Bibliografias temáticas / Cisplatin

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Literatura científica selecionada sobre o tema "Cisplatin"

Autor: Grafiati
Publicado: 4 de junho de 2021
Última modificação: 25 de julho de 2025

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Artigos de revistas sobre o assunto "Cisplatin"

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Ozdemir-sanci, Tuba, and Ebru Alimogullari. "Effect of naringin and cisplatin combination on cell viability and cell death in bladder cancer cells." Journal of Research in Pharmacy 29, no. 2 (2025): 673–81. https://doi.org/10.12991/jrespharm.1664894.

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Bladder cancer is a prevalent malignancy characterized by high recurrence rates and limited therapeutic options, particularly due to resistance and toxicity associated with cisplatin therapy. Bladder cancer remains a significant global health concern, and while cisplatin is a cornerstone of treatment, its efficacy is often limited by resistance and toxicity. Therefore, there is a critical need for novel agents that can enhance cisplatin's effects while mitigating its drawbacks. This study investigates the potential of naringin, a natural flavonoid, to exhibit antiproliferative and proapoptotic
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Demirel, Özge, and Serap Şahin. "Duloksetin Hidroklorür’ün DU-145 İnsan Prostat Kanser Hücreleri Üzerine Sitotoksik Etkisinin Araştırılması." Black Sea Journal of Health Science 8, no. 4 (2025): 132–38. https://doi.org/10.19127/bshealthscience.1649481.

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Prostat kanseri (PKa), Dünya çapında en yaygın kanserlerden birisi olup kansere bağlı ölümlerin büyük bir kısmını oluşturmaktadır. Bir serotonin-nöradrenalin geri alım intibitörü (SNGI) olan duloksetin, hem norepinefrin hem de serotoninin geri alımının inhibisyonu yoluyla antidepresan bir rol oynamaktadır. Duloksetin hidroklorürün DU-145 prostat kanser hücrelri üzerine sitotoksik etkisi ile ilgili çalışma bulunmamaktadır. Çalışmamızda, duloksetin hidroklorür ve cisplatinin DU-145 prostat kanseri ve L-929 sağlıklı fare fibroblast hücreleri üzerine sitotoksik etkileri 3-[4,5-Dimetiltiyazol-2-İl]
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ATAMAN, J. E., and D. BAXTER-GRILLO. "MORPHOLOGICAL EVALUATION OF CISPLATIN-INDUCED TESTICULAR DAMAGE IN WISTAR RATS." Nigerian Journal of Life Sciences (ISSN: 2276-7029) 4, no. 1 (2022): 16–25. http://dx.doi.org/10.52417/njls.v4i1.151.

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Twenty-one Wistar rats weighing 200 - 250 mg were categorized into three treatment groups- the control, saline and the cisplatin groups, each comprising of seven rats per group, to assess the effects of cisplatin-induced testicular damage in Wistar rats. The control group received feed mash and water ad libitum, the saline group received equal volumes of physiological saline intraperitoneally with normal feeds and water, while the cisplastin group received 8 mg/kg body weight of cisplastin only for five days and had normal feeds and water for twelve weeks before sacrifice. The effects of these
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Monroe, Jerry D., Denis Hodzic, Matthew H. Millay, Blaine G. Patty, and Michael E. Smith. "Anti-Cancer and Ototoxicity Characteristics of the Curcuminoids, CLEFMA and EF24, in Combination with Cisplatin." Molecules 24, no. 21 (2019): 3889. http://dx.doi.org/10.3390/molecules24213889.

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In this study, we investigated whether the curcuminoids, CLEFMA and EF24, improved cisplatin efficacy and reduced cisplatin ototoxicity. We used the lung cancer cell line, A549, to determine the effects of the curcuminoids and cisplatin on cell viability and several apoptotic signaling mechanisms. Cellular viability was measured using the MTT assay. A scratch assay was used to measure cell migration and fluorescent spectrophotometry to measure reactive oxygen species (ROS) production. Western blots and luminescence assays were used to measure the expression and activity of apoptosis-inducing f
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Heiserman, James Patrick, Zenab Minhas, Elahe Nikpayam, and Dong-Joo Cheon. "Targeting Heat Shock Protein 27 and Fatty Acid Oxidation Augments Cisplatin Treatment in Cisplatin-Resistant Ovarian Cancer Cell Lines." International Journal of Molecular Sciences 24, no. 16 (2023): 12638. http://dx.doi.org/10.3390/ijms241612638.

