Gotowe bibliografie tematyczne / Spherocytosis

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  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji

Gotowa bibliografia na temat „Spherocytosis”

Autor: Grafiati
Data publikacji: 3 czerwca 2025
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Spherocytosis"

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LI, Ying, Liyuan Lu, and Juan LI. "Topological Structures and Membrane Nanostructures of Erythrocytes after Splenectomy in Hereditary Spherocytosis Patients via Atomic Force Microscopy." Cell Biochemistry and Biophysics 74, no. 3 (2016): 365–71. http://dx.doi.org/10.1007/s12013-016-0755-4.

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Abstract Hereditary spherocytosis is an inherited red blood cell membrane disorder resulting from mutations of genes encoding erythrocyte membrane and cytoskeletal proteins. Few equipments can observe the structural characteristics of hereditary spherocytosis directly expect for atomic force microscopy In our study, we proved atomic force microscopy is a powerful and sensitive instrument to describe the characteristics of hereditary spherocytosis. Erythrocytes from hereditary spherocytosis patients were small spheroidal, lacking a well-organized lattice on the cell membrane, with smaller cell
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Shah, S., and R. Vega. "Hereditary Spherocytosis." Pediatrics in Review 25, no. 5 (2004): 168–72. http://dx.doi.org/10.1542/pir.25-5-168.

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Mahajan, Vasudha, and Sunil K. Jain. "Hereditary Spherocytosis." NeoReviews 17, no. 12 (2016): e697-e704. http://dx.doi.org/10.1542/neo.17-12-e697.

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Kalyan, Meenakshi, ShubhangiA Kanitkar, AnuN Gaikwad, and Harsh Kumar. "Hereditary spherocytosis." Medical Journal of Dr. D.Y. Patil University 7, no. 2 (2014): 229. http://dx.doi.org/10.4103/0975-2870.126357.

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Koju, Surendra, and Ramesh Makaju. "Hereditary Spherocytosis." Journal of Lumbini Medical College 6, no. 1 (2018): 41–43. http://dx.doi.org/10.22502/jlmc.v6i1.202.

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Introduction: Hereditary spherocytosis is a red cell membrane disorder that causes hemolytic anemia. Due to defective cell membrane, red cells are spherical shaped and result in their early lysis. Osmotic fragility of spherocytic red cell is increased.
 Case report: A 22 year old female presented with chief complain of abdominal pain. Initially she was diagnosed as cholelithiasis. Under laboratory evaluation she was found to be anemic with reticulocytosis. In peripheral blood smear, spherocytes were moderately distributed. Antihuman globulin test was negative but osmotic fragility was hig
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CROOM, ROBERT D., CAMPBELL W. MCMILLAN, GEORGE F. SHELDON, and EUGENE P. ORRINGER. "Hereditary Spherocytosis." Annals of Surgery 203, no. 1 (1986): 34–39. http://dx.doi.org/10.1097/00000658-198601000-00007.

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Perrotta, Silverio, Patrick G. Gallagher, and Narla Mohandas. "Hereditary spherocytosis." Lancet 372, no. 9647 (2008): 1411–26. http://dx.doi.org/10.1016/s0140-6736(08)61588-3.

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Iolascon, A., R. A. Avvisati, and C. Piscopo. "Hereditary spherocytosis." Transfusion Clinique et Biologique 17, no. 3 (2010): 138–42. http://dx.doi.org/10.1016/j.tracli.2010.05.006.

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Trompeter, Sara, and May-Jean King. "Hereditary spherocytosis." Paediatrics and Child Health 25, no. 8 (2015): 381–86. http://dx.doi.org/10.1016/j.paed.2015.04.003.

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Trompeter, Sara, and May-Jean King. "Hereditary spherocytosis." Paediatrics and Child Health 29, no. 8 (2019): 359–64. http://dx.doi.org/10.1016/j.paed.2019.05.006.

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Rozprawy doktorskie na temat "Spherocytosis"

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Rocha, Susana Maria Santos. "Biological Aspects Underlying the Clinical Outcome of Hereditary Spherocytosis." Doctoral thesis, Faculdade de Farmácia da Universidade do Porto, 2009. http://hdl.handle.net/10216/63809.

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Rocha, Susana Maria Santos. "Biological Aspects Underlying the Clinical Outcome of Hereditary Spherocytosis." Tese, Faculdade de Farmácia da Universidade do Porto, 2009. http://hdl.handle.net/10216/63809.

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Simionato, Greta [Verfasser]. "The influence of hypoxia in erythropoiesis and morphology of red blood cells in sickle cell disease and hereditary spherocytosis. / Greta Simionato." Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2020. http://d-nb.info/1221599666/34.

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Poolla, Radhika. "A Reinforcement Learning Approach To Obtain Treatment Strategies In Sequential Medical Decision Problems." [Tampa, Fla.] : University of South Florida, 2003. http://purl.fcla.edu/fcla/etd/SFE0000215.

