopathological studies have demonstrated a generalized increase in extracellular connective tissue in spastic muscles, which can decrease its compliance, and reduce the threshold for stimulation of the spindle receptors. Here we propose and provide preliminary evidence for a novel hypothesis for exacerbation of spasticity in an immobilized limb - the hyaluronan hypothesis. We hypothesize that the extracellula
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Rouleau, André-Jean. "Influence de la rigidité du microenvironnement sur les cellules progénitrices myogéniques du muscle squelettique." Mémoire, Université de Sherbrooke, 2016. http://hdl.handle.net/11143/9491.

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La matrice extracellulaire (MEC) subit plusieurs modifications au cours du vieillissement, ce qui altère ses propriétés biomécaniques. Les cellules responsables de la régénération de la portion myogénique du muscle sont les cellules satellites, qui, une fois activées, sont appelées les cellules progénitrices myogéniques (CPM). La rigidité du muscle, influence le devenir des CPM. La capacité régénérative du muscle squelettique diminue lors du vieillissement. Nous avons posé l’hypothèse selon laquelle la rigidité observée dans le tissu âgé pourrait nuire à la capacité régénérative des CPM. Nous
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Belozertseva, Ekaterina. "Effets du récepteur minéralocorticoïde, de l’intégrine αv et de vimentine sur les fonctions des cellules musculaires lisses vasculaires et la rigidité artérielle". Thesis, Université de Lorraine, 2016. http://www.theses.fr/2016LORR0165/document.

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La rigidité artérielle et la fibrose ont une valeur prédictive dans le développement des maladies cardiovasculaires (CV). Ces 2 phénotypes impliquent les cellules musculaires lisses vasculaires (CMLVs) notamment des récepteurs membranaires et les protéines du cytosquelette. Les objectifs ont été d’étudier : (i) l’influence du récepteur minéralocorticoïde (MR) sur la réactivité vasculaire, (ii) le rôle de l’intégrine αvβ3 dans le développement de la rigidité artérielle et la fibrose vasculaire, et (iii) l’impact de la vimentine et la synémine sur la structure et la fonction artérielle. Ces troi
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Stuyvers, Bruno. "Modélisation et description du comportement instantané de la rigidité du muscle cardiaque : application à l'hypertrophie ventriculaire gauche expérimentale consécutive à une hypertension rénovasculaire chez le rat (modèle 2K-1C R.H.R)." Bordeaux 2, 1991. http://www.theses.fr/1991BOR28154.

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La methodologie developpee au cours de ce travail permet la modelisation du comportement de la rigidite face a la contrainte d'un muscle papillaire (isole de cur de rat) a un moment determine de son activation. Appliquee a differents instants de l'activite du muscle stimule, cette methodologie rend compte de l'evolution de la relation rigidite-contrainte en fonction du degre d'activation. La meme procedure est appliquee au muscle papillaire de cur hypertrophie (hypertrophie induite par hypertension renovasculaire: modele 2k-1c rhr). Les resultats obtenus montrent que dans certaines conditions
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Hovhannisyan, Yeranuhi. "Modélisation cardiaque des myopathies myofibrillaires à l'aide de cellules souches pluripotentes induites pour explorer la pathogenèse cardiaque Polyacrylamide Hydrogels with Rigidity-Independent Surface Chemistry Show Limited Long-Term Maintenance of Pluripotency of Human Induced Pluripotent Stem Cells on Soft Substrates Modéliser la myopathie myofibrillaire pour élucider la pathogenèse cardiaque Synemin-related skeletal and cardiac myopathies: an overview of pathogenic variants Desmin prevents muscle wasting, exaggerated weakness and fragility, and fatigue in dystrophic mdx mouse Effects of the selective inhibition of proteasome caspase-like activity by CLi a derivative of nor-cerpegin in dystrophic mdx mice." Thesis, Sorbonne université, 2020. http://www.theses.fr/2020SORUS095.

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La myopathie myofibrillaire est une maladie neuromusculaire à évolution lente caractérisée par de graves troubles musculaires causés par des mutations dans le gène codant pour des protéines du cytosquelette. L'un des gènes affectés en relation avec le développement de la MFM est DES. Des mutations dans le gène de la desmine entraînent des myopathies des muscles squelettiques et cardiaques. Cependant, les évènements qu'elles entraînent et qui sont à l’origine des phénotypes pathologiques cardiaques restent mal connus. Mon objectif est de créer un modèle in vitro de MFM basé sur des cellules sou
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Książki na temat "Muscle rigidity"

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Avela, Janne. Stretch-reflex adaptation in man. University of Jyväskylä, 1998.

