Gotowe bibliografie tematyczne / Stevens Johnson's syndrome

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  3. Książki
  4. Części książek
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Gotowa bibliografia na temat „Stevens Johnson's syndrome”

Autor: Grafiati
Data publikacji: 3 czerwca 2025
Data aktualizacji: 6 lipca 2025

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Artykuły w czasopismach na temat "Stevens Johnson's syndrome"

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Comparin, Cristiane, Günter Hans Filho, Luiz Carlos Takita, Nayara de Castro Wiziack Costa, Roberta Ayres Ferreira do Nascimento, and Lidiane de Oliveira Costa Nanni. "Treatment of toxic epidermal necrolysis with intravenous immunoglobulin: a series of three cases." Anais Brasileiros de Dermatologia 87, no. 3 (2012): 477–81. http://dx.doi.org/10.1590/s0365-05962012000300022.

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Stevens-Johnson's syndrome (SJS) and toxic epidermal necrolysis (TEN) are life-threatening dermatoses, that lead to keratinocyte apoptosis induced by interactions between Fas (cell death receptor) and soluble Fas-ligand, present in serum of Stevens-Johnson's syndrome / toxic epidermal necrolysis patients. Anti-Fas antibodies in intravenous immunoglobulin (IVIG) would block the apoptosis cascade. Three cases of toxic epidermal necrolysis occurred in one male and two female patients, after use of allopurinol, leprosy multidrug therapy concomitant with dipyrone, and diclofenac. The cases were treated with intravenous immunoglobulin 2-3 mg/kg and prednisone 20-50 mg/day. The interruption of new lesions outbreak and reepithelization were extremely fast after the use of intravenous immunoglobulin, without adverse effects. Controlled studies are needed to confirm the efficacy of intravenous immunoglobulin in Stevens-Johnson's syndrome / toxic epidermal necrolysis, but the results seem promising.
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V.Vijaya, Prasad* P.Venkata Sravan Kumar Dr R. Siddarama. "DEATH DUE TO STEVENS JOHNSON SYNDROME; A RARE CASE REPORT." Indo American Journal of Pharmaceutical Sciences 04, no. 07 (2017): 2072–76. https://doi.org/10.5281/zenodo.835176.

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Stevens Johnson’s syndrome is a life threatening drug induced hypersensitivity reaction. The drugs that cause SJS commonly are antibacterials (sulfonamides), anticonvulsants (phenytoin, phenobarbital, and carbamazepine), non-steroidal anti-inflammatory drugs (oxicam derivatives) and oxide inhibitors (allopurinol).Clinical symptoms are urticarial skin eruptions, arthralgia or arthritis, lymphadenopathy and fever.Treatment of Stevens Johnsons Syndrome includes systemic steroids, cyclosporine, intravenous immunoglobulin’s and supportive therapy.A male patient of 58 years was admitted in cardiology unit with chief complaints of skin reactions (Erythema andrashes) all over the body and oral erosions. The patient was diagnosed to have drug induced stevens Johnson’s syndromeandthe suspected drug was amoxicillin potassium clavulanate. By using Naranjo scale and WHO-UMC scale it was found to be a probable ADR and it was managed by drug dechallenge, dexamethasone, dew on ultra-lotion, diprovate ointment. During treatment patientdeveloped severe breathlessness and died.Hence there is a need of clinical pharmacy services to detect, manage and prevent adverse drug reactions. Key words: Stevens Johnson’s syndrome, amoxicillin+potassium clavulanate, Naranjo scale, Dechallenge.
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HAMADA, Yoshiki, Akiko HAMADA, Michihiko KINOSHITA, Norihiko TAKADA, and Kanichi SETO. "A case of Stevens-Johnson's syndrome attributed to Sulpyrine." Japanese Journal of Oral & Maxillofacial Surgery 41, no. 7 (1995): 659–61. http://dx.doi.org/10.5794/jjoms.41.659.

