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Artykuły w czasopismach na temat „PEG10”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Yalçın, Arif Hakan, and Ercan Şimşir. "Production of Wood Pyrolysis Oil for Use as an Alternative Fuel in the Automotive Sector and Improvement of Its Physicochemical Properties." Journal of Materials and Mechatronics: A 6, no. 1 (2025): 262–73. https://doi.org/10.55546/jmm.1581683.

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Biomass resources have the potential to replace petroleum-based fuels. Biomass can be converted into pyrolysis oil by pyrolysis method and this oil is of interest as an alternative to fossil fuels used in many areas such as automotive sector. However, pyrolysis oil is difficult to use directly in diesel engines due to its low energy density, high viscosity and water content. The easiest solution is to create mixtures with high cetane content. In this study, polyethylene glycol 400 (PEG), Wood Pyrolysis oil (WPO), n-butanol (B) and 2-ethylhexyl nitrate (2-EHN) (PEG0/PY10/B85/2-EHN5) were obtain

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Campodonico, Will, Harihar M. Mohan, Phuoc T. Huynh, et al. "The gag-like gene RTL8 antagonizes PEG10-mediated virus like particles." PLOS ONE 19, no. 12 (2024): e0310946. https://doi.org/10.1371/journal.pone.0310946.

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PEG10 is a retroelement-derived Mart-family gene that is necessary for placentation and has been implicated in neurological disease. PEG10 resembles both retrotransposon and retroviral proteins and forms virus-like particles (VLPs) that can be purified using iodixanol ultracentrifugation. It is hypothesized that formation of VLPs is crucial to the biological roles of PEG10 in reproduction and neurological health. Here, we describe the regulation of PEG10 VLP formation and release in human cells with a role for the related Mart gene RTL8. RTL8 resembles a truncated form of PEG10 that shares hom

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Kim, Soojin, Daksh Thaper, Samir Bidnur, et al. "PEG10 is associated with treatment-induced neuroendocrine prostate cancer." Journal of Molecular Endocrinology 63, no. 1 (2019): 39–49. http://dx.doi.org/10.1530/jme-18-0226.

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Neuroendocrine (NE) differentiation of advanced prostate adenocarcinoma following androgen receptor (AR) axis-directed therapy is becoming increasingly recognized. Several models of this transdifferentiation provide insight into its molecular pathogenesis and have highlighted the placental gene PEG10 for further study. Using our unique model of enzalutamide resistance (ENZR) and NE differentiation, we studied PEG10/AR interplay in enzalutamide treatment-resistant cell lines 42DENZR and 42FENZR compared to LNCaP and castration-resistant 16DCRPC cells. ENZR cell lines with positive terminal NE m

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Xiong, Jie, Jian Qin, Yingcheng Zheng, Xiaofan Peng, Yixing Luo, and Xiangyu Meng. "PEG10 promotes the migration of human Burkitt’s lymphoma cells by up-regulating the expression of matrix metalloproteinase-2 and -9." Clinical & Investigative Medicine 35, no. 3 (2012): 117. http://dx.doi.org/10.25011/cim.v35i3.16587.

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Purpose: Paternally expressed gene 10 (PEG10) is important for apoptosis resistance in cancer cells; however, the effect of PEG10 on tumor cell migration remains poorly understood. In this study, we investigated the effects of PEG10 on proliferation, apoptosis, adhesion and migration in the Burkitt’s lymphoma cell line, Raji.   Methods: Apoptosis was induced by 5-fluorouracil (5-FU) in pcDNA3.0/PEG10 transiently transfected HEK293T cells and PEG10-suppressed Raji cells. siRNAPEG10 was used to inhibit PEG10 expression. Fluorescence-activated cell sorting (FACS) were performed to ana

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5

Kaneko-Ishino, Tomoko, and Fumitoshi Ishino. "Retrovirus-Derived RTL/SIRE Genes: Their Diverse Roles in the Current Eutherian Developmental System and Contribution to Eutherian Evolution." Biomolecules 13, no. 10 (2023): 1436. http://dx.doi.org/10.3390/biom13101436.

