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Artykuły w czasopismach na temat „Oxidative stress and”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Sun, Qi-An, Nageswara Madamanchi, and Marschall Runge. "Oxidative stress, NADPH oxidases, and arteries." Hämostaseologie 36, no. 02 (2016): 77–88. http://dx.doi.org/10.5482/hamo-14-11-0076.

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ZusammenfassungDie Atherosklerose und ihre wichtigsten Komplikationen – Myokardinfarkt und Schlaganfall – sind die Hauptursachen für Tod und Behinderung in den USA und weltweit. Eine dramatische Zunahme bei Adipositas und Diabetes mellitus wird wahrscheinlich auch in Zukunft zu einer hohen Prävalenz kardiovaskulärer Erkrankungen (CVD) und deren Auswirkungen auf das Gesundheitswesen führen. Große Fortschritte gibt es bei der Entwicklung neuer Therapien zur Senkung der Inzidenz von Atherosklerose und CVD, besonders bei der Behandlung der Hypercholesterinämie und Hypertonie. Der gemeinsame mechan

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Naziya, Dr Saba. "MITOCHONDRIAL CARBON STRESS AND OXIDATIVE STRESS: IMPLICATIONS FOR HUMAN HEALTH." Era's Journal of Medical Research 11, no. 1 (2024): 85–92. http://dx.doi.org/10.24041/ejmr2024.14.

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Avital component of cellular redox equilibrium, mitochondria also play a crucial role in the synthesis of energy within cells. Disturbances in the metabolism of carbon inside the mitochondria may cause malfunction and oxidative stress, which in turn may contribute to the etiology of a number of human diseases. This is supported by newly emerging research. The interaction of oxidative stress and mitochondrial carbon stress is examined in this review, with particular attention on how these effects affect human health. We go over the processes that underlie oxidative stress caused by mitochondria

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Malenica, Maja, and Neven Meseldžić. "Oxidative stress and obesity." Arhiv za farmaciju 72, no. 2 (2022): 166–83. http://dx.doi.org/10.5937/arhfarm72-36123.

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Obesity is a disease of excessive accumulation of adipose tissue due to an increased energy intake which is disproportionate to the energy expenditure in the body. The visceral adipose tissue in the obese accumulated in that way increases the risk of developing a number of metabolic and cardiovascular diseases. Disorders such as diabetes, dyslipidemia, inflammation, endothelial dysfunction and mitochondria can contribute to the development of oxidative stress, which is especially pronounced in the abdominal type of obesity. Obesity can induce systemic oxidative stress through a variety of bioc

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Dorovskikh, V. A., N. V. Simonova, E. Yu Yurtaeva, R. A. Anokhina, and M. A. Shtarberg. "PHYTOCORRECTION OF OXIDATIVE STRESS IN EXPERIMENT." Amur Medical Journal, no. 15-16 (2016): 35–37. http://dx.doi.org/10.22448/amj.2016.15-16.35-37.

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Nsonwu, Magnus, SJ Ozims, and JK Nnodim. "Perspective of Cataract and Oxidative Stress." Series of Clinical and Biomedical Research 1, no. 1 (2024): 1–10. http://dx.doi.org/10.54178/2997-2701.v1i1a1994.

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One of the main causes of blindness is the multifactorial condition known as cataract. It is believed that oxidative stress plays a significant role in starting the cataractogenesis process. Today, it is a well-established fact that oxidative stress plays a role in both diabetes-induced cataract (diabetic) and age-related cataract (senile). The most likely cause of oxidative damage to the lens is a compromised antioxidant defense system brought on by age and diabetes-related increases in reactive oxygen species (ROS) production. The main factor contributing to cataract formation is systemic ox

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6

BÜYÜKOĞLU, Tülay, and Nurcanan ASLAN. "Oxidative Stress and Effects of Oxidative Stress on the Dairy Cattle During Transition Period." Turkiye Klinikleri Journal of Veterinary Sciences 9, no. 2 (2018): 33–41. http://dx.doi.org/10.5336/vetsci.2018-60899.

