Gotowe bibliografie tematyczne / Systemic Auto-Immune Disease

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Gotowa bibliografia na temat „Systemic Auto-Immune Disease”

Autor: Grafiati
Data publikacji: 29 marca 2025

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Artykuły w czasopismach na temat "Systemic Auto-Immune Disease"

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Naha, K., S. Thakare, G. Vivek, and M. Prabhu. "Adenocarcinoma of lung masquerading as systemic auto-immune disease." Case Reports 2012, jun13 1 (2012): bcr0220125822. http://dx.doi.org/10.1136/bcr.02.2012.5822.

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Leslie, R. D. G., and M. Hawa. "Twin Studies in Auto-immune Disease." Acta geneticae medicae et gemellologiae: twin research 43, no. 1-2 (1994): 71–81. http://dx.doi.org/10.1017/s000156600000297x.

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AbstractImmune-mediated diseases affect up to 5% of the population and are a major cause of morbidity and mortality. These diseases can be organ specific, such as insulin-dependent diabetes (IDDM) and non-organ specific, such as Rheumatoid Arthritis (RA). Identical and non-identical twins have been used to establish whether these diseases are determined by genetic or environmental factors. The results of these studies have been collated in a new section of the Mendel Institute in Rome.Diseases included in these studies included IDDM, RA, Systemic Lupus Erythematosus (SLE), Multiple Sclerosis (
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Chen, Yunying, Fredrik Wermeling, Sven Petersen, Ylva Kaiser, and Mikael Karlsson. "Memory to self-antigens in Systemic Lupus Erythematosus (P4020)." Journal of Immunology 190, no. 1_Supplement (2013): 42.11. http://dx.doi.org/10.4049/jimmunol.190.supp.42.11.

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Abstract Systemic Lupus Erythematosus (SLE) is a systemic autoimmune disease associated with auto-IgG antibodies, which are produced due to defective clearance of apoptotic cells (AC) and loss of tolerance to self-antigens. SLE follows flare and remission courses; disease symptoms can come rapidly and unpredictably, suggesting the presence of immune memory to self-antigens in patients. However, experimental evidence and molecular mechanisms behind this are not known. We set up a mouse model by injecting normal mice with syngenic AC weekly for 4 weeks that breaks tolerance and induces auto-IgG
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J.P, Pramod. "Decoding the Mysteries of Auto-Immune Disorders." International Journal for Research in Applied Science and Engineering Technology 12, no. 8 (2024): 1366–71. http://dx.doi.org/10.22214/ijraset.2024.64123.

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An autoimmune disease (AI) happens when the immune system doesn’t recognise its cells. This leads to a problem with how it reacts to these cells. Some of these diseases can be due to your genes, the environment, or even certain infections. They can affect specific organs or the whole body. Examples of autoimmune diseases are insulin-dependent diabetes mellitus, rheumatoid arthritis, systemic lupus erythematosus, scleroderma, thyroiditis, and multiple sclerosis. But wait—there's more! Other conditions like arteriosclerosis, inflammatory bowel disease, schizophrenia, & some types of infertil
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Hsu, Eugene, and Manishi Desai. "Glaucoma and Systemic Disease." Life 13, no. 4 (2023): 1018. http://dx.doi.org/10.3390/life13041018.

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Glaucoma is the leading cause of irreversible blindness in the world. Due to its potential to cause permanent vision loss, it is important to understand how systemic conditions and their respective treatments can be associated with or increase the risk for developing glaucoma. In this review, we examined the literature for up-to-date discussions and provided commentary on glaucoma, its pathophysiology, and associated risk factors. We discuss systemic diseases and the impact, risk, and mechanism for developing glaucoma, including pharmacologically induced glaucoma; inflammatory and auto-immune
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Kamiya, Koji, Megumi Kishimoto, Junichi Sugai, Mayumi Komine, and Mamitaro Ohtsuki. "Risk Factors for the Development of Psoriasis." International Journal of Molecular Sciences 20, no. 18 (2019): 4347. http://dx.doi.org/10.3390/ijms20184347.

