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Artykuły w czasopismach na temat „Sleep-wake cycle”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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1

Martell, M. "Sleep-Wake Cycle." Acta Scientific Paediatrics 5, no. 3 (2022): 25–26. http://dx.doi.org/10.31080/aspe.2022.05.0506.

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Mallika, M. C. Vasantha, and Ajay Jayakumar Nair. "Effect of Sleep-Wake Cycles on Academic Performances and Behavioural Changes among Undergraduate Medical Students." Healthline 15, no. 1 (2024): 86–90. http://dx.doi.org/10.51957/healthline5772023.

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Introduction: Sleep wake cycles form major part in the life of every student, starting from the school ages itself. This cycle has a major relationship in ensuring the proper functioning and day to day activities of the individual in all walks of life. Objectives: To assess the quality of sleep wake cycle among undergraduate medical students and to find out the association of sleep wake cycle with academic performances and behavioural changes among undergraduate medical students Results: In a cross sectional study among 300 participants, 35.3 % of the participants had good sleep-wake cycle. Th

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Claustrat, B., J. Brun, M. Geoffriau, G. Chazot, and M. J. Challarmel. "Melatonin, sleep-wake cycle and sleep." Biological Psychiatry 42, no. 1 (1997): 226S. http://dx.doi.org/10.1016/s0006-3223(97)87830-4.

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Kniazkina, Marina, and Vyacheslav Dyachuk. "Does EGFR Signaling Mediate Orexin System Activity in Sleep Initiation?" International Journal of Molecular Sciences 24, no. 11 (2023): 9505. http://dx.doi.org/10.3390/ijms24119505.

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Sleep–wake cycle disorders are an important symptom of many neurological diseases, including Parkinson’s disease, Alzheimer’s disease, and multiple sclerosis. Circadian rhythms and sleep–wake cycles play a key role in maintaining the health of organisms. To date, these processes are still poorly understood and, therefore, need more detailed elucidation. The sleep process has been extensively studied in vertebrates, such as mammals and, to a lesser extent, in invertebrates. A complex, multi-step interaction of homeostatic processes and neurotransmitters provides the sleep–wake cycle. Many other

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Putilov, Arcady A. "Can the Brain’s Thermostatic Mechanism Generate Sleep-Wake and NREM-REM Sleep Cycles? A Nested Doll Model of Sleep-Regulating Processes." Clocks & Sleep 6, no. 1 (2024): 97–113. http://dx.doi.org/10.3390/clockssleep6010008.

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Evidence is gradually accumulating in support of the hypothesis that a process of thermostatic brain cooling and warming underlies sleep cycles, i.e., the alternations between non-rapid-eye-movement and rapid-eye-movement sleep throughout the sleep phase of the sleep-wake cycle. A mathematical thermostat model predicts an exponential shape of fluctuations in temperature above and below the desired temperature setpoint. If the thermostatic process underlies sleep cycles, can this model explain the mechanisms governing the sleep cyclicities in humans? The proposed nested doll model incorporates

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Marita, P., and R. Acharya Pandey. "Prevalence of sleep – wake cycle disturbance among cancer patients of Bhaktapur cancer hospital, Nepal." Journal of Chitwan Medical College 6, no. 2 (2017): 6–13. http://dx.doi.org/10.3126/jcmc.v6i2.16678.

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Cancer patients are at great risk for developing insomnia and disorders of the sleep-wake cycle. Insomnia is the most common sleep disturbance in this population and is most often secondary to physical and/or psychological factors related to cancer and/or cancer treatment. It is estimated that nearly 45% of cancer patients experience sleep disturbances; this is nearly three times the estimate of its occurrence in the general population. The purpose of the study is to determine the prevalence of sleep-wake cycle disturbance in patient receiving chemotherapy. A descriptive cross-sectional study

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Duclos, Catherine, Marie Dumont, Caroline Arbour, et al. "Parallel recovery of consciousness and sleep in acute traumatic brain injury." Neurology 88, no. 3 (2016): 268–75. http://dx.doi.org/10.1212/wnl.0000000000003508.