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Chen, Chuck T., Marc-Olivier Trépanier, Kathryn E. Hopperton, Anthony F. Domenichiello, Mojgan Masoodi та Richard P. Bazinet. "Inhibiting Mitochondrial β-Oxidation Selectively Reduces Levels of Nonenzymatic Oxidative Polyunsaturated Fatty Acid Metabolites in the Brain". Journal of Cerebral Blood Flow & Metabolism 34, № 3 (2013): 376–79. http://dx.doi.org/10.1038/jcbfm.2013.221.

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Schönfeld and Reiser recently hypothesized that fatty acid β-oxidation is a source of oxidative stress in the brain. To test this hypothesis, we inhibited brain mitochondrial β-oxidation with methyl palmoxirate (MEP) and measured oxidative polyunsaturated fatty acid (PUFA) metabolites in the rat brain. Upon MEP treatment, levels of several nonenzymatic auto-oxidative PUFA metabolites were reduced with few effects on enzymatically derived metabolites. Our finding confirms the hypothesis that reduced fatty acid β-oxidation decreases oxidative stress in the brain and β-oxidation inhibitors may be

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8

Kibel, Aleksandar, Ana Marija Lukinac, Vedran Dambic, Iva Juric, and Kristina Selthofer Relatic. "Oxidative Stress in Ischemic Heart Disease." Oxidative Medicine and Cellular Longevity 2020 (December 28, 2020): 1–30. http://dx.doi.org/10.1155/2020/6627144.

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One of the novel interesting topics in the study of cardiovascular disease is the role of the oxidation system, since inflammation and oxidative stress are known to lead to cardiovascular diseases, their progression and complications. During decades of research, many complex interactions between agents of oxidative stress, oxidation, and antioxidant systems have been elucidated, and numerous important pathophysiological links to na number of disorders and diseases have been established. This review article will present the most relevant knowledge linking oxidative stress to vascular dysfunctio

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9

Moroz, A. А. "EFFECT OF CORNUS FRUIT EXTRACTS ON OXIDATIVE STRESS IN DIABETES." Biotechnologia Acta 18, no. 2 (2025): 60–62. https://doi.org/10.15407/biotech18.02.060.

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The biological effects of fruits from different cultivars of the cornelian cherry under diabetes mellitus remain unclear. Aim. This study was purposed to investigate the effect of fruit extracts from various cultivars of Cornus mas L. on primary biomarkers of oxidative stress. Materials and Methods: oxidative stress metabolites in erythrocytes were assessed by measuring the level of lipid peroxidation and protein oxidation products. Results. The administration of C. mas and C. mas × C. officinalis hybrids fruit extracts to diabetic rats significantly reduced oxidative stress metabolite levels

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10

LATHA, A. "OXIDATIVE STRESS." NARAYANA NURSING JOURNAL 3, no. 2 (2014): 13. http://dx.doi.org/10.5455/nnj.2014-06-4.

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Poncin, Sylvie, Sandrine Van Eeckoudt, Kevin Humblet, Ides M. Colin, and Anne-Catherine Gérard. "Oxidative Stress." American Journal of Pathology 176, no. 3 (2010): 1355–63. http://dx.doi.org/10.2353/ajpath.2010.090682.

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Dabrowski, Andrzej, and Antoni Gabryelewicz. "Oxidative stress." International Journal of Pancreatology 12, no. 3 (1992): 193–99. http://dx.doi.org/10.1007/bf02924357.

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Finaud, Julien, G??rard Lac, and Edith Filaire. "Oxidative Stress." Sports Medicine 36, no. 4 (2006): 327–58. http://dx.doi.org/10.2165/00007256-200636040-00004.

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RAMASARMA, T. "Oxidative Stress." Biochemical Society Transactions 14, no. 3 (1986): 666. http://dx.doi.org/10.1042/bst0140666a.

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Faraci, Frank M. "Oxidative Stress." Stroke 36, no. 2 (2005): 186–88. http://dx.doi.org/10.1161/01.str.0000153067.27288.8b.

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Sies, Helmut, Carsten Berndt, and Dean P. Jones. "Oxidative Stress." Annual Review of Biochemistry 86, no. 1 (2017): 715–48. http://dx.doi.org/10.1146/annurev-biochem-061516-045037.

