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Artykuły w czasopismach na temat „Psoriasiform”

Autor: Grafiati

Data publikacji: 3 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Sehgal, VirendraN, Sunil Dogra, Govind Srivastava, and AshokK Aggarwal. "Psoriasiform dermatoses." Indian Journal of Dermatology, Venereology and Leprology 74, no. 2 (2008): 94. http://dx.doi.org/10.4103/0378-6323.39688.

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Carbone, Angelo, Clara De Simone, Francesco Valenzano, Pierluigi Amerio, and Guido Massi. "Psoriasiform keratosis." European Journal of Dermatology 19, no. 6 (2009): 631–32. http://dx.doi.org/10.1684/ejd.2009.0760.

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3

Walsh, Sarah N., Mark A. Hurt, and Daniel J. Santa Cruz. "Psoriasiform Keratosis." American Journal of Dermatopathology 29, no. 2 (2007): 137–40. http://dx.doi.org/10.1097/01.dad.0000246177.63145.b3.

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4

Choi, Jung Chul, Kyoung Ae Jang, Jee-Ho Choi, Kyung-Jeh Sung, Kee-Chan Moon, and Jai-Kyoung Koh. "Psoriasiform Sarcoidosis." Annals of Dermatology 12, no. 4 (2000): 303. http://dx.doi.org/10.5021/ad.2000.12.4.303.

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5

Ramezani, Mazaher, Atefeh Shamshiri, Elisa Zavattaro, et al. "Immunohistochemical expression of P53, Ki-67, and CD34 in psoriasis and psoriasiform dermatitis." BioMedicine 9, no. 4 (2019): 26. http://dx.doi.org/10.1051/bmdcn/2019090426.

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Background: Psoriasis is the prime example of psoriasiform tissue pattern and should be differentiated from other psoriasiform dermatoses both clinically and histopathologically. Aim: To evaluate immunohistochemical expression of P53, Ki-67, and CD34 in psoriasis and psoriasiform dermatitis for diagnostic purposes. Methods: An analytical cross-sectional study was performed on the paraffin blocks of 60 psoriasis and 31 psoriasiform dermatitis patients between 2014 and 2017. The selected formalin-fixed paraffin-embedded tissues from each biopsy specimen were cut into 4-micron sections. Initial s

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6

Evdokimov, Evgeny Yu, L. M. Smirnova, and M. A. Golubev. "PSORIASIFORM CUTANEOUS SARCOIDOSIS." Russian Journal of Skin and Venereal Diseases 22, no. 1-2 (2019): 11–14. http://dx.doi.org/10.17816/dv42930.

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Psoriasiform cutaneous sarcoidosis relates to atypical forms of sarcoidosis. The clinical case with a typical manifestation and the formation of noncaseating granulomas and a specific histological pattern associated with vulgar psoriasis is described. Despite the relative prevalence of cutaneous forms of sarcoidosis, clinical recognition of the disease sometimes presents certain difficulties and requires close attention of clinicians.

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7

Nair, PragyaA, and NidhiB Jiwani. "Psoriasiform lupus vulgaris." Egyptian Journal of Dermatology and Venerology 37, no. 1 (2017): 30. http://dx.doi.org/10.4103/1110-6530.207491.

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8

Guo, Hao, Xue-Gang Xu, Xiu-Hao Guan, Xing-Hua Gao, Hong-Duo Chen, and Jiu-Hong Li. "Psoriasiform cutaneous sporotrichosis." International Journal of Dermatology 54, no. 9 (2015): e364-e365. http://dx.doi.org/10.1111/ijd.12814.

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9

Patalay, R., P. Fields, M. Kazmi, N. D. Francis, and C. B. Bunker. "A psoriasiform rash." Clinical and Experimental Dermatology 35, no. 2 (2010): 209–10. http://dx.doi.org/10.1111/j.1365-2230.2009.03244.x.

