Gotowe bibliografie tematyczne / Porphyromonas gingivalis infections

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  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji

Gotowa bibliografia na temat „Porphyromonas gingivalis infections”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 1 lutego 2022

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Artykuły w czasopismach na temat "Porphyromonas gingivalis infections"

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Winkelhoff, Arie J., and Jørgen Slots. "Actinobacillus actinomycetemcomitans and Porphyromonas gingivalis in nonoral infections." Periodontology 2000 20, no. 1 (June 1999): 122–35. http://dx.doi.org/10.1111/j.1600-0757.1999.tb00160.x.

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Condorelli, Francesca, Guido Scalia, Giuditta Calì, Bruno Rossetti, Giuseppe Nicoletti, and Anna M. Lo Bue. "Isolation of Porphyromonas gingivalisand Detection of Immunoglobulin A Specific to Fimbrial Antigen in Gingival Crevicular Fluid." Journal of Clinical Microbiology 36, no. 8 (1998): 2322–25. http://dx.doi.org/10.1128/jcm.36.8.2322-2325.1998.

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The present study evaluated the prevalence of Porphyromonas gingivalis and the correlation between the bacterial culture method and the detection of immunoglobulin A (IgA) specific to theP. gingivalis fimbrial antigen in gingival crevicular fluid (GCF). P. gingivalis was isolated from 78.3% of subgingival plaque samples obtained from active sites and 34.7% of those from inactive sites of periodontal patients. P. gingivalis was isolated from only 4.7% of healthy subjects (control group). Immunoglobulins specific to the P. gingivalis fimbrial antigen were detected by enzyme-linked immunosorbent
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Hirasawa, Masaaki, and Tomoko Kurita-Ochiai. "Porphyromonas gingivalis Induces Apoptosis and Autophagy via ER Stress in Human Umbilical Vein Endothelial Cells." Mediators of Inflammation 2018 (July 29, 2018): 1–8. http://dx.doi.org/10.1155/2018/1967506.

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It has been reported that periodontitis is associated with an increased risk of atherosclerosis. Accumulating evidence suggests that endothelial dysfunction is an early marker for atherosclerosis. To determine how periodontal infections contribute to endothelial dysfunction, we examined the effect of Porphyromonas gingivalis on human umbilical vein endothelial cells (HUVEC). P. gingivalis significantly suppressed the viability of HUVEC, induced DNA fragmentation and annexin V staining, and increased caspase-3, caspase-8, and caspase-9 activities. P. gingivalis also increased the expression of
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Wu, Jie, and Hua Xie. "Role of Arginine Deiminase of Streptococcus cristatus in Porphyromonas gingivalis Colonization." Antimicrobial Agents and Chemotherapy 54, no. 11 (July 26, 2010): 4694–98. http://dx.doi.org/10.1128/aac.00284-10.

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ABSTRACT The ability to attach to a variety of oral surfaces is an important characteristic of Porphyromonas gingivalis. Previous studies have demonstrated that expression and production of FimA, a major subunit protein of the long fimbriae, is required for P. gingivalis colonization. Here we report that a surface protein, arginine deiminase (ArcA) of Streptococcus cristatus, represses FimA production and inhibits biofilm formation of P. gingivalis. This inhibitory function of ArcA is also observed in the formation of heterotypic P. gingivalis-Streptococcus gordonii biofilms. P. gingivalis is
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Rôças, Isabela N., and José F. Siqueira. "Distribution of Porphyromonas gingivalis fimA genotypes in primary endodontic infections." Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontology 109, no. 3 (March 2010): 474–78. http://dx.doi.org/10.1016/j.tripleo.2009.11.009.

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Odell, Lynnetta J., J. Craig Baumgartner, Tian Xia, and Larry L. David. "Survey for collagenase gene prtC in Porphyromonas gingivalis and Porphyromonas endodontalis isolated from endodontic infections." Journal of Endodontics 25, no. 8 (August 1999): 555–58. http://dx.doi.org/10.1016/s0099-2399(99)80379-3.

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Fiorillo, Luca, Gabriele Cervino, Luigi Laino, Cesare D’Amico, Rodolfo Mauceri, Tolga Fikret Tozum, Michele Gaeta, and Marco Cicciù. "Porphyromonas gingivalis, Periodontal and Systemic Implications: A Systematic Review." Dentistry Journal 7, no. 4 (December 11, 2019): 114. http://dx.doi.org/10.3390/dj7040114.

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In recent scientific literature, oral infections and systemic manifestations, or correlations between oral health and systemic diseases are a topic of discussion. Porphyromonas gingivalis is one of the bacteria implicated in the biofilm formation of bacterial plaque, and plays an important role in the progression of periodontal disease. In this systematic review authors have evaluated the literature of the last 10 years on P. gingivalis and all the systemic implications proven. This study therefore evaluates all the districts of the organism in which this bacterium may have implications. From
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Maezono, H., Y. Noiri, Y. Asahi, M. Yamaguchi, R. Yamamoto, N. Izutani, H. Azakami, and S. Ebisu. "Antibiofilm Effects of Azithromycin and Erythromycin on Porphyromonas gingivalis." Antimicrobial Agents and Chemotherapy 55, no. 12 (September 12, 2011): 5887–92. http://dx.doi.org/10.1128/aac.05169-11.

