Gotowe bibliografie tematyczne / Tumor Necrosis Factor/metabolism

Spis treści

  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
  6. Raporty organizacyjne

Gotowa bibliografia na temat „Tumor Necrosis Factor/metabolism”

Autor: Grafiati
Data publikacji: 2 czerwca 2025
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Tumor Necrosis Factor/metabolism"

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Kaji, Keisuke, Rei Katogi, Yoshiaki Azuma, Asuka Naito, Jun-Ichiro Inoue та Akira Kudo. "Tumor Necrosis Factor α-Induced Osteoclastogenesis Requires Tumor Necrosis Factor Receptor-Associated Factor 6". Journal of Bone and Mineral Research 16, № 9 (2001): 1593–99. http://dx.doi.org/10.1359/jbmr.2001.16.9.1593.

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Balkwill, Fran. "Tumor necrosis factor or tumor promoting factor?" Cytokine & Growth Factor Reviews 13, no. 2 (2002): 135–41. http://dx.doi.org/10.1016/s1359-6101(01)00020-x.

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Wu, Shanshan, Kai Dong, Jiajia Wang, and Yaxin Bi. "Tumor necrosis factor alpha improves glucose homeostasis in diabetic mice independent with tumor necrosis factor receptor 1 and tumor necrosis factor receptor 2." Endocrine Journal 65, no. 6 (2018): 601–9. http://dx.doi.org/10.1507/endocrj.ej17-0539.

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Holeček, M. "Leucine metabolism in fasted and tumor necrosis factor-treated rats." Clinical Nutrition 15, no. 2 (1996): 91–93. http://dx.doi.org/10.1016/s0261-5614(96)80028-8.

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Battelino, Tadej, Masakatsu Goto, Ciril Krzisnik та W. Patrick Zeller. "Tumor necrosis factor-α alters glucose metabolism in suckling rats". Journal of Laboratory and Clinical Medicine 133, № 6 (1999): 583–89. http://dx.doi.org/10.1016/s0022-2143(99)90188-9.

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Li, Yi-Ping, та Michael B. Reid. "Effect of tumor necrosis factor-α on skeletal muscle metabolism". Current Opinion in Rheumatology 13, № 6 (2001): 483–87. http://dx.doi.org/10.1097/00002281-200111000-00005.

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Shiraki, Makoto, Yoichi Terakura, Junpei Iwasa та ін. "Elevated serum tumor necrosis factor-α and soluble tumor necrosis factor receptors correlate with aberrant energy metabolism in liver cirrhosis". Nutrition 26, № 3 (2010): 269–75. http://dx.doi.org/10.1016/j.nut.2009.04.016.

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Leeper-Woodford, S. K., and B. W. Tobin. "Tumor necrosis factor activity of pancreatic islets." American Journal of Physiology-Endocrinology and Metabolism 273, no. 2 (1997): E433—E437. http://dx.doi.org/10.1152/ajpendo.1997.273.2.e433.

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Tumor necrosis factor (TNF) is involved in the pathogenesis of acute sepsis-induced organ injury and has been implicated as a mediator of metabolic alterations observed during sepsis. Pancreatic islet cell function may be significantly compromised during sepsis or endotoxemia, and sepsis also increases plasma levels of epinephrine, a modifier of islet insulin secretion. We proposed that islets exposed to bacterial lipopolysaccharide (LPS) produce TNF and that epinephrine attenuates islet secretory activity. We monitored the effects of LPS and epinephrine on TNF and insulin activity of isolated
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Deuss, Ulrich, Massimo Buscema, Heike Schumacher та Werner Winkelmann. "In vitro effects of tumor necrosis factor-α on human thyroid follicular cells". Acta Endocrinologica 127, № 3 (1992): 220–25. http://dx.doi.org/10.1530/acta.0.1270220.

