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Livros sobre o tema "Anticancer drug treatment"

Autor: Grafiati

Publicado: 14 de março de 2023

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1

Link, Wolfgang. Principles of Cancer Treatment and Anticancer Drug Development. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-18722-4.

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2

Manuel, Hidalgo. Principles of anticancer drug development. New York: Springer, 2011.

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3

Parfenov, E. A. Biometals and ligands for anticancer drug design. Commack, N.Y: Nova Science Publishers, 1998.

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4

1946-, Lippert Bernhard, ed. Cisplatin: Chemistry and biochemistry of a leading anticancer drug. Zürich: Verlag Helvetica Chimica Acta, 1999.

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5

Avendaño, Carmen. Medicinal chemistry of anticancer drugs. Amsterdam: Elsevier, 2008.

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6

J, Houghton Peter, e Houghton Janet A, eds. Preclinical and clinical modulation of anticancer drugs. Boca Raton: CRC Press, 1993.

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7

1945-, Ojima Iwao, Vite Gregory D, Altmann Karl-Heinz, American Chemical Society. Division of Organic Chemistry, American Chemical Society. Division of Medicinal Chemistry e American Chemical Society Meeting, eds. Anticancer agents: Frontiers in cancer chemotherapy. Washington, DC: American Chemical Society, 2001.

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8

Cho, William C. S. Evidence-based Anticancer Materia Medica. Dordrecht: Springer Science+Business Media B.V., 2011.

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9

Lipp, H. P. Prevention and management of anticancer drug toxicity: The significance of clinical pharmacokinetics. Jena: Univ.-Verlag, 1995.

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10

Snapka, Robert M. The SV40 replicon model for analysis of anticancer drugs. Austin, TX: R.G. Landes, 1996.

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11

The SV40 replicon model for analysis of anticancer drugs. [San Diego, Calif.]: Academic Press, 1996.

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12

J, Zeller W., D'Incalci M, Newell D. R e Novel approaches in cancer therapy (Heidelburg : 1993), eds. Novel approaches in anticancer drug design: Molecular modelling - new treatment strategies, novel approaches in cancer therapy. Basel: Karger, 1994.

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13

International Symposium on Novel Approaches in Cancer Therapy (1993 Heidelberg, Germany). Novel approaches in anticancer drug design: Molecular modelling, new treatment strategies : International Symposium on Novel Approaches in Cancer Therapy, Heidelberg, December 1-4, 1993. Editado por Zeller W. J, D'Incalci Maurizio e Newell David R. Basel: Karger, 1995.

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14

Frederick, Valeriote, e Baker Laurence H, eds. Biochemical modulation of anticancer agents: Experimental and clinical approaches : proceedings of the 18th Annual Detroit Cancer Symposium, Detroit, Michigan, USA, June 13-14, 1986. Boston: Nijhoff, 1986.

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15

Convention, United States Pharmacopeial. Fact sheets on anticancer drugs. [Washington, D.C.?]: National Cancer Institute [distributor], 1994.

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16

Anticancer herbal drugs of India with special reference to ayurveda. Delhi: Sri Satguru Publications, 2002.

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17

Stephen, Neidle, e Waring Michael J, eds. Molecular aspects of anticancer drug-DNA interactions. Basingstoke: Macmillan, 1993.

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18

Neidle, Stephen, e Michael J. Waring. Molecular Aspects of Anticancer Drug-DNA Interactions. Taylor & Francis Group, 1994.

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19

Link, Wolfgang. Principles of Cancer Treatment and Anticancer Drug Development. Springer International Publishing AG, 2020.

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20

Link, Wolfgang. Principles of Cancer Treatment and Anticancer Drug Development. Springer, 2019.

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21

Lipp. Anticancer Drug Toxicity: Prevention, Management, and Clinical Pharmacokinetics. Informa Healthcare, 1999.

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22

(Editor), Lori J. Goldstein, e Robert F. Ozols (Editor), eds. Anticancer Drug Resistance: Advances in Molecular and Clinical Research (Cancer Treatment and Research). Springer, 1994.

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23

Medicinal Chemistry of Anticancer Drugs. Elsevier Science & Technology Books, 2015.

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24

Avendano, Carmen, e J. Carlos Menendez. Medicinal Chemistry of Anticancer Drugs. Elsevier Science & Technology Books, 2021.

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25

Avendano, Carmen, e J. Carlos Menendez. Medicinal Chemistry of Anticancer Drugs. Elsevier Science & Technology Books, 2008.

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26

Medicinal Chemistry of Anticancer Drugs. Elsevier Science, 2008.

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27

Avendano, Carmen, e J. Carlos Menendez. Medicinal Chemistry of Anticancer Drugs. Elsevier Science & Technology, 2021.

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28

Singh, Rajesh Kumar, ed. Key Heterocyclic Cores for Smart Anticancer Drug–Design Part I. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/97898150400741220101.