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Psoriasis is an immune-mediated genetic skin disease. The underlying pathomechanisms involve complex interaction between the innate and adaptive immune system. T cells interact with dendritic cells, macrophages, and keratinocytes, which can be mediated by their secreted cytokines. In the past decade, biologics targeting tumor necrosis factor-α, interleukin (IL)-23, and IL-17 have been developed and approved for the treatment of psoriasis. These biologics have dramatically changed the treatment and management of psoriasis. In contrast, various triggering factors can elicit the disease in geneti
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Bird, Anna Kathleen, Javier Rangel-Moreno, Nida Meednu, Jennifer Hossler, and Jennifer Anolik. "Neutrophils modulate the progression of B cell auto-reactivity in systemic lupus erythematosus." Journal of Immunology 196, no. 1_Supplement (2016): 117.3. http://dx.doi.org/10.4049/jimmunol.196.supp.117.3.

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Abstract The central feature of systemic lupus erythematosus (SLE) pathogenesis is loss of immunologic self-tolerance. Here, we address a current controversy regarding whether neutrophils influence the development of B cell auto-reactivity SLE. While both human and murine lupus neutrophils produce factors known to fuel adaptive immune dysregulation, including type I interferon, B cell survival factors, and extracellular traps (a putative source of self-antigen), some studies have contended that neutrophils in peripheral lymphoid organs (PLO) suppress B cell activation and differentiation. In o
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Hossain, Md Ismail, Shah Md Sarwer Jahan, Md Ashraful Haque, ABM Mobasher Alam, Mainuddin Ahmed, and Md Zakir Hossain. "Systemic lupus erythematosus in male: two case reports." Bangladesh Journal of Medicine 24, no. 2 (2014): 82–85. http://dx.doi.org/10.3329/bjmed.v24i2.20222.

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Systemic lupus erythematosus (SLE) is the most common multisystem connective tissue disease. Around 90% of affected individuals are women, with peak onset in the second and third decades. Tissues of all system are damaged by pathogenic auto-antibodies and immune complexes. We report here two cases of SLE in male patient, presented with typical features of SLE. Though the disease is rare in male, but such type of manifestations should be investigated properly to exclude SLE. DOI: http://dx.doi.org/10.3329/bjmed.v24i2.20222 Bangladesh J Medicine 2013; 24 : 82-85
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Yang, Yening. "The comparison between different therapies of Systemic Lupus Erythematosus." Theoretical and Natural Science 49, no. 1 (2024): 9–15. http://dx.doi.org/10.54254/2753-8818/49/20241257.

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Abstract. The immune system has three basic functions: defense, surveillance and homeostasis. It helps us monitoring and timely eliminate mutant cells and distinguishing between alien components to maintain self-stability. However, once it loses these abilities, it could cause the autoimmune disease. In this article, I mainly discuss about the Systemic Lupus Erythematosus (SLE), a type of autoimmune diseases which is mediated by type hypersensitivity. SLE patients suffer from chronic inflammatory responses, leading to tissue damage and organ dysfunction. Therefore, purpose of therapeutic sched
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Hudspeth, Kelly L., Wang Shu, Jingya Wang, et al. "NK cell phenotype and proliferation in Systemic Lupus Erythematosus." Journal of Immunology 196, no. 1_Supplement (2016): 194.5. http://dx.doi.org/10.4049/jimmunol.196.supp.194.5.

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Abstract Systemic Lupus Erythematosus (SLE) is a complex autoimmune disorder whose pathology appears to involve many immune cell types. While it is clear that autoantibody producing B cells as well as CD4+ T cell help are key contributors to disease, little is known regarding the role of innate lymphoid cells such as Natural Killer (NK) cells in the pathogenesis of SLE. We have characterized the phenotype of NK cells by multicolor flow cytometry in a large cohort of SLE patients. While the overall percentage of NK cells was similar or slightly decreased compared to healthy controls, a subset o
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Rozprawy doktorskie na temat "Systemic Auto-Immune Disease"

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Depaire, Agathe. "Altérations de l’efferocytose des macrophages induits par les cellules endothéliales : analyse des mécanismes et approche thérapeutique pour corriger la vasculopathie et la fibrose au cours de la sclérodermie systémique." Electronic Thesis or Diss., Bordeaux, 2024. http://www.theses.fr/2024BORD0481.

