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Bibliografias temáticas / H. pylori

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Literatura científica selecionada sobre o tema "H. pylori"

Autor: Grafiati

Publicado: 4 de junho de 2021

Última modificação: 25 de julho de 2025

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Artigos de revistas sobre o assunto "H. pylori"

1

. "NSAID, gebruikNSAID-gebruik en H. pylori, tetracycline H. pylori, naproxen H. pylori, metronidazol H. pylori, bismutoxideH. Pylori-eradicatie." Medisch-Farmaceutische Mededelingen 38, no. 4 (2000): 72. http://dx.doi.org/10.1007/bf03057514.

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2

Assumpção, Paulo Pimentel de, Robert M. Genta, Maria Constanza Camargo, Jessica Manoelli Costa da Silva, Manuel Ricardo Amieva, and Massimo Rugge. "The Landscape of Helicobacter pylori-related Gastric Carcinogenesis." Journal of Gastrointestinal and Liver Diseases 33, no. 4 (2024): 524–34. https://doi.org/10.15403/jgld-5959.

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The relationship between Helicobacter pylori (H. pylori) and humans remains a complex enigma. While other factors contribute to gastric cancer (GC), their impact pales in comparison to the central role of H. pylori. Various cofactors, such as dietary carcinogens and Epstein-Barr virus infection, can lead to GC independently of H. pylori. However, it is likely the combination of mechanisms, especially those driven by H. pylori, that represents the primary force behind GC development. Identifying individuals at high risk of developing H. pylori-related GC or detecting the disease in its earliest

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3

Sakaguchi, Takuki, Takaaki Sugihara, Ken Ohnita, et al. "Pyloric Incompetence Associated with Helicobactor pylori Infection and Correlated to the Severity of Atrophic Gastritis." Diagnostics 12, no. 3 (2022): 572. http://dx.doi.org/10.3390/diagnostics12030572.

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Duodenogastric reflux (DGR) causes bile reflux gastritis (BRG) and may develop into gastric cancer. DGR is classified as primary in non-operated stomachs or secondary to surgical intervention. Primary DGR and Helicobacter pylori (H. pylori) infection are reportedly related. However, the mechanism is not fully understood. This study aimed to elucidate the relationship between H. pylori infection and pyloric incompetence in a non-operated stomach. A total of 502 non-operated participants who underwent an upper intestinal endoscopy were prospectively enrolled. Endoscopic findings (EAC, endoscopic

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Garcés-Duran, R., S. Kindt, K. Kotilea, et al. "Belgian consensus for Helicobacter pylori management 2023." Acta Gastro Enterologica Belgica 86, no. 1 (2023): 74–91. http://dx.doi.org/10.51821/86.1.11327.

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Helicobacter pylori (H. pylori) infection causes chronic gastritis, peptic ulcers and gastric cancer. Although H. pylori prevalence is decreasing worldwide, regional variations exist in Europe, with the lowest infection prevalence in Northern Europe, and the highest in Eastern and Southern Europe (1). Changes in the treatment recommendations and the increasing available evidence have justified the implementation of new recommendations since last Belgian consensus in 1998 (2). Several non-H. pylori Helicobacter species (NH.PYLORI-H), colonizing the stomach of domestic animals, also have the abi

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5

Öztekin, Merve, Birsen Yılmaz, Duygu Ağagündüz, and Raffaele Capasso. "Overview of Helicobacter pylori Infection: Clinical Features, Treatment, and Nutritional Aspects." Diseases 9, no. 4 (2021): 66. http://dx.doi.org/10.3390/diseases9040066.

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Helicobacter pylori (H. pylori) is a 0.5–1 µm wide, 2–4 µm long, short helical, S-shaped Gram-negative microorganism. It is mostly found in the pyloric region of the stomach and causes chronic gastric infection. It is estimated that these bacteria infect more than half of the world’s population. The mode of transmission and infection of H. pylori is still not known exactly, but the faecal–oral and oral–oral routes via water or food consumption are thought to be a very common cause. In the last three decades, research interest has increased regarding the pathogenicity, microbial activity, genet

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Blasiak, Janusz, Jan Chojnacki, and Cezary Chojnacki. "Inflammation, oxidative stress, DNA damage response and epigenetic modifications interact behind the beneficial actions of melatonin on H. pylori-mediated gastric disorders." Melatonin Research 6, no. 2 (2023): 135–47. http://dx.doi.org/10.32794/mr112500145.

