Gotowe bibliografie tematyczne / Normobaric oxygen paradox (NOP)

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Gotowa bibliografia na temat „Normobaric oxygen paradox (NOP)”

Autor: Grafiati
Data publikacji: 16 listopada 2024
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Normobaric oxygen paradox (NOP)"

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Lafère, Pierre, Thomas Schubert, David De Bels, Peter Germonpré, and Costantino Balestra. "Can the normobaric oxygen paradox (NOP) increase reticulocyte count after traumatic hip surgery?" Journal of Clinical Anesthesia 25, no. 2 (2013): 129–34. http://dx.doi.org/10.1016/j.jclinane.2012.06.021.

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Fratantonio, Deborah, Fabio Virgili, Alessandro Zucchi, et al. "Increasing Oxygen Partial Pressures Induce a Distinct Transcriptional Response in Human PBMC: A Pilot Study on the “Normobaric Oxygen Paradox”." International Journal of Molecular Sciences 22, no. 1 (2021): 458. http://dx.doi.org/10.3390/ijms22010458.

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The term “normobaric oxygen paradox” (NOP), describes the response to the return to normoxia after a hyperoxic event, sensed by tissues as oxygen shortage, and resulting in up-regulation of the Hypoxia-inducible factor 1α (HIF-1α) transcription factor activity. The molecular characteristics of this response have not been yet fully characterized. Herein, we report the activation time trend of oxygen-sensitive transcription factors in human peripheral blood mononuclear cells (PBMCs) obtained from healthy subjects after one hour of exposure to mild (MH), high (HH) and very high (VHH) hyperoxia, c
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Balestra, Costantino, Sara Baldelli, Fabio Virgili, Michele Salvagno, Simona Mrakic-Sposta, and Deborah Fratantonio. "Pulsed Hyperoxia Acts on Plasmatic Advanced Glycation End Products and Advanced Oxidation Protein Products and Modulates Mitochondrial Biogenesis in Human Peripheral Blood Mononuclear Cells: A Pilot Study on the “Normobaric Oxygen Paradox”." International Journal of Molecular Sciences 25, no. 4 (2024): 2394. http://dx.doi.org/10.3390/ijms25042394.

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The “normobaric oxygen paradox” (NOP) describes the response to the return to normoxia after a hyperoxic event, sensed by tissues as an oxygen shortage, up-regulating redox-sensitive transcription factors. We have previously characterized the time trend of oxygen-sensitive transcription factors in human PBMCs, in which the return to normoxia after 30% oxygen is sensed as a hypoxic trigger, characterized by hypoxia-induced factor (HIF-1) activation. On the contrary, 100% and 140% oxygen induce a shift toward an oxidative stress response, characterized by NRF2 and NF-kB activation in the first 2
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Balestra, Costantino, Kate Lambrechts, Simona Mrakic-Sposta, et al. "Hypoxic and Hyperoxic Breathing as a Complement to Low-Intensity Physical Exercise Programs: A Proof-of-Principle Study." International Journal of Molecular Sciences 22, no. 17 (2021): 9600. http://dx.doi.org/10.3390/ijms22179600.

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Inflammation is an adaptive response to both external and internal stimuli including infection, trauma, surgery, ischemia-reperfusion, or malignancy. A number of studies indicate that physical activity is an effective means of reducing acute systemic and low-level inflammation occurring in different pathological conditions and in the recovery phase after disease. As a proof-of-principle, we hypothesized that low-intensity workout performed under modified oxygen supply would elicit a “metabolic exercise” inducing a hormetic response, increasing the metabolic load and oxidative stress with the s
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Debevec, Tadej, Michail E. Keramidas, Barbara Norman, Thomas Gustafsson, Ola Eiken, and Igor B. Mekjavic. "No Evidence For The "Normobaric Oxygen Paradox"." Medicine & Science in Sports & Exercise 43, Suppl 1 (2011): 151. http://dx.doi.org/10.1249/01.mss.0000403127.23300.30.

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Balestra, Costantino, Peter Germonpré, Jacques R. Poortmans, and Alessandro Marroni. "Serum erythropoietin levels in healthy humans after a short period of normobaric and hyperbaric oxygen breathing: the “normobaric oxygen paradox”." Journal of Applied Physiology 100, no. 2 (2006): 512–18. http://dx.doi.org/10.1152/japplphysiol.00964.2005.

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Renal (peritubular) tissue hypoxia is a well-known physiological trigger for erythropoietin (EPO) production. We investigated the effect of rebound relative hypoxia after hyperoxia obtained under normo- and hyperbaric oxygen breathing conditions. A group of 16 healthy volunteers were investigated before and after a period of breathing 100% normobaric oxygen for 2 h and a period of breathing 100% oxygen at 2.5 ATA for 90 min (hyperbaric oxygen). Serum EPO concentration was measured using a radioimmunoassay at various time points during 24–36 h. A 60% increase ( P < 0.001) in serum EPO was ob
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Salvagno, Michele, Giacomo Coppalini, Fabio Silvio Taccone, et al. "The Normobaric Oxygen Paradox—Hyperoxic Hypoxic Paradox: A Novel Expedient Strategy in Hematopoiesis Clinical Issues." International Journal of Molecular Sciences 24, no. 1 (2022): 82. http://dx.doi.org/10.3390/ijms24010082.

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Hypoxia, even at non-lethal levels, is one of the most stressful events for all aerobic organisms as it significantly affects a wide spectrum of physiological functions and energy production. Aerobic organisms activate countless molecular responses directed to respond at cellular, tissue, organ, and whole-body levels to cope with oxygen shortage allowing survival, including enhanced neo-angiogenesis and systemic oxygen delivery. The benefits of hypoxia may be evoked without its detrimental consequences by exploiting the so-called normobaric oxygen paradox. The intermittent shift between hypero
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Keramidas, Michail E., Ola Eiken, and Igor B. Mekjavic. "Prevailing evidence contradicts the notion of a “normobaric oxygen paradox”." European Journal of Applied Physiology 112, no. 12 (2012): 4177–78. http://dx.doi.org/10.1007/s00421-012-2394-9.

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Balestra, C., and P. Germonpré. "Hypoxia, a multifaceted phenomenon: the example of the “Normobaric Oxygen Paradox”." European Journal of Applied Physiology 112, no. 12 (2012): 4173–75. http://dx.doi.org/10.1007/s00421-012-2392-y.

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Balestra, C., and P. Germonpré. "Increasing EPO using the normobaric oxygen paradox: a ‘not so simple’ task." Acta Physiologica 203, no. 2 (2011): 287–88. http://dx.doi.org/10.1111/j.1748-1716.2011.02282.x.

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Rozprawy doktorskie na temat "Normobaric oxygen paradox (NOP)"

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Leveque, Clément. "Etude des variations de pressions partielles d'oxygène sur les réactions cellulaires chez le sujet sain." Electronic Thesis or Diss., Brest, 2024. http://www.theses.fr/2024BRES0040.

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La compréhension de l'impact de l'oxygène sur les organismes révèle des réponses cellulaires complexes. Des études exposant des sujets sains pendant 1 heure à diverses pressions partielles d'oxygène révèlent des différences quantitatives et cinétiques significatives des réponses cellulaires. En hypoxie normobare, un pic de radicaux libres d’oxygène est observé 30 minutes après l’exposition, sans activation immédiate de réponses antioxydantes. L’interleukine-6 augmente progressivement, atteignant un pic à 8h, reflétant une inflammation évolutive. En hyperoxie normobare, la production de radicau
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