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Eutherians have 11 retrotransposon Gag-like (RTL)/sushi-ichi retrotransposon homolog (SIRH) genes presumably derived from a certain retrovirus. Accumulating evidence indicates that the RTL/SIRH genes play a variety of roles in the current mammalian developmental system, such as in the placenta, brain, and innate immune system, in a eutherian-specific manner. It has been shown that the functional role of Paternally Expressed 10 (PEG10) in placental formation is unique to the therian mammals, as are the eutherian-specific roles of PEG10 and PEG11/RTL1 in maintaining the fetal capillary network a

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6

Yin, Dong, Xinyi Yao, Jingyuan Zhang, et al. "Abstract 5399: Placental gene PEG10 promotes onco-fetal metabolic reprogramming in hepatocellular carcinoma." Cancer Research 85, no. 8_Supplement_1 (2025): 5399. https://doi.org/10.1158/1538-7445.am2025-5399.

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Abstract Tumorigenesis is a complex biological process, accompanied by cellular dedifferentiation and metabolic reprogramming, which similarities to the metabolic characteristics of embryonic development stages. In our research, we focused particularly on the RNA-binding protein PEG10, which is highly expressed in the placenta and found to be similarly overexpressed in liver cancer cells, playing a key role in the process of aerobic glycolysis in tumor cells. This discovery suggests that there may be a phenomenon of fetal-like metabolic reprogramming in hepatocellular carcinoma (HCC), which is

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7

Kainz, Birgit, Martin Bilban, Medhat Shehata, et al. "The Paternally Expressed Gene 10 (PEG10) on Chromosome 7q21 Is Overexpressed and Imprinted in High-Risk B-CLL." Blood 106, no. 11 (2005): 1221. http://dx.doi.org/10.1182/blood.v106.11.1221.1221.

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Abstract Introduction: To establish surrogate markers for IgVH mutation status in B-CLL, we performed microarray analysis with mutated and unmutated patient samples. Among the most differentially expressed genes was PEG10, a maternally imprinted gene located on chromosome 7q21. PEG10 is known to be expressed during placental development and in hepatocellular carcinoma. Here we have investigated the association of PEG10 with B-CLL subtypes and risk factors as well as its transcriptional regulation. In addition, we studied its expression in other hematologic malignancies. Methods and Results: In

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8

Liu, Fengjie, Yumei Gao, Bufang Xu, et al. "PEG10 amplification at 7q21.3 potentiates large-cell transformation in cutaneous T-cell lymphoma." Blood 139, no. 4 (2022): 554–71. http://dx.doi.org/10.1182/blood.2021012091.

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Abstract Mycosis fungoides (MF), the most common form of cutaneous T-cell lymphoma, undergo large-cell transformation (LCT) in the late stage, manifesting aggressive behavior, resistance to treatments, and poor prognosis, but the mechanisms involved remain unclear. To identify the molecular driver of LCT, we collected tumor samples from 133 MF patients and performed whole-transcriptome sequencing on 49 advanced-stage MF patients, followed by integrated copy number inference and genomic hybridization. Tumors with LCT showed unique transcriptional programs and enriched expressions of genes at ch

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9

Byrne, Raphael M., Rebecca Ruhl, Christian Lanciault, Sudarshan Anand, Abhinav Nellore, and Vassiliki Liana Tsikitis. "Age-related differences in gene expression in colorectal cancer (CRC)." Journal of Clinical Oncology 36, no. 4_suppl (2018): 654. http://dx.doi.org/10.1200/jco.2018.36.4_suppl.654.

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654 Background: Colorectal cancer (CRC) in young patients is increasing in incidence and is associated with worse outcomes than CRC in older patients. While distinct molecular subtypes of CRC have been recently characterized, it is unclear whether there are molecular differences between the tumors of young and old patients. We sought to identify differences in gene expression of CRC between these two groups. Our discovery analysis identified a gene signature of several differentially expressed RNAs, from which we validated PEG10. The PEG10 gene on chromosome 7q21.3 has been implicated in liver

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10

Segel, Michael, Blake Lash, Jingwei Song, et al. "Mammalian retrovirus-like protein PEG10 packages its own mRNA and can be pseudotyped for mRNA delivery." Science 373, no. 6557 (2021): 882–89. http://dx.doi.org/10.1126/science.abg6155.

