Gotowe bibliografie tematyczne / P53 Alterations

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  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
  6. Raporty organizacyjne

Gotowa bibliografia na temat „P53 Alterations”

Autor: Grafiati
Data publikacji: 3 czerwca 2025
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "P53 Alterations"

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SAFFARI, B., L. BERNSTEIN, D. C. HONG, et al. "Association of p53 mutations and a codon 72 single nucleotide polymorphism with lower overall survival and responsiveness to adjuvant radiotherapy in endometrioid endometrial carcinomas." International Journal of Gynecologic Cancer 15, no. 5 (2005): 952–63. http://dx.doi.org/10.1136/ijgc-00009577-200509000-00038.

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p53 genetic alterations are associated with advanced stage and aggressive tumors in a variety of human malignancies. The aim of this study was to examine p53 for genetic alterations and to evaluate the association of these alterations with clinical outcome and response to adjuvant radiotherapy in endometrioid endometrial carcinomas. p53 mutations in exons 2–11 were assessed in 59 endometrioid carcinomas by polymerase chain reaction–single-strand conformational polymorphism and sequence analysis. Twelve mutations (20.3%) and nine polymorphisms were identified. Seven of the nine polymorphisms we
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Chapuy, Bjoern, Stefano Monti, Kunihiko Takeyama, et al. "Integrative Analysis Reveals Multiple Alterations of p53 Signaling Pathway Components In Primary Diffuse Large B-Cell Lymphomas." Blood 116, no. 21 (2010): 635. http://dx.doi.org/10.1182/blood.v116.21.635.635.

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Abstract Abstract 635 Diffuse large B-cell lymphoma (DLBCL) is a genetically heterogeneous disease with infrequent alterations of multiple apoptotic, developmental and signaling pathways. Inactivating somatic mutations of the p53 tumor suppressor are uncommon in DLBCL, prompting speculation regarding alternative mechanisms of modulating p53 activity in this disease. As part of a comprehensive analysis of genetic alterations in DLBCL, we recently integrated high-resolution copy number data and transcription profiles in a series of 81 newly diagnosed previously untreated DLBCLs. Copy number (CN)
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Arranz-Salas, Isabel, Andres Sanz-Trelles, and Dolores Bautista Ojeda. "p53 alterations in porokeratosis." Journal of Cutaneous Pathology 30, no. 7 (2003): 455–58. http://dx.doi.org/10.1034/j.1600-0560.2003.00097.x.

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Muto, T., S. Wakui, H. Takahashi, et al. "p53 Gene Mutations Occurring in Spontaneous Benign and Malignant Mammary Tumors of the Dog." Veterinary Pathology 37, no. 3 (2000): 248–53. http://dx.doi.org/10.1354/vp.37-3-248.

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Sixty-three cases of benign and malignant canine mammary tumors were analyzed to define the alteration of exons 5–8 for the p53 tumor suppressor gene using polymerase chain reaction direct sequence analysis with paraffin-embedded tissues. Four missense mutations were found in 38 benign mammary tumors (11%), and five missense (one tumor had two missense mutations) and one nonsense mutations were found in 25 mammary carcinomas (20%). These data suggest that the p53 gene alterations might be initiated at an early stage of canine mammary carcinogenesis and p53 mutations might be associated with ma
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Huang, Hsuan-Ying, Peter B. Illei, Zhiquan Zhao, et al. "Ewing Sarcomas Withp53Mutation orp16/p14ARFHomozygous Deletion: A Highly Lethal Subset Associated With Poor Chemoresponse." Journal of Clinical Oncology 23, no. 3 (2005): 548–58. http://dx.doi.org/10.1200/jco.2005.02.081.

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PurposeEWS-FLI1 fusion type, p53 mutation, and homozygous deletion of p16/p14ARF have each been shown to be prognostically significant in Ewing sarcoma (ES). We provide the first combined prognostic analysis of these three molecular parameters in ES.Patients and MethodsWe studied 60 patients with ES (stage: localized in 54, metastatic in six). All cases were confirmed to contain the EWS-FLI1 (29 type 1, 12 type 2, 14 other types) or EWS-ERG fusions (five cases). Homozygous deletion of p16/p14ARF, and p53 mutations were determined by fluorescent in situ hybridization and Affymetrix (Santa Clara
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Yoshimoto, T., M. Naruse, Z. Zeng, et al. "The relatively high frequency of p53 gene mutations in multiple and malignant phaeochromocytomas." Journal of Endocrinology 159, no. 2 (1998): 247–55. http://dx.doi.org/10.1677/joe.0.1590247.

