Gotowe bibliografie tematyczne / Sensorimotor polyneuropathy

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Gotowa bibliografia na temat „Sensorimotor polyneuropathy”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Sensorimotor polyneuropathy"

Sedlić, Marija, Gordana Sičaja, Krešimir Luetić, and Hrvoje Budinčević. "Cryoglobulinemia and sensorimotor polyneuropathy." Infektološki glasnik 40, no. 1 (2020): 35–37. http://dx.doi.org/10.37797/ig.40.1.5.

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Secondary cryoglobulinemia is the most common extrahepatic manifestation of hepatitis C. There are different genotypes of hepatitis C, and the ones that are most frequently associated with cryoglobulinemia are genotypes 1b and 2a. Cryoglobulinemia affects various organs and can cause vasculitis, arthralgia, skin changes, glomerulonephritis and neurological manifestations, including peripheral neuropathy. The aim of this report was to emphasize the importance of early diagnosis of hepatitis C infection in patients who are presenting with sensomotor polyneuropathy.

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Gorczyca, Daiva, and Ulrike Schwirten. "Sensorimotor polyneuropathy after hexavalent vaccination." Scandinavian Journal of Infectious Diseases 46, no. 1 (2013): 66–68. http://dx.doi.org/10.3109/00365548.2013.831183.

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Nardone, Raffaele, Thomas Buratti, Stefan Golaszewski, et al. "Delayed Oxaliplatin-Induced Sensorimotor Polyneuropathy." Onkologie 32, no. 5 (2009): 10. http://dx.doi.org/10.1159/000209321.

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Ocampo-Navia, Maria Isabel, María Andrea Negret Noreña, Luis Rafael Chaparro Santos, José Ricardo Morera Afanador, Ricardo Andrés Quintero Farías, and José Luis Bustos Sánchez. "POEMS Syndrome Diagnosis in a Patient with Mixed Polyneuropathy: Case Report." Prague Medical Report 123, no. 1 (2022): 27–34. http://dx.doi.org/10.14712/23362936.2022.3.

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POEMS syndrome is a rare condition of paraneoplasic origin characterized by the presence of a sensorimotor polyneuropathy associated with the presence of a proliferative disorder of plasmatic monoclonal cells and overproduction of vascular endothelial growth factor. The acronym “POEMS” represents multisystem findings including polyneuropathy, organomegaly, endocrinopathy, monoclonal plasma cell disorder and skin changes; nevertheless, clinical presentation is heterogeneous. We describe a clinical case, the diagnostic and therapeutic approach in a patient with sensorimotor polyneuropathy in who

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Jastrzębski, Karol, Nina Romanowska, Marta Rek-Pacześ, et al. "Stiff-person syndrome with sensorimotor polyneuropathy – case report." Aktualności Neurologiczne 21, no. 1 (2021): 47–55. http://dx.doi.org/10.15557/an.2021.0007.

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Stiff-person syndrome (SPS) is a rare disorder with an estimated prevalence in the general population of 1–2 cases/1,000,000. It is 2–3 times more common in females, with symptom onset at the age of 20–50 years in most cases. Although stiff-person syndrome is associated with antibodies against glutamic acid decarboxylase and amphiphysin, their presence is not necessary for the diagnosis. The treatment should be multidirectional and include immunomodulation, symptomatic treatment as well as monitoring and treatment of overlapping autoimmune, and surgery. We present a case report of a patient di

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Puri, Vinod, AshishKumar Duggal, and Neera Chaudhry. "Idiopathic CD4 lymphocytopenia with sensorimotor polyneuropathy." Annals of Indian Academy of Neurology 19, no. 3 (2016): 381. http://dx.doi.org/10.4103/0972-2327.165470.

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Dunnigan, S. K., H. Ebadi, A. Breiner, et al. "Conduction Slowing in Diabetic Sensorimotor Polyneuropathy." Diabetes Care 36, no. 11 (2013): 3684–90. http://dx.doi.org/10.2337/dc13-0746.

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A. Kles, Keri, and Vera Bril. "Diagnostic Tools for Diabetic Sensorimotor Polyneuropathy." Current Diabetes Reviews 2, no. 3 (2006): 353–61. http://dx.doi.org/10.2174/157339906777950598.

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Cornblath, D. R., A. Vinik, E. Feldman, R. Freeman, and A. J. M. Boulton. "Surgical Decompression for Diabetic Sensorimotor Polyneuropathy." Diabetes Care 30, no. 2 (2007): 421–22. http://dx.doi.org/10.2337/dc06-2324.

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Choi, Jong Kyoung, Won Jun Kim, and Jae Yong Jeon. "Paraneoplastic sensorimotor polyneuropathy in prostatic adenocarcinoma." Medicine 97, no. 15 (2018): e0030. http://dx.doi.org/10.1097/md.0000000000010030.

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Książki na temat "Sensorimotor polyneuropathy"

Katirji, Bashar. Case 26. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0030.