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17

Stevanovic, Jelka, Suncica Borozan, Tatjana Bozic, Slavoljub Jovic, Tatjana Djekic, and Blagoje Dimitrijevic. "Oxidative stress." Veterinarski glasnik 66, no. 3-4 (2012): 273–83. http://dx.doi.org/10.2298/vetgl1204273s.

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The unceasing need for oxygen is in contradiction to the fact that it is in fact toxic to mammals. Namely, its monovalent reduction can have as a consequence the production of short-living, chemically very active free radicals and certain non-radical agents (nitrogen-oxide, superoxide-anion-radicals, hydroxyl radicals, peroxyl radicals, singlet oxygen, peroxynitrite, hydrogen peroxide, hypochlorous acid, and others). There is no doubt that they have numerous positive roles, but when their production is stepped up to such an extent that the organism cannot eliminate them with its antioxidants (

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18

Preiser, Jean-Charles. "Oxidative Stress." Journal of Parenteral and Enteral Nutrition 36, no. 2 (2012): 147–54. http://dx.doi.org/10.1177/0148607111434963.

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19

Nugent, Kenneth. "Oxidative stress." Southwest Respiratory and Critical Care Chronicles 7, no. 27 (2019): 1–3. http://dx.doi.org/10.12746/swrccc.v7i27.518.

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Storz, Gisela, and James A. Imlayt. "Oxidative stress." Current Opinion in Microbiology 2, no. 2 (1999): 188–94. http://dx.doi.org/10.1016/s1369-5274(99)80033-2.

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Halliwell, B. "Oxidative stress." FEBS Letters 216, no. 1 (1987): 170–71. http://dx.doi.org/10.1016/0014-5793(87)80784-6.

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Henry, Yann. "Oxidative stress." Biochimie 69, no. 2 (1987): 166. http://dx.doi.org/10.1016/0300-9084(87)90256-2.

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Opara, Emmanuel C. "Oxidative Stress." Disease-a-Month 52, no. 5 (2006): 183–98. http://dx.doi.org/10.1016/j.disamonth.2006.05.003.

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Bast, A., and R. J. A. Goris. "Oxidative stress." Pharmaceutisch Weekblad Scientific Edition 11, no. 6 (1989): 199–206. http://dx.doi.org/10.1007/bf01959411.

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Soffler, Carl. "Oxidative Stress." Veterinary Clinics of North America: Equine Practice 23, no. 1 (2007): 135–57. http://dx.doi.org/10.1016/j.cveq.2006.11.004.

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Burton, Graham J., and Eric Jauniaux. "Oxidative stress." Best Practice & Research Clinical Obstetrics & Gynaecology 25, no. 3 (2011): 287–99. http://dx.doi.org/10.1016/j.bpobgyn.2010.10.016.

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Catanzaro, Orlando. "Diabetic oxidative stress and bone loss complications." Endocrinology and Disorders 5, no. 1 (2021): 01–04. http://dx.doi.org/10.31579/2640-1045/056.

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Diabetes mellitus is a group of metabolic disorder characterize by and absolute or partial insulin deficiency. Diabetic hyperglycemia is produce by the effect of homeostasis between proteolytic enzymes, their inhibitors and the antioxidants defense that protect and repair vital tissues and molecular components. Bone consist of both component and trabecular bone tissue. Organic matrix and albumin form part of noncollagenous of bone .Initiation of mineralization and collagen fibrils form the phase of mineral matrix. Calcium flux into and out of bone depend of osteoclastic and osteoblastic activi

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28

Tiwari, Supriya. "Oxidative Stress and Antioxidant Defense in Cells." Global Journal For Research Analysis 3, no. 8 (2012): 11–14. http://dx.doi.org/10.15373/22778160/august2014/4.

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Zayyanu, Tukur. "Antioxidant in Oxidative Stress and Neurodegenerative Diseases." Journal of Medical Science And clinical Research 11, no. 08 (2023): 10–22. http://dx.doi.org/10.18535/jmscr/v11i8.02.