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Helicobacter pylori (H. pylori) infection is associated with several disorders of the gastrointestinal tract, including gastric cancer. Studies of ours and others suggest that H. pylori infection may affect melatonin synthesis in the gastric epithelial cells. On the other hand, melatonin ameliorates gastric disorders as shown in clinical trials and experimental studies. Moreover, melatonin not only suppresses the DNA-damaging reaction of diet-related mutagens that can initiate carcinogenesis in gastric mucosa, but also the oxidative DNA damage evoked by reactive oxygen and nitrogen species pro

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Asonuma, Sho, Akira Imatani, Naoki Asano, et al. "Helicobacter pylori induces gastric mucosal intestinal metaplasia through the inhibition of interleukin-4-mediated HMG box protein Sox2 expression." American Journal of Physiology-Gastrointestinal and Liver Physiology 297, no. 2 (2009): G312—G322. http://dx.doi.org/10.1152/ajpgi.00518.2007.

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Helicobacter pylori is a major cause of the transdifferentiation into intestinal metaplasia that may develop gastric cancer. However, the molecular pathogenesis of this transdifferentiation is poorly understood. A SRY-related HMG box protein Sox2 is an essential transcription factor of organ development in brain, lung, and stomach. Our aim of this study was to investigate the mechanism responsible for regulation of Sox2 in host Th1-dominant response to H. pylori. Sox2 protein was immunohistochemically expressed in both human oxyntic and pyloric glands with H. pylori infection, but not in intes

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Dr., Tarak Banik, Prasenjit Kumar Bar Dr., and Saikat Mandal Dr. "Helicobacter pylori chronic gastritis: correlation between endoscopic findings and histopathology with special reference to updated sydney system." International Journal of Medical Research and Review 8, no. 1 (2020): 7–13. https://doi.org/10.17511/ijmrr.2020.i01.02.

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Aims of the study: Helicobacter pylori (H. pylori) induces inflammatory changes in the gastric mucosa variably correlated to different endoscopic and histologic features. The present prospective study aimed to correlate different endoscopic findings with histomorphological changes and the presence of H. pylori in the gastro-duodenal mucosa, in samples of dyspeptic patients. Methods: 60 dyspeptic patients of 18 years to 60 years of age were selected from outpatient department and screened with gastro-duodenoscopy and biopsy. The presence of H. pylori was determined by urease test on fresh biops

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Imam, Alam Imam Bakhsh Irshad Ahmad Zakir Hussain Sanjay Kumar. "Association Of Helicobacter Pylori With Perforated Peptic Ulcer In Pakistani Population An Institution Based Study." Multicultural Education 8, no. 3 (2022): 130. https://doi.org/10.5281/zenodo.6344724.

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<strong><em>Background: </em></strong><em>Most patients with chronic peptic ulcer disease have Helicobacter pylori (H. pylori) infection. In the past, immediate acid-reduction surgery has been strongly advocated for perforated peptic ulcers because of the high incidence of ulcer relapse after simple closure. Simple over sewing procedures either by an open or laparoscopic approach together with H. pylori eradication appears to supersede definitive ulcer surgery.</em> <strong><em>Objective:</em></strong><em> To determine the frequency of H pylori in patients with perforated duodenal ulcer.</em>

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Takatsuna, Masafumi, Rie Azumi, Takeshi Mizusawa, et al. "A case of Helicobacter pylori-negative early gastric adenocarcinoma with gastrointestinal phenotype." Endoscopy International Open 09, no. 06 (2021): E863—E866. http://dx.doi.org/10.1055/a-1396-3854.