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La sclérodermie systémique (ScS) est une maladie auto-immune fibrotique chronique incurable. Le concept de réparation tissulaire non résolue, menant à une fibrose persistante, a émergé sur la base d'une inflammation stérile chronique qui transforme une réponse de réparation contrôlée en fibrose pathologique. La résolution efficace de l'inflammation repose notamment sur l’élimination des cellules apoptotiques par les macrophages (Mϕ) via l’efferocytose. Récemment, mon équipe a montré le rôle de la stimulation des cellules endothéliales microvasculaires (CEMV) cutanées par l’IL-1β dans la modula
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Nguyen, Vinh. "Développement d’un nouveau modèle murin expérimental de sclérodermie." Thèse, 2016. http://hdl.handle.net/1866/13888.

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La sclérodermie (SSc) est une maladie rare affectant les personnes génétiquement prédisposées d’une réponse immunitaire défectueuse. Malgré les derniers avancements et développements dans le domaine, l’étiologie et la pathogénèse de la maladie demeurent peu comprises. Par ailleurs, il y a un ralentissement dans la compréhension de cette maladie à cause du manque de modèle animal représentatif de la SSc humaine. Malgré plusieurs lacunes, les souris traitées avec la bléomycine ou portant des modifications génétiques (TSK-1) sont très utilisées dans les études précliniques de la SSc mais elles ne
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Książki na temat "Systemic Auto-Immune Disease"

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Berden, Jo H. M., and Jack F. M. Wetzels. Immunological investigation of the patient with renal disease. Edited by Christopher G. Winearls. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0017.

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Laboratory techniques (electrophoresis, indirect immunofluorescence, ELISA, and immunoblotting) required for immunological investigation of the patient with renal disease are described. Renal disease-related aspects of immunoglobulins (immunoglobulin A, paraproteins, cryoglobulins), complement, antinuclear antibodies, anti-C1q antibodies, antineutrophil cytoplasmic antibodies, anti-glomerular basement membrane antibodies, antipodocyte antibodies, antiphospholipid antibodies, and antimicrobial responses (streptococci, hepatitis C, hepatitis B) are reviewed. Laboratory assays which evaluate the
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Multi-systemic auto-immune diseases: An integrated approach : dermatological and internal aspects. Elsevier, 1995.

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Kater, Louis, and Harold Baart De LA Faille. Multi-Systemic Auto-Immune Diseases: An Integrated Approach : Dermatological and Internal Aspects. Elsevier Science Pub Co, 1999.

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Klenerman, Paul. The Immune System: A Very Short Introduction. Oxford University Press, 2017. http://dx.doi.org/10.1093/actrade/9780198753902.001.0001.

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The Immune System: A Very Short Introduction describes the immune system and how it works in health and disease. It focuses on the human immune system, considering how it evolved, and the basic rules that govern its behaviour. The immune system comprises a series of organs, cells, and chemical messengers that work together as a team to provide defence against infection. These components are discussed along with the critical signals that trigger them and how they exert their protective effects, including innate and adaptive responses. The consequences of too little immunity (immunodeficiency),
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Dalbeth, Nicola. Pathophysiology of gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0039.

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The clinical features of gout occur in response to monosodium urate (MSU) crystals. Gout should be considered a chronic disease of MSU crystal deposition. A number of pathophysiological checkpoints are required for development of gout. First, elevated urate concentrations are required: urate overproduction and underexcretion contribute to total urate balance. Overproduction occurs due to alterations in the purine synthesis and degradation pathways. Renal underexcretion is an important cause of elevated serum urate concentrations (hyperuricaemia), and occurs through alterations in the urate tra
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Części książek na temat "Systemic Auto-Immune Disease"

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Greco, Raffaella, and Dominique Farge. "CART Cells and Other Cell Therapies (ie MSC, Tregs) in Autoimmune Diseases." In The EBMT Handbook. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-44080-9_93.