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Jensen, Søren. "A CASE OF STEVENS-JOHNSON'S SYNDROME FOLLOWING ANTIEPILEPTIC MEDICATION." Acta Ophthalmologica 45, no. 4 (2009): 576–81. http://dx.doi.org/10.1111/j.1755-3768.1967.tb06524.x.

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GOMI, Hiroko, Harumi MIMURA, Junko SHIOJIMA, et al. "A Case of Esophagitis Associated with Stevens‐Johnson's Syndrome." Digestive Endoscopy 9, no. 4 (1997): 309–13. http://dx.doi.org/10.1111/j.1443-1661.1997.tb00507.x.

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Tammiraju, Iragavarapu, B. Srinivas, K. Nirupama, and KPruthvi Naresh. "Recurrent cardiac tamponade with stevens–Johnson's syndrome – A rare deadly combo." JOURNAL OF INDIAN COLLEGE OF CARDIOLOGY 11, no. 2 (2021): 82. http://dx.doi.org/10.4103/jicc.jicc_20_20.

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Катилов, А. В. "Principles of diagnostics and intensive therapy of a Stevens-Johnson's syndrome at children." Pain, anesthesia and intensive care, no. 4(69) (December 21, 2014): 77–82. http://dx.doi.org/10.25284/2519-2078.4(69).2014.84670.

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NISHIGUCHI, Hiroaki, Hiroko HAGINO, Shinichi TSURUSAKO, Iwai TOHNAI, Takeshi USAMI, and Minoru UEDA. "A case of Stevens-Johnson's syndrome showing initial symptoms in the oral cavity." Japanese Journal of Oral & Maxillofacial Surgery 49, no. 1 (2003): 19–22. http://dx.doi.org/10.5794/jjoms.49.19.

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Ström, Justus. "OCULAR SYMPTOMS IN FEBRILE MUCOCUTANEOUS REACTIONS (ECTODERMOSIS EROSIVA PLURIORIFICIALIS, STEVENS-JOHNSON'S SYNDROME, MUCOCUTANEOUS-OCULAR SYNDROME ETC.)." Acta Ophthalmologica 44, no. 3 (2009): 411–14. http://dx.doi.org/10.1111/j.1755-3768.1966.tb08051.x.

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Dahake, Shubham, Bibin Kurian, and Archana Maurya. "A Rare Low-Grade Glioma with Stevens Jonson’s Syndrome - Case Report." International Journal of Neonatal Care and Pediatric Nursing 4, no. 2 (2023): 1–7. http://dx.doi.org/10.46610/ijncpn.2023.v04i02.001.

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Introduction: The brain has a complicated structure within the body, but the CNS develops during growth and development in three stages: cytogenesis, histogenesis, and organogenesis, which determine the CNS's final makeup and shape. Organogenesis occurs in children when neural and extra-neural tissues mingle, causing a growing lesion inside the skull. Nearly 20% of the second-most prevalent group of paediatric neoplasms after leukaemia either a benign or malignant primary brain tumour may exist. The majority of infratentorial and close-to-midline brain tumours result in hydrocephalus. Astrocytomas, ependymomas, craniopharyngiomas, and malignant gliomas are the most prevalent supratentorial tumours. Additionally, toxic epidermal necrolysis (TEN) and Stevens Johnson's syndrome (SJS) are now thought to be variations of the same hypertension illness. These conditions affect 1 to 3 million children each year. The degree of epidermal detachment determines the classification; a transitional SJS with less than 10% and 30% epidermal detachment is most frequently confused with Kawasaki illnesses. Case presentation: 9-year-old male child came with complaints of ataxic gait in the past 7 days and vomiting in the past 20 days. As narrated by the father patient was well 7 days back then the patient had difficulty walking due to an ataxic gait, the patient fell on the ground and had a head injury and had no vomiting, fever, loss of consciousness, and no altered sensorium. The patient had 4 episodes of vomiting yesterday and 2 episodes today morning, non-bilious, non-projectile, and containing sputum, patient had a headache that was progressive, intermittent no blurring of vision, not fever but vomiting. The patient then came for further management. Conclusion: The second most prevalent type of paediatric neoplasm after leukaemia, organogenesis in the kid occurs when neural and extra neural tissue combines to form a growing lesion inside the skull either a benign or malignant primary brain tumour may exist. The majority of infratentorial and close-to-midline brain tumours result in hydrocephalus. Additionally, toxic epidermal necrolysis (TEN) and Stevens Johnson's syndrome (SJS) are now thought to be variations of the same hypertension illness. With the development of newer diagnostic methods like MRI, CT, SPECT, PET, and newer electroencephalography techniques as well as evoked potential, it is now possible to accurately diagnose and delineate a variety of neurological alignments. The incidence of these conditions ranges from 1 to 3 million per child lower the risk of morbidity.
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Rozprawy doktorskie na temat "Stevens Johnson's syndrome"