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Most ovarian cancer patients develop recurrent cancers which are often resistant to commonly employed chemotherapy agents, such as cisplatin. We have previously shown that the inhibition of heat shock protein 27 (HSP27) or fatty acid oxidation (FAO) sensitizes cisplatin-resistant ovarian cancer cell lines to cisplatin and dual inhibition of both HSP27 and FAO induces substantial cell death in vitro. However, it is unclear how HSP27 and FAO promote cisplatin resistance, and if dual inhibition of both HSP27 and FAO would augment cisplatin treatment in vivo. Here we showed that HSP27 knockdown in
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CHEN, PING, QING-SHENG WU, YA-PING DING, and ZI-CHUN ZHU. "PREPARATION OF CISPLATIN COMPOSITE MICRO/NANOFIBERS AND ANTITUMOR ACTIVITY IN VITRO AGAINST HUMAN TUMOR spc-a-1 CELLS." Nano 06, no. 04 (2011): 325–32. http://dx.doi.org/10.1142/s1793292011002688.

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In this paper, the cisplatin composite micro/nanofibers were prepared by electrospinning. Average diameter of the typical products was about 700 nm, and cisplatins were incorporated in biodegradable poly (L-lactic acid) fibers. The controlled release of cisplatin can be gained for long time. The possible mechanisms of cisplatin release in the PBS and the PBS with proteinase K were discussed. 3-(4, 5)-dimethylthiahiazo-(-z-y1)-3, 5-di-phenytetrazoliumromide (MTT) method was used to test antitumor activities in vitro against human lung tumor spc-a-1 cells. When incubation time was 24 h, the same
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Moon, Hyeon-Min, Jin-Sung Park, Il-Buem Lee, et al. "Cisplatin fastens chromatin irreversibly even at a high chloride concentration." Nucleic Acids Research 49, no. 21 (2021): 12035–47. http://dx.doi.org/10.1093/nar/gkab922.

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Abstract Cisplatin is one of the most potent anti-cancer drugs developed so far. Recent studies highlighted several intriguing roles of histones in cisplatin's anti-cancer effect. Thus, the effect of nucleosome formation should be considered to give a better account of the anti-cancer effect of cisplatin. Here we investigated this important issue via single-molecule measurements. Surprisingly, the reduced activity of cisplatin under [NaCl] = 180 mM, corresponding to the total concentration of cellular ionic species, is still sufficient to impair the integrity of a nucleosome by retaining its c
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Karayay, Betül, Heidi Olze, and Agnieszka J. Szczepek. "Degranulation of Murine Resident Cochlear Mast Cells: A Possible Factor Contributing to Cisplatin-Induced Ototoxicity and Neurotoxicity." International Journal of Molecular Sciences 24, no. 5 (2023): 4620. http://dx.doi.org/10.3390/ijms24054620.

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Permanent hearing loss is one of cisplatin’s adverse effects, affecting 30–60% of cancer patients treated with that drug. Our research group recently identified resident mast cells in rodents’ cochleae and observed that the number of mast cells changed upon adding cisplatin to cochlear explants. Here, we followed that observation and found that the murine cochlear mast cells degranulate in response to cisplatin and that the mast cell stabilizer cromoglicic acid (cromolyn) inhibits this process. Additionally, cromolyn significantly prevented cisplatin-induced loss of auditory hair cells and spi
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Bridget, SAMUEL1 Victor SAMUEL2* Otuamiobhedio Messiah WILFRED3 Victor Nnaemeka OGBONNA4 Igbanam Michael URANGIKOR5 Eugene ITIRI6 Emeka Emmanuel EZEALISIOBI7 Chinemerem Ulu IKPE8 Bliss ANYALEBECHI9 Benita Basil EGBE10. "CISPLATIN-INDUCED NEPHROTOXICITY IN WISTAR RATS." ISRG Journal of Clinical Medicine and Medical Research [ISRGJCMMR] II, no. II (2025): 5–9. https://doi.org/10.5281/zenodo.14955099.