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Gelb, Harold. Killing painwithout prescription: A new and simple way to free yourself from headache, backache and other sources of chronic pain. Thorsons, 1987.

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Dreaver, Jim. Somatic therapy: A neuromuscular approach to chronic pain and stiffness. 2nd ed. Wild Goose Press, 1991.

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Fujimoto, Yasushi. He ji rou suan tong shuo bai bai! man hua tu jie jian, jing, yao, bei man xing teng tong quan xiao chu. Ren lei zhi ku shu wei ke ji wen hua you xian gong si, 2016.

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Muscles, Masses and Motion: The Physiology of Normality, Hypotonicity, Spasticity and Rigidity (Clinics in Developmental Medicine (Mac Keith Press)). Cambridge University Press, 1993.

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Stafstrom, Carl E. Disorders Caused by Botulinum Toxin and Tetanus Toxin. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0156.

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Anaerobic organisms of the genus Clostridia (C) can cause significant human disease. Exotoxins secreted by C botulinum and C tetani cause botulism and tetanus, respectively (summarized in Table 156.1). Botulinum neurotoxin causes neuromuscular blockade by interfering with vesicular acetylcholine release, leading to cholinergic blockade at the neuromuscular junctions of skeletal muscle, and consequently, symmetric flaccid paralysis. Tetanus toxin prevents release of inhibitory neurotransmitters at central synapses, leading to overactivity of motor neurons and muscle rigidity and spasms. This ch
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Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 30-Year-Old Male with Severe Cramps. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0029.

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Muscle cramps are a common complaint. When not associated with weakness, the course is usually benign but can still be frustrating to patients. Diffuse cramping may be indicative of a neuromuscular disorder. Cramps can be associated with a metabolic or endocrine disorder or medications. Normal nerve conduction studies and electromyography are reassuring for a benign cause of cramps. In motor neuron disease cramps are common. There are rare spontaneous activity syndromes associated with cramping. Stiff person syndrome is rare and usually has severe axial rigidity. It can be treated with immune
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Suzzen, Marrinaz Ton. Serotonin Syndrome Symptoms: Hyperthermia,Agitation and Restlessness,Ocular Clonus,Muscle Rigidity,Headaches,Tremor,Nausea, Vomiting, and Diarrhea,Heavy Sweating,Shivering and Goose Bumps,High Blood Pressure and Rapid Heart Rate. Independently Published, 2021.

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Muscles, Masses and Motion:The Physiology of Normality, Hypotonicity, Spasticity and Rigidity (Classics in Developmental Medicine). MacKeith Press, 1992.

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Części książek na temat "Muscle rigidity"

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Struppler, A., and C. Jakob. "Measurement of Muscle Tone — Demarcation Between Spasticity and Rigidity." In Instrumental Methods and Scoring in Extrapyramidal Disorders. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-78914-4_6.

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Hemsley, Kim M., and Ann D. Crocker. "Antipsychotic Drug-Induced Muscle Rigidity and D2 Receptor Occupancy in the Basal Ganglia of the Rat." In Advances in Behavioral Biology. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0179-4_52.

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Goldberg, Joshua A., Thomas Boraud, Sharon Maraton, Eilon Vaadia, and Hagai Bergman. "Enhanced Synchrony in the Primary Motor Cortex of Mptp Primates May Underlie Muscle Co-Contraction and Rigidity." In Advances in Behavioral Biology. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0715-4_11.

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Lorenc-Koci, E., G. Schulze, M. Śmiałowska, et al. "Chronic Treatment with 1,2,3,4-Tetrahydroiso-Quinoline (TIQ), an Environmental and Endogenous Substance, Induces Parkinsonian like Muscle Rigidity in Young and Old Rats." In Neurotoxic Factors in Parkinson’s Disease and Related Disorders. Springer US, 2000. http://dx.doi.org/10.1007/978-1-4615-1269-1_40.

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"Fever, muscle rigidity, mental confusion." In 100 Cases in Psychiatry. CRC Press, 2010. http://dx.doi.org/10.1201/b13316-44.

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Vargas-Patron, Luis A., and Jessica A. Lovich-Sapola. "Malignant Hyperthermia and Masseter Muscle Rigidity." In Anesthesia Oral Board Review, 2nd ed. Cambridge University Press, 2023. http://dx.doi.org/10.1017/9781316181492.081.

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Baliga, Narayan, and Theodore J. Sanford. "Difficult Airway: Opiate-Induced Muscle Rigidity." In Complications in Anesthesia. Elsevier, 2007. http://dx.doi.org/10.1016/b978-1-4160-2215-2.50046-6.

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Miller, Aaron E., Tracy M. DeAngelis, Michelle Fabian, and Ilana Katz Sand. "Gad-Awful Spasms." In Neuroimmunology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190693190.003.0019.

