Gotowe bibliografie tematyczne / Oxidative damage

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Gotowa bibliografia na temat „Oxidative damage”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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Artykuły w czasopismach na temat "Oxidative damage"

Krisko, Anita, and Miroslav Radman. "Protein damage, ageing and age-related diseases." Open Biology 9, no. 3 (2019): 180249. http://dx.doi.org/10.1098/rsob.180249.

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Ageing is considered as a snowballing phenotype of the accumulation of damaged dysfunctional or toxic proteins and silent mutations (polymorphisms) that sensitize relevant proteins to oxidative damage as inborn predispositions to age-related diseases. Ageing is not a disease, but it causes (or shares common cause with) age-related diseases as suggested by similar slopes of age-related increase in the incidence of diseases and death. Studies of robust and more standard species revealed that dysfunctional oxidatively damaged proteins are the root cause of radiation-induced morbidity and mortalit

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Nunomura, Akihiko, Kazuhiro Honda, Atsushi Takeda, et al. "Oxidative Damage to RNA in Neurodegenerative Diseases." Journal of Biomedicine and Biotechnology 2006 (2006): 1–6. http://dx.doi.org/10.1155/jbb/2006/82323.

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Since 1999, oxidative damage to RNA molecules has been described in several neurological diseases including Alzheimer's disease, Parkinson's disease, Down syndrome, dementia with Lewy bodies, prion disease, subacute sclerosing panencephalitis, and xeroderma pigmentosum. An early involvement of RNA oxidation of vulnerable neuronal population in the neurodegenerative diseases has been demonstrated, which is strongly supported by a recent observation of increased RNA oxidation in brains of subjects with mild cognitive impairment. Until recently, little is known about consequences and cellular han

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Rice-Evans, C., S. C. Omorphos, and E. Baysal. "Sickle cell membranes and oxidative damage." Biochemical Journal 237, no. 1 (1986): 265–69. http://dx.doi.org/10.1042/bj2370265.

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Sickle erythrocytes and their membranes are susceptible to endogenous free-radical-mediated oxidative damage which correlates with the proportion of irreversibly sickled cells. The suppression of incubation-induced oxidative stress by antioxidants, free radical scavengers and an iron chelator suggest that oxidation products of membrane-bound haemoglobin contribute towards the pathology of the disease.

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Singh, Abhishek Kumar, Sandeep Singh, Geetika Garg, and Syed Ibrahim Rizvi. "Rapamycin alleviates oxidative stress-induced damage in rat erythrocytes." Biochemistry and Cell Biology 94, no. 5 (2016): 471–79. http://dx.doi.org/10.1139/bcb-2016-0048.

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An imbalanced cellular redox system promotes the production of reactive oxygen species (ROS) that may lead to oxidative stress-mediated cell death. Erythrocytes are the best-studied model of antioxidant defense mechanism. The present study was undertaken to investigate the effect of the immunosuppressant drug rapamycin, an inducer of autophagy, on redox balance of erythrocytes and blood plasma of oxidatively challenged rats. Male Wistar rats were oxidatively challenged with HgCl2 (5 mg/kg body mass (b.m.)). A significant (p < 0.05) induction in ROS production, plasma membrane redox system (

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Skrypnyk, N. V., and O. O. Maslova. "Oxidative DNA damage." Biopolymers and Cell 23, no. 3 (2007): 202–14. http://dx.doi.org/10.7124/bc.000766.

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Davenport, R. J. "Trash Cache: Secret mitochondrial weapon fights oxidative damage (Oxidative damage)." Science of Aging Knowledge Environment 2002, no. 12 (2002): 41nw—41. http://dx.doi.org/10.1126/sageke.2002.12.nw41.

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DAS, Nilanjana, Rodney L. LEVINE, William C. ORR, and Rajindar S. SOHAL. "Selectivity of protein oxidative damage during aging in Drosophila melanogaster." Biochemical Journal 360, no. 1 (2001): 209–16. http://dx.doi.org/10.1042/bj3600209.