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Peripheral polyneuropathy is a common clinical presentation, and most cases seen in clinical practice are axonal, dying-back, sensorimotor polyneuropathies. Axonal polyneuropathies should be distinguished from the acquired demyelinating peripheral polyneuropathies which are often immune-mediated and amenable to treatment. This case presents a typical patient with a dying-back sensorimotor axonal peripheral polyneuropathy and presents a practical approach to the etiologic diagnoses of peripheral neuropathy. It then highlights its distinguishing features on nerve conduction studies. A comparison

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Pitt, Matthew. Pathophysiological correlations in neuropathies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198754596.003.0004.

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This chapter begins with an explanation of the pathophysiological correlations between the recorded changes and the underlying diagnosis which allow classification into demyelinating and axonal neuropathy. Demyelinating neuropathies are discussed first. The extensive and ever expanding literature in hereditary neuropathies is highlighted. The different variants of the acute inflammatory demyelinating polyneuropathy encountered in children are discussed along with the electrodiagnostic criteria for the diagnosis. Chronic inflammatory demyelinating polyneuropathy is then covered, both in its cli

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Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 45-Year-Old Male with Toxin Exposure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0004.

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A patient presents with a sensorimotor neuropathy and believes he has been poisoned. The approach to the differential diagnosis of arsenic toxicity is presented. Comparisons with mimics of this entity are made, and clinical clues to its early detection are provided. There are typical skin and nail changes with can occur with arsenic poisoning. Arsenic poisoning can appear similar to Guillain-Barre syndrome with gastrointestinal symptoms and later an ascending paralysis. Urine arsenic levels are more reliable than blood levels. Hair and nail samples are very useful in confirming the diagnosis.

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Części książek na temat "Sensorimotor polyneuropathy"

"Diabetic Sensorimotor Polyneuropathy." In Encyclopedia of Pain. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_100583.

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Mahajan, Shalini, and Daniel J. Wallace. "Management of Peripheral Nervous System Complications in Sjögren’s." In The Sjögren's Book. Oxford University Press, 2022. http://dx.doi.org/10.1093/oso/9780197502112.003.0029.

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Sjögren’s disease can affect the central, peripheral, and autonomic nervous systems. Peripheral neuropathy, which is more common than central nervous system complications, may include sensory polyneuropathy, sensorimotor polyneuropathy, small fiber neuropathy, sensory ganglionopathy, or cranial neuropathies. Autonomic dysfunction may be characterized by feelings of lightheadedness, dizziness, changes in blood pressure, sweating abnormalities, and more. When managing these conditions, a three-pronged approach may be considered that includes treating the underlying pathophysiology and providing

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Marra, Christina M. "Peripheral Neuropathy." In The HIV Manual. Oxford University PressNew York, NY, 1996. http://dx.doi.org/10.1093/oso/9780195100365.003.0057.

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Abstract Peripheral neuropathy describes the clinical syndrome of weakness, sensory loss, diminished tendon reflexes, or some combination of these findings caused by lesions of peripheral nerves. It may symmetrically involve the distal extremities, usually lower extremity greater than upper extremity (distal sensory, motor, or sensorimotor polyneuropathy), or may involve one or more named nerves (mononeuritis or mononeuritis multiplex). Peripheral neuropathy can also involve nerve roots (polyradiculopathy). From a pathophysiologic perspective, peripheral neuropathies are commonly divided into

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Pina-Escudero, Stefanie. "Uremic Neuropathy." In Pain. Oxford University Press, 2022. http://dx.doi.org/10.1093/med/9780197542873.003.0034.

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Uremic neuropathy is a distal symmetric sensorimotor polyneuropathy that presents in patients with chronic kidney disease as a consequence of severe axonal degeneration in distal nerve trunks with secondary segmental demyelination. Nerve conduction velocity tests show a reduction of motor and sensory-motor unit potential amplitudes. Regarding treatment, in patients who are not receiving dialytic therapy, conservative management of the kidney disease might have a positive impact on uremic neuropathy. Physical therapy may prevent the loss of muscle power and prevent or improve functional loss ca

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Streszczenia konferencji na temat "Sensorimotor polyneuropathy"

Tessaro, Camila Lorenzini, Renata Dal-Prá Ducci, Cláudia Suemi Kamoi Kay, et al. "Clinical and electrophysiological characteristics of the peripheral polyneuropathies from a single specialized center in the Southern Brazil." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.359.

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Background: The peripheral polyneuropathies have etiological heterogeneity, with more than a hundred known causes. In addition, they have a lack of information related to their epidemiology. Objectives: The aim of this study is to determine the prevalence of each etiology of polyneuropathy in a single specialized center from Southern Brazil and to correlate main clinical manifestations and electrophysiological aspects. Design and setting: Observational cross-sectional study. Neuromuscular disorder center from a tertiary service. Methods: This study comprised individuals with electrodiagnostic

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Nery, Julia Pio Fernandes, Rafael Elian Alvares, Taianne Fiore Schumann, Cintia Alvarenga Pereira Vieira, and Glaucia Lara Resende. "Poems syndrome: a case report." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.677.

