Bibliografias temáticas / Endometrial hyperplasia

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Literatura científica selecionada sobre o tema "Endometrial hyperplasia"

Autor: Grafiati
Publicado: 4 de junho de 2021
Última modificação: 22 de fevereiro de 2023

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Artigos de revistas sobre o assunto "Endometrial hyperplasia"

1

Arai, Y., e M. Nishida. "Differential diagnosis between normal endometrium and endometrial hyperplasia with immunostaining cytology using anti-LeY monoclonal antibody". International Journal of Gynecologic Cancer 13, n.º 1 (2003): 42–46. http://dx.doi.org/10.1136/ijgc-00009577-200301000-00008.

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We have previously reported that both endometrial cancer and endometrial hyperplasia stain positively for the anti-LeY monoclonal antibody, whereas normal endometrium does not. Endometrial hyperplasia is a premalignant change associated with the eventual development of endometrial carcinoma. However, it can be difficult to differentiate hyperplasia from normal endometrium in cytology. This study illustrates the use of immunocytochemical cytology using anti-LeY monoclonal antibody to differentiate between endometrial hyperplasia and normal endometrium. Immunostaining using anti-LeY monoclonal antibody was performed on cytologic specimens obtained from 17 normal endometria, 25 endometria with endometrial hyperplasia, and 13 endometria with endometrial carcinoma. All normal endometria displayed negative staining for anti-LeY monoclonal antibody, whereas all endometria with endometrial carcinoma displayed positive staining. Of the endometrial hyperplasia cases, 21 displayed positive staining. However, four displayed negative staining due to the small number of cells available for diagnosis. We believe that immunostaining cytology using anti-LeY monoclonal antibody is a useful method for differentiating between normal endometrium and endometrial hyperplasia.
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Quddus, M. Ruhul, Predrag Latkovich, William J. Castellani, C. James Sung, Margaret M. Steinhoff, Robert C. Briggs e Roberto N. Miranda. "Expression of Cyclin D1 in Normal, Metaplastic, Hyperplastic Endometrium and Endometrioid Carcinoma Suggests a Role in Endometrial Carcinogenesis". Archives of Pathology & Laboratory Medicine 126, n.º 4 (1 de abril de 2002): 459–63. http://dx.doi.org/10.5858/2002-126-0459-eocdin.

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Abstract Context.—Endometrioid carcinoma is often preceded by characteristic histopathologic lesions known as endometrial hyperplasia. Estrogen appears to be involved in the development of endometrioid carcinoma. Other mechanisms of endometrial carcinogenesis include mutations in p53 and PTEN tumor suppressor genes and overexpression of cyclin D1. However, the pattern of cyclin D1 expression is not well defined in normal, hyperplastic, neoplastic, and metaplastic endometrium. Design.—Cyclin D1 immunohistochemical analysis was used to evaluate 108 fixed, paraffin-embedded endometrial biopsy specimens and uterine resections obtained from 108 patients. Specimens included proliferative and secretory endometria, simple and complex hyperplastic lesions, and endometrioid adenocarcinoma. Normal and metaplastic surface epithelia were also evaluated independently of glandular morphologic features. Results.—Cyclin D1 was significantly overexpressed in glands with complex hyperplasia and endometrioid adenocarcinoma compared with proliferative or secretory endometrium and simple hyperplasia. Significant overexpression was also noted in papillary, syncytial, and squamous metaplasias compared with normal surface epithelium or epithelium with tubal metaplasia. Conclusion.—Overexpression of cyclin D1 increases from normal endometrium to hyperplasia and carcinoma, suggesting that it may play a role in endometrial carcinogenesis. Overexpression of cyclin D1 in endometrial glands was independent from overexpression of cyclin D1 in surface metaplastic epithelium.
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Benyuk, V., V. Goncharenko e O. Zabudskyi. "Features of the post-operating period after the endometrial ablation in post-menopausal aged women with hyperplastic processes of endometrium". HEALTH OF WOMAN, n.º 6(142) (29 de julho de 2019): 60–67. http://dx.doi.org/10.15574/hw.2019.142.60.

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The paper presents the results of surgical treatment of 45 postmenopausal women with non-atypical forms of endometrial hyperplasia. The objective: is to establish the features of the postoperative period depending on the method of endometrial ablation in postmenopausal patients with endometrial hyperplastic processes. Materials and methods. The study included 45 women with non-atypical forms of endometrial hyperplasia: 25 women of postmenopausal age with non-atypical forms of endometrial hyperplasia, who were treated with hysteroscopic monopolar ablation of the endometrium and 20 women of postmenopausal age with non-atypical forms of endometrial hyperplasia, who underwent radiowave endometrial ablation. In the postoperative period, on 3, 7 days and after 1, 3, 6, 12, 24 months, the complaints, the general condition, the nature of the discharge from the genital tract were evaluated, gynecological examination with ultrasound examination of the pelvis were provided, office hysteroscopy or endometrial aspiration from the uterus were provided according to indications. Results. It is advisable to recommend endometrial ablation for the patients with non-atypical forms of postmenopausal endometrial hyperplasia as an alternative to hormonal treatment or hysterectomy. The efficiency of radiowave ablation of the endometrium with non-atypical forms of endometrial hyperplasia is 85.0%. The effectiveness of monopolar hysteroscopic ablation of the endometrium in women with non-atypical forms of endometrial hyperplasia is 96.0%. In the case of recurrence of the endometrial hyperplastic process (bleeding, thickening of the M-echo according to ultrasound data), hysteroscopy is shown with mandatory hystopathological studies and diagnosis verification. Conclusions. After ablation of the endometrium, follow-up is advisable for two years. The method of choice for follow-up observation of the state of the endometrium of the uterus in women after endometrial ablation is transvaginal ultrasound, which is performed after 1, 3, 6, 12 and 24 months of observation. Key words: endometrial hyperplastic processes, postmenopausal women, endometrial ablation.
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Krut, Yuri, Nataly Zemlyana, Natalie Gaidai, Mykhailo Pavliuchenko e Iryna Amro. "The proliferation markers in patients with different forms of hyperplastic endometrial processes". Journal of Education, Health and Sport 12, n.º 6 (5 de maio de 2022): 80–86. http://dx.doi.org/10.12775/jehs.2022.12.06.007.

