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Gotowe bibliografie tematyczne / Pediatric Cancer Susceptibility

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Gotowa bibliografia na temat „Pediatric Cancer Susceptibility”

Autor: Grafiati

Data publikacji: 3 czerwca 2025

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Pediatric Cancer Susceptibility"

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Plon, Sharon E., and Katherine Nathanson. "Inherited Susceptibility for Pediatric Cancer." Cancer Journal 11, no. 4 (2005): 255–67. http://dx.doi.org/10.1097/00130404-200507000-00002.

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McReynolds, Lisa J., and Sharon A. Savage. "Pediatric leukemia susceptibility disorders: manifestations and management." Hematology 2017, no. 1 (2017): 242–50. http://dx.doi.org/10.1182/asheducation-2017.1.242.

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Abstract The clinical manifestations of inherited susceptibility to leukemia encompass a wide phenotypic range, including patients with certain congenital anomalies or early-onset myelodysplastic syndrome (MDS) and some with no obvious medical problems until they develop leukemia. Leukemia susceptibility syndromes occur as a result of autosomal dominant, autosomal recessive, or X-linked recessive inheritance, or de novo occurrence, of germline pathogenic variants in DNA repair, ribosome biogenesis, telomere biology, hematopoietic transcription factors, tumor suppressors, and other critical cel

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Kennedy, Richard D., and Alan D. D'Andrea. "DNA Repair Pathways in Clinical Practice: Lessons From Pediatric Cancer Susceptibility Syndromes." Journal of Clinical Oncology 24, no. 23 (2006): 3799–808. http://dx.doi.org/10.1200/jco.2005.05.4171.

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Human cancers exhibit genomic instability and an increased mutation rate due to underlying defects in DNA repair. Cancer cells are often defective in one of six major DNA repair pathways, namely: mismatch repair, base excision repair, nucleotide excision repair, homologous recombination, nonhomologous endjoining and translesion synthesis. The specific DNA repair pathway affected is predictive of the kinds of mutations, the tumor drug sensitivity, and the treatment outcome. The study of rare inherited DNA repair disorders, such as Fanconi anemia, has yielded new insights to drug sensitivity and

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Godinez Paredes, Jesica M., Claudia Garrido, Patricia Calderón, et al. "Abstract 2273: The spectrum of germline cancer gene mutations in Central American pediatric cancer." Cancer Research 85, no. 8_Supplement_1 (2025): 2273. https://doi.org/10.1158/1538-7445.am2025-2273.

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Abstract Pediatric cancers are a diverse set of both hematologic and solid malignancies. Approximately 5% of pediatric cancer cases are caused by known genetic conditions, but little is known about genetic susceptibility in Central American children. Most pediatric leukemia and lymphoma cases have high response and cure rates, and many solid malignancies have better outcomes than similar adult cancers. We evaluated the germline genetic variation of pediatric solid tumor cases in Guatemala and Nicaragua in reference hospitals, capturing most of the cancer cases nationwide. We performed exome se

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Cornel, Annelisa M., Loutje van der Sman, Jip T. van Dinter, et al. "Targeting pediatric cancers via T-cell recognition of the monomorphic MHC class I-related protein MR1." Journal for ImmunoTherapy of Cancer 12, no. 3 (2024): e007538. http://dx.doi.org/10.1136/jitc-2023-007538.

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Human leukocyte antigen (HLA) restriction of conventional T-cell targeting introduces complexity in generating T-cell therapy strategies for patients with cancer with diverse HLA-backgrounds. A subpopulation of atypical, major histocompatibility complex-I related protein 1 (MR1)-restricted T-cells, distinctive from mucosal-associated invariant T-cells (MAITs), was recently identified recognizing currently unidentified MR1-presented cancer-specific metabolites. It is hypothesized that the MC.7.G5 MR1T-clone has potential as a pan-cancer, pan-population T-cell immunotherapy approach. These cells

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Nirmalkar, Nikita, Ishani Arora, Vaishnavi H. Mishra, Gulshan R. Bandre, and Yugeshwari R. Tiwade. "Genetic Susceptibility and Treatment Personalization in Acute Lymphoblastic Leukemia: A Comprehensive Review of Genetic Susceptibility and Targeted Therapies." Journal of Applied Hematology 15, no. 3 (2024): 163–68. http://dx.doi.org/10.4103/joah.joah_40_24.