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The purpose of the present study was to determine whether oxidation of various proteins during the aging process occurs selectively or randomly, and whether the same proteins are damaged in different species. Protein oxidative damage to the proteins, present in the matrix of mitochondria in the flight muscles of Drosophila melanogaster and manifested as carbonyl modifications, was detected immunochemically with anti-dinitrophenyl-group antibodies. Aconitase was found to be the only protein in the mitochondrial matrix that exhibited an age-associated increase in carbonylation. The accrual of ox

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Soria-Meneses, Pedro Javier, Alejandro Jurado-Campos, Virgilio Gómez-Rubio, et al. "Determination of Ram (Ovis aries) Sperm DNA Damage Due to Oxidative Stress: 8-OHdG Immunodetection Assay vs. SCSA®." Animals 12, no. 23 (2022): 3286. http://dx.doi.org/10.3390/ani12233286.

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Conventional DNA analysis techniques can hardly detect DNA damage in ruminant spermatozoa due to high DNA compaction in these cells. Furthermore, these techniques cannot discriminate whether the damage is due to oxidative stress. The main purpose of this study was to evaluate the efficacy of two techniques for determining DNA damage in ovine sperm when the source of that damage is oxidative stress. Semen samples from twenty Manchega rams (Ovis aries) were collected and cryopreserved. After thawing, the samples were subjected to different levels of oxidative stress, and DNA oxidation was quanti

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Huang, Yue, Zhiling Li, En Lin, Pei He та Gaizhen Ru. "Oxidative damage-induced hyperactive ribosome biogenesis participates in tumorigenesis of offspring by cross-interacting with the Wnt and TGF-β1 pathways in IVF embryos". Experimental & Molecular Medicine 53, № 11 (2021): 1792–806. http://dx.doi.org/10.1038/s12276-021-00700-0.

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AbstractIn vitro fertilization (IVF) increases the risk of tumorigenesis in offspring. The increased oxidative damage during IVF may be involved in tumor formation. However, the molecular mechanisms underlying this phenomenon remain largely unclear. Using a well-established model of oxidatively damaged IVF mouse embryos, we applied the iTRAQ method to identify proteins differentially expressed between control and oxidatively damaged zygotes and explored the possible tumorigenic mechanisms, especially with regard to the effects of oxidative damage on ribosome biogenesis closely related to tumor

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Jeshan, Milad, Fatemeh Yousefbeyk, Hiva Rahmati, Amir Hosein Shoormeij, Mitra Rezazadeh, and Ehsan Zamani. "Salvia spinosa L. Protects against Diabetes-Induced Nephropathy by Attenuation of Mitochondrial Oxidative Damage in Mice." Advances in Pharmacological and Pharmaceutical Sciences 2021 (December 26, 2021): 1–10. http://dx.doi.org/10.1155/2021/4657514.

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Mitochondrial oxidative damage is a crucial factor in the pathogenesis of diabetic nephropathy (DN), which is among the most prevalent problems of diabetes, and there hasn’t been an effective treatment for DN yet. This study planned to investigate the effects of Salvia spinosa L. on mitochondrial function along with its protection against streptozotocin-induced nephropathy in diabetic mice. After the injection of streptozotocin (STZ) and verification of the establishment of diabetes, mice (n = 30) were randomly divided into the following groups: control group, diabetic-control, S. spinosa-trea

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Rozprawy doktorskie na temat "Oxidative damage"

Yang, Weidong. "Oxidative damage of endothelial cells." Thesis, University of Leicester, 1999. http://hdl.handle.net/2381/29603.

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This study sought to investigate the consequences of different degrees of oxidative stress on endothelial cells, using a cultured endothelial cell model; principally bovine aortic endothelial cells, subjected to oxidative stress. High concentrations of H2O2 or a superoxide generating system caused rapid endothelial cell death, as evidenced by increased membrane permeability, which could be partially protected by myoglobin. Extracellular H2O2 caused a rapid increase in intracellular peroxidation but was also eliminated by endothelial cells. However, the anti-oxidant capacity of the bovine endot

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Soman, Sony. "OXIDATIVE DAMAGE TO DNA IN ALZHEIMER'S DISEASE." UKnowledge, 2013. http://uknowledge.uky.edu/chemistry_etds/28.

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Previous studies from our laboratory and others show a significant increase in levels of both nuclear and mitochondrial DNA and RNA oxidation in vulnerable brain regions in the progression of Alzheimer’s disease (AD). Although total DNA oxidation is increased in AD it remains unclear whether oxidative damage is widespread throughout the genome or is concentrated to specific genes. To test the hypothesis that specific genes are more highly oxidized in the progression of AD, we propose to quantify the percent oxidative damage in genes coding for proteins shown to be altered in the progression of

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Farooq, Sabya. "Free radical induced oxidative DNA damage." Thesis, University of Leicester, 1997. http://hdl.handle.net/2381/30749.