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Introduction: Polyneuropathy, organomegaly, endocrinopathy, monoclonal gammopathy and skin changes syndrome (POEMS) is a rare monoclonal disease with multisystemic presentation and therefore challenging diagnosis. The aim is to report a case of a rare hematological disease with unusual characteristic neurological manifestation. Case report: Male, 59-year-old, with a report of anesthesia and paresthesia in the feet that started in 2018. On examination, he presented tactile and painful hypoesthesia in a path innervated by L5 to S1, in addition to hypoactive Achilles tendon reflexes. Electroneuro

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Khalil, Nadia, and Anthony Bradshaw. "Kappa Light Chain Deposition Disease Presenting with Axonal Sensorimotor Polyneuropathy in the Absence of Clinically Significant Renal Pathology (P11-8.012)." In 2023 Annual Meeting Abstracts. Lippincott Williams & Wilkins, 2023. http://dx.doi.org/10.1212/wnl.0000000000201819.

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Oliveira, Victória Nunes, Mellyssa Cota Elias, Heloiza Castilhoni Belique, et al. "Case of aluminium poisoning, with differential diagnosis for amyotrophic lateral sclerosis (als)." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.246.

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Introduction: Essential metals in high quantities can accumulate and reach toxicity. In the CNS, they lead to neurodegeneration, causing movement and cognitive disorders. Case Report: White man, 59 years, sanitation agent, uses PPE irregularly. For 1 year presented progressive speech disorder, paresis, emaciation, dysphonia, tongue atrophy, limbs fasciculations and abnormal reflexes; normal tonus/ sensibility. Bulbar/limb electroneuromyography and serum dosage of metals in contact were requested, revealing muscles denervation, axonal diffuse polyneuropathy with postganglionic injury and elevat

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Morais, Gabriela Lopes de, Manoella Guerra de Albuquerque Bueno, Guilherme Otero Brum, et al. "Anti-hu neuropathy as a paraneoplastic syndrome: the pursuit for the primary site." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.418.

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A 53-year-old man with a history of smoking and recent weight loss presented with progressive weakness and difficulty walking. Physical exam evidenced dysmetria on finger-to-nose test, worsened with eyes closed, pseudoathetosis, absent deep tendon reflexes and a neuropathic pattern of hypoesthesia and hypopalesthesia. He was unable to stand due to the severity of the ataxia. Initial investigation was positive for syphilis, with no history of treatment, and cerebrospinal fluid analysis showed hyperproteinrachia with normal cell count. Despite adequate treatment with penicillin, symptoms continu

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Pinto, Wladimir Bocca Vieira de Rezende, Bruno de Mattos Lombardi Badia, Igor Braga Farias, et al. "Expanding the neurological and imaging phenotype of women with adult-onset X- linked Adrenoleukodystrophy." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.019.

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Background: X-linked Adrenoleukodystrophy (X-ALD) represents a key inherited metabolic disorder in neurological practice, representing an important differential diagnosis in different neurological contexts. Symptomatic female patients have been scarcely studied in large cohorts. Objectives: Evaluation of clinical, laboratory and genetic findings from a Brazilian cohort of women with X-ALD. Methods, design and setting: We performed a retrospective observational study of clinical, biochemical, genetic, neuroimaging and neurophysiological aspects of 10 Brazilian female patients with X-linked Adre

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Moura, Fernanda Maria Gonçalves de Sousa, Amanda Freitas Alves, João Paulo Barile, Sephora Sabrina Cândido de Almeida, Arthur da Veiga Kalil Coelho, and Marcele Schettini de Almeida. "Hallucinations and psychosis in Guillain-Barré syndrome: case report." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.394.

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Introduction: Guillain-Barré syndrome (GBS) is an acute inflammatory peripheral neuropathy that occurs after infection or immunization. Wernicke’s encephalopathy (WE) is caused by thiamine deficiency that classically presents with altered mental status, ataxic gait and ophthalmoplegia. Case report: Male, 64 years old, presented with diarrhea, vomiting and hyporexia. Three weeks later, he developed concomitant acute tetraparesis and cognitive impairment. On examination, he presented with persecutory delusions, hallucinations, tetraparesis with global areflexia. The cerebrospinal fluid analysis

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Raporty organizacyjne na temat "Sensorimotor polyneuropathy"

Lu, Qi, Haili Wang, Weizheng Wang, et al. Efficacy of Electroacupuncture in Painful Diabetic Peripheral Neuropathy: A protocol of systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0040.

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Review question / Objective: The aim of this study is to perform a meta-analysis to evaluate the effectiveness of electroacupuncture in the treatment of painful diabetic peripheral neuropathy (PDNP).And to provide data support for electroacupuncture as an effective means to treat pain of nervous system diseases. Condition being studied: Diabetes mellitus (DM) affects more than six hundred million population worldwide till 2045. The most common form is chronic, distal, and symmetric sensorimotor polyneuropathy, while other uncommon forms include asymmetric or focal neuropathy, such as diabetic

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