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Objective:To investigate whether the progressive recuperation of consciousness was associated with the reconsolidation of sleep and wake states in hospitalized patients with acute traumatic brain injury (TBI).Methods:This study comprised 30 hospitalized patients (age 29.1 ± 13.5 years) in the acute phase of moderate or severe TBI. Testing started 21.0 ± 13.7 days postinjury. Consciousness level and cognitive functioning were assessed daily with the Rancho Los Amigos scale of cognitive functioning (RLA). Sleep and wake cycle characteristics were estimated with continuous wrist actigraphy. Mixed

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Wexler, D. B., and M. C. Moore-Ede. "Circadian sleep-wake cycle organization in squirrel monkeys." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 248, no. 3 (1985): R353—R362. http://dx.doi.org/10.1152/ajpregu.1985.248.3.r353.

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To investigate the relationship between circadian rhythms of body temperature and sleep-wake stages, four squirrel monkeys were prepared for unrestrained monitoring of temperature, locomotor activity, electroencephalogram, electroculogram, and electromyogram. Continuous records for each animal were made for several 12-h light-dark (LD) cycles and then after a few days in constant illumination (LL). All animals maintained consolidated sleep-wake cycles and had a longer circadian period (mean 24.7 h) in LL than in LD (mean 24.1 h). The increased period reflected greater time per circadian cycle

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Westmark, Pamela R., Timothy J. Swietlik, Ethan Runde, et al. "Adult Inception of Ketogenic Diet Therapy Increases Sleep during the Dark Cycle in C57BL/6J Wild Type and Fragile X Mice." International Journal of Molecular Sciences 25, no. 12 (2024): 6679. http://dx.doi.org/10.3390/ijms25126679.

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Sleep problems are a significant phenotype in children with fragile X syndrome. Our prior work assessed sleep–wake cycles in Fmr1KO male mice and wild type (WT) littermate controls in response to ketogenic diet therapy where mice were treated from weaning (postnatal day 18) through study completion (5–6 months of age). A potentially confounding issue with commencing treatment during an active period of growth is the significant reduction in weight gain in response to the ketogenic diet. The aim here was to employ sleep electroencephalography (EEG) to assess sleep–wake cycles in mice in respons

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Murillo-Rodriguez, Eric, Oscar Arias-Carrion, Katya Sanguino-Rodriguez, Mauricio Gonzalez-Arias, and Reyes Haro. "Mechanisms of Sleep-Wake Cycle Modulation." CNS & Neurological Disorders - Drug Targets 8, no. 4 (2009): 245–53. http://dx.doi.org/10.2174/187152709788921654.

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Shadan, Farhad F. "Sleep-wake cycle, aging and cancer." Journal of Applied Biomedicine 6, no. 3 (2008): 131–38. http://dx.doi.org/10.32725/jab.2008.016.

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Louzada, Fernando, and Luiz Menna-Barreto. "Sleep–Wake Cycle in Rural Populations." Biological Rhythm Research 35, no. 1-2 (2004): 153–57. http://dx.doi.org/10.1080/09291010412331313304.

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Westmark, Pamela R., Aaron K. Gholston, Timothy J. Swietlik, Rama K. Maganti, and Cara J. Westmark. "Ketogenic Diet Affects Sleep Architecture in C57BL/6J Wild Type and Fragile X Mice." International Journal of Molecular Sciences 24, no. 19 (2023): 14460. http://dx.doi.org/10.3390/ijms241914460.

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Nearly half of children with fragile X syndrome experience sleep problems including trouble falling asleep and frequent nighttime awakenings. The goals here were to assess sleep–wake cycles in mice in response to Fmr1 genotype and a dietary intervention that reduces hyperactivity. Electroencephalography (EEG) results were compared with published rest–activity patterns to determine if actigraphy is a viable surrogate for sleep EEG. Specifically, sleep–wake patterns in adult wild type and Fmr1KO littermate mice were recorded after EEG electrode implantation and the recordings manually scored for

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14

Dijk, Derk-Jan, and Steven W. Lockley. "Invited Review: Integration of human sleep-wake regulation and circadian rhythmicity." Journal of Applied Physiology 92, no. 2 (2002): 852–62. http://dx.doi.org/10.1152/japplphysiol.00924.2001.