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AbstractAuto-immune diseases (AD) are heterogeneous conditions, characterized by polyclonal activation of the immune system with a defect of B or T lymphocyte selection and altered lymphocytic reactions to auto-antigens components (Burnet 1959a, b), although it is rare to identify a single antigenic epitope. The native immune system and its tissue environment play an important role to determine if exposure to a given antigen will induce an immune response or tolerance or anergy. The role of the genes coding for the major histocompatibility system molecules, but also of many other genes, is important in the regulation of the immune response, although this does not explain all the observed phenomena during loss of tolerance (Matzinger 1994; Rioux and Abbas 2005).
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Hall, Angela, Chris Scott, and Matthew Buckland. "Autoimmune skin disease." In Clinical Immunology. Oxford University Press, 2016. http://dx.doi.org/10.1093/hesc/9780199657650.003.0008.

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This chapter looks at autoimmune skin diseases such as pemphigoid and pemphigus. It notes how auto-antibodies are directed against protein at the dermal-epidermal junction in the pemphigoid, while pemphigus has auto-antibodies directed against cell junctions in the epidermis. The chapter indicates that specific auto-antibodies are associated with disruption of the skin structure leading to blistering. Biopsies or serums can demonstrate the presence of auto-antibodies as well. The chapter shows how treatment can include steroid, topic, systemic, steroid-sparing systemic immune suppression, high dose intravenous immunoglobulin, plasmapheresis in conjunction with systemic immune suppression, or monoclonal antibody therapy targeting B lymphocytes. In addition, it references other autoimmune blistering skin disorders such as dermatitis herpetiformis and toxic epidermal necrolysis.
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Monroy-Trujillo, Jose Manuel, and Duvuru Geetha. "Systemic Inflammatory Diseases and the Kidney." In Kidney Protection, edited by Vijay Lapsia, Bernard G. Jaar, and A. Ahsan Ejaz. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190611620.003.0033.

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The kidneys are targets of systemic autoimmunity as well as pathogenic immune responses against renal auto antigens. Systemic autoimmunity against ubiquitous antigens leading to renal inflammation is seen in antineutrophil cytoplasmic antibodies (ANCA) associated vasculitis, immunoglobulin (Ig) A vasculitis, systemic lupus erythematosis, scleroderma and IgG4 disease while immune responses against specific renal antigens is seen in antiglomerular basement membrane disease. Renal involvement can be the presenting feature in these diseases and can manifest as a rise in serum creatinine or asymptomatic urinary abnormalities or can present with rapidly progressive renal failure. For the majority of systemic inflammatory disorders, renal involvement heralds a poor prognosis and warrants timely initiation of immunosuppressive therapy. This chapter will review the clinical, laboratory, and histologic features and discuss management of renal disease associated with ANCA-associated vasculitis, IgA vasculitis, antiglomerular basement membrane disease, polyarteritis nodosa, scleroderma, and IgG4 disease.
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Shimpi, Pooja, Smita Pillewan, and Vandana S. Nikam. "Diseases and Disorders Associated with Immune System." In Natural Immunomodulators: Promising Therapy for Disease Management. BENTHAM SCIENCE PUBLISHERS, 2023. http://dx.doi.org/10.2174/9789815123258123010005.

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The human immune system is one of the complex systems of the body, which works against both external and internal invasion. It has two parts: the innate and the acquired immune systems. We have been born with the innate system which gives a quick response for the invading pathogen non-specifically. To deal with the typical environmental antigens, immune system adapts to changes. The acquired (or adaptive) component develops over time and produces antibodies that “remember” invaders to fight them if they return. Failure of it could be due to genetic defect (weak natural immunity), inability to adapt to the change, hyper-responsiveness, or inability to distinguish self from foreign, leading to various diseases and disorders. Various genetic defects of the immune system are at the core of Primary Immune disorders (PIDs), while overactivity is responsible for allergic diseases. Autoimmune diseases are mostly due to malfunction of the adaptive immune system, while in Systemic Auto-inflammatory Disorders (SAIDs), the innate immune system is affected. Advancements in technology and genetics have improved our understanding of the pathogenesis, diagnosis, and management of these diseases.
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Reséndiz-Mora, Albany, Alonso Tescucano, Giovanna Barrera-Aveleida, et al. "Anti-Non-Bilayer Phospholipid Arrangement Antibodies Trigger an Autoimmune Disease Similar to Systemic Lupus Erythematosus in Mice." In Systemic Lupus Erythematosus - Pathogenesis and Management [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.106373.