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10

Sánchez-Salas, M. P., F. J. García-Latasa de Aranibar, and M. A. Ania. "Acral Psoriasiform Lesions." Actas Dermo-Sifiliográficas (English Edition) 100, no. 4 (2009): 325–26. http://dx.doi.org/10.1016/s1578-2190(09)70071-x.

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11

Veraldi, Stefano, Chiara Galloni, Raffaella Cremonesi, and Riccardo Cavalli. "Psoriasiform cutaneous leishmaniasis." International Journal of Dermatology 45, no. 2 (2006): 129–30. http://dx.doi.org/10.1111/j.1365-4632.2006.02691.x.

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12

Sutarjono, Bayu, and Herman Lebovitch. "Psoriasiform spongiotic dermatitis." BMJ Case Reports 12, no. 3 (2019): e228690. http://dx.doi.org/10.1136/bcr-2018-228690.

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13

Hall, M. J., R. Kovach, and C. Parsons. "Recalcitrant Psoriasiform Rash." Pediatric Dermatology 19, no. 2 (2002): 180–82. http://dx.doi.org/10.1046/j.1525-1470.2002.00037.x.

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14

Boje Rasmussen, H., H. Hagdrup, and H. Schmidt. "Psoriasiform napkin dermatitis." Acta Dermato-Venereologica 66, no. 6 (1986): 534–36. http://dx.doi.org/10.2340/0001555566534536.

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A prognostic study of 18 patients with a diagnosis of psoriasiform napkin dermatitis was performed. The observation period had a duration of 7 to 15 years. Two patients developed psoriasis and two developed atopic dermatitis.

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15

Almutairi, Dalal, Christopher Sheasgreen, Adam Weizman, and Afsaneh Alavi. "Generalized Pustular Psoriasis Induced by Infliximab in a Patient With Inflammatory Bowel Disease." Journal of Cutaneous Medicine and Surgery 22, no. 5 (2018): 507–10. http://dx.doi.org/10.1177/1203475418758986.

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The incidence of anti–tumour necrosis factor (TNF)–induced psoriasiform eruptions has ranged in the literature between 0.5% and 10.9% of patients with inflammatory bowel disease (IBD). The morphology of the rash varies, with plaque psoriasis being the most common form. Generalized pustular eruption is reported in 10.2% of cases of anti–TNF-induced psoriasis. We are reporting a unique case of anti–TNF-associated psoriasiform eruption due to the severity of the rash presenting as a generalized pustular eruption in association with plaque psoriasiform rash requiring systemic therapy.

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16

Choudhary, Vivek, Ismail Kaddour-Djebbar, Victoria E. Custer, et al. "Glycerol Improves Skin Lesion Development in the Imiquimod Mouse Model of Psoriasis: Experimental Confirmation of Anecdotal Reports from Patients with Psoriasis." International Journal of Molecular Sciences 22, no. 16 (2021): 8749. http://dx.doi.org/10.3390/ijms22168749.

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Glycerol is used in many skin care products because it improves skin function. Anecdotal reports by patients on the National Psoriasis Foundation website also suggest that glycerol may be helpful for the treatment of psoriasis, although to date no experimental data confirm this idea. Glycerol entry into epidermal keratinocytes is facilitated by aquaglyceroporins like aquaporin-3 (AQP3), and its conversion to phosphatidylglycerol, a lipid messenger that promotes keratinocyte differentiation, requires the lipid-metabolizing enzyme phospholipase-D2 (PLD2). To evaluate whether glycerol inhibits in

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17

Tanita, Kayo, Taku Fujimura, Aya Kakizaki, Sadanori Furudate, Yoshiyuki Kusakari, and Setsuya Aiba. "Psoriasiform Drug Eruption Caused by Abatacept: Immunohistochemical Investigation of STAT Signaling." Case Reports in Dermatology 7, no. 2 (2015): 166–70. http://dx.doi.org/10.1159/000437415.