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Chartier, Claire Patey Olivier. "Infection à Mycoplasma pneumoniae et syndrome de Stevens-Johnson." Créteil : Université de Paris-Val-de-Marne, 2006. http://doxa.scd.univ-paris12.fr:80/theses/th0236637.pdf.

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Leclercq, Emmanuel. "Syndrome de stevens-johnson associee a un syndrome de detresse respiratoire de type adulte : un cas pediatrique." Lille 2, 1989. http://www.theses.fr/1989LIL2M392.

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Gaultier, Frédérick. "Contribution à l'étude de la nécrolyse épidermique toxique et du syndrome de Stevens-Johnson." Paris 5, 2006. http://www.theses.fr/2006PA05M001.

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La nécrolyse épidermique toxique et le Syndrome de Stevens-Johnson sont des toxidermies médicamenteuses rares d'étiologies inconnues. Au cours de notre étude nous avons cherché à préciser les mécanismes physiopathogéniques impliqués dans la genèse de ces pathologies. L'histologie a montré une atteinte et une désorganisation modérées de la matrice extracellulaire. L'immunohistochimie et les études biochimiques nous ont permis de mettre en évidence un déséquilibre de la balance MMPs/TIMPs en faveur des MMPs avec un schéma d'expression essentiellement épidermique. Enfin, nous proposons un processus étio-pathogénique compatible avec nos résultats et ceux de la littérature qui permet d'envisager une alternative thérapeutique<br>Toxic Epidermal Necrolysis and Stevens-Johnson Syndrome are rare toxic mucocutaneous drugs reactions withunknown etiologies. In our study, we tried to determine the mechanism involved in these diseases. Histological studies points out few disorganisation of the extracellular matrix. Histochemistry and biochemistry studies led us to evidence a break down of the MMPs/TIMPS balance with more MMPs than TIMPs especially localised in the epidermis. At last, we proposed a theory in accordance with scientific literature and our results in order to get an alternative therapy
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Van, Zyl Lourens Marthinus. "Prevalence of chronic ocular complications in Stevens-Johnson Syndrome and toxic epidermal necrolysis." Master's thesis, University of Cape Town, 2013. http://hdl.handle.net/11427/2899.

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Includes abstract.<br>Includes bibliographical references.<br>The main objective of the study is to identify and grade the severity of chronic ocular complications in patients who were treated for SJS and TEN at Groote Schuur Hospital. The secondary objective is to identify patients who need referrals to specialist ophthalmic clinics for treatable ocular complications of SJS and TEN.
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Wolkenstein, Pierre. "Prédisposition métabolique et mécanisme de mort cellulaire au cours du syndrome de Stevens-Johnson et de la nécrolyse épidermique toxique." Paris 12, 1996. http://www.theses.fr/1996PA120079.