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Oxidative DNA damage has been implicated in processes such as carcinogenesis, mutagenesis, ageing and cell death. Reactive oxygen species (ROS) such as superoxide (O2), hydrogen peroxide (H2O 2) and hydroxyl radical (OH*) are produced in mammalian cells as a result of aerobic metabolism. However excess generation of these species by endogenous or exogenous sources can result in damage to DNA, producing a large number of sugar and base lesions. In order to understand the biological consequences of such free radical induced damage it is essential to characterise and quantitate this damage. This

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Furness, Lindsay Jayne. "Energetics, oxidative damage and ageing in birds." Thesis, Available from the University of Aberdeen Library and Historic Collections Digital Resources, 2009. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=25473.

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Jansson, Kristina. "Oxidative damage and the DNA glycosylase MutYH /." Göteborg : Department of Cell and Molecular Biology, University of Gothenburg, 2010. http://hdl.handle.net/2077/22092.

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Davies, John McCartan Caswell. "Oxidative damage in the colon and rectum." Thesis, University of Leeds, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.493554.

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There is considerable supportive evidence to suggest that increased levels of DNA damage are associated with an increased risk of developing neoplastic lesions in the human colon and rectum. Within this thesis, several different topics related to DNA damage were explored in detail, principally using the single cell gel electrophoresis assay (the comet assay) to measure DNA damage both in cell line studies and in human colorectal mucosal biopsies from patients undergoing routine endoscopic examinations of the colon and rectum.

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Konz, John O. (John Otto) 1971. "Oxidative damage to recombinant proteins during production." Thesis, Massachusetts Institute of Technology, 1998. http://hdl.handle.net/1721.1/17472.

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Thesis (Ph.D.)--Massachusetts Institute of Technology, Dept. of Chemical Engineering, 1998.<br>Includes bibliographical references (p. 209-222).<br>Since the introduction of recombinant human insulin nearly two decades ago, recombinant proteins have increasingly been utilized as therapeutic agents. In addition, expression of recombinant proteins is now a common tool used in basic research. Recombinant proteins are subject to many subtle modifications that can affect their properties; among these modifications, oxidative damage is one of the most ubiquitous. Oxidative damage, however, is only

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Renganathan, Kutralanathan. "Oxidative Damage and Age Related Macular Degeneration." Case Western Reserve University School of Graduate Studies / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=case1193002743.

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Carroll, Luke Dean. "Modulation of oxidative damage by selenium compounds." Thesis, The University of Sydney, 2015. http://hdl.handle.net/2123/14124.

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Myeloperoxidase (MPO) is the primary enzyme responsible for the production of strong oxidants by neutrophils in response to pathogens. One of the major oxidants produced is hypochlorous acid (HOCl), which can react with amine groups to form the secondary oxidants N-chloramines. These oxidants play a role in the destruction of pathogens, however they also have the potential to damage host cells, and have been implicated in numerous inflammatory diseases. This Thesis explores the potential for selenium containing compounds and enzymes to act as catalytic oxidant scavengers. Reaction rates betwe

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Cao, Huachuan. "Probe Oxidative Damage in DNA Charge Transfer Process." Diss., Georgia Institute of Technology, 2005. http://hdl.handle.net/1853/6983.

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As a hydrophilic biopolymer, a DNA molecule is surrounded by water molecules in aqueous solution. The charge hopping mechanism indicates the competition between radical cation quenching by water molecules and migration along DNA partially determines the distance and efficiency of charge transport in DNA. Lipid can effectively bind DNA to induce hydrophobic environment around the DNA helix and reduce the water contact with bases in the DNA duplex. Therefore, the effect of water molecules on charge transport can be studied by comparison between nature DNA and DNA-lipid complexes. We synthesized

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Książki na temat "Oxidative damage"

Evans, Mark D., and Marcus S. Cooke, eds. Oxidative Damage to Nucleic Acids. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-72974-9.

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1962-, Evans Mark D., and Cooke Marcus S, eds. Oxidative damage to nucleic acids. Landes Bioscience, 2007.