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Introduction: Endometrial hyperplastic processes (EHP) are characterized by recurrent course, high risk of malignancy, difficulties in diagnosing and predicting the course, which determines their great medical and social significance Objective: To evaluate the levels of proliferation markers Ki-67 and APRIL in serum of women with various forms of endometrial hyperplastic processes (EHP) and their correlation with clinical and anamnestic features. Materials and Methods: The study examined 95 women with endometrial hyperplasia and endometrial adenocarcinoma who underwent treatment in the gynecology department "City Clinical Hospital №7» in Zaporizhzhya and "Zaporozhye Regional Clinical Oncology Center". The first group consisted of 32 women with simple endometrial hyperplasia. The second group included 43 patients with complex endometrial hyperplasia. The third group consisted of 22 women with endometrial adenocarcinoma. Results: The analysis of Ki-67 level revealed an increasing in the patients with simple hyperplasia (p <0.05) and complex hyperplasia of endometrium (p <0.05) compare with control group. The level of the APRIL in serum is increased in group of complex hyperplasia compared with the control group (p <0,05) and simple hyperplasia (p <0,05). Conclusions: Level of APRIL in serum increased in patients with complex endometrial hyperplasia and endometrial adenocarcinoma. Level of Ki-67 in serum was highest in patients with complex endometrial hyperplasia. We found a significant positive association between Ki-67 levels in serum and thickness of M-echo in endometrium, duration of hyperplastic endometrial processes anamnesis, between APRIL in serum and age of patients, duration of hyperplastic endometrial processes anamnesis.
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Xu, Jian-Ying, Wei-Jie Zhu, Xiao-Zhe Cao, Xian-Feng Li e Jin Wu. "Aberrant Expression of the Von Hippel-Lindau Gene in Human Endometrial Hyperplasia and Endometrial Carcinoma". International Journal of Gynecologic Cancer 21, n.º 3 (abril de 2011): 430–34. http://dx.doi.org/10.1097/igc.0b013e31820c5903.

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IntroductionThe purpose of this study was to determine whether aberrant expression of the von Hippel-Lindau (VHL) gene in human hyperplastic and malignant endometrial tissues was involved in endometrial carcinogenesis.MethodsFresh tissue samples of endometrial hyperplasia consisting of simple (n = 26), complex (n = 23), and atypical hyperplasia (n = 20); endometrial carcinoma (n = 17); and normal endometrium (n = 40) were measured using Western blotting and real-time reverse transcription polymerase chain reaction. Paraffin-embedded sections of endometrial hyperplasia (n = 90), endometrial carcinoma (n = 30), and normal endometrium (n = 60) were detected by immunohistochemical method.ResultsVon Hippel-Lindau staining was present in the cytoplasm of epithelial cells and stroma. A decreased expression of VHL mRNA in endometrial hyperplasia from simple, complex, to atypical hyperplasia was observed. There were statistical differences on VHL messenger RNA (mRNA) levels among simple, complex, and atypical hyperplasia (P < 0.01). The VHL mRNA levels in endometrial carcinoma were significantly lower than those in normal endometrium, simple hyperplasia, or complex hyperplasia (P < 0.01) but similar to those in atypical hyperplasia (P > 0.05). Von Hippel-Lindau protein levels by Western blotting and staining intensity by immunohistochemistry were coincident with the VHL mRNA levels.ConclusionsAberrant expression of the VHL gene is associated with the risk of endometrial hyperplasia progressing to endometrial carcinoma, and its expression levels are useful as a predictive indicator for endometrial carcinoma.
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Levakov, S. A., N. A. Sheshukova, A. G. Kedrova, A. S. Fedotova e E. A. Obukhova. "MOLECULAR PROFILES OF ENDOMETRIAL HYPERPLASIA AND ENDOMETRIAL INTRAEPITHELIAL NEOPLASIA". Tumors of female reproductive system 14, n.º 2 (15 de agosto de 2018): 76–81. http://dx.doi.org/10.17650/1994-4098-2018-14-2-76-81.