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Anti-lipid antibodies are present in some infectious and autoimmune diseases, such as Systemic Lupus Erythematosus (SLE). Particularly, anti-non-bilayer phospholipid arrangement (NPA) antibodies have been detected in patients with SLE, and these antibodies trigger a disease similar to human lupus in mice. NPA are lipid associations different from the lipid bilayer of cellular membranes and, since they are transient, they are not immunogenic. However, if NPA are stabilized by drugs, they induce an immune response with the production of anti-NPA antibodies, which bind to NPA on cell membranes and generate cell lysis. As a result, intracellular antigens are exposed and trigger an immune response that generates more auto-antibodies. In this chapter, we describe the formation and stabilization of NPA, the induction of B cell responses to generate anti-NPA antibodies, and the characteristics that the disease caused by these antibodies in mice shares with human lupus.
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Vanditha, Mohan, Sonu Das, and Mathew John. "Lipid Metabolism and Associated Molecular Signaling Events in Autoimmune Disease." In Fatty Acids - Recent Advances [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.105746.

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Lipid metabolism, when dysregulated paves the way to many autoimmune disease conditions. One such recently explored mechanism was that of Liver X receptor (LXR) signaling which acts as a molecular link between lipid metabolism and inflammation. LXR plays a critical role in coupling immune cell lipid homeostasis with systemic immune responses. In this chapter, we will discuss how an altered lipid metabolite environment causes inflammation signaling via LXR-mediated molecular events which could lead to autoimmune disease. In a hyperlipidemic environment, Interferon regulatory factor 3(IRF3) mediated downregulation of LXR signaling in innate immune cells leading to an inflammatory auto-immune response. Meanwhile, dendritic cell-mediated cytokine generation amidst LXR downregulation leads to the differentiation of autoreactive T cells and B cells, conferring an autoimmune response. Recent advances in the therapeutic management of autoimmune diseases target specific metabolic events as a strategy to limit inflammation and the autoimmune outcome. Novel treatment regimes in autoimmune diseases featuring lipid metabolic pathways are also discussed.
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Martínez-Jiménez, Santiago. "Introduction to Connective Tissue Disorders and Autoimmune Conditions." In Chest Imaging. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780199858064.003.0058.

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A variety of pathologic processes affect the connective tissues. The chapters that follow this one address some of these conditions with special emphasis on autoimmune disorders (e.g. rheumatoid arthritis, scleroderma, systemic lupus erythematosus, dermatomyositis/polymyositis, Sjögren syndrome, mixed connective tissue disease, and vasculitis), amyloidosis and eosinophilic lung disease. An autoimmune response is an immune response that targets an antigen within the host and attacks healthy body tissues. This often involves T and B lymphocytes in a response that is very similar to an allergic reaction. While the presence of an auto-antibody is often an essential component of the autoimmune response, its mere presence does not define the disease. In addition to the presence of auto-antibodies, the presence of soft tissue damage is required, thus auto-antibodies may be present in the absence of an inflammatory process. Amyloidosis is a pathologic entity with protean manifestations, often thought of as a single disease rather a group of diseases that share a similar pathophysiologic event: the deposition of proteins within the soft tissues. The term eosinophilic lung disease encompasses a wide variety of pathologic conditions that range from idiopathic diseases to systemic vasculitides and, in some cases, a response to parasitic infestation.
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Pathak, Soumya, and Dr Shambaditya Goswami. "ANTIPSORIATIC GEL FORMULATIONS: AN UPDATED WRITE UP." In Futuristic Trends in Pharmacy & Nursing Volume 3 Book 17. Iterative International Publishers, Selfypage Developers Pvt Ltd, 2024. http://dx.doi.org/10.58532/v3bkpn17p5ch2.