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Connor, James R., ed. Metals and Oxidative Damage in Neurological Disorders. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-0197-2.

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R, Connor James, ed. Metals and oxidative damage in neurological disorders. Plenum Press, 1997.

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Gadoth, Natan, and Hans Hilmar Göbel, eds. Oxidative Stress and Free Radical Damage in Neurology. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60327-514-9.

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J, Jesaitis Algirdas, Dratz Edward A, and Montana State University (Bozeman, Mont.), eds. The molecular basis of oxidative damage by leukocytes. CRC Press, 1992.

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Beal, M. Flint. Mitochondrial dysfunction and oxidative damage in neurodegenerative diseases. R.G. Landes Co., 1995.

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Hannon-Fletcher, Mary Philomena Anne. Oxidative stress and biomolecule damage in human IDDM. The Author], 1999.

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J, Lunec, ed. Measuring in vivo oxidative damage: A practical approach. Wiley, 2000.

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-H, Goebel H., ed. Oxidative stress and free radical damage in neurology. Humana Press, 2011.

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Części książek na temat "Oxidative damage"

Guengerich, F. Peter. "Oxidative DNA Damage." In Molecular Life Sciences. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-6436-5_441-1.

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Collins, Andrew R. "Oxidative DNA Damage." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_4306.

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Guengerich, Frederick Peter. "Oxidative DNA Damage." In Molecular Life Sciences. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-1531-2_441.

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Khelfi, A. "Biomarkers of Oxidative Damage." In Biomarkers of Oxidative Stress. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-60738-7_3.

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Cchjda, Koh. "ROS damage to protein." In Experimental protocols for reactive oxygen and nitrogen species. Oxford University PressOxford, 2000. http://dx.doi.org/10.1093/oso/9780198506683.003.0074.

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Abstract Several lines of evidence indicate that protein oxidation by free radicals and the subsequent accumulation of oxidatively modified proteins, which could be an early indication of oxygen radical-mediated tissue damage, have been found in cells during ageing, after oxidative stress, and in various pathological states including premature ageing diseases, muscular dystrophy, rheumatoid arthritis, and atherosclerosis (1). Furthermore, free radical-mediated oxidative inactivation of enzymes, especially by metal-catalysed oxidation systems, have been postulated as marking steps in enzyme tur

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"Oxidative Damage." In Encyclopedia of Biophysics. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-16712-6_100715.

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Balin, A. K., and M. Vilenchik. "Oxidative Damage." In Encyclopedia of Gerontology. Elsevier, 2007. http://dx.doi.org/10.1016/b0-12-370870-2/00144-x.

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"Oxidative Damage." In Dictionary of Concrete Technology. Springer Nature Singapore, 2025. https://doi.org/10.1007/978-981-97-2998-2_528.

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STERN, ARNOLD. "Red Cell Oxidative Damage." In Oxidative Stress. Elsevier, 1985. http://dx.doi.org/10.1016/b978-0-12-642760-8.50018-1.

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"DNA Oxidative Damage." In Dictionary of Toxicology. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-99-9283-6_762.

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Streszczenia konferencji na temat "Oxidative damage"

Liu, Qi, Jie Wei, and Yuan Wang. "Triphenyltin induced oxidative damage in sea urchin Glyptocidaris crenularis." In Fifth International Conference on Green Energy, Environment, and Sustainable Development, edited by Mohammadreza Aghaei, Hongyu Ren, and Xiaoshuan Zhang. SPIE, 2024. http://dx.doi.org/10.1117/12.3044595.

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Alejolowo, Omokolade, Gloria Austin, Charles Nwonuma, et al. "Puerarin: A Stimulator of NRF-2 Against Oxidative Damage in Diseases." In 2024 International Conference on Science, Engineering and Business for Driving Sustainable Development Goals (SEB4SDG). IEEE, 2024. http://dx.doi.org/10.1109/seb4sdg60871.2024.10630141.

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Wang, Shuwei, Can Cheng, Fan Yang, et al. "Study on the Mechanical Properties of Ginsenoside Rg3 in Protecting Cardiomyocytes from Oxidative Damage." In 2024 IEEE International Conference on Manipulation, Manufacturing and Measurement on the Nanoscale (3M-NANO). IEEE, 2024. https://doi.org/10.1109/3m-nano61605.2024.10769633.