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To explore the clinical significance of p53 in the pathogenesis of adrenal neoplasms, we investigated the incidence of p53 gene mutations in functioning human adrenal tumours using the polymerase chain reaction-single strand conformation polymorphism (PCR-SSCP) technique to screen p53 exons 4 to 9. We examined 29 adrenocortical adenomas (primary aldosteronism, n=17; Cushing's syndrome, n=12, all benign), and 33 phaeochromocytomas (benign solitary, n=18; benign multiple, n=5; malignant, n=10) in Japanese and Chinese patients. PCR-SSCP did not show any abnormal band-shifts in any of the adrenoco
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Curtin, Karen, Martha L. Slattery, Richard Holubkov, Sandra Edwards, Joseph A. Holden, and Wade S. Samowitz. "p53 Alterations in Colon Tumors." Applied Immunohistochemistry & Molecular Morphology 12, no. 4 (2004): 380–86. http://dx.doi.org/10.1097/00129039-200412000-00017.

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Boyapati, Anita, Eiki Kanbe, and Dong-Er Zhang. "p53 Alterations in Myeloid Leukemia." Acta Haematologica 111, no. 1-2 (2003): 100–106. http://dx.doi.org/10.1159/000074489.

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Seruca, Raquel, Leonor David, Sérgio Castedo, Isabel Veiga, Anne-Lise Børresen, and Manuel Sobrinho-Simões. "p53 alterations in gastric carcinoma:." Cancer Genetics and Cytogenetics 75, no. 1 (1994): 45–50. http://dx.doi.org/10.1016/0165-4608(94)90214-3.

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Ichikawa, A., T. Hotta, N. Takagi, et al. "Mutations of p53 gene and their relation to disease progression in B- cell lymphoma." Blood 79, no. 10 (1992): 2701–7. http://dx.doi.org/10.1182/blood.v79.10.2701.2701.

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Abstract The alteration of p53 tumor suppressor gene was studied in 48 patients with B-cell lymphoma. A sequential combined technique of polymerase chain reaction-mediated single-strand conformational polymorphism (PCR- SSCP) or reverse transcription (RT)-PCR-SSCP and direct sequencing were used as a simple and sensitive approach to analyze nucleotide changes. By these methods, we identified 8 missense point mutations and 2 codon deletions in 9 of the 48 patients. These mutations were located in or close to the evolutionally highly conserved regions of the p53 gene. Eight of nine patients havi
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Rozprawy doktorskie na temat "P53 Alterations"

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Reles, Angela. "Molecular genetic alterations in ovarian cancer." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2001. http://dx.doi.org/10.18452/13801.

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Einleitung: Das p53 Tumorsuppressorgen spielt eine zentrale Rolle für Regulation des Zellzyklus und die Induktion der Apoptose. MDM2, das Protein des mdm2 Gens, bindet an p53, hemmt seine Funktion als Transkriptionsfaktor und bewirkt den raschen Abbau des Proteins. Methode: Gefriergewebe von 178 primären Ovarialkarzinomen wurde mittels PCR, SSCP Single Strand Conformation Polymorphism), DNA-Sequenzierung und Immunhistochemie auf p53 Mutationen (exon 2-11) und p53 Proteinüberexpression untersucht. Das mdm2-Gen wurde an 92 Ovarialkarzinomen, neun Borderline-Tumoren, sechs Cystadenomen und 2
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Saffroy, Raphael. "Instabilite genomique et alterations de p53 dans le carcinome hepatocellulaire." Paris 5, 2000. http://www.theses.fr/2000PA05N117.

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Ling, Gao. "p53 alterations in human skin : a molecular study based on morphology /." Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5131-4/.

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Gao, Ling. "p53 Alterations in Human Skin : A Molecular Study Based on Morphology." Doctoral thesis, Uppsala University, Department of Genetics and Pathology, 2001. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-1465.

