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Artykuły w czasopismach na temat "Pylori"

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Chiba, N., A. Matisko, P. Sinclair, and ABR Thomson. "Helicobacter pylori: From Bench to Bedside." Canadian Journal of Gastroenterology 11, no. 7 (1997): 589–96. http://dx.doi.org/10.1155/1997/975469.

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With the exponential increase in research in the field ofHelicobacter pyloria paradigm shift has occurred. It is now recognized thatH pyloriis a chronic infection of the stomach causing inflammation. Some patients remain asymptomatic, while others may develop dyspepsia, duodenal or gastric ulcer, gastric cancer or a mucosa-associated lymphoid tissue lymphoma. However, the role ofH pyloriin contributing to nonulcer dyspepsia or nonsteroidal anti-inflammatory drug gastropathy remains controversial. An effective vaccine againstH pyloriis years away. Major interest has focused on the questions "wh
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Sakaguchi, Takuki, Takaaki Sugihara, Ken Ohnita, et al. "Pyloric Incompetence Associated with Helicobactor pylori Infection and Correlated to the Severity of Atrophic Gastritis." Diagnostics 12, no. 3 (2022): 572. http://dx.doi.org/10.3390/diagnostics12030572.

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Duodenogastric reflux (DGR) causes bile reflux gastritis (BRG) and may develop into gastric cancer. DGR is classified as primary in non-operated stomachs or secondary to surgical intervention. Primary DGR and Helicobacter pylori (H. pylori) infection are reportedly related. However, the mechanism is not fully understood. This study aimed to elucidate the relationship between H. pylori infection and pyloric incompetence in a non-operated stomach. A total of 502 non-operated participants who underwent an upper intestinal endoscopy were prospectively enrolled. Endoscopic findings (EAC, endoscopic
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Assumpção, Paulo Pimentel de, Robert M. Genta, Maria Constanza Camargo, Jessica Manoelli Costa da Silva, Manuel Ricardo Amieva, and Massimo Rugge. "The Landscape of Helicobacter pylori-related Gastric Carcinogenesis." Journal of Gastrointestinal and Liver Diseases 33, no. 4 (2024): 524–34. https://doi.org/10.15403/jgld-5959.

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The relationship between Helicobacter pylori (H. pylori) and humans remains a complex enigma. While other factors contribute to gastric cancer (GC), their impact pales in comparison to the central role of H. pylori. Various cofactors, such as dietary carcinogens and Epstein-Barr virus infection, can lead to GC independently of H. pylori. However, it is likely the combination of mechanisms, especially those driven by H. pylori, that represents the primary force behind GC development. Identifying individuals at high risk of developing H. pylori-related GC or detecting the disease in its earliest
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Garcés-Duran, R., S. Kindt, K. Kotilea, et al. "Belgian consensus for Helicobacter pylori management 2023." Acta Gastro Enterologica Belgica 86, no. 1 (2023): 74–91. http://dx.doi.org/10.51821/86.1.11327.

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Helicobacter pylori (H. pylori) infection causes chronic gastritis, peptic ulcers and gastric cancer. Although H. pylori prevalence is decreasing worldwide, regional variations exist in Europe, with the lowest infection prevalence in Northern Europe, and the highest in Eastern and Southern Europe (1). Changes in the treatment recommendations and the increasing available evidence have justified the implementation of new recommendations since last Belgian consensus in 1998 (2). Several non-H. pylori Helicobacter species (NH.PYLORI-H), colonizing the stomach of domestic animals, also have the abi
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Chen, Yang. "The overview of Clarithromycin treatment for Helicobacter pylori infection." Theoretical and Natural Science 46, no. 1 (2024): 142–48. http://dx.doi.org/10.54254/2753-8818/46/20240604.