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Tumor necrosis factor-α is assumed to be an important mediator in thyroid autoimmunity. In the present study we have shown that human thyrocytes possess a single specific binding site for recombinant tumor necrosis factor-α with an average of 9,300 receptors/cell (Kd = 1.9 · 10−10 mol). The effects of the cytokine on thyroid cell proliferation were assessed by 3H-thymidine uptake as well as by the protein and DNA content of cell monolayers. Low dose tumor necrosis factor-α resulted in a moderate stimulation of cell proliferation with an increase of 3H-thymidine incorporation from 44,613±7,989
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Watanabe, Naoki, Naoki Tsuji, Yasushi Tsuji, et al. "Endogenous Tumor Necrosis Factor Inhibits the Cytotoxicity of Exogenous Tumor Necrosis Factor and Adriamycin in Pancreatic Carcinoma Cells." Pancreas 13, no. 4 (1996): 395–400. http://dx.doi.org/10.1097/00006676-199611000-00009.

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Rozprawy doktorskie na temat "Tumor Necrosis Factor/metabolism"

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Miller, Katherine. "Genetic susceptibility in Alzheimer's Disease and the role of lipid metabolism." Connect to text online, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=case1164830757.

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Thesis (Ph. D.)--Case Western Reserve University, 2006.<br>[School of Medicine] Department of Epidemiology and Biostatistics. Includes bibliographical references. Available online via OhioLINK's ETD Center.
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Kerzic, Patrick James. "Inhibition of NF-[kappa]B by the benzene metabolite hydroquinone /." Connect to full text via ProQuest. Limited to UCD Anschutz Medical Campus, 2006.

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Thesis (Ph.D. in Toxicology) -- University of Colorado at Denver and Health Sciences Center, 2006.<br>Typescript. Includes bibliographical references (leaves 121-141). Free to UCDHSC affiliates. Online version available via ProQuest Digital Dissertations;
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Puliti, Elisa. "Role of sphingosine 1-phosphate metabolism and signalling in skeletal muscle atrophy and fibrosis." Doctoral thesis, Università di Siena, 2022. http://hdl.handle.net/11365/1195603.

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In the last 30 years, multiple roles of S1P have been demonstrated in the regulation of skeletal muscle biology. The presented study is focused on the role of S1P metabolism in myogenic differentiation, where SPL was found playing a crucial role in regulating S1P cellular levels and responsible for onset of myogenic program. The role of S1P axis was also confirmed in skeletal muscle atrophy induced by TNF-alpha. S1P signalling pathwayplays a crucial role in the development and maintenance of the fibrotic process. New S1P3 antagonists were tested to antagonise the receptor involved in fibrosis,
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Molgat, André. "The Effect of Macrophage-secreted Factors on Preadipocyte Survival." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/23628.

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Adipose tissue (AT) expansion and remodeling that maintains healthy function relies on stromal preadipocytes capable of differentiating into new adipocytes (adipogenesis). During chronic positive energy balance, a relative deficit in adipogenesis, from either a decrease in preadipocyte number or their capacity to differentiate, leads to excessive adipocyte hypertrophy and AT dysfunction. AT contains macrophages whose number and activation state is dynamically regulated with changes in AT mass. This study aims to investigate the effect of macrophage-secreted factors on preadipocyte survival. To
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Ferreira, João Marcos Bemfica Barbosa. "Avaliação do metabolismo e atividade inflamatória nas diversas formas evolutivas da doença de Chagas: correlação com disfunção autonômica." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-07022014-144651/.

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INTRODUÇÃO: A cardiopatia chagásica crônica (CCC) apresenta características específicas, tais como: disfunção autonômica e atividade inflamatória exacerbada. Esta fisiopatologia sugere que alguns parâmetros metabólicos podem estar alterados em pacientes chagásicos. O objetivo deste estudo foi avaliar os parâmetros metabólicos e inflamatórios nas diversas formas evolutivas de doença de Chagas e sua correlação com medidas de avaliação do Sistema Nervoso Autônomo (SNA). MÉTODOS: Foram avaliados 60 indivíduos divididos em 4 grupos (n=15): Grupo controle (GC), Grupo FI - forma indeterminada, Grupo
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Boussouar, Fayçal. "Production et transport de lactate au cours de la spermatogenèse : rôle des facteurs de croissance et des hormones sur la lactate déshydrogénase A et les transporteurs de monocarboxylates I et II." Lyon 1, 1999. http://www.theses.fr/1999LYO1T198.