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<p>Mutation of the p53 gene appears to be an early event in skin cancer development. The present study is based on morphology and represents a cellular and genetic investigation of p53 alterations in normal human skin and basal cell cancer.</p><p>Using double immunofluorescent labelling, we have demonstrated an increase in thymine dimers and p53 protein expression in the same keratinocytes following ultraviolet radiation. Large inter-individual differences in the kinetics of thymine dimer repair and subsequent epidermal p53 response were evident in both sunscreen-protected and non-protected s
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Froggatt, Nicola Jane. "Alterations to the tumour suppressor genes p53 and dcc in colorectal neplasia." Thesis, University of York, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.385322.

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Wäster, Larsson Petra. "UVA/B induced redox alterations and apoptosis in human melanocytes." Doctoral thesis, Linköpings universitet, Dermatologi och venerologi, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-8880.

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Malignant melanoma is one of the most rapidly increasing cancers and accounts for about three-quarter of all skin cancer deaths worldwide. Despite compelling evidence that ultraviolet (UV) irradiation causes melanoma the knowledge how various wavelength spectra affect the balance between proliferation and apoptosis controlling the homeostasis of the melanocyte population is still limited. The aim of this thesis was to elucidate the regulation of UVA/B induced apoptotic signaling in human epidermal melanocytes in vitro in relation to redox alterations and antioxidant photoprotection. UVA irradi
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Sivertsson, Åsa. "Detection and analysis of genetic alterations in normal skin and skin tumours." Doctoral thesis, KTH, Biotechnology, 2002. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-3432.

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<p>The investigation of genetic alterations in cancer-relatedgenes is useful for research, prognostic and therapeuticpurposes. However, the genetic heterogeneity that often occursduring tumour progression can make correct analysischallenging. The objective of this work has been to develop,evaluate and apply techniques that are sufficiently sensitiveand specific to detect and analyse genetic alterations in skintumours as well as in normal skin.</p><p>Initially, a method based on laser-assisted microdissectionin combination with conventional dideoxy sequencing wasdeveloped and evaluated for the
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Heminger, Katherine Ann. "LOSS OF HDMX LEADS TO ALTERATIONS IN GENE EXPRESSION AND INHIBITION OF CELL GROWTH IN TUMOR CELLS WITH WILD-TYPE p53." Wright State University / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=wright1175282827.

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Rafki, Beljebbar Naïma. "Etude des alterations du phenotype nucleaire liees a la resistance mdr dans des cellules leucemiques humaines sensibles et resistantes a la chimiotherapie." Reims, 1996. http://www.theses.fr/1996REIMP213.

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Tammemagi, Martin Carl. "Tobacco smoking, p53 tumour suppressor gene alterations, and clinicopathologic features and prognosis in non-small cell lung cancer." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0013/NQ35341.pdf.

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Książki na temat "P53 Alterations"

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Cooper, Melissa Erin. Investigation of methods to evaluate p53 genetic alterations in archival tissue. National Library of Canada, 2002.

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Pontén, Fredrik. Growth characteristics and p53 alterations in human basal cell cancer: Between bewildering basics & BCC biology. Univ., 1996.

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Tammemagi, Martin Carl. Tobacco smoking, p53 tumour suppressor gene alterations, and clinicopathologic features and prognosis in non-small cell lung cancer. National Library of Canada = Bibliothèque nationale du Canada, 1998.

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Breisgau, Universität Freiburg im, ed. P53-Alterations in squamous cell carcinoma of the head and neck: A molecular biological, immunohistochemical and clinical study. 1997.

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Ling, Gao. P53 Alterations in Human Skin: A Molecular Study Based on Morphology (Comprehensive Summaries of Uppsala Dissertations from the Faculty of Medicine, 1082). Uppsala Universitet, 2001.

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Hasan, David. The Natural History of Cerebral Aneurysms. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0109.

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Subarachnoid hemorrhage (SAH) secondary to rupture of cerebral aneurysms represents a relatively small fraction of strokes (5%) but morbidity and mortality associated with aneurysm rupture remain very high despite advances in the treatment of aneurysmal SAH. Cerebral vasospasm (CV) is the leading cause of delayed morbidity and mortality following aneurysmal subarachnoid hemorrhage, as well as delayed neurological dysfunction 1 to 2 weeks after rupture. Endothelial dysfunction is one of the primary contributing factors to CV following aneurysmal SAH, and this is associated with alterations in i
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Części książek na temat "P53 Alterations"

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Ul Haq, Burhan, Asma Jan, and Manzoor Ahmad Mir. "Mutational Alterations in p53." In p53 in Breast Cancer. CRC Press, 2024. http://dx.doi.org/10.1201/9781003520771-4.