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<strong>Abstract</strong> <em>This study examined the morphological changes in the kidneys of Wistar rats exposed to cisplatin over short-term (14 days) and long-term (28 days) periods. We randomly assigned 25 albino rats to three groups: a control group (Group A) receiving distilled water and rat pellets, a low-dose cisplatin group (Group B) receiving 7.5mg/ml of cisplatin twice weekly, and a high-dose cisplatin group (Group C) receiving 16mg/ml of cisplatin twice weekly. 24 hours after the last treatment, animals were sacrificed and kidneys were collected into sterile bottles for histologica
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Kasturi Pangarkar. "Development of cisplatin as an anti-cancer drug." GSC Advanced Research and Reviews 23, no. 1 (2025): 005–11. https://doi.org/10.30574/gscarr.2025.23.1.0101.

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Cisplatin, a platinum-based chemotherapeutic agent, has been a cornerstone in the treatment of various cancers since its FDA approval in 1978. It is particularly effective against cancers such as ovarian, colorectal, prostate, bladder, and non-small cell lung cancer (NSCLC). Cisplatin exerts its anticancer effects by forming platinum-DNA adducts that inhibit DNA replication and transcription, leading to cell cycle arrest and apoptosis. However, its clinical application is limited by severe side effects, including renal toxicity, nausea, vomiting, and the development of drug resistance. To miti
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Teses / dissertações sobre o assunto "Cisplatin"

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Amaral, Catia Lira do. "Efeito do resveratrol na nefrotoxicidade induzida pela cisplatina em ratos." Universidade de São Paulo, 2006. http://www.teses.usp.br/teses/disponiveis/60/60134/tde-22052007-090133/.

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O resveratrol (Res), um polifenol presente no vinho tinto, é conhecido por possuir potente atividade antioxidante. O efeito do resveratrol (Res) frente à nefrotoxicidade do antineoplásico cisplatina (cDDP) foi avaliado em ratos neste estudo. Os animais foram tratados com Res (25 mg/Kg de peso copóreo, ip., dose única) 30 minutos antes da administração de cisplatina (5 mg/Kg de peso copóreo, ip., dose única) e foram sacrificados depois de 2 ou 5 dias do tratamento. Após 5 dias, o aumento da creatinina sérica, volume urinário e proteinúria, que são marcadores de alterações renais, apresentaram s
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Pan, Zhenrui. "Functional characterization of the GATOR1 complex in cisplatin resistance." Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASL053.

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L'administration de cisplatine constitue la principale approche de chimiothérapie pour de nombreux cancers épithéliaux. Cependant, la résistance à ce médicament pose un défi considérable à un traitement efficace. Malgré l'identification de nombreux facteurs associés à la résistance aux médicaments, des biomarqueurs fiables permettant de prédire la réponse au traitement restent insaisissables. Auparavant, une faible expression du suppresseur de tumeur NPRL2 était liée à la résistance au cisplatine. NPRL2, ainsi que NPRL3 et DEPDC5, forment le complexe GATOR1, un régulateur en amont du complexe
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Santos, Graciela Cristina dos [UNESP]. "Avaliação do efeito protetor do urucum e da bixina sobre a genotoxicidade induzida pelo antitumoral cisplatina em células da linhagem PC12." Universidade Estadual Paulista (UNESP), 2008. http://hdl.handle.net/11449/100973.

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Made available in DSpace on 2014-06-11T19:31:05Z (GMT). No. of bitstreams: 0 Previous issue date: 2008-12-12Bitstream added on 2014-06-13T18:41:39Z : No. of bitstreams: 1 santos_gc_dr_arafcf.pdf: 2386901 bytes, checksum: b46aa55b36f051a8683831df1b69f150 (MD5)<br>Universidade Estadual Paulista (UNESP)<br>A neuropatia induzida por drogas quimioterápicas é uma complicação no tratamento do câncer e outras doenças por ser freqüentemente dolorosa e requerer a interrupção da terapia. O antitumoral cisplatina é comumente usado contra muitas formas de câncer há aproximadamente 40 anos. Entretanto, su
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Santos, Graciela Cristina dos. "Avaliação do efeito protetor do urucum e da bixina sobre a genotoxicidade induzida pelo antitumoral cisplatina em células da linhagem PC12 /." Araraquara : [s.n.], 2008. http://hdl.handle.net/11449/100973.

