Gotowe bibliografie tematyczne / Signaling lipid

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  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
  6. Raporty organizacyjne

Gotowa bibliografia na temat „Signaling lipid”

Autor: Grafiati
Data publikacji: 9 listopada 2024
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Signaling lipid"

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Korbelik, Mladen, and Albert W. Girotti. "Tumor Lipid Signaling Involved in Hyperoxidative Stress Response: Insights for Therapeutic Advances." Journal of Cellular Signaling 6, no. 2 (2025): 39–47. https://doi.org/10.33696/signaling.6.132.

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Most malignantly transformed cells are metabolically rewired to promote their survival and progression, even under conditions that would be unfavorable for normal counterparts. Arguably the most impactful metabolic transformation and recognized cancer hallmark is the reprogrammed lipid metabolism. Lipids are not only primary constituents of cell membranes but essential participants in fundamental cellular functions including cell signaling, protein regulation, energy provision, inflammation, and cell-cell interaction. Engagement of lipids in critical physiological functions in cells is additio
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Kumar, Vikash, and Sidra Khan. "The Intersection of Lipid Signaling and Metabolism in Cancer and Tuberculosis." Journal of Cellular Signaling 6, no. 2 (2025): 71–82. https://doi.org/10.33696/signaling.6.135.

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Lipids are an essential class of complex biomolecules involved in maintaining cellular energy homeostasis, structural organization and signal transduction. Dysregulated lipid signaling and metabolism have increasingly been reported in various pathological settings like diabetes, cardiomyopathy, neurological pathologies, malignancies and infectious diseases. Recent technological advances in metabolomics and lipidomics have shown enormous complexities and functionalities of lipids. The role of lipid metabolism in maintaining cancer heterogeneity and plasticity is an important hallmark in the dis
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Wang, Xuemin. "Lipid signaling." Current Opinion in Plant Biology 7, no. 3 (2004): 329–36. http://dx.doi.org/10.1016/j.pbi.2004.03.012.

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Bickel, Perry E. "Lipid rafts and insulin signaling." American Journal of Physiology-Endocrinology and Metabolism 282, no. 1 (2002): E1—E10. http://dx.doi.org/10.1152/ajpendo.2002.282.1.e1.

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Lipid rafts are domains within the plasma membrane that are enriched in cholesterol and lipids with saturated acyl chains. Specific proteins, including many signaling proteins, segregate into lipid rafts, and this process is important for certain signal transduction events in a variety of cell types. Within the past decade, data have emerged from many laboratories that implicate lipid rafts as critical for proper compartmentalization of insulin signaling in adipocytes. A subset of lipid rafts, caveolae, are coated with membrane proteins of the caveolin family. Direct interactions between resid
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Höglinger, Doris, André Nadler, Per Haberkant, et al. "Trifunctional lipid probes for comprehensive studies of single lipid species in living cells." Proceedings of the National Academy of Sciences 114, no. 7 (2017): 1566–71. http://dx.doi.org/10.1073/pnas.1611096114.

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Lipid-mediated signaling events regulate many cellular processes. Investigations of the complex underlying mechanisms are difficult because several different methods need to be used under varying conditions. Here we introduce multifunctional lipid derivatives to study lipid metabolism, lipid−protein interactions, and intracellular lipid localization with a single tool per target lipid. The probes are equipped with two photoreactive groups to allow photoliberation (uncaging) and photo–cross-linking in a sequential manner, as well as a click-handle for subsequent functionalization. We demonstrat
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Bazan, Nicolas G. "Synaptic lipid signaling." Journal of Lipid Research 44, no. 12 (2003): 2221–33. http://dx.doi.org/10.1194/jlr.r300013-jlr200.

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Irvine, R. "Nuclear Lipid Signaling." Science Signaling 2000, no. 48 (2000): re1. http://dx.doi.org/10.1126/stke.2000.48.re1.

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Irvine, R. F. "Nuclear Lipid Signaling." Science Signaling 2002, no. 150 (2002): re13. http://dx.doi.org/10.1126/stke.2002.150.re13.