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AbstractA 40-year-old man with slightly depressed (0-IIc) type gastric cancer of the pyloric anterior gastric area underwent pre-operative screening for tetralogy of Fallot and endoscopic submucosal dissection (ESD) and was tested for Helicobacter pylori antigens and antibodies. Both tests were negative. He did not have a history of eradication. Pathological diagnosis of ESD showed a well-differentiated adenocarcinoma. The tumor was CD10-positive, MUC5AC-negative, and MUC6-confocal positive; it showed differentiation with gastrointestinal phenotype. Moreover, the tumor cells were lysozyme-posi

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Teses / dissertações sobre o assunto "H. pylori"

1

Warren, John Robin. "The discovery and pathology of H pylori /." Title page and contents only, 1999. http://web4.library.adelaide.edu.au/theses/09MD/09mdw289.pdf.

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Сорокман, Т. В. "Кіотський глобальний консенсус щодо H. pylori-асоційованого гастриту". Thesis, БДМУ, 2017. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/17155.

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Leslie, Kevin Alexander. "Investigation of Prophage in Clinical Isolates of H pylori." W&M ScholarWorks, 2013. https://scholarworks.wm.edu/etd/1539626941.

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Arebi, Naila. "Identification of novel factors regulating mucosal somatostatin in H. pylori gastritis." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289867.

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Zhou, Yong. "An antimicrobial agent from celery seed active against H. pylori." Thesis, Sheffield Hallam University, 2008. http://shura.shu.ac.uk/20709/.

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As well as peptic ulcers, Helicobacter pylori is associated with the development of gastritis, gastric adenocarcinoma and lymphoma, and has been classified as a class I carcinogen in humans (International Agency for Research on Cancer Working Group, 1994). Although the bacteria can be eradicated in up to 90% of patients, side effects, poor compliance and the resistance of the bacteria to antibiotics are common causes of frequent treatment failure. Celery seed extracts (CSE) from a unique source in India has been used as herbal medicine since antiquity and found to have anti-inflammatory and ga

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Iwano, Masahiro. "Characteristics of gastric MALT lymphoma resistant to eradication of H. pylori." Kyoto University, 2008. http://hdl.handle.net/2433/124330.

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Jimenez-Guerra, Francisco. "Prevalence of, and risk factors for, Helicobacter pylori infection and its effect on growth of children in Mexico." Thesis, London School of Hygiene and Tropical Medicine (University of London), 1999. http://researchonline.lshtm.ac.uk/4649359/.

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Helicobacter pylori (H. pylori) infection causes achlorydria, depressed gastric acid barrier, impaired immune response and is suspected in bacterial overgrowth and diarrhoea. These features of the infection are known to cause significant malabsorption of nutrients and impairment of linear growth in children. The prevalence of H. pylori infection in children is known to be much higher in developing countries, especially among the lower socio-economic groups. The true prevalence of infection in urban children in Mexico and its impact on their growth are largely unknown. This study examined the p

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Vallström, Anna. "Helicobacter pylori : molecular mechanisms for variable adherence properties." Doctoral thesis, Umeå universitet, Institutionen för medicinsk kemi och biofysik, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-25931.

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More than half of all people worldwide are infected with H. pylori. The infection always cause a gastric inflammation that may develop into peptic ulcer disease or gastric cancer. Attachment proteins, adhesins, mediate specific adherence of H. pylori to receptor structures on the human gastric mucosa. The best-characterized H. pylori adhesin-receptor interactions are the BabA adhesin and the binding to the fucosylated blood group antigens ABO/Lewis b (Leb) and the SabA adhesin and its binding to the inflammation associated sialyl-Lewis x antigen. During H. pylori infection the availability of

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de, Albuquerque Mello Luciano. "Análise histomorfométrica de lesões gástricas relacionadas à infecção pelo Hlicobacter pylori." Universidade Federal de Pernambuco, 2007. https://repositorio.ufpe.br/handle/123456789/9072.

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Made available in DSpace on 2014-06-12T23:04:25Z (GMT). No. of bitstreams: 2 arquivo8874_1.pdf: 1552701 bytes, checksum: ca1f64cd2a9ab7f1dd8b3732c2cc688d (MD5) license.txt: 1748 bytes, checksum: 8a4605be74aa9ea9d79846c1fba20a33 (MD5) Previous issue date: 2007<br>O bacilo Helicobacter pylori é atualmente considerado como o agente etiológico mais importante das gastrites em seres humanos e como um fator de risco para o surgimento das doenças ulcerativas do carcinoma gástrico. Estima-se que 50% da população mundial está infectada por esta bactéria. Atualmente, apenas estudos subjetivos e qualit

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Benigno, Tiago Gomes da Silva. "Análise da resposta humoral contra H. pylori em pacientes portadores e não portadores de câncer gástrico." reponame:Repositório Institucional da UFC, 2013. http://www.repositorio.ufc.br/handle/riufc/10642.