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Resumo: A neuropatia induzida por drogas quimioterápicas é uma complicação no tratamento do câncer e outras doenças por ser freqüentemente dolorosa e requerer a interrupção da terapia. O antitumoral cisplatina é comumente usado contra muitas formas de câncer há aproximadamente 40 anos. Entretanto, sua aplicação é associada a muitos efeitos tóxicos, como neurotoxicidade, nefrotoxicidade, perda da audição e vômitos. Estes efeitos adversos têm levado ao desenvolvimento de agentes específicos para amenizar a toxicidade do fármaco. Alguns estudos sugerem que a administração de antioxidantes é capaz
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Kullmann, Maximilian [Verfasser]. "Identifying intracellular cisplatin interaction partners and assessing their contribution to cisplatin resistance / Maximilian Kullmann." Bonn : Universitäts- und Landesbibliothek Bonn, 2016. http://d-nb.info/111988876X/34.

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Hadi, Sutopo. "The chemistry of cisplatin metabolites /." [St. Lucia, Qld.], 2007. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19800.pdf.

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Zhang, Jin-Gang. "Cisplatin nephrotoxicity : mechanisms and antidotes." Thesis, University of Liverpool, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.307635.

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Castro, João Humberto Teotônio de [UNESP]. "Avaliação do espermograma de cães submetidos à administração de cisplatina." Universidade Estadual Paulista (UNESP), 2007. http://hdl.handle.net/11449/89030.

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Made available in DSpace on 2014-06-11T19:23:42Z (GMT). No. of bitstreams: 0 Previous issue date: 2007-02-28Bitstream added on 2014-06-13T18:51:10Z : No. of bitstreams: 1 castro_jht_me_jabo.pdf: 401295 bytes, checksum: 5932eead976a77a78dee0fb6c3fb9169 (MD5)<br>Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)<br>A correta orientação do Médico Veterinário, aos proprietários de cães, usados com finalidades reprodutivas, submetidas à quimioterapia com cisplatina, é importante na medida que este agente citostático age nas células em constante divisão, podendo ser citotóxicos pa
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Oliva, Carlos Alfredo Calpa [UNESP]. "Hemograma e teores séricos de Na, K, Mg, Ca e P de cães hígidos submetidos à administração de cisplatina." Universidade Estadual Paulista (UNESP), 2007. http://hdl.handle.net/11449/89087.

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Made available in DSpace on 2014-06-11T19:23:43Z (GMT). No. of bitstreams: 0 Previous issue date: 2007-06-19Bitstream added on 2014-06-13T18:51:13Z : No. of bitstreams: 1 oliva_cac_me_jabo.pdf: 178983 bytes, checksum: f310a85e1eb63818fa51dba6cafeadd8 (MD5)<br>A cisplatina é um fármaco antineoplásico utilizado como adjuvante no tratamento de diversas neoplasias. Neste estudo foram avaliados o hemograma e os teores de sódio, potássio, magnésio, cálcio e fósforo do soro sanguíneo de cães submetidos à terapia com cisplatina. Foram utilizados oito cães, machos, sem raça definida, com 10 a 15 kg d
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Li, Yan Julia. "Cisplatin-induced cytotoxicity in MDCK cells." Thesis, University of Ottawa (Canada), 2002. http://hdl.handle.net/10393/6408.

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Background. The mechanism of cisplatin-induced nephrotoxicity is not well understood. The distal tubules are affected in both human and animal studies, although the majority of cisplatin-induced renal damage is in proximal tubules. Platinum (Pt) forms intra- and interstrand cross-links with DNA in cancer cells. Hypothesis. A mechanism of cisplatin-induced cytotoxicity in MDCK cells relates to its ability to bind to DNA and interfere with its synthesis. Methods. The canine distal renal tubular epithelial cell line, MDCK was used as an in vitro model to investigate the mechanism of cisplatin-ind
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Livros sobre o assunto "Cisplatin"

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Ozols, Robert F. High-dose Platinol (cisplatin for infection) in hypertonic saline. Bristol-Myers Oncology Division, 1985.