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Abstract Acute lymphoblastic leukemia (ALL) in children is a complicated and heterogeneous disease impacted by various genetic susceptibility factors. The significance of genetic testing in pediatric ALL diagnosis and management, the role of minimal residual disease (MRD) monitoring, and ethical issues and problems in pediatric genetic testing are discussed in this narrative review. It also looks ahead to the future of genetic susceptibility research, focusing on data integration, artificial intelligence-driven insights, and the possible finding of novel treatment targets. We hope to provide a

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Liu, Zan, Zhenghui Xiao, Ming Li, et al. "Association Between Arg72Pro Polymorphism in TP53 and Malignant Abdominal Solid Tumor Risk in Hunan Children." Cancer Control 28 (January 1, 2021): 107327482110048. http://dx.doi.org/10.1177/10732748211004880.

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Pediatric solid tumors are heterogeneous and comprise various histological subtypes. TP53, a tumor suppressor, orchestrates the transcriptional activation of anti-cancer genes. The gene coding for this protein is highly polymorphic, and its mutations are associated with cancer development. The Arg72Pro polymorphism in TP53 has been associated with susceptibility to various types of cancer. Here, in this hospital-based study, we evaluated the association of this polymorphism with susceptibility toward malignant abdominal solid tumors in children in the Hunan province of China. We enrolled 162 p

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Patenaude, A. F., L. Basili, D. L. Fairclough, and F. P. Li. "Attitudes of 47 mothers of pediatric oncology patients toward genetic testing for cancer predisposition." Journal of Clinical Oncology 14, no. 2 (1996): 415–21. http://dx.doi.org/10.1200/jco.1996.14.2.415.

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PURPOSE To assess attitudes toward testing for cancer susceptibility genes, we interviewed mothers of pediatric oncology patients about their cancer causation theories, interest in hypothetical predisposition testing for themselves and their healthy children, and anticipated impact of testing. PATIENTS AND METHODS The subjects were 47 mothers of two or more living children, one of whom was 6 to 24 months postdiagnosis of cancer. Potential risks and benefits of hypothetical genetic predisposition testing for cancer susceptibility were described. A semistructured interview assessed the following

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Kara, Buket, and Yavuz Koksal. "Pediatric Lymphoma and Solid Tumors Associated With Cancer Susceptibility Syndromes." Journal of Pediatric Hematology/Oncology 42, no. 7 (2020): 438–45. http://dx.doi.org/10.1097/mph.0000000000001798.

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Hadi, Dalal Abbas. "Spectrum of Bacterial Infection and Antibiotic Susceptibility Profile among Clinical Samples of Febrile Pediatric Cancer Patients under Chemotherapy." International Journal of Psychosocial Rehabilitation 24, no. 5 (2020): 1189–203. http://dx.doi.org/10.37200/ijpr/v24i5/pr201794.

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Części książek na temat "Pediatric Cancer Susceptibility"

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Sherborne, Amy L., and Richard S. Houlston. "Risk of Childhood Acute Lymphoblastic Leukemia: Identification of Inherited Susceptibility." In Pediatric Cancer, Volume 4. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6591-7_11.

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Kunkle, Brian, David Sandberg, Prasanna Jayakar, Quentin Felty, and Deodutta Roy. "Gene–Environment Interaction and Susceptibility to Pediatric Brain Tumors." In Environmental Factors, Genes, and the Development of Human Cancers. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6752-7_9.

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Patenaude, Andrea Farkas, and Katherine A. Schneider. "Genetic Issues." In Comprehensive Handbook of Childhood Cancer and Sickle Cell Disease. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195169850.003.0028.