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The human sleep-wake cycle is generated by a circadian process, originating from the suprachiasmatic nuclei, in interaction with a separate oscillatory process: the sleep homeostat. The sleep-wake cycle is normally timed to occur at a specific phase relative to the external cycle of light-dark exposure. It is also timed at a specific phase relative to internal circadian rhythms, such as the pineal melatonin rhythm, the circadian sleep-wake propensity rhythm, and the rhythm of responsiveness of the circadian pacemaker to light. Variations in these internal and external phase relationships, such

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15

Martoni, Monica, Marco Fabbri, Annalisa Grandi, Luisa Sist, and Lara Colombo. "Self-Care Practices as a Mediator between Workaholism and Sleep–Wake Problems during COVID-19." Sustainability 15, no. 16 (2023): 12603. http://dx.doi.org/10.3390/su151612603.

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Self-care practices are considered an important resource for workers’ psychophysical well-being. These resources were especially relevant during the COVID-19 outbreak, during which both workaholism and sleep–wake problems were documented. Our study aimed to examine whether workaholism could predict sleep–wake quality through the mediating effects of self-care practices. A convenient sample of 405 Italian workers (71.1% females; mean age = 42.58 ± 10.68 years) completed the Self-Care Practices Scale, Mini-Sleep Questionnaire, and Working Excessively and Working Compulsively Scale during the fir

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16

Kinoshita, Fukuaki L., Rikuhiro G. Yamada, Koji L. Ode, and Hiroki R. Ueda. "A unified framework to model synaptic dynamics during the sleep–wake cycle." PLOS Biology 23, no. 6 (2025): e3003198. https://doi.org/10.1371/journal.pbio.3003198.

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Understanding synaptic dynamics during the sleep–wake cycle in the cortex is crucial yet remains controversial. The synaptic homeostasis hypothesis (SHY) suggests synaptic depression during non-rapid eye movement (NREM) sleep, while other studies report synaptic potentiation or synaptic changes during NREM sleep depending on activities in wakefulness. To find boundary conditions between these contradictory observations, we focused on learning rules and firing patterns that contribute to the synaptic dynamics. Using computational models considering mammalian cortical neurons, we found that unde

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Gabova, A. V., E. A. Fedosova, and K. Yu Sarkisova. "Spindles in WAG/Rij Rats with Absence Epilepsy and Comorbid Depression." Rossijskij fiziologičeskij žurnal im. I.M. Sečenova 110, no. 6 (2024): 1037–54. http://dx.doi.org/10.31857/s0869813924060115.

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WAG/Rij rats are a valid model of absence epilepsy and comorbid depression. We have previously shown that WAG/Rij rats have disturbances in the sleep-wake cycle and changes in the characteristics of sleep spindles. A negative correlation was also found between the number of spike-wave discharges (SWD) and the duration of rapid eye movement (REM) sleep. Clinical evidence suggests that the traditional antidepressants imipramine and fluoxetine are effective in suppressing symptoms of depression, but may have a negative impact on the sleep-wake cycle and comorbid epilepsy in patients. Our previous

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18

Xu, Jiayang. "Analysis of the role of clock genes in the sleep-wake cycle and other biological processes." Theoretical and Natural Science 23, no. 1 (2023): 235–41. http://dx.doi.org/10.54254/2753-8818/23/20231067.

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Clock genes, forming the crux of the body's circadian system, underpin the molecular basis of circadian rhythms. These rhythms, following approximately 24-hour cycles, regulate an array of biological processes, enabling organisms to adjust to environmental shifts. The sleep-wake cycle, a fundamental manifestation of this, alongside crucial brain functions and basic physiological processes, demonstrates significant links to circadian rhythms. This paper explores the interplay between clock genes and the sleep-wake cycle, illustrating that these genes modulate the cycle by managing associated ho

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Holth, Jerrah K., Sarah K. Fritschi, Chanung Wang, et al. "The sleep-wake cycle regulates brain interstitial fluid tau in mice and CSF tau in humans." Science 363, no. 6429 (2019): 880–84. http://dx.doi.org/10.1126/science.aav2546.