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Abatacept is a biological immune modifier that is used for the treatment of rheumatoid arthritis. Although psoriasiform drug eruption is reported as one of the cutaneous adverse effects of abatacept, the precise mechanisms are not fully understood. In this report, we describe a 65-year-old Japanese man with psoriasiform drug eruption caused by abatacept. Interestingly, immunohistochemical staining revealed that the epidermal keratinocytes in the basal layer and lower layers of the stratum spinosum were positive for pSTAT3, partially positive for pSTAT1 and negative for pSTAT6, which is similar

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18

Novice, Karlee, Jesse Veenstra, Dana Siegel, and Holly Kerr. "Dupilumab-Induced Psoriasiform Dermatitis." SKIN The Journal of Cutaneous Medicine 4, no. 1 (2020): 72–75. http://dx.doi.org/10.25251/skin.4.1.11.

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Atopic dermatitis (AD) is a common inflammatory skin condition. The newly FDA approved biologic dupilumab has demonstrated significant improvement in the signs and symptoms of AD, including pruritus as well as those of anxiety and depression1. The most frequently reported adverse effects of dupilumab include injection site reactions, nasopharyngitis, upper respiratory infections, and conjunctivitis 1-5. We report a case of dupilumab-induced psoriasiform dermatitis. To our knowledge, there have been no reports to date of a dupilumab-induced psoriasiform dermatitis. There has been one report of

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19

Tirumalae, Rajalakshmi. "Psoriasiform dermatoses: Microscopic approach." Indian Journal of Dermatology 58, no. 4 (2013): 290. http://dx.doi.org/10.4103/0019-5154.113945.

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20

Pires, Carla Andréa Avelar, Brena Andrade de Sousa, Carla do Socorro Silva do Nascimento, Ana Thais Machado Moutinho, Mario Fernando Ribeiro de Miranda, and Francisca Regina Oliveira Carneiro. "Psoriasiform Keratosis - Case report." Anais Brasileiros de Dermatologia 89, no. 2 (2014): 318–19. http://dx.doi.org/10.1590/abd1806-4841.20142504.

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21

MITSUISHl, TSUYOSHI, TOSHITATSU NOGITA, and MAKOTO KAWASHIMA. "PSORIASIFORM SARCOIDOSIS WITH ULCERATION." International Journal of Dermatology 31, no. 5 (1992): 339–40. http://dx.doi.org/10.1111/j.1365-4362.1992.tb03950.x.

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22

Yiu, Zenas, Aysha Javed, Rajani Nalluri, et al. "Infliximab-Associated Psoriasiform Alopecia." American Journal of Gastroenterology 110 (October 2015): S300—S301. http://dx.doi.org/10.14309/00000434-201510001-00681.

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23

Sakemi, Hideta, and Hiroshi Oiwa. "Psoriasiform Plaques of Sarcoidosis." Internal Medicine 48, no. 5 (2009): 391. http://dx.doi.org/10.2169/internalmedicine.48.1686.

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24

Sladden, Michael J., Christopher S. Sladden, and Wayne P. F. Gulliver. "Secukinumab-Induced Psoriasiform Eruption." JAMA Dermatology 153, no. 11 (2017): 1194. http://dx.doi.org/10.1001/jamadermatol.2017.2826.

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25

Chan, Lawrence S. "Generalized Pruritic Psoriasiform Lesions." Archives of Dermatology 127, no. 12 (1991): 1831. http://dx.doi.org/10.1001/archderm.1991.04520010078016.

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26

Sripathi, H., M. Dahal, and A. Basu. "SUBACUTE CUTANEOUS LUPUS ERYTHEMATOSUS WITH PSEUDO-PORPHYRIA AND VITILIGO." Journal of Nepal Medical Association 40, no. 140 (2003): 209–11. http://dx.doi.org/10.31729/jnma.675.

