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Bibliografias temáticas / Cell death

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Literatura científica selecionada sobre o tema "Cell death"

Autor: Grafiati

Publicado: 4 de junho de 2021

Última modificação: 25 de julho de 2025

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Artigos de revistas sobre o assunto "Cell death"

1

Fernández-Lázaro, Diego, César Ignacio Fernández-Lázaro, and Martínez Alfredo Córdova. "Cell Death: Mechanisms and Pathways in Cancer Cells." Cancer Medicine Journal 1, no. 1 (2018): 12–23. http://dx.doi.org/10.46619/cmj.2018.1-1003.

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Programmed cell death is an essential physiological and biological process for the proper development and functioning of the organism. Apoptosis is the term that describes the most frequent form of programmed cell death and derives from the morphological characteristics of this type of death caused by cellular suicide. Apoptosis is highly regulated to maintain homeostasis in the body, since its imbalances by increasing and decreasing lead to different types of diseases. In this review, we aim to describe the mechanisms of cell death and the pathways through apoptosis is initiated, transmitted,

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2

Lockshin, Richard A., and Zahra Zakeri. "Cell Death (Apoptosis, Programmed Cell Death)." Directions in Science 1 (February 27, 2002): 41–44. http://dx.doi.org/10.1100/tsw.2002.161.

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3

Yao, Nan, and Jean T. Greenberg. "Arabidopsis ACCELERATED CELL DEATH2 Modulates Programmed Cell Death." Plant Cell 18, no. 2 (2005): 397–411. http://dx.doi.org/10.1105/tpc.105.036251.

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4

Deniz, Özdemir. "KAN0438757: A NOVEL PFKFB3 INHIBITOR THAT INDUCES PROGRAMMED CELL DEATH AND SUPPRESSES CELL MIGRATION IN NON-SMALL CELL LUNG CARCINOMA CELLS." Biotechnologia Acta 16, no. 5 (2023): 34–44. http://dx.doi.org/10.15407/biotech16.05.034.

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Aim. PFKFB3 is glycolytic activators that is overexpressed in human lung cancer and plays a crucial role in multiple cellular functions including programmed cell death. Despite the many small molecules described as PFKFB3 inhibitors, some of them have shown disappointing results in vitro and in vivo. On the other hand KAN0438757, selective and potent, small molecule inhibitor has been developed. However, the effects of KAN0438757, in non-small cell lung carcinoma cells remain unknown. Herein, we sought to decipher the effect of KAN0438757 on proliferation, migration, DNA damage, and programmed

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5

Sarosiek, Kristopher. "Blocking cell death to enhance cell death." Science Translational Medicine 9, no. 408 (2017): eaao6129. http://dx.doi.org/10.1126/scitranslmed.aao6129.

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6

M, Aloysius Dhivya, Kishore A, and Bharathidevi SR. "Down-Regulation of NRF2 Signaling Triggers Cell Death in Y79 Cells upon Copper Chelation." Open Access Journal of Ophthalmology 10, no. 1 (2025): 1–10. https://doi.org/10.23880/oajo-16000329.

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Background: Retinoblastoma is a rare intraocular malignancy that leads to vision loss in children. While copper (Cu) chelation has been reported as a therapy in many cancers, its relevance to retinoblastoma has not yet been explored. Objectives: In this study, we have explored the role of penicillamine (a Cu chelator) as a therapeutic target for retinoblastoma using Y79 cells as a model. Methods: The effect of the Cu chelator on the viability of Y79 was assessed using the MTT assay. Additionally, we performed nuclear fractionation to assess Nuclear factor erythroid 2–related factor 2 (NRF2) ac

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7

Medema, J. P., H. Walczak, M. Hahne, and V. de Laurenzi. "Cell Death." Cell Death & Differentiation 17, no. 4 (2010): 730–32. http://dx.doi.org/10.1038/cdd.2010.11.

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8

Hotchkiss, Richard S., Andreas Strasser, Jonathan E. McDunn, and Paul E. Swanson. "Cell Death." New England Journal of Medicine 361, no. 16 (2009): 1570–83. http://dx.doi.org/10.1056/nejmra0901217.

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9

Vogel, Michael W. "Cell Death." American Journal of Psychiatry 162, no. 8 (2005): 1503. http://dx.doi.org/10.1176/appi.ajp.162.8.1503.

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10

MALORNI, WALTER, and GIANFRANCO DONELLI. "Cell Death." Annals of the New York Academy of Sciences 663, no. 1 Aging and Cel (1992): 218–33. http://dx.doi.org/10.1111/j.1749-6632.1992.tb38666.x.

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Teses / dissertações sobre o assunto "Cell death"

1

Pat, Sze Wa. "Cell metabolism in cell death and cell growth." HKBU Institutional Repository, 2007. http://repository.hkbu.edu.hk/etd_ra/775.

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2

Crisby, Milita. "Cell death in atherosclerosis /." Stockholm, 1998. http://diss.kib.ki.se/1998/91-628-3191-7/.

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3

Ellison, David William. "Cell proliferation, cell death, and differentiation in gliomas." Thesis, University of Southampton, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295912.