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Le but de notre travail etait d'etudier les predispositions metaboliques et la mort keratinocytaire au cours de la necrolyse epidermique toxique. Nous avons montre que: 1) la necrolyse epidermique toxique aux sulfamides et aux anticomitiaux etait liee a un deficit hautement specifique de la detoxication des metabolites reactifs du medicament responsable, constitutionnel et genetiquement transmis ; 2) un genotype d'acetylation lente est un facteur de risque de la necrolyse epidermique toxique induite par les sulfamides ; 3) le cytochrome p450 3a4 a un role majeur dans la formation des metabolites reactifs de la carbamazepine et est present dans l'epiderme humain ; 4) le mecanisme de mort cellulaire au cours la necrolyse epidermique toxique est apoptotique. En conclusion, la necrolyse epidermique toxique est associee a une predisposition metabolique conduisant a une accumulation des metabolites reactifs du medicament responsable ; chez certains sujets, la formation d'un neoantigene (metabolites-p450) declencherait une cytotoxicite a mediation cellulaire qui conduirait a l'apoptose des keratinocytes
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Poletti, Jabbour Jamil, Rospigliosi Andrés Wiegering, Elías Reneé Pereyra, and Barrera Carmen Cecilia Elías. "Carbamazepine and oxcarbazepine: reflections after an oxcarbazepine-induced Stevens-Johnson syndrome/toxic epidermal necrolysis overlap." Springer International Publishing, 2016. http://hdl.handle.net/10757/609483.

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AMRAOUI, ABDERRAHIM. "L'erytheme polymorphe majeur ou syndrome de stevens-johnson associe a une infection a mycoplasma pneumoniae : une observation pediatrique." Lille 2, 1993. http://www.theses.fr/1993LIL2M149.

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Giron, Sabrina Delverdier Maxence. "L'Érythème polymorphe chez le chien et le chat données bibliographiques récentes /." [S.l.] : [s.n.], 2008. http://oatao.univ-toulouse.fr/1147/1/jan_1147.pdf.

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Cooper, Ryan. "Intravenous Immunoglobulin Use in the Treatment of Toxic Epidermal Necrolysis and Stevens-Johnson Syndrome: A 10-year Retrospective Analysis of Patients of a Single Burn Center." Thesis, The University of Arizona, 2014. http://hdl.handle.net/10150/315845.

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A Thesis submitted to The University of Arizona College of Medicine - Phoenix in partial fulfillment of the requirements for the Degree of Doctor of Medicine.<br>Stevens - Johnson syndrome and Toxic Epidermal Necrolysis Syndrome are rare, but serious conditions affecting skin and mucous membranes that are primarily treated with supportive care. Other more specific therapies have limited evidence to support the benefit of their use; one such treatment is intravenous immunoglobulin (IVIG). The use of IVIG in the treatment of these syndromes remain controversial due to mixed results demonstrated in the literature, and at present is not considered a component of the standard of care. This study seeks to provide additional data regarding the efficacy of IVIG treatment on mortality in a small cohort of patients presenting with these syndromes at a regional burn center over a 10-year period; data was retrospectively collected from patient medical records. On analysis of this data, IVIG use showed a potential, but not significant. improvement on mortality in comparison to the non-treatment group. Compared with the non-treatment group, odds ratios for death were 0.81 (95% CI 0.3-2.0) for IVIG. There is ultimately no new evidence that the benefit of IVIG in the treatment of Stevens - Johnson syndrome and Toxic Epidermal Necrolysis Syndrome is anything more than potential. Further investigation should include a rigorous analysis and comparison of different dosing regimens.
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Tanno, Luciana Kase. "Estudo de associação de fatores genéticos em indivíduos com reações de hipersensibilidade tardia induzida por anticonvulsivantes aromáticos." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/5/5146/tde-01122014-113756/.