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Sub acute Cutaneous Lupus Erythematosus (SCLE) is a distinct subset of LupusErythematosus. It can present as non scarring psoriasiform or annular polycycliclesions. About half the patients fulfil the criteria of the American RheumatismAssociation for systemic lupus erythematosus. In this report, we present the case of a45 years old male who came with psoriasiform depigmented plaques and papulo-vesicular lesions developed after taking some medication. Clinically diagnosed as subacute cutaneous lupus erythematosus with acral vitiligo. A histopathological studyand urine analysis however revealed

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27

Rafia Bilquis Siddiqui, Shahnaz Imdad Kehar, Mansoorul Aziz, and Naila Zahir. "HISTOPATHOLOGICAL SPECTRUM OF PSORIASIFORM SKIN LESIONS- A CENTRE BASED STUDY." JMMC 6, no. 1 (2015): 1–6. http://dx.doi.org/10.62118/jmmc.v6i1.360.

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Objective: To study frequencies, morphological patterns, and the spectrum of psoriasiform lesions and to assess diagnostic accuracy of histopathology.Study design: Cross-sectional.Place and Duration of Study: This study was conducted from January 2007 to December 2011 at the Department of Histopathology, Basic Medical Science Institute, Jinnah Postgraduate Medical Centre, Karachi.Materials and Methods: All the biopsies of psoriasiform dermatitis were selected for detailed study. The clinicalhistory and relevant data was recorded from the surgical pathology record registers. Haematoxylin and eo

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28

Di Brizzi, Eugenia Veronica, Stefano Caccavale, Caterina Mariarosaria Giorgio, Giuseppe Argenziano, and Anna Balato. "Psoriasiform Dermatitis: From Pathogenesis to New Therapeutic Opportunities." Life 15, no. 7 (2025): 1026. https://doi.org/10.3390/life15071026.

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Psoriasiform dermatitis refers to a spectrum of inflammatory skin disorders that resemble psoriasis both clinically and histologically. These conditions can occur idiopathically or as paradoxical reactions to biologic or targeted therapies, particularly in patients with atopic or autoimmune backgrounds. Histologic features often include acanthosis, parakeratosis, and lymphocytic infiltrates, but without the full molecular signature of classical psoriasis. This review provides an overview of psoriasiform dermatitis with a focus on its clinical presentation, differential diagnosis, and the immun

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29

WANG, ZHEN, HONG ZHOU, HUAPING ZHENG, XIU TENG, XIAOQIONG WEI, and JIONG LI. "Autophagy-based unconventional secretion of alarmin HMGB1 by keratinocytes plays a pivotal role in psoriatic skin inflammation." Journal of Immunology 202, no. 1_Supplement (2019): 59.1. http://dx.doi.org/10.4049/jimmunol.202.supp.59.1.

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Abstract The precise mechanism by which autophagy affects psoriasis is poorly understood. Here we show that keratinocytes (KCs) autophagy was activated in psoriatic lesions of patients and mice model, positively correlating with psoriatic severity, and could be inhibited by MAPK family (ERK, p38, JNK) inactivation, implying autophagy was associated with psoriatic inflammation. Indeed, impaired autophagy flux, caused by autophagy inhibitors or KC-specific deletion of Atg5, alleviated psoriasisform inflammation, demonstrating autophagy positively regulated psoriatic inflammation. We then found a

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30

Tosti, A., P. A. Fanti, R. Morelli, and F. Bardazzi. "Psoriasiform acral dermatitis. Report of three cases." Acta Dermato-Venereologica 72, no. 3 (1992): 206–7. http://dx.doi.org/10.2340/0001555572206207.

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The authors report 3 patients affected by psoriasiform acral dermatitis, a distinctive clinical entity characterized by a chronic dermatitis of the terminal phalanges, associated with marked shortening of the nail beds of the affected fingers. The skin biopsy showed in all cases the pathological features of a subacute spongiotic dermatitis. X-ray examination of affected fingers showed no bone or soft tissue changes. Differential diagnosis of psoriasiform acral dermatitis included psoriasis, atopic or contact dermatitis and corticosteroid-induced distal phalangeal atrophy.