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Eukaryotic genomes contain domesticated genes from integrating viruses and mobile genetic elements. Among these are homologs of the capsid protein (known as Gag) of long terminal repeat (LTR) retrotransposons and retroviruses. We identified several mammalian Gag homologs that form virus-like particles and one LTR retrotransposon homolog, PEG10, that preferentially binds and facilitates vesicular secretion of its own messenger RNA (mRNA). We showed that the mRNA cargo of PEG10 can be reprogrammed by flanking genes of interest with Peg10’s untranslated regions. Taking advantage of this reprogram

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11

Ahn, Jinsoo, In-Sul Hwang, Mi-Ryung Park, In-Cheol Cho, Seongsoo Hwang, and Kichoon Lee. "The Landscape of Genomic Imprinting at the Porcine SGCE/PEG10 Locus from Methylome and Transcriptome of Parthenogenetic Embryos." G3: Genes|Genomes|Genetics 10, no. 11 (2020): 4037–47. http://dx.doi.org/10.1534/g3.120.401425.

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In mammals, imprinted genes often exist in the form of clusters in specific chromosome regions. However, in pigs, genomic imprinting of a relatively few genes and clusters has been identified, and genes within or adjacent to putative imprinted clusters need to be investigated including those at the SGCE/PEG10 locus. The objective of this study was to, using porcine parthenogenetic embryos, investigate imprinting status of genes within the genomic region spans between the COL1A2 and ASB4 genes in chromosome 9. Whole-genome bisulfite sequencing (WGBS) and RNA sequencing (RNA-seq) were conducted

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12

Tatarūnas, Vacis, Ieva Čiapienė, and Agnė Giedraitienė. "Precise Therapy Using the Selective Endogenous Encapsidation for Cellular Delivery Vector System." Pharmaceutics 16, no. 2 (2024): 292. http://dx.doi.org/10.3390/pharmaceutics16020292.

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Interindividual variability in drug response is a major problem in the prescription of pharmacological treatments. The therapeutic effect of drugs can be influenced by human genes. Pharmacogenomic guidelines for individualization of treatment have been validated and used for conventional dosage forms. However, drugs can often target non-specific areas and produce both desired and undesired pharmacological effects. The use of nanoparticles, liposomes, or other available forms for drug formulation could help to overcome the latter problem. Virus-like particles based on retroviruses could be a po

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13

Golda, Mária, János András Mótyán, Mohamed Mahdi, and József Tőzsér. "Study of the Retrotransposon-Derived Human PEG10 Protease." Proceedings 50, no. 1 (2020): 110. http://dx.doi.org/10.3390/proceedings2020050110.

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Paternally expressed gene 10 (PEG10) is a human retrotransposon-derived imprinted gene. Previous works have demonstrated that a mutation in the coding sequence of this gene is lethal with regard to embryological age due to defects of placental development. In addition, PEG10 is implicated in several malignancies, such as pancreatic cancer and hepatocellular carcinoma. The PEG10 gene encodes two protein isoforms, which are translated by a typical retroviral frameshift mechanism. The Gag-like protein (RF1PEG10) is encoded by reading frame 1, whilst reading frames 1 and 2 accounts for the Gag-Pol

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14

Shu, Dan, Shengchun Liu, Kang Li, et al. "Abstract P1-04-22: Loss of SMAD4 Drives Breast Cancer Invasion and Metastasis through E2F1-Mediated Activation of the PEG10-ERK Axis." Clinical Cancer Research 31, no. 12_Supplement (2025): P1–04–22—P1–04–22. https://doi.org/10.1158/1557-3265.sabcs24-p1-04-22.

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Abstract Objective: Although SMAD4 mutations are rare in breast cancer, its loss is strongly associated with metastasis and poor prognosis. This highlights the need to elucidate the mechanisms by which SMAD4 influences breast cancer progression. In this study, we examined the invasive and metastatic properties of SMAD4-deficient breast cancer cell lines, alongside transcriptomic changes in SMAD4 knockout (KO) models, with the aim of uncovering the molecular pathways through which SMAD4 regulates breast cancer recurrence and metastasis.Methods: We analyzed SMAD4 expression across multiple bioin

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15

Qi, Shiyu, Yibo Wang, Zhimei Liu, et al. "Construction of a TAT-Cas9-EGFP Site-Specific Integration Eukaryotic Cell Line Using Efficient PEG10 Modification." International Journal of Molecular Sciences 26, no. 3 (2025): 1331. https://doi.org/10.3390/ijms26031331.