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Nunes, Felipe G., Elber V. Bendinelli, Mark D. Soucek, and Idalina V. Aoki. "Synthesis of High Solids Alkyds Modified with Reactive Diluents for Encapsulation and Use in Self-healing Coatings." In LatinCORR 2023. AMPP, 2023. https://doi.org/10.5006/lac23-20430.

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A great effort has been devoted to the research of self-healing coatings, as they provide anticorrosive protection even when the painting is damaged [1]. The encapsulation of drying oils is an established strategy to promote self-healing. Microcapsules release the seed oil upon mechanical damage, which triggers self-healing in the coating defect as the oil undergoes oxidative polymerization [2]. Alkyds are also a great candidate for microencapsulation due to their green nature, oxidative-driven polymerization, and better anticorrosive properties than seed oils [3]. However, the high viscosity

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Rajalakshmi, N. R., L. Sharmila, Aluri Yuva Krishna, Shreedhar B, and Pranshu Chourasia. "Real-Time Monitoring of Oxidative Damage and Physiological Signals in the Human Body Using Chemical Biosensors." In 2025 7th International Conference on Inventive Material Science and Applications (ICIMA). IEEE, 2025. https://doi.org/10.1109/icima64861.2025.11073858.

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Shikov, A. E., V. V. Lastochkin, T. V. Chirkova, and V. V. Emelyanov. "Oxidative damage to plant lipids and proteins bynatural and artificial oxidative stress." In IX Congress of society physiologists of plants of Russia "Plant physiology is the basis for creating plants of the future". Kazan University Press, 2019. http://dx.doi.org/10.26907/978-5-00130-204-9-2019-481.

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Watson, Walter Philip, Carl W. Aften, and David J. Previs. "Delayed-Release Coatings for Oxidative Breakers." In SPE International Symposium and Exhibition on Formation Damage Control. Society of Petroleum Engineers, 2010. http://dx.doi.org/10.2118/127895-ms.

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Bonetta, Sa, V. Gianotti, D. Scozia, et al. "Genotoxic and oxidative damage related to PM2.5chemical fraction." In AIR POLLUTION 2008. WIT Press, 2008. http://dx.doi.org/10.2495/air080551.

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Xuena Zhu, P. Shah, and Chenzhong Li. "Paper-based Immunosensor for Oxidative DNA Damage Biomarker Detection." In 2013 29th Southern Biomedical Engineering Conference (SBEC 2013). IEEE, 2013. http://dx.doi.org/10.1109/sbec.2013.71.

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Ding, Ning, Sam Miller, and Heather O'Hagan. "Abstract 5317: JAK2 regulates oxidative damage-induced epigenetic alterations." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-5317.

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Raporty organizacyjne na temat "Oxidative damage"

Beal, M. F. Oxidative Damage in Parkinson's Disease. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada434051.

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Beal, M. F. Oxidative Damage in Parkinson's Disease. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada416957.

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Goth-Goldstein, Regine. Oxidative Damage, CYP1B1 and Breast Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada409396.

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Browne, Susan E. Bioenergetic Defects and Oxidative Damage in Transgenic Mouse Models of Neurodegenerative Disorders. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada430585.

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Goodwin, Edwin H. Biological Effects of LLIR and Normal Oxidative Damage: The Same or Different? Office of Scientific and Technical Information (OSTI), 2000. http://dx.doi.org/10.2172/833474.

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Browne, Susan E. Bioenergetic Defects and Oxidative Damage in Transgenic Mouse Models of Neurodegenerative Disorders. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada419306.

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Browne, Susan E. Bioenergetic Defects and Oxidative Damage in Transgenic Mouse Models of Neurodegenerative Disorders. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada460659.

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Wu, Guangyu. Preventive Role of Specific Dietary Factors and Natural Compounds Against DNA Damage and Oxidative Stress. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada377925.

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Barragan Echenique, Diego. ?-TEA?s Tumor Toxicity may be Attributed to its Capability of Inducing Oxidative Damage in the Endoplasmic Reticulum. Portland State University Library, 2015. http://dx.doi.org/10.15760/honors.172.

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Wen, Qin, Xueqin Hong, Kunze He, and Min Li. Can acupuncture reverse oxidative stress and neuroinflammatory damage in animal models of vascular Dementia? A preclinical systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.3.0114.

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