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Yoshihiro, Kikuchi, ed. Mechanism of cisplatin resistance and its circumvention. Nova Science Publishers, 1998.

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Chu, Wendy. Mechanism of cisplatin resistance inhuman malignant melanoma. National Library of Canada, 1998.

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Merazga, Yamina. Aspects of the interaction between cisplatin and renal glutathione. Brunel University, 1990.

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1946-, Lippert Bernhard, ed. Cisplatin: Chemistry and biochemistry of a leading anticancer drug. Verlag Helvetica Chimica Acta, 1999.

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Morley, Christopher. Analogues of cisplatin from diamino carbohydrates and related compounds. University of East Anglia, 1986.

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Evans, Dyfed Llyr. The induction of apoptosis by the anticancer agent cisplatin. Universityof Manchester, 1994.

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Laurell, Göran. Ototoxicity of the anticancer drug cisplatin: Clinical and experimental aspects. Distributed by Almqvist & Wiksell Periodical Co., 1991.

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Institut für Qualität und Wirtschaftlichkeit im Gesundheitswesen. Pembrolizumab (nicht kleinzelliges Lungenkarzinom: Nutzenbewertung gemäß § 35a SGB V. Institut für Qualität und Wirtschaftlichkeit im Gesundheitswesen, 2017.

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Bryant, Eric Edward. Systems Genetics of DNA Damage Tolerance – Cisplatin, RAD5 & CRISPR-mediated Nonsense. [publisher not identified], 2019.

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Capítulos de livros sobre o assunto "Cisplatin"

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Rosenberg, Barnett. "Platinum Complexes for the Treatment of Cancer: Why the Search Goes On." In Cisplatin. Verlag Helvetica Chimica Acta, 2006. http://dx.doi.org/10.1002/9783906390420.ch1.

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Ano, Susan O., Zsuzsanna Kuklenyik, and Luigi G. Marzilli. "Structure and Dynamics of Pt Anticancer Drug Adducts from Nucleotides to Oligonucleotides as Revealed by NMR Methods." In Cisplatin. Verlag Helvetica Chimica Acta, 2006. http://dx.doi.org/10.1002/9783906390420.ch10.

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Chen, Yu, Zijian Guo, and Peter J. Sadler. "195Pt- and 15N-NMR Spectroscopic Studies of Cisplatin Reactions with Biomolecules." In Cisplatin. Verlag Helvetica Chimica Acta, 2006. http://dx.doi.org/10.1002/9783906390420.ch11.

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Bau, Robert, and Michal Sabat. "Structural Aspects of Pt-Purine Interactions: From Models to DNA." In Cisplatin. Verlag Helvetica Chimica Acta, 2006. http://dx.doi.org/10.1002/9783906390420.ch12.

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Reedijk, Jan, and Jan Maarten Teuben. "Platinum-Sulfur Interactions Involved in Antitumor Drugs, Rescue Agents, and Biomolecules." In Cisplatin. Verlag Helvetica Chimica Acta, 2006. http://dx.doi.org/10.1002/9783906390420.ch13.

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Appleton, Trevor G. "Diammine- and Diamineplatinum Complexes with Non-Sulfur-Containing Amino Acids and Peptides." In Cisplatin. Verlag Helvetica Chimica Acta, 2006. http://dx.doi.org/10.1002/9783906390420.ch14.

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Lippert, Bernhard. "Platinum Blues: On the Way toward Unraveling a Mystery." In Cisplatin. Verlag Helvetica Chimica Acta, 2006. http://dx.doi.org/10.1002/9783906390420.ch15.

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Randaccio, Lucio, and Ennio Zangrando. "Heteronuclear PtII Complexes with Pyrimidine Nucleobases." In Cisplatin. Verlag Helvetica Chimica Acta, 2006. http://dx.doi.org/10.1002/9783906390420.ch16.