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The CRISPR/Cas9 system enables precise and efficient modification of eukaryotic genomes. Among its various applications, homology-directed repair (HDR) mediated knock-in (KI) is crucial for creating human disease models, gene therapy, and agricultural genetic enhancements. Despite its potential, HDR-mediated knock-in efficiency remains relatively low. This study investigated the impact of 5′ end PEG10 modification on site-specific integration of the target gene. The HEK293 cell line is considered a highly attractive expression system for the production of recombinant proteins, with the constru

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Nishihara, T., A. Shiomi, S. Kadotani, T. Nokami, and T. Itoh. "Remarkably improved stability and enhanced activity of a Burkholderia cepacia lipase by coating with a triazolium alkyl-PEG sulfate ionic liquid." Green Chem. 19, no. 21 (2017): 5250–56. http://dx.doi.org/10.1039/c7gc02319g.

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Golda, Mária, János András Mótyán, Mohamed Mahdi, and József Tőzsér. "Functional Study of the Retrotransposon-Derived Human PEG10 Protease." International Journal of Molecular Sciences 21, no. 7 (2020): 2424. http://dx.doi.org/10.3390/ijms21072424.

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Paternally expressed gene 10 (PEG10) is a human retrotransposon-derived imprinted gene. The mRNA of PEG10 encodes two protein isoforms: the Gag-like protein (RF1PEG10) is coded by reading frame 1, while the Gag-Pol-like polyprotein (RF1/RF2PEG10) is coded by reading frames 1 and 2. The proteins are translated by a typical retroviral frameshift mechanism. The protease (PR) domain of RF2PEG10 contains an -Asp-Ser-Gly- sequence, which corresponds to the consensus -Asp-Ser/Thr-Gly- active-site motif of retroviral aspartic proteases. The function of the aspartic protease domain of RF2PEG10 remains

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18

Mulaudzi, Anzai, Caven Mguvane Mnisi, and Victor Mlambo. "Enhancing the Utility of Dietary Moringa oleifera Leaf Meal for Sustainable Jumbo quail (Coturnix sp.) Production." Sustainability 14, no. 9 (2022): 5067. http://dx.doi.org/10.3390/su14095067.

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The effect of pre-treating Moringa oleifera leaf powder (MOLP) with different levels of polyethylene glycol (PEG) on the growth performance, serum biochemistry, hematology, and meat quality parameters of Jumbo quail was evaluated. Two-week-old quail chicks (n = 432; 239.6 ± 6.48 g live-weight) were randomly allocated to six diets formulated by incorporating (10% w/w) untreated MOLP (PEG0) or MOLP pre-treated with PEG at 2.5% (PEG25), 5% (PEG50), 7.5% (PEG75), and 10% (PEG100) (w/w) into a standard grower diet (CON). Overall feed intake linearly increased with PEG levels. At week 4, significant

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Mou, Dachao, Shasha Wu, Yanqiong Chen, et al. "Roles of PEG10 in cancer and neurodegenerative disorder (Review)." Oncology Reports 53, no. 5 (2025): 1–9. https://doi.org/10.3892/or.2025.8893.

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Nakamura, Akie, Koji Muroya, Hiroko Ogata-Kawata, et al. "A case of paternal uniparental isodisomy for chromosome 7 associated with overgrowth." Journal of Medical Genetics 55, no. 8 (2018): 567–70. http://dx.doi.org/10.1136/jmedgenet-2017-104986.

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BackgroundPaternal uniparental disomy for chromosome 7 (upd(7)pat) is extremely rare, and only four cases have been previously reported. As these cases were accompanied by autosomal-recessive disorders which are likely to be involved in growth restriction, the relevance of upd(7)pat to the overgrowth phenotype remains unclear. Here we describe one case of upd(7)pat with no additional genetic diseases, which may answer the question.MethodsA 5-year-old Japanese boy presented with a tall stature of unknown causes. To detect the genetic cause of the tall stature, we performed Sanger sequencing, ta

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Chen, Haiying, Manni Sun, Ge Zhao, et al. "Elevated expression of PEG10 in human placentas from preeclamptic pregnancies." Acta Histochemica 114, no. 6 (2012): 589–93. http://dx.doi.org/10.1016/j.acthis.2011.11.003.

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Akamatsu, Shusuke, Alexander W. Wyatt, Dong Lin, et al. "The Placental Gene PEG10 Promotes Progression of Neuroendocrine Prostate Cancer." Cell Reports 12, no. 6 (2015): 922–36. http://dx.doi.org/10.1016/j.celrep.2015.07.012.

