Gotowe bibliografie tematyczne / Neuroretinal organoids

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  1. Artykuły w czasopismach
  2. Rozprawy doktorskie

Gotowa bibliografia na temat „Neuroretinal organoids”

Autor: Grafiati
Data publikacji: 21 grudnia 2024
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Neuroretinal organoids"

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Atac, David, Kevin Maggi, Silke Feil, et al. "Identification and Characterization of ATOH7-Regulated Target Genes and Pathways in Human Neuroretinal Development." Cells 13, no. 13 (2024): 1142. http://dx.doi.org/10.3390/cells13131142.

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The proneural transcription factor atonal basic helix–loop–helix transcription factor 7 (ATOH7) is expressed in early progenitors in the developing neuroretina. In vertebrates, this is crucial for the development of retinal ganglion cells (RGCs), as mutant animals show an almost complete absence of RGCs, underdeveloped optic nerves, and aberrations in retinal vessel development. Human mutations are rare and result in autosomal recessive optic nerve hypoplasia (ONH) or severe vascular changes, diagnosed as autosomal recessive persistent hyperplasia of the primary vitreous (PHPVAR). To better un
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Takata, Nozomu, Deepti Abbey, Luciano Fiore, et al. "An Eye Organoid Approach Identifies Six3 Suppression of R-spondin 2 as a Critical Step in Mouse Neuroretina Differentiation." Cell Reports 21, no. 6 (2017): 1534–49. http://dx.doi.org/10.1016/j.celrep.2017.10.041.

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Afting, Cassian, Tobias Walther, Oliver M. Drozdowski, et al. "DNA microbeads for spatio-temporally controlled morphogen release within organoids." Nature Nanotechnology, September 9, 2024. http://dx.doi.org/10.1038/s41565-024-01779-y.

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AbstractOrganoids are transformative in vitro model systems that mimic features of the corresponding tissue in vivo. However, across tissue types and species, organoids still often fail to reach full maturity and function because biochemical cues cannot be provided from within the organoid to guide their development. Here we introduce nanoengineered DNA microbeads with tissue mimetic tunable stiffness for implementing spatio-temporally controlled morphogen gradients inside of organoids at any point in their development. Using medaka retinal organoids and early embryos, we show that DNA microbe
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Liu, Wei, Rupendra Shrestha, Albert Lowe, Xusheng Zhang, and Ludovic Spaeth. "Self-formation of concentric zones of telencephalic and ocular tissues and directional retinal ganglion cell axons." eLife 12 (September 4, 2023). http://dx.doi.org/10.7554/elife.87306.3.

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The telencephalon and eye in mammals are originated from adjacent fields at the anterior neural plate. Morphogenesis of these fields generates telencephalon, optic-stalk, optic-disc, and neuroretina along a spatial axis. How these telencephalic and ocular tissues are specified coordinately to ensure directional retinal ganglion cell (RGC) axon growth is unclear. Here, we report self-formation of human telencephalon-eye organoids comprising concentric zones of telencephalic, optic-stalk, optic-disc, and neuroretinal tissues along the center-periphery axis. Initially-differentiated RGCs grew axo
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Isla-Magrané, Helena, Maddalen Zufiaurre-Seijo, José García-Arumí, and Anna Duarri. "All-trans retinoic acid modulates pigmentation, neuroretinal maturation, and corneal transparency in human multiocular organoids." Stem Cell Research & Therapy 13, no. 1 (2022). http://dx.doi.org/10.1186/s13287-022-03053-1.

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Abstract Background All-trans retinoic acid (ATRA) plays an essential role during human eye development, being temporally and spatially adjusted to create gradient concentrations that guide embryonic anterior and posterior axis formation of the eye. Perturbations in ATRA signaling can result in severe ocular developmental diseases. Although it is known that ATRA is essential for correct eye formation, how ATRA influences the different ocular tissues during the embryonic development of the human eye is still not well studied. Here, we investigated the effects of ATRA on the differentiation and
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Ye, Xiuhong, Sihui Chen, Wei Xiong, et al. "Magnetic‐Guided Delivery of Antisense Oligonucleotides for Targeted Transduction in Multiple Retinal Explant and Organoid Models." Advanced Science, April 25, 2025. https://doi.org/10.1002/advs.202417363.

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AbstractAntisense oligonucleotide (ASO) therapy holds promise in gene therapy but faces challenges due to poor delivery efficiency and limited evaluation models. This investigation employs magnetic nanoparticles (MNPs) to augment the delivery efficiency of ASOs. It assesses their distribution and therapeutic efficacy across various models, including retinal explants from mice and macaques or human retinal and inner ear organoids. Retinal explants from both mice and monkeys are methodically arranged to expose the ganglion cell layer (GCL) or the photoreceptor layer (PL). MNPs markedly enhanced
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Rozprawy doktorskie na temat "Neuroretinal organoids"

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Frank, Elie. "Modélisation du Syndrome d'Alström à partir de cellules souches pluripotentes humaines pour l'identification de cibles moléculaires d'intérêt thérapeutique." Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASQ041.

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Le syndrome d'Alström (SA) est une maladie monogénique récessive multi-systémique, caractérisée notamment par une perte de l'audition et de la vue, une obésité, un diabète de type 2, une cardiomyopathie et une insuffisance hépatique et rénale progressive. Les symptômes affectant la vision se développent dès les premières semaines après la naissance et mènent progressivement à une perte totale de la vue. À l'heure actuelle, aucun traitement ne permet de soigner cette maladie et seules des solutions permettant de réduire les effets des symptômes peuvent être proposées.L'objet de la thèse est de
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