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BENIGNO, Tiago Gomes da Silva. Análise da resposta humoral contra H. pylori em pacientes portadores e não portadores de câncer gástrico. 2013. 71 f. Dissertação (Mestrado em Cirurgia) - Universidade Federal do Ceará. Faculdade de Medicina, Fortaleza, 2013.<br>Submitted by denise santos (denise.santos@ufc.br) on 2015-02-19T11:35:41Z No. of bitstreams: 1 2013_dis_tgsbenigno.pdf: 896697 bytes, checksum: daa07ef38bffc56879c4e5d12ca866fd (MD5)<br>Approved for entry into archive by denise santos(denise.santos@ufc.br) on 2015-02-19T11:36:17Z (GMT) No. of bitstreams: 1 2013_dis_tgsbenigno.pdf: 896697

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Mais fontes

Livros sobre o assunto "H. pylori"

1

B, Ernst Peter, Michetti Pierre, and Smith, Phillip D., M.D., eds. The immunobiology of H. pylori: From pathogenesis to prevention. Lippincott-Raven, 1997.

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2

Satō, Eisuke. H10-tokubetsu-023, H. pylori no kassei sanso taisha tokusei to idenshi ihen kikō no bunshironteki kaiseki: Heisei 10-nendo kenkyū hōkokusho : Kōseishō kōsei kagaku kenkyūhi hojokin kōsei kagaku tokubetsu kenkyū jigyō. s.n.], 1999.

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3

H. Pylori Rescue Guide. Independently Published, 2020.

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4

Benjamin, James. H. Pylori: The Pro Guide on How to Treat and Prevent H. Pylori. Independently Published, 2022.

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5

LOUIS, Gerald. Helicobacter Pylori : Essential Manual about H. Pylori: All Information Included. Independently Published, 2022.

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6

H. Pylori: Learn and Master the Quick Guide on How to Heal H. Pylori. Independently Published, 2021.

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7

Mandela, Gregory. Helicobacter Pylori: The Complete Guide on How to Treat and Prevent H. Pylori. Independently Published, 2021.

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8

H. Pylori Cure Manual: The Ultimate Guide on Complete Cure from Helicobacter Pylori. Independently Published, 2021.

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9

Helicobacter Pylori: The Effective Cure Guide on the Effective Treatment of H. Pylori. Independently Published, 2021.

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10

ALDO, Adler. H. Pylori Cure Guide: Understanding the Disease, Everything on How to Strive, Prevent and Reverse H. Pylori Completely. Independently Published, 2022.

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Capítulos de livros sobre o assunto "H. pylori"

1

Mobley, H. L. T., and P. A. Foxall. "H. pylori urease." In Helicobacter pylori. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1418-9_3.

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2

Tytgat, G. N. J., and L. A. Noach. "H. pylori eradication." In Helicobacter pylori. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1418-9_53.

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3

Takafuta, Toshiro, and Kingo Fujimura. "Helicobacter pylori (H. pylori) Eradication." In Autoimmune Thrombocytopenia. Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-4142-6_12.

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4

Lee, A., and H. Mitchell. "Basic bacteriology of H. pylori: H. pylori colonization factors." In Helicobacter pylori. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1418-9_4.

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Figura, N., and J. E. Crabtree. "H. pylori vacuolating toxin." In Helicobacter pylori. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1418-9_19.

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Mégraud, F. "H. pylori species heterogeneity." In Helicobacter pylori. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1418-9_2.

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Salaspuro, M. "H. pylori alcohol dehydrogenase." In Helicobacter pylori. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1418-9_20.

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Graham, D. Y., and R. M. Genta. "Reinfection with H. pylori." In Helicobacter pylori. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1418-9_8.

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Rübe, Claudia E., Bernadine R. Donahue, Jay S. Cooper, et al. "Helicobacter (H.) pylori." In Encyclopedia of Radiation Oncology. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-540-85516-3_367.