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Junkins, Sadie, Gabrielle Westenberger, Jacob Sellers, et al. "MKP-2 Deficiency Leads to Lipolytic and Inflammatory Response to Fasting in Mice." Journal of Cellular Signaling 5, no. 1 (2024): 10–23. http://dx.doi.org/10.33696/signaling.5.108.

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The liver plays a crucial role in maintaining homeostasis for lipid and glucose. Hepatic lipid synthesis is regulated by nutritional signals in response to fasting and refeeding. It is known that overnutrition regulates MAPK-dependent pathways that control lipid metabolism in the liver by activating MAPK phosphatase-2 (MKP-2). Uncertainty still exists regarding the regulatory mechanisms and effects of MKP-2 on hepatic response to fasting. We investigated the effect of fasting on the expression of MKP-2 and the impact on hepatic inflammatory response to feeding a high-fat diet (HFD). In this st
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Dowds, C. Marie, Sabin-Christin Kornell, Richard S. Blumberg, and Sebastian Zeissig. "Lipid antigens in immunity." Biological Chemistry 395, no. 1 (2014): 61–81. http://dx.doi.org/10.1515/hsz-2013-0220.

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Abstract Lipids are not only a central part of human metabolism but also play diverse and critical roles in the immune system. As such, they can act as ligands of lipid-activated nuclear receptors, control inflammatory signaling through bioactive lipids such as prostaglandins, leukotrienes, lipoxins, resolvins, and protectins, and modulate immunity as intracellular phospholipid- or sphingolipid-derived signaling mediators. In addition, lipids can serve as antigens and regulate immunity through the activation of lipid-reactive T cells, which is the topic of this review. We will provide an overv
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Rozprawy doktorskie na temat "Signaling lipid"

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Benarab, Ammar. "Harnessing endothelial lipid signaling for ischemic stroke protection." Electronic Thesis or Diss., Université Paris Cité, 2021. http://www.theses.fr/2021UNIP5197.

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Justification : La fonction cérébrovasculaire est essentielle à la santé du cerveau, et les voies de protection vasculaire endogènes peuvent fournir des cibles thérapeutiques pour les troubles neurologiques. La signalisation S1P (sphingosine 1-phosphate) coordonne les fonctions vasculaires dans d'autres organes et les modulateurs S1P1 (récepteur S1P-1), y compris le fingolimod, sont prometteurs pour le traitement de l'AVC ischémique et hémorragique. Cependant, S1P1 coordonne également le trafic lymphocytaire, et les lymphocytes sont actuellement considérés comme la principale cible thérapeutiq
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Cheong, Fei Ying. "Regulation of lipid signaling at the Golgi by the lipid phosphatases hSAC1 and OCRL1." [S.l. : s.n.], 2007. http://nbn-resolving.de/urn:nbn:de:bsz:16-opus-71011.

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Herman, Moreno Maria Dolores. "Structural studies of proteins in apoptosis and lipid signaling." Doctoral thesis, Stockholms universitet, Institutionen för biokemi och biofysik, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-8212.

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Signaling pathways control the fate of the cell. For example, they promote cell survival or commit the cell to death (apoptosis) in response to cell injury or developmental stimuli, decisions, which are vital for the proper development and functioning of metazoan. Tight control of such pathways is essential; dysregulation of apoptosis can disrupt the delicate balance between cell proliferation and cell death ending up in pathological processes, including cancer, autoimmunity diseases, inflammatory diseases, or degenerative disorders. We have used a structural genomic approach to study the stru
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Aivazian, Dikran A. (Dikran Arvid) 1971. "Lipid-protein interactions of immunoreceptor signaling subunit cytoplasmic domains." Thesis, Massachusetts Institute of Technology, 2001. http://hdl.handle.net/1721.1/8583.

