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Artigos de revistas sobre o tema "Cell death"

Autor: Grafiati

Publicado: 4 de junho de 2021

Última modificação: 25 de julho de 2025

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1

Fernández-Lázaro, Diego, César Ignacio Fernández-Lázaro, and Martínez Alfredo Córdova. "Cell Death: Mechanisms and Pathways in Cancer Cells." Cancer Medicine Journal 1, no. 1 (2018): 12–23. http://dx.doi.org/10.46619/cmj.2018.1-1003.

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Programmed cell death is an essential physiological and biological process for the proper development and functioning of the organism. Apoptosis is the term that describes the most frequent form of programmed cell death and derives from the morphological characteristics of this type of death caused by cellular suicide. Apoptosis is highly regulated to maintain homeostasis in the body, since its imbalances by increasing and decreasing lead to different types of diseases. In this review, we aim to describe the mechanisms of cell death and the pathways through apoptosis is initiated, transmitted,

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2

Lockshin, Richard A., and Zahra Zakeri. "Cell Death (Apoptosis, Programmed Cell Death)." Directions in Science 1 (February 27, 2002): 41–44. http://dx.doi.org/10.1100/tsw.2002.161.

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3

Yao, Nan, and Jean T. Greenberg. "Arabidopsis ACCELERATED CELL DEATH2 Modulates Programmed Cell Death." Plant Cell 18, no. 2 (2005): 397–411. http://dx.doi.org/10.1105/tpc.105.036251.

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4

Deniz, Özdemir. "KAN0438757: A NOVEL PFKFB3 INHIBITOR THAT INDUCES PROGRAMMED CELL DEATH AND SUPPRESSES CELL MIGRATION IN NON-SMALL CELL LUNG CARCINOMA CELLS." Biotechnologia Acta 16, no. 5 (2023): 34–44. http://dx.doi.org/10.15407/biotech16.05.034.

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Aim. PFKFB3 is glycolytic activators that is overexpressed in human lung cancer and plays a crucial role in multiple cellular functions including programmed cell death. Despite the many small molecules described as PFKFB3 inhibitors, some of them have shown disappointing results in vitro and in vivo. On the other hand KAN0438757, selective and potent, small molecule inhibitor has been developed. However, the effects of KAN0438757, in non-small cell lung carcinoma cells remain unknown. Herein, we sought to decipher the effect of KAN0438757 on proliferation, migration, DNA damage, and programmed

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5

Sarosiek, Kristopher. "Blocking cell death to enhance cell death." Science Translational Medicine 9, no. 408 (2017): eaao6129. http://dx.doi.org/10.1126/scitranslmed.aao6129.

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6

M, Aloysius Dhivya, Kishore A, and Bharathidevi SR. "Down-Regulation of NRF2 Signaling Triggers Cell Death in Y79 Cells upon Copper Chelation." Open Access Journal of Ophthalmology 10, no. 1 (2025): 1–10. https://doi.org/10.23880/oajo-16000329.

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Background: Retinoblastoma is a rare intraocular malignancy that leads to vision loss in children. While copper (Cu) chelation has been reported as a therapy in many cancers, its relevance to retinoblastoma has not yet been explored. Objectives: In this study, we have explored the role of penicillamine (a Cu chelator) as a therapeutic target for retinoblastoma using Y79 cells as a model. Methods: The effect of the Cu chelator on the viability of Y79 was assessed using the MTT assay. Additionally, we performed nuclear fractionation to assess Nuclear factor erythroid 2–related factor 2 (NRF2) ac

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7

Medema, J. P., H. Walczak, M. Hahne, and V. de Laurenzi. "Cell Death." Cell Death & Differentiation 17, no. 4 (2010): 730–32. http://dx.doi.org/10.1038/cdd.2010.11.

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8

Hotchkiss, Richard S., Andreas Strasser, Jonathan E. McDunn, and Paul E. Swanson. "Cell Death." New England Journal of Medicine 361, no. 16 (2009): 1570–83. http://dx.doi.org/10.1056/nejmra0901217.

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9

Vogel, Michael W. "Cell Death." American Journal of Psychiatry 162, no. 8 (2005): 1503. http://dx.doi.org/10.1176/appi.ajp.162.8.1503.

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10

MALORNI, WALTER, and GIANFRANCO DONELLI. "Cell Death." Annals of the New York Academy of Sciences 663, no. 1 Aging and Cel (1992): 218–33. http://dx.doi.org/10.1111/j.1749-6632.1992.tb38666.x.

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11

Giampietri, Claudia, Alessio Paone, and Alessio D’Alessio. "Cell Death." International Journal of Cell Biology 2014 (2014): 1–2. http://dx.doi.org/10.1155/2014/864062.

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12

Danial, Nika N., and Stanley J. Korsmeyer. "Cell Death." Cell 116, no. 2 (2004): 205–19. http://dx.doi.org/10.1016/s0092-8674(04)00046-7.