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Yin, Shaoman, and Erwin G. Van Meir. "p53 Pathway Alterations in Brain Tumors." In CNS Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-553-8_14.

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Högberg, Johan, Ilona Silins, and Ulla Stenius. "Chemical induced alterations in p53 signaling." In Experientia Supplementum. Birkhäuser Basel, 2009. http://dx.doi.org/10.1007/978-3-7643-8336-7_7.

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Weirich, G., P. Schneider, C. Fellbaum, et al. "p53-Alterations in Thymic Epithelial Tumors." In Epithelial Tumors of the Thymus. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-0033-3_6.

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Soussi, Thierry. "Analysis of p53 Gene Alterations in Cancer: A Critical View." In 25 Years of p53 Research. Springer Netherlands, 2007. http://dx.doi.org/10.1007/978-1-4020-2922-6_12.

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Jabar, Alia, Nusrat Jan, Iqra Jan, and Manzoor Ahmad Mir. "Genetic Alterations Affecting p53 and Emerging Perspectives on Epigenetic Control of p53 in Breast Cancer." In p53 in Breast Cancer. CRC Press, 2024. http://dx.doi.org/10.1201/9781003520771-11.

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Girardini, Javier E., Dawid Walerych, and Giannino Del Sal. "Cooperation of p53 Mutations with Other Oncogenic Alterations in Cancer." In Subcellular Biochemistry. Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-017-9211-0_3.

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Hollstein, M., T. Soussi, G. Thomas, M. C. von Brevern, and H. Bartsch. "P53 Gene Alterations in Human Tumors: Perspectives for Cancer Control." In Recent Results in Cancer Research. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60393-8_26.

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Fukuyama, Kouzou, Mamoru Oh-uchida, Toshihiro Mineta, Nobuaki Momozaki, Katsuji Hori, and Kazuo Tabuchi. "An Analysis of Genetic Alterations of the p53 Gene in Human Glioma." In Biological Aspects of Brain Tumors. Springer Japan, 1991. http://dx.doi.org/10.1007/978-4-431-68150-2_42.

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Mass, M. J., and L. Wang. "Alterations of methylation of the tumor suppressor gene p53: insights into potential mechanisms of arsenic carcinogenesis." In Arsenic. Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5864-0_27.

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Streszczenia konferencji na temat "P53 Alterations"

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Das, Viswanath, Narendran Annadurai, and Marián Hajdúch. "Abstract 4474: An intricate role of p53 and p21 in cellular alterations and drug penetration in spheroids of colorectal cancer cells." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-4474.

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Liu, Jinyun, Marcia A. Ogasawara, Gang Chen, Helene Pelicano, and Peng Huang. "Abstract 3072: Characterization of TCL1-Tg:p53-/- mice that resemble human chronic lymphocytic leukemia with 17p-deletion: alterations in p53→Mir30→EZH2 axis." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-3072.

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Mukhopadhyay, Asima, Nicola Curtin, and Richard Edmondson. "Evaluation of different methods to assess homologous recombination status and sensitivity to PARP inhibitors in ovarian cancer." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685289.

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Methods: Matched samples of ascites and tumor tissue were taken from patients undergoing surgery for epithelial ovarian cancer. Tumor samples were formalin fixed and paraffin embedded (FFPE); ascites samples were used to generate primary cultures (PC). HR status was determined in PCs as previously described.[1] IC50 for the PARP inhibitor Rucaparib was estimated using SRB assays. DNA was extracted from the FFPE tissue. The following techniques were evaluated in PCs or paired FFPE samples: DR-GFP reporter assay, PARP activity assay, BRCA1 expression on immunohistochemistry, BRCA1 methylation st
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Abdulrahman, Azheen, and Inaam Mustafa. "Hepatotoxicity and Nephrotoxicity Provoked by an Anti-Androgen Drug (Bicalutamide) in Rat." In 5th International Conference on Biomedical and Health Sciences. Cihan University-Erbil, 2024. http://dx.doi.org/10.24086/biohs2024/paper.1073.