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Thesis (Ph.D.)--Massachusetts Institute of Technology, Dept. of Biology, 2001.<br>Vita.<br>Includes bibliographical references (leaves 116-131).<br>Protein-lipid interactions are emerging as key components of cellular processes such as protein and membrane trafficking and cell-cell signaling. Many proteins bind lipid reversibly, including cytoplasmic proteins involved in signal transduction, such as Ras and Src. Membrane binding is vital for the function of these signaling proteins both through co-localization with other signaling proteins as well as effects of lipid on intrinsic activities. I
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Secor, Jordan Douglas. "Phytochemical Antioxidants Induce Membrane Lipid Signaling in Vascular Endothelial Cells." The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1338391553.

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Cody, West Kime. "Autotaxin-mediated lipid signaling intersects with LIF and BMP signaling to promote the naive pluripotency transcription factor program." Kyoto University, 2018. http://hdl.handle.net/2433/232302.

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Kline, Michelle A. "Membrane cholesterol regulates vascular endothelial cell viability, function, and lipid signaling." Connect to resource, 2008. http://hdl.handle.net/1811/32175.

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Sadhukhan, Sushabhan. "Metabolism & Signaling of 4-Hydroxyacids: Novel Metabolic Pathways and Insight into the Signaling of Lipid Peroxidation Products." Case Western Reserve University School of Graduate Studies / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=case1339171892.

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Kilaru, Aruna. "Discovery of Anandamide, a Novel Lipid Signaling Molecule in Moss and Its Implications." Digital Commons @ East Tennessee State University, 2015. https://dc.etsu.edu/etsu-works/4771.

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Herrera-Velit, Patricia. "Bacterial lipopolysaccharides signaling pathways in mononuclear phagocytes involve protein and lipid kinases." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0034/NQ27161.pdf.

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Książki na temat "Signaling lipid"

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Banafshé, Larijani, Woscholski Rudiger, and Rosser Colin A, eds. Lipid signaling protocols. Humana, 2009.

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Larijani, Banafshé, Rudiger Woscholski, and Colin A. Rosser, eds. Lipid Signaling Protocols. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-115-8.

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Waugh, Mark G., ed. Lipid Signaling Protocols. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3170-5.

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Murphy, Eric J. Lipid-mediated signaling. CRC Press/Taylor & Francis, 2010.

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1963-, Murphy Eric J., and Rosenberger Thad A, eds. Lipid-mediated signaling. CRC Press/Taylor & Francis, 2009.

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Munnik, Teun, and Ingo Heilmann, eds. Plant Lipid Signaling Protocols. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-401-2.

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Capelluto, Daniel G. S., ed. Lipid-mediated Protein Signaling. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6331-9.

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Munnik, Teun, ed. Lipid Signaling in Plants. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-03873-0.

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Kihara, Yasuyuki, ed. Druggable Lipid Signaling Pathways. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50621-6.

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service), SpringerLink (Online, ed. Lipid Signaling in Plants. Springer-Verlag Berlin Heidelberg, 2010.

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Części książek na temat "Signaling lipid"

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Luna, Elizabeth J., Thomas Nebl, Norio Takizawa, and Jessica L. Crowley. "Lipid Raft Membrane Skeletons." In Membrane Microdomain Signaling. Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-803-x:047.

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Mak, Lok Hang. "Lipid Signaling and Phosphatidylinositols." In Encyclopedia of Biophysics. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-16712-6_537.

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Michaelson, Louise V., and Johnathan A. Napier. "Sphingolipid Signaling in Plants." In Lipid Signaling in Plants. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-03873-0_20.

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Yanagida, Keisuke, and William J. Valentine. "Druggable Lysophospholipid Signaling Pathways." In Druggable Lipid Signaling Pathways. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50621-6_7.

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Mattson, Mark P. "Dietary Modulation of Lipid Rafts." In Membrane Microdomain Signaling. Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-803-x:191.

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Mosblech, Alina, Ivo Feussner, and Ingo Heilmann. "Oxylipin Signaling and Plant Growth." In Lipid Signaling in Plants. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-03873-0_18.

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Kihara, Yasuyuki. "Introduction: Druggable Lipid Signaling Pathways." In Druggable Lipid Signaling Pathways. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50621-6_1.