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LI, XINRAN, RUIJING XIAO, KINGSLEY TEMBO, et al. "PEG10 promotes human breast cancer cell proliferation, migration and invasion." International Journal of Oncology 48, no. 5 (2016): 1933–42. http://dx.doi.org/10.3892/ijo.2016.3406.

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Li, Chi-Ming, Adam A. Margolin, Martha Salas, et al. "PEG10 Is a c-MYC Target Gene in Cancer Cells." Cancer Research 66, no. 2 (2006): 665–72. http://dx.doi.org/10.1158/0008-5472.can-05-1553.

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Jie, X., C. Lang, Q. Jian, et al. "Androgen activates PEG10 to promote carcinogenesis in hepatic cancer cells." Oncogene 26, no. 39 (2007): 5741–51. http://dx.doi.org/10.1038/sj.onc.1210362.

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Akamatsu, Shusuke, Alexander W. Wyatt, Dong Lin, et al. "The Placental Gene PEG10 Promotes Progression of Neuroendocrine Prostate Cancer." Cell Reports 44, no. 4 (2025): 115600. https://doi.org/10.1016/j.celrep.2025.115600.

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Argyraki, Maria, Pauliina Damdimopoulou, Katerina Chatzimeletiou, et al. "In-utero stress and mode of conception: impact on regulation of imprinted genes, fetal development and future health." Human Reproduction Update 25, no. 6 (2019): 777–801. http://dx.doi.org/10.1093/humupd/dmz025.

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AbstractBACKGROUNDGenomic imprinting is an epigenetic gene regulatory mechanism; disruption of this process during early embryonic development can have major consequences on both fetal and placental development. The periconceptional period and intrauterine life are crucial for determining long-term susceptibility to diseases. Treatments and procedures in assisted reproductive technologies (ART) and adverse in-utero environments may modify the methylation levels of genomic imprinting regions, including insulin-like growth factor 2 (IGF2)/H19, mesoderm-specific transcript (MEST), and paternally

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Amin, Muhammad Talal, Louis Coussement, and Tim De Meyer. "Characterization of Loss-of-Imprinting in Breast Cancer at the Cellular Level by Integrating Single-Cell Full-Length Transcriptome with Bulk RNA-Seq Data." Biomolecules 14, no. 12 (2024): 1598. https://doi.org/10.3390/biom14121598.

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Genomic imprinting, the parent-of-origin-specific gene expression, plays a pivotal role in growth regulation and is often dysregulated in cancer. However, screening for imprinting is complicated by its cell-type specificity, which bulk RNA-seq cannot capture. On the other hand, large-scale single-cell RNA-seq (scRNA-seq) often lacks transcript-level detail and is cost-prohibitive. Here, we address this gap by integrating bulk RNA-seq with full-length transcript scRNA-seq to investigate imprinting dynamics in breast cancer. By analyzing scRNA-seq data from 486 cancer cells across subtypes, we i

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Ip, Wai-Ki, Paul B. S. Lai, Navy L. Y. Wong, et al. "Identification of PEG10 as a progression related biomarker for hepatocellular carcinoma." Cancer Letters 250, no. 2 (2007): 284–91. http://dx.doi.org/10.1016/j.canlet.2006.10.012.

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Ge, Hua, Yan Yan, Di Wu, Yongsheng Huang, and Fei Tian. "Prognostic value of PEG10 in Asian solid tumors: A meta-analysis." Clinica Chimica Acta 483 (August 2018): 197–203. http://dx.doi.org/10.1016/j.cca.2018.04.041.

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Hishida, Tomoaki, Kumiko Naito, Shigehiro Osada, Makoto Nishizuka, and Masayoshi Imagawa. "peg10, an imprinted gene, plays a crucial role in adipocyte differentiation." FEBS Letters 581, no. 22 (2007): 4272–78. http://dx.doi.org/10.1016/j.febslet.2007.07.074.

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Hossain, Md Nazmul, Yao Gao, Michael J. Hatfield, Jeanene M. de Avila, Matthew C. McClure, and Min Du. "PSV-9 Cold Exposure Impacts Dna Methylation Patterns of Genes in Cattle Sperm Involved in Early Embryonic and Bone Development." Journal of Animal Science 101, Supplement_3 (2023): 399–400. http://dx.doi.org/10.1093/jas/skad281.475.