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4

Uppington, Kay Marie. "Cell death in prion disease." Thesis, University of Bath, 2008. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.488879.

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Prion diseases are a group of fatal neurodegenerative diseases, including CJD and scrapie, which are thought to be caused by a protein termed a prion (PrP). As manganese has previously been suggested to be involved in prion disease we have investigated manganese binding to PrP and its role in the toxicity of the protein. We have shown that manganese bound PrP (MnPrP) has several of the characteristics of the disease form of PrP, including protease resistance and toxicity that is dependent on cellular PrP expression. Further investigation into the mechanism of toxicity revealed that MnPrP is si

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5

Beeharry, Neil. "Cell death in insulin-containing cells : induction and prevention." Thesis, University of Brighton, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.401600.

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6

Gorak-Stolinska, Patricia. "Activation induced cell death in human T cell subsets." Thesis, King's College London (University of London), 2002. http://kclpure.kcl.ac.uk/portal/en/theses/activation-induced-cell-death-in-human-t-cell-subsets(eb708e24-eccb-42fc-8930-d62ddf6794c1).html.

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7

Cheng, Jade. "Regulation of cell division and cell death by GRASP65." Thesis, University of Bristol, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.544414.

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8

RUNYAN, CHRISTOPHER MICHAEL. "The Role of Cell Death in Germ Cell Migration." University of Cincinnati / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1210732680.

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9

Courtois-Moreau, Charleen Laetitia. "Programmed Cell Death in Xylem Development." Doctoral thesis, Umeå universitet, Umeå Plant Science Centre, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1831.

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Concerns about climate changes and scarcity of fossil fuels are rising. Hence wood is becoming an attractive source of renewable energy and raw material and these new dimensions have prompted increasing interest in wood formation in trees, in both the scientific community and wider public. In this thesis, the focus is on a key process in wood development: programmed cell death (PCD) in the development of xylem elements. Since secondary cell wall formation is dependent, inter alia, upon the life time of xylem elements, the qualitative features of wood will be affected by PCD in xylem, about whi

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10

Klassen, Shaun Scott. "Nitric oxide-induced cardiomyocyte cell death." Thesis, University of British Columbia, 2006. http://hdl.handle.net/2429/31539.

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Nitric oxide (NO), a regulator of diverse cardiovascular functions, modifies cardiac cell viability through mechanisms that remain uncertain. Several pathways were studied to understand these effects. The possibility that the protein p53 is involved in the cardiomyocyte response to the NO donor s-nitrosoglutathione (GSNO) or the peroxynitrite donor 3- morpholinosydnonimine (SIN-1) was explored. These donors induced a concentration-dependent increase of cell death in cultured embryonic chick cardiomyocytes. Expression of p53 protein was increased in response to GSNO, specifically in the n

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Mais fontes

Livros sobre o assunto "Cell death"

1

Melino, Gerry. Cell death. Wiley-Blackwell, 2010.

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2

Wu, Hao, ed. Cell Death. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9302-0.

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3

Gerry, Melino, and Vaux David, eds. Cell death. John Wiley & Sons, 2010.

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4

Jahani-Asl, Arezu, ed. Neuronal Cell Death. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2409-8.

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5

Shen, Han-Ming, and Peter Vandenabeele, eds. Necrotic Cell Death. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-8220-8.

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6

Puthalakath, Hamsa, and Christine J. Hawkins, eds. Programmed Cell Death. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3581-9.

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Lossi, Laura, and Adalberto Merighi, eds. Neuronal Cell Death. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2152-2.

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8

Shi, Yun-Bo, Yufang Shi, Yonghua Xu, and David W. Scott, eds. Programmed Cell Death. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-0072-2.

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9

Paul, Mattson Mark, Estus Steven, and Rangnekar Vivek, eds. Programmed cell death. Elsevier, 2001.

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10

Yun-Bo, Shi, and International Symposium on Programmed Cell Death (1996 : Shanghai, China), eds. Programmed cell death. Plenum Press, 1997.

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Mais fontes

Capítulos de livros sobre o assunto "Cell death"

1

Lockshin, Richard A. "Cell Death." In Studies of Aging. Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59916-3_5.

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2

Burke, Thomas J., and Robert W. Schrier. "Cell Death." In Molecular Biology of Membrane Transport Disorders. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-1143-0_24.

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3

Knudsen, T. B. "Cell Death." In Drug Toxicity in Embryonic Development I. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60445-4_8.

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4

Vaux, David L. "Historical Perspective: The Seven Ages of Cell Death Research." In Cell Death. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9302-0_1.

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5

Chan, Francis Ka-Ming. "Programmed Necrosis/Necroptosis: An Inflammatory Form of Cell Death." In Cell Death. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9302-0_10.

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6

Gavathiotis, Evripidis. "Structural Perspectives on BCL-2 Family of Proteins." In Cell Death. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9302-0_11.

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7

Li, Jixi, and Hao Wu. "Structural Basis of Death Receptor Signaling." In Cell Death. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9302-0_12.