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Topical monotherapy in patients with mild to moderate psoriasis has proved to be practically constructive along with their adjuvant role in moderate to severe psoriasis under treatment with systemic medications and phototherapy. However mild psoriasis can usually be controlled by topical therapies exclusively. Therapeutic armamentarium of topical drug delivery especially gel formulations for such debilitating auto-immune disease is ever expanding aiming at increasing patient acceptability and adherence to treatment regimen which is likely to improve ease of formulation application and its outcome based upon selection of drugs, vehicles and frequency of application. The review emphasizes on various gel formulations engaged in topical mono and/or combined therapy in treatment of psoriasis.
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Pandya, Purvi M., Ekta N. Jayswal, and Yash Shah. "Spread of Tuberculosis Among Smokers." In Mathematical Models of Infectious Diseases and Social Issues. IGI Global, 2020. http://dx.doi.org/10.4018/978-1-7998-3741-1.ch003.

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Smoking tobacco has some hazardous implications on an individual's physical, physiological, and psychological health; health of the passive smokers near him or her; and on the surrounding environment. From carcinomas to auto-immune disorders, smoking has a role to play. Therefore, there arises a need to frame a systemic pathway to decipher relationship between smoking and a perilous disease such as tuberculosis. This research work focuses on how drugs or medications can affect individuals who are susceptible to tuberculosis because of smoking habits and also on individuals who have already developed symptoms of tuberculosis due to their smoking addiction. The mathematical model is formulated using non-linear ordinary differential equations, and then threshold is calculated for different equilibrium points using next generation matrix method. Stability analysis along with numerical simulations are carried out to validate the data.
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Bhowmik, Diptendu, Venu Kola, Subba Rao Chamakuri, and Chiranjib Bhattacharjee. "Rheumatoid Arthritis: Introduction." In Natural Products for the Management of Arthritic Disorders. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/9789815050776122010003.

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Rheumatoid Arthritis elaborated as Rheumatoid Arthritis is a systemic chronic inflammatory condition that might affect numerous organs and tissues in a human body, but mainly it attacks the synovial joints. These methods result in the inflammatory synovitis (synovium) response. Factors that lead to an increase in the risk of rheumatoid arthritis are age, sex, family history, smoking, obesity, and exposure to pollutants. RA holds the ability to put a person at a higher risk of developing other medical conditions if it is not controlled timely. A syndrome named carpal tunnel is another common condition found in people suffering from rheumatoid arthritis. Trauma, infection, smoking cigarettes are some of the examples of external triggers which can trigger the reaction of the auto-immune system, which results in chronic joint inflammation and synovial hypertrophy in addition to a potential of other manifestations, which will be theorized for going on in people prone genetically. The pathological process of the disease usually results in destructing the articular cartilage as well as the joints ankylosis. Rheumatoid Arthritis could also result in diffusive lung inflammation, sclera, pleura, pericardium, as well as nodular lesions, which are also common in subcutaneous tissue. However, the causes of RA are still not known, the autoimmunity holds an important part in both the progression and chronicity. Rheumatoid Arthritis is a systemic disorder. RA can be prevented by using coldwater, living fish oil, from the herring, cod, mackerel, and salmon fish, which contains omega-3 fatty acids in high amounts, Vitamin-D supplements, and adopting the lifestyle modifications such as avoiding smokingand weight loss.
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Streszczenia konferencji na temat "Systemic Auto-Immune Disease"

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Stemeier, K., J. Mertin, J. Pill, and F. Hartig. "EFFECTS OF THROMBOXANE RECEPTOR BLOCKER BM 13.505 ON THE DEVELOPMENT OF PROTEINURIA IN AUTOIMMUNE NZB/W MICE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643757.

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Female F1 hybrid of New Zealand black and white mice(NZB/W) spontaneouslydevelop an autoimmune disease characterize by afatal immune complex glomerulonephritis.Theyare considered to be a relevant model of human systemic lupus erythematosus. We observeda doubling of the concentration of TXB2 in urine at the same time when onset of proteinuria was noticed. This suggests that TXA2 synthetized by mesangial and epithelial cells of the glomeruli as well as by some inflammatory cells and platelets might be an important mediator in the pathogenesis of thi auto immune-mediated glomerular disease. Weuse
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Kechida, M., R. Mesfar, R. Klii, S. Hammami, and I. Khochtali. "PS6:128 Influence of associated auto immune diseases in systemic lupus erythematosus." In 11th European Lupus Meeting, Düsseldorf, Germany, 21–24 March 2018, Abstract presentations. Lupus Foundation of America, 2018. http://dx.doi.org/10.1136/lupus-2018-abstract.171.

