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Gotowa bibliografia na temat „Spastic therapy”

Autor: Grafiati

Data publikacji: 3 czerwca 2025

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Spastic therapy"

Sutton, Andrew. "PHYSICAL THERAPY IN SPASTIC DIPLEGIA." Lancet 332, no. 8608 (1988): 460. http://dx.doi.org/10.1016/s0140-6736(88)90457-6.

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THYGESEN, PAUL, and ERNA CHRISTENSEN. "CURARE THERAPY IN SPASTIC CONDITIONS." Acta Psychiatrica Scandinavica 24, no. 2 (2007): 251–80. http://dx.doi.org/10.1111/j.1600-0447.1949.tb03496.x.

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Wardhani, Rizky Kusuma, Luh Karunia Wahyuni, Budiati Laksmitasari, and Alvin Lakmudin. "Effect of session frequency of radial extracorporeal shockwave therapy on gastrocnemius muscle spasticity in children with spastic cerebral palsy: a double-blinded, randomised clinical trial." International Journal of Therapy and Rehabilitation 29, no. 1 (2022): 1–6. http://dx.doi.org/10.12968/ijtr.2021.0047.

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Background/aims Extracorporeal shockwave therapy has been effective in reducing spasticity in patients with spastic cerebral palsy, but there has yet to be a recommended guideline for the treatment of spasticity in children using extracorporeal shockwave therapy. The aim of this study was to evaluate the effect of session frequency of radial extracorporeal shockwave therapy on gastrocnemius muscle spasticity in children with spastic cerebral palsy. Methods This was a double-blinded, randomised controlled trial consisting of 14 patients with spastic cerebral palsy (8 boys and 6 girls), aged 5 t

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Sardina, Francesca, Alessandra Pisciottani, Manuela Ferrara, et al. "Spastin recovery in hereditary spastic paraplegia by preventing neddylation-dependent degradation." Life Science Alliance 3, no. 12 (2020): e202000799. http://dx.doi.org/10.26508/lsa.202000799.

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Hereditary Spastic Paraplegia (HSP) is a neurodegenerative disease most commonly caused by autosomal dominant mutations in the SPG4 gene encoding the microtubule-severing protein spastin. We hypothesise that SPG4-HSP is attributable to reduced spastin function because of haploinsufficiency; thus, therapeutic approaches which elevate levels of the wild-type spastin allele may be an effective therapy. However, until now, how spastin levels are regulated is largely unknown. Here, we show that the kinase HIPK2 regulates spastin protein levels in proliferating cells, in differentiated neurons and i

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Hadders-Algra, Mijna, Jolanda C. van der Heide, Johanna M. Fock, Elisabeth Stremmelaar, Leo A. van Eykern, and Bert Otten. "Effect of Seat Surface Inclination on Postural Control During Reaching in Preterm Children With Cerebral Palsy." Physical Therapy 87, no. 7 (2007): 861–71. http://dx.doi.org/10.2522/ptj.20060330.

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Background and Purpose Because it is debatable whether seat surface inclination improves motor function in children with cerebral palsy (CP), the effect of seat surface tilting on postural control and quality of reaching was studied. Subjects The subjects were 58 children with CP aged 2 to 11 years (34 with unilateral spastic CP, 24 with bilateral spastic CP). Methods During the task of reaching movements, surface electromyographic and kinematic data were recorded for posture and reaching with the dominant arm in 3 sitting conditions: horizontal seat surface, seat surface tilted forward 15 deg

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Filippetti, Mirko, Rita Di Censo, Valentina Varalta, et al. "Is the Outcome of Diagnostic Nerve Block Related to Spastic Muscle Echo Intensity? A Retrospective Observational Study on Patients with Spastic Equinovarus Foot." Journal of Rehabilitation Medicine 54 (March 29, 2022): jrm00275. http://dx.doi.org/10.2340/jrm.v54.85.