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ABSTRACTAntibiotic resistance of biofilm-grown bacteria contributes to chronic infections, such as marginal and periapical periodontitis, which are strongly associated withPorphyromonas gingivalis. Concurrent azithromycin (AZM) administration and mechanical debridement improve the clinical parameters of periodontal tissuein situ. We examined thein vitroefficacy of AZM againstP. gingivalisbiofilms. The susceptibilities of adherentP. gingivalisstrains 381, HW24D1, 6/26, and W83 to AZM, erythromycin (ERY), ampicillin (AMP), ofloxacin (OFX), and gentamicin (GEN) were investigated using a static mo
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Siqueira, José F., Isabela N. Rôças, and Marlei G. Silva. "Prevalence and Clonal Analysis of Porphyromonas gingivalis in Primary Endodontic Infections." Journal of Endodontics 34, no. 11 (November 2008): 1332–36. http://dx.doi.org/10.1016/j.joen.2008.08.021.

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Loos, B. G., D. Mayrand, R. J. Genco, and D. P. Dickinson. "Genetic Heterogeneity of Porphyromonas (Bacteroides) gingivalis by Genomic DNA Fingerprinting." Journal of Dental Research 69, no. 8 (August 1990): 1488–93. http://dx.doi.org/10.1177/00220345900690080801.

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This study describes the use of total genomic DNA fingerprinting with the use of restriction endonucleases to characterize clinical isolates of Porphyromonas gingivalis (Bacteroides gingivalis) obtained from patients with periodontitis or with root-canal infections. The majority of independent isolates had a unique DNA fingerprint, indicating extensive genetic heterogeneity within this species. Twenty-nine distinct DNA fingerprints were found among the 33 isolates investigated. This is in contrast to biotyping and serotyping, where only one type and three types, respectively, have been reporte
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Rozprawy doktorskie na temat "Porphyromonas gingivalis infections"

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Xie, Hua. "Regulation of fimbrillin expression in Porphyromonas gingivalis." Thesis, Connect to this title online; UW restricted, 1998. http://hdl.handle.net/1773/6392.

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Díaz, Patricia I. "Studies on the oxidative stress response of porphyromonas gingivalis : a thesis submitted in fulfillment of the requirements for admission to the degree of Doctor of Philosophy /." Title page, summary and table of contents only, 2002. http://web4.library.adelaide.edu.au/theses/09PH/09phd5426.pdf.

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Leclerc, Julia. "Etude d’un système respiratoire de Porphyromonas gingivalis, pathogène impliqué dans les infections parodontales." Thesis, Rennes 1, 2015. http://www.theses.fr/2015REN1B032/document.

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Les parodontites sont des maladies chroniques inflammatoires causées par un biofilm bactérien. Elles sont la première cause de perte des dents dans les pays industrialisés et représentent donc un coût important pour la société. Le biofilm buccal est composé de plus de 500 espèces différentes, parmi lesquelles Porphyromonas gingivalis est reconnue comme une cause majeure du développement des symptômes. Cette bactérie à Gram négatif est considérée comme anaérobie bien qu’elle tolère des concentrations faibles en oxygène, ce qui favorise la colonisation de la cavité orale. Notre objectif était de
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Belvin, Benjamin R. "Nitrosative stress sensing in Porphyromonas gingivalis: structure and function of the heme binding transcriptional regulator HcpR." VCU Scholars Compass, 2017. https://scholarscompass.vcu.edu/etd/5123.

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Porphyromonas gingivalis, a Gram negative anaerobe implicated in the progression of periodontal disease, is capable of surviving and causing infection despite high levels of reactive nitrogen species found in the oral cavity due to its efficient nitrosative stress response. HcpR is an important sensor-regulator that plays a vital step in the initiation of the nitrosative stress response in many Gram negative anaerobic bacteria. We employ a combination of X-ray crystallography, SAXS, resonance Raman spectroscopy, UV-Vis spectroscopy, and molecular biology techniques to better understand this ke
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Huck, Olivier. "Infection et stimulation de cellules endothéliales par Porphyromonas gingivalis et son lipopolysaccharide : lien entre maladies parodontales et athérosclérose." Thesis, Strasbourg, 2013. http://www.theses.fr/2013STRAJ021.