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13

Newton, Kim, Andreas Strasser, Nobuhiko Kayagaki, and Vishva M. Dixit. "Cell death." Cell 187, no. 2 (2024): 235–56. http://dx.doi.org/10.1016/j.cell.2023.11.044.

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14

Klishch, M., N. Skorokhyd, R. Panchuk, and R. Stoika. "Biochemical and cellular mechanisms of immunogenic cell death." Ukrainian Biochemical Journal 96, no. 6 (2024): 5–16. https://doi.org/10.15407/ubj96.06.005.

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Immunogenic cell death (ICD) is a mode of programmed cell death that leads to the activation of anticancer immune response and determines the long-term success of anticancer therapies. Here, we provide a review of the known molecular and cellular mechanisms of ICD. Usually, solid tumor experimental models have been used in ICD studies. However, ascites tumor models may possess some advantages over them. The results of our investigation on the approbation of murine Nemeth-Kellner lymphoma as an experimental ascites tumor model for ICD studies are presented. Keywords: ascites tumor model, bioche

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15

Momoi, Takashi. "Conformational Diseases and ER Stress-Mediated Cell Death: Apoptotic Cell Death and Autophagic Cell Death." Current Molecular Medicine 6, no. 1 (2006): 111–18. http://dx.doi.org/10.2174/156652406775574596.

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16

Panaretakis, T., L. Hjortsberg, J. M. R. Lambert, and B. Joseph. "Second Cell Death Network symposium: the vital cell death." Cell Death & Differentiation 16, no. 9 (2009): 1300–1302. http://dx.doi.org/10.1038/cdd.2009.93.

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17

Imig, Dirke, Karsten Kuritz, Nadine Pollak, Markus Rehm, and Frank Allgöwer. "Death patterns resulting from cell cycle-independent cell death." IFAC-PapersOnLine 51, no. 19 (2018): 90–93. http://dx.doi.org/10.1016/j.ifacol.2018.09.028.

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18

Maddox, Amy Shaub, and Jan M. Skotheim. "Cell cycle, cell division, cell death." Molecular Biology of the Cell 30, no. 6 (2019): 732. http://dx.doi.org/10.1091/mbc.e18-12-0819.

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19

Miura, Masayuki, Shin Hisahara, Riya Takano, Shin'ichi Shoji, and Hideyuki Okano. "Oligodendrocyte cell death by death factors." Neuroscience Research 31 (January 1998): S53. http://dx.doi.org/10.1016/s0168-0102(98)81804-x.

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20

Samali, Afshin, Simone Fulda, Adrienne M. Gorman, Osamu Hori, and Srinivasa M. Srinivasula. "Cell Stress and Cell Death." International Journal of Cell Biology 2010 (2010): 1–2. http://dx.doi.org/10.1155/2010/245803.

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21

Roy, Arun K. "Cell ageing and cell death." Trends in Biochemical Sciences 10, no. 9 (1985): 371–72. http://dx.doi.org/10.1016/0968-0004(85)90124-0.

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22

Pillai, Harikrishna, Anuraj K. S. Anuraj K.S, and Harikumar S. Kartha. "Guanine Nucleotide Oxidation and Cell Death by Bactericidal Antibiotics." Global Journal For Research Analysis 3, no. 7 (2012): 273–74. http://dx.doi.org/10.15373/22778160/july2014/98.

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23

Neves, A. A., and K. M. Brindle. "Imaging Cell Death." Journal of Nuclear Medicine 55, no. 1 (2014): 1–4. http://dx.doi.org/10.2967/jnumed.112.114264.

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24

Fulton, Alice B. "Programmed Cell Death." Science 274, no. 5284 (1996): 20. http://dx.doi.org/10.1126/science.274.5284.20.b.

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25

Novak, Jan. "Programmed Cell Death." Science 274, no. 5284 (1996): 20. http://dx.doi.org/10.1126/science.274.5284.20.a.

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26

Cahill, Larry, and Henry J. Haigler. "Hippocampal Cell Death." Science 272, no. 5266 (1996): 1251. http://dx.doi.org/10.1126/science.272.5266.1251.a.

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27

Zaidel, Dahlia W., and Margaret M. Esiri. "Hippocampal Cell Death." Science 272, no. 5266 (1996): 1249. http://dx.doi.org/10.1126/science.272.5266.1249.a.

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28

Landfield, Philip W., Bruce S. McEwen, Robert M. Sapolsky, and Michael J. Meaney. "Hippocampal Cell Death." Science 272, no. 5266 (1996): 1249–51. http://dx.doi.org/10.1126/science.272.5266.1249.b.

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29

Cahill, Larry, and Henry J. Haigler. "Hippocampal Cell Death." Science 272, no. 5266 (1996): 1251. http://dx.doi.org/10.1126/science.272.5266.1251-a.