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Objective: to assess molecular profiles of endometrial hyperplasia and endometrial intraepithelial neoplasia.Materials and methods. We conducted a retrospective study that included 77 patients with a morphologically verified hyperplastic process in the endometrium. Of them, 34 patients had endometrial hyperplasia and 33 patients had endometrial intraepithelial neoplasia. The control group comprised 30 women with no endometrial disorders according to the results of histological examination.Results. Patients with endometrial intraepithelial neoplasia are at high risk of developing cancer. High angiogenic activity, pronounced vascularization, and endometrial hypoxia are believed to be important risk factors contributing to tumor proliferation and transformation. The expression of inhibitors of apoptosis, such as survivin and Bcl-2, is usually increased in atypical cells, which may indicate their involvement in malignant transformation of cells and tumor invasive growth.Conclusion. Our findings confirm the important role of survivin and Bcl-2 in hyperplastic processes in the endometrium.
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Kundu, Parveen R., Anjali Sindhu, Swaran Kaur, Amrita Kulhria e Ritika Hooda. "Expression of Cyclin D1 in normal and hyperplastic endometrium". International Journal of Reproduction, Contraception, Obstetrics and Gynecology 8, n.º 11 (23 de outubro de 2019): 4474. http://dx.doi.org/10.18203/2320-1770.ijrcog20194878.

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Background: Endometrial hyperplasia is characterised by increased gland to stroma ratio with varying degree of atypia. Cyclin D1 is a protein playing important role during the G1→S phase transition in the cell cycle. The present study evaluated the expression of Cyclin D1 in normal and hyperplastic endometrium.Methods: A cross sectional study was conducted over a period of 1 year. We evaluated and compared the expression of Cyclin D1 in 56 endometrial samples including 24 cases of simple hyperplasia, 12 cases of complex hyperplasia and 10 cases each of secretory and proliferative endometrium.Results: A substantial increase in expression of Cyclin D1 was seen in hyperplastic endometrium compared to normal endometrium. Moreover, complex hyperplasia showed the maximum positivity for Cyclin D1.Conclusions: Cyclin D1 may play a stimulatory role in the proliferation of endometrial glands and hence may be involved in endometrial tumorigenesis.
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Gad, Mohamed S., Bahgat A. Elfeky e Ahmed M. Nofal. "Stem cells and endometrial hyperplasia". Journal of Biochemistry, Microbiology and Biotechnology 2, n.º 2 (31 de dezembro de 2014): 70–75. http://dx.doi.org/10.54987/jobimb.v2i2.151.

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Endometrial hyperplasia is characterized by a proliferation of endometrial glands that may progress to or coexist with endometrial carcinoma. Previous studies proved the presence of endometrial stem cells that may have a role in endometrial regeneration and may be responsible for proliferative disorders as in endometrial hyperplasia and endometrial carcinoma. The presentstudy carried out to evaluate the potentiality of Umbilical cord blood Hematopoietic CD34+ and Mesenchymal stem cells to regress endometrial hyperplastic cells proliferation in vitro under certain condition. The Study carried out on 40 Umbilical cord blood samples collected from term delivering women, mononuclear cell were separated. Endometrial samples from the 40 cases (20normal and 20 cases with endometrial hyperplasia) were collected by dilatation and curettage or by curetting endometrium after hysterectomy operation. Then treatment of the hyperplastic cells with the Mesenchymal stem cells extract and CD34+ Hematopoietic stem cells extract was done. Results showed that both CD34+ Hematopoietic stem cells extract and mesenchymal stem cellsextract both had decreased the rate of growth of endometrial hyperplastic cell culture reaching near to the rate of growth of normal endometrial primary cell culture but the mesenchymal stem cells extract had a higher degree of control making endometrial growth rate nearer to the rate of growth of normal endometrial primary cell culture.
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Shapoval, Yu S. "Expression of Estrogen and Progesterone Receptors in the Endometrial Stroma of Women with Different Types of Endometrial Hyperplasia and Benign Breast Tumors". Ukraïnsʹkij žurnal medicini, bìologìï ta sportu 7, n.º 3 (2 de julho de 2022): 206–11. http://dx.doi.org/10.26693/jmbs07.03.206.

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The purpose of the study was to determine the level of expression of estrogen and progesterone receptors in endometrial tissue and to determine their relationship with the level of vitamin D in the blood of women of childbearing age with endometrial hyperplasia and benign breast tumors. Materials and methods. Examination of women of reproductive age who were diagnosed with hyperplastic processes in the endometrium was carried out. The examined patients were divided into two groups: Group I – the comparison group, which included patients with endometrial hyperplasia without concomitant pathology; Group II – women who, along with endometrial hyperplasia, were diagnosed with benign breast disease in the form of mastopathy. An immunohistochemical method using the En Vision imaging system with diaminobenzidine was used to detect estrogen and progesterone receptors in the endometrium. Determination of 25-hydroxyvitamin D (25OHD) content in blood plasma was performed by enzyme-linked immunosorbent assay using the 25-Hydroxy test kit. Results and discussion. It was found that the level of estrogen receptors expression increases in all types of endometrial hyperplasia and is exacerbated in concomitant mastopathy. Along with the increase in the level of estrogen receptors expression in endometrial cells, the expression of progesterone receptors decreases. It was found that the amount of progesterone receptors in the main group decreased by 38.3% – in endometrial hyperplasia without atypia, by 46.3% – in atypical endometrial hyperplasia and by 20.4% – in endometrial polyps compared with the control group of women. Analysis of the results shows an association between the form of hyperplasia and the receptor phenotype of the endometrium. It is established that the greatest changes are observed in atypical endometrial hyperplasia, which are exacerbated during the development of proliferative processes in the mammary glands. The relationship between vitamin D deficiency and estrogen and progesterone receptor expression in women with endometrial hyperplasia has been shown. Thus, when the level of estrogen and progesterone receptors changes in the endometrium of women with endometrial hyperplasia and mastopathy, the concentration of vitamin D in blood plasma decreases. Conclusion. The development of hyperplastic processes in the endometrium and mammary glands occurs against the background of increased expression of estrogen receptors and decreased expression of progesterone receptors. At the same time in the blood plasma of patients a decrease in vitamin D was revealed
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Buccoliero, A. M., F. Castiglione, C. F. Gheri, F. Garbini, M. Fambrini, G. Bargelli, S. Pappalardo, G. Scarselli, M. Marchionni e G. L. Taddei. "Liquid-based endometrial cytology: its possible value in postmenopausal asymptomatic women". International Journal of Gynecologic Cancer 17, n.º 1 (janeiro de 2007): 182–87. http://dx.doi.org/10.1111/j.1525-1438.2006.00757.x.