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Andersson, Jonas. "Inflammation and lifestyle in cardiovascular medicine." Doctoral thesis, Umeå universitet, Medicin, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-36221.

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Despite major advances in the treatment and prevention of atherosclerosis the last several decades, cardiovascular disease still accounts for the majority of deaths in Sweden. With the population getting older, more obese and with rising numbers of diabetics, the cardiovascular disease burden may increase further in the future. The focus in cardiovascular disease has shifted with time from calcification and narrowing of arteries to the biological processes within the atherosclerotic plaque. C-reactive protein (CRP) has emerged as one of many proteins that reflect a low grade systemic inflammat
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Björnberg, Flemming. "Processing of TNF-receptors to soluble receptor forms in myeloid cells." Lund : Dept. of Hematology, Lund University, 1998. http://catalog.hathitrust.org/api/volumes/oclc/39176479.html.

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Engelberts, Ingeborg. "Tumor necrosis factor during sepsis king of cytokines? /." Maastricht : Maastricht : Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 1994. http://arno.unimaas.nl/show.cgi?fid=6955.

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Krugten, Michiel Volkert van. "Tumor necrosis factor gene polymorphisms and rheumatic diseases /." Leiden, 2003. http://catalogue.bnf.fr/ark:/12148/cb40223074h.

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Książki na temat "Tumor Necrosis Factor/metabolism"

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Corti, Angelo, and Pietro Ghezzi. Tumor Necrosis Factor. Humana Press, 2004. http://dx.doi.org/10.1385/1592597718.

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National Institutes of Health (U.S.), ed. Tumor necrosis factor. National Institutes of Health, 1989.

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National Institutes of Health (U.S.), ed. Tumor necrosis factor. National Institutes of Health, 1989.

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F, Oettgen Herbert, and International Cancer Research Data Bank., eds. Selected abstracts on tumor necrosis factor. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, International Cancer Research Data Bank, National Cancer Institute, 1987.

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Angelo, Corti, and Ghezzi P, eds. Tumor necrosis factor: Methods and protocols. Humana Press, 2004.

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Benjamin, Bonavida, ed. Tumor necrosis factor/cachectin and related cytokines. Karger, 1988.

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Sanjay, Khare, ed. TNF superfamily. Landes Bioscience, 2007.

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Gregory, Bock, Marsh Joan, and Symposium on Tumour Necrosis Factor and Related Cytotoxins (1987 : London, England), eds. Tumour necrosis factor and related cytotoxins. Wiley, 1987.

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Sanjay, Khare, ed. TNF superfamily. Landes Bioscience, 2007.

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Bruce, Beutler, ed. Tumor necrosis factors: The molecules and their emerging role in medicine. Raven Press, 1992.

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Części książek na temat "Tumor Necrosis Factor/metabolism"

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Proietti, E., F. Belardelli, G. Carpinelli, et al. "Interferon and Tumor Necrosis Factor Alter the Phospholipid Metabolism in Friend Leukemia Cell Tumors in Mice." In The Biology of the Interferon System 1986. Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3543-3_39.

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Yokoyama, Hiroomi, Seiichiro Kano, Keiichi Okamoto, and Yoshiyuki Shinagawa. "Modification of Tumor Necrosis Factor (TNF) Production and Survival Rate by a Nucleoside Mixture in Lipopolysaccharide-Injected Rats." In Purine and Pyrimidine Metabolism in Man VIII. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-2584-4_164.

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Janabi, Mohamed, Shizuya Yamashita, Ken-ichi Hirano, Kengo Matsumoto, Shuichi Nozaki, and Yuji Matsuzawa. "Reduced Secretion of Tumor Necrosis Factor-α and Interleukin-1β from Monocyte-Derived Macrophages of CD36-Deficient Subjects in Response to Oxidized LDL." In Lipoprotein Metabolism and Atherogenesis. Springer Japan, 2000. http://dx.doi.org/10.1007/978-4-431-68424-4_48.