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The sleep-wake cycle regulates interstitial fluid (ISF) and cerebrospinal fluid (CSF) levels of β-amyloid (Aβ) that accumulates in Alzheimer’s disease (AD). Furthermore, chronic sleep deprivation (SD) increases Aβ plaques. However, tau, not Aβ, accumulation appears to drive AD neurodegeneration. We tested whether ISF/CSF tau and tau seeding and spreading were influenced by the sleep-wake cycle and SD. Mouse ISF tau was increased ~90% during normal wakefulness versus sleep and ~100% during SD. Human CSF tau also increased more than 50% during SD. In a tau seeding-and-spreading model, chronic SD

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20

Bochkarev, M. V., L. S. Korostovtseva, A. B. Tataraidze, et al. "Sleep-wake cycle regularity and cardiometabolic indicators." Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova 121, no. 4 (2021): 57. http://dx.doi.org/10.17116/jnevro202112104157.

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Cantero, Jose L., Eva Hita-Yañez, Bernardo Moreno-Lopez, Federico Portillo, Alicia Rubio, and Jesus Avila. "Tau Protein Role in Sleep-Wake Cycle." Journal of Alzheimer's Disease 21, no. 2 (2010): 411–21. http://dx.doi.org/10.3233/jad-2010-100285.

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Geladze, Tina S., Kote S. Dzamashvili, and Maya G. Djibladze. "Sleep-Wake Cycle in Cluster Headache Patients." Cephalalgia 11, no. 11_suppl (1991): 260–61. http://dx.doi.org/10.1177/0333102491011s11139.

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Buguet, A., J. Bert, P. Tapie, et al. "Sleep-Wake Cycle in Human African Trypanosomiasis." Journal of Clinical Neurophysiology 10, no. 2 (1993): 190–96. http://dx.doi.org/10.1097/00004691-199304000-00006.

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Wagner, Daniel R. "DISORDERS OF THE CIRCADIAN SLEEP–WAKE CYCLE." Neurologic Clinics 14, no. 3 (1996): 651–70. http://dx.doi.org/10.1016/s0733-8619(05)70278-4.

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Karlsson, K. Æ., J. C. Kreider, and M. S. Blumberg. "Hypothalamic contribution to sleep–wake cycle development." Neuroscience 123, no. 2 (2004): 575–82. http://dx.doi.org/10.1016/j.neuroscience.2003.09.025.

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Ono, Daisuke, and Akihiro Yamanaka. "Hypothalamic regulation of the sleep/wake cycle." Neuroscience Research 118 (May 2017): 74–81. http://dx.doi.org/10.1016/j.neures.2017.03.013.

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Lemmer, Björn. "The sleep–wake cycle and sleeping pills." Physiology & Behavior 90, no. 2-3 (2007): 285–93. http://dx.doi.org/10.1016/j.physbeh.2006.09.006.

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Beersma, Domien G. M., and Marijke C. M. Gordijn. "Circadian control of the sleep–wake cycle." Physiology & Behavior 90, no. 2-3 (2007): 190–95. http://dx.doi.org/10.1016/j.physbeh.2006.09.010.

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DiNuzzo, Mauro, and Maiken Nedergaard. "Brain energetics during the sleep–wake cycle." Current Opinion in Neurobiology 47 (December 2017): 65–72. http://dx.doi.org/10.1016/j.conb.2017.09.010.

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Portas, Chiara M., Karsten Krakow, Phillip Allen, Oliver Josephs, Jorge L. Armony, and Chris D. Frith. "Auditory Processing across the Sleep-Wake Cycle." Neuron 28, no. 3 (2000): 991–99. http://dx.doi.org/10.1016/s0896-6273(00)00169-0.

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Székely, Miklós. "Orexins, energy balance, temperature, sleep-wake cycle." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 291, no. 3 (2006): R530—R532. http://dx.doi.org/10.1152/ajpregu.00179.2006.

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Wollman, M., and P. Lavie. "Hypernychthemeral Sleep-Wake Cycle: Some Hidden Regularities." Sleep 9, no. 2 (1986): 324–34. http://dx.doi.org/10.1093/sleep/9.2.324.