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Helicobacter pylori (H. pylori) is a Gram-negative bacillus that causes infections worldwide. Helicobacter pylori's drug resistance and infectivity are vital factors causing Helicobacter pylori infection. The virulence factors of Helicobacter pylori include acid escape virulence factors and epithelial cell colonizing factors, which can directly or indirectly cause chronic gastritis or gastric ulcer. As the resistance of Helicobacter pylori increases, a combination of multiple antibiotics is now the recommended therapy, and clarithromycin is one of them. The production process of clarithromycin
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Asonuma, Sho, Akira Imatani, Naoki Asano, et al. "Helicobacter pylori induces gastric mucosal intestinal metaplasia through the inhibition of interleukin-4-mediated HMG box protein Sox2 expression." American Journal of Physiology-Gastrointestinal and Liver Physiology 297, no. 2 (2009): G312—G322. http://dx.doi.org/10.1152/ajpgi.00518.2007.

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Helicobacter pylori is a major cause of the transdifferentiation into intestinal metaplasia that may develop gastric cancer. However, the molecular pathogenesis of this transdifferentiation is poorly understood. A SRY-related HMG box protein Sox2 is an essential transcription factor of organ development in brain, lung, and stomach. Our aim of this study was to investigate the mechanism responsible for regulation of Sox2 in host Th1-dominant response to H. pylori. Sox2 protein was immunohistochemically expressed in both human oxyntic and pyloric glands with H. pylori infection, but not in intes
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Takatsuna, Masafumi, Rie Azumi, Takeshi Mizusawa, et al. "A case of Helicobacter pylori-negative early gastric adenocarcinoma with gastrointestinal phenotype." Endoscopy International Open 09, no. 06 (2021): E863—E866. http://dx.doi.org/10.1055/a-1396-3854.

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AbstractA 40-year-old man with slightly depressed (0-IIc) type gastric cancer of the pyloric anterior gastric area underwent pre-operative screening for tetralogy of Fallot and endoscopic submucosal dissection (ESD) and was tested for Helicobacter pylori antigens and antibodies. Both tests were negative. He did not have a history of eradication. Pathological diagnosis of ESD showed a well-differentiated adenocarcinoma. The tumor was CD10-positive, MUC5AC-negative, and MUC6-confocal positive; it showed differentiation with gastrointestinal phenotype. Moreover, the tumor cells were lysozyme-posi
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Saler, Tayyibe, Şakir Özgür Keşkek, Sibel Kırk, Süleyman Ahbab, and Gülay Ortoğlu. "H. pyloriMay Not Be Associated with Iron Deficiency Anemia in Patients with Normal Gastrointestinal Tract Endoscopy Results." Advances in Hematology 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/375915.

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Background. The aim of this study was to investigate the association between iron deficiency anemia andH. pyloriin patients with normal gastrointestinal tract endoscopy results.Materials and Methods. A total of 117 male patients with normal gastrointestinal tract endoscopy results were included in this retrospective study. The study and control groups included 69 and 48 patients with and without iron deficiency anemia, respectively. The prevalence ofH. pylori, the number of RBCs, and the levels of HGB, HTC, MCV, iron, and ferritin were calculated and compared.Results. There was no statisticall
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Kosai, Nik Ritza, Hardip Singh Gendeh, Abdul Rashid Norfaezan, Jamin Razman, Paul Anthony Sutton, and Srijit Das. "Prolapsing Gastric Polyp Causing Intermittent Gastric Outlet Obstruction." International Surgery 100, no. 6 (2015): 1148–52. http://dx.doi.org/10.9738/intsurg-d-14-00205.1.

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Gastric polyps are often an incidental finding on upper gastrointestinal endoscopy, with an incidence up to 5%. The majority of gastric polyps are asymptomatic, occurring secondary to inflammation. Prior reviews discussed Helicobacter pylori (H pylori)–associated singular gastric polyposis; however, we present a rare and unusual case of recurrent multiple benign gastric polyposis post H pylori eradication resulting in intermittent gastric outlet obstruction. A 70-year-old independent male, Chinese in ethnicity, with a background of diabetes mellitus, hypertension, and a simple renal cyst prese
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Öztekin, Merve, Birsen Yılmaz, Duygu Ağagündüz, and Raffaele Capasso. "Overview of Helicobacter pylori Infection: Clinical Features, Treatment, and Nutritional Aspects." Diseases 9, no. 4 (2021): 66. http://dx.doi.org/10.3390/diseases9040066.