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Descamps-Latscha, Béatrice, André Herbelin, Anh Thu Nguyen, and P. Urena. "Respective Influence of Uremia and Hemodialysis on Whole Blood Phagocyte Oxidative Metabolism, and Circulating Interleukin-1 and Tumor Necrosis Factor." In Advances in Experimental Medicine and Biology. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4899-3629-5_16.

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Sasajima, T., K. Mineura, H. Shioya, et al. "Effects of intracarotid recombinant human tumour necrosis factor-α on kinetics of glucose metabolism in malignant gliomas." In Proceedings of the XV Symposium Neuroradiologicum. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79434-6_77.

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Chu, Wen-Ming. "Tumor Necrosis Factor." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27841-9_6040-8.

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Barger, Steven W. "Tumor Necrosis Factor." In Neuroprotective Signal Transduction. Humana Press, 1998. http://dx.doi.org/10.1007/978-1-59259-475-7_9.

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Chu, Wen-Ming. "Tumor Necrosis Factor." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_6040.

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Ulich, Thomas R. "Tumor Necrosis Factor." In Cytokines of the Lung. CRC Press, 2022. http://dx.doi.org/10.1201/9781003066927-11.

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Manogue, Kirk R., and Anthony Cerami. "Cachectin (Tumor Necrosis Factor)." In Cellular and Molecular Aspects of Inflammation. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5487-1_8.

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Streszczenia konferencji na temat "Tumor Necrosis Factor/metabolism"

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Rahmawaty, Annis, Akrom, Endang Darmawan, Titis Indri Wahyuni, Dian Kumalasari та Adnan. "Tumor Necrosis Factor Alpha (Tnf-α) and Creatinine Levels in Patients at Risk of Metabolic Syndrome with Standard Therapy Combined with Black Cumin Seed Oil". У 1st Paris Van Java International Seminar on Health, Economics, Social Science and Humanities (PVJ-ISHESSH 2020). Atlantis Press, 2021. http://dx.doi.org/10.2991/assehr.k.210304.142.

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Goel, Raghav, Guilio F. Paciotti, and John C. Bischof. "Tumor necrosis factor-alpha induced enhancement of cryosurgery." In Biomedical Optics (BiOS) 2008, edited by Nikiforos Kollias, Bernard Choi, Haishan Zeng, et al. SPIE, 2008. http://dx.doi.org/10.1117/12.764020.

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Rivas, MA, M. Tkach, CJ Proietti, et al. "Tumor necrosis factor transactivates ErbB2 in breast cancer cells." In CTRC-AACR San Antonio Breast Cancer Symposium: 2008 Abstracts. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-4056.

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Tavares, Thayna Monteiro, EDUARDA OLIVEIRA PERIN, MARYEL CRISTIN SEDOVSKI, GIOVANNA CHARRON DE MACEDO, and CAMILA RIGOBELLO. "ADIPOCINAS E O QUADRO INFLAMATÓRIO CRÔNICO EM OBESOS." In II Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conbrai/6770.

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Introdução: O tecido adiposo produz alguns hormônios importantes no metabolismo que são enviados para a circulação sanguínea e atuam na inflamação, podendo ser antagônicos ou promotores deste processo, sendo assim entende-se que a obesidade altera a produção destas substâncias as quais pode afetar no sistema imunológico. Objetivo: Compreender a resposta imunológica correlacionado a obesidade. Material e métodos: Foi realizada busca nas plataformas Google acadêmico e Scielo de artigos desde 2018 em português com os termos “adipocinas e sistema imune”. Resultados: O aumento da quantidade da cama
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abla, hedia ben, Sonia Rekik, Soumaya Boussaid, et al. "AB0699 EFFECT OF SWITCHING BETWEEN TUMOR NECROSIS FACTOR INHIBITOR IN SPONDYLOARTHRITIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.3834.

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Pass, Harvey I., Steven Evans, Roger Perry, and Wilbert Matthews. "Kinetics of tumor necrosis factor production by photodynamic-therapy-activated macrophages." In OE/LASE '90, 14-19 Jan., Los Angeles, CA, edited by Thomas J. Dougherty. SPIE, 1990. http://dx.doi.org/10.1117/12.17660.