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Intrdodução: As terapias com anticonvulsivantes de anel aromático (ACA) são freqüentemente associadas a reações adversas. No entanto, reações de hipersensibilidade (RH) não-imediatas (tardias) a estes fármacos são raras, imprevisíveis e geralmente relacionadas à alta morbidade e mortalidade. Foi demonstrado que estas RH aos ACA estão fortemente associadas ao Antígenio de Leucócitos Humanos (HLA)-B*1502 em pacientes chineses e ao HLA-A*3101 em caucasianos. Polimorfismos de genes do metabolismo do Citocromo P450 (CYP)2C9 foram mais associados a estas reações em pacientes orientais. Objetivo: Nosso objetivo é analisar a associação das reações de hipersensibilidade a anticonvulsivantes de anel aromático com os polimorfismos descritos e de interesse, bem como realizar a tipificação de HLA em uma população de São Paulo, Brasil. Métodos: Estudo tipo caso-controle com genotipagem dos polimorfismos de interesse por reação em cadeia da polimerase (PCR) em tempo real e tificação de HLA A, B, C, DRB, DQA, DQB por PCR seguido de deteção utilizando método LuminexR. A avaliação fenotípica se baseou em sistemas de escores padronizados, utilizando um questionário adaptado da ENDA (Rede Européia de Alergia a Medicamentos), em registros médicos e no acompanhamento clínico. O teste de contato com o medicamento suspeito foi realizado de acordo com as recomendações da ENDA, nos pacientes que apresentaram reação. Resultados: Foram estudados 506 pacientes, 65% do gênero feminino e a idade média foi de 43,6 anos. Oitenta por cento era de etnia mista. Polimorfismos de HLA-A*3101, HLA-B*1502, CYP2C9, CYP2C19 e CYP3A5 foram analisados de 55 indivíduos com reações de hipersensibilidade (RH) a antiepilépticos, de 85 tolerantes e de 366 controles sadios. Dos 55 casos foram validados como RH, 32 apresentaram Reação a Drogas com eosinofilia e sintomas sistêmicos (DRESS), 12 Síndrome de Stevens-Johnson (SSJ) e 11 exantema maculo-papular. De todos os 46 testes de contato com medicamento, 29 (63%) foram positivos, tanto em SSJ como em DRESS. Houve associação significativa entre polimorfismo de HLA-A*1502 e casos. Nenhum de nossos grupos de estudo apresentou associação positiva com polimorfismos de HLAA* 3101. Verificamos uma forte associação entre a atividade normal do CYP3A5 e indivíduos tolerantes quando comparado com casos (p = 0,0002, OR = 4,8). A tipificação de HLA demonstrou associação significante de HLA-A*31, HLA-A*74, HLA-B*35 e HLA-B*53 com reações graves aos ACA e de HLA-B*44 e HLA-C*03 com indivíduos tolerantes. Conclusão: Estes resultados sugere fortemente a existência de fatores genéticos de risco e/ou de proteção a RH a ACA em indivíduos brasileiros, mas não devem ser considerados de forma isolada. Assim, a relevância deste estudo extrapola o objetivo de estudo caso-controle e sugere um modelo como forma de prevenção primária às RH aos ACA.<br>Background: Antiepileptics with aromatic ring (AAR) therapies are frequently associated with adverse reactions. Nevertheless non-immediate (late) hypersensitivity reactions (HR) to these drugs are rare, unpredictable and usually related with high morbidity and mortality. A strong pharmacogenetic association has been reported in Chinese patients with these HR and Human Leukocyte Antigen (HLA)-B*1502 and with HLA- A*3101 in caucasians. Polymorphism of genes of P450 Cytocrome (CYP)2C9 has been related to these reactions in patients of oriental origin. Objective: Our aim is to analyze the association between hypersensitivity reactions due to AAR and the described polymorphisms, as well as perform the typification of HLA in a population of São Paulo, Brazil. Methods: Case-control study genotyping the polymorphisms of interest by polymerase chain reaction (PCR) real time and typifying HLA A, B, C, DRB, DQA, DQB by PCR followed by LuminexR .The phenotype evaluation was based on standardized scoring systems using an adapted ENDA (European Network of Drug Allergy) questionnaire, medical records and on the clinical follow-up in our Allergy Clinic. The patch test with the culprit drug was performed in patients who experienced HR according to the ENDA recommendations. Results: We studied 506 subjects, 65% female and mean age was 43,6 years. Eighty percent had mixed ethnicity. Polymorphisms of HLA-B*1502, HLA- A*3101, CYP2C9, CYP2C19 e CYP3A5 were studied in 55 subjects with antiepileptics HR, 85 tolerants, and 366 control subjects. Of 55 cases were validated as AHR, 32 presented Drug Reaction with Eosinophilia and Systemic Symptoms (DRESS), 12 Stevens-Johnson Syndrome (SJS) and 11 maculopapular exanthema. Of all 46 drug patch tests, 29 (63%) were positive, in both SJS and DRESS. A significant association between polymorphism of HLA-A*1502 and cases was found. None of our study groups presented positive association with HLA-A*3101 polymorphisms. We found a strong association between the normal activity of CYP3A5 and tolerants subjects when compared to HR (p=0.0002, OR=4.8). The HLA typification showed a significant association between HLA-A*31, HLA-A*74, HLAB* 35 e HLA-B*53 and severe AAR reactions and HLA-B*44 and HLA-C*03 in tolerants subjects. Conclusion: These results strongly suggests the existence of genetic risk and/or protective factors to the development of HR to AAR AAR in Brazilian subjects, but it should not be considered in a isolated manner. So, the relevance of this study extrapolates the aim of a case-control study and suggests a system of primary prevention to HR due to AAR
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Książki na temat "Stevens Johnson's syndrome"