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Gregus, Ann M., and Matthew W. Buczynski. "Druggable Targets in Endocannabinoid Signaling." In Druggable Lipid Signaling Pathways. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50621-6_8.

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Scherer, Günther F. E. "Phospholipase A in Plant Signal Transduction." In Lipid Signaling in Plants. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-03873-0_1.

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Im, Yang Ju, Brian Q. Phillippy, and Imara Y. Perera. "InsP3 in Plant Cells." In Lipid Signaling in Plants. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-03873-0_10.

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Streszczenia konferencji na temat "Signaling lipid"

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Maiti, Sudipta. "Extra-receptor signaling: how the lipid bilayer transduces neurotransmitter signals." In Multiphoton Microscopy in the Biomedical Sciences XXIV, edited by Ammasi Periasamy, Peter T. So, and Karsten König. SPIE, 2024. http://dx.doi.org/10.1117/12.3010037.

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Stamm, N., H. Asperger, M. Ludescher, T. Fehm, and H. Neubauer. "PGRMC1 alters de novo lipid biosynthesis resulting in enhanced oncogenic signaling." In Kongressabstracts zur Tagung 2020 der Deutschen Gesellschaft für Gynäkologie und Geburtshilfe (DGGG). © 2020. Thieme. All rights reserved., 2020. http://dx.doi.org/10.1055/s-0040-1718184.

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Taylor, Graham, Donald Leo, and Andy Sarles. "Detection of Botulinum Neurotoxin/A Insertion Using an Encapsulated Interface Bilayer." In ASME 2012 Conference on Smart Materials, Adaptive Structures and Intelligent Systems. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/smasis2012-8101.

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Many signaling mechanisms in living cells occur at biological boundaries via cell surface receptors and membrane proteins embedded in lipid bilayers. The coordination of actions of sensory and motor neurons in the nervous system represents one example of many that heavily depends on lipid membrane bound receptor mediated signaling. As a result, chemical and biological toxins that disrupt these neural signals can have severe physiological effects, including paralysis and death. Botulinum neurotoxin Type A (BoNT/A) is a proteolytic toxin that inserts through vesicle membranes and cleaves membran
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Parchaykina, M. V., I. D. Molchanov, E. V. Chudaikina, et al. "THE ROLE OF LIPID METABOLITES IN THE REGULATION OF REGENERATIVE PROCESSES IN DAMAGED SOMATIC NERVES." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-266.

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Under the action of various physiologically active substances, lipid metabolites in the damaged nerve trigger key signaling cascades that are responsible for the regeneration of damaged somatic nerves. Activation of this pathway leads to the regulation of many biological processes such as cell migration, proliferation, differentiation and apoptosis.
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Rong, Xi, Kenneth M. Pryse, Jordan A. Whisler, et al. "Confidence Intervals for Estimation of the Concentration and Brightness of Multiple Diffusing Species." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80921.

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Lipid nanodomains in cell membranes are believed to play a significant role in replication of enveloped viruses such as bird flu and HIV and signaling mechanisms underlying pathological conditions such as cancer. However, the forces that govern the formation and availability of these “membrane rafts” are uncertain, and even their existence is questioned. The central challenge is that no suitable imaging modalities exist (Elson, et al., 2010). We are developing tools to characterize and visualize dynamics of lipid nanodomains on idealized systems called giant unilamellar vesicles (GUVs) using f
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Jiang, Yanfei, Guy M. Genin, Srikanth Singamaneni, and Elliot L. Elson. "Interfacial Phases on Giant Unilamellar Vesicles." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80942.

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Lipid nanodomains in cell membranes are believed to play a significant role in a number of critical cellular processes (Elson, et al., 2010). These include, for example, replication processes in enveloped viruses such as bird flu and HIV and signaling mechanisms underlying pathological conditions such as cancer. Due to the potential for developing new disease treatments through the control of these membrane rafts, the biophysics underlying their formation has been the subject of intense study, much of this focused on domain formation in giant unilamellar lipid vesicles (GUVs), a simplified mod
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Bukowski, Michael, Brij Singh, James Roemmich, and Kate Larson. "Lipidomic analysis of TRPC1 Ca2+-permeable channel-knock out mouse demonstrates a vital role in placental tissue sphingolipid and triacylglycerol homeostasis under high-fat diet." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/tjdt4839.