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Huang, Yun-Ju, Yao-Fan Fang, Shue-Fen Luo, et al. "AB0383 LATENT TUBERCULOSIS INFECTION SHOULD BE MONITORED IN BOTH TUMOR NECROSIS FACTOR INHIBITORS AND NON-TUMOR NECROSIS FACTOR INHIBITORS IN BIOLOGICAL-NAïVE PATIENTS WITH RHEUMATOID ARTHRITIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.3054.

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Rego, Stephen, Krista Ricci, Muthulekha Swamydas, and Didier Dreau. "Abstract 397: Soluble Tumor Necrosis Factor Receptor shed by breast tumor cells modulates macrophage migration." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-397.

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Iwona, Grądzka, Sikorska Katarzyna, and Brzóska Kamil. "Interference of Silver Nanoparticles with Tumor Necrosis Factor Action in Epithelial Cells." In The 2nd World Congress on Recent Advances in Nanotechnology. Avestia Publishing, 2017. http://dx.doi.org/10.11159/icnb17.116.

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Membriani, Evangelina, Erika Cuenca, Leticia Limongi, Ana Putruele, and Carlos Luna. "Latent tuberculosis screening and entering antibody therapy monoclonares against tumor necrosis factor." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa2701.

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Raporty organizacyjne na temat "Tumor Necrosis Factor/metabolism"

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Meidan, Rina, and Joy Pate. Roles of Endothelin 1 and Tumor Necrosis Factor-A in Determining Responsiveness of the Bovine Corpus Luteum to Prostaglandin F2a. United States Department of Agriculture, 2004. http://dx.doi.org/10.32747/2004.7695854.bard.

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The corpus luteum (CL) is a transient endocrine gland that has a vital role in the regulation of the estrous cycle, fertility and the maintenance of pregnancy. In the absence of appropriate support, such as occurs during maternal recognition of pregnancy, the CL will regress. Prostaglandin F2a (PGF) was first suggested as the physiological luteolysin in ruminants several decades ago. Yet, the cellular mechanisms by which PGF causes luteal regression remain poorly defined. In recent years it became evident that the process of luteal regression requires a close cooperation between steroidogenic,
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Larrick, James W., Vera Morhenn, Yawen L. Chiang, and Tim Shi. Activated Langerhans Cells Release Tumor Necrosis Factor. Defense Technical Information Center, 1988. http://dx.doi.org/10.21236/ada206646.

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Gao, Li-nan, Lian-gang Ge, Ming-zhe Zhu та Xin-xin Yao. Association between tumor necrosis factor α and uterine fibroids: a protocol of systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.7.0010.

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Behbakht, Kian. Modulators of Response to Tumor Necrosis-Factor-Related Apoptosis Inducing Ligand (TRAIL) Therapy in Ovarian Cancer. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada532993.

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Li, Peng, and Junjun Liu. Effect of tumor necrosis factor inhibitors on the risk of adverse cardiovascular events in patients with psoriasis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.8.0090.

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Review question / Objective: Previous studies have indicated a cardioprotective effect of tumor necrosis factor inhibitor (TNFi) therapy in adult patients with psoriasis (Pso). However, most were retrospective studies, and the association between cardiometabolic comorbidities and major adverse cardiovascular events (MACE) has not been validated in randomized controlled trials (RCTs). Condition being studied: Because the available evidence has recently increased, we performed the present updated meta-analysis and meta-regression of cohort studies and RCTs to evaluate whether TNFi therapy can de
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Borra, Himabindu, Daniel F. Battafarano, Ramon Arroyo, Michael J. Morris, Michelle Sit, and Gerald Merrill. Reliability of Tuberculosis Screening Test in Patients Receiving Tumor Necrosis Factor Antagonist Therapy in a United States Rheumatology Clinic. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada577631.

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Ni, Xiaofeng, and Lingli Zhang. Efficacy and Safety of Anti-Tumor Necrosis Factor Alpha in Very Early Onset Inflammatory Bowel Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. https://doi.org/10.37766/inplasy2024.12.0012.