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This book provides an update on heterocyclic compounds that serve as key components of anti-cancer agents administered in pre-clinical settings. Many of the compounds highlighted in the book are being actively investigated for the bioactive properties against a range of cancer cell lines. There is potential for heterocyclic compounds to design agents that can target specific molecules to treat different types of cancers. Chapters are contributed by experts in pharmaceutical chemistry and are written to give a general overview of the topic to readers involved in all levels of research and decision-making in pharmaceutical chemistry and anti-cancer drug design. Part 1 of the book set covers these topics: - Heterocyclic anticancer compounds derived from natural sources with their mechanism of action - The role of terpenoids as anticancer compounds: an insight into prevention and treatment - Recent advances in synthesis and anticancer activity of benzothiazole hybrids as anticancer agents - Structure-activity relationship studies of novel hybrid quinoline and quinolone derivatives as anticancer agents - Tetrazoles: structure and activity relationship as anticancer agents - Progress in nitrogen and oxygen-based heterocyclic compounds for their anticancer activity: an update (2017-2020)

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29

(Editor), Iwao Ojima, Gregory D. Vite (Editor) e Karl-Heinz Altmann (Editor), eds. Anticancer Agents: Frontiers in Cancer Chemotherapy (Acs Symposium Series). An American Chemical Society Publication, 2001.

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30

Zaikov, Gennadii Efremovich, e E. A. Parfenov. Biometals and Ligands for Anticancer Drug Design: Superoxide Dimutase Models in Combined Tumor Therapy. 2^a ed. Nova Science Pub Inc, 2000.

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31

Parfenov, E. A. Biometals And Ligands For Anticancer Drug Design: Molecular Mechanisms of Superoxide Dimutase Models Antitumor Effects. NOVA SCIENCE PUBLISHERS, 1998.

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32

Biochemical Modulation of Anticancer Agents: Experimental and Clinical Approaches (Developments in Oncology). Springer, 1986.

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33

Zeller, W. J., e M. D'Incalci. Novel Approaches in Anticancer Drug Design: Molecular Modelling - New Treatment Strategies : International Symposium on Novel Approaches in Cancer T (Contributions ... Oncology = Beitrage Zur Onkologie, Vol 49). S. Karger AG (Switzerland), 1995.

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34

Snapka, Robert M. SV40 Replicon Model for Analysis of Anticancer Drugs. Elsevier Science & Technology Books, 1996.

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35

Avendano, Carmen, e J. Carlos Menendez. Medicinal Chemistry of Anticancer Drugs, Second Edition. Elsevier Science, 2015.

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36

Atta-ur-Rahman e Khurshid Zaman, eds. Topics in Anti-Cancer Research: Volume 8. BENTHAM SCIENCE PUBLISHERS, 2019. http://dx.doi.org/10.2174/97898114043821190801.

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Topics in Anti-Cancer Research covers new developments in the field of cancer. Novel drugs as anticancer agents include natural and synthetic phenazines and other anti-cancer compounds. It also encompasses the role of estrogen as endocrine disruptors and strategies targeting cancer stem cells for the treatment of different types of cancers, including myeloma and renal cell cancer. The diversity of researches and topics published in this eBook Series will be valuable to cancer researchers, clinicians, and cancer professionals aiming to develop novel anti-cancer targets for the treatment of various cancers. The topics covered in the eighth volume of this series are as follows: Novel Drugs for Multiple Myeloma Synthetic Estrogens are Endocrine Disruptors via Inhibition of AF1 Domain of ERs Recent Progress of Phenazines as Anticancer Agents Cancer Stem Cell Targeting for Anticancer Therapy: Strategies and Challenges

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37

Freifeld, Alison G. An Introduction. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199938568.003.0100.

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This chapter focuses on solid tumors and how they can be treated. Solid tumors, lymphomas, and leukemias represent a widely diverse array of cancers. Until recently, the general approach to treating all of them was to administer cytotoxic anticancer drugs that damage proliferating cells by interfering with mitosis and other essential steps in cellular replication. Localized solid tumors are largely treated by surgical resection and radiotherapy, with cytotoxic chemotherapy being commonly used adjunctively or in cases of metastatic disease. A major drawback of this approach has been the lack of specificity in that cytotoxic drugs will destroy actively dividing normal cells as well as malignant cells. The “shotgun approach” of using intensive cytotoxic chemotherapies has been the mainstay of cancer treatment for at least 6 decades. The chapter concludes that in addition to the tissue damage and immunomodulating effects of anticancer drugs, it should be remembered that cancers themselves may increase the chances for infection.

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38

Innominato, Pasquale F., e David Spiegel. Circadian rhythms, sleep, and anti-cancer treatments. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198778240.003.0016.

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The circadian timing system temporally regulates biological functions relevant for psycho-physical wellbeing, spanning all the systems related to health. Hence, disruption of circadian rhythms, along with sleep cycles, is associated with the development of several diseases, including cancer. Moreover, altered circadian and sleep functions negatively impact on cancer patients’ quality of life and survival, above and beyond known determinants of outcome. This alteration can occur as a consequence of cancer, but also of anti-cancer treatments. Indeed, circadian rhythms govern also the ability of detoxifying chemotherapy agents across the 24 hours. Hence, adapting chemotherapy delivery to the molecular oscillations in relevant drug pathways can decrease toxicity to healthy cells, while increasing the number of cancer cells killing. This chronomodulated chemotherapy approach, together with the maintenance of proper circadian function throughtout the whole disease challenge, would finally result in safer and more active anticancer treatments, and in patients experiencing better quality and quantity of life.

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