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The incidence of endometrial adenocarcinoma in asymptomatic women is low. Nevertheless, some of these women might require endometrial surveillance. In this study, we evaluated the accuracy of liquid-based endometrial cytology compared to biopsy in asymptomatic postmenopausal women. Three hundred twenty women scheduled for hysteroscopy were enrolled for this study. After hysteroscopy, patients were submitted to endometrial cytology and to biopsy. Two hundred ninety-three (92%) women had sonographically thickened endometrium (>5 mm), 53 (17%) were on tamoxifen, and 16 (5%) were on hormonal substitutive treatment. The evaluation of the biopsies determined that six (2%) women had adenocarcinoma, one (<1%) had adenomatous atypical hyperplasia, and eight (3%) had simple nonatypical hyperplasia. Endometrial cytology evidenced 5 (2%) neoplastic cases, 2 (<1%) hyperplastic with atypia cases, and 25 (8%) hyperplastic without atypia cases. Two hundred twenty-two biopsies (69%) and 17 (5%) cytologies were inadequate. One adenocarcinoma and one simple nonatypical hyperplasia were underrated by cytology resulting, respectively, as atypical hyperplasia and as negative. Four cases were false positive (simple nonatypical hyperplasias on cytology, negative on biopsy). The sensitivity and specificity were estimated, respectively, at 94% and 95%; the positive and negative predictive value were estimated, respectively, at 80% and 99%. Endometrial cytology provided sufficient material more often than biopsy (P < 0.01). We suggest to introduce liquid-based endometrial cytology in the management of some subpopulations of asymptomatic postmenopausal women. Particularly, the combination of liquid-based endometrial cytology and transvaginal sonography may improve their diagnostic accuracy and reduce unnecessary more invasive and expensive procedures.
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Mais fontes

Teses / dissertações sobre o assunto "Endometrial hyperplasia"

1

Gallos, Ioannis D. "Management & prognosis of endometrial hyperplasia". Thesis, University of Birmingham, 2014. http://etheses.bham.ac.uk//id/eprint/5157/.

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This thesis investigates the management and prognosis of endometrial hyperplasia. The literature on conservative therapies for endometrial hyperplasia is systematically reviewed and a meta-analysis is performed to identify the most effective treatment. Further meta-analysis is performed for young women with severe endometrial hyperplasia or cancer to explore the effectiveness of fertility-sparing treatment. A national survey of Gynaecologists is performed to evaluate current and the need for further research. A large cohort study is included that defines the regression and relapse of endometrial hyperplasia with two popular conservative therapies, the Levonorgestrel-releasing intrauterine system (LNG-IUS) and oral progestogens. The LNG-IUS is found to induce regression more often with fewer events of relapse than oral progestogens. A prediction model based on clinical characteristics and biomarkers finds that morbid obesity is an independent predictor for relapse. This research has major implications for clinical practice and a national guideline in process is based on its findings.
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2

Sivridis, E. "An immunohistochemical study of endometrial hyperplasia and neoplasia". Thesis, University of Manchester, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.374781.

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Vaskivuo, T. (Tommi). "Regulation of apoptosis in the female reproductive system". Doctoral thesis, University of Oulu, 2002. http://urn.fi/urn:isbn:9514266676.

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Abstract Apoptosis is a genetically programmed mechanism for a multicellular organism to remove cells that are unnecessary, or potentially harmful. The female reproductive system is characterised by a high rate of cellular proliferation. At the same time, apoptosis is also abundant during the normal physiological function of the ovary and endometrium. More than half of the 7 million oocytes that are produced during human ovarian development are deleted before birth and only about 400 oocytes reach the stage of ovulation during the female fertile lifespan. The fate of the non-ovulatory follicles is atresia, occurring through the mechanism of apoptosis. The endometrium goes through radical renewal processes during each menstrual cycle. Apoptosis has been suggested to participate in the regulation of endometrial cellular homeostasis. Errors in this mechanism can result in endometrial diseases such as hyperplasia and cancer. In this work, apoptosis and its regulation were studied in the human fetal and adult ovary, normal endometrium and endometrial pathologies. In fetal ovaries, apoptosis was already abundantly present in oocytes at 13 weeks of gestation. The maximum rate of apoptosis was seen between the 14th and 20th weeks, after which apoptosis decreased towards term. Ovarian Bcl-2 expression was detected in early fetal life during weeks 13 and 14. Bax expression was observed throughout the studied period, from week 13 to 40. The expression of transcription factor GATA-4, which is linked to follicular survival, was localised to the granulosa cells and was high in early fetal life and decreased somewhat towards term. In adult life apoptosis was located in the granulosa cells of the growing follicles. In ovarian biopsies from women homozygous for the inactivating C566T mutation of the FSH receptor, apoptosis or GATA-4 expression was not detected. During corpus luteum regression a peak in apoptosis was detected 10 - 12 days after the LH surge, and was preceded by an increase in 17HSD type 1 and TNF-α expression. During normal menstrual cycles, the highest rate of apoptosis was observed in the menstrual endometrium. This increase in apoptosis was preceded by a decreased Bcl-2/Bax ratio. In endometrial hyperplasia, the rate of apoptosis was similar to that seen during normal proliferation of the endometrium, but an apparent increase was observed in grade II endometrial carcinoma. In grade III carcinoma, the rate of apoptosis was lower than in grade II carcinoma but higher than in hyperplasia. These results indicate that apoptosis is the mechanism behind the substantial oocyte demise during ovarian development. During adult life, apoptosis was mainly localised to the granulosa cells of the growing follicles which do not reach the stage of a dominant follicle. In ovaries where FSH action is abolished, folliculogenesis was impaired and ovarian apoptosis was negligible. Apoptosis is also the underlying mechanism of corpus luteum regression. In the endometrium, apoptosis has a role in rejuvenating the endometrium for growth during the next endometrial cycle and in regulating cellular homeostasis.
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HAMID, ATIA AKHTAR. "Cyclical Change of hMSH2 Protein Expression in Normal endometrium during the Menstrual Cycle and its Overexpression in Endometrial Hyperplasia and Sporadic Endometrial Carcinoma". Kyoto University, 2002. http://hdl.handle.net/2433/149704.