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Abstract—Bicalutamide (Casodex) is prescribed for the management of prostate metastatic carcinoma. The current investigation was designed to evaluate the physiological and histological impact of bicalutamide on the liver and kidney of rats. Twelve male albino rats were divided randomly into two groups. The first group represents the control and received 1ml of distilled water. The second group received bicalutamide (0.8mg/kg body weight/day) by gavage for six weeks. After the experiment wraps up, histological, biochemical and physiological evaluation of BCT treatment was achieved. Unpaired T-t
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Okano, Larissa Miyuki, Alexandre Luiz Korte de Azevedo, Tamyres Mingorance Carvalho, Tathiane Maistro Malta, Mauro Antonio Alves Castro, and Luciane Regina Cavalli. "Characterization of an epigenetic regulatory network on basal-like breast cancer subtype and its impact on signaling pathways and biological processes." In Brazilian Breast Cancer Symposium 2024. Mastology, 2024. http://dx.doi.org/10.29289/259453942024v34s1029.

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Objective: The main objective of this study was to identify DNA methylation at the distal cis-regulatory genomic regions associated with the basal-like breast cancer (BLBC) subtype, construct an epigenetic regulatory network, and determine its impact on cancer-associated signaling pathways and biological processes. Methodology: BLBC (n=134) and non- -tumoral breast (n=84) samples with DNA methylation, mRNA, and miRNA expression data were downloaded from The Cancer Genome Atlas (TCGA) database using a pipeline of computational tools. DNA methylation patterns on cancer- -specific enhancers enric
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Morrison, Bethanie L., Amanda L. Whiting, and Federico Bernal. "Abstract 4071: SAH-p53-mediated inhibition of cell migration via alteration of actin dynamics." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4071.

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Kim, Hoon, Siyuan Zheng, Seyed S. Amini, et al. "Abstract 984: Alteration of the p53 pathway is associated with subclonal tumor progression in glioblastoma." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-984.

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Gonzalez-Angulo, Ana Maria, Ana Lluch, Agda K. Eterovic, et al. "Abstract PD3-6: ConvertHER: Evolution of genomic alterations from primary to metastatic breast cancer." In Thirty-Seventh Annual CTRC-AACR San Antonio Breast Cancer Symposium; December 9-13, 2014; San Antonio, TX. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.sabcs14-pd3-6.

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Tan, Huey, Paula Boeira, Matthew Cramp, and Ashwin Dhanda. "P03 Mortality from severe alcoholic hepatitis is associated with increased oxidative stress and epigenetic alterations." In Abstracts of the British Association for the Study of the Liver Annual Meeting, 20–23 September 2022. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2022. http://dx.doi.org/10.1136/gutjnl-2022-basl.54.

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Chia, SKL, M. Martin, FA Holmes, et al. "Abstract PD3-12: PIK3CA alterations and benefit with neratinib after trastuzumab-based adjuvant therapy in early-stage HER2+ breast cancer: Correlative analyses of the phase III ExteNET trial." In Abstracts: 2017 San Antonio Breast Cancer Symposium; December 5-9, 2017; San Antonio, Texas. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.sabcs17-pd3-12.

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Raporty organizacyjne na temat "P53 Alterations"

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Gao, Da-Qing, and Michael F. Press. Assessment of mdm2 Alterations on p53 Expression in Breast Cancer. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada394016.

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Blackburn, Anneke C., and Joseph Jerry. Development of Spontaneous Mammary Tumors in BALB/c-p53+-Mice: Detection of Early Genetic Alterations and the Mapping of BALB/c Susceptibility Genes. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada410279.

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Smith, Sallie, and Joseph Jerry. Development of Spontaneous Mammary Tumors in BALB/c-p53+/-Mice: Detection of Early Genetic Alterations and the Mapping of BALB/c Susceptibility Genes. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada424523.

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Connolly, Denise C. Modeling Human Epithelial Ovarian Cancer in Mice by Alteration of Expression of the BRCA1 and/or P53 Genes. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada436423.

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Connolly, Denise C. Modeling Human Epithelial Ovarian Cancer in Mice by Alteration of Expression of the BRCA1 and/or p53 Genes. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada485053.

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