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Stiff person syndrome (SPS) is a rare autoimmune neurological disease that causes progressive, fluctuating, painful muscle rigidity and spasms, generally beginning in the axial muscles, truncal region, and progressing to proximal musculature. A hyperlordotic posture is considered an early clinical hallmark of the condition. The most common pathological correlate is with anti-glutamic acid decarboxylase (GAD) 65 antibodies, but several other associated antibodies have been identified There is a clinical spectrum of disease with variants including stiff leg syndrome, progressive encephalomyeliti
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Shibasaki, Hiroshi, Mark Hallett, Kailash P. Bhatia, Stephen G. Reich, and Bettina Balint. "Disorders of Increased Muscle Stiffness or Overactivity." In Involuntary Movements. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190865047.003.0009.

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Increased muscle stiffness or overactivity (which differs from parkinsonian rigidity or dystonia) can be caused by a variety of disorders of central or peripheral nervous system origin and genetic, autoimmune, or infectious etiology. Increased muscle stiffness may be the cause of some joint movements, particularly when such stiffness is associated with stimulus sensitivity that causes involuntary movements. The conditions discussed in this chapter include stiff person syndrome, progressive encephalomyelitis with rigidity and myoclonus (PERM), acquired neuromyotonia (Isaacs syndrome), hereditar
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Devine, Michelle F., and A. Sebastian Lopez Chiriboga. "Stiffness, Spasms, and Frequent Falls in a 41-Year-Old Man." In Mayo Clinic Cases in Neuroimmunology, edited by Andrew McKeon, B. Mark Keegan, and W. Oliver Tobin. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780197583425.003.0037.

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A 41-year-old man sought care for 3 years of right-sided muscle stiffness. He also had 5- to 10-minute episodes of severe muscle spasms. He noted development of daily episodes of sudden, severe stiffness, often triggered by unexpected stimuli (eg, a touch or loud sound). He started using a walker and stopped driving. He stopped working because of increasing difficulty with mobility and cognition. On neurologic examination, he had a Kokmen Short Test of Mental Status score of 28/38, with points lost for orientation, attention, calculation, and recall. Cranial nerve examination showed bilateral
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Streszczenia konferencji na temat "Muscle rigidity"

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Ueda, Shodai, and Atsushi Sakuma. "Finite Element Analysis of Dynamics of Human Muscle Compressed by Fabric Sleeve." In ASME 2018 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/imece2018-87304.

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Recently, compression wear has become the preferred performance material for many athletes, where it has the effect of reducing the burden on the body by suppressing muscle vibrations and improves athletic performance by providing the body with suitable moderate pressure. This study concerns thigh sleeves formed of compression wear. The optimal level of compression is studied in order to improve athletic performance and reduce muscular strain. Subsequently, the mechanics of the thigh compression sleeve are discussed. Here, the optimal tensile rigidity of the sleeve, which is calculated using t
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Ayuni, C. Z. Noor, Takashi Komeda, Cheng Yee Low, and Kaoru Inoue. "Emulation of muscle tone of upper limb spasticity and rigidity." In 2013 6th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2013. http://dx.doi.org/10.1109/ner.2013.6696252.

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Song, Seung Yun, Yinan Pei, Jiahui Liang, and Elizabeth T. Hsiao-Wecksler. "Design of a Portable Position, Velocity, and Resistance Meter (PVRM) for Convenient Clinical Evaluation of Spasticity or Rigidity." In 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3503.

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Spasticity is a common consequence of the upper motor neuron syndrome and usually associated with brain lesion, stroke, cerebral palsy, spinal cord injury, and etc. On the other hand, rigidity is a neuromuscular disorder often found in Parkinson’s disease patients. Both of spasticity and rigidity are characterized by abnormal hypertonic muscle behaviors that will cause discomfort and hinder daily activities. Worldwide, the estimated affected population of spasticity is around 12 million [1], and rigidity affects more than 10 million people [2]. Clinical evaluation of spasticity or rigidity inv
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He, Maxine, Mahshid Mansouri, Yinan Pei, Isaac Pedroza, Christopher M. Zallek, and Elizabeth T. Hsiao-Wecksler. "Clinical Validation Testing Of An Upper Limb Robotic Medical Education Training Simulator For Rigidity Assessment." In 2022 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2022. http://dx.doi.org/10.1115/dmd2022-1073.