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Background: Natural substances have long been used for developing traditional medicines, and the production of innovative drugs is encouraged by the use of these natural ingredients. The key interaction between oxidative stress and inflammation in disease etiology is supported by mounting research. Reactive oxygen species (ROS) generated by inflammatory cells cause oxidative stress, which has been recognized as the key mediator of the relationship between inflammation and the spread for diseases. Curcumin (DFM), demethoxycurcumin (DMC), and bisdemethoxycurcumin (BDMC) are the three main compon

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Ussipbek, B. A., L. C. López, N. T. Ablaikhanova, and M. K. Murzakhmetova. "OXIDATIVE STRESS AND MITOCHONDRIAL DYSFUNCTION." Series of biological and medical 2, no. 338 (2020): 31–40. http://dx.doi.org/10.32014/10.32014/2020.2519-1629.10.

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The process of cell damage resulting from the action of free radicals – reactive oxygen species (ROS) – is called oxidative stress. Most ROS are constantly formed in the cell – about 5 % of the oxygen consumed by tissues is converted into free radicals, but their level is normally so small that the cell inactivates them with the help of an antioxidant system. Different organs and tissues are exposed to different degrees of ROS and demonstrate different stability during the implementation of oxidative stress. The mechanisms of ROS formation by mitochondria under oxidative stress are still uncle

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Hao, Yue, Mingjie Xing, and Xianhong Gu. "Research Progress on Oxidative Stress and Its Nutritional Regulation Strategies in Pigs." Animals 11, no. 5 (2021): 1384. http://dx.doi.org/10.3390/ani11051384.

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Oxidative stress refers to the dramatic increase in the production of free radicals in human and animal bodies or the decrease in the ability to scavenging free radicals, thus breaking the antioxidation–oxidation balance. Various factors can induce oxidative stress in pig production. Oxidative stress has an important effect on pig performance and healthy growth, and has become one of the important factors restricting pig production. Based on the overview of the generation of oxidative stress, its effects on pigs, and signal transduction pathways, this paper discussed the nutritional measures t

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Basu, Samar. "Fatty acid oxidation and isoprostanes: Oxidative strain and oxidative stress." Prostaglandins, Leukotrienes and Essential Fatty Acids (PLEFA) 82, no. 4-6 (2010): 219–25. http://dx.doi.org/10.1016/j.plefa.2010.02.031.

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Naviaux, Robert K. "Oxidative Shielding or Oxidative Stress?" Journal of Pharmacology and Experimental Therapeutics 342, no. 3 (2012): 608–18. http://dx.doi.org/10.1124/jpet.112.192120.

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Miglioranza Scavuzzi, Bruna, and Joseph Holoshitz. "Endoplasmic Reticulum Stress, Oxidative Stress, and Rheumatic Diseases." Antioxidants 11, no. 7 (2022): 1306. http://dx.doi.org/10.3390/antiox11071306.

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Background: The endoplasmic reticulum (ER) is a multi-functional organelle responsible for cellular homeostasis, protein synthesis, folding and secretion. It has been increasingly recognized that the loss of ER homeostasis plays a central role in the development of autoimmune inflammatory disorders, such as rheumatic diseases. Purpose/Main contents: Here, we review current knowledge of the contribution of ER stress to the pathogenesis of rheumatic diseases, with a focus on rheumatoid arthritis (RA) and systemic lupus erythematosus (SLE). We also review the interplay between protein folding and

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Shang, F., and A. Taylor. "Oxidative stress and recovery from oxidative stress are associated with altered ubiquitin conjugating and proteolytic activities in bovine lens epithelial cells." Biochemical Journal 307, no. 1 (1995): 297–303. http://dx.doi.org/10.1042/bj3070297.