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Natile, Giovanni, Francesco P. Intini, and Concetta Pacifico. "Diplatinum(III) Complexes: Chemical Species More Widely Spread Than Suspected." In Cisplatin. Verlag Helvetica Chimica Acta, 2006. http://dx.doi.org/10.1002/9783906390420.ch17.

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Matsumoto, Kazuko. "Inorganic and Organometallic Chemistry of Cisplatin-Derived Diplatinum(III) Complexes." In Cisplatin. Verlag Helvetica Chimica Acta, 2006. http://dx.doi.org/10.1002/9783906390420.ch18.

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Trabalhos de conferências sobre o assunto "Cisplatin"

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Absar, Saheem, Mujibur Khan, Kyle Edwards, and David Calamas. "Electrospinning of Cisplatin-Loaded Cellulose Nanofibers for Cancer Drug Delivery." In ASME 2014 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/imece2014-37182.

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Cellulosic nanofibers have been electrospun with an antitumor agent Cisplatin. Cellulose acetate (CA) and Cisplatin were co-electrospun using a coaxial electrospinning system. For the outer sheath, a solution of 7.5wt% CA in Acetone and DMAc (2:1) was used. The inner core consisted of Cisplatin dissolved in DMF at a concentration of 5mg/ml. Drug-loaded nanofibers from Cellulose pulp (2wt%) dissolved in NMMO. H2O were also produced. The solutions were electrospun in a high voltage electric field of 25–30 kV. Characterization of neat and drug-loaded nanofibers was performed using Scanning electr
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Camargo, Luana Cristina, Joao Paulo Figueiro Longo, Karen Letycia Rodrigues de Paiva, Marina Mesquita Simões, Thais Bergmann, and Victor Carlos Mello da Silva. "Immunotherapy vaccines for triple-negative breast cancer and its influence on the tumor microenvironment." In Brazilian Breast Cancer Symposium 2023. Mastology, 2023. http://dx.doi.org/10.29289/259453942023v33s1024.

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Objective: Cancer is still a complex and debilitating disease even though advances in treatment have occurred. Triplenegative breast cancer (TNBC) is an aggressive subtype of breast cancer with a poor prognosis and occurs more frequently in young women. Due to its metastatic features and unique tumor microenvironment, TNBC treatment is limited. In this study, we evaluated how three chemotherapy drugs could be used to produce vaccines with cells under immunogenic cell death. Methodology: For that, 4T1-luc2 cells were treated with cisplatin (100 μM), mitoxantrone (MTX) (15 μM), and doxorubicin (
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Lau, KingWun. "Brief summary of cisplatin nephrotoxicity." In International Conference on Biological Engineering and Medical Science (ICBIOMed2022), edited by Gary Royle and Steven M. Lipkin. SPIE, 2023. http://dx.doi.org/10.1117/12.2669859.

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Soodvilai, Sirima, Sunhapas Soodvilai, Warayuth Sajomsang, Theerasak Rojanarata, Prasopchai Patrojanasophon, and Praneet Opanasopit. "Chitosan Polymeric Micelles for Prevention of Cisplatin-Induced Nephrotoxicity and Anticancer Activity of Cisplatin." In ICBET 2020: 2020 10th International Conference on Biomedical Engineering and Technology. ACM, 2020. http://dx.doi.org/10.1145/3397391.3397438.

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Lee, Chunman, Masao Sasai, and Yoshiki Sawa. "Abstract 4452: Controlled release complex of cisplatin for mesothelioma: gamma-PGA/Cisplatin complex formulation." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-4452.

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Liang, Xiaobing, Michael D. Mueller, and Jing Jie Yu. "Abstract 2982: Activation of checkpoint kinase Chk2 by cisplatin and its role in cisplatin resistance." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2982.

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Abdullah, N., N. Al Balushi, S. Al-Bahlani, et al. "EP765 The effect of malformina1 on cisplatin-sensitive and cisplatin-resistant ovarian cancer cell lines." In ESGO Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-esgo.816.