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Objective: To investigate the relationship between spastic calf muscles echo intensity and the outcome of tibial nerve motor branches selective block in patients with spastic equinovarus foot.Design: Retrospective observational study. Patients: Forty-eight patients with spastic equinovarus foot.Methods: Each patient was given selective diagnostic nerve block (lidocaine 2% perineural injection) of the tibial nerve motor branches. All patients were evaluated before and after block. Outcomes were: spastic calf muscles echo intensity measured with the Heckmatt scale; affected ankle dorsiflexion pa

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Yulianti, Atika, Elita Hafi Dhiyah, and Anita Faradilla Rahim. "HUBUNGAN SELECTIVE CONTROL LOWER EXTREMITY DAN KEMAMPUAN BERJALAN PADA ANAK CEREBRAL PALSY TIPE SPASTIK." Jurnal Ilmiah Keperawatan (Scientific Journal of Nursing) 9, no. 2 (2023): 357–64. https://doi.org/10.33023/jikep.v9i2.1527.

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Pendahuluan: Cerebral Palsy (CP) merupakan gangguan karena lesi pada otak yang mengakibatkan beberapa permasalahan pada tonus otot, postur, dan gerakan. Tipe yang sering ditemui adalah tipe spastik. Pada tipe spastik ini letak kerusakannya pada bagian motor korteks yang akan menimbulkan spastisitas bahkan kontraktur sehingga sulit bergerak dan melakukan aktivitasnya. Gangguan pada CP spastik yang belum menjadi perhatian adalah gangguan pada selective motor control (SMC) yang merupakan salah satu faktor dalam perkembangan kemampuan motorik pada anak, dimana akan mempengaruhi kemampuan berjalann

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Hummelsheim, H., B. Münch, C. Bütefisch, and S. Neumann. "Influence of sustained stretch on late muscular responses to magnetic brain stimulation in patients with upper motor neuron lesions." Journal of Rehabilitation Medicine 26, no. 1 (1994): 3–9. http://dx.doi.org/10.2340/16501977199426139.

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Late muscular responses to transcranial magnetic stimulation occur in healthy subjects only in tonically active muscles with a proportional relationship between the amount of the EMG-response and the voluntary innervation strength. In hemiparetic stroke patients late potentials are elicited in chronically spastic hand flexors without voluntary background muscular activity probably reflecting enhanced excitability of spinal alpha-motoneurons in the spastic state. When spastic muscle hypertonus has been diminished by sustained muscle stretch late EMG-potentials are reduced or have disappeared co

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Королев and A. Korolev. "Scientific Basis of the Use of Medical Rehabilitation and Botulinum Therapy during Rehabilitation Post-Stroke Spasticity." Journal of New Medical Technologies 21, no. 1 (2014): 25–29. http://dx.doi.org/10.12737/3305.

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Among the factors contributing to disability after stroke is the most important movement disorder, major clinical manifestation of which is the central spastic paresis. Spasticity is revealed in the study of passive movements of the limbs as increased resistance to muscle in response to its rapid stretching. Once developed spastic paresis usually persists throughout the life of the patient. The treatment of spastic paresis is intractable problem. In recent years the treatment of post-stroke spasticity started using botulinum toxin type A. In comparison with the existing methods of treating mus

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Duncan, J., and P. Rudge. "Methylprednisolone therapy in tropical spastic paraparesis." Journal of Neurology, Neurosurgery & Psychiatry 53, no. 2 (1990): 173–74. http://dx.doi.org/10.1136/jnnp.53.2.173-a.

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Rozprawy doktorskie na temat "Spastic therapy"

Fox, Cynthia Marie. "Intensive voice treatment for children with spastic cerebral palsy." Diss., The University of Arizona, 2002. http://hdl.handle.net/10150/280175.

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Purpose. The purpose of this study was to examine the effects of an intensive speech treatment for children with spastic cerebral palsy (CP). Background. Children with spastic CP have speech and voice disorders, which may limit functional communication and negatively impact quality of life. There are limited published outcome data on speech treatment approaches for these children. Recent advances in theories of motor development and behavioral gait and limb treatment provide a solid framework (consistent with theories of motor learning) from which to test different speech treatment concepts (e

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Berman, Barbara. "Assessment of spastic patients before and after selective posterior lumbar rhizotomy." Master's thesis, University of Cape Town, 1988. http://hdl.handle.net/11427/25839.

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For many decades people working with spastic patients have searched for better methods of alleviating spasticity and its debilitating effects. Although many methods have been used, no single method has been completely successful. It was not until the nineteen seventies that Selective Posterior Lumbar Rhizotomy (SPLR) was used successfully with spastic patients. In 1980 Peacock started doing this procedure on a large number of spastic patients in South Africa. I became interested in SPLR after seeing the dramatic reduction in spasticity and the improvement in function in my patients who had und

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Hoving, Marjanke Aaltje. "Intrathecal baclofen therapy in children with intractable spastic cerebral palsy a Dutch national study /." [Maastricht] : Maastricht : Universitaire Pers Maastricht ; University Library, Universiteit Maastricht [host], 2008. http://arno.unimaas.nl/show.cgi?fid=10712.