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Kyoto University (京都大学)
0048
新制・課程博士
博士(医学)
甲第9461号
医博第2474号
新制||医||795(附属図書館)
UT51-2002-G219
京都大学大学院医学研究科外科系器官外科学(婦人科学産科学)専攻
(主査)教授 中畑 龍俊, 教授 丹羽 太貫, 教授 藤井 信吾
学位規則第4条第1項該当
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Сміян, Світлана Анатоліївна, Светлана Анатольевна Смиян, Svitlana Anatoliivna Smiian, Анатолій Борисович Сухарєв, Анатолий Борисович Сухарев, Anatolii Borysovych Sukhariev, Ірина Миколаївна Нікітіна et al. "Рецептори стероїдних гормонів і морфофункціональний стан ендометрія у хворих з гіперпластичними процесами ендометрія". Thesis, Сумський державний університет, 2014. http://essuir.sumdu.edu.ua/handle/123456789/36521.

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Гіперплазія ендометрія – найбільш часта патологія слизової матки у жінок різних вікових груп. Інтерес до проблеми гіперпластичних процесів ендометрія зумовлена досить високим ризиком малігнізації, а також постійним зростанням частоти гіперплазії і рака ендометрія за останні десятиріччя у багатьох країнах світу, зокрема в Україні. Рак ендометрія серед злоякісних пухлин у жінок займає третє місце. За даними F.Degeinhard (1995), 50 % випадків рака ендометрія в пери- і пост менопаузі є результатом малігнізації гіперпластичних процесів. Бохман Я.В. і співавтори (1983) обгрунтували концепцію 2-х основних патогенетичних варіантів рака ендометрія: І – гормонозалежний, який зустрічається у 60–70 % випадках рака і характеризуется хронічною гіперестрогенією, ІІ – автономний, який визначається у 30–40 %, і не асоційований з естрогенним впливом. При цитуванні документа, використовуйте посилання http://essuir.sumdu.edu.ua/handle/123456789/36521
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Martins, Danilo Gama [UNESP]. "Complexo hiperplasia endometrial cística/piometra em cadelas: fisiopatogenia, características clínicas e laboratoriais e abordagem terapêutica". Universidade Estadual Paulista (UNESP), 2007. http://hdl.handle.net/11449/89044.

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Made available in DSpace on 2014-06-11T19:23:42Z (GMT). No. of bitstreams: 0 Previous issue date: 2007-07-06Bitstream added on 2014-06-13T19:48:09Z : No. of bitstreams: 1 martins_dg_me_jabo.pdf: 254602 bytes, checksum: f45d1583561d8115c503fe5bb6c3321d (MD5)
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Na prática da clínica de pequenos animais, os médicos veterinários são freqüentemente confrontados com o quadro ou com o diagnóstico diferencial da piometra em cadelas, e devem decidir rapidamente sobre a melhor forma de tratamento, pois se trata de uma situação de risco para a vida da paciente. Esta revisão tem como objetivo a descrição dos conceitos práticos e atuais da etiopatogenia da hiperplasia endometrial cística em cadelas, a qual precede o desenvolvimento do quadro de piometra. Abordam-se tanto a classificação quanto os sintomas, os achados clínicos, bem como aspectos diagnósticos dessa síndrome. Com relação ao tratamento, são consideradas tanto a abordagem cirúrgica quanto a medicamentosa, bem como as vantagens e desvantagens de cada opção.
In clinics, pyometra in bitches is one of the most common disorders. Veterinarians must decide about the therapeutical approach and the best way to conduct treatment. So, it is important to know the complex physiopathogeny of this disorder, as well as the concomitant diseases, laboratorial alterations and types of treatment. The aim of this dissertation is to describe the new concepts of the etiopathogenia of the cystic endometrial hyperplasia-pyometra complex in bitches, evaluate the clinical and laboratorial alterations and also the options of the treatments found in animals consulted at the obstetrics department at the Veterinary Hospital Governador Laudo Natel, FCAV-UNESP-Jaboticabal and confront them with the results published in literature and also with cases from other national and international universities. Thus, it was described the classification of clinical signs, diagnosis and therapeutics of this syndrome.
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Martins, Danilo Gama. "Complexo hiperplasia endometrial cística/piometra em cadelas : fisiopatogenia, características clínicas e laboratoriais e abordagem terapêutica /". Jaboticabal : [s.n.], 2007. http://hdl.handle.net/11449/89044.