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Theisler, Charles. "H. Pylori Infection." In Adjuvant Medical Care. CRC Press, 2022. http://dx.doi.org/10.1201/b22898-164.

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Trabalhos de conferências sobre o assunto "H. pylori"

1

Rangel, Gabriela Flores, Lorena Diaz de León Martinez, and Boris Mizaikoff. "Non-Invasive Helicobacter Pylori Detection via Mid-Infrared iHWG Sensor." In Latin America Optics and Photonics Conference. Optica Publishing Group, 2024. https://doi.org/10.1364/laop.2024.tu4a.31.

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Helicobacter pylori infection is linked to gastrointestinal disorders, including gastric cancer, necessitating efficient detection technologies. This study evaluates substrate-integrated hollow waveguide (iHWG) sensors using mid-infrared Fourier-transform infrared (FTIR) spectroscopy for precise isotopic ratio measurement of ¹³CO₂ to ¹²CO₂ in simulated breath analysis conditions. Three iHWGs of varying lengths were tested, optimizing the gas-light interaction to enhance sensitivity. Results show high precision and stability, establishing the FTIR-iHWG system as a promising platform for non-inv

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2

Yuan, Sishen, Baijia Liang, Po Wa Wong, et al. "Magnetic-Guided Flexible Origami Robot toward Long-Term Phototherapy of H. pylori in the Stomach." In 2024 IEEE International Conference on Robotics and Automation (ICRA). IEEE, 2024. http://dx.doi.org/10.1109/icra57147.2024.10611027.

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Kittner, JM, L. Volksdorf, CC Adarkwah, and M. Menges. "H. pylori-Eradikation: Versorgungsrealität in Deutschland." In Viszeralmedizin 2021 Gemeinsame Jahrestagung Deutsche Gesellschaft für Gastroenterologie, Verdauungs- und Stoffwechselkrankheiten (DGVS), Sektion Endoskopie der DGVS, Deutsche Gesellschaft für Allgemein und Viszeralchirurgie (DGAV). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1733507.

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Koumoutsos, Ioannis, John Klein, Elisabeta Compot, Sungjae Hwang, Alida Finze, and Giovanni Tritto. "PTU-062 “The only good H. pylori is a dead H. pylori” – challenges in isolation and eradication." In British Society of Gastroenterology Annual Meeting, 17–20 June 2019, Abstracts. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2019. http://dx.doi.org/10.1136/gutjnl-2019-bsgabstracts.278.

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Atikov, A. S. "THE ASSOCIATION OF LIVER PATHOLOGIES WITH THE CONTAMINATION OF THE GASTROINTESTINAL TRACT WITH H.PYLORI." In SPbVetScience. FSBEI HE St. Petersburg SUVM, 2023. http://dx.doi.org/10.52419/3006-2023-11-9-16.

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Currently, one of the most promising areas in bacteriology is the study of the bacterium Helicobacter pylori. New studies show a link between H. pylori and some liver diseases, but their exact mechanisms and connections are still not fully understood. Despite this, studies of H. pylori and its association with liver diseases remain relevant to this day. This article presents an analysis of the association of H. Pylori with liver diseases.

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Шишкина, Виктория, Светлана Клочкова, Наталия Алексеева та ін. "Морфофункциональные особенности тучных клеток при инфицировании Helicobacter pylori". У Международная научная и методическая конференция, посвященная году фундаментальных наук: "Современные аспекты морфологии, патоморфологии и онкопатологии организма человека". ФГБОУ ВО КГМУ Минздрава России, 2022. http://dx.doi.org/10.21626/cb.22.humanmorphology/32.

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Одними из ключевых промоторов в регуляции каскада воспалительных медиаторов являются тучные клетки, обладающие широким арсеналом биологически активных веществ. В патогенезе гастрита, ассоциированного с H. pylori принимают активное участие различные виды иммунокомпетентных клеток - макрофаги, нейтрофилы, эозинофилы, дендритные клетки, Т- и В-лимфоциты, а также тучные клетки. Антигенпредставляющие свойства тучных клеток представляют интерес в аспекте взаимодействия с Helicobacter pylori (H. Pylori)

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Çavuş, B., T. Çavuş, B. Akyüz Erdoğan, and E. Kumcu. "ERYTHROCYTE DISTRIBUTION WIDTH (RDW) AND HELICOBACTER PYLORI (H. PYLORI) INFECTION: IS THERE AN ASSOCIATION?" In ESGE Days 2018 accepted abstracts. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1637463.