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Parker, James N., and Philip M. Parker. Stevens-Johnson Syndrome: A medical dictionary, bibliography, and annotated research guide to internet references. ICON Health Publications, 2004.

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Mehta, Jodhbir, Mayumi Ueta, and Shigeru Kinoshita, eds. Update on Stevens Johnson Syndrome. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-314-8.

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Taghipour, Kathy. Mucosal disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0255.

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This chapter discusses the following mucous membrane disorders: pemphigus vulgaris, lichen planus, and Stevens–Johnson syndrome. Pemphigus vulgaris is an autoimmune disease that affects the skin and the mucosal membranes with blisters and erosions. Lichen planus is a cell-mediated immunological mucocutaneous disease; oral lichen planus may present with erosions, white streaks, or plaques in the oral cavity. Stevens–Johnson syndrome is an emergency dermatological condition in which an immunological hypersensitivity causes erosions and inflammation of mucosal membranes and the skin. As well as providing definitions of these diseases, this chapter discusses their etiology, typical symptoms, uncommon symptoms, demographics, natural history, complications, diagnostic approach, other diagnoses that should be considered, prognosis, and treatment.
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Breisgau, Universität Freiburg im, ed. Letalität und Risikofaktoren der toxisch epidermalen Nekrolyse und des Stevens-Johnson-Syndroms. 1991.

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Publications, ICON Health. Stevens-Johnson Syndrome - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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Schupp, Peter. Die Differentialdiagnosen der schweren Hautreaktionen Erythema exsudativum multiforme majus, Stevens-Johnson-Syndrom und Toxisch epidermale Nekrolyse. 1997.

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Kānsưksā wičhai ʻubatkān kānkœ̄t læ khwāmsamphan kap kānchai yā khō̜ng kānphǣ thāng phiunang nai klum Stevens-Johnson syndrome. Sūn Tittām ʻĀkān ʻAn Mai Phưng Prasong Čhāk Kānchai Yā, Kō̜ng Wichākān, Samnakngān Khana Kammakān ʻĀhān læ Yā, Krasūang Sāthāranasuk, 1999.

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Jolly, Elaine, Andrew Fry, and Afzal Chaudhry, eds. Dermatology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199230457.003.0006.

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Chapter 6 covers the basic science and clinical topics relating to dermatology which trainees are required to learn as part of their basic training and demonstrate in the MRCP. It covers eczema/dermatitis, psoriasis, blistering skin disorders, bacterial and viral infections of the skin, fungal infections and infestations, erythema multiforme, Stevens-Johnson syndrome/toxic epidermal necrolysis, erythema nodosum, drug eruptions, benign skin tumours, malignant skin tumours, the skin and systemic disease, cutaneous lupus erythematosus, systemic sclerosis (scleroderma), vasculitis, structure and function of the skin, hair, and nails, nail disorders, alopecia, hirsutism and hypertrichosis, acne vulgaris, rosacea, hidradenitis suppurativa, disorders of pigmentation, urticaria and angio-oedema, and photosensitivity.
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Banerjee, Ashis, and Clara Oliver. Dermatology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198786870.003.0016.