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Dotsenko, S. S., L. N. Shilova, A. S. Trofimenko, S. A. Bedina, E. A. Tikhomirova та M. A. Mamus. The role of cytokines in predicting the effectiveness of combined treatment with tumor necrosis factor α inhibitors in rheumatoid arthritis. ООО "ИМА-Пресс", 2018. http://dx.doi.org/10.18411/1995-4484-2018-56-33-17.

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กาญจนทัต, อภิชาติ. ความเป็นพิษและการเหนี่ยวนำการตายแบบอะโพโทซิสต่อเซลล์มะเร็ง โดยเพปไทด์จากเกสรผึ้งพันธุ์ Apis mellifera. สถาบันวิจัยเทคโนโลยีชีวภาพและวิศวกรรมพันธุศาสตร์ จุฬาลงกรณ์มหาวิทยาลัย, 2018. https://doi.org/10.58837/chula.res.2018.100.

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งานวิจัยนี้เตรียมโปรตีนไฮโดรไลเสตจากเกสรผึ้งพันธุ์ ที่ได้จากปฏิกิริยาย่อยสลายด้วยเอนไซม์ 3 ชนิด ได้แก่ แอลคาเลส ฟลาโวไซม์ และนิวเทรส พบว่าเมื่อใช้นิวเทรสในอัตราส่วนของเอนไซม์ต่อสับสเตรต 1:1 (NH1) โดยปริมาตร จะให้แสดงค่าการยับยั้งการสร้างอนุมูลอิสระด้วยวิธีไนตริกออกไซต์ได้ดีที่สุด คัดแยกเพปไทด์ที่มีขนาดโมเลกุลต่ำกว่า 0.65 กิโลดาลตัน (MW1) มีฤทธิ์ในการขจัดอนุมูลอิสระไนตริกออกไซด์ได้ดีที่สุด จากนั่นได้ทำการตรวจสอบความเป็นพิษของ MW1 ด้วยวิธี MTT และตรวจสอบ ผลการยับยั้งการสร้างไนตริกออกไซด์ในเซลล์แมคโครฟาจ RAW 264.7 ที่ถูกกระตุ้นด้วยไลโปโพลิแซคคาไรด์ พบว่า MW1 ไม่มีความเป็นพิษต่อเซลล์ และมีฤทธิ์ในก
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พงษ์เลาหพันธุ์, ศุภวิวัธน์, та ธีรพงศ์ ยะทา. การพัฒนาพาหะนำส่งยีนเหนี่ยวนำการตายของเซลล์แบบอะพอพโทซิสระดับนาโน เพื่อการคุมกำเนิดสัตว์เพศผู้แบบไม่ผ่าตัดทำหมัน : ต้นแบบเพื่อการคุมจำนวนประชากรสุนัขและแมวจรจัด. คณะสัตวแพทยศาสตร์ จุฬาลงกรณ์มหาวิทยาลัย, 2019. https://doi.org/10.58837/chula.res.2019.42.

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การศึกษาวิจัยนี้มีวัตถุประสงค์ เพื่อตรวจสอบการนำไปใช้ของอนุภาคไคโตซานระดับนาโนที่ถูกดัดแปลงให้มีคุณสมบัติเป็นตัวนำส่งยีนเหนี่ยวนำการตายแบบอะพอพโทซิสเข้าสู่เซลล์อัณฑะที่มีตัวรับฮอร์โมนโกนาโดโทรปินรีลิสซิ่ง โดยการออกแบบตัวนำส่งยีนนี้สามารถนำไปใช้ประโยชน์ในการทำหมันสัตว์เพศผู้แบบไม่ผ่าตัด การศึกษานี้ได้มีการรายงานผลของอนุภาคไคโตซานระดับนาโนเชื่อมติดกับฮอร์โมนโกนาโดโทรปินรีลิสซิ่ง (Gonadotropin Releasing Hormone-modified Chitosan; GnRH-CS) เพื่อนำส่งยีนอย่างมีเป้าหมาย และการใช้เปปไทด์โกนาโดโทรปินรีลิสซิ่งในการระบุเป้าหมายการนำส่งยีนไปสู่เซลล์ที่มีตัวรับฮอร์โมนโกนาโดโทรปินรีลิสซิ่ง (GnRH receptor;
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