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Rousseau, Aletta Helena. "The use of play activities in eliciting specific components of movement in the development of handfunction of the spastic cerebral palsied child." Master's thesis, University of Cape Town, 1985. http://hdl.handle.net/11427/26356.

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Fung, Joyce. "The muscle activation and reflex modulation pattern during locomotion in normal and spastic paretic subjects." Thesis, McGill University, 1992. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=39327.

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The lower limb electromyographic (EMG) patterns of 5 normal and 8 spastic paretic subjects during walking were compared. A dynamic EMG profile index, proposed to quantify spastic locomotor dysfunction, was found to be homogeneously low in normal subjects and abnormally high in spastic subjects. In another study, the normally phase-dependent modulation of the soleus H-reflex during walking was found to be decreased or absent in 21 spastic paretic subjects, suggesting the possibility of defective reflex gating mechanisms. The conditioning effects of plantar cutaneomuscular stimulation were furth

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Unger, Marianne. "The impact of an eight-week progressive resisted exercise program in adolescents with spastic cerebral palsy." Thesis, Stellenbosch : Stellenbosch University, 2004. http://hdl.handle.net/10019.1/50155.

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Thesis (MScPhysio)--Stellenbosch University, 2004.<br>ENGLISH ABSTRACT: Muscle weakness is a problem for many young people with spastic cerebral palsy (CP). Many studies have reported that selective strength-training programs can improve muscle strength. However, most of these studies are of single group design and do not adequately control for confounding variables. Objective: To determine the impact of a comprehensive strength training program targeting multiple muscle groups on adolescents with CP, using basic inexpensive free weights and resistance devices. Method: A randomised cl

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Novak, Iona, University of Western Sydney, and College of Social and Health Sciences. "The effect of a standardised occupational therapy home program for children with spastic hemiplegic cerebral palsy." THESIS_CSHS_XXX_Novak_I.xml, 2004. http://handle.uws.edu.au:8081/1959.7/694.

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Despite the popularity of home program interventions there is little evidence to demonstrate effectiveness, particularly when an explicit family centred framework is adopted. This single-group pre-post design study evaluates the impact of a standardised occupational therapy home program implemented with a group of 20 children (2-7 years, mean age 3.8)with spastic hemiplegic cerebral palsy. The study measured the effect of the program using: goal attainment scaling(GAS);pediatric evaluation of disability inventory (PEDI); and quality of upper extremity test(QUEST). In addition, parent participa

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Novak, Iona E. "The effect of a standardised occupational therapy home program for children with spastic hemiplegic cerebral palsy." View thesis, 2004. http://library.uws.edu.au/adt-NUWS/public/adt-NUWS20050615.162942/index.html.

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Mirbagheri, M. Mehdi (Mohammad Mehdi). "Intrinsic and reflex stiffness in normal & spastic spinal cord injured subjects." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=36823.

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The contributions of stretch reflexes to overall joint stiffness are unknown in normal and pathological function. The main reason for this is difficulty in distinguishing the mechanical contributions of the two mechanisms. We used a new non-linear system identification technique to achieve this and address three specific objectives.<br>First, we identified intrinsic and reflex contributions to dynamic ankle stiffness over a wide range of tonic voluntary contraction levels and ankle positions in healthy human subjects (normals). Intrinsic and reflex dynamic stiffness were strongly modulated wit

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Visintin, Martha. "The effect of body weight support on the locomotor pattern of spastic paraparetic subjects walking on a treadmill /." Thesis, McGill University, 1989. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=59238.

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This study investigated the effects of providing body weight support (BWS) on the gait pattern of 15 incomplete spinal cord lesioned subjects. Electromyographic (EMG), joint angular displacement and temporal distance parameters were simultaneously recorded as subjects walked on a treadmill while 0% and 40% of their body weight was mechanically supported by an overhead harness. The effects of 0% and 40% BWS while walking with and without parallel bars and at different treadmill speeds was further investigated in a subgroup of 8 subjects.<br>In general, 40% BWS led to a decrease in prolonged EMG

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Książki na temat "Spastic therapy"

Doman, Glenn J. What to do about your brain-injured child: Or your brain-damaged, mentally retarded, mentally deficient, cerebral-palsied, emotionally disturbed, spastic, flaccid, rigid, epileptic, autistic, athetoid, hyperactive, down's child. M. Evans, 1990.

