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Rozprawy doktorskie na temat „TGIFLX”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Ayyar, Savita. "Analysis of TGIF function in Drosophila." Thesis, University of Cambridge, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.620360.

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2

Demange, Christine. "Mécanismes de régulation de la protéine TGIF." Paris 6, 2008. http://www.theses.fr/2008PA066032.

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Le TGIF (TG Interacting Factor) est un répresseur de différents facteurs transcriptionnels qui sont impliqués dans la prolifération cellulaire et l’apoptose. J’ai étudié l’implication du TGIF dans l’apoptose induite par le TNF-alpha(Tumor Necrosis Factor alpha). Mes résultats montrent que la surexpression du TGIF entraîne une augmentation de l’apoptose suite à un traitement au TNF-alpha. L’ubiquitine ligase E3 AIP4/Itch (Atrophin-1 Interacting Proteine 4) est connue pour son rôle de médiateur apoptotique car elle provoque la dégradation de la protéine anti-apoptotique c-FLIP via le protéasome.

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3

Krüger, Melanie [Verfasser]. "Die Rolle von TGIF1 in der peripheren T-Zellaktivierung und Differenzierung / Melanie Krüger." Berlin : Freie Universität Berlin, 2012. http://d-nb.info/1027815316/34.

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4

Santos, Tatiana Nayara Libório dos. "Expressão de variantes transcricionais do gene Homeobox TGIF1 em carcinomas epidermóides de boca." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-09042009-115621/.

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Genes da família homeobox têm sido alvo de intensas pesquisas científicas relacionadas ao câncer. Recentemente, mostramos que o gene homeobox TGIF1 está expresso no carcinoma epidermóide de boca na sua forma genérica. Porém, não foi feita uma discriminação entre quais variantes transcricionais, ou subtipos, do TGIF1 estariam expressas. Neste estudo, propusemo-nos a verificar diferenças na freqüência de expressão das variantes transcricionais do TGIF1 em carcinomas epidermóides de boca (CEB) em relação a tecidos morfologicamente não tumorais (TN), avaliar possíveis associações entre essas varia

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5

Maeda, Miki [Verfasser], and Eric [Akademischer Betreuer] Hesse. "Role of Tgif1 in osteoclast differentiation and bone resorption / Miki Maeda ; Betreuer: Eric Hesse." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2019. http://d-nb.info/1183262426/34.

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6

Matthies, Levi [Verfasser], and Eric [Akademischer Betreuer] Hesse. "The Role of Tgif1 in Bone Anabolic Signal Transduction / Levi Matthies ; Betreuer: Eric Hesse." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2018. http://d-nb.info/116227526X/34.

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7

Suñol, Moreno David. "Structural studies of recombinant TGIF1 and FBP28 WW domains using NMR and peptide ligation strategies." Doctoral thesis, Universitat de Barcelona, 2016. http://hdl.handle.net/10803/400299.

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The present thesis is divided in three different but related projects. In the first project, the interaction between TGIF1 and SMAD proteins is investigated. TGIF1 is a transcriptional suppressor that prevents the gene transcription due to its interaction with DNA and protein suppressor complexes. TGIF1 has a role in different signalling pathways such as retinoid, Wnt or TGF-β. Focusing on TGF-β, this is one of the main signalling pathways that regulates a plethora of functions in metazoans, including cell differentiation, proliferation and tissue homeostasis, among many others. SMAD proteins

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8

Rocha, Ana Laís Bignotto da. "Sequenciamento direto dos genes SIX3, SHH, TGIF1, ZIC2 e array-CGH no estudo de pacientes com holoprosencefalia." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/61/61132/tde-12112013-150520/.

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Objetivos: Analisar por meio da técnica de sequenciamento direto a presença de alterações moleculares nos genes SHH, SIX3, ZIC2 e TGIF1 em indivíduos com diagnóstico clínico de HPE. Analisar por meio da técnica de CGH-array a presença de alterações moleculares em indivíduos com diagnóstico clínico de HPE previamente submetidos à análise por sequenciamento direto. Local: Laboratório de Genética e Citogenética Humana HRAC/USP, Bauru-SP. Casuística e metodologia: Foram selecionados 50 indivíduos, de ambos os sexos com idades entre 03 meses a 50 anos com diagnóstico clínico para HPE. Todos foram a

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9

Lasse, Samuel. "Tgif and Smad2 mutations increase mammalian sensitivity to retinoic acid teratogenesis." Connect to this title online, 2005. http://hdl.handle.net/1811/3543.