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TOUCHARD, ELISABETH. "Etude du cytosquelette de la membrane erythrocytaire lors de la spherocytose hereditaire (maladie de minkowski - chauffard) : a propos de 4 observations chez l'enfant." Saint-Etienne, 1994. http://www.theses.fr/1994STET6223.

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Dubois-Honoré, Marie, and Jeanne Pelzer. "La maladie de Minkowski-Chauffard (sphérocytose héréditaire) : manifestations néonatales : à propos de cinq cas." Lille 2, 1991. http://www.theses.fr/1991LIL2M156.

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ACHKAR, ELIE. "Une maladie de minkowski-chauffard revelee par une crise erythroblastopenique due au parvovirus humain b 19 : a propos d'une observation." Toulouse 3, 1991. http://www.theses.fr/1991TOU31071.

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Saada, Véronique. "Incidence de la sphérocytose héréditaire dans une population de nouveau-nés ictériques : étude des paramètres erythrocytaires dans la sphérocytose héréditaire et dans la sphérocytose acquise allo-immune en période néonatale : rôle de la maladie de Gilbert dans l'hyperbilirubinémie." Paris 5, 1999. http://www.theses.fr/1999PA05P084.

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Bridgemohan, Roshini. "Red cell membrane abnormalities in hereditary spherocytosis patients of KwaZulu-Natal." Thesis, 2006. http://hdl.handle.net/10413/2055.

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Hereditary Spherocytosis (HS) is a common inherited haemolytic anaemia with variable clinical expression. Fifty subjects with HS from KwaZulu-Natal were studied with the aim of providing further information on the protein abnormalities of the red blood cell (RBC) membrane and their relationship with clinical presentations. Haematological and biochemical tests were performed by routine procedures. Mean Corpuscular Haemoglobin Concentration ( MCHC) in the HS group was 35.1g /dl. This was significantly higher than in normal control subjects (33.6g /dl) (p value < 0.001); indicating its usefulness
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Babu, Wilson Anand. "Pyruvate Kinase Activator Mitapivat Ameliorates Anemia and Prevents Iron Overload in a Mouse Model of Hereditary Spherocytosis." Doctoral thesis, 2021. http://hdl.handle.net/11562/1045441.

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Hereditary spherocytosis (HS) is the most common cause of inherited red cell membranopathy, due to mutations in genes encoding for membrane or cytoskeletal proteins, including band 3, ankyrin, spectrin, band 4.1 or band 4.2. Membrane instability results in membrane surface area loss and generation of spherocytic red cells with elevated MCHC, decreased cellular deformability and reduced red cell survival, due to splenic sequestration. Clinical management of the hemolytic anemia due to HS depends on the age of the patient and the severity of anemia. Splenectomy is indicated in children with symp
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Książki na temat "Spherocytosis"

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Carton, James. Haematopathology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199591633.003.0014.

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Iron deficiency anaemia 256Anaemia of chronic disease 257Megaloblastic anaemias 258Hereditary spherocytosis 259Glucose-6-phosphate dehydrogenase deficiency 260Thalassaemias 261Sickle cell disorders 262Idiopathic thrombocytopenic purpura 264Thrombotic thrombocytopenic purpura 265von Willebrand disease 266Haemophilia 267Thrombophilia 268Acute B-lymphoblastic leukaemia 269...
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Carton, James. Haematopathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0015.

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This chapter discusses haematopathology, including iron deficiency anaemia, anaemia of chronic disease, megaloblastic anaemias, hereditary spherocytosis, glucose-6-phosphate dehydrogenase deficiency, thalassaemias, sickle-cell disorders, idiopathic thrombocytopenic purpura (ITP), thrombotic thrombocytopenic purpura (TTP), von Willebrand disease, haemophilia, thrombophilia, acute B-lymphoblastic leukaemia, acute myeloid leukaemias, chronic lymphocytic leukaemia (CLL), chronic myelogenous leukaemia, polycythaemia vera (PV), essential thrombocythaemia (ET), primary myelofibrosis (PMF), myelodyspl
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Provan, Drew, Trevor Baglin, Inderjeet Dokal, and Johannes de Vos. Red cell disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0002.

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The peripheral blood film in anaemias - Anaemia in renal disease - Anaemia in endocrine disease - Anaemia in joint disease - Anaemia in gastrointestinal disease - Anaemia in liver disease - Iron (Fe) deficiency anaemia - Vitamin B12 deficiency - Folate deficiency - Other causes of megaloblastic anaemia - Anaemia in other deficiency states - Haemolytic syndromes - Genetic control of haemoglobin production - Sickling disorders - HbS—sickle-modifying therapies - Sickle cell trait (HbAS) - Other sickling disorders - Other haemoglobinopathies - Unstable haemoglobins - Thalassaemias - α thalassaemia
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Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, Banu Kaya, and Angela Theodoulou. Red cell disorders. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0002_update_001.