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Ferreira, Shirley Cristina Reis, Luísa Neiva Araújo, Sérgio Rubens Lacerda Morais, Maria Fernanda Soares Batista, and Silvia Fernandes Ribeiro da Silva. "MÉTODOS DIAGNÓSTICOS DE INFECÇÃO POR H. PYLORI: UMA REVISÃO LITERÁRIA." In I Congresso Brasileiro de Doenças Infectocontagiosas On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/2226.

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Introdução: A infecção pela bactéria Helicobacter pylori (H. pylori) é mundialmente frequente e relaciona-se a doenças, como dispepsia, úlcera péptica e neoplasias gástricas. É um bastonete espiralado, multiflagelado e gram negativo, que provoca a destruição gradativa das camadas gástricas e duodenais. Objetivo: Revisar os principais métodos que identificam a presença da bactéria H. pylori no TGI. Material e métodos: Este resumo foi realizado com base em uma pesquisa bibliográfica qualitativa exploratória feita nas bases de dados PubMed e SciELO utilizando H. pylori, pathogenicity e diagnosis

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M.Hussein, Ali, Nadir Nanakali, and Mohammed M.Hussein. "EFFECT OF HYPERICUM PERFORATUM ON GASTRIC ULCER IN RAT." In 4th International Conference on Biological & Health Sciences (CIC-BIOHS’2022). Cihan University, 2022. http://dx.doi.org/10.24086/biohs2022/paper.742.

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Gastric ulcer is a chronic condition that occurs when the mucosa of the stomach is broken. There is a physiological equilibrium between aggressive factors and mucosal defense. This study aimed to determine the prevention level and efficiency of herbal medicinal plants (Hypericum perforatum) and compared with the omeprazole drug.Many groups were prepared from Albino male rats, first control group (inoculate with H. pylori and fed with standard pellet), Second group, rats inoculated by H. pylori and prevented with aqueous extract H. perforatum in two dosages (250mg/kg, 500mg/kg), Third group ino

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Devyatko, A. O., T. A. Noskova, and N. V. Shevchenko. "EXPERIENCE OF USING THE TEST SYSTEM «GASTROPANEL» IN THE PREVENTIVE MEDICAL EXAMINATION OF WORKFORCE IN JSC RZD." In The 16th «OCCUPATION and HEALTH» Russian National Congress with International Participation (OHRNC-2021). FSBSI “IRIOH”, 2021. http://dx.doi.org/10.31089/978-5-6042929-2-1-2021-1-173-176.

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Abstract: Non-invasive diagnosis of the gastric mucosa was carried out in 181 people in the preventive medical examination of workforce in JSC RZD. A set of diagnostics from Vector-Best (Novosibirsk, Russia) was used for enzyme immunoassay of pepsinogen I, pepsinogen II, and immunoglobulins for Helicobacter pylori. The prevalence of H. pylori infection in more than half of the employees, the presence of deviations of biomarkers from the norm in more than one in three employees were revealed. Laboratory signs of atrophy were detected eight times less frequently than signs of inflammation of the

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Relatórios de organizações sobre o assunto "H. pylori"

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Tumwasorn, Somying, and Versalovic, James. Identification of probiotic lactobacillus with anti-inflammatory activity in the gastrointestinal tract. Chulalongkorn University, 2009. https://doi.org/10.58837/chula.res.2009.15.

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In order to identify probiotic Lactobacillus for potential use as immunoprobiotics for the amelioration or treatment of inflammatory diseases in the gastrointestinal tract such as Helicobacter pylori-induced gastro-duodenal diseases and Clostridium difficile-associated disease (CDAD), we chose to test for the ability of human-derived Lactobacillus to modulate interleukin-8 (IL8) production in gastrointestinal epithelial cells. Eighty-seven gastric biopsy-derived Lactobacillus isolates were tested for their ability to modulate IL-8 production in H.pylori-stimulated AGS cells and 37 feces-derive

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