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Verbitsky, E. V., and M. G. Poluektov. "Energy processes in the sleep—wake cycle." S.S. Korsakov Journal of Neurology and Psychiatry 125, no. 5 (2025): 8. https://doi.org/10.17116/jnevro20251250528.

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An attempt was made to broadly consider energy exchange in the sleep—wake fulness cycle of cells not only in the brain, but also in other tissues of the body. For this purpose, the results of experiments on energy supply of tissues in sleep models performed on animals were generalized with the data of polysomnography and mass spectrometry of exhaled air of subjects examined during the night. Such an integrated approach allowed us to clarify the mechanisms of sleep, as well as to come closer to understanding its disorders in humans and to outline ways of their effective pharmacotherapy.

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Hsu, Chung-Yao, Yao-Chung Chuang, Fang-Chia Chang, Hung-Yi Chuang, Terry Ting-Yu Chiou, and Chien-Te Lee. "Disrupted Sleep Homeostasis and Altered Expressions of Clock Genes in Rats with Chronic Lead Exposure." Toxics 9, no. 9 (2021): 217. http://dx.doi.org/10.3390/toxics9090217.

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Sleep disturbance is one of the neurobehavioral complications of lead neurotoxicity. The present study evaluated the impacts of chronic lead exposure on alteration of the sleep–wake cycle in association with changes of clock gene expression in the hypothalamus. Sprague–Dawley rats with chronic lead exposure consumed drinking water that contained 250 ppm of lead acetate for five weeks. Electroencephalography and electromyography were recorded for scoring the architecture of the sleep–wake cycle in animals. At six Zeitgeber time (ZT) points (ZT2, ZT6, ZT10, ZT14, ZT18, and ZT22), three clock gen

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Gavrilov, Yuri V., Kristina Z. Derevtsova, and Elena A. Korneva. "Morphofunctional alterations of the hypothalamic neurons activity during sleep-wake cycle regulation disturbances after experimental traumatic brain injury." Medical academic journal 19, no. 3 (2019): 47–56. http://dx.doi.org/10.17816/maj19347-56.

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Relevance. The study of sleep disorders mechanisms after traumatic brain injury is complicated and poorly understood. Traumatic damage to the structures that are responsible for the sleep-wake cycle regulation is a common cause of sleep disorders after traumatic brain injury. The number of hypothalamic neurotransmitter systems, which are involved in the sleep-wake cycle regulation, could change its functional activity after trauma that suggests their key role in the development of disturbances of this process. The aim of the study was to assess the morphological alterations of the hypoth

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Martín-Olalla, José María. "The long term impact of Daylight Saving Time regulations in daily life at several circles of latitude." Scientific Reports 9 (December 5, 2019): 18466. https://doi.org/10.5281/zenodo.14855081.

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6000 words, 7 figures, supp material Winter daily rhythms and summer daily rhythms under seasonal clock regulations are compared. Lack of response to clock regulations is reported.   Abstract We analyze large scale (N &sim; 10 000) time use surveys in United States, Spain, Italy, France and Great Britain to ascertainseasonal variations in the sleep/wake cycle and the labor cycle after daylight saving time regulations have stood up for at leastforty years. That is, not the usual search for the impact of the biannual transitions, but a search for how industrialized societieshave answered to

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Yamanaka, Yujiro, Satoko Hashimoto, Yusuke Tanahashi, Shin-ya Nishide, Sato Honma, and Ken-ichi Honma. "Physical exercise accelerates reentrainment of human sleep-wake cycle but not of plasma melatonin rhythm to 8-h phase-advanced sleep schedule." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 298, no. 3 (2010): R681—R691. http://dx.doi.org/10.1152/ajpregu.00345.2009.

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Effects of timed physical exercise were examined on the reentrainment of sleep-wake cycle and circadian rhythms to an 8-h phase-advanced sleep schedule. Seventeen male adults spent 12 days in a temporal isolation facility with dim light conditions (<10 lux). The sleep schedule was phase-advanced by 8 h from their habitual sleep times for 4 days, which was followed by a free-run session for 6 days, during which the subjects were deprived of time cues. During the shift schedule, the exercise group ( n = 9) performed physical exercise with a bicycle ergometer in the early and middle waking per

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Culebras, Antonio. "Update on Disorders of Sleep and the Sleep-Wake Cycle." Psychiatric Clinics of North America 15, no. 2 (1992): 467–86. http://dx.doi.org/10.1016/s0193-953x(18)30250-8.