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Roles for ubiquitin (an 8.5 kDa polypeptide) involve its conjugation to proteins as a signal to initiate degradation and as a stress protein. We investigated ubiquitin conjugation and ubiquitin-dependent proteolytic activities in cultured bovine lens epithelial cells (BLECs) upon oxidative challenge. A 44% decrease in intracellular glutathione confirmed oxidative stress upon incubation with 1 mM H2O2. After 30 min incubation, endogenous high-molecular-mass ubiquitin conjugates decreased 73%, and intracellular proteolysis decreased about 50%. In the supernatants of the oxidatively treated BLECs

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Wang, Xiaole, Chuqiao Shen, Jie Zhu, Guoming Shen, Zegeng Li, and Jingcheng Dong. "Long Noncoding RNAs in the Regulation of Oxidative Stress." Oxidative Medicine and Cellular Longevity 2019 (February 17, 2019): 1–7. http://dx.doi.org/10.1155/2019/1318795.

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Oxidative stress takes responsibility for various diseases, such as chronic obstructive pulmonary disease (COPD), Alzheimer’s disease (AD), and cardiovascular disease; nevertheless, there is still a lack of specific biomarkers for the guidance of diagnosis and treatment of oxidative stress-related diseases. In recent years, growing studies have documented that oxidative stress has crucial correlations with long noncoding RNAs (lncRNAs), which have been identified as important transcriptions involving the process of oxidative stress, inflammation, etc. and been regarded as the potential specifi

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Soria-Meneses, Pedro Javier, Alejandro Jurado-Campos, Virgilio Gómez-Rubio, et al. "Determination of Ram (Ovis aries) Sperm DNA Damage Due to Oxidative Stress: 8-OHdG Immunodetection Assay vs. SCSA®." Animals 12, no. 23 (2022): 3286. http://dx.doi.org/10.3390/ani12233286.

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Conventional DNA analysis techniques can hardly detect DNA damage in ruminant spermatozoa due to high DNA compaction in these cells. Furthermore, these techniques cannot discriminate whether the damage is due to oxidative stress. The main purpose of this study was to evaluate the efficacy of two techniques for determining DNA damage in ovine sperm when the source of that damage is oxidative stress. Semen samples from twenty Manchega rams (Ovis aries) were collected and cryopreserved. After thawing, the samples were subjected to different levels of oxidative stress, and DNA oxidation was quanti

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Ibiene, Sarah Kalio, and Davis Tamunoemine. "Antioxidants and Oxidative Stress in Diabetes." Pharmaceutical and Chemical Journal 5, no. 3 (2018): 55–60. https://doi.org/10.5281/zenodo.13908748.

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Oxidative stress occurs when there is a disturbance in the balance between production of reactive oxygen species and antioxidant defense system. Reactive oxygen species are radicals such as superoxide and hydroxyl while non radical reactive oxygen species are hydrogen peroxide, hypochlorite and singlet oxygen. Auto oxidation of glucose in diabetes, glycation of anti oxidative enzymes and leakage of reactive oxygen species from mitochondria cause increase in oxidative stress in diabetes. Impaired activities of antioxidant defense enzymes such as super oxide dismutase and catalase enhance oxidat

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Thomas, Megan M., Maricela Haghiac, Catalin Grozav, Judi Minium, Virtu Calabuig-Navarro, and Perrie O’Tierney-Ginn. "Oxidative Stress Impairs Fatty Acid Oxidation and Mitochondrial Function in the Term Placenta." Reproductive Sciences 26, no. 7 (2018): 972–78. http://dx.doi.org/10.1177/1933719118802054.

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Placental fatty acid oxidation (FAO) is impaired and lipid storage is increased in pregnancy states associated with chronic oxidative stress. The effect of acute oxidative stress, as seen in pregnancies complicated with asthma, on placental lipid metabolism is unknown. We hypothesized that induction of acute oxidative stress would decrease FAO and increase esterification. We assessed [3H]-palmitate oxidation and esterification in term placental explants from lean women after exposure to hydrogen peroxide (H2O2) for 4 hours. Fatty acid oxidation decreased 16% and 24% in placental explants expos

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Saha, Atanu. "Assessment of Oxidative Stress and Association of Oxidative Markers among High School Teachers." Indian Journal of Applied Research 4, no. 2 (2011): 7–9. http://dx.doi.org/10.15373/2249555x/feb2014/179.

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Kiffin, Roberta, Christopher Christian, Erwin Knecht, and Ana Maria Cuervo. "Activation of Chaperone-mediated Autophagy during Oxidative Stress." Molecular Biology of the Cell 15, no. 11 (2004): 4829–40. http://dx.doi.org/10.1091/mbc.e04-06-0477.