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31

Venna, Ashok Babu, Sravan Chittla, and Shashikant Malkud. "A Clinico-Pathological Study of Psoriasis and Psoriasiform Dermatitis." Journal of Evidence Based Medicine and Healthcare 7, no. 51 (2020): 3085–89. http://dx.doi.org/10.18410/jebmh/2020/629.

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BACKGROUND Psoriasis is a chronic skin condition, which can have varied presentation either per se or because of various treatment modalities, which can closely simulate any different dermatological conditions. Hence, a clinicohistopathological correlation is necessary for confirmation of diagnosis and treatment. Very few studies are available in the indexed journals on this subject matter. The present study is aimed to study the clinical and histological features of psoriasiform dermatitis and psoriasis. METHODS This was a longitudinal study consisting of 60 subjects divided into two classes

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32

Mochizuki, Toshihiko, Tomomitsu Miyagaki, Mayumi Tamaki, Sora Takeuchi та Takafumi Kadono. "β-Hydroxybutyrate Reduces Psoriasiform Dermatitis". Journal of St. Marianna University 11, № 2 (2020): 153–59. http://dx.doi.org/10.17264/stmarieng.11.153.

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33

Rambhia, KinjalDeepak, AmitkumarSureshchandra Gulati, and Sushil Pande. "Psoriasis versus psoriasiform drug eruption." Indian Journal of Drugs in Dermatology 2, no. 1 (2016): 48. http://dx.doi.org/10.4103/2455-3972.184083.

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Verea, María M., Jesús Del Pozo, M. Teresa Yebra-Pimentel, Angeles Porta, and Eduardo Fonseca. "Psoriasiform Eruption Induced by Infliximab." Annals of Pharmacotherapy 38, no. 1 (2004): 54–57. http://dx.doi.org/10.1345/aph.1c477.

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35

Vernali, Steven, Eva Vertes George, Tania Gonzalez Santiago, and Kiran Motaparthi. "Psoriasiform Graft-Versus-Host Disease." American Journal of Dermatopathology 40, no. 7 (2018): 511–14. http://dx.doi.org/10.1097/dad.0000000000001078.

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36

Kwan, Zhenli, Rokiah Binti Che Ismail, Su-Ming Wong, Leng Leng Tan, Suganthy Robinson, and Kheng Seang Lim. "Sodium valproate-aggravated psoriasiform eruption." International Journal of Dermatology 53, no. 10 (2014): e477-e479. http://dx.doi.org/10.1111/ijd.12579.

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STASSIY, ANYA, and AMOR KHACHEMOUNE. "What is this psoriasiform plaque?" Journal of the American Academy of Physician Assistants 24, no. 7 (2011): 15. http://dx.doi.org/10.1097/01720610-201107000-00003.

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Barr, R. J., and E. M. Young. "Psoriasiform and related papulosquamous disorders." Journal of Cutaneous Pathology 12, no. 5 (1985): 412–25. http://dx.doi.org/10.1111/j.1600-0560.1985.tb00439.x.

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Kunzler, Elaine, Christina Cernik, and Joshua Weaver. "Psoriasiform Rash in a Toddler." Journal of Pediatrics 170 (March 2016): 332–332. http://dx.doi.org/10.1016/j.jpeds.2015.11.032.

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Jayalakshmy, PL, AM Babitha, S. Sankar, and G. Nandakumar. "Histopathological spectrum of Psoriasiform dermatitis." Journal of Pathology of Nepal 6, no. 12 (2016): 975–80. http://dx.doi.org/10.3126/jpn.v6i12.16265.

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Background: Psoriasiform dermatitis is a frequently encountered terminology in a wide variety of inflammatory dermatoses. It often poses challenges to both dermatologists and pathologists alike. Clinical features when considered alone may not be reliable, as they vary with both disease duration and treatment. On the contrary, histopathological material constitutes definite hard evidence, which can be preserved and will continue to be available for future review. The objective of the study is to study the histopathological findings in Psoriasiform dermatitis.Materials and Methods: This is a ret

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41

Jinno, Naoko, Yayoi Yamana, Makoto Kawashima, and Yuichiro Tsunemi. "Mycosis fungoides with psoriasiform lesions." Journal of Dermatology 42, no. 2 (2015): 227–29. http://dx.doi.org/10.1111/1346-8138.12749.