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Orientador: Wilter Ricardo Russiano Vicente
Banca: Elzylene Léga
Banca: Marcos Lania de Araujo
Resumo: Na prática da clínica de pequenos animais, os médicos veterinários são freqüentemente confrontados com o quadro ou com o diagnóstico diferencial da piometra em cadelas, e devem decidir rapidamente sobre a melhor forma de tratamento, pois se trata de uma situação de risco para a vida da paciente. Esta revisão tem como objetivo a descrição dos conceitos práticos e atuais da etiopatogenia da hiperplasia endometrial cística em cadelas, a qual precede o desenvolvimento do quadro de piometra. Abordam-se tanto a classificação quanto os sintomas, os achados clínicos, bem como aspectos diagnósticos dessa síndrome. Com relação ao tratamento, são consideradas tanto a abordagem cirúrgica quanto a medicamentosa, bem como as vantagens e desvantagens de cada opção.
Abstract: In clinics, pyometra in bitches is one of the most common disorders. Veterinarians must decide about the therapeutical approach and the best way to conduct treatment. So, it is important to know the complex physiopathogeny of this disorder, as well as the concomitant diseases, laboratorial alterations and types of treatment. The aim of this dissertation is to describe the new concepts of the etiopathogenia of the cystic endometrial hyperplasia-pyometra complex in bitches, evaluate the clinical and laboratorial alterations and also the options of the treatments found in animals consulted at the obstetrics department at the Veterinary Hospital "Governador Laudo Natel", FCAV-UNESP-Jaboticabal and confront them with the results published in literature and also with cases from other national and international universities. Thus, it was described the classification of clinical signs, diagnosis and therapeutics of this syndrome.
Mestre
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Замкова, В. О., e К. О. Швидун. "Морфологічні особливості атипової гіперплазії ендометрія". Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/26861.

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Dhaliwal, Gurmeet Kaur. "The pathogenesis of cystic endometrial hyperplasia (pyometra) in the bitch; with particular reference to the role of steroid receptors". Thesis, Royal Veterinary College (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.522193.

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Bentham, Zoe. "A novel progesterone oral formulation for the treatment of endometrial hyperplasia with reduced adverse signalling compared to synthetic progestins". Thesis, Swansea University, 2015. https://cronfa.swan.ac.uk/Record/cronfa42514.

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Background: Natural progesterone is currently unavailable for the treatment of endometrial hyperplasia. The first line of treatment for this condition is with the synthetic progestins Medroxyprogesterone Acetate (MPA) and Levonorgestrel (LNG). However, these hormones frequently trigger side effects which occur as a result of adverse gene and protein regulation. The current unavailability of natural progesterone is down to its poor bioavailability which is due to the low aqueous solubility and extensive first-pass metabolism of this compound. Aims: The first aim of this thesis is to compare natural progesterone with MPA and LNG in their regulation of a subset of genes and proteins in the endometrium whilst also assessing the selected proteins in biopsies from patients with endometrial hyperplasia before and after progestin therapy. The second aim is to study progesterone metabolism within the gastrointestinal tract and liver in order to direct the production of progesterone amorphous solid dispersions. Methodology: During this thesis both microbiological and pharmacological laboratory techniques were used. For microbiology investigations immunohistochemistry, polymerase chain reaction, InCell Analysis and enzyme-linked immunosorbent assay were the key research methods. For pharmacological research the main methods used were stability assays analysed by tandem mass spectrometry and high performance liquid chromatography, solvent emulsion evaporation, powder x-ray diffraction and scanning electron microscopy. Conclusions: Amorphous solid dispersions are an attractive option for the future production of a natural progesterone oral formulation with enhanced solubility. Such a formulation can be targeted for delivery to the distal small intestine where progesterone metabolism is reduced compared to other intestinal regions. This formulation would be of particular benefit to patients with endometrial hyperplasia as the most commonly used progestins to treat this disease were shown to differentially regulate genes and proteins in the human endometrium compared to the natural product.
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Livros sobre o assunto "Endometrial hyperplasia"

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E, Poulsen H., ed. Atlas of endometrial histopathology. 2a ed. Berlin: Springer, 1996.

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Malpica, Anais. Biopsy interpretation of the uterine cervix and corpus. Philadelphia: Wolters Kluwer/Lippincott Williams & Wilkins, 2010.

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Saffry, Olga Barbara. Endometrial Hyperplasia as Observed in Experimental Guinea Pigs. Hassell Street Press, 2021.

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Publications, ICON Health. Endometrial Hyperplasia - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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BIOPSY INTERP UTERINE CERVIX CORP 2E CB. WOLTERS KLUWER, 2015.

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Capítulos de livros sobre o assunto "Endometrial hyperplasia"

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Williams, Kristina, e Emily Ko. "Endometrial Hyperplasia". In Handbook of Gynecology, 1–15. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-17002-2_3-1.

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Dotto, Jorge Enrique, e Miguel A. Bigozzi. "Endometrial Hyperplasia". In Atlas of Hysteroscopy, 117–23. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-29466-3_14.