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Abstract An upper limb robotic training simulator was developed to replicate the haptic feeling of lead-pipe rigidity of the biceps. Rigidity is the increased muscle tone observed during passive movement of a joint. To validate the realism of our training simulator, a clinical validation study was conducted with 11 experienced clinicians. Testing involved two parts: Blinded Assessment followed by Disclosed Assessment. There were 12 randomized trials (4 levels of rigidity with 3 repetitions each) in the Blind Assessment. The participants were asked to rate the rigidity level using the Unified P
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Garavaglia, Lorenzo, Elena Beretta, Sandra Strazzer, et al. "Dynamic Splints, Functionally-Customized With Nitinol, Can Reduce Joint Rigidity in Pediatric Subjects With Spasticity." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14246.

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Neuromuscular diseases as a consequence of brain damage are complex phenomena involving disuse, immobility, brain tissue remodeling and cortical function remapping. They may have various causes and strike any part of the population. The vicious circle leading to a worsening of the patients’ conditions proceeds through muscle shortening by contractures, disruption of the normal reflex behavior and sensory problems, development of spasticity [1]. Physical rehabilitation alone or in association with surgery or pharmacological treatments can be useful in limiting those degenerations. Besides manua
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Song, Seung Yun, Yinan Pei, Steven R. Tippett, Dronacharya Lamichhane, Christopher M. Zallek, and Elizabeth T. Hsiao-Wecksler. "Validation of a Wearable Position, Velocity, and Resistance Meter for Assessing Spasticity and Rigidity." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6906.

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Patients with neuromuscular disorders such as Parkinson’s disease (PD), traumatic brain or spinal cord injury, or multiple sclerosis (MS) can develop different levels of abnormal muscle behavior (hypertonia) such as rigidity and spasticity [1], [2]. Hypertonia can affect different parts of the body such as upper or lower extremities. Symptoms include pain, increased muscle tone, spasms, and decreased functional abilities. Hypertonia can interfere with many activities of daily living, greatly affecting the quality of life in patients and causing anxiety, depression, and social isolation [2].
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Nyugen, Joey, Shenbagaraj Kannapiran, Subhrajyoti Chaudhuri, Valerie Lane Gentz, and Panagiotis Polygerinos. "Design of a Soft Ankle Joint Device for Correction of Inversion/Eversion Angle During Aquatic Therapy." In 2019 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/dmd2019-3206.

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According to statistical data, approximately 800,000 individuals across the United States have strokes each year [2]. A stroke event causes neurological and orthopedic deficits, such as weak muscles, decreased proprioception, and spasticity [6]. To regain function, increase motor skills, and retrain muscles, many stoke survivors utilize aquatic therapy as a form of rehabilitation [14]. Typically inside water, the lower body part of a person has to carry 75% less weight, This decreases the effect of gravity allowing increased joint range of motion [6], [13]. This also helps increase muscle stre
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Oppold, Julia, Alexander Grimm, Philipp Klocke, et al. "Muscle ultrasound in idiopathic Parkinson's disease with deep brain stimulation: Rigidity can be quantified by shear wave elastography." In Interdisziplinärer Kongress | Ultraschall 2022. Georg Thieme Verlag, 2022. http://dx.doi.org/10.1055/s-0042-1749497.

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Oliveira Júnior, Rocymar Rebouças, Ana Carolina Soares de Lira, Nilson Batista Lemos, et al. "The Non-motor Effects of Deep Brain Stimulation of the Subthalamic Nucleus in Patients with Motor Disorders Caused by Parkinson’s Disease." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.655.

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Introduction: Parkinson’s disease is a disease caused by the degeneration of dopaminergic neurons in the substantia nigra, and is characterized by a triad of symptoms: bradykinesia, muscle rigidity and rest tremors; which worsen progressively, leading the patient to resort to surgical treatment to ensure a better drug response. However, surgical intervention has proven to be efficient not only to alleviate Movement Disorders, but also to control the nonmotor symptoms of the disease. Objective: To evaluate the non-motor effects of Deep Brain Stimulation (DBS) in patients who do not respond adeq
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Maynard, Jacqueline A., Ahmad S. Arabiyat, Anna Elefante, Lucas Shearer, Eoin King, and Andrea Kwaczala. "Using Acoustic Waves to Modulate Stem Cell Growth and Differentiation." In ASME 2017 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/imece2017-71341.

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During spaceflight, the loss of mechanical loads due to microgravity leads to rapid bone loss, where bone deteriorates at a rate of 1–2% per month, where some astronauts can lose as much as 20% of their skeletal mass in a single expedition [NASA, 2001]. In order to prevent muscle and bone loss, long-term space flight exercise regimes are strictly implemented [Shackleford, 2004]. Current research has demonstrated that mechanical vibrations can help to maintain or improve bone mass [Chan, 2013] and reduce adiposity [Chen, 2015, Sen, 2011] when signals are applied at the appropriate frequency and
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