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Abstract DNA methylation and other epigenetic modifications are indispensable for maintaining sperm quality, fertilization capacity, and embryonic and postnatal development. In mice, environmental factors, such as stress, nutrition, or exposure to the cold condition, have been demonstrated as factors that alter methylation marks of sperm that are passed to the subsequent generation through transgenerational inheritance and genomic imprinting. Breeding bulls from northern regions of the USA are exposed to extreme cold for 2 to 3 months during the winter; however, no single study has analyzed th

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Renfree, Marilyn B., Eleanor I. Ager, Geoff Shaw, and Andrew J. Pask. "Genomic imprinting in marsupial placentation." REPRODUCTION 136, no. 5 (2008): 523–31. http://dx.doi.org/10.1530/rep-08-0264.

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Genomic imprinting is a widespread epigenetic phenomenon in eutherian mammals, which regulates many aspects of growth and development. Parental conflict over the degree of maternal nutrient transfer is the favoured hypothesis for the evolution of imprinting. Marsupials, like eutherian mammals, are viviparous but deliver an altricial young after a short gestation supported by a fully functional placenta, so can shed light on the evolution and time of acquisition of genomic imprinting. All orthologues of eutherian imprinted genes examined have a conserved expression in the marsupial placenta reg

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Berland, Siren, Cecilie F. Rustad, Mariann H. L. Bentsen, et al. "Double paternal uniparental isodisomy 7 and 15 presenting with Beckwith–Wiedemann spectrum features." Molecular Case Studies 7, no. 6 (2021): a006113. http://dx.doi.org/10.1101/mcs.a006113.

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Here we describe for the first time double paternal uniparental isodisomy (iUPD) 7 and 15 in a baby boy with features in the Beckwith–Wiedemann syndrome spectrum (BWSp) (placentomegaly, hyperinsulinism, enlarged viscera, hemangiomas, and earlobe creases) in addition to conjugated hyperbilirubinemia. His phenotype was also reminiscent of genome-wide paternal uniparental isodisomy. We discuss the most likely origin of the UPDs: a maternal double monosomy 7 and 15 rescued by duplication of the paternal chromosomes after fertilization. So far, paternal UPD7 is not associated with an abnormal pheno

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Renfree, Marilyn B., Shunsuke Suzuki, and Tomoko Kaneko-Ishino. "The origin and evolution of genomic imprinting and viviparity in mammals." Philosophical Transactions of the Royal Society B: Biological Sciences 368, no. 1609 (2013): 20120151. http://dx.doi.org/10.1098/rstb.2012.0151.

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Genomic imprinting is widespread in eutherian mammals. Marsupial mammals also have genomic imprinting, but in fewer loci. It has long been thought that genomic imprinting is somehow related to placentation and/or viviparity in mammals, although neither is restricted to mammals. Most imprinted genes are expressed in the placenta. There is no evidence for genomic imprinting in the egg-laying monotreme mammals, despite their short-lived placenta that transfers nutrients from mother to embryo. Post natal genomic imprinting also occurs, especially in the brain. However, little attention has been pa

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LIANG, Xiao-Yan. "Genetic imprinted genePEG10expression in deciduas from inevitable abortion." Hereditas (Beijing) 30, no. 6 (2008): 735–40. http://dx.doi.org/10.3724/sp.j.1005.2008.00735.

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Jie, X., C. Lang, Q. Jian, et al. "Retraction Note: Androgen activates PEG10 to promote carcinogenesis in hepatic cancer cells." Oncogene 30, no. 24 (2011): 2798. http://dx.doi.org/10.1038/onc.2011.66.

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Zang, W., T. Wang, J. Huang, et al. "Long noncoding RNA PEG10 regulates proliferation and invasion of esophageal cancer cells." Cancer Gene Therapy 22, no. 3 (2015): 138–44. http://dx.doi.org/10.1038/cgt.2014.77.

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Xiaoyan, Liang, Chen Xiong, Zuo Wanxing, and He Junlin. "Genetic imprinted gene PEG10 expression in deciduas of normal early pregnant women." Journal of Medical Colleges of PLA 24, no. 6 (2009): 329–36. http://dx.doi.org/10.1016/s1000-1948(10)60003-2.

