Gotowe bibliografie tematyczne / Receptor cross-interference

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Gotowa bibliografia na temat „Receptor cross-interference”

Autor: Grafiati
Data publikacji: 2 czerwca 2025
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Receptor cross-interference"

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Dondi, Elisabetta, Els Pattyn, Georges Lutfalla, et al. "Down-modulation of Type 1 Interferon Responses by Receptor Cross-competition for a Shared Jak Kinase." Journal of Biological Chemistry 276, no. 50 (2001): 47004–12. http://dx.doi.org/10.1074/jbc.m104316200.

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In contrast to the large number of class I and II cytokine receptors, only four Janus kinase (Jak) proteins are expressed in mammalian cells, implying the shared use of these kinases by many different receptor complexes. Consequently, if receptor numbers exceed the amount of available Jak, cross-interference patterns can be expected. We have engineered two model cellular systems expressing two different exogenous Tyk2-interacting receptors. A receptor chimera was generated wherein the extracellular part of the interferon type 1 receptor (Ifnar1) component of the interferon-α/β receptor is repl
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Kutoh, E., P. E. Strömstedt, and L. Poellinger. "Functional interference between the ubiquitous and constitutive octamer transcription factor 1 (OTF-1) and the glucocorticoid receptor by direct protein-protein interaction involving the homeo subdomain of OTF-1." Molecular and Cellular Biology 12, no. 11 (1992): 4960–69. http://dx.doi.org/10.1128/mcb.12.11.4960-4969.1992.

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The ubiquitous and constitutive octamer transcription factor OTF-1 (Oct 1) is the target of positive regulation by the potent herpes simplex virus trans-activator VP16, which forms a complex with the homeodomain of OTF-1. Here we present evidence that the glucocorticoid receptor can negatively regulate OTF-1 function by a mechanism that is independent of DNA binding. In vivo-expressed glucocorticoid receptor inhibited in a hormone-dependent manner activation of a minimal promoter construct carrying a functional octamer site. Moreover, expression of the receptor in vivo resulted in hormone-depe
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Kutoh, E., P. E. Strömstedt, and L. Poellinger. "Functional interference between the ubiquitous and constitutive octamer transcription factor 1 (OTF-1) and the glucocorticoid receptor by direct protein-protein interaction involving the homeo subdomain of OTF-1." Molecular and Cellular Biology 12, no. 11 (1992): 4960–69. http://dx.doi.org/10.1128/mcb.12.11.4960.

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The ubiquitous and constitutive octamer transcription factor OTF-1 (Oct 1) is the target of positive regulation by the potent herpes simplex virus trans-activator VP16, which forms a complex with the homeodomain of OTF-1. Here we present evidence that the glucocorticoid receptor can negatively regulate OTF-1 function by a mechanism that is independent of DNA binding. In vivo-expressed glucocorticoid receptor inhibited in a hormone-dependent manner activation of a minimal promoter construct carrying a functional octamer site. Moreover, expression of the receptor in vivo resulted in hormone-depe
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De Coster, Sam, and Nicolas van Larebeke. "Endocrine-Disrupting Chemicals: Associated Disorders and Mechanisms of Action." Journal of Environmental and Public Health 2012 (2012): 1–52. http://dx.doi.org/10.1155/2012/713696.

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The incidence and/or prevalence of health problems associated with endocrine-disruption have increased. Many chemicals have endocrine-disrupting properties, including bisphenol A, some organochlorines, polybrominated flame retardants, perfluorinated substances, alkylphenols, phthalates, pesticides, polycyclic aromatic hydrocarbons, alkylphenols, solvents, and some household products including some cleaning products, air fresheners, hair dyes, cosmetics, and sunscreens. Even some metals were shown to have endocrine-disrupting properties. Many observations suggesting that endocrine disruptors do
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Vegna, Serena, Damien Gregoire, Marie Moreau, et al. "NOD1 Participates in the Innate Immune Response Triggered by Hepatitis C Virus Polymerase." Journal of Virology 90, no. 13 (2016): 6022–35. http://dx.doi.org/10.1128/jvi.03230-15.

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ABSTRACTHepatitis C virus (HCV) triggers innate immunity signaling in the infected cell. Replication of the viral genome is dispensable for this phenotype, and we along with others have recently shown that NS5B, the viral RNA-dependent RNA polymerase, synthesizes double-stranded RNA (dsRNA) from cellular templates, thus eliciting an inflammatory response, notably via activation of type I interferon and lymphotoxin β. Here, we investigated intracellular signal transduction pathways involved in this process. Using HepaRG cells, a model that largely recapitulates thein vivocomplexities of the inn
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Leis, Hugo, Angustias Page, Angel Ramírez, et al. "Glucocorticoid Receptor Counteracts Tumorigenic Activity of Akt in Skin through Interference with the Phosphatidylinositol 3-Kinase Signaling Pathway." Molecular Endocrinology 18, no. 2 (2004): 303–11. http://dx.doi.org/10.1210/me.2003-0350.