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42

Wick, Mark R. "Psoriasiform dermatitides: A brief review." Seminars in Diagnostic Pathology 34, no. 3 (2017): 220–25. http://dx.doi.org/10.1053/j.semdp.2016.12.006.

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Chan, L. S. "Generalized pruritic psoriasiform lesions. Scabies." Archives of Dermatology 127, no. 12 (1991): 1833. http://dx.doi.org/10.1001/archderm.127.12.1833.

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Prendiville, Julie. "Psoriasiform Plaque on the Buttock." Archives of Dermatology 125, no. 1 (1989): 117. http://dx.doi.org/10.1001/archderm.1989.01670130119021.

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Horiuchi, Yasuhiro. "Psoriasiform Eruptions: An Etiological Perspective." American Journal of Dermatopathology 45, no. 9 (2023): 671–73. http://dx.doi.org/10.1097/dad.0000000000002509.

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SEQUEIRA, VIOLA ELVIA, CAROL LOBO, and SOWMYA KAIMAL. "Psoriasiform plaques: A great mimic." National Medical Journal of India 37 (April 16, 2025): 357. https://doi.org/10.25259/nmji_1111_2023.

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Pesqué, D., B. Sanchez-Gonzalez, F. Gallardo, S. Segura, and R. Pujol. "Psoriasiform Eruption Secondary to PI3K-delta Inhibitor: Expanding the Spectrum of Psoriasiform Paradoxical Reactions?" Acta Dermato Venereologica 101, no. 3 (2021): adv00418. http://dx.doi.org/10.2340/00015555-3783.

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Ganjoo, Shikhar, and Tulika Gupta. "Autoeczematization Id reaction following candidal diaper dermatitis." Our Dermatology Online 12, no. 2 (2022): 187–89. http://dx.doi.org/10.7241/ourd.20222.17.

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Id reactions are secondary inflammatory reactions that develop from a remote, localized immunological insult. They may be caused by various fungal, bacterial, viral, and parasitic infections. Diaper dermatitis with psoriasiform Id eruptions is a rarely reported phenomenon. Herein, we report the case of an infant who developed candidal diaper dermatitis followed by generalized psoriasiform Id eruptions. Most authors report the resolution of diaper area lesions with topical antifungals with or without steroids and Id eruptions with topical steroids alone. Our patient showed complete resolution o

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49

M.K., Kumari Kalpana. "A Detailed Study of Histomorphological Spectrum of Psoriasiform Dermatitis." Indian Journal of Pathology: Research and Practice 6, no. 2 (Part-2) (2017): 410–14. http://dx.doi.org/10.21088/ijprp.2278.148x.62(pt-ii)17.12.

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Baggio, Raphaëlle, Claire Le Treut, Laure Darrieux, Amélie Vareliette, and Gilles Safa. "Psoriasiform Diaper Rash Possibly Induced by Oral Propranolol in an 18-Month-Old Girl with Infantile Hemangioma." Case Reports in Dermatology 8, no. 3 (2016): 369–73. http://dx.doi.org/10.1159/000455021.

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Propranolol, a nonselective blocker of β-adrenergic receptors, has become the first-line treatment for complicated infantile hemangiomas. Therefore, its use in the pediatric population has expanded in recent years. In adults, β-blockers have been reported to be the most common causative agents for drug-induced psoriasis. In infants treated with propranolol for infantile hemangioma, the onset of psoriasiform diaper rash has not yet been reported. Here, to the best of our knowledge, we report the first case of psoriasiform diaper rash possibly induced by oral propranolol in an 18-month-old girl

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