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Adil Mansoor, AL-Husnah. "Role of Interleukin-35 In The Pathogenesis of Hashimoto’s Thyroiditis Disease." In X INTERNATIONAL CONGRESS OF PURE AND APPLIED TECHNOLOGICAL SCIENCES. Rimar Academy, 2023. http://dx.doi.org/10.47832/minarcongress10-1.

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The immune system has the ability to distinguish between self and non-self. Also, Regulatory T-cells are a portion of Tcells that are in charge of preserving regulation of the immune system and immune tolerance through active immunerepression. Immune tolerance is very important to prevent the immune system from interacting against itself and thus to avoid the development of autoimmune diseasesInterleukin-35 (IL-35), which is a member of the interleukin-12 (IL12) cytokine family, is an anti-inflammatory immune response that plays the important function in the obstruction of the development of a
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Scherlinger, M., J. Lutz, J. Sibilia, E. Chatelus, and M. E. Truchetet. "SAT0499 Association between systemic sclerosis and other systemic auto-immune diseases: study in two university hospitals cohorts." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.2227.

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Scherlinger, Marc, Philippe Mertz, Flora Sagez, et al. "O5 Worldwide trends in all-cause mortality of auto-immune systemic diseases between 2001 and 2014." In 12th European Lupus Meeting. Lupus Foundation of America, 2020. http://dx.doi.org/10.1136/lupus-2020-eurolupus.19.

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Bussel, J. "FOR MODULATION AS A MEANS OF ELEVATING THE PLATELET COUNT IN ITP." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644761.

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ITP is an autoantibody-mediated disease which would logically be treated by decreasing the level of autoantibody. However, the most exciting developments in understanding the pathophysiology of the thrombocytopenia and its treatment involve a better understanding of the MPS FcR system and ways in which it can be modulated. This work has focussed on phagocytic paralysis or FcR blockade (FcRBl): the slowing of destruction of antibody-coated platelets despite the persistent presence of antibody on the surface of the platelet.Several areas have been explored in learning about the MPS system. Inves
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Raporty organizacyjne na temat "Systemic Auto-Immune Disease"

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LI, jianhong, Zhuang LI, Yalin SHE, and Guohua LIN. Assessment of acupuncture for treating herpes zoster:a protocol for an umbrella systematic review and meta analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.4.0010.

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Review question / Objective: Patients who suffer from HZ in line with the consensus of Chinese experts will be included, regardless of sex, race and time of onset . Those who diagnosed with PHN, auto-immune diseases, pregnant women will be excluded.Acupuncture, electroacupuncture, fire needle, skin acupuncture, plum blossom needle, auriculo-acupuncture all these such therapies in treating herpes zoster will be included.The control group’s treatment includes drug therapy (such as antiviral acyclovir nutritional nerve medicine or traditional Chinese medicine, etc.) ，sham acupuncture, placebo, no
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Wongpiyabovorn, Jongkonnee, Nattiya Hirankarn, Yingyos Avihingsanon, Tewin Tencomnao, Yong Poovorawan, and Kriangsak Ruchusatsawat. The association between immunogenetics and genetic susceptibility of psoriasis in Thai population. Chulalongkorn University, 2006. https://doi.org/10.58837/chula.res.2006.27.

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Psoriasis is T-cell-mediated skin autoimmunity, required environmental triggers and genetic susceptibility factors to become manifested. Psoriasis is a chronic skin disease characterized by the abnormal hyperproliferation and differentiation of the epidermis, elongated and prominent blood vessels and a thick perivascular lymphocytic infiltrate. Vascular endothelial growth factor (VEGF) gene play important role in pathogenesis of various diseases with angiogenic basis such as breast cancer and autoimmune disease including psoriasis. Many studies analyzed the association of VEGF gene polymorphis
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