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Helicobacter pylori (H. pylori) is a 0.5–1 µm wide, 2–4 µm long, short helical, S-shaped Gram-negative microorganism. It is mostly found in the pyloric region of the stomach and causes chronic gastric infection. It is estimated that these bacteria infect more than half of the world’s population. The mode of transmission and infection of H. pylori is still not known exactly, but the faecal–oral and oral–oral routes via water or food consumption are thought to be a very common cause. In the last three decades, research interest has increased regarding the pathogenicity, microbial activity, genet
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Rozprawy doktorskie na temat "Pylori"

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Ferreira, Pedro Manuel Negreiro de Moura. "Helicobacter pylori." Master's thesis, Universidade da Beira Interior, 2008. http://hdl.handle.net/10400.6/801.

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O Helicobacter pylori (HP) constitui uma entidade fundamental quando nos reportamos à patologia gastro-duodenal. As indicações para a erradicação desta bactéria constituem, actualmente, uma das grandes controvérsias na área da Gastroenterologia. Com o objectivo principal de estabelecer o actual ponto de situação no que diz respeito às indicações para a erradicação do HP, realizámos uma revisão sistemática aprofundada do tema com base em artigos científicos de boa qualidade metodológica. Existem duas indicações inequívocas para efectuar o tratamento de erradicação do HP: a doença péptic
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Illingworth, David Simon. "Studies on Helicobacter pylori." Thesis, University of Reading, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.333335.

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Salamina, M. "Helicobacter pylori Pathogenic Factors." Doctoral thesis, Università degli studi di Padova, 2014. http://hdl.handle.net/11577/3423803.

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From 1994, Helicobacter pylori was classified by WHO (World Health Organization) as a class I carcinogen and its infection has been associated to gastroduodenal disease. It colonizes more than half of worldwide population, with a prevalent infection rate in developed countries. In spite of the majority of infected people are asymptomatic, around 20% develop severe pathologies like peptic ulcers and the 1% lymphoma of the mucosa-associated lymphoid tissue (MALT) and stomach cancer. This significant epidemiological study both of the unique characteristics of H. pylori inspired many scientists, a
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Coelho, Filipa Maria Meireles da Cunha. "Helicobacter pylori : eficácia da terapêutica." Master's thesis, Universidade da Beira Interior, 2013. http://hdl.handle.net/10400.6/1412.

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Introdução: O Helicobacter pylori adquiriu grande importância durante as últimas décadas, ao ser reconhecido como um importante patogénio que infeta uma grande parte da população humana. Esta bactéria, localizada na mucosa gástrica, apresenta uma relação causal com algumas patologias, tais como: carcinoma gástrico, úlcera péptica, entre outras. A sua erradicação representa um grande desafio para os clínicos. O aumento gradual da falha do tratamento obriga a utilizar novos esquemas terapêuticos e a procurar novas soluções. As taxas de eficácia de erradicação do Helicobacter pylori têm vindo a d
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Loos, Chantal. ""Campylobacter pylori" : apport du laboratoire de bactériologie dans une étude multidisciplinaire." Paris 5, 1989. http://www.theses.fr/1989PA05P015.

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Garrett, June Kazumi. "Inhibition of Helicobacter pylori by Wild Blueberry Phenolics." Fogler Library, University of Maine, 2009. http://www.library.umaine.edu/theses/pdf/GarrettJK2009.pdf.

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Queralt, i. Díaz Núria. "Detecció d'Helicobacter pylori en aigua." Doctoral thesis, Universitat de Barcelona, 2013. http://hdl.handle.net/10803/104263.