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García-García, Fabio, Mario Eduardo Acosta-Hernández, Luis Beltrán-Parrazal, and Juan Carlos Rodríguez-Alba. "The Role of Neuroglobin in the Sleep-Wake Cycle." Sleep Science 16, no. 03 (2023): e362-e367. http://dx.doi.org/10.1055/s-0043-1772806.

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AbstractNeuroglobin (Ngb) is a protein expressed in the central and peripherical nervous systems of the vertebrate. The Ngb has different functions in neurons, including regulating O2 homeostasis, oxidative stress, and as a neuroprotector after ischemia/hypoxia events. The Ngb is a hemoprotein of the globin family, structurally like myoglobin and hemoglobin. Ngb has higher expression in the cortex, hypothalamus, thalamus, brainstem, and cerebellum in mammals. Interestingly, Ngb immunoreactivity oscillates according to the sleep-wake cycle and decreases after 24 hours of sleep deprivation, sugg

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Assadzadeh, S., and P. A. Robinson. "Necessity of the sleep–wake cycle for synaptic homeostasis: system-level analysis of plasticity in the corticothalamic system." Royal Society Open Science 5, no. 10 (2018): 171952. http://dx.doi.org/10.1098/rsos.171952.

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Neural field theory is used to study the system-level effects of plasticity in the corticothalamic system, where arousal states are represented parametrically by the connection strengths of the system, among other physiologically based parameters. It is found that the plasticity dynamics have no fixed points or closed cycles in the parameter space of the connection strengths, but parameter subregions exist where flows have opposite signs. Remarkably, these subregions coincide with previously identified regions that correspond to wake and slow-wave sleep, thus demonstrating state dependence of

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Kunz, Dieter, and Werner Martin Herrmann. "Sleep-wake cycle, sleep-related disturbances, and sleep disorders: A chronobiological approach." Comprehensive Psychiatry 41, no. 2 (2000): 104–15. http://dx.doi.org/10.1016/s0010-440x(00)80016-4.

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De Martino, Milva Maria Figueiredo, Ana Cristina Basto Abreu, Manuel Fernando dos Santos Barbosa, and João Eduardo Marques Teixeira. "The relationship between shift work and sleep patterns in nurses." Ciência & Saúde Coletiva 18, no. 3 (2013): 763–68. http://dx.doi.org/10.1590/s1413-81232013000300022.

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The scope of this study was to evaluate the sleep/wake cycle in shift work nurses, as well as their sleep quality and chronotype. The sleep/wake cycle was evaluated by keeping a sleep diary for a total of 60 nurses with a mean age of 31.76 years. The Horne & Östberg Questionnaire (1976) for the chronotype and the Pittsburgh Sleep Quality Index (PSQI) for sleep quality were applied. The results revealed a predominance of indifferent chronotypes (65.0%), followed by moderately evening persons (18.3%), decidedly evening persons (8.3%), moderately morning persons (6.6%) and decidedly morning p

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Kanarskii, Mikhail, Julia Nekrasova, Pranil Pradhan, et al. "The High-Dose of Exogenous Melatonin Did Not Alter the Sleep-Wake Cycle in Anoxic Brain Injury Patients." Sleep Medicine Research 13, no. 2 (2022): 112–17. http://dx.doi.org/10.17241/smr.2022.01361.

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Disturbance in circadian rhythms and the sleep-wake cycle is typical for patients in the intensive care unit, which retards rehabilitation. To assess the effect of exogenous melatonin and simultaneous mitigation of intensive care unit environmental factors on sleep duration. We studied five patients with chronic disorder of consciousness caused by anoxic brain injury. In addition, we varied the level of melatonin secretion in blood plasma to assess melatonin’s bioavailability and elimination time. We evaluated the sleep-wake cycle using continuous videoelectroencephalogram monitoring with the

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Sattari, N., K. Simon, and S. Mednick. "0110 Fluctuations Across the Menstrual Cycle in Cardiac Autonomic Activity During Sleep and Wake May Affect Memory Consolidation." Sleep 43, Supplement_1 (2020): A43—A44. http://dx.doi.org/10.1093/sleep/zsaa056.108.