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Cioni, Giovanni, and Adriano Ferrari. The Spastic Forms of Cerebral Palsy. Springer, 2010.

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Araujo, Abelardo Q.-C. Neurological Manifestations of the Human T-lymphotropic Virus Type 1. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0161.

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The human T cell lymphotropic virus type 1 (HTLV-1) is a retrovirus that infects about 20 million individuals worldwide. Its typical neurological presentation is of a chronic, slowly progressive myelopathy named “HTLV-1-associated myelopathy/tropical spastic paraparesis” (HAM/TSP). HAM/TSP emerges as the tip of the iceberg among numerous other neurological clinical syndromes caused by this virus, such as inflammatory myopathies, polyneuropathies, ALS-like syndromes, dysautonomia, etc. HAM/TSP designates a spastic paraparesis with neurogenic bladder, and minor sensory signs. Pathologically, HAM

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Doman, Glenn. What to Do About Your Brain Injured Child: Or Your Brain-damaged, Mentally Retarded, Mentally Deficient, Cerebral-palsied, Spastic, Flaccid, Rigid, Epileptic, Autistic, Athetoid, Hyperactive Child. 3rd ed. Gentle Revolution Press, 2003.

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What to do about your brain-injured child: Or your brain-damaged, mentally retarded, mentally deficient, cerebral-palsied, spastic, flaccid, rigid, epileptic, autistic, athetoid, hyperactive, Down's child. Avery Pub. Group, 1994.

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Wolf, Barry, and Sara Elrefai. Biotinidase Deficiency and Biotin Holocarboxylase Synthetase Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0026.

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Biotin holocarboxylase synthetase and biotinidase deficiencies are biotin-dependent, autosomal recessively inherited disorders. Untreated individuals with both disorders usually develop neurological and cutaneous symptoms during childhood. There is limited information about older children or adults with holocarboxylase synthetase deficiency. Older individuals with biotinidase deficiency may exhibit spastic paresis, scotomata, and progressive optic neuropathy. Both disorders are readily treated with oral biotin supplementation. Asymptomatic individuals with biotinidase deficiency are at risk of

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Joneja, Janice M. Vickerstaff. Digestion, Diet, and Disease: Irritable Bowel Syndrome and Gastrointestinal Function. Rutgers University Press, 2004.

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Części książek na temat "Spastic therapy"

Wissel, J., G. Ebersbach, and L. Gutjahr. "Biosignal Processing: An Electromyographic Biofeedback-Aided Therapy in Spastic Hemiparesis." In Adapted Physical Activity. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74873-8_51.

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Herrlich, Simon, S. Ziolek, H. Hoefemann, R. Zengerle, and S. Haeberle. "Adjustable Diffusion Barrier for Controlled Drug Release in Spastic and Pain Therapy." In IFMBE Proceedings. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-89208-3_568.

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Kollewe, Katja, and Reinhard Dengler. "Therapie der Spastik." In Die neurologisch-neurochirurgische Frührehabilitation. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-24886-3_11.

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Kappel, Stefan. "Multimodale Therapie der Spastik." In Assistive Technologien, technische Rehabilitation und Unterstützte Kommunikation. Springer Berlin Heidelberg, 2023. http://dx.doi.org/10.1007/978-3-662-64118-7_13.

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Čumlivski, Radomir. "Intrathekale Therapie der Spastik – Neuromodulation." In Neuroorthopädie - Disability Management. Springer Berlin Heidelberg, 2021. http://dx.doi.org/10.1007/978-3-662-61330-6_31.

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Benecke, R., and D. Dressler. "Diagnostik, Klassifikation und Therapie der Spastik bei Multipler Sklerose." In Multiple Sklerose. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-59453-3_15.

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Elenz, St, H. J. Böhm, and G. Hierholzer. "Indikationen und Ergebnisse der Baclofenpumpen-Implantation zur Therapie der Spastik bei Rückenmarkverletzten." In 53. Jahrestagung der Deutschen Gesellschaft für Unfallheilkunde e.V. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75920-8_214.