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Aquesta Tesi Doctoral té com objectius principals l’estudi d’Helicobacter pylori en mostres aquàtiques de Catalunya, en aigües procedents de sistemes dentals de consultes de dentistes, en saliva i en femtes de pacients amb símptomes gastrointestinals mitjançant el mètode de la PCR. També es va estudiar la supervivència d’Helicobacter pylori en aigua dolça usant un model de laboratori aplicant diferents tècniques d’anàlisi i es va interpretar el canvi de morfologia, la viabilitat i la culturabilitat de la bactèria així com la detecció i quantificació del seu ADN durant un període de temps concr
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Goto, Hidemi. "Helicobacter pylori and gastric diseases." Nagoya University School of Medicine, 2003. http://hdl.handle.net/2237/5386.

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Phillips, Rosemary Helen. "Metal ions and Helicobacter pylori." Thesis, King's College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.408125.

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Kivi, Mårten. "Aspects of Helicobacter pylori transmission /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-422-8/.

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Książki na temat "Pylori"

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Mobley, Harry L. T., George L. Mendz, and Stuart L. Hazell, eds. Helicobacter pylori. ASM Press, 2001. http://dx.doi.org/10.1128/9781555818005.

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Smith, Sinead M., ed. Helicobacter Pylori. Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1302-3.

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Kim, Nayoung, ed. Helicobacter pylori. Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-287-706-2.

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Hunt, Richard H., and Guido N. J. Tytgat, eds. Helicobacter pylori. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1418-9.

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Suzuki, Hidekazu, Robin Warren, and Barry Marshall, eds. Helicobacter pylori. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-55705-0.

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Menge, H., M. Gregor, G. N. J. Tytgat, and B. J. Marshall, eds. Campylobacter pylori. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-83322-9.

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Hunt, Richard H., and Guido N. J. Tytgat, eds. Helicobactor pylori. Springer Netherlands, 2003. http://dx.doi.org/10.1007/978-94-017-1763-2.

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Hunt, Richard H., and Guido N. J. Tytgat, eds. Helicobacter pylori. Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1792-7.

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Hunt, Richard H., and Guido N. J. Tytgat, eds. Helicobacter pylori. Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-011-4882-5.

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Hunt, Richard H., and Guido N. J. Tytgat, eds. Helicobacter pylori. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-3927-4.

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Części książek na temat "Pylori"

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Takafuta, Toshiro, and Kingo Fujimura. "Helicobacter pylori (H. pylori) Eradication." In Autoimmune Thrombocytopenia. Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-4142-6_12.

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Marshall, Barry J., Helen M. Windsor, and Kazufumi Kimura. "Helicobacter pylori." In Practical Gastroenterology and Hepatology: Esophagus and Stomach. Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444327311.ch44.

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Smith, Karen L., and Julie Parsonnet. "Helicobacter pylori." In Bacterial Infections of Humans. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5327-4_18.

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Coia, John, and Heather Cubie. "Helicobacter pylori." In The Immunoassay Kit Directory. Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-009-0359-3_19.

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Bateson, Malcolm C., and Ian A. D. Bouchier. "Helicobacter pylori." In Clinical Investigations in Gastroenterology. Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5630-1_1.

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Welcome, Menizibeya Osain. "Helicobacter Pylori." In Gastrointestinal Physiology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91056-7_14.

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Crowell, Trevor A. "Helicobacter pylori." In Encyclopedia of Immigrant Health. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-5659-0_356.

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Kawakubo, Masatomo, Yuki Ito, Minoru Fukuda, and Jun Nakayama. "Helicobacter pylori." In Glycoscience: Biology and Medicine. Springer Japan, 2014. http://dx.doi.org/10.1007/978-4-431-54836-2_145-1.

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Kawakubo, Masatomo, Yuki Ito, Minoru Fukuda, and Jun Nakayama. "Helicobacter pylori." In Glycoscience: Biology and Medicine. Springer Japan, 2014. http://dx.doi.org/10.1007/978-4-431-54841-6_145.

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Bateson, Malcolm C., and Ian A. D. Bouchier. "Helicobacter pylori." In Clinical Investigations in Gastroenterology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53786-3_1.

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Streszczenia konferencji na temat "Pylori"

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Khanna, Palak, Shanvi, and Debendra Kumar Dhir. "Solutions to Detect Helicobacter Pylori Using AI." In 2024 6th International Conference on Computational Intelligence and Networks (CINE). IEEE, 2024. https://doi.org/10.1109/cine63708.2024.10881029.