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Oxidatively damaged proteins accumulate with age in almost all cell types and tissues. The activity of chaperone-mediated autophagy (CMA), a selective pathway for the degradation of cytosolic proteins in lysosomes, decreases with age. We have analyzed the possible participation of CMA in the removal of oxidized proteins in rat liver and cultured mouse fibroblasts. Added to the fact that CMA substrates, when oxidized, are more efficiently internalized into lysosomes, we have found a constitutive activation of CMA during oxidative stress. Oxidation-induced activation of CMA correlates with highe

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Singh, Abhishek Kumar, Sandeep Singh, Geetika Garg, and Syed Ibrahim Rizvi. "Rapamycin alleviates oxidative stress-induced damage in rat erythrocytes." Biochemistry and Cell Biology 94, no. 5 (2016): 471–79. http://dx.doi.org/10.1139/bcb-2016-0048.

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An imbalanced cellular redox system promotes the production of reactive oxygen species (ROS) that may lead to oxidative stress-mediated cell death. Erythrocytes are the best-studied model of antioxidant defense mechanism. The present study was undertaken to investigate the effect of the immunosuppressant drug rapamycin, an inducer of autophagy, on redox balance of erythrocytes and blood plasma of oxidatively challenged rats. Male Wistar rats were oxidatively challenged with HgCl2 (5 mg/kg body mass (b.m.)). A significant (p < 0.05) induction in ROS production, plasma membrane redox system (

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Mazumdar, Pinku, Jadab Kishore Phukan, and Vidhya Srinivasan. "OXIDATIVE STRESS MARKERS IN TYPE 2 DIABETES MELLITUS." International Journal of Advanced Research 12, no. 06 (2024): 1225–32. http://dx.doi.org/10.21474/ijar01/18987.

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Insulin resistance and high blood glucose are hallmarks of type 2 diabetes mellitus (T2DM), a serious global health concern. In the pathogenesis of T2DM, oxidative stress-a mismatch between antioxidant defenses and reactive oxygen species (ROS)-is a major factor. The mechanisms causing oxidative stress and its clinical consequences are highlighted in this review, which offers a thorough overview of oxidative stress markers in T2DM. We classify oxidative stress according to its origins, pathways, impacted biomolecules, and related illnesses. We stress the significance of keeping an eye on indic

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Kobiela, Jarek, Jacek Krajewski, Beata Kalińska-Błach, and Tomasz Stefaniak. "Selectivity of oxidative stress targeting in estrogen-induced experimental nephrocarcinogenesis." Acta Biochimica Polonica 49, no. 1 (2002): 51–58. http://dx.doi.org/10.18388/abp.2002_3820.

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Specificity of targeting of the oxidative stress towards lipid and protein fractions in a model of estrogen-induced Syrian hamster nephrocarcinogenesis was evaluated. The amount of proteins modified by oxidative stress was significantly elevated as early as one month after the initial implantation of estradiol to the experimental versus the control group, while the stress did not affect lipids. Subcellular localization of the oxidative stress target was determined by the analysis of protein oxidation in subcellular fractions of kidney cells. The endoplasmic reticulum membranes were the fractio

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Desbruslais, Alexandra, and Alexandra L. Wealleans. "Oxidation in Poultry Feed: Impact on the Bird and the Efficacy of Dietary Antioxidant Mitigation Strategies." Poultry 1, no. 4 (2022): 246–77. http://dx.doi.org/10.3390/poultry1040022.

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Oxidative reactions in premixes or final-compound feed pose considerable challenges to the poultry industry, both in terms of rancidity reducing palatability and shelf life and in vivo oxidative stress negatively impacting animal performance. Whilst there has been broad awareness of feed oxidation for many years, recognition of the impact of oxidative stress on the poultry industry has grown in the past twenty years. The appearance of woody breast and associated muscle meat myopathies has led to a rapid increase in research output and awareness of in vivo oxidative reactions. Oxidative stress

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Zieniewska, Izabela, Mateusz Maciejczyk, and Anna Zalewska. "The Effect of Selected Dental Materials Used in Conservative Dentistry, Endodontics, Surgery, and Orthodontics as Well as during the Periodontal Treatment on the Redox Balance in the Oral Cavity." International Journal of Molecular Sciences 21, no. 24 (2020): 9684. http://dx.doi.org/10.3390/ijms21249684.