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30

Landfield, Philip W., Bruce S. McEwen, Robert M. Sapolsky, and Michael J. Meaney. "Hippocampal Cell Death." Science 272, no. 5266 (1996): 1249–51. http://dx.doi.org/10.1126/science.272.5266.1249-b.

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31

Zaidel, Dahlia W., and Margaret M. Esiri. "Hippocampal Cell Death." Science 272, no. 5266 (1996): 1249. http://dx.doi.org/10.1126/science.272.5266.1249-a.

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32

Green, Douglas R., and Fabien Llambi. "Cell Death Signaling." Cold Spring Harbor Perspectives in Biology 7, no. 12 (2015): a006080. http://dx.doi.org/10.1101/cshperspect.a006080.

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33

David, Rachel. "'Cheating' cell death." Nature Reviews Molecular Cell Biology 11, no. 7 (2010): 462–63. http://dx.doi.org/10.1038/nrm2922.

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34

Garg, Abhishek D., Aleksandra M. Dudek-Peric, Erminia Romano, and Patrizia Agostinis. "Immunogenic cell death." International Journal of Developmental Biology 59, no. 1-2-3 (2015): 131–40. http://dx.doi.org/10.1387/ijdb.150061pa.

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35

Greenwood, Emma. "Survivin' cell death." Nature Reviews Cancer 1, no. 2 (2001): 97. http://dx.doi.org/10.1038/35101029.

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36

Sinclair, Meeghan. "Modeling cell death." Nature Biotechnology 18, no. 7 (2000): 703. http://dx.doi.org/10.1038/77234.

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37

Zaidel, D. W., M. M. Esiri;, P. W. Landfield, et al. "Hippocampal Cell Death." Science 272, no. 5266 (1996): 1247d—1251. http://dx.doi.org/10.1126/science.272.5266.1247d.

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38

Zaidel, D. W., and M. M. Esiri. "Hippocampal Cell Death." Science 272, no. 5266 (1996): 1249a. http://dx.doi.org/10.1126/science.272.5266.1249a.

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39

Landfield, P. W., B. S. McEwen, R. M. Sapolsky, and M. J. Meaney. "Hippocampal Cell Death." Science 272, no. 5266 (1996): 1249b—1251b. http://dx.doi.org/10.1126/science.272.5266.1249b.

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40

Cahill, L., and H. J. Haigler. "Hippocampal Cell Death." Science 272, no. 5266 (1996): 1251a. http://dx.doi.org/10.1126/science.272.5266.1251a.

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41

Medema, Jan Paul, Michael Hahne, Henning Walczak, and Vincenzo De Laurenzi. "Rocking cell death." Cell Death & Differentiation 6, no. 3 (1999): 297–300. http://dx.doi.org/10.1038/sj.cdd.4400480.

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42

Seçer, Merve Nur Şahan, and Mine Dosay-Akbulut. "Cell Death Mechanisms." Journal of Advances in Biology & Biotechnology 27, no. 11 (2024): 241–70. http://dx.doi.org/10.9734/jabb/2024/v27i111609.

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Cells and their building blocks ensure the maintenance of life by performing complex tasks such as taking in nutrients, excreting waste materials, producing energy, growth, division and reproduction. The form of intercellular communication and coordination is critical for the organism to grow, develop, and adapt to its environment optimally. The numerical balance of the cells that make up the organism is very important for it to survive in a healthy way. While new cells are being created in the living being, some of the existing cells are also eliminated through cell death, thus ensuring a sta

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43

Adler, E. M. "Silencing Cell Death?" Science Signaling 2007, no. 389 (2007): tw192. http://dx.doi.org/10.1126/stke.3892007tw192.

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44

Cory, S. "Cell death throes." Proceedings of the National Academy of Sciences 95, no. 21 (1998): 12077–79. http://dx.doi.org/10.1073/pnas.95.21.12077.

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45

Novak, J., A. B. Fulton, and J. C. Ameisen. "Programmed Cell Death." Science 274, no. 5284 (1996): 17–21. http://dx.doi.org/10.1126/science.274.5284.17c.

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46

Novak, J. "Programmed Cell Death." Science 274, no. 5284 (1996): 20. http://dx.doi.org/10.1126/science.274.5284.20.

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47

Fulton, A. B. "Programmed Cell Death." Science 274, no. 5284 (1996): 20. http://dx.doi.org/10.1126/science.274.5284.20-a.

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48

Novak, J. "Programmed Cell Death." Science 274, no. 5284 (1996): 20a. http://dx.doi.org/10.1126/science.274.5284.20a.

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49

Fulton, A. B. "Programmed Cell Death." Science 274, no. 5284 (1996): 20b. http://dx.doi.org/10.1126/science.274.5284.20b.

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50

Golstein, P. "Controlling Cell Death." Science 275, no. 5303 (1997): 1081–82. http://dx.doi.org/10.1126/science.275.5303.1081.

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