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Thesis (Honors)--Ohio State University, 2005.<br>Title from first page of PDF file. Document formattted into pages: contains vi, 43 p.; also includes graphics. Includes bibliographical references (p. 41-43). Available online via Ohio State University's Knowledge Bank.

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10

Carrel, Tessa Lyn. "The use of the spontaneous Bn mouse mutant and targeted alleles of Smad2 and Tgif to understand axial specification and neural development." Connect to this title online, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1085415535.

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Thesis (Ph. D.)--Ohio State University, 2004.<br>Title from first page of PDF file. Document formatted into pages; contains xiv, 111 p.; also includes graphics (some col.). Includes bibliographical references (p. 98-111). Available online via OhioLINK's ETD Center

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11

Faresse, Nourdine. "Identification de PCTA, un nouvel effecteur de la voie de signalisation du TGF-beta." Paris 6, 2007. http://www.theses.fr/2007PA066428.

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La stimulation des cellules au TGF- entraîne l’assemblage des protéines cytoplasmiques Smad2/3 avec Smad4. Ce complexe activé se transloque dans le noyau et régule la transcription des gènes cibles du TGF-. Dans le noyau, le complexe Smad peut soit recruter un co-répresseur tel que TGIF. Le TGIF peut inhiber la transcription dépendante du TGF- par différents mécanismes. Récemment, il a été démontré que le TGIF est capable de séquestrer la protéine cytoplasmique cPML dans le noyau inhibant ainsi la phosphorylation de Smad2/3. Dans cette étude, nous avons identifié un nouveau partenaire du TG

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12

Antonio, Luciana Fasanella Matizonkas. ""Análise da presença de transcritos dos genes HOXA7, HOXC6 e TGIF em carcinomas epidermóides de boca"." Universidade de São Paulo, 2006. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-09032006-161432/.

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Os genes homeobox são uma família de genes reguladores que são vitais para vários aspectos do crescimento e diferenciação celular. Recentemente, implicações dos genes HOXA7, HOXC6 e TGIF na gênese e progressão tumoral vêm sendo verificadas. Entretanto, o envolvimento desses genes em carcinomas epidermóides (CE) de boca ainda não foi demonstrado. A possível presença de transcritos dos genes HOXA7, HOXC6 e TGIF em carcinomas epidermóides de boca e em tecidos não tumorais adjacentes (TN) foi analisada. Os transcritos dos genes foram amplificados por RT-PCR e sua localização celular determinada po

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13

Powers, Shannon Elizabeth. "Regulation of development by Tgifs." 2009. http://wwwlib.umi.com/dissertations/fullcit/3393535.

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14

Fang-ChengSu and 蘇芳正. "Involvement of TGIF in a model of traumatic brain injury." Thesis, 2012. http://ndltd.ncl.edu.tw/handle/77073735544450020882.

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碩士<br>國立成功大學<br>醫學檢驗生物技術學系碩博士班<br>100<br>Traumatic brain injury (TBI), the leading cause of death and disability in young children and adults, is a highly complex disorder, including neuroinflammation, blood-brain barrier disruption, raise of oxidative stress, and other physiological alterations. Collectively, these effects result in the secondary damage, including neurodegeneration, apoptosis or autophagy of neurons, as well as neurological impairment. TG-interacting factor (TGIF) is a transcriptional co-repressor to inhibit TGF-β signaling pathway which plays a role of protector to maintain

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15

Mar, Lynn. "Role of TGIF in cell cycle control and establishment of laterality." Thesis, 2006. http://hdl.handle.net/2429/18552.

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Holoprosencephaly (HPE) is the most common structural anomaly of the human brain, resulting from incomplete cleavage of the developing forebrain during embryogenesis. Haploinsufficient mutations in TG-Interacting Factor (TGIF) were previously identified in a subset of HPE families and sporadic patients, and this gene is located within a region of Chromosome 18 that is associated with non-random chromosomal aberrations in HPE patients. TGIF is a transcription factor that contains a three amino acid loop extension (TALE) homeodomain and functions both as a co-repressor of the TGF-β pathway and a

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16

Pei-ChiChen and 陳佩祺. "Reactive oxygen species are involved in TGIF-regulatedmigration of bladder cancer cells." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/92974317642412105356.

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17

""Análise da presença de transcritos dos genes HOXA7, HOXC6 e TGIF em carcinomas epidermóides de boca"." Tese, Biblioteca Digital de Teses e Dissertações da USP, 2006. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-09032006-161432/.

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