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Placental function including oxygen delivery and nutrient transport are critical determinants of fetal growth, moderating the risks of obesity and metabolic diseases later in life. Previously, we demonstrated in a mouse model that parental diet and exercise play important roles in placental lipid content and inflammation. Transient receptor potential canonical channel 1 (TRPC1) is a Ca2+-permeable integral membrane protein. We have demonstrated that TRPC1 increases total body adiposity in mice by decreasing the efficacy of exercise to limit adipose accumulation under a high fat (HF) diet. Impo
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Wadgaonkar, R., and X. Jiang. "Sphingolipid Dependent Integration of TNF Receptor Signaling in Endothelial Cell Lipid Microdomains." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2478.

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Hawk, Ernest T., David G. Menter, Sherri Patterson, Michael W. Swank, and Raymond N. DuBois. "Abstract 3251: Linking prostaglandin E2 signaling, lipid rafts, and DNA protein kinase." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-3251.

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Shokat, Kevan M. "Abstract SY19-01: Chemical genetic investigations of protein and lipid kinase signaling." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-sy19-01.

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Raporty organizacyjne na temat "Signaling lipid"

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Gatley, S. J. Radiotracers For Lipid Signaling Pathways In Biological Systems. Office of Scientific and Technical Information (OSTI), 2016. http://dx.doi.org/10.2172/1326385.

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Brown Horowitz, Sigal, Eric L. Davis, and Axel Elling. Dissecting interactions between root-knot nematode effectors and lipid signaling involved in plant defense. United States Department of Agriculture, 2014. http://dx.doi.org/10.32747/2014.7598167.bard.

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Root-knot nematodes, Meloidogynespp., are extremely destructive pathogens with a cosmopolitan distribution and a host range that affects most crops. Safety and environmental concerns related to the toxicity of nematicides along with a lack of natural resistance sources threaten most crops in Israel and the U.S. This emphasizes the need to identify genes and signal mechanisms that could provide novel nematode control tactics and resistance breeding targets. The sedentary root-knot nematode (RKN) Meloidogynespp. secrete effectors in a spatial and temporal manner to interfere with and mimic multi
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Osathanon, Thanaphum, and Prasit Pavasant. Notch signaling in adipogenic differentiation of single-clone-derived mesenchymal stem cells isolated from human adipose tissue. Chulalongkorn University, 2013. https://doi.org/10.58837/chula.res.2013.5.

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Stem cells can be isolated from various tissues, including bone marrow, dental pulp, as well as adipose tissues. Due to the non-invasive isolation procedure, the adipose-derived mesenchymal stem cells (ADSCs) are introduced as an alternative stem cell source for regenerative medicine. In addition, it has been shown that Notch signaling participates in the control of ADSCs’ behavior. However, those studies were performed in the heterogeneous population of ADSCs. In the present study, human adipose tissue derived single-cell clones were isolated using a cloning ring technique and characterized f
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Barg, Rivka, Kendal D. Hirschi, Avner Silber, Gozal Ben-Hayyim, Yechiam Salts, and Marla Binzel. Combining Elevated Levels of Membrane Fatty Acid Desaturation and Vacuolar H+ -pyrophosphatase Activity for Improved Drought Tolerance. United States Department of Agriculture, 2012. http://dx.doi.org/10.32747/2012.7613877.bard.

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Background to the topic: In previous works we have shown that Arabidopsis and tomato over-expressing H+-pyrophosphatase show increased tolerance to drought imposed by withholding irrigation of young plants in pots (Park et al. 2005). In addition, young tobacco plants over-expressing fatty acid desaturase 3 (OEX-FAD3) also showed increasing tolerance to drought stress (Zhang et al 2005), and similarly OEX-FAD3 young tomato plants (unpublished data from ARO), hence raising the possibility that pyramiding the two could further improve drought tolerance in tomato. Based on these findings the speci
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