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Rangel, Gabriela Flores, Lorena Diaz de León Martinez, and Boris Mizaikoff. "Non-Invasive Helicobacter Pylori Detection via Mid-Infrared iHWG Sensor." In Latin America Optics and Photonics Conference. Optica Publishing Group, 2024. https://doi.org/10.1364/laop.2024.tu4a.31.

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Helicobacter pylori infection is linked to gastrointestinal disorders, including gastric cancer, necessitating efficient detection technologies. This study evaluates substrate-integrated hollow waveguide (iHWG) sensors using mid-infrared Fourier-transform infrared (FTIR) spectroscopy for precise isotopic ratio measurement of ¹³CO₂ to ¹²CO₂ in simulated breath analysis conditions. Three iHWGs of varying lengths were tested, optimizing the gas-light interaction to enhance sensitivity. Results show high precision and stability, establishing the FTIR-iHWG system as a promising platform for non-inv
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Popov, E. E., A. V. Polishchuk, A. V. Kovalev, and V. V. Vitkin. "Raman spectroscopy as a tool for helicobacter pylori diagnostics." In 2024 International Conference Laser Optics (ICLO). IEEE, 2024. http://dx.doi.org/10.1109/iclo59702.2024.10624505.

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Coelho, Luiz, and Maria Coelho. "Helicobacter pylori no idoso." In XVIII Semana Brasileira do Aparelho Digestivo. Editora Manole, 2019. http://dx.doi.org/10.22288/978857868372600005.

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Atikov, A. S. "THE ASSOCIATION OF LIVER PATHOLOGIES WITH THE CONTAMINATION OF THE GASTROINTESTINAL TRACT WITH H.PYLORI." In SPbVetScience. FSBEI HE St. Petersburg SUVM, 2023. http://dx.doi.org/10.52419/3006-2023-11-9-16.

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Currently, one of the most promising areas in bacteriology is the study of the bacterium Helicobacter pylori. New studies show a link between H. pylori and some liver diseases, but their exact mechanisms and connections are still not fully understood. Despite this, studies of H. pylori and its association with liver diseases remain relevant to this day. This article presents an analysis of the association of H. Pylori with liver diseases.
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Boraeva, T. T., O. V. Remizov, A. A. Revazova, F. S. Dzebisova, F. V. Bazrova, and A. M. Grigorian. "Helicobacter Pylori Infection in Children." In Proceedings of the International Conference on Health and Well-Being in Modern Society (ICHW 2019). Atlantis Press, 2019. http://dx.doi.org/10.2991/ichw-19.2019.8.

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Çavuş, B., T. Çavuş, B. Akyüz Erdoğan, and E. Kumcu. "ERYTHROCYTE DISTRIBUTION WIDTH (RDW) AND HELICOBACTER PYLORI (H. PYLORI) INFECTION: IS THERE AN ASSOCIATION?" In ESGE Days 2018 accepted abstracts. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1637463.

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Шишкина, Виктория, Светлана Клочкова, Наталия Алексеева та ін. "Морфофункциональные особенности тучных клеток при инфицировании Helicobacter pylori". У Международная научная и методическая конференция, посвященная году фундаментальных наук: "Современные аспекты морфологии, патоморфологии и онкопатологии организма человека". ФГБОУ ВО КГМУ Минздрава России, 2022. http://dx.doi.org/10.21626/cb.22.humanmorphology/32.

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Одними из ключевых промоторов в регуляции каскада воспалительных медиаторов являются тучные клетки, обладающие широким арсеналом биологически активных веществ. В патогенезе гастрита, ассоциированного с H. pylori принимают активное участие различные виды иммунокомпетентных клеток - макрофаги, нейтрофилы, эозинофилы, дендритные клетки, Т- и В-лимфоциты, а также тучные клетки. Антигенпредставляющие свойства тучных клеток представляют интерес в аспекте взаимодействия с Helicobacter pylori (H. Pylori)
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Абрамян, Мария Владимировна, Алина Викторовна Свиридова, and Елена Николаевна Веселова. "HELICOBACTER PYLORI INFECTION AND ITS ROLE IN THE PATHOGENESIS OF NONALCOHOLIC FATTY LIVER DISEASE." In Наука. Исследования. Практика: сборник избранных статей по материалам Международной научной конференции (Санкт-Петербург, Февраль 2021). Crossref, 2021. http://dx.doi.org/10.37539/srp295.2021.30.76.006.