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Dermatology encompasses a large number of conditions including both primary skin diseases as well as multisystem disease. This chapter covers the pertinent areas of dermatology required for the Intermediate FRCEM examination. It is highly possible that skin conditions could appear in the short-answer question (SAQ) paper and therefore this chapter provides candidates with the tools to describe a rash in terms of nomenclature as well as constructing a differential diagnosis and management plan. This chapter covers the common life-threatening rashes such as Steven-Johnson syndrome, as well as rashes associated with multisystem disease such as erythema nodosum and primary skin conditions.
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MELVIN, Mike. Stevens - Johnson Syndrome Active Remedy: Ultimate Guide on How to Completely Defeat Symptoms Effectively, and Get Your Immediate and Restorative Healing. Independently Published, 2022.

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Części książek na temat "Stevens Johnson's syndrome"

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Siddiqui, Aazim A., and Allen O. Eghrari. "Stevens Johnson Syndrome." In Encyclopedia of Ophthalmology. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-35951-4_594-1.

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Siddiqui, Aazim A., and Allen O. Eghrari. "Stevens Johnson Syndrome." In Encyclopedia of Ophthalmology. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_594.

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Coster, D. J. "Stevens-Johnson Syndrome." In Cicatrising Conjunctivitis. KARGER, 1997. http://dx.doi.org/10.1159/000060696.

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Pollard, Margaret C., Laura M. Le, and Deepinder K. Dhaliwal. "Stevens–Johnson Syndrome." In Corneal Emergencies. Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-5876-1_17.

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Leung, Alexander K. C., Cham Pion Kao, Andrew L. Wong, et al. "Stevens-Johnson Syndrome." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1672.

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Gammanpila, Ranthilaka R., and Eckart Haneke. "Stevens–Johnson syndrome." In Dermatoses in Dark Vs Light Skin. CRC Press, 2025. https://doi.org/10.1201/9781003479512-114.

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Cardon, Zachary E., Colin G. Kaide, and Jason J. Bischof. "Stevens Johnson Syndrome – “Steven Who? And Why I Should Care About His Johnson?”." In Case Studies in Emergency Medicine. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-22445-5_55.

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Abdel-Naser, Mohamed Badawy. "Erythema Multiforme and Stevens-Johnson Syndrome." In Pigmented Ethnic Skin and Imported Dermatoses. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-69422-1_27.

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Yoshida, Satoshi. "Stevens–Johnson Syndrome/Toxic Epidermal Necrolysis." In Challenging Cases in Allergic and Immunologic Diseases of the Skin. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-60761-296-4_6.

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Ferguson, Nkanyezi N. "Stevens-Johnson Syndrome/Toxic Epidermal Necrolysis." In Inpatient Dermatology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-18449-4_4.

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Streszczenia konferencji na temat "Stevens Johnson's syndrome"

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Strukar, Nedim, Verica Mišanović, Adisa Čengić, Aida Karačić, Alma Mujić, and Emina Ribić. "147 Stevens-Johnson syndrome." In 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.147.

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Walsh, B. C. "Chlordiazepoxide Induced Steven-Johnson Syndrome." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6125.

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Stockheim, A., E. Mann, S. Schäd-Trcka, and B. Gerber. "Kasuistik eines Postpartalen Stevens-Johnson-Syndroms." In 28. Deutscher Kongress für Perinatale Medizin. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1607813.

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Ramasli Gursoy, Tugba, Ayse Tana Aslan, Hacer Ilbilge Ertoy Karagol, et al. "A case of Stevens-Johnson Syndrome associated with bronchiolitis obliterans." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3523.

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Gans, A., J. Taylor, and N. N. Goel. "A Case of Drug-Induced Stevens-Johnson Syndrome With Primarily Visceral Involvement." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a5678.

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Sabogal, Carlos, Katarzyna Zarzycki, and Daniel F. Garcia. "Use Of Inflixamab On The Treatment Of Bronchiolitis Obliterans After Stevens-Johnson Syndrome." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a1871.