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Depuis plusieurs années, l’influence des pathologies parodontales sur certaines pathologies systémiques, notamment les maladies cardio-vasculaires et l’athérosclérose apparait de plus en plus évidente. Dans notre étude, nous nous sommes intéressés à l’évaluation des effets induits par Porphyromonas gingivalis, une des principales bactéries parodontopathogènes, et son lipopolysaccharide sur les cellules endothéliales, qui forment une interface entre le flux sanguin et la paroi vasculaire, d’où un rôle important dans l’initiation et le développement de la plaque d’athérome. Nous avons surtout ci
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Nakka, Sravya Sowdamini. "Development of novel tools for prevention and diagnosis of Porphyromonas gingivalis infection and periodontitis." Doctoral thesis, Örebro universitet, Institutionen för medicinska vetenskaper, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-52056.

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Periodontitis is a chronic inflammatory disease caused by exaggerated host immune responses to dysregulated microbiota in dental biofilms leading to degradation of tissues and alveolar bone loss. Porphyromonas gingivalis is a major periodontal pathogen and expresses several potent virulence factors. Among these factors, arginine and lysine gingipains are of special importance, both for the bacterial survival/proliferation and the pathological outcome. The major aim of this thesis was to develop and test novel methods for diagnosis and prevention of P. gingivalis infection and periodontitis. In
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Reader, Brenda Faye. "Social Stress Induces Immunoenhancement During Allergic Airway Inflammation and Infection." The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1385475903.

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Gross, Jane Elizabeth. "Local and systemic host immune responses to Porphyromonas gingivalis A7436 infection in a subcutaneous tissue chamber in untreated and lipopolysaccharide-treated mice." 1999. http://catalog.hathitrust.org/api/volumes/oclc/48199463.html.

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Díaz, Patricia I. "Studies on the oxidative stress response of porphyromonas gingivalis : a thesis submitted in fulfillment of the requirements for admission to the degree of Doctor of Philosophy." 2002. http://web4.library.adelaide.edu.au/theses/09PH/09phd5426.pdf.

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Lin, Yuh-Yih. "Characterization of the molecular and cellular events that determine outcome of infection with Porphyromonas gingivalis in a mouse chamber model." Diss., 1998. http://catalog.hathitrust.org/api/volumes/oclc/47976721.html.

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Książki na temat "Porphyromonas gingivalis infections"

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N, Shah Haroun, Mayrand D, and Genco Robert J, eds. Biology of the species Porphyromonas gingivalis. Boca Raton: CRC Press, 1993.

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J, Genco Robert, ed. Molecular pathogenesis of periodontaldisease. Washington, D.C: ASM Press, 1994.

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J, Genco Robert, ed. Molecular pathogenesis of periodontal disease. Washington, D.C: ASM Press, 1994.

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Części książek na temat "Porphyromonas gingivalis infections"

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Joly, Sophie, Myriam Bélanger, Georgia K. Johnson, Ann Progulske-Fox, and Kim A. Brogden. "Infection with Porphyromonas gingivalis, a Potential Risk Factor for Chronic Systemic Disease." In Sequelae and Long-Term Consequences of Infectious Diseases, 443–57. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815486.ch25.

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Han, Xiaozhe, Xiaoping Lin, Toshihisa Kawai, Karen B. LaRosa, and Martin A. Taubman. "Porphyromonas gingivalis infection elicits immune-mediated RANKL-dependent periodontal bone loss in rats." In Interface Oral Health Science 2009, 400–402. Tokyo: Springer Japan, 2010. http://dx.doi.org/10.1007/978-4-431-99644-6_114.

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Khalaf, Hazem, Eleonor Palm, and Torbjörn Bengtsson. "Cellular Response Mechanisms in Porphyromonas gingivalis Infection." In Periodontitis - A Useful Reference. InTech, 2017. http://dx.doi.org/10.5772/intechopen.69019.

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Streszczenia konferencji na temat "Porphyromonas gingivalis infections"

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Jenning, M., B. Marklein, Z. Konthur, U. Nonhoff, G.-R. Burmester, and K. Skriner. "P043 Infection with citrullinating porphyromonas gingivalis can induce autoimmunity to human ribosomal proteins." In 39th European Workshop for Rheumatology Research, 28 February–2 March 2019, Lyon, France. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2019.35.

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Jenning, M., B. Marklein, Y. Ytterberg, A. Catarina, D. Schaardenburg, G. Bürmester, and K. Skriner. "P013 Porphyromonas gingivalis infection linked to ra onset and ANTI TNF alpha treatment non-response." In 38th European Workshop for Rheumatology Research, 22–24 February 2018, Geneva, Switzerland. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2018.38.

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Jenning, Madeleine, Bianka Marklein, Ute Nonhoff, Zoltan Konthur, Gerd Rüdiger Burmester, and Karl Skriner. "SAT0049 INFECTION WITH CITRULLINATING PORPHYROMONAS GINGIVALIS CAN INDUCE AUTOIMMUNITY TO HUMAN RIBOSOMAL PROTEINS AND TNF ALPHA TREATMENT NONRESPONSE." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.4653.

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