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The peripheral blood film in anaemias - Anaemia in renal disease - Anaemia in endocrine disease - Anaemia in joint disease - Anaemia in gastrointestinal disease - Anaemia in liver disease - Iron (Fe) deficiency anaemia - Vitamin B12 deficiency - Folate deficiency - Other causes of megaloblastic anaemia - Anaemia in other deficiency states - Haemolytic syndromes - Genetic control of haemoglobin production - Sickling disorders - HbS—sickle-modifying therapies - Sickle cell trait (HbAS) - Other sickling disorders - Other haemoglobinopathies - Unstable haemoglobins - Thalassaemias - α‎ thalassaemi
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Bunch, Chris. Haemolytic anaemia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0280.

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Haemolytic anaemias occur when the rate of red-cell breakdown is increased and exceeds the marrow’s capacity to generate new cells. Increased red-cell destruction, or haemolysis, may reflect a broad range of disorders. Some involve intrinsic defects in the red cell itself; in others, the red cells are normal but are subjected to external factors which lead to premature destruction. Many of the intrinsic defects are due to inherited disorders affecting the red-cell membrane, its enzymes, or haemoglobin. The marrow can normally compensate for moderate haemolysis by increasing red-cell production
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Części książek na temat "Spherocytosis"

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Taylor, Jeffrey S. "Hereditary Spherocytosis." In Pediatric Surgery. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-04340-1_52.

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Gupta, Arun. "Hereditary Spherocytosis." In Decision Making Through Problem Based Learning in Hematology. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-99-8933-1_7.

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Leung, Anskar Y. H., and Edmond S. K. Ma. "Hereditary Spherocytosis." In Haematology and the Asian Patient. CRC Press, 2024. http://dx.doi.org/10.1201/9781003325413-46.

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Leung, Alexander K. C., Cham Pion Kao, Andrew L. Wong, et al. "Spherocytosis, Hereditary." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1649.

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Metze, Dieter, Vanessa F. Cury, Ricardo S. Gomez, et al. "Hereditary Spherocytosis." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_9223.

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Granjo, Elisa, Alice Santos-Silva, Irene Rebelo, et al. "Hematological and Biochemical Parameters in Hereditary Spherocytosis Under Oxidative Stress." In Molecular Biology of Hematopoiesis 6. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4797-6_41.

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Lecomte, M. C., J. Delaunay, and D. Dhermy. "Spectrin and other red cell membrane proteins in hereditary elliptocytosis and spherocytosis." In Protein Dysfunction in Human Genetic Disease. Garland Science, 2024. http://dx.doi.org/10.1201/9781003579953-11.

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Granjo, Elisa, Ilídia Moreira, Alice Santos-Silva, et al. "Lymphocyte Populations in Hereditary Spherocytosis Pre and Post Splenectomy and Under Oxidative Stress." In Molecular Biology of Hematopoiesis 6. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4797-6_42.

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Halterman, Jill S., and George B. Segel. "Spherocytosis." In Pediatric Clinical Advisor. Elsevier, 2007. http://dx.doi.org/10.1016/b978-032303506-4.10306-2.

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"Hereditary Spherocytosis." In Diagnostic Pathology: Blood and Bone Marrow. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-39254-9.50032-5.

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Streszczenia konferencji na temat "Spherocytosis"

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Ahel, Ivona Butorac, Kristina Baraba Dekanić, Goran Palčevski, and Jelena Roganović. "289 Hereditary spherocytosis associated with Gilbert syndrome – a case report." In 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.289.

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Hoxha, Gladiola, Donjeta Bali, Mirzana Kapllanaj, Mirela Xhafa, Elizana Petrela, and Anila Godo. "P342 Clinical course in children diagnosed with hereditary spherocytosis in albania." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.691.

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Shaukat, Memoona, Mareike Lankeit, Ding Cao, Nicola Benjamin, Ekkehard Grünig, and Christina Eichstaedt. "Genetic findings in a family with hereditary spherocytosis, haemolytic anaemia and pulmonary hypertension." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa596.

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Rahim Sipae, Abdulmunaf Abdul, Hassan Al-Moaseeb, Tedimola Olushola, and Debdutta Roy. "8050 Myocarditis and dilated cardiomyopathy as a consequence of parvovirus B19 infection in a patient with hereditary spherocytosis – case study." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference, Glasgow, 26 March 2025 – 28 March 2025. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2025. https://doi.org/10.1136/archdischild-2025-rcpch.536.

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Ivančić, Petra, and Maja Vrdoljak. "325 Aplastic crisis induced by human parvovirus B19 as an initial presentation of hereditary spherocytosis in a child – a case report." In 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.325.

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Li, He, and George Lykotrafitis. "Modeling Diffusion and Vesiculation in Defective Human Erythrocyte Membrane." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14203.

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The hemolytic disorders of hereditary spherocytosis (HS) and hereditary elliptocytosis (HE) affect the lives of millions of individuals worldwide. In HS and HE, connections in the vertical and horizontal directions between components of the RBC membrane (see Fig. 1(a)), are disrupted due to defective proteins, leading to loss of the structural and functional integrity of the membrane (1–2). Moreover, disruptions of either the vertical interactions or horizontal interactions affect the lateral diffusivity of the mobile band 3 proteins, as the motion of band 3 in the RBC membrane is confined by
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