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8

Jiang, Xuejun. "The Intrinsic Apoptotic Pathway." In Cell Death. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9302-0_2.

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9

Lucas, Carrie L., and Michael J. Lenardo. "Molecular Basis of Cell Death Programs in Mature T Cell Homeostasis." In Cell Death. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9302-0_3.

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10

Liu, Qian, Xiaoke Chi, Brian Leber, and David W. Andrews. "Bcl-2 Family and Their Therapeutic Potential." In Cell Death. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9302-0_4.

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Trabalhos de conferências sobre o assunto "Cell death"

1

Satio, Makoto, Rinko Kurogi, Satoshi Yoshimoto, et al. "Microfluidic Non-Stationary Process for Live Cell Imaging of Triggered Cell Death." In 2025 International Conference on Manipulation, Automation and Robotics at Small Scales (MARSS). IEEE, 2025. https://doi.org/10.1109/marss65887.2025.11072802.

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2

Bilgic, Elif. "Endocannabinoid induced apoptotic cell death on endometriotic cells." In 15th International Congress of Histochemistry and Cytochemistry. LookUs Scientific, 2017. http://dx.doi.org/10.5505/2017ichc.op-12.

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3

Wright, Neil T. "Parameter Correlation in Models of Hyperthermic Cell Death." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53933.

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A number of mathematical models have been developed to predict the survival of cells after heating. Some of these models have been based on first principle arguments, while others have been empirically motivated. Some models have been inspired by analogs of damage to cells by ionizing radiation. Evidence exists for multiple targets leading to cell death, although precise definition of the pathways for the various temperature ranges and environmental conditions remains in question. For reviews of the cellular targets of heating, see [1], [2], or [3].

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4

Jain, Shruti, Pradeep K. Naik, and Sunil V. Bhooshan. "BiCMOS Implementation of Cell Signaling for Cell Survival/Death." In 2010 International Conference on Signal Acquisition and Processing (ICSAP). IEEE, 2010. http://dx.doi.org/10.1109/icsap.2010.36.

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5

Gu, Ying, Shanxiang Jiang, Elahe Mahdavian, and Shile Huang. "Abstract 4566: Fusarochromanone inhibits cell proliferation and induces cell death in COS7 cells." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4566.

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6

Pearce, John A. "Considerations in Modeling Cell Death Processes." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80191.

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Traditional single-reaction Arrhenius models have been successfully used for many years in burn studies[1–3] and have been adapted and used to predict quantitative histologic results in laser, RF and microwave heating at high temperatures.[4–6] The single reaction kinetics model also forms the basis for the time scaling ratio as is currently used in calculating the cumulative equivalent minutes (CEM) assessment of tumor hyperthermia treatments.[7] Recently, it has been clearly demonstrated that these models are not acceptably accurate predictors of the early stages of cell death processes in h

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7

"Regulated cell death in Hetherocephalus glaber." In Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-365.

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8

Kessel, David, and John J. Reiners, Jr. "Cell death pathways associated with PDT." In Biomedical Optics 2006, edited by David Kessel. SPIE, 2006. http://dx.doi.org/10.1117/12.639925.

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9

Zitvogel, Laurence, and Guido Kroemer. "Abstract SY03-01: The desirable death of the cancer cell: Immunogenic cell death for optimal chemotherapy." In Proceedings: AACR 101st Annual Meeting 2010; Apr 17-21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-sy03-01.

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10

Hadji, Abbas, Annika Hau, and Marcus E. Peter. "Abstract 4847: CD95/Fas protects cancer cells from cell death." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4847.

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Relatórios de organizações sobre o assunto "Cell death"

1

Baker, Nicholas E. Cell Proliferation, Cell Death, and Size Regulation. Defense Technical Information Center, 1998. http://dx.doi.org/10.21236/adb248354.

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2

Distelhorst, Clark W. Programmed Cell Death in Breast Cancer. Defense Technical Information Center, 1995. http://dx.doi.org/10.21236/ada300581.

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3

Distelhorst, Clark W. Programmed Cell Death in Breast Cancer. Defense Technical Information Center, 1997. http://dx.doi.org/10.21236/ada340671.

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4

Chung, Leland W. K. Accelerated Tumor Cell Death by Anglogenic Modifiers. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada441865.

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5

Drews, Gary, N. Programmed Cell Death During Female Gametophyte Development. Office of Scientific and Technical Information (OSTI), 2004. http://dx.doi.org/10.2172/1014978.

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6

Chung, Leland W., Chia-Ling Hsieh, Michael Bradley, and Mitchell H. Sokoloff. Accelerated Tumor Cell Death by Angiogenic Modifiers. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada403672.

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7

Tyler, Kenneth L. Mechanisms of Virus-Induced Neural Cell Death. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada435392.

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8

Chung, Leland W. Accelerated Tumor Cell Death by Angiogenic Modifiers. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada418654.

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9

Kornbluth, Sally. Metabolic Regulation of Ovarian Cancer Cell Death. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada570124.

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10

Tyler, Kenneth L. Mechanisms of Virus-Induced Neural Cell Death. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada419455.

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