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Mencaglia, Luca, Francesca Ciociola e Stefania Magnolfi. "Endometrial Hyperplasia". In Hysteroscopy, 261–67. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57559-9_27.

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Williams, Kristina, e Emily Ko. "Endometrial Hyperplasia". In Handbook of Gynecology, 877–91. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-17798-4_3.

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Hermann, Catherine, Kristina Williams e Emily M. Ko. "Endometrial Hyperplasia". In Handbook of Gynecology, 1–19. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-319-17002-2_3-2.

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Deligdisch, Liane. "Endometrial Hyperplasia and Endometrial Adenocarcinoma". In Clinical Perspectives in Obstetrics and Gynecology, 84–101. New York, NY: Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4613-9086-2_7.

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Goeckenjan, Maren, Michael von Wolff e Pauline Wimberger. "Endometrial Hyperplasia and Endometrial Carcinoma". In Fertility Preservation in Oncological and Non-Oncological Diseases, 85–91. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-47568-0_11.

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Hasson, Brian F., Charlie Ma, Lu Wang, David E. Wazer, Jay E. Reiff, Jay E. Reiff, Brandon J. Fisher et al. "Atypical Endometrial Hyperplasia". In Encyclopedia of Radiation Oncology, 23. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-540-85516-3_482.

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Gupta, Krishnendu, e Bhaskar Pal. "Hysteroscopy: Endometrial Hyperplasia and Endometrial Malignancies". In Hysteroscopy Simplified by Masters, 177–83. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-2505-6_22.

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Dragun, Anthony E., Paul J. Schilling, Tod W. Speer, Feng-Ming Kong, Jingbo Wang, Hedvig Hricak, Oguz Akin et al. "Endometrial Hyperplasia with Atypia". In Encyclopedia of Radiation Oncology, 214. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-540-85516-3_1099.

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Trabalhos de conferências sobre o assunto "Endometrial hyperplasia"

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Qureshi, Sabuhi. "Study of PTEN immunohistochemical expression in endometrial hyperplasia". In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685337.

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Objective: The incidence of endometrial hyperplasia & carcinoma is increasing in developing nations. Newer techniques are being tried to recognise endometrial hyperplasia. One of these is tumor suppressor gene phosphatase & tensin homologue (PTEN). It is frequently inactivated i.e turned off in endometrial hyperplasia lesions. This is an early event in endometrial tumorigenesis that may occur in response to known endocrine risk factors & offers an informative immunohistochemical marker for premalignant disease. The present study was planned to study PTEN immunohistochemical expression in endometrial hyperplasia. Methods: Women of >40 years of age presenting with abnormal uterine bleeding in the OPD of OBGYN Department of KG Medical University underwent endometrial biopsy. The histopathology of the biopsy tissue was done in department of Pathology of KG Medical University. The cases of endometrial hyperplasia were studied for PTEN immunohistochemical expression. Results: 168 women of >40 years of age with abnormal uterine bleeding underwent endometrial biopsy. 50 women were diagnosed as endometrial hyperplasia. Of these, PTEN evaluation was done in 27 cases. Loss of PTEN expression was found in 11 cases (40.74%) of endometrial hyperplasia. Loss of PTEN expression was more in complex hyperplasia with atypia (66.66%) as compared to simple hyperplasia without atypia (29.4%). Conclusion: There is positive correlation between loss of PTEN expression and grade of morphological differentiation of hyperplasia.
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Lisova, E. S., L. A. Shpagina e L. A. Panacheva. "IMMUNOHISTOCHEMICAL FEATURES OF ENDOMETRIAL HYPERPLASIA IN PRE – AND POSTMENOPAUSAL CONDITIONS UNDER THE INFLUENCE OF CHEMICAL FACTORS". In The 16th «OCCUPATION and HEALTH» Russian National Congress with International Participation (OHRNC-2021). FSBSI “IRIOH”, 2021. http://dx.doi.org/10.31089/978-5-6042929-2-1-2021-1-317-320.

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Abstract. Objective: To study the frequency of endometrial hyperplasia in women with I-II degree obesity exposed to toxic-chemical factors, and to identify the most important immunohistochemical markers of the proliferative activity of endometrial cells. 60 women aged 45-65 years were examined, 30 of them worked in contact with a complex of toxic factors (the main group) and 30 – without them (the comparison group). The maximum single concentrations of toxicants exceeded the MPC by 2.1-5.4 times. The criterion of obesity was a BMI > 30.0. All underwent hysteroscopy and histological examination of scrapings. Immunohistochemical characteristics of the endometrial prescription apparatus were carried out by evaluating the proliferation marker Ki 67, estrogen (Eg) and progesterone receptors (Pr). Results. In all women, simple endometrial hyperplasia was most common, more often diagnosed in the main group in the presence of obesity and in the comparison group. The highest indicators of Ki 67 were found in workers of harmful production with complex and simple atypical endometrial hyperplasia in combination with obesity of I-II degree. Eg parameters were high in women of the main group with obesity, having complex atypical (71/48) and complex endometrial hyperplasia (66/30). Among the obese workers of the comparison group, high Eg values were found in simple and complex atypical hyperplasia (64/32 and 67/40, respectively). High values of Rg indicators in obese women who come into contact with chemical agents, who have complex (21/136) and simple atypical hyperplasia (26/124), as well as in obese workers of the comparison group and the presence of complex atypical hyperplasia (29/135). Conclusion. In women with endometrial hyperplasia, working
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Oliver, MDLR, A. Olloqui-Escalona, C. González-Macho, C. Pérez-Sagaseta, C. Guillen-Gamez, M. Martínez- Lopez e A. Tejerizo-García. "497 Endometrial Hyperplasia: Risk of coexistence and progression to endometrial Carcinoma. Retrospective cohort study". In ESGO 2021 Congress. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/ijgc-2021-esgo.158.