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Kleopa, Kleopas. "Hereditary Spastic Paraplegia." In Neurological Disease and Therapy. CRC Press, 2005. http://dx.doi.org/10.1201/9780849358364.ch12.

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Tilton, Ann H. "The Use of Botulinum Neurotoxin in Spastic Infantile Cerebral Palsy." In Manual of Botulinum Toxin Therapy, 3rd ed. Cambridge University Press, 2023. http://dx.doi.org/10.1017/9781009105033.018.

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Nihonmatsu, Akira. "Somatosensory stimulation (acupuncture) modulates spinal and supraspinal motor neuron excitability." In Physical Therapy - Towards Evidence-Based Practice [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.1002498.

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It has been reported that acupuncture is effective in alleviating abnormalities of muscle tone caused by abnormal motor neuron excitability such as spastic paralysis caused by cerebrovascular disorder. However, the underlying mechanism is unclear. Thus, we examined the effect of acupuncture stimulation on long-latency reflexes (LLR) to determine the site of action of acupuncture stimulation in modulating motor reflexes. The amplitude ratio of LLR/M was reduced by the acupuncture stimulation of LI4 (hand). Furthermore, we examined the effect of acupuncture stimulation on blink reflexes. The R2

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Streszczenia konferencji na temat "Spastic therapy"

Lyubov, Rychkova, Mikhnovich Vanda, Belogorova Tatyana, Vlasenko Anastasiya, Dutova Natalya, and Taskaeva Tatyana. "P300 Rehabilitation of children with spastic cerebral palsy after botulinum toxin therapy." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.388.

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Mukhopadhyay, R., M. Mahadevappa, P. K. Lenka, and A. Biswas. "Correction of Toe-Walking Gait in Children with Spastic Cerebral Palsy by using Electrical Stimulation Therapy." In 2018 40th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2018. http://dx.doi.org/10.1109/embc.2018.8513043.

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Shidik, Beta Arya Ash, and Hartomo Soewardi. "Innovative design of the foot health therapy appliance for the cerebral palsy patients of spastic diplegia." In 3RD CONFERENCE ON INNOVATION IN TECHNOLOGY AND ENGINEERING SCIENCE 2022 (CITES2022): Innovation in Technology and Science for New Era of Engineering Professionalism. AIP Publishing, 2024. http://dx.doi.org/10.1063/5.0201567.

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de Almeida, M. F., A. Obrecht, M. B. Zonta, and A. C. Crippa. "The Effect of Constraint-Induced Movement Therapy on the Rest-activity Patterns in Children with Spastic Hemiparetic Cerebral Palsy." In XIX Congresso Brasileiro do Sono. Thieme Revinter Publicações Ltda., 2023. http://dx.doi.org/10.1055/s-0043-1770233.

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Silva, Diógenes Emanuel Dantas da, Roberta Lessa Ribeiro, Iony Santana Santos Dantas, et al. "Atypical presentation of progressive multifocal leukoencephalopathy in a patient with ataxic syndrome and retrovirus with infra-tentorial involvement: a case report." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.615.

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Introduction: Progressive Multifocal Leukoencephalopathy (PML) is a rare demyelinating disease of the central nervous system caused by reactivation of the JC virus. It usually occurs in immunocompromised patients. The majority of PML cases present with supratentorial involvement. In contrast, infratentorial involvement is exceedingly rare in PML. The aim to report an atypical presentation of PML in a patient with ataxic syndrome and retrovirus with infra-tentorial involvement. This is a case report study. The information of this work was obtained through review of the medical record. Case repo

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Trivellato, Stella de Angelis, Joao Lucas Gomes Salgado, Hendrick Henrique Fernandes Gramasco, et al. "Thoracolumbar progressive myelopathy due to extra-dural arteriovenous fistula." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.223.

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Context: Arteriovenous dural fistulas are the most common spinal vascular malformations and constitute a reversible cause of progressive myelopathy. This disease affects elderly men and are classically found at the thoracolumbar region. Case report: A 69year-old man was admitted to the Neurology outpatient clinic with weakness in his left inferior limb for the past 10 years; 2 years before the evaluation, the weakness progressed also to the right inferior limb, causing inability to walk, and 1 year after the patient noticed urinary incontinence and sexual dysfunction. Physical evaluation showe

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