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Mehdi, N. F., and R. Katz. "Bronchiolitis Obliterans Secondary to Steven Johnson Syndrome, Diagnostic and Therapeutic Challenges." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4931.

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Chaudhary, Himanshi, Dharmagat Bhattarai, Pandiarajan Vignesh, et al. "159 Pediatric onset lupus with stevens johnson syndrome/toxic epidermal necrolysis: an unusual association." In 13th International Congress on Systemic Lupus Erythematosus (LUPUS 2019), San Francisco, California, USA, April 5–8, 2019, Abstract Presentations. Lupus Foundation of America, 2019. http://dx.doi.org/10.1136/lupus-2019-lsm.159.

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Ortí Juan, C., A. Pérez Plasencia, A. Fayet Pérez, et al. "4CPS-154 Stevens–Johnson syndrome in a pregnant woman caused by pyrimethamine and sulfadizine." In 26th EAHP Congress, Hospital pharmacists – changing roles in a changing world, 23–25 March 2022. British Medical Journal Publishing Group, 2022. http://dx.doi.org/10.1136/ejhpharm-2022-eahp.168.

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Sandhya Rani, D. C. "Foresight Gives Eyesight...Amniotic Membrane Transplantation for Steven Johnson Syndrome-Anaesthetic Management." In ISACON KARNATAKA 2017 33rd Annual Conference of Indian Society of Anaesthesiologists (ISA), Karnataka State Chapter. Indian Society of Anaesthesiologists (ISA), 2017. http://dx.doi.org/10.18311/isacon-karnataka/2017/ep086.

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Raporty organizacyjne na temat "Stevens Johnson's syndrome"

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Stewart, Thomas, Hemali Shah, and John Frew. Infectious complications of Stevens Johnson syndrome and toxic epidermal necrolysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.1.0124.

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Stewart, Thomas, Joshua Farrell, and John Frew. Psychological complications of Stevens Johnson syndrome and toxic epidermal necrolysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.1.0097.

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Stewart, Thomas, Jeremy Chan, and John Frew. A systematic review and meta-analysis of Non-SCORTEN mortality predictors in Stevens Johnson syndrome and toxic epidermal necrolysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.4.0005.

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Stewart, Thomas, Nicole Seebacher, and John Frew. A systematic review and meta-analysis of case-control studies on cytokines in blister fluid and skin of patients with Stevens Johnson syndrome and toxic epidermal necrolysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.2.0123.

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Severe Cutaneous Adverse Reactions. A consensus by a CIOMS Working Group. CIOMS, 2025. https://doi.org/10.56759/lrty1600.

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Streszczenie:
In clinical practice, there is mounting concern about the burden of SCAR in relation to novel biologics as well as the increasing cost of diagnosis and management. This consensus report provides unique insights and the latest thinking from renowned experts on this important topic. The skin is among the parts of the body most commonly affected by adverse drug reactions (ADRs). Cutaneous ADRs affect 2% to 3% of all hospitalized patients and have a wide spectrum of clinical manifestations, are caused by various medicinal products, and result from different pathophysiologic mechanisms. Hence, their diagnosis and management are challenging. However, approximately 0.1% to 1% of patients with medicinal product eruptions have serious ADRs which can lead to disabling sequelae and in some cases, fatalities. Although Severe Cutaneous Adverse Reactions (SCAR) are rare, they are a significant health challenge and hinder the safe and effective use of medicines. In short, they pose substantial hurdles to drug developers, medicines regulators and health professionals. SCAR include Stevens-Johnson syndrome (SJS), toxic epidermal necrolysis (TEN), drug reaction with eosinophilia and systemic symptoms (DRESS), acute generalized exanthematous pustulosis (AGEP), and generalized bullous fixed drug eruptions (GBFDE). Premarketing randomized clinical trials have limited power to detect SCAR. There is also a lack of specific diagnostic tests for SCAR, which today, depend on subjective causality assessment methods. These factors highlight the urgent need for guidelines, including how to predict, prevent, detect and diagnose SCAR either during drug development or in the post-marketing phase.
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