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Sharif, B., A. Ahmadian, M. A. Oghabian e N. Izadi. "Texture Segmentation of Endometrial Images for Aiding Diagnosis of Hyperplasia". In EUROCON 2005 - The International Conference on "Computer as a Tool". IEEE, 2005. http://dx.doi.org/10.1109/eurcon.2005.1630112.

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D’Angelo, E., I. Espinosa, V. Cipriani, M. Barbareschi e J. Prat. "257 Atypical endometrial hyperplasia, low-grade: ‘much ADO About nothing’". In IGCS Annual 2019 Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-igcs.257.

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Sideris, M., A. Darwish, K. Rallis, EI Emin e T. Mould. "548 Prognostic biomarkers for atypical endometrial hyperplasia: a mini review". In ESGO 2021 Congress. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/ijgc-2021-esgo.596.

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Atalay, Cemal Resat, e Funda Atalay. "2022-RA-1017-ESGO Concurrent endometrial carcinoma in hysterectomy specimens in patients with atypical endometrial hyperplasia". In ESGO 2022 Congress. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/ijgc-2022-esgo.280.

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Akkour, K., N. Alali, H. Alhalal, A. Bogis, H. Alanazi, M. Alhulwah e M. Arafah. "238 When endometrial sampling is not an option, the value of endometrial thickness in predicting endometrial hyperplasia in patients with postmenopausal bleeding". In IGCS Annual 2019 Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-igcs.238.

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Singhal, Seema, Sunesh Kumar Jain, D. N. Sharma, Sandeep Mathur, Juhi Bharti, Anshu Yadav, K. K. Roy, Neeta Singh e Jyoti Meena. "Clinicopathological analysis of early endometrial cancers". In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685344.

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Aim: The study objectives were evaluation of clinicopathological characteristics, correlations between the preoperative and postoperative tumor assessment in early stage endometrial cancer. Materials and Methods: We conducted a prospective descriptive study of 30 cases of endometrial cancer stage 1 examined and treated at a tertiary care teaching institute between the years 2014-15. Results: The patients’ mean age at the time of diagnosis was 56.4 years. The mean parity was two. Postmenopausal bleeding with or without abnormal vaginal discharge was the most frequent symptom; it was present in 84.7% of patients. Co morbidities like hypertension and diabetes were seen in 65% of women. 6/30 patients had family history of some malignancy. All the patients underwent Type I extrafascial hysterectomy with bilateral salpingo oophorectomy, one case had Type I extrafascial hysterectomy with infracolic omentectomy. A total of 10.6% cases had lymph nodes metastasis and none of these patients had ovarian metastasis or positive peritoneal cytology. None of the patients with superficial myometrial invasion (MI) had lymph node metastasis. None of the cases showed positive peritoneal cytology. Staging upgraded fom 1a to 1b in 50% of subjects after final histopathological analysis. One patient who was operated as endometrial hyperplasia with atypia actually had endometrial adenocarcinoma in the postoperative specimen. Conclusions: There is a poor correlation between the preoperative and the postoperative tumor assessment.
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Singhal, Seema, Sunesh Kumar Jain, D. N. Sharma, Sandeep Mathur, Juhi Bharti, Anshu Yadav, K. K. Roy, Neeta Singh e Jyoti Meena. "Clinicopathological analysis of early endometrial cancers". In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685338.

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Aim: The study objectives were evaluation of clinicopathological characteristics, correlations between the preoperative and postoperative tumor assessment in early stage endometrial cancer. Materials and Methods: We conducted a prospective descriptive study of 30 cases of endometrial cancer stage 1 examined and treated at a tertiary care teaching institute between the years 2014-15. Results: The patients’ mean age at the time of diagnosis was 56.4 years. The mean parity was two. Postmenopausal bleeding with or without abnormal vaginal discharge was the most frequent symptom; it was present in 84.7% of patients. Co morbidities like hypertension and diabetes were seen in 65% of women. 6/30 patients had family history of some malignancy. All the patients underwent Type I extrafascial hysterectomy with bilateral salpingo oophorectomy, one case had Type I extrafascial hysterectomy with infracolic omentectomy. A total of 10.6% cases had lymph nodes metastasis and none of these patients had ovarian metastasis or positive peritoneal cytology. None of the patients with superficial myometrial invasion (MI) had lymph node metastasis. None of the cases showed positive peritoneal cytology. Staging upgraded from 1a to 1b in 50% of subjects after final histopathological analysis. One patient who was operated as endometrial hyperplasia with atypia actually had endometrial adenocarcinoma in the postoperative specimen. Conclusions: There is a poor correlation between the preoperative and the postoperative tumor assessment.
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Relatórios de organizações sobre o assunto "Endometrial hyperplasia"

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Franchi, Dorella. Role of transvaginal ultrasonography in the management of young patients affected by atypical endometrial hyperplasia and welldifferentiated endometrial cancer undergoing hormonal fertility-sparing treatment. Science Repository OÜ, março de 2019. http://dx.doi.org/10.31487/j.jso.2019.01.005.

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