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Johansson, A., A. Gogoll, and J. Tegenfeldt. "Diffusion and ionic conductivity in Li(CF3SO3)PEG10 and LiN(CF3SO2)2PEG10." Polymer 37, no. 8 (1996): 1387–93. http://dx.doi.org/10.1016/0032-3861(96)81136-0.

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Zhao, Lixia, Gaoping Zhao, Haiyan Xi, Yiyi Liu, Kaifeng Wu, and Huanmin Zhou. "Molecular and DNA methylation analysis of Peg10 and Xist gene in sheep." Molecular Biology Reports 38, no. 5 (2010): 3495–504. http://dx.doi.org/10.1007/s11033-010-0460-0.

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SU, Jian-Min, Wen-Bing XU, Yan-Yan LI, Li-Jun WANG, Yong-Sheng WANG, and Yong ZHANG. "The methylation status of PEG10 in placentas of cloned transgenic calves." Hereditas (Beijing) 33, no. 5 (2011): 533–38. http://dx.doi.org/10.3724/sp.j.1005.2011.00533.

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Zhang, Minfeng, Chengjun Sui, Binghua Dai, Weifeng Shen, Jiongjiong Lu та Jiamei Yang. "PEG10 is imperative for TGF-β1-induced epithelial-mesenchymal transition in hepatocellular carcinoma". Oncology Reports 37, № 1 (2016): 510–18. http://dx.doi.org/10.3892/or.2016.5282.

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Kawai, Yoshihisa, Kenjiro Imada, Shusuke Akamatsu, et al. "Paternally Expressed Gene 10 (PEG10) Promotes Growth, Invasion, and Survival of Bladder Cancer." Molecular Cancer Therapeutics 19, no. 10 (2020): 2210–20. http://dx.doi.org/10.1158/1535-7163.mct-19-1031.

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Piedrahita, J., S. Bischoff, J. Estrada, et al. "263 USE OF PORCINE PARTHENOTES AND GENE EXPRESSION PROFILING USING MICROARRAYS FOR IDENTIFICATION OF IMPRINTED GENES." Reproduction, Fertility and Development 18, no. 2 (2006): 239. http://dx.doi.org/10.1071/rdv18n2ab263.

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Streszczenie:

Genomic imprinting arises from differential epigenetic markings including DNA methylation and histone modifications and results in one allele being expressed in a parent-of-origin specific manner. For further insight into the porcine epigenome, gene expression profiles of parthenogenetic (PRT; two maternally derived chromosome sets) and biparental embryos (BP; one maternal and one paternal set of chromosomes) were compared using microarrays. Comparison of the expression profiles of the two tissue types permits identification of both maternally and paternally imprinted genes and thus the degree

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Bang, Heejin, Sang Yun Ha, Soo Hyun Hwang, and Cheol-Keun Park. "Expression of PEG10 Is Associated with Poor Survival and Tumor Recurrence in Hepatocellular Carcinoma." Cancer Research and Treatment 47, no. 4 (2015): 844–52. http://dx.doi.org/10.4143/crt.2014.124.

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Liang, X. Y., X. Chen, Y. Z. Jin, X. O. Chen, and Q. Z. Chen. "Expression and significance of the imprinted gene PEG10 in placenta of patients with preeclampsia." Genetics and Molecular Research 13, no. 4 (2014): 10607–14. http://dx.doi.org/10.4238/2014.december.18.2.

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DENG, XINZHOU, YI HU, QIANSHAN DING, et al. "PEG10 plays a crucial role in human lung cancer proliferation, progression, prognosis and metastasis." Oncology Reports 32, no. 5 (2014): 2159–67. http://dx.doi.org/10.3892/or.2014.3469.

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Ono, Ryuichi, Kenji Nakamura, Kimiko Inoue, et al. "Deletion of Peg10, an imprinted gene acquired from a retrotransposon, causes early embryonic lethality." Nature Genetics 38, no. 1 (2005): 101–6. http://dx.doi.org/10.1038/ng1699.

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Tsuji, Kazuhiro, Kohichiroh Yasui, Yasuyuki Gen, et al. "PEG10 is a probable target for the amplification at 7q21 detected in hepatocellular carcinoma." Cancer Genetics and Cytogenetics 198, no. 2 (2010): 118–25. http://dx.doi.org/10.1016/j.cancergencyto.2010.01.004.

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