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The hallmark of genetic medicine is that medical concerns of one individual are germane not only for that person, but also for their offspring and future generations. Pediatric cancer patients, survivors of childhood cancers, their parents, and their siblings all have concerns that are likely to be increasingly affected by advances in genetics. It has been estimated that between 10% and 15% of pediatric cancers are either hereditary or familial in origin (Quesnel & Malkin, 1997). Advances in genetics will help identify pediatric cancers that have a hereditary origin as well as genetic synd

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Farnaz, Dr Huma, and Dr Dipanshu Aggarwal. "CANCER CHRONICLES: UNDERSTANDING, TREATING, AND OVERCOMING: NARRATIVE REVIEW." In Futuristic Trends in Medical Sciences Volume 3 Book 20. Iterative International Publisher, Selfypage Developers Pvt Ltd, 2024. http://dx.doi.org/10.58532/v3bfms20p1ch8.

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This abstract delves into the nuanced concept of health, focusing on the pivotal stages of treatment planning and the profound implications of cancer within this context. Health, encompassing both mental and physical well-being, stands as a cornerstone of human existence. The article underscores the initial significance of delineating treatment objectives to devise effective strategies that target the fundamental causes of health issues and foster sustainable wellness. The judicious selection of treatment regimens necessitates a holistic understanding of the underlying ailment and the distinct

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Swain, Nicole F., and Scott W. Powers. "Pain Management of Sickle Cell Disease." In Comprehensive Handbook of Childhood Cancer and Sickle Cell Disease. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195169850.003.0036.

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Acute and chronic pain episodes are common experiences for many individuals living with sickle cell disease (SCD). The management of pain related to SCD is primary palliative and includes pharmacological, nonpharmacological, and preventive therapies (Ballas, 2002). This chapter focuses on issues relevant to the assessment and treatment of sickle cell pain in primarily pediatric populations, although some of the adult literature is reviewed, particularly as it relates to children and adolescents. SCD is a hereditary disorder. The disorder primarily affects people of Caribbean and African origin

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Streszczenia konferencji na temat "Pediatric Cancer Susceptibility"

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Chang, Vivian, Hane Lee, Tom Davidson, et al. "Abstract 08: Clinical exome sequencing in the diagnosis of pediatric cancer predisposition." In Abstracts: AACR Special Conference: Cancer Susceptibility and Cancer Susceptibility Syndromes; January 29-February 1, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.cansusc14-08.

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Gramatges, Maria, Ninad Oak, David Wheeler, Deborah Ritter, Donna Muzny, and Sharon Plon. "Abstract 01: Genetic variation within genes related to telomere maintenance and DNA repair in a cohort of pediatric acute myeloid leukemia (AML) subjects." In Abstracts: AACR Special Conference: Cancer Susceptibility and Cancer Susceptibility Syndromes; January 29-February 1, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.cansusc14-01.

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Plon, Sharon E., Sarah Scollon, Katie Bergstrom, et al. "Abstract 11: Evaluating cancer susceptibility mutations and incidental findings from whole exome sequencing of sequentially diagnosed pediatric solid and brain tumor patients: Early results of the BASIC3 study." In Abstracts: AACR Special Conference: Cancer Susceptibility and Cancer Susceptibility Syndromes; January 29-February 1, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.cansusc14-11.

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Grunewald, Thomas, Marie-Ming Aynaud, Franck Tirode, et al. "Abstract A10: Functional characterization of Ewing's sarcoma susceptibility loci." In Abstracts: AACR Special Conference: Pediatric Cancer at the Crossroads: Translating Discovery into Improved Outcomes; November 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.pedcan-a10.

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Machiela, Mitchell J., Thomas G. P. Grünewald, Didier Surdez, et al. "Abstract A13: Genome-wide association study identifies multiple new loci associated with Ewing sarcoma susceptibility." In Abstracts: AACR Special Conference: Pediatric Cancer Research: From Basic Science to the Clinic; December 3-6, 2017; Atlanta, Georgia. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.pedca17-a13.

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