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Larisch, C., T. Markowiak, S. Golovchenko, et al. "Dosis-abhängige Cisplatin-Konzentration und -Eindringtiefe in humanes Lungengewebe bei Inkubation in hyperthermer Cisplatin-Lösung." In 32. Jahrestagung der Deutschen Gesellschaft für Thoraxchirurgie. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1771119.

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Gupta, Vikas, Ashok Kumar Chauhan, Paramjeet Kaur, Anil Khurana, Yashpal Verma, and Nupur Bansal. "Comparative evaluation of concomitant chemoradiation with weekly cisplatin and gemcitabine versus weekly cisplatin in the management of locally advanced carcinoma of uterine cervix." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685264.

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Aim: To evaluate feasibility of concomitant chemoradiation with weekly cisplatin and gemcitabine, and comparing the advantage of using this regimen over cisplatin alone in terms of disease control and toxicities in management of locally advanced carcinoma cervix. Materials and Methods: The study has been conducted on fifty previously untreated, histopathologically proven FIGO stage II B - IV A patients of carcinoma cervix, attending the Department of Radiotherapy, Post Graduate Institute of Medical Sciences, Rohtak for definitive treatment by radiation therapy. The patients were divided random
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Richards, Elizabeth J., William O. Cookson, Sanjay Popat, and Miriam F. Moffatt. "Transcriptome Changes Accompanying Induced Cisplatin Resistance." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4918.

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Relatórios de organizações sobre o assunto "Cisplatin"

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Turchi, John J. Proteomic Analysis of Cisplatin-Resistant Ovarian Cancer. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada425620.

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Turchi, John. Proteomic Analysis of Cisplatin-Resistant Ovarian Concers. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada462560.

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Turchi, John J. Proteomic Analysis of Cisplatin-Resistant Ovarian Concers. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada463194.

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Balaji, Kethandapatti C. MT 2A Phosphorylation by PKC Mu/PKD Influences Chemosensitivity to Cisplatin in Prostate Cancer. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada495664.

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Allworth, Ann E. The Role of Terbium and Gadolinium in Reversal of Cisplatin Resistance in Cultured Human Breast Cancer Cells. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada414791.

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Miao, Fang, Yaru Guo, Yan Yuan, Juzhou Chen, and Yong Xin. Bevacizumab combined with pemetrexed plus carboplatin or cisplatin in the treatment of malignant pleural effusion of lung cancer : A meta-analysis of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.4.0096.

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Wang, Dandan, Shuaihang Hu, Kangdi Cao, Chenxi Qiao, Zhuo Wang, and Wei Hou. Clinical efficacy and safety of Chinese herb injections combination with Docetaxel combined with cisplatin (DP) chemotherapy for advanced non-small cell lung cancer: A protocol for Bayesian network meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.9.0081.

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Li, Qian, Hua Xiao, Ren-long Liang, et al. Efficacy and safety of Cinobufacini injection combined with vinorelbine and cisplatin regimen chemotherapy for stage III/IV non-small cell lung cancer A protocol for systematic review and meta-analysis of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.6.0091.

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Parikh, Romil R., Alexander Troester, Bronwyn Southwell, et al. Treatment of Stages I–III Squamous Cell Anal Cancer: A Systematic Review. Agency for Healthcare Research and Quality (AHRQ), 2024. http://dx.doi.org/10.23970/ahrqepccer273.

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Objectives. To evaluate the comparative effectiveness and harms of initial treatment and posttreatment surveillance strategies for stages I–III squamous cell anal cancer. Data sources. MEDLINE®, Embase®, Cochrane Register of Controlled Trials, and ClinicalTrials.gov from January 2000 through March 2024; reference lists of systematic reviews and included studies; and a Federal Register notice. Review methods. Using predefined criteria and dual review, we selected randomized controlled trials (RCTs) and nonrandomized studies of interventions (NRSIs) comparing strategies for chemotherapy, radiati
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Health hazard evaluation report: HETA-2009-0121-3106, evaluation of exposures to healthcare personnel from cisplatin during a mock interperitoneal operation, University Medical Center, Las Vegas, Nevada. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 2010. http://dx.doi.org/10.26616/nioshheta200901213106.

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