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Oxidative stress (OS) is a redox homeostasis disorder that results in oxidation of cell components and thus disturbs cell metabolism. OS is induced by numerous internal as well as external factors. According to recent studies, dental treatment may also be one of them. The aim of our work was to assess the effect of dental treatment on the redox balance of the oral cavity. We reviewed literature available in PubMed, Medline, and Scopus databases, including the results from 2010 to 2020. Publications were searched according to the keywords: oxidative stress and dental monomers; oxidative stress

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Karanikas, Evangelos, Nikolaos P. Daskalakis, and Agorastos Agorastos. "Oxidative Dysregulation in Early Life Stress and Posttraumatic Stress Disorder: A Comprehensive Review." Brain Sciences 11, no. 6 (2021): 723. http://dx.doi.org/10.3390/brainsci11060723.

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Traumatic stress may chronically affect master homeostatic systems at the crossroads of peripheral and central susceptibility pathways and lead to the biological embedment of trauma-related allostatic trajectories through neurobiological alterations even decades later. Lately, there has been an exponential knowledge growth concerning the effect of traumatic stress on oxidative components and redox-state homeostasis. This extensive review encompasses a detailed description of the oxidative cascade components along with their physiological and pathophysiological functions and a systematic presen

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Lozano-Picazo, Carmen María, and Francisco Fernández-Belda. "Especies reactivas de oxígeno y su implicación en Biomedicina." Anales de Veterinaria de Murcia 34 (December 16, 2020): 17–26. http://dx.doi.org/10.6018/analesvet.332621.

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Las especies reactivas de oxígeno (ROS) actúan como regulador intracelular cuando se generan de forma controlada en puntos concretos de la célula. Modifican la función de proteínas mediante la oxidación reversible de cisteínas. Hay quinasas y fosfatasas de proteínas, factores de transcripción y canales iónicos que están regulados por ROS. Estrés oxidativo y daño celular aparecen cuando los mecanismos antioxidantes de protección son incapaces de mantener bajo el nivel intracelular de ROS. En estas condiciones, ROS inducen pérdida de viabilidad celular en patologías degenerativas de corazón y ce

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Breusing, Nicolle, and Tilman Grune. "Regulation of proteasome-mediated protein degradation during oxidative stress and aging." Biological Chemistry 389, no. 3 (2008): 203–9. http://dx.doi.org/10.1515/bc.2008.029.

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Abstract Protein degradation is a physiological process required to maintain cellular functions. There are distinct proteolytic systems for different physiological tasks under changing environmental and pathophysiological conditions. The proteasome is responsible for the removal of oxidatively damaged proteins in the cytosol and nucleus. It has been demonstrated that proteasomal degradation increases due to mild oxidation, whereas at higher oxidant levels proteasomal degradation decreases. Moreover, the proteasome itself is affected by oxidative stress to varying degrees. The ATP-stimulated 26

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Zhang, Weiran, Ranwei Zhong, Xiangping Qu, Yang Xiang, and Ming Ji. "Effect of 8-Hydroxyguanine DNA Glycosylase 1 on the Function of Immune Cells." Antioxidants 12, no. 6 (2023): 1300. http://dx.doi.org/10.3390/antiox12061300.

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Excess reactive oxygen species (ROS) can cause an imbalance between oxidation and anti-oxidation, leading to the occurrence of oxidative stress in the body. The most common product of ROS-induced base damage is 8-hydroxyguanine (8-oxoG). Failure to promptly remove 8-oxoG often causes mutations during DNA replication. 8-oxoG is cleared from cells by the 8-oxoG DNA glycosylase 1 (OGG1)-mediated oxidative damage base excision repair pathway so as to prevent cells from suffering dysfunction due to oxidative stress. Physiological immune homeostasis and, in particular, immune cell function are vulne

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