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Велика ли роль инфекции Helicobacter pylori на патогенез неалкогольной жировой болезни печени (НАЖБП)? Последние данные свидетельствуют о наличии их взаимосвязи [1, 2]. Однако до сих пор данная тема остается под вопросом. Цель данной работы - исследовать возможную связь и клиническое значение Helicobacter pylori в патогенезе неалкогольной жировой болезни печени. Is the role of Helicobacter pylori infection on the pathogenesis of non-alcoholic fatty liver disease (NAFLD) significant? Recent data indicate the presence of their relationship [1, 2]. However, until now, this topic remains in questi
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Kittner, JM, L. Volksdorf, CC Adarkwah, and M. Menges. "H. pylori-Eradikation: Versorgungsrealität in Deutschland." In Viszeralmedizin 2021 Gemeinsame Jahrestagung Deutsche Gesellschaft für Gastroenterologie, Verdauungs- und Stoffwechselkrankheiten (DGVS), Sektion Endoskopie der DGVS, Deutsche Gesellschaft für Allgemein und Viszeralchirurgie (DGAV). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1733507.

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Raporty organizacyjne na temat "Pylori"

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López-Valverde, Nansi, Antonio López-Valverde, Ana Suarez, Bruno Macedo de Sousa, and Juan Manuel Aragoneses. Association of gastric infection and periodontal disease through Helicobacter pylori as a common denominator: A systematic review and meta-analysi. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.10.0097.

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Review question / Objective: Is gastric helicobacter pylori infection related to periodontal diseases? Condition being studied: Therefore, the aim of this systematic review and meta-analysis was to identify and analyze clinical studies to determine the direct correlation between Helicobacter Pylori gastric infection andPeriodontal Disease. Study designs to be included: Clinical studies that provided data on Helicobacter Pylori infection in both the stomach and oral cavity, confirmed by polymerase chain reaction (PCR), rapid urease test (RUT) or enzyme-linked immunosorbent assay (ELISA). Clinic
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Boyanova, Lyudmila, Radka Koumanova, Petyo Hadzhiyski, et al. Helicobacter pylori Infection in Bulgarian Pediatric Patients. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2018. http://dx.doi.org/10.7546/crabs.2018.07.14.

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Gilbreath, Jeremy J. Novel Insights into Fur Regulation in Helicobacter pylori. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ad1012921.

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Jones, Kathy R. Heliobactor pylori Virulence Factors and Their Role in Pathogenesis. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ad1013348.

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Yordanov, Daniel, Lyudmila Boyanova, Rumyana Markovska, Petyo Hadzhiyski, Galina Gergova, and Ivan Mitov. Seroprevalence of Helicobacter pylori IgG and CagA IgG in Bulgarian Children. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2018. http://dx.doi.org/10.7546/crabs.2018.08.15.

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Tang, Jia, guangming Tang, and weiqiang Wang. Association of Coffee Consumption and Helicobacter pylori Infection A Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.7.0095.

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Carpenter, Beth M. Structure Function Analysis of the Ferric Uptake Regulator (Fur) of Helicobacter pylori. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ad1013395.

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Cai, Zhaolun. Tailored Therapy for Helicobacter Pylori Eradication: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.3.0166.

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Gao, Ying, Xiao-jing Yang, Yun Zhu, Ming Yang, and Fei Gu. A systematic review and meta-analysis of rosacea and helicobacter pylori infection. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.11.0011.

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LI, YINGHAO, and HS SHI. Helicobacter pylori infection and atherosclerotic disease progression: a systematic review and meta‑analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.10.0032.

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