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Abstract Introduction Prior studies have shown that benefits of sleep for memory consolidation may be influenced by menstrual phase. Menstrual phase also impact autonomic regulation during sleep, and autonomic activity has been recently shown to play a role in sleep-dependent memory consolidation. Methods We investigated the interaction of menstrual cycle and autonomic activity measured by heart rate-variability (HRV) on sleep-dependent memory consolidation among 18-healthy females. Using a within-subjects design, we investigated episodic memory improvement with a nap paradigm during two phase

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Hajak, G. "Chronobiological issues of sleep and circadian rhythms." European Psychiatry 26, S2 (2011): 2133. http://dx.doi.org/10.1016/s0924-9338(11)73836-6.

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Progress in unravelling the cellular and molecular basis of mammalian circadian regulation over the past decade has provided us with data that deteriorations in measurable circadian output parameters, such as sleep/wake deficits and dysregulation of circulating hormone levels, are common features of most central nervous system disorders.At the core of the mammalian circadian system is a complex of molecular oscillations within the hypothalamic suprachiasmatic nucleus. These oscillations are modifiable by afferent signals from the environment, and integrated signals are subsequently conveyed to

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Yamanaka, Yujiro, Satoko Hashimoto, Satoru Masubuchi, et al. "Differential regulation of circadian melatonin rhythm and sleep-wake cycle by bright lights and nonphotic time cues in humans." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 307, no. 5 (2014): R546—R557. http://dx.doi.org/10.1152/ajpregu.00087.2014.

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Our previous study demonstrated that physical exercise under dim lights (<10 lux) accelerated reentrainment of the sleep-wake cycle but not the circadian melatonin rhythm to an 8-h phase-advanced sleep schedule, indicating differential effects of physical exercise on the human circadian system. The present study examined the effects of bright light (>5,000 lux) on exercise-induced acceleration of reentrainment because timed bright lights are known to reset the circadian pacemaker. Fifteen male subjects spent 12 days in temporal isolation. The sleep schedule was advanced from habitual sle

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Fierro, Adriela, Carmen Cortés, and José Eguibar. "0262 Sleep-wake cycle circadian disruption after chronic alcohol intake in a rat model of anxiety." Sleep 45, Supplement_1 (2022): A117—A118. http://dx.doi.org/10.1093/sleep/zsac079.260.

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Abstract Introduction Alcohol intake can produce disruptions in sleep-wake cycle, including circadian alterations like phase shifts. Alcohol intake is increased in subjects with anxiety to diminish its symptoms. We have selectively bred two sublines from Sprague-Dawley rats that differs on its yawning frequency. The high-yawning (HY) rats have a mean of 20 yawns/h, whereas the low-yawning (LY) rats have only 2 yawns/hour. LY male rats had high anxiety-like behavior in standardized tests and high preference for alcohol intake. The aim of this study was to assess circadian disruption of sleep-wa

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Gomes, Matheus Antonio, Fernanda Veruska Narciso, Marco Tulio de Mello, and Andrea Maculano Esteves. "Identifying electronic-sport athletes’ sleep-wake cycle characteristics." Chronobiology International 38, no. 7 (2021): 1002–9. http://dx.doi.org/10.1080/07420528.2021.1903480.

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Semenova, N. V., I. M. Madaeva, and L. I. Kolesnikova. "Clock Gene, Melatonin, and the Sleep–Wake Cycle." Russian Journal of Genetics 57, no. 3 (2021): 251–57. http://dx.doi.org/10.1134/s1022795421030121.

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Sinha, Prabhat, Susmita Chowdhuri, and James A. Rowley. "Sleep-Wake Cycle Diagnosed by CPAP Compliance Study." Journal of Clinical Sleep Medicine 4, no. 1 (2008): 70–72. http://dx.doi.org/10.5664/jcsm.27084.

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