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Abstract The skin-targeted overexpression of the glucocorticoid receptor (GR) in transgenic mice dramatically impairs the inflammatory responses to tumor promoter agents and suppresses skin tumor development. The antiinflammatory, rapid effects of corticosteroids are partially exerted through interference of GR with the phosphatidylinositol 3-kinase (PI3K)/Akt signaling pathway in several tissues, a highly relevant pathway in the mouse skin tumor progression process. In this work, we aimed to elucidate whether a cross-talk mechanism between GR and PI3K/Akt occurred in intact skin as well as th
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Boleij, Annemarie, Coby M. Laarakkers, Jolein Gloerich, Dorine W. Swinkels, and Harold Tjalsma. "Surface-Affinity Profiling To Identify Host-Pathogen Interactions." Infection and Immunity 79, no. 12 (2011): 4777–83. http://dx.doi.org/10.1128/iai.05572-11.

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ABSTRACTProteolytic treatment of intact bacterial cells has proven to be a convenient approach for the identification of surface-exposed proteins. This class of proteins directly interacts with the outside world, for instance, during adherence to human epithelial cells. Here, we aimed to identify host receptor proteins by introducing a preincubation step in which bacterial cells were first allowed to capture human proteins from epithelial cell lysates. UsingStreptococcus gallolyticusas a model bacterium, liquid chromatography-tandem mass spectrometry (LC-MS/MS) analysis of proteolytically rele
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Matthews, Jason, Björn Wihlén, Jane Thomsen та Jan-Åke Gustafsson. "Aryl Hydrocarbon Receptor-Mediated Transcription: Ligand-Dependent Recruitment of Estrogen Receptor α to 2,3,7,8-Tetrachlorodibenzo- p-Dioxin-Responsive Promoters". Molecular and Cellular Biology 25, № 13 (2005): 5317–28. http://dx.doi.org/10.1128/mcb.25.13.5317-5328.2005.

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ABSTRACT Using chromatin immunoprecipitation assays, we studied the 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-mediated recruitment of the aryl hydrocarbon receptor (AhR) and several coregulators to the CYP1A1 promoter. AhR displayed a time-dependent recruitment, reaching a peak at 75 min and maintaining promoter occupancy for the remainder of the time course. Recruitment of AhR was followed by TIF2/SRC2, which preceded CBP, histone H3 acetylation, and RNA polymerase II (RNAPII). Simultaneous recruitment to the enhancer and the TATA box region suggests the formation of a large multiprotein com
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Albee, Lauren J., Heather M. LaPorte, Xianlong Gao, et al. "Identification and functional characterization of arginine vasopressin receptor 1A : atypical chemokine receptor 3 heteromers in vascular smooth muscle." Open Biology 8, no. 1 (2018): 170207. http://dx.doi.org/10.1098/rsob.170207.

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Recent observations suggest that atypical chemokine receptor (ACKR)3 and chemokine (C-X-C motif) receptor (CXCR)4 regulate human vascular smooth muscle function through hetero-oligomerization with α 1 -adrenoceptors. Here, we show that ACKR3 also regulates arginine vasopressin receptor (AVPR)1A. We observed that ACKR3 agonists inhibit arginine vasopressin (aVP)-induced inositol trisphosphate (IP 3 ) production in human vascular smooth muscle cells (hVSMCs) and antagonize aVP-mediated constriction of isolated arteries. Proximity ligation assays, co-immunoprecipitation and bioluminescence resona
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Lin, Yuting, John Kokontis, Fangming Tang, et al. "Androgen and Its Receptor Promote Bax-Mediated Apoptosis." Molecular and Cellular Biology 26, no. 5 (2006): 1908–16. http://dx.doi.org/10.1128/mcb.26.5.1908-1916.2006.

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ABSTRACT Androgen and its receptor (AR) have been reported to have pro- or antiapoptotic functions. However, the underlying molecular mechanism is incompletely understood. We report here that androgen and AR promote Bax-mediated apoptosis in prostate cancer cells. UV irradiation and ectopic expression of Bax induce apoptosis in AR-positive, but not AR-negative prostate cancer cells. UV- and Bax-induced apoptosis is abrogated in AR-positive cells that express small interference RNA (siRNA) of AR and is sensitized by reintroduction of AR into AR-negative cells. Although AR is able to promote Bax
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Części książek na temat "Receptor cross-interference"

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Jarausch, Konrad H. "Running Away." In The Rush to German Unity. Oxford University PressNew York, NY, 1994. http://dx.doi.org/10.1093/oso/9780195072754.003.0002.

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Abstract A hole in the Iron Curtain started the mass exodus. When Hungarian soldiers cut the barbed wire fence on May 2, 1989, desperate East Germans began to cross the Austrian frontier. Those who were turned back or were less daring crowded into West German embassies in Budapest, Prague, and East Berlin. Though the GDR protested against “crude interference in [its] sovereign affairs,” Bonn welcomed the refugees from poverty and repression: “When they come to us, we accept them as Germans. We will not throw them out by force.”1 Sure of a warm reception, more and more East Germans sought to le
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Streszczenia konferencji na temat "Receptor cross-interference"

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Liu, Wei, Yu Xia, Ming Xu, Jian Xie, Rong Luo, and Daqing Huang. "Distributed and Accurate Packet Reception Rate Estimation under Cross-Technology Interference." In GLOBECOM 2020 - 2020 IEEE Global Communications Conference. IEEE, 2020. http://dx